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Artigos de revistas sobre o tema "Etiology"

Autor: Grafiati
Publicado: 4 de junho de 2021
Última modificação: 29 de julho de 2024

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1

Pratama, Bangkit Ary, e Wahyuni. "LITERATURE REVIEW: IDENTIFIKASI PENYEBAB HEPATITIS AKUT TANPA ETIOLOGI PADA ANAK". KOSALA : Jurnal Ilmu Kesehatan 10, n.º 2 (17 de maio de 2022): 63–75. http://dx.doi.org/10.37831/kjik.v10i2.233.

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Kasus hepatitis akut tanpa etiologi yang menyerang anak-anak telah dikaitkan dengan beberapa kemungkinan etiologi termasuk penyakit coronavirus 2019 (COVID-19), vaksinasi COVID-19, agen infeksi baru, racun, dan kemungkinan etiologi terkait makanan. Masih perlu banyak penelitian yang dilakukan untuk secara tepat mengidentifikasi agen penyebab hepatitis akut tanpa etiologi. Penelitian ini bertujuan untuk mengidentifikasi kemungkinan penyebab hepatitis akut tanpa etiologi pada anak. Penelitian ini menggunakan kajian literatur (literature review). Sumber data pada penelitian ini adalah jurnal ilmiah yang tersedia di PubMed dengan tema hepatitis akut tanpa etiologi pada anak. Berdasarkan pencarian jurnal menggunaan database di PubMed dengan menggunakan kata kunci “acute hepatitis of unknown aetiology” dan “children”, didapatkan 14 jurnal dan hanya 4 jurnal saja yang memenuhi kriteria dan selanjutnya dianalisis. Hasil penelitian menunjukkan terdapat beberapa kemungkinan penyebab kasus hepatitis akut tanpa etiologi meliputi infeksi Human Adenovirus 41 Subtipe F (HAdV41-F), infeksi coronavirus–2 (SARS-CoV-2), vaksinasi COVID-19, infeksi virus lainnya, dan faktor non-infeksi. Kesimpulan pada penelitian ini adalah etiologi hepatitis akut tanpa etiologi pada anak-anak belum dapat ditentukan sehingga perlu adanya penelitian lebih lanjut. Kata kunci : anak, hepatitis akut tanpa etiologi Cases of acute hepatitis with no etiology affecting children have been associated with several possible etiologies including coronavirus disease 2019 (COVID-19), COVID-19 vaccination, new infectious agents, toxins, and possible food-related etiologies. More research was needed to accurately identify the causative agent of acute hepatitis without an etiology. This study aimed to identify possible causes of acute hepatitis without etiology in children. This study used a literature review. The data sources in this study were scientific journals available on PubMed with the theme of acute hepatitis without etiology in children. Based on a journal search using the PubMed database using the keywords “acute hepatitis of unknown aetiology” and “children”, 14 journals were obtained and only 4 journals met the criteria and were then analyzed. The results showed that there were several possible causes of acute hepatitis cases without etiology including Human Adenovirus 41 Subtype F (HAdV41-F) infection, coronavirus-2 (SARS-CoV-2) infection, COVID-19 vaccination, other viral infections, and other factors. non-infectious. The conclusion in this study is that the etiology of acute hepatitis without an etiology in children has not been determined so that further research is needed. Keywords: acute hepatitis of unknown aetiologi, children Korespondensi: Bangkit Ary Pratama, Poltekkes Bhakti Mulia, Jl. Solo-Sukoharjo No.KM. 9, Sukoharjo, Jawa Tengah, email: bangkit@poltekkesbhaktimulia.ac.id, 085326333050
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2

White, Keith C. "Etiology". Clinical Obstetrics and Gynecology 31, n.º 1 (março de 1988): 141–52. http://dx.doi.org/10.1097/00003081-198803000-00017.

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3

Crowther, Mark, e Jeffrey Weitz. "ETIOLOGY". Evidence-based Cardiovascular Medicine 3, n.º 2 (junho de 1999): 34–35. http://dx.doi.org/10.1054/ebcm.1999.0207.

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4

Field, Teresa. "ETIOLOGY". Evidence-based Oncology 1, n.º 2 (junho de 2000): 44–45. http://dx.doi.org/10.1054/ebon.2000.0021.

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5

White, John W. "Etiology". Journal of Burn Care & Rehabilitation 7, n.º 5 (setembro de 1986): 446. http://dx.doi.org/10.1097/00004630-198609000-00037.

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6

Morgan, Rachel. "Etiology". Prairie Schooner 91, n.º 2 (2017): 116. http://dx.doi.org/10.1353/psg.2017.0028.

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7

Dr. Nupur Sah, Dr Nupur Sah, Dr Hemant Bhutani e Dr Priyadarshini Shetty. "Halitosis – Etiology and Diagnosis". Indian Journal of Applied Research 3, n.º 11 (1 de outubro de 2011): 352–54. http://dx.doi.org/10.15373/2249555x/nov2013/109.

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8

Vaish, Sunidhi, Sunil K Pollipalli e Gurumurthy DM. "Genetic Etiology of Stillbirths". International Journal of Science and Research (IJSR) 13, n.º 5 (5 de maio de 2024): 617–22. http://dx.doi.org/10.21275/sr24508190240.

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9

Nagai, Atsushi, e Kazutetsu Aoshiba. "2. Etiology". Nihon Naika Gakkai Zasshi 97, n.º 6 (2008): 1177–83. http://dx.doi.org/10.2169/naika.97.1177.

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10

Davidson, Joseph. "Bacterial Etiology". Journal of the American Dental Association 122, n.º 1 (janeiro de 1991): 28. http://dx.doi.org/10.14219/jada.archive.1991.0013.

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11

Gillison, M. "Viral etiology". Radiotherapy and Oncology 82 (fevereiro de 2007): S4. http://dx.doi.org/10.1016/s0167-8140(07)80017-x.

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12

Chen, Cynthia, Neil Kaushal, David M. Scher, Shevaun M. Doyle, John S. Blanco e Emily R. Dodwell. "Clubfoot Etiology". Journal of Pediatric Orthopaedics 38, n.º 8 (setembro de 2018): e462-e469. http://dx.doi.org/10.1097/bpo.0000000000001191.

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13

Wahlgren, H., W. Mortensson, M. Eriksson, Y. Finkel, M. Forsgren e M. Leinonen. "Radiological findings in children with acute pneumonia: age more important than infectious agent". Acta Radiologica 46, n.º 4 (julho de 2005): 431–36. http://dx.doi.org/10.1080/02841850510021238.

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Purpose: To evaluate whether radiological findings and healing time in children with pneumonia are correlated to etiologic agent. Material and Methods: A total of 346 children with radiologically verified acute pneumonia, and with accomplished serological tests for bacteria and viruses, were included in the study. Five etiological groups were analysed: children with bacterial etiology only, with viral etiology only, with mixed bacterial and viral etiology, with Mycoplasma only, and children with no etiology. Results: The chest films of each etiological group were analysed and the findings were correlated to the children's age. The radiological findings did not differ between the etiological groups. Radiological findings correlated significantly with the patient's age. The radiological healing frequency at check‐up X‐ray was found to be significantly lower in children with mixed bacterial and viral etiology compared to children in each of the other groups and to the material as a whole. Conclusion: Conclusions about the etiology could not be drawn from the chest X‐ray findings.
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14

Poedjiastoeti, Wiwiek. "Komunikasi oroantral: etiologi dan penatalaksanaannya Oroantral communication: the etiology and management". Journal of Dentomaxillofacial Science 10, n.º 2 (30 de junho de 2011): 116. http://dx.doi.org/10.15562/jdmfs.v10i2.267.

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Oroantral communication (OAC)may occur when upper posterior teeth are removed, and occasionally, as a resultof trauma. This sinus perforation formed particularly when a maxillary molar with widely divergent roots adjacentto edentulous spaces is extracted. In this instance the sinus is likely to be pneumatized into the edentulous alveolarprocessus surrounding the tooth, which weakens the entire alveolus and brings the tooth apices into a closerrelationship with the sinus cavity. In order to avoid OAC, preoperative radiograph is needed. When perforation, ifsinus opening is small and disease free, effort should be made to establish blood clot in the extraction site andpreserve it in place. Soft tissue flap elevation is not required. Sutures are placed to reposition the soft tissue, and agauze pack is placed over the surgical site for 1-2 hours. Majority of patients treated in this manner showeduneventful healing when no evidence of preexisting sinus disease.
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15

De Araújo, Daniel Alencar, Denise Teixeira Santos, Larisse Yara De Carvalho e Daniela Moura Parente. "Etiologia genética do aborto recorrente / Genetic etiology of recurrent abortion". Brazilian Journal of Health Review 4, n.º 6 (18 de novembro de 2021): 25673–77. http://dx.doi.org/10.34119/bjhrv4n6-166.

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16

Berdysh, D. S., V. I. Shaposhnikov e M. A. Maksimenko. "ETIOLOGY DEVELOPMENT VARICOTSEL". EurasianUnionScientists 4, n.º 63 (2019): 6–8. http://dx.doi.org/10.31618/esu.2413-9335.2019.4.63.207.

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17

Petersen, Hermod, e Mikko Salaspuro. "Etiology and pathophysiology". Scandinavian Journal of Gastroenterology 20, sup109 (janeiro de 1985): 49. http://dx.doi.org/10.3109/00365528509103936.

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18

Ohyama, M. "Etiology and Pathology." Nihon Kikan Shokudoka Gakkai Kaiho 48, n.º 2 (1997): 138–39. http://dx.doi.org/10.2468/jbes.48.138.

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19

Rubin, Harry. "Etiology of AIDS". Science 240, n.º 4858 (10 de junho de 1988): 1389–90. http://dx.doi.org/10.1126/science.240.4858.1389.d.

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20

Nazish, Zahra, Muhammad Inayatullah e Muhammad Younus Khan. "ETIOLOGY OF DYSPHAGIA". Professional Medical Journal 23, n.º 09 (10 de setembro de 2016): 1039–44. http://dx.doi.org/10.29309/tpmj/2016.23.09.1692.

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Objectives: To determine the etiology of dysphagia based on upper GI endoscopyin Nishtar Hospital Multan. Study design: Retrospective study. Place and Duration of study:This study was conducted at gastroenterology unit of Nishtar Hospital Multan from Feb 2013 toAugust 2014. Patients and methods: Three hundred and twenty three patients, ≥ 13 years old,who presented with history of dysphagia to the gastroenterology unit of Nishtar Hospital Multan.Results: Out of 323 patients, 43.7% were males and 56.3% were females. Mean age of patientswas 44.37±17.395 years. Most common finding was benign stricture (28.5% cases) followedby no abnormality (21.7%), carcinoma esophagus (20.7%), achalasia (6.5%), esophagealweb (4%), ulcers (3.7%), multiple pathologies (3.1%), pharyngeal cancer (2.2%), esophagealcandidiasis (1.9%), reflux esophagitis (1.5%) and hiatus hernia (1.2%). Uncommon findingswere incompetent LES (0.9%), extrinsic compression (0.9%), vocal cord paralysis (0.6%),barrett’s esophagus (0.6%), herpes simplex esophagitis (0.6%), shatzki ring (0.3%), diverticulum(0.3%) and thick aryepiglottic folds (0.3%.). Conclusion: Esophagogastroduodenoscopy is theinvestigation of choice for patients of dysphagia. Most common finding in our study was benignstricture in young females, followed by carcinoma esophagus, achalasia, web, ulcer, pharyngealcancer, reflux esophagitis, esophageal candidiasis and hiatus hernia. Incompetent LES,extrinsic compression, vocal cord paralysis, barrett esophagus, herpes simplex esophagitis,ring, diverticulum and thick aryepiglottic folds were rare causes. Measures should be taken toavoid the preventable causes by patient awareness and adequate treatment of predisposingfactors.
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21

Metzger, Deborah A., e A. F. Haney. "Etiology of Endometriosis". Obstetrics and Gynecology Clinics of North America 16, n.º 1 (março de 1989): 1–14. http://dx.doi.org/10.1016/s0889-8545(21)00134-0.

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22

Isbit, J. "Etiology of Diverticulosis". North Carolina Medical Journal 77, n.º 4 (1 de julho de 2016): 303. http://dx.doi.org/10.18043/ncm.77.4.303.

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23

Rubin, H. "Etiology of AIDS". Science 240, n.º 4858 (10 de junho de 1988): 1389–90. http://dx.doi.org/10.1126/science.240.4858.1389-c.

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24

Ahmetagic, Aldina, Fatima Numanovic, Sead Ahmetagic, Lejla RakovacTupkovic e Humera PorobicJahic. "Etiology of Peritonitis". Medical Archives 67, n.º 4 (2013): 278. http://dx.doi.org/10.5455/medarh.2013.67.278-281.

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25

Sullivan, L. P., J. C. Stears e S. P. Ringel. "Etiology of syringomyelia". Neurology 39, n.º 2 (1 de fevereiro de 1989): 308. http://dx.doi.org/10.1212/wnl.39.2.308.

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26

Milstein, J. M., e D. M. Olson. "Etiology of syringomyelia". Neurology 39, n.º 2 (1 de fevereiro de 1989): 308. http://dx.doi.org/10.1212/wnl.39.2.308-a.

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27

Pitts, Susan L. "Etiology of Blindness". Public Library Quarterly 18, n.º 3-4 (junho de 2000): 81–93. http://dx.doi.org/10.1300/j118v18n03_11.

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28

Grobler, Leon J., Peter A. Robertson, John E. Novotny e Malcolm H. Pope. "Etiology of Spondylolisthesis". Spine 18, n.º 1 (janeiro de 1993): 80–91. http://dx.doi.org/10.1097/00007632-199301000-00013.

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29

Nanay, Bence. "Teleosemantics without Etiology". Philosophy of Science 81, n.º 5 (dezembro de 2014): 798–810. http://dx.doi.org/10.1086/677684.

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30

&NA;. "Etiology and pathogenesis". Current Opinion in Rheumatology 3, n.º 1 (fevereiro de 1991): 32–35. http://dx.doi.org/10.1097/00002281-199102000-00007.

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31

DETORAKIS, ET. "Anatomy & Etiology". Acta Ophthalmologica 89, s248 (setembro de 2011): 0. http://dx.doi.org/10.1111/j.1755-3768.2011.1311.x.

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32

Rubin, H. "Etiology of AIDS". Science 240, n.º 4858 (10 de junho de 1988): 1389–90. http://dx.doi.org/10.1126/science.3163849.

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33

Thomas, Rhys, e Philip Smith. "Etiology of Epilepsy". Seminars in Neurology 35, n.º 03 (10 de junho de 2015): 191–92. http://dx.doi.org/10.1055/s-0035-1552626.

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34

Fuchs, Bruno, e Douglas J. Pritchard. "Etiology of Osteosarcoma". Clinical Orthopaedics and Related Research 397 (abril de 2002): 40–52. http://dx.doi.org/10.1097/00003086-200204000-00007.

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35

Bhatia, M. S., N. K. Dhar, P. K. Singhal, V. R. Nigam, S. C. Malik e D. N. Mullick. "Prevalence and Etiology". Clinical Pediatrics 29, n.º 6 (junho de 1990): 311–15. http://dx.doi.org/10.1177/000992289002900603.

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36

Cammisa, Kathryne M., e Stephen H. Hobbs. "Etiology of Autism". Occupational Therapy in Mental Health 12, n.º 2 (4 de junho de 1993): 39–67. http://dx.doi.org/10.1300/j004v12n02_03.

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37

Panel, J. P. Mohr, Gregory W. Albers, Pierre Amarenco, Viken L. Babikian, José Biller, Robin L. Brey et al. "Etiology of Stroke". Stroke 28, n.º 7 (julho de 1997): 1501–6. http://dx.doi.org/10.1161/01.str.28.7.1501.

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38

Tomatsu, Shunji, Shaukat Khan, Hira Peracha, Alfred Wiesbauer e Tadao Orii. "Etiology of mucopolysaccharidoses". Molecular Genetics and Metabolism 120, n.º 1-2 (janeiro de 2017): S132. http://dx.doi.org/10.1016/j.ymgme.2016.11.347.

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39

Sharma, Om, e Darshana Kadakia. "Etiology of Sarcoidosis". Seminars in Respiratory and Critical Care Medicine 8, n.º 01 (julho de 1986): 95–102. http://dx.doi.org/10.1055/s-2007-1012644.

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40

Meares, E. "Etiology of Prostatitis". Journal of Urology 133, n.º 6 (junho de 1985): 1126. http://dx.doi.org/10.1016/s0022-5347(17)49394-3.

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41

Edwards, I. Keith. "Endometriosis Etiology Questioned". AORN Journal 49, n.º 2 (fevereiro de 1989): 478. http://dx.doi.org/10.1016/s0001-2092(07)66666-6.

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42

Redwine, David B. "Endometriosis Etiology Questioned". AORN Journal 49, n.º 2 (fevereiro de 1989): 478. http://dx.doi.org/10.1016/s0001-2092(07)66667-8.

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43

Kelly, Ellen M. "Modeling stuttering etiology". Journal of Fluency Disorders 25, n.º 4 (dezembro de 2000): 359–68. http://dx.doi.org/10.1016/s0094-730x(00)00085-1.

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44

Sanders, Richard J., e Sharon L. Hammond. "Etiology and pathology". Hand Clinics 20, n.º 1 (fevereiro de 2004): 23–26. http://dx.doi.org/10.1016/s0749-0712(03)00079-9.

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45

Neidner, Kenneth H. "Etiology and pathogenesis". Clinics in Dermatology 6, n.º 1 (janeiro de 1988): 123–35. http://dx.doi.org/10.1016/0738-081x(88)90018-1.

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46

Carras, Porto. "Etiology of insomnia". International Journal of Psychophysiology 18, n.º 2 (novembro de 1994): 155–56. http://dx.doi.org/10.1016/0167-8760(94)90474-x.

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47

Bonnet, Michael. "Etiology of insomnia". Sleep Medicine 7, n.º 7 (outubro de 2006): 544. http://dx.doi.org/10.1016/j.sleep.2006.05.012.

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48

Moller, David R. "ETIOLOGY OF SARCOIDOSIS". Clinics in Chest Medicine 18, n.º 4 (dezembro de 1997): 695–706. http://dx.doi.org/10.1016/s0272-5231(05)70413-5.

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Meeuwisse, Willem H. "Athletic Injury Etiology". Clinical Journal of Sport Medicine 4, n.º 3 (julho de 1994): 171–75. http://dx.doi.org/10.1097/00042752-199407000-00005.

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Raveenthiran, V. "Etiology of Gastroschisis". Journal of Neonatal Surgery 1, n.º 4 (1 de outubro de 2012): 53. http://dx.doi.org/10.47338/jns.v1.4.

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