Literatura científica selecionada sobre o tema "Embryons non-C. elegans"
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Artigos de revistas sobre o assunto "Embryons non-C. elegans"
Nance, Jeremy, e James R. Priess. "Cell polarity and gastrulation inC. elegans". Development 129, n.º 2 (15 de janeiro de 2002): 387–97. http://dx.doi.org/10.1242/dev.129.2.387.
Texto completo da fonteOlson, Sara K., Joseph R. Bishop, John R. Yates, Karen Oegema e Jeffrey D. Esko. "Identification of novel chondroitin proteoglycans in Caenorhabditis elegans: embryonic cell division depends on CPG-1 and CPG-2". Journal of Cell Biology 173, n.º 6 (19 de junho de 2006): 985–94. http://dx.doi.org/10.1083/jcb.200603003.
Texto completo da fonteSchroeder, D. F., e J. D. McGhee. "Anterior-posterior patterning within the Caenorhabditis elegans endoderm". Development 125, n.º 24 (15 de dezembro de 1998): 4877–87. http://dx.doi.org/10.1242/dev.125.24.4877.
Texto completo da fonteVan Auken, Kimberly, Daniel Weaver, Barbara Robertson, Meera Sundaram, Tassa Saldi, Lois Edgar, Ulrich Elling, Monica Lee, Queta Boese e William B. Wood. "Roles of the Homothorax/Meis/Prep homolog UNC-62 and the Exd/Pbx homologs CEH-20 and CEH-40 in C. elegans embryogenesis". Development 129, n.º 22 (15 de novembro de 2002): 5255–68. http://dx.doi.org/10.1242/dev.129.22.5255.
Texto completo da fonteSchierenberg, Einhard. "Early development of nematode embryos: differences and similarities". Nematology 2, n.º 1 (2000): 57–64. http://dx.doi.org/10.1163/156854100508890.
Texto completo da fonteCoomans, August, Myriam Claeys, Gaëtan Borgonie e Christopher Link. "Lysosomal and pseudocoelom routing protects Caenorhabditis elegans from ricin toxicity". Nematology 5, n.º 3 (2003): 339–50. http://dx.doi.org/10.1163/156854103769224331.
Texto completo da fonteFerreira, Helder C., Benjamin D. Towbin, Thibaud Jegou e Susan M. Gasser. "The shelterin protein POT-1 anchors Caenorhabditis elegans telomeres through SUN-1 at the nuclear periphery". Journal of Cell Biology 203, n.º 5 (2 de dezembro de 2013): 727–35. http://dx.doi.org/10.1083/jcb.201307181.
Texto completo da fonteLabouesse, M., E. Hartwieg e H. R. Horvitz. "The Caenorhabditis elegans LIN-26 protein is required to specify and/or maintain all non-neuronal ectodermal cell fates". Development 122, n.º 9 (1 de setembro de 1996): 2579–88. http://dx.doi.org/10.1242/dev.122.9.2579.
Texto completo da fonteOsório, Daniel S., Fung-Yi Chan, Joana Saramago, Joana Leite, Ana M. Silva, Ana F. Sobral, Reto Gassmann e Ana Xavier Carvalho. "Crosslinking activity of non-muscle myosin II is not sufficient for embryonic cytokinesis in C. elegans". Development 146, n.º 21 (3 de outubro de 2019): dev179150. http://dx.doi.org/10.1242/dev.179150.
Texto completo da fonteKowalski, M. P., H. A. Baylis e T. Krude. "Non-coding stem-bulge RNAs are required for cell proliferation and embryonic development in C. elegans". Journal of Cell Science 128, n.º 11 (23 de abril de 2015): 2118–29. http://dx.doi.org/10.1242/jcs.166744.
Texto completo da fonteTeses / dissertações sobre o assunto "Embryons non-C. elegans"
Samandar, eweis Dureen. "Asymmetric division in single cell nematode embryos outside the Caenorhabditis genus". Electronic Thesis or Diss., Université Paris sciences et lettres, 2021. http://www.theses.fr/2021UPSLS063.
Texto completo da fonteAsymmetric cell division is an essential process of development. The process and its regulation have been studied extensively in the Caenorhabditis elegans embryo. Asymmetric division of the single-cell embryo is a conserved process in nematode species, however, the cellular features leading up to division are surprisingly variable. During my PhD, I aimed to study these differences by using two non-C. elegans embryos: Diploscapter pachys and Pristionchus pacificus. D. pachys is the closest parthenogenetic relative to C. elegans. Since the polarity cue in C. elegans is brought by the sperm, how polarity is triggered in D. pachys remains unknown. My results show that the nucleus inhabits principally the hemisphere of the D. pachys embryo that will become the posterior pole. Moreover, in embryos where the nucleus is forced to one pole by centrifugation, it returns to its preferred pole. Although the embryo is polarized, cortical ruffling and actin cytoskeleton at both poles appear identical. Interestingly, the location of the meiotic spindle also correlates with the future posterior cell. In some oocytes, a slight actin enrichment along with unusual microtubule structures emanating from the meiotic spindle are observed at the future posterior pole. Overall, my main PhD project shows that polarity of the D. pachys embryo is attained during meiosis wherein the meiotic spindle could potentially be playing a role by a mechanism that may be present but suppressed in C. elegans. For P. pacificus, biolistic transgenesis has been shown recently successful. However, due to a lack of a stringent selection marker, the continuation of this project was unfeasible during my PhD. Altogether, the results of my PhD add to the understanding of non-C. elegans early embryogenesis and emphasizes on the importance of using these species for comparative studies
Molnar, Kelly. "Contribution of non-muscle myosins to C. elegans embryonic elongation". Electronic Thesis or Diss., Sorbonne université, 2023. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2023SORUS091.pdf.
Texto completo da fonteThe morphogenesis in the C. elegans embryo is characterized by a four-fold elongation, which occurs without any cell division or intercalation. This process of cell-shape change occurs in two distinct stages, the second of which is triggered by an initial mechanical input from the muscles. Both stages require actomyosin in the epidermis. This work is an investigation of the second stage, especially the interplay between the muscles and the epidermis, and the precise role of the two non-muscle myosins NMY-1 and NMY-2. This pair of molecular motors is essential for the late stage elongation, their inhibition using temperature sensitive mutants having been shown to cause immediate arrest, despite continued mechanical input from the muscles. Furthermore, after arrest, the myosins can return to their functional state and elongation is able to resume. Inactive myosin motors also have been shown here to form aggregates in the epidermis. It is likely that this myosin pair is responsible for pulling circumferential actin cables in the epidermis towards one another, this providing the force necessary for elongation
Capítulos de livros sobre o assunto "Embryons non-C. elegans"
Schnabel, Ralf. "Microscopy". In C.elegans, 119–42. Oxford University PressOxford, 1999. http://dx.doi.org/10.1093/oso/9780199637393.003.0007.
Texto completo da fonteBurggren, Warren W. "Complexity Change during Physiological Development". In Comparative Developmental Physiology, 174–90. Oxford University PressNew York, NY, 2006. http://dx.doi.org/10.1093/oso/9780195168594.003.0012.
Texto completo da fonte