Bibliografias temáticas / Drosophila

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Literatura científica selecionada sobre o tema "Drosophila"

Autor: Grafiati
Publicado: 4 de junho de 2021
Última modificação: 30 de julho de 2024

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Artigos de revistas sobre o assunto "Drosophila"

1

BAŞPINAR, Hüseyin, Tülin AKŞİT, Alper KESİCİ, Ferenc DEUTSCH, Balazs KİSS e Laszlo PAPP. "Aydın İli (Türkiye) meyve bahçelerindeki Drosophilidae (Diptera) familyası türlerinin mevsimsel yoğunlukları ve tür çeşitliliği ve birlikte saptanan diğer Diptera türleri". Turkish Journal of Entomology 46, n.º 3 (1 de setembro de 2022): 289–98. http://dx.doi.org/10.16970/entoted.1088263.

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Bu çalışmada Aydın İli’ndeki üç meyve bahçesinde Drosophilidae (Diptera) familyası türlerinin belirlenmesi ve bunların mevsimsel yoğunluklarının araştırılması amaçlanmıştır. Aynı zamanda çalışmada saptanan diğer diptera türleri de incelenmiştir. Çalışmalar Eylül 2018-Ocak 2020 tarihleri arasında içerisinde üzüm sirkesi bulunan besin cezbedici tuzaklar kullanılarak yürütülmüştür. Çalışma sonunda, tuzaklarda 11 Drosophilidae türü ve ayrıca yedi familyadan 10 farklı sinek türü belirlenmiştir. Drosophilidae türlerinden Drosophila subobscura Collin, 1936 toplam 1 964 birey olarak çalışma bahçelerinde belirlenmiş ve en çok yakalanan tür olmuştur. Bunu sayısal olarak Drosophila immigrans Sturtevant, 1921, Drosophila melanogaster Meigen, 1830, Zaprionus tuberculatus Malloch, 1932 ve Drosophila suzukii Matsumura, 1931 izlemiştir. Bahçelerde en çok drosophilid 1 836 birey ile Nisan (2019) ayında elde edilmiştir. Drosophilid türleri sayısal olarak birlikte dikkate alındığında, mevsimsel dalgalanmalar göstermiş olup, bunlardan ilk tepe noktası Nisan (2019) ayında ve ikincisi Kasım-Aralık (2019) aylarında ortaya çıkmıştır. Ancak, drosophilid türleri yaz ayları süresince oldukça düşük sayılarda tuzaklara yakalanmıştır. Çalışmada saptanan Aulacigaster falcata Papp, 1997 (Diptera: Aulacigastridae) Türkiye faunası için ilk kayıt niteliğindedir.
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Hotimah, Husnul, Purwatiningsih Purwatiningsih e Kartika Senjarini. "Morphological Description of Drosophila melanogaster Wild Type (Diptera:Drosophilidae), Sepia and Plum Strain". Jurnal ILMU DASAR 18, n.º 1 (1 de fevereiro de 2017): 55. http://dx.doi.org/10.19184/jid.v18i1.3113.

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Drosophila melanogaster is one of the insects which have a very important role in the development of the genetic science. Drosophila melanogaster have many mutation, recently there are many mutant such as sepia and plum strain. Morgan et al have found 85 strain mutan of Drosophila melanogaster. The purpose of these research was to characterize of morphological from the head, thorax, and abdomen. The result show that the morphological of Drosophilla melanogaster wild type, sepia and plum mutant have many similarities. The difference of them are the eyes color, Drosophila melanogaster wild type has red eye, sepia strain has a dark brown eye and plum has a dark purple eye.
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3

VAN DER LINDE, KIM, DAVID HOULE, GREG S. SPICER e SCOTT J. STEPPAN. "A supermatrix-based molecular phylogeny of the family Drosophilidae". Genetics Research 92, n.º 1 (fevereiro de 2010): 25–38. http://dx.doi.org/10.1017/s001667231000008x.

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SummaryThe genus Drosophila is diverse and heterogeneous and contains a large number of easy-to-rear species, so it is an attractive subject for comparative studies. The ability to perform such studies is currently compromised by the lack of a comprehensive phylogeny for Drosophila and related genera. The genus Drosophila as currently defined is known to be paraphyletic with respect to several other genera, but considerable uncertainty remains about other aspects of the phylogeny. Here, we estimate a phylogeny for 176 drosophilid (12 genera) and four non-drosophilid species, using gene sequences for up to 13 different genes per species (average: 4333 bp, five genes per species). This is the most extensive set of molecular data on drosophilids yet analysed. Phylogenetic analyses were conducted with maximum-likelihood (ML) and Bayesian approaches. Our analysis confirms that the genus Drosophila is paraphyletic with 100% support in the Bayesian analysis and 90% bootstrap support in the ML analysis. The subgenus Sophophora, which includes Drosophila melanogaster, is the sister clade of all the other subgenera as well as of most species of six other genera. This sister clade contains two large, well-supported subclades. The first subclade contains the Hawaiian Drosophila, the genus Scaptomyza, and the virilis-repleta radiation. The second contains the immigrans-tripunctata radiation as well as the genera Hirtodrosophila (except Hirtodrosophila duncani), Mycodrosophila, Zaprionus and Liodrosophila. We argue that these results support a taxonomic revision of the genus Drosophila.
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Boycheva Woltering, Svetlana, Jörg Romeis e Jana Collatz. "Influence of the Rearing Host on Biological Parameters of Trichopria drosophilae, a Potential Biological Control Agent of Drosophila suzukii". Insects 10, n.º 6 (25 de junho de 2019): 183. http://dx.doi.org/10.3390/insects10060183.

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Trichopria drosophilae is a pupal parasitoid that can develop in a large number of drosophilid host species including the invasive pest Drosophila suzukii, and is considered a biological control agent. We investigated the influence of the rearing host on the preference and performance of the parasitoid, using two different strains of T. drosophilae, reared on D. melanogaster or D. suzukii for approximately 30 generations. Host switching was employed to assess the impact of host adaptation on T. drosophilae performance. In a no-choice experimental setup, T. drosophilae produced more and larger offspring on the D. suzukii host. When given a choice, T. drosophilae showed a preference towards D. suzukii, and an increased female ratio on this host compared to D. melanogaster and D. immigrans. The preference was independent from the rearing host and was confirmed in behavioral assays. However, the preference towards D. suzukii increased further after a host switch from D. melanogaster to D. suzukii in just one generation. Our data indicate that rearing T. drosophilae for several years on D. melanogaster does not compromise its performance on D. suzukii in the laboratory. However, producing a final generation on D. suzukii prior to release could increase its efficacy towards the pest.
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Karaningannavar, Shwetha, Rajat Hegde e Ramesh Babu Yarajarla. "Simple and Rapid PCR-RFLP based species identification in Drosophila suzukii and Drosophila immigrans larvae". Research Journal of Biotechnology 19, n.º 3 (31 de janeiro de 2024): 48–50. http://dx.doi.org/10.25303/1903rjbt048050.

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Wild fruit fly larvae can be distinguished at the family level such as Drosophilidae and Tephritidae larvae are based on size or spiracles arrangement, but such visual distinction is more intricate between Drosophilid species. In the present study, to identify the Drosophila immigrans and Drosophila suzukii species at the larval stage, we employed a simple, rapid PCR-RFLP experiment. Wild suspected larvae from Drosophila immigrans and Drosophila suzukii species were collected from 3 different geographical regions. DNA was isolated and a PCR-RFLP profile was developed using the XmnI restriction enzyme. Sequencing of the mitochondrial COI gene from larvae was performed to evaluate the species. PCR-RFLP analysis from Drosophila suzukii wild larvae generated three distinct bands with 120bp, 210bp and 250bp. Similarly, Drosophila immigrans generated 2 distinct bands with 270bp and 310bp. Restriction digestion products from both the larvae and adult flies were the same, hence the accuracy of the PCR-RFLP profile developed from both suspected wild larvae and adult flies was 100%. Sequencing analysis confirms that wild field collected larvae belong to Drosophila suzukii and Drosophila immigrans. Present study clearly shows that PCR‐RFLP is a fast, simple, inexpensive and accurate molecular method and it can be successfully used for species identification at the early stages of the life cycle of Drosophila species.
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Liu, Xuxiang, Yongbang Yang, Qingwen Fan, Qinyuan Zhang e Qinge Ji. "Effect of Ultraviolet-B Radiating Drosophila melanogaster as Host on the Quality of Trichopria drosophilae, a Pupal Parasitoid of Drosophila suzukii". Insects 14, n.º 5 (28 de abril de 2023): 423. http://dx.doi.org/10.3390/insects14050423.

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The pupal parasitoid, Trichopria drosophilae Perkins (Hymenoptera: Diapriidae), is an ectoparasitoid of the genus Drosophila with great potential for application in biological control based on its excellent control efficiency for Drosophila suzukii Matsumura (Diptera: Drosophilidae), and it has has even been commercialized by biofactories. Due to its characteristics of short life cycle, large number of offspring, easy rearing, rapid reproduction, and low cost, Drosophila melanogaster (Diptera: Drosophilidae) is currently being utilized as a host to mass produce T. drosophilae. To simplify the mass rearing process and omit the separation of hosts and parasitoids, ultraviolet-B (UVB) was used as an irradiation source to irradiate D. melanogaster pupae, and the effects on T. drosophilae were studied. The results showed that UVB radiation significantly reduces host emergence and affects the duration of parasitoid development (female: F0 increased from 21.50 to 25.80, F1 from 23.10 to 26.10; male: F0 decreased from 17.00 to 14.10, F1 from 17.20 to 14.70), which has great significance for the separation of hosts and parasitoids as well as of females and males. Of the various studied conditions, UVB irradiation was ideal when the host was supplied with parasitoids for 6 h. The selection test results showed that the female-to-male ratio of emerging parasitoids in this treatment was highest at 3.47. The no-selection test resulted in the highest rates of parasitization and parasitoid emergence rate, maximized inhibition of host development, and allowed the omission of the separation step. Finally, the results of the semi-field test showed that the parasitoids bred in this treatment could search for their hosts normally and could therefore be directly applied in the biological control of Drosophila pests in the field.
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Trivellone, Valeria, Michela Meier, Corrado Cara, Lucia Pollini Paltrinieri, Felix Gugerli, Marco Moretti, Sarah Wolf e Jana Collatz. "Multiscale Determinants Drive Parasitization of Drosophilidae by Hymenopteran Parasitoids in Agricultural Landscapes". Insects 11, n.º 6 (30 de maio de 2020): 334. http://dx.doi.org/10.3390/insects11060334.

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(1) The management of agricultural landscapes for pest suppression requires a thorough understanding of multiple determinants controlling their presence. We investigated the ecological preferences of indigenous parasitoids and their drosophilid hosts to understand the role of native parasitoids as biological control agents of the invasive frugivorous Drosophila suzukii. (2) Using data from an extensive field survey across different habitat types we analyzed the influence of abiotic and biotic factors on parasitoid and drosophilid communities at multiscale levels. (3) Eight parasitoid and 27 drosophilid species were identified. Thirty-four percent variation in drosophilid communities was explained by factors at the landscape scale, and 52% of significant variation of parasitoids by local distribution of three drosophilid species, mainly collected in woodland. Parasitoid communities were significantly influenced by microhabitat type (ground versus canopy) rather than habitat type. All parasitoids except Pachycrepoideus vindemmiae preferred the ground microhabitat. All parasitoids, with the exception of Trichopria drosophilae and Spalangia erythromera, displayed significant preferences among the drosophilid species used in the baited traps. (4) Since they can tolerate a broad range of habitat factors, altogether pupal parasitoids investigated in this study could play a role in biological control programs to suppress D. suzukii, but non-target effects have to be regarded.
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Chassagnard, Μ. Τ., e L. Tsacas. "Drosophila schmidti Duda: redescription et iconographie des genitalia (Diptera, Drosophilidae)". ENTOMOLOGIA HELLENICA 5 (31 de maio de 2017): 69. http://dx.doi.org/10.12681/eh.13950.

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Drosophila schmidti Duda, n’a jamais été retrouvée depuis sa description. Les genitalia, structure diagnostique importante, n’ont jamais été iconographiês. Est donnée ici une re­description détaillée de l’espêw avec des dessins des genitalia et de l’ovipositeur. La posi­tion de cette espèce par rapport aux divers sous-genres du genre Drosophile est discutée.
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Buonocore Biancheri, María Josefina, Segundo Ricardo Núñez-Campero, Lorena Suárez, Marcos Darío Ponssa, Daniel Santiago Kirschbaum, Flávio Roberto Mello Garcia e Sergio Marcelo Ovruski. "Implications of the Niche Partitioning and Coexistence of Two Resident Parasitoids for Drosophila suzukii Management in Non-Crop Areas". Insects 14, n.º 3 (23 de fevereiro de 2023): 222. http://dx.doi.org/10.3390/insects14030222.

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Understanding the mechanisms associated with the coexistence of competing parasitoid species is critical in approaching any biological control strategy against the globally invasive pest spotted-wing drosophila (=SWD), Drosophila suzukii (Matsumura). This study assessed the coexistence of two resident pupal parasitoids, Trichopria anastrephae Lima and Pachycrepoideus vindemiae Rondani, in SWD-infested fruit, in disturbed wild vegetation areas of Tucumán, northwestern Argentina, based on niche segregation. Drosophilid puparia were collected between December/2016 and April/2017 from three different pupation microhabitats in fallen feral peach and guava. These microhabitats were “inside flesh (mesocarp)”, “outside flesh”, but associated with the fruit, and “soil”, i.e., puparia buried close to fruit. Saprophytic drosophilid puparia (=SD) belonging to the Drosophila melanogaster group and SWD were found in all tested microhabitats. SD predominated in both inside and outside flesh, whereas SWD in soil. Both parasitoids attacked SWD puparia. However, T. anastrephae emerged mainly from SD puparia primarily in the inside flesh, whereas P. vindemiae mostly foraged SWD puparia in less competitive microhabitats, such as in the soil or outside the flesh. Divergence in host choice and spatial patterns of same-resource preferences between both parasitoids may mediate their coexistence in non-crop environments. Given this scenario, both parasitoids have potential as SWD biocontrol agents.
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Wilson, Carolyn. "A review of the monitoring and management of Spotted-Wing Drosophila (Drosophila suzukii) in lowbush blueberrie". Proceedings of the Nova Scotian Institute of Science (NSIS) 49, n.º 1 (30 de março de 2017): 145. http://dx.doi.org/10.15273/pnsis.v49i1.6984.

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The recent arrival of spotted-wing drosophilia (Drosophila suzukii) to eastern Canada is a major threat to the million dollar lowbush blueberry industry. The highly fecund female fly lays her eggs in ripe soft-skinned fruit. The maturing larvae consume the fruit interior, ultimately resulting in fruit collapse and decreased yield. Effective monitoring and management of this pest is essential for reducing the risk of export market closure and economic losses. In this literature review, an integrated pest management plan is developed that outlines current monitoring and management practices for the pest and considers preventative physical, chemical and biological controls.
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Mais fontes

Teses / dissertações sobre o assunto "Drosophila"

1

Le, Thomas Adrien. "Piwi function and piRNA cluster regulation : Drosophila melanogaster". Electronic Thesis or Diss., Paris 6, 2014. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2014PA066688.pdf.

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Les piRNAs sont une population de petit ARNs très diverse, que l'on retrouve dqns la lignée germinales des animaux pour réprimer les éléments génétiques mobiles : agissant de pair avec les protéines Piwi, ils guident le clivage des transposons actif. Chez la Drosophile, 3 protéines Piwi sont présentes, dont deux d'entre elles, AUB et AGO3, sont cytoplasmique et la dernière, PIWI, est nucléaire cependant son mécanisme d'action reste inconnu. La source principale de piRNAs sont des régions du génome bien particulière, appelé cluster de piRNAs. Cependant, il n'est pas encore connu a ce jour qu'est ce qui différentie ces région du reste du génome. Durant mon doctorant mon travail s'est focalisé sur ces deux questions centrales :Quel est le rôle de PIWI dans le noyau? Nous avons montré que PIWI était responsable de répression transcriptionnelle des transposons par l'intermédiaire de la déposition de marques chromatiniennes répressive, H3K9me3, grâce à la spécificité des piRNAs.Comment sont définit les régions générant des piRNA et comment sont régules leur expression ?Nous avons trouvé que les piRNAs qui sont transmis par la mère aux progénitures sont responsables de l'identification des régions génomiques donnant naissances à de nouveau piRNAs, grâce à la déposition de H3K9me3 dans le noyau et par l'initiation du cycle ping-pong dans le cytoplasme.Nous avons aussi mis en évidence les régions promoteurs des clusters de piRNAs, et trouve qu'elles sont nécessaires pour la production de piRNAs
PiRNAs are a diverse population of small RNA found in the animal germline to silence mobile genetic elements: loaded into Piwi proteins, they guide homology-dependent cleavage of active transposon mRNAs. In Drosophila, three Piwi proteins are expressed, from which two, AUB and AGO3, are known to destroy transposon transcripts in the cytoplasm. The third one, Piwi itself, is nuclear and the molecular mechanism of its function remains unknown. The main sources of piRNAs are discrete genomic loci called piRNA clusters, however it is not known what differentiate them from non-piRNA producing loci. During my PhD, I focused my work on two central questions:1) What is the role of Piwi in the nucleus? We showed that Piwi is responsible for transcriptional silencing by mediating installment of repressive marks, especially H3K9me3, over active transposons copies in a piRNA dependent manner.2) How are piRNA clusters defined, and what regulates their expression? Analyzing what features differentiate a piRNA producing loci from any non-producing loci in the genome, we were able to single out some specific characteristics: . We showed that maternally inherited piRNAs are responsible to define germline clusters at the next generation through two mechanisms: in the nucleus, by deposition of H3K9me3 onto complementary genomic sequence, and, in the cytoplasm, by initiating the ping-pong cycle using cluster transcripts as substrates, leading to their processing into mature piRNAs.. We found that cluster promoters are essential to mediate full cluster transcription, which is allowed thanks to a very specific chromatin signature necessary to ensure piRNA production
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2

Le, Thomas Adrien. "Piwi function and piRNA cluster regulation : Drosophila melanogaster". Thesis, Paris 6, 2014. http://www.theses.fr/2014PA066688/document.

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Les piRNAs sont une population de petit ARNs très diverse, que l'on retrouve dqns la lignée germinales des animaux pour réprimer les éléments génétiques mobiles : agissant de pair avec les protéines Piwi, ils guident le clivage des transposons actif. Chez la Drosophile, 3 protéines Piwi sont présentes, dont deux d'entre elles, AUB et AGO3, sont cytoplasmique et la dernière, PIWI, est nucléaire cependant son mécanisme d'action reste inconnu. La source principale de piRNAs sont des régions du génome bien particulière, appelé cluster de piRNAs. Cependant, il n'est pas encore connu a ce jour qu'est ce qui différentie ces région du reste du génome. Durant mon doctorant mon travail s'est focalisé sur ces deux questions centrales :Quel est le rôle de PIWI dans le noyau? Nous avons montré que PIWI était responsable de répression transcriptionnelle des transposons par l'intermédiaire de la déposition de marques chromatiniennes répressive, H3K9me3, grâce à la spécificité des piRNAs.Comment sont définit les régions générant des piRNA et comment sont régules leur expression ?Nous avons trouvé que les piRNAs qui sont transmis par la mère aux progénitures sont responsables de l'identification des régions génomiques donnant naissances à de nouveau piRNAs, grâce à la déposition de H3K9me3 dans le noyau et par l'initiation du cycle ping-pong dans le cytoplasme.Nous avons aussi mis en évidence les régions promoteurs des clusters de piRNAs, et trouve qu'elles sont nécessaires pour la production de piRNAs
PiRNAs are a diverse population of small RNA found in the animal germline to silence mobile genetic elements: loaded into Piwi proteins, they guide homology-dependent cleavage of active transposon mRNAs. In Drosophila, three Piwi proteins are expressed, from which two, AUB and AGO3, are known to destroy transposon transcripts in the cytoplasm. The third one, Piwi itself, is nuclear and the molecular mechanism of its function remains unknown. The main sources of piRNAs are discrete genomic loci called piRNA clusters, however it is not known what differentiate them from non-piRNA producing loci. During my PhD, I focused my work on two central questions:1) What is the role of Piwi in the nucleus? We showed that Piwi is responsible for transcriptional silencing by mediating installment of repressive marks, especially H3K9me3, over active transposons copies in a piRNA dependent manner.2) How are piRNA clusters defined, and what regulates their expression? Analyzing what features differentiate a piRNA producing loci from any non-producing loci in the genome, we were able to single out some specific characteristics: . We showed that maternally inherited piRNAs are responsible to define germline clusters at the next generation through two mechanisms: in the nucleus, by deposition of H3K9me3 onto complementary genomic sequence, and, in the cytoplasm, by initiating the ping-pong cycle using cluster transcripts as substrates, leading to their processing into mature piRNAs.. We found that cluster promoters are essential to mediate full cluster transcription, which is allowed thanks to a very specific chromatin signature necessary to ensure piRNA production
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3

Entrevan, Marianne. "Caractérisation de la diversité des sites de fixation des protéines du groupe Polycomb chez la Drosophile". Thesis, Montpellier, 2017. http://www.theses.fr/2017MONTT029/document.

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Les protéines du groupe Polycomb (PcG) ont initialement été identifiées chez la drosophile comme répresseurs transcriptionnels des gènes homéotiques. Aujourd’hui, nous savons que ces protéines jouent un rôle bien plus large puisqu’elles régulent des gènes dont les produits sont impliqués dans de nombreux processus biologiques (régulation des gènes HOX, maintien de la plasticité des cellules souches, la différenciation cellulaire, l’inactivation du chromosome X, la régulation des gènes soumis à empreintes). Leur dérégulation est source de nombreux cancers chez l’homme. Hautement conservées, elles forment deux principaux complexes : PRC 1 et 2 (Polycomb repressive complex 1 and 2), dont l’activité est respectivement reflétée par la mono-ubiquitinylation de la lysine 118 l’histone H2A (H2AK118Ub) et la tri-méthylation de la lysine 27 de l’histone H3 (H3K27me3). Chez la Drosophile, les sites de fixation de ces complexes sont appelés PRE (Polycomb Responsive Elements) où ils sont recrutés via des facteurs de transcription (FT).La complexité du recrutement des complexes du PcG, chez la Drosophile comme chez les mammifères, est visible à différents niveaux : au niveau de la séquence même de leurs sites de fixations, au niveau des facteurs de transcription qui les recrutent, au niveau de l’interface entre les deux complexes PRC1 et PRC2 et enfin au niveau global, part le présence de ces complexes au niveau de sites transcriptionnellement actifs. L’ensemble de ces résultats démontre clairement la nature hétérogène des PRE. Ces derniers diffèrent non seulement par leur séquence, mais également par les FT qui les recrutent et enfin par la manière dont les complexes PcG sont recrutés (PRC2 recrute PRC1 ou le contraire). Mon projet de thèse s’est donc dessiné autour d’une hypothèse : il existe différentes classes de PRE chez la Drosophile. Mon travail a donc consisté à définir ces différentes classes et à les caractériser pour en déduire des rôles spécifiques à l’échelle génomique. En effet, l’implication des complexes du PcG dans l’apparition de cancer chez l’Homme requière que l’on comprenne comment ces protéines sont recrutées à la chromatine.Mes travaux de thèse ont permis d’identifier six classes différentes de sites de fixation aux protéines du PcG. Nous avons retrouvé une classe correspondant aux sites de fixations canoniques fixés par les protéines du PcG et présents au sein de larges domaines répressifs marqués par H3K27me3. Une seconde classe correspond à des éléments de régulation marqués par un état de pause transcriptionnelle. De façon surprenante, nous avons démontré qu’une grande partie des sites de fixation des complexes du PcG était localisée au niveau de régions transcriptionnellement actives. Ces classes de PRE diffèrent en particulier en éléments génomiques qui les composent. Deux classes correspondent à des enhancers développementaux. Une classe correspond à des promoteurs actifs pouvant réguler des gènes de ménage. Enfin, une dernière classe correspond à des bordures de TAD. Les sites actifs et réprimés fixés par le PcG fixent également des combinaisons différentes de FT. Des analyses in vivo associées à un transcriptome réalisé à partir de cellules mutantes pour une protéine du PcG révèlent que les complexes du PcG jouent également un rôle de répresseur transcriptionnel au niveau des sites actifs. L’ensemble de ces résultats suggère une hétérogénéité inattendue des sites de fixation des complexes du PcG et permettra de mieux comprendre les caractéristiques liées à ces protéines dont la dérégulation mène à l’apparition de cancers chez l’Homme marqués par leur agressivité
Polycomb group (PcG) complexes were initially discovered in Drosophila as transcriptionnal repressors of homeotic genes. To date, we know that they are involves in a large pleithora of biological processes including the maintenance of stem cells plasticity, differentiation, X chromosome inactivation and imprinting. PcG complexes are highly conserved from Drosophila to Humans and can be divided into two main complexes: PRC1 and PRC2 (Polycomb repressive complex 1 and 2). Both complexes have a histone modifying activity: PRC1 catalyses the mono-ubiquitination of the lysine 118 on histone H2A (H2AK118Ub) and PRC2 catalyses the tri-methylation of the lysine 27 on histone H3 (H3K27me3).In Drosophila, these complexes are recruited to cis regulatory elements named Polycomb Responsive Elements (PREs) that drive the epigenetic inheritance of silent chromatin states throughout development. Importantly, PcG complexes do not contain DNA-binding activity but are recruited to PREs via their interaction with Transcription Factors (TF) recognizing DNA motifs clustered at PREs. However the mechanism how PREs target PcG complexes is still not well understood due to the complexity of PcG recruitment, which is reflected at different levels: The DNA signature between PREs can differ significantly and several TF are implicated in PcG recruitment, but none of them is sufficient to recruit PcG complexes to PREs. Moreover PcG complexes can cooperate in different ways to stabilize each other’s binding. Finally, another layer of complexity is found at a more global level since PcG complexes do not only bind repressed sites, but they are also found at active regions.Therefore, our working hypothesis is that different classes of PREs exist in Drosophila. My PhD work was thus to define these different classes of PREs on a genome-wide scale and to functionally characterize them in order to get a complete molecular description of PRE function. Understanding how PcG complexes are recruited is of high importance, since deregulation of both, PcG complexes and their recruiting factors can led to cancer and diseases. My work led to the identification of six different classes of PREs that are characterized by different chromatin and genomic features. Interestingly the majority of PREs are associated with active genes that can be divided into housekeeping regulatory regions and developmental enhancers. In addition another class comprises bona fide chromatin domain boundaries. On the other hand PREs associated with repressed chromatin states shows features of previously described PREs and associate with repressed genes and PcG-associated histone marks. Finally another class comprises PREs that are likely in a poised chromatin state. We further demonstrated that PREs located at repressed and active regions differ in their combination of TF. In vivo analyses along with a transcriptomic analysis performed in cell lines mutated for a member of PcG complexes revealed that PcG complexes play a repressive role at both, active and repressed PREs.Taken together, our result suggest an unexpected heterogeneity of PREs and contributes to the better understanding of their characteristics and function
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4

Barthez, Marine. "Functional characterisation of Drosophila M1BP". Thesis, Aix-Marseille, 2019. http://www.theses.fr/2019AIXM0298.

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Le facteur de transcription Motif 1 Binding Protein (M1BP) est une protéine à doigt de zinc décrite pour contrôler la pause de l’ARN polymérase II aux sites d’initiation de la transcription de milliers de gènes chez la Drosophile. J’ai démontré une interaction directe entre M1BP et les protéines Hox AbdA et Ubx chez la Drosophile, interaction essentielle au mécanisme de levée de la pause transcriptionnelle. L’expression de M1BP est maintenue au cours du développement, mais une perte de son expression induit de façon précoce l’autophagie, ceci sans affecter l’expression des protéines Hox. Des approches transcriptomiques ont par ailleurs révélé que M1BP régule 25% des voies métaboliques de la cellule. En accord avec ces observations, nous avons remarqué que la perte de fonction de M1BP induit de nombreux phénotypes et anomalies mitochondriales, notamment dans la chaine respiratoire avec la mise en place d’une respiration anaérobie. L’ensemble de ces résultats mettent en évidence que M1BP est un régulateur transcriptionnel jouant un rôle clé dans le métabolisme mitochondrial et cellulaire. La recherche d’un homologue de la protéine de Drosophile M1BP chez les vertébrés a conduit à l’identification de ZKSCAN3, capable de corriger l’induction précoce de l’autophagie résultant de la perte de M1BP, et de rétablir l’expression de la majorité des gènes dérégulés dans ce contexte. Dans l’ensemble, ces données montrent que les protéines M1BP et ZKSCAN3 sont des homologues fonctionnels dans la régulation de l’autophagie
The transcription factor Motif 1 Binding protein (M1BP) is a zinc finger protein known to be involved in the pausing of RNA Polymerase II (Pol II) at the transcription start site of thousands of Drosophila genes. I was able to demonstrate direct protein interaction between M1BP and the Drosophila Hox proteins AbdA and Ubx, providing evidence that Hox-M1BP collaborate to regulate gene expression. While M1BP expression is maintained during all larval stages, loss of M1BP expression in the feeding stage induces premature autophagy. In characterising the diverse functions of M1BP, I determined that M1BP transcriptionally regulates 25% of all cellular metabolic pathways. Indeed, many severe mitochondrial defects and phenotypes are observed upon M1BP knock down in the Drosophila fat body and indirect flight muscle. One of the major consequences of M1BP knock down is that the respiratory chain is strongly impacted forcing the cell to switch to anaerobic respiration for the production of ATP. Together, these data provide evidence that M1BP is an essential transcriptional regulator of mitochondrial and cellular metabolic processes. In searching for a vertebrate homolog of Drosophila M1BP, I identified ZKSCAN3 as a vertebrate functional homolog of M1BP in autophagy repression in Drosophila fat body or in human cell lines. Additionally, transcriptomic studies demonstrate that ZKSCAN3 expression in the Drosophila fat body reverses the deregulation of the majority of genes observed upon M1BP knock down. Together with the identification that ZKSCAN3 binds to 90% of M1BP genomic targets, these data provide evidence that M1BP and ZKSCAN3 are functionally homologous proteins
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5

Ducuing, Antoine. "Signalling and morphogenesis during Drosophila dorsal closure". Thesis, Lyon, 2016. http://www.theses.fr/2016LYSEN002/document.

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La fermeture dorsale est un événement majeur de l’embryogénèse de la drosophile durant lequel les cellules les plus dorsales de l’épiderme se différencient et agissent de concert pour refermer une ouverture dorsale temporairement recouverte par l’amnioséreuse. Ce processus présente de nombreuses similarités avec la cicatrisation cellulaire. J’ai montré que les voies JNK et DPP forment une boucle cohérente appelée « feed-forward loop » (boucle d’anticipation) qui contrôle la différentiation des cellules de la marge active. La branche DPP de cette boucle filtre les signaux non désirés de la voix JNK quand les embryons sont soumis à un stress thermique. Je me suis ensuite concentré sur le câble d'actine, une structure supra-cellulaire produite par les cellules de la marge active lors de la fermeture dorsale. J’ai montré que le câble d’actine est une structure discontinue qui n’est pas nécessaire pour la fermeture dorsale ou pour la cicatrisation cellulaire. Le câble d’actine homogénéise les forces et stabilise la géométrie cellulaire pour que la fermeture se fasse de manière parfaite et sans cicatrice. Sans le câble, les cellules ont une forme irrégulière, associé à des défauts de patterning et des défauts de polarité planaire qui ressemblent aux défauts que l’on trouve lors de la formation d’une cicatrice. Nous proposons donc que le câble empêche la formation de cicatrice en « congelant » les propriétés mécaniques des cellules afin de les protéger des forces qui agissent au niveau tissulaire lors de la fermeture dorsale.En conclusion, mon travail apporte un regard neuf sur la signalisation et la morphogenèse lors de la fermeture dorsale de l’embryon de Drosophile
Drosophila dorsal closure is a key embryonic process during which the dorsal-most epidermal cells called leading edge cells differentiate and act in a coordinated manner to close a transient dorsal hole covered by the amnioserosa in a process reminiscent of wound healing. I showed that JNK and DPP are wired in a network motif called ‘feed-forward loop’ (FFL) that controls leading edge cell specification and differentiation. The DPP branch of the FFL filters unwanted JNK activity that occurs during thermal stress. Next, I focused on the actin cable, a supra-cellular structure produced by the leading edge cells during dorsal closure or wound healing from fly to humans. My data suggest that the actin cable does not provide a major contractile force. Rather, the actin cable balances forces and stabilizes cell geometry so that closure resolves in a perfectly structured and scar-free tissue. The absence of the cable leads to cell shape irregularities as well as patterning and planar cell polarity defects that are reminiscent of scarring. We propose that the cable prevents scaring by acting as a mechanical freeze field that protects fine cellular structures from the major closure forces that operate at tissue level. Altogether, my work brings new insights on the signalling and morphogenesis during dorsal closure
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6

Lepot, Frédérique. "Recherches sur l'apprentissage associatif chez la drosophile (drosophila melanogaster)". Toulouse 3, 1986. http://www.theses.fr/1986TOU30033.

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Etude de l'inhibition conditionnee de la reponse tarsale (extension du proboscis, induite par stimulation sucree des chemorecepteurs tarsaux) chez la drosophile. Conditions optimales pour l'acquisition et l'extinction de ce comportement (importance de certains indices visuels). Existence d'un apprentissage associatif revele par l'acquisition d'une discrimination visuelle successive, avec renforcement regulier de l'un des indices visuels ou en apprentissage "probabiliste". La mutation dunce **(2), qui deteriore l'acquisition d'une discrimination olfactive n'affecte pas la maitrise d'une discrimination visuelle
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7

Lepot, Frédérique. "Recherches sur l'apprentissage associatif chez la Drosophile, Drosophila melanogaster". Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb37599134t.

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8

Ruby, Vincent. "Étude des évènements mitochondriaux impliqués dans le contrôle de l'apoptose par rbf1, l'homologue de drosophile du gène suppresseur de tumeur rb". Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLV039/document.

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Le gène rb est le premier suppresseur de tumeur découvert chez l’homme. Il prévient l’apparition de tumeurs notamment en régulant négativement le cycle cellulaire. Le rôle de pRb dans le contrôle de l’apoptose est plus complexe et les mécanismes moléculaires contrôlés par ce facteur de transcription ne sont pas complétement élucidés. Il existe un homologue de rb chez la drosophile : rbf1. J’ai contribué à caractériser les évènements mitochondriaux induits au cours de l’activation de l’apoptose par Rbf1 dans le disque imaginal d'aile, un tissu en prolifération de la larve de drosophile. Dans cette voie d’apoptose, la protéine Debcl, seule membre pro-apoptotique de la famille Bcl-2 chez la drosophile, est activée et induit le recrutement et l’oligomérisation de Drp1, protéine effectrice principale de la fission mitochondriale. C’est ainsi qu’est déclenchée la fragmentation mitochondriale et l’accumulation d’espèces activées de l’oxygène (EAOs) mitochondriales. Ces deux évènements participent à la transmission du signal apoptotique. J’ai par ailleurs pu mettre en évidence l’implication de facteurs participant au maintien du contrôle qualité mitochondriale. Celui-ci s’assure de l’intégrité des mitochondries et, le cas échéant, déclenche la digestion des éléments défaillants par mitophagie. Enfin, j’ai contribué à l’étude des liens entre la traduction et l’apoptose induite par Rbf1. Dans cette étude, nous montrons que la poly-A binding protein (PABP) peut supprimer le phénotype d’encoche induit par Rbf1 chez l’adulte alors que la mort cellulaire induite au cours du stade larvaire n’est pas inhibée mais augmentée. Ces résultats nous ont poussé à étudier les mécanismes de compensation induits par l’appareil traductionnel, ce qui nous a permis de montrer qu'une modulation de la traduction pourrait permettre de compenser la perte de tissu consécutive à l'apoptose induite par Rbf1 sans impliquer une inhibition de l'apoptose
The gene rb is the first tumor suppressor discovered in humans. Its prevents the appearance of tumors by regulating negatively the cell cycle. The role of pRb in apoptosis is more complex and the molecular mechanisms triggered by this transcription factor are not completely elucidated. There is a rb homologue in drosophila: rbf1. I participated in the characterization of mitochondrial events induced during activation of apoptosis by Rbf1 in a proliferating tissue of this model organism, the wing disc. In this apoptosis pathway, the Debcl protein, the only drosophila pro-apoptotic member of the Bcl-2 family, is activated and induces recruitment and oligomerization of Drp1, the main effector of mitochondrial fission. This triggers the mitochondrial fragmentation and the accumulation of mitochondrial reactive oxygen species (ROS). Both events participate to the transmission of the apoptotic signal. I have also been able to highlight the implication of factors involved in maintaining mitochondrial quality control which ensures the integrity of the mitochondria and, if necessary, triggers the degradation of damaged elements by mitophagy. Finally, I have contributed to the study of the links between translation and apoptosis induced by Rbf1. In this study, we show that the Poly-A Binding Protein (PABP) can suppress the Rbf1-induced notch phenotype in adults while cell death induced during larval stage was not inhibited but increased. These results prompted us to study the compensation mechanisms induced by the translational apparatus, which allowed us to show that a mRNA translation-related mechanism could counteract the loss of tissue resulting from Rbf1-induced apoptosis independently of apoptosis inhibition
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9

Garrido, Damien. "Etude de l’homéostasie lipidique chez Drosophila melanogaster". Thesis, Université Paris-Saclay (ComUE), 2015. http://www.theses.fr/2015SACLS030.

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Le métabolisme des acides gras (AG) est crucial dans le maintien de l’homéostasie. Son implication dans des processus tels que la signalisation, le stockage énergétique, l’isolation thermique, la régulation du comportement ne révèle qu’une fraction de la complexité et de la variabilité des rôles dans lesquels il peut être associé. En outre, ce métabolisme est dérégulé dans de nombreuses pathologies, diabète, obésité, cancers,... C’est pourquoi les enzymes de ce métabolisme constituent des cibles attractives pour développer de nouveaux traitements. Cependant les conséquences de ces dérégulations sur l’organisme sain sont encore mal connues, surtout à l’échelle de chaque organe.L’objectif de ma thèse était d’évaluer comment le métabolisme des AGs participe à la régulation de l’homéostasie au sein d’un organisme entier. Pour cela, j’ai utilisé les possibilités génétiques du modèle drosophile dont le métabolisme est comparable à celui des mammifères. J’ai ainsi montré que la synthèse d’AGs contribue à neutraliser les effets toxiques du sucre alimentaire. Ce processus se fait en coopération avec la voie de la détoxification du méthylglyoxal qui permet de prévenir la formation de composés issus de la glycation non enzymatique. J’ai aussi contribué à montrer que les précurseurs des hydrocarbures et phéromones ont une origine flexible, qui dépend du maintien de l’homéostasie et qui peut perturber les interactions entre individus. Je suis actuellement en train d’étudier la sensibilité à l’inhibition de la synthèse d’AG de différents modèles de croissance dérégulée. Enfin, dans un travail préliminaire, j’ai montré que le métabolisme des AGs est essentiel dans le tube digestif, possiblement en perturbant l’homéostasie hydrique de la larve.L’ensemble de ces résultats aidera à mieux cerner l’importance du métabolisme des AGs dans le maintien de l’homéostasie d’un organisme sain et dans des processus dérégulés
Fatty acid (FA) metabolism is crucial in maintaining homeostasis, but also in a numerous of processes including signaling, energy storage, protection to temperature loss, regulation of behavior... In addition, FA metabolism is deregulated in several pathologies including diabetes, obesity, and cancers... Therefore, the enzymes that catalyze the reactions of the FA metabolic pathways constitute attractive targets to develop novel therapies. However the consequences of these deregulations in healthy organism are still poorly known, in particular at the level of each organ.The aim of my PhD was to estimate how FA metabolism participates in the regulation of homeostasis within a whole body organism. To address these issues, I used the genetic possibilities of the Drosophila model, whose metabolism is similar to that of mammals.I showed that FA synthesis contributes to neutralize the toxic effects of dietary sugar. This process operates in cooperation with the methylglyoxal detoxification pathway, which prevents the formation of compounds resulting from the non-enzymatic glycation. I also contributed to a project showing that the precursors of hydrocarbons and pheromones have a flexible origin, which depends on lipid homeostasis and may affect sexual recognition between individuals. Currently, I’m studying the consequences of FA synthesis inhibition in various deregulated growth models. Finally, in a preliminary work, I showed that the FA metabolism is essential in the digestive tract, possibly by disrupting water homeostasis in larvae. Taken together, these results will help to characterize the importance of FA metabolism in healthy organism as well as in deregulated processes
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10

Lamiré, Laurie-Anne. "Identification and characterization of the mechanical role of germline growth in Drosophila melanogaster epithelial morphogenesis". Thesis, Lyon, 2019. http://www.theses.fr/2019LYSEN002.

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La morphogenèse épithéliale est essentielle à la formation des organes. J'utilise le follicule ovarien de Drosophila comme modèle d'étude de l’aplatissement des cellules. Un follicule est composé de cellules germinales en croissance entourées d'une monocouche de cellules épithéliales cuboïdes. À un stade de développement spécifique, une part de ces cellules s'aplatit en suivant une vague régulée. Cet aplatissement est en partie contrôlé par un gradient de pression provenant d’un groupe de cellules germinales (les cellules nourricières). Toutes les cellules germinales sont connectées via des ponts cytoplasmiques. Cette thèse étudie les mécanismes conduisant à la génération du gradient de pression, et à la modulation moléculaire induite par cette force mécanique pour permettre l'aplatissement. J'ai montré que le nombre et le diamètre des ponts cytoplasmiques influaient sur la pression. En utilisant des reconstructions tridimensionnelles de follicules, j’ai étudié le rôle de la croissance différentielle des cellules nourricières en mesurant le changement de volume des cellules germinales lors de l'aplatissement des cellules. Enfin, j’ai cherché le mécanisme moléculaire conduisant à l’aplatissement des cellules et influencé par un stimulus mécanique à partir de la pression germinale, en proposant un rôle de la voie Hippo dans ce processus. En conclusion, nous proposons que la croissance des cellules germinales influe de manière mécanique et génétique sur les cellules épithéliales pour permettre l’élongation, et donc l'acquisition de la forme finale du follicule
Epithelial morphogenesis is essential for organ formation. I use the Drosophila ovarian follicle as a model for studying cell flattening. A follicle is composed of growing germ cells surrounded by a monolayer of cuboidal epithelial cells. At a specific stage of development, some of these cells flatten out following a regulated wave. This flattening is partly controlled by a pressure gradient from part of the germ cells (the nurse cells). All germ cells are connected via cytoplasmic bridges. This thesis studies the mechanisms leading to the generation of the gradient of pressure, and to the molecular modulation induced by this mechanical force to allow flattening. I have shown that the number and diameter of cytoplasmic bridges affect the pressure. Using three-dimensional reconstructions of follicles, I studied the role of differential growth of nurse cells by measuring the change in germ cell volume during epithelial cell flattening. Finally, I looked for the molecular mechanism leading to the flattening and influenced by a mechanical stimulus from the germinal pressure, supporting a role of the Hippo pathway in this process. In conclusion, we propose that germ cell growth mechanically and genetically influences epithelial cells to allow elongation, and thus the acquisition of the final form of the follicle
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Livros sobre o assunto "Drosophila"

1

Dahmann, Christian, ed. Drosophila. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2541-5.

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Dahmann, Christian, ed. Drosophila. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-583-1.

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Dahmann, Christian, ed. Drosophila. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6371-3.

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4

Ashburner, M. Drosophila. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1989.

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5

Ashburner, M. Drosophila. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1989.

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6

Ashburner, M. Drosophila. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1989.

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7

Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. Drosophila Genetics. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7.

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8

Bratu, Diana P., e Gerard P. McNeil, eds. Drosophila Oogenesis. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2851-4.

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9

Arkhipova, Irina R. Drosophila retrotransposons. New York: Springer-Verlag, 1995.

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10

Giedt, Michelle S., e Tina L. Tootle, eds. Drosophila Oogenesis. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2970-3.

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Capítulos de livros sobre o assunto "Drosophila"

1

Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "General". In Drosophila Genetics, 1–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_1.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Morphology of Drosophila Melanogaster". In Drosophila Genetics, 33–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_2.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Transmission Genetics". In Drosophila Genetics, 55–101. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_3.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Phenogenetics". In Drosophila Genetics, 103–34. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_4.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Mutation Genetics". In Drosophila Genetics, 135–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_5.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Population Genetics". In Drosophila Genetics, 163–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_6.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Cytology and Cytogenetics". In Drosophila Genetics, 177–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_7.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Molecular Biology". In Drosophila Genetics, 189–202. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_8.

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Graf, Ulrich, Nancy van Schaik e Friedrich E. Würgler. "Results and Answers". In Drosophila Genetics, 203–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_9.

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Romeo, Yves, e Bruno Lemaitre. "Drosophila Immunity". In Innate Immunity, 379–94. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-570-1_22.

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Trabalhos de conferências sobre o assunto "Drosophila"

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Zhang, Aijun. "Attractive blend for spotted wing drosophila,Drosophila suzukii(Matsumura)". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.111493.

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Liang, Jingdong, Jixiang Sun e Yuancheng Xie. "Research of drosophila detection and courtship drosophila differentiation against complicated background". In 2011 International Conference on Electrical and Control Engineering (ICECE). IEEE, 2011. http://dx.doi.org/10.1109/iceceng.2011.6057886.

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Sward, Grace. "An exploration into pesticide resistance in spotted wing drosophila, Drosophila suzukii". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.114081.

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Yi, Hoonbok. "Drosophila suzukiiin South Korea". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93351.

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GÖPFERT, M. C., e D. ROBERT. "MICROMECHANICS OF DROSOPHILA AUDITION". In Proceedings of the International Symposium. WORLD SCIENTIFIC, 2003. http://dx.doi.org/10.1142/9789812704931_0042.

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Becher, Paul G. "Detangling behavioral responses to semiochemicals in the spotted wing drosophila,Drosophila suzukii". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93348.

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Guédot, Christelle. "Using CSIA to identify unexpected hosts for spotted wing drosophila,Drosophila suzukii". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94563.

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Tang, Siew Bee. "Screening of RNAi targets and impacts on spotted wing drosophila,Drosophila suzukii". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.115004.

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Kirkpatrick, Danielle M. "Improving the efficiency of monitoring tools for spotted wing drosophila,Drosophila suzukii". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.108543.

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Wells, Simon, e Chris Reed. "A drosophila for computational dialectics". In the fourth international joint conference. New York, New York, USA: ACM Press, 2005. http://dx.doi.org/10.1145/1082473.1082724.

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Relatórios de organizações sobre o assunto "Drosophila"

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Audsley, Neil, Gonzalo Avila, Claudio Ioratti, Valerie Caron, Chiara Ferracini, Tibor Bukovinszki, Marc Kenis et al. Spotted wing drosophila, Drosophila suzukii (Matsumura). Euphresco, 2023. http://dx.doi.org/10.1079/20240228462.

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Resumo:
The spotted wing Drosophila, (Drosophila suzukii) native to East Asia, has become an invasive species in Hawaii, USA, since 1980 and has spread globally, affecting economically important crops, such as berries and cherries. The high reproductive rate and polyphagous nature of this species, facilitated by a unique serrated ovipositor, lead to significant crop damage and economic losses. Control measures, including insecticides and biological controls, have limited effectiveness due to frequent re-infestations. Research in Asia identified 3 promising parasitoid species for classical biological control, i.e. Ganaspis brasiliensis, Leptopilina japonica and Asobara japonica. Among them, G. brasiliensis (particularly genetic group G1) shows the highest host specificity to D. suzukii and has been approved for release in several regions. Other parasitoids, such as Trichopria drosophilae and Pachycrepoideus vindemiae, have limited field effectiveness. Classical biological control using specific parasitoids offers a potential solution to reduce D. suzukii populations and mitigate economic damage.
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Bernards, Andre. Functional Analysis of Drosophila NF1. Fort Belvoir, VA: Defense Technical Information Center, julho de 2005. http://dx.doi.org/10.21236/ada444270.

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Walker, James A. Developing a Drosophila Model of Schwannomatosis. Fort Belvoir, VA: Defense Technical Information Center, agosto de 2012. http://dx.doi.org/10.21236/ada575950.

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Walker, James A. Developing a Drosophila Model of Schwannomatosis. Fort Belvoir, VA: Defense Technical Information Center, fevereiro de 2013. http://dx.doi.org/10.21236/ada575951.

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Zhong, Yi. Functional Analysis of Human NF1 in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, dezembro de 2008. http://dx.doi.org/10.21236/ada488787.

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Zhong, Yi. Functional Analysis of Human NF1 in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, janeiro de 2009. http://dx.doi.org/10.21236/ada532314.

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Zhong, Yi. Functional Analysis of Human NF1 in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, janeiro de 2007. http://dx.doi.org/10.21236/ada465210.

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Greenspan, Ralph J. Gene Networks Underlying Chronic Sleep Deprivation in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, junho de 2014. http://dx.doi.org/10.21236/ada610340.

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Gerald M. Rubin. Resources for Biological Annotation of the Drosophila Genome. Office of Scientific and Technical Information (OSTI), agosto de 2005. http://dx.doi.org/10.2172/842216.

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Zhong, Yi. NF-1 Dependent Gene Regulation in Drosophila Melanogaster. Fort Belvoir, VA: Defense Technical Information Center, abril de 2004. http://dx.doi.org/10.21236/ada471891.

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