Literatura científica selecionada sobre o tema "Dicarbonyl stress"
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Artigos de revistas sobre o assunto "Dicarbonyl stress"
Csongová, Melinda, Jean L. J. M. Scheijen, Marjo P. H. van de Waarenburg, Radana Gurecká, Ivana Koborová, Tamás Tábi, Éva Szökö, Casper G. Schalkwijk e Katarína Šebeková. "Association of α-Dicarbonyls and Advanced Glycation End Products with Insulin Resistance in Non-Diabetic Young Subjects: A Case-Control Study". Nutrients 14, n.º 22 (21 de novembro de 2022): 4929. http://dx.doi.org/10.3390/nu14224929.
Texto completo da fonteNigro, Cecilia, Alessia Leone, Francesca Fiory, Immacolata Prevenzano, Antonella Nicolò, Paola Mirra, Francesco Beguinot e Claudia Miele. "Dicarbonyl Stress at the Crossroads of Healthy and Unhealthy Aging". Cells 8, n.º 7 (19 de julho de 2019): 749. http://dx.doi.org/10.3390/cells8070749.
Texto completo da fonteAhmad, Khurshid, Sibhghatulla Shaikh, Eun Ju Lee, Yong-Ho Lee e Inho Choi. "Consequences of Dicarbonyl Stress on Skeletal Muscle Proteins in Type 2 Diabetes". Current Protein & Peptide Science 21, n.º 9 (11 de dezembro de 2020): 878–89. http://dx.doi.org/10.2174/1389203720666191119100759.
Texto completo da fonteRabbani, Naila, Mingzhan Xue e Paul J. Thornalley. "Methylglyoxal-induced dicarbonyl stress in aging and disease: first steps towards glyoxalase 1-based treatments". Clinical Science 130, n.º 19 (23 de agosto de 2016): 1677–96. http://dx.doi.org/10.1042/cs20160025.
Texto completo da fonteTatone, Carla, Ursula Eichenlaub-Ritter e Fernanda Amicarelli. "Dicarbonyl stress and glyoxalases in ovarian function". Biochemical Society Transactions 42, n.º 2 (20 de março de 2014): 433–38. http://dx.doi.org/10.1042/bst20140023.
Texto completo da fonteMasania, Jinit, Malgorzata Malczewska-Malec, Urszula Razny, Joanna Goralska, Anna Zdzienicka, Beata Kiec-Wilk, Anna Gruca et al. "Dicarbonyl stress in clinical obesity". Glycoconjugate Journal 33, n.º 4 (24 de junho de 2016): 581–89. http://dx.doi.org/10.1007/s10719-016-9692-0.
Texto completo da fonteAlouffi, Sultan, e Mohd Wajid Ali Khan. "Dicarbonyls Generation, Toxicities, Detoxifications and Potential Roles in Diabetes Complications". Current Protein & Peptide Science 21, n.º 9 (11 de dezembro de 2020): 890–98. http://dx.doi.org/10.2174/1389203720666191010155145.
Texto completo da fonteRabbani, Naila, e Paul J. Thornalley. "Dicarbonyls linked to damage in the powerhouse: glycation of mitochondrial proteins and oxidative stress". Biochemical Society Transactions 36, n.º 5 (19 de setembro de 2008): 1045–50. http://dx.doi.org/10.1042/bst0361045.
Texto completo da fonteMey, Jacob T., Brian K. Blackburn, Edwin R. Miranda, Alec B. Chaves, Joan Briller, Marcelo G. Bonini e Jacob M. Haus. "Dicarbonyl stress and glyoxalase enzyme system regulation in human skeletal muscle". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 314, n.º 2 (1 de fevereiro de 2018): R181—R190. http://dx.doi.org/10.1152/ajpregu.00159.2017.
Texto completo da fonteAntognelli, Cinzia, Andrea Perrelli, Tatiana Armeni, Vincenzo Nicola Talesa e Saverio Francesco Retta. "Dicarbonyl Stress and S-Glutathionylation in Cerebrovascular Diseases: A Focus on Cerebral Cavernous Malformations". Antioxidants 9, n.º 2 (1 de fevereiro de 2020): 124. http://dx.doi.org/10.3390/antiox9020124.
Texto completo da fonteTeses / dissertações sobre o assunto "Dicarbonyl stress"
Ashour, Amal. "Dicarbonyl stress and dysfunction of the glyoxalase system in periodontal diseases". Thesis, University of Warwick, 2016. http://wrap.warwick.ac.uk/80026/.
Texto completo da fonteKimzey, Michael John. "Identification, Characterization, and Quantification of Dicarbonyl Adducts in the Plasma Proteome in Type-2 Diabetes". Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/145123.
Texto completo da fonteRosenstock, Philip [Verfasser], Iris [Gutachter] Thondorf, Rüdiger [Gutachter] Horstkorte e Lars-Oliver [Gutachter] Klotz. "Untersuchungen humaner natürlicher Killer-Zellen und ihrer Sialyltransferasen nach Dicarbonyl-Stress / Philip Rosenstock ; Gutachter: Iris Thondorf, Rüdiger Horstkorte, Lars-Oliver Klotz". Halle (Saale) : Universitäts- und Landesbibliothek Sachsen-Anhalt, 2020. http://d-nb.info/1215098855/34.
Texto completo da fonteHalkoum, Rym. "Rôle du glyoxal dans la sénescence cellulaire : implications dans le vieillissement de la peau". Electronic Thesis or Diss., Sorbonne université, 2021. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2021SORUS016.pdf.
Texto completo da fonteSenescence is a well-characterized cellular state associated with specific markers such as permanent cell proliferation arrest, and the secretion of messenger molecules by cells expressing the Senescence-Associated Secretory Phenotype (SASP). The SASP can display autocrine and paracrine effects which contribute to the senescent phenotype reinforcement and propagation. The SASP composition depends on many factors such as the cell type or the nature of the stress that induces senescence. Since the skin constitutes a barrier with the external environment, it is particularly subjected to different types of stresses, and consequently prone to premature cellular aging. Glyoxal, a dicarbonyl compound produced during glucose metabolism and lipid peroxidation, is a precursor of Advanced Glycation End-products (AGEs), whose presence marks normal and pathological aging. My thesis work showed that glyoxal treatment provokes oxidative stress by increasing reactive oxygen species and AGEs levels and induce senescence in human keratinocytes. Furthermore, glyoxal-induced senescence bears a unique molecular progression profile: an “early-stage” when AKT-FOXO3a-p27KIP1 pathway mediates cell-cycle arrest, and a “late-stage” senescence maintained by the p16INK4/pRb pathway. Moreover, we characterized the resulting secretory phenotype during early senescence by mass spectrometry in order to find new targets for senomorphic ingredients. Our study provides evidence that glyoxal can affect keratinocyte functions and act as a driver of human skin aging
Yang, Kai. "Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes". Thesis, 2011. http://hdl.handle.net/1807/31650.
Texto completo da fonteBanach, Monica Sofia. "Hepatocyte Cytotoxicity Induced by Hydroperoxide (Oxidative Stress Model) or Dicarbonyls (Carbonylation Model): Prevention by Bioactive Nut Extracts or Catechins". Thesis, 2009. http://hdl.handle.net/1807/18164.
Texto completo da fonteCapítulos de livros sobre o assunto "Dicarbonyl stress"
Kovacic, Peter, e Ratnasamy Somanathan. "Novel Mechanism for Advanced Glycation End Product (AGE) Toxicity: α-Dicarbonyls, Electron Transfer, Radicals, Oxidative Stress, and Antioxidants". In Systems Biology of Free Radicals and Antioxidants, 3405–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-30018-9_153.
Texto completo da fonteRabbani, Naila, Mingzhan Xue e Paul J. Thornalley. "Dicarbonyl stress and the glyoxalase system". In Oxidative Stress, 759–77. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-818606-0.00036-5.
Texto completo da fonteTrabalhos de conferências sobre o assunto "Dicarbonyl stress"
Bulkescher, R., S. Herzig, J. Szendrödi, PP Nawroth e J. Zemva. "Dicarbonyl stress in endothelial cells alters mitochondrial protein homeostasis". In Late Breaking Abstracts Diabetes Kongress 2021 – 55. Jahrestagung der DDG Präzisionsmedizin – Eine Reise in die Zukunft der Diabetologie www.diabeteskongress.de. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1730861.
Texto completo da fonteBellier, J., MJ Nokin, F. Durieux, F. Journe, G. Ghanem, V. Castronovo e A. Bellahcène. "PO-219 Methylglyoxal-induced dicarbonyl stress: role in melanoma progression and response to therapy". In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.254.
Texto completo da fonteBellahcene, A., J. Bellier, O. Peulen, G. Rademaker, B. Charloteaux, S. Van Laere, M. Herfs, C. Lambert, V. Castronovo e MJ Nokin. "PO-226 Dicarbonyl stress induces ECM remodelling and MAPK signalling activation in metastatic breast cancer cells". In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.260.
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