Literatura científica selecionada sobre o tema "D2-mdx"
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Artigos de revistas sobre o assunto "D2-mdx"
De Giorgio, Daria, Deborah Novelli, Francesca Motta, Marianna Cerrato, Davide Olivari, Annasimon Salama, Francesca Fumagalli et al. "Characterization of the Cardiac Structure and Function of Conscious D2.B10-Dmdmdx/J (D2-mdx) mice from 16–17 to 24–25 Weeks of Age". International Journal of Molecular Sciences 24, n.º 14 (22 de julho de 2023): 11805. http://dx.doi.org/10.3390/ijms241411805.
Texto completo da fonteHassani, Medhi, Dylan Moutachi, Mégane Lemaitre, Alexis Boulinguiez, Denis Furling, Onnik Agbulut e Arnaud Ferry. "Beneficial effects of resistance training on both mild and severe mouse dystrophic muscle function as a preclinical option for Duchenne muscular dystrophy". PLOS ONE 19, n.º 3 (8 de março de 2024): e0295700. http://dx.doi.org/10.1371/journal.pone.0295700.
Texto completo da fonteHayes, Holly M., Julie Angerosa, Adam T. Piers, Jason D. White, Jane Koleff, Madeline Thurgood, Jessica Moody, Michael M. Cheung e Salvatore Pepe. "Preserved Left Ventricular Function despite Myocardial Fibrosis and Myopathy in the Dystrophin-Deficient D2.B10-Dmdmdx/J Mouse". Oxidative Medicine and Cellular Longevity 2022 (16 de março de 2022): 1–19. http://dx.doi.org/10.1155/2022/5362115.
Texto completo da fonteKrishna, Swathy, Tiffany Quindry, Matthew B. Hudson, John C. Quindry e Joshua T. Selsby. "Defective Autophagic Degradation in Aged D2‐mdx Diaphragms". FASEB Journal 34, S1 (abril de 2020): 1. http://dx.doi.org/10.1096/fasebj.2020.34.s1.04955.
Texto completo da fonteYarlagadda, Sai, Christina Kulis, Peter G. Noakes e Mark L. Smythe. "Hematopoietic Prostaglandin D Synthase Inhibitor PK007 Decreases Muscle Necrosis in DMD mdx Model Mice". Life 11, n.º 9 (21 de setembro de 2021): 994. http://dx.doi.org/10.3390/life11090994.
Texto completo da fonteSpaulding, Hannah R., Tiffany Quindry, Kayleen Hammer, John C. Quindry e Joshua T. Selsby. "Nutraceutical and pharmaceutical cocktails did not improve muscle function or reduce histological damage in D2-mdx mice". Journal of Applied Physiology 127, n.º 4 (1 de outubro de 2019): 1058–66. http://dx.doi.org/10.1152/japplphysiol.00162.2019.
Texto completo da fonteMartins-Bach, A., E. Araujo, B. Matot, Y. Fromes, P. Baudin, I. Richard e P. Carlier. "Nuclear magnetic resonance relaxometry characterization of D2-mdx mice". Neuromuscular Disorders 27 (outubro de 2017): S124. http://dx.doi.org/10.1016/j.nmd.2017.06.120.
Texto completo da fonteWard, Christopher W., Frederick Sachs, Ernest D. Bush e Thomas M. Suchyna. "GsMTx4-D provides protection to the D2.mdx mouse". Neuromuscular Disorders 28, n.º 10 (outubro de 2018): 868–77. http://dx.doi.org/10.1016/j.nmd.2018.07.005.
Texto completo da fontePandeya, Sarbesh R., Janice A. Nagy, Daniela Riveros, Carson Semple, Rebecca S. Taylor, Benjamin Sanchez e Seward B. Rutkove. "Relationships between in vivo surface and ex vivo electrical impedance myography measurements in three different neuromuscular disorder mouse models". PLOS ONE 16, n.º 10 (29 de outubro de 2021): e0259071. http://dx.doi.org/10.1371/journal.pone.0259071.
Texto completo da fonteEnglish, Katherine G., Andrea L. Reid, Adrienne Samani, Gerald J. F. Coulis, S. Armando Villalta, Christopher J. Walker, Sharon Tamir e Matthew S. Alexander. "Next-Generation SINE Compound KPT−8602 Ameliorates Dystrophic Pathology in Zebrafish and Mouse Models of DMD". Biomedicines 10, n.º 10 (26 de setembro de 2022): 2400. http://dx.doi.org/10.3390/biomedicines10102400.
Texto completo da fonteTeses / dissertações sobre o assunto "D2-mdx"
Monceau, Alexandra. "Effet de l'exercice physique, combiné ou non à une thérapie génique, sur la fonction musculaire de modèles murins de dystrophie musculaire de Duchenne". Electronic Thesis or Diss., Sorbonne université, 2021. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2021SORUS172.pdf.
Texto completo da fonteDuchenne muscular dystrophy (DMD) is a neuromuscular disease caused by mutations in the DMD gene encoding dystrophin, a protein essential for the integrity of the skeletal muscle fiber. Its absence causes increased muscle fragility, and extreme muscle weakness. Currently, there is no curative treatment, but the use of several combined therapeutic approaches seems promising. We tested the idea that regular physical activity could be a way to decrease dystrophic symptoms of skeletal muscle, especially those related to muscle function, in mice models of DMD. First, we evaluated the effect of chronic endurance exercise when combined with overexpression of Prox1, a transcription factor known to promote slower fibers in healthy muscle, which are less affected in DMD. We demonstrated that this combination allowed to decrease muscle fragility in mdx mice, the classical mouse model of DMD, and thus had the potential to stop the progression of the disease. Subsequently, we were interested in the effect of chronic endurance exercise when combined with gene therapy, which restores dystrophin expression, in D2-mdx mouse, a severe model of DMD. We showed that chronic endurance exercise decreased the efficiency of the gene therapy, by decreasing the restoration of dystrophin. Finally, we characterized the effects of chronic resistance exercise in D2-mdx mice. Our results indicate an incredibly significant improvement in muscle function in response to mechanical overload, without obvious muscle damage in this severe model
(11191884), Bohyun Ro. "IMPACT OF HEAT THERAPY ON SKELETAL MUSCLE FUNCTION IN A MODEL OF DUCHENNE MUSCULAR DYSTROPHY". Thesis, 2021.
Encontre o texto completo da fonteCapítulos de livros sobre o assunto "D2-mdx"
Kennedy, Tahnee L., e Hannah F. Dugdale. "Cardiac and Skeletal Muscle Pathology in the D2/mdx Mouse Model and Caveats Associated with the Quantification of Utrophin". In Methods in Molecular Biology, 55–66. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2772-3_4.
Texto completo da fonteKennedy, Tahnee L., e Hannah F. Dugdale. "Correction to: Cardiac and Skeletal Muscle Pathology in the D2/mdx Mouse Model and Caveats Associated with the Quantification of Utrophin". In Methods in Molecular Biology, C1. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2772-3_31.
Texto completo da fonte