Literatura científica selecionada sobre o tema "Complement-like pathway"
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Artigos de revistas sobre o assunto "Complement-like pathway"
Blandin, Stéphanie A., Eric Marois e Elena A. Levashina. "Antimalarial Responses in Anopheles gambiae: From a Complement-like Protein to a Complement-like Pathway". Cell Host & Microbe 3, n.º 6 (junho de 2008): 364–74. http://dx.doi.org/10.1016/j.chom.2008.05.007.
Texto completo da fonteDelvaeye, Mieke, Astrid DeVriese, Michael Moons, Naomi Esmon, Charles Esmon e Edward M. Conway. "Regulation of Complement Activation by Thrombomodulin." Blood 114, n.º 22 (20 de novembro de 2009): 5127. http://dx.doi.org/10.1182/blood.v114.22.5127.5127.
Texto completo da fonteZhang, Kang, Jingyan Zhang, Lei Wang, Qiang Liang, Yuhui Niu, Linlin Gu, Yanming We e Jianxi Li. "Integrative Transcriptomics and Proteomics Analysis Reveals Immune Response Process in Bovine Viral Diarrhea Virus-1-Infected Peripheral Blood Mononuclear Cells". Veterinary Sciences 10, n.º 10 (28 de setembro de 2023): 596. http://dx.doi.org/10.3390/vetsci10100596.
Texto completo da fonteKim, Sook Young, Sang Eun Lee, Man Sup Kwak e Jeon-Soo Shin. "Regulatory Role of HMGB1 on complement activation via the classical pathway (169.3)". Journal of Immunology 188, n.º 1_Supplement (1 de maio de 2012): 169.3. http://dx.doi.org/10.4049/jimmunol.188.supp.169.3.
Texto completo da fonteDe Marco Verissimo, Carolina, Heather L. Jewhurst, József Dobó, Péter Gál, John P. Dalton e Krystyna Cwiklinski. "Fasciola hepatica is refractory to complement killing by preventing attachment of mannose binding lectin (MBL) and inhibiting MBL-associated serine proteases (MASPs) with serpins". PLOS Pathogens 18, n.º 1 (10 de janeiro de 2022): e1010226. http://dx.doi.org/10.1371/journal.ppat.1010226.
Texto completo da fonteIrmscher, Sarah, Nadia Döring, Luke D. Halder, Emeraldo A. H. Jo, Isabell Kopka, Christine Dunker, Ilse D. Jacobsen et al. "Kallikrein Cleaves C3 and Activates Complement". Journal of Innate Immunity 10, n.º 2 (14 de dezembro de 2017): 94–105. http://dx.doi.org/10.1159/000484257.
Texto completo da fonteKoethe, S. M., K. E. Nelson e C. G. Becker. "Activation of the classical pathway of complement by tobacco glycoprotein (TGP)." Journal of Immunology 155, n.º 2 (15 de julho de 1995): 826–35. http://dx.doi.org/10.4049/jimmunol.155.2.826.
Texto completo da fonteLee, Garam, Yonghyan Nam, Manu Shivakumar, Apoorva Joshi, Weixuan Fu, Rebecca Anne Simmons, Paul Wang, Dokyoon Kim e Sara Elizabeth Pinney. "A Novel Graph Based Semi-Supervised Learning Approach to Identify Pathways Contributing to the Development of Diabetes and Obesity". Journal of the Endocrine Society 5, Supplement_1 (1 de maio de 2021): A656—A657. http://dx.doi.org/10.1210/jendso/bvab048.1339.
Texto completo da fonteGyörffy, Balázs A., Judit Kun, György Török, Éva Bulyáki, Zsolt Borhegyi, Péter Gulyássy, Viktor Kis et al. "Local apoptotic-like mechanisms underlie complement-mediated synaptic pruning". Proceedings of the National Academy of Sciences 115, n.º 24 (29 de maio de 2018): 6303–8. http://dx.doi.org/10.1073/pnas.1722613115.
Texto completo da fonteStaels, F., W. Meersseman, P. Stordeur, K. Willekens, S. Van Loo, A. Corveleyn, I. Meyts, G. Meyfroidt e R. Schrijvers. "Terminal Complement Pathway Deficiency in an Adult Patient with Meningococcal Sepsis". Case Reports in Immunology 2022 (23 de maio de 2022): 1–6. http://dx.doi.org/10.1155/2022/9057000.
Texto completo da fonteTeses / dissertações sobre o assunto "Complement-like pathway"
Zmarlak, Natalia Marta. "Regulation of immune signalling in the malaria mosquito vector, Anopheles : the secreted mosquito leucine-rich repeat protein APL1C is a pathogen binding factor essential for immunity to Plasmodium ookinetes and sporozoites". Electronic Thesis or Diss., Sorbonne université, 2021. http://www.theses.fr/2021SORUS053.
Texto completo da fonteAnopheles mosquito is a vector of Plasmodium parasite, the causative agent of malaria. The Anopheles leucine-rich repeat (LRR) proteins were described as key antagonists of Plasmodium parasites in Anopheles mosquito midgut. APL1C (Anopheles Plasmodium-responsive factor) is a representative of LRR members which specifically protects against rodent malaria parasites by stabilizing the complement-like protein TEP1. By combining cell biology with functional genomic approaches, this study shows that mosquito bloodmeal induce the presence of an extracellular layer of APL1C protein surrounding the midgut beneath of the basal lamina. Consistently with the formation of this layer, APL1C binds to the ookinetes that emerged on the basal side of the midgut. This presence occurs independently from TEP1 function, requires the contribution of the phagocytic cells and nitration pathway. In addition, APL1C defence function is not restricted to the ookinete in the midgut but it also acts against the latest Plasmodium stage, the sporozoites. APL1C inhibits salivary glands infection prevalence, and consistently, it also binds to the surface of the sporozoites in the hemocoel. However, unlike to the midgut stages, anti-sporozoites APL1C-dependent mechanism involves different partners. Moreover, RNAseq study revealed APL1C gene targets, including genes with immune-like function. These results generate novel biological insight for the function of APL1C, and probably other LRR family members, as a pathogen recognition receptor inducing immune response against pathogens that come in contact with mosquito hemolymph compartment
Livros sobre o assunto "Complement-like pathway"
Noris, Marina, e Tim Goodship. The patient with haemolytic uraemic syndrome/thrombotic thrombocytopenic purpura. Editado por Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0174.
Texto completo da fonteCapítulos de livros sobre o assunto "Complement-like pathway"
Kojouharova, Mihaela. "Classical Complement Pathway Component C1q: Purification of Human C1q, Isolation of C1q Collagen-Like and Globular Head Fragments and Production of Recombinant C1q—Derivatives. Functional Characterization". In The Complement System, 25–42. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-724-2_3.
Texto completo da fonteKumar Chatterjee, Swapan, e Snigdha Saha. "Glycan and Its Role in Combating COVID-19". In Biotechnology to Combat COVID-19 [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97240.
Texto completo da fonteMichael, A., Newton Hyuna Yang, Patricia A. Gorman Ian Tomlinson e Rebecca R. Roylance. "A Statistical Approach to Modeling Genomic Aberrations in Cancer Cells". In Bayesian Statistics 7, 293–305. Oxford University PressOxford, 2003. http://dx.doi.org/10.1093/oso/9780198526155.003.0016.
Texto completo da fonteAraújo, Kathleen. "French Nuclear Energy: Concentrated Power". In Low Carbon Energy Transitions. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199362554.003.0008.
Texto completo da fonteTuryamuhika, Laban, Agaba Bosco, Asiimwe Moses, Musinguzi Benson e Okek Erick. "Functioning and Control of Phagocytosis". In Phagocytosis - Main Key of Immune System. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.110511.
Texto completo da fonteTrabalhos de conferências sobre o assunto "Complement-like pathway"
Povelones, Michael. "Specificity of complement-like pathway activation inAnopheles gambiae". In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.92449.
Texto completo da fonteRelatórios de organizações sobre o assunto "Complement-like pathway"
Friedman, Haya, Julia Vrebalov, James Giovannoni e Edna Pesis. Unravelling the Mode of Action of Ripening-Specific MADS-box Genes for Development of Tools to Improve Banana Fruit Shelf-life and Quality. United States Department of Agriculture, janeiro de 2010. http://dx.doi.org/10.32747/2010.7592116.bard.
Texto completo da fonteWeiss, David, e Neil Olszewski. Manipulation of GA Levels and GA Signal Transduction in Anthers to Generate Male Sterility. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7580678.bard.
Texto completo da fonte