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Livros sobre o tema "Cancer Age factors"

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Publicado: 4 de junho de 2021
Última modificação: 30 de janeiro de 2023

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1

1932-, Macieira-Coelho Alvaro, e Nordenskjöld Bo, eds. Cancer and aging. Boca Raton: CRC Press, 1990.

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2

Zhong lao nian ren ai zheng jiang zuo: Cancer discussion for middle-old age. Taibei Shi: Taiwan xin sheng bao chu ban bu, 1986.

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3

Cancer 2000 Task Force. Expert Panel on Palliative Care. Report to Cancer 2000 Task Force. [Ontario: The Panel, 1991.

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4

International Association of Biomedical Gerontology. International Congress, ed. Aging, cancer and age-related diseases: Common mechanisms? Boston, MA: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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5

Cancer risk by social class and occupation: A survey of 109,000 cancer cases among Finns of working age. Basel: Karger, 1995.

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6

V, Zenser Terry, e Coe Rodney M, eds. Cancer and aging: Progress in research and treatment. New York: Springer Pub. Co., 1989.

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7

Carcinogenesis and aging. Boca Raton, Fla: CRC Press, 1987.

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8

United States. Congress. House. Select Committee on Aging. Skin cancer: Older Americans at risk : hearing before the Select Committee on Aging, House of Representatives, Ninety-ninth Congress, second session, May 21, 1986. Washington: U.S. G.P.O., 1986.

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9

Gosney, Margot, e Keith Bellizzi. Cancer and aging handbook. Hoboken, N.J: Wiley-Blackwell, 2012.

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10

Oktay, Julianne S. Breast cancer in the life course: Women's experiences. New York: Springer Pub. Co., 1991.

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11

Hunt, Jennie P. Breast cancer screening in women ages 40-49: January 1985 through November 1996 : 334 selected citations. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Library of Medicine, Reference Section, 1996.

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12

Frank, Steven A. Dynamics of cancer: Incidence, inheritance, and evolution. Princeton, N.J: Princeton University Press, 2007.

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13

1919-, Pullman Bernard, Ts'o, Paul O. P. 1929-, Schneider Edward L, Fondation Edmond de Rothschild e National Institute for Cancer Research., eds. Interrelationship among aging, cancer, and differentiation: Proceedings of the Eighteenth Jerusalem Symposium on Quantum Chemistry and Biochemistry held in Jerusalem, Israel, April 29-May 2, 1985. Dordrecht: D. Reidel Pub. Co., 1985.

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14

The cancer generation: Baby boomers facing a perfect storm. Monroe, Me: Common Courage Press, 2009.

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15

Schrijvers, Dirk. ESMO handbook of cancer in the senior patient. New York: Informa Healthcare, 2010.

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16

Society, Canadian Cancer, Cancer Care Ontario e Public Health Agency of Canada., eds. Cancer in young adults in Canada. Toronto: Cancer Care Ontario, 2006.

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17

Aging, National Institute on, ed. Cancer facts for people over 50. [Bethesda, Md.]: National Institute on Aging, U.S. Dept. of Health and Human Services, National Institutes of Health, 2005.

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18

Aging, National Institute on, ed. Cancer facts for people over 50. [Bethesda, Md.?]: National Institute on Aging, 2000.

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19

ed, Likhachev A., Anisimov Vladimir N. ed, Montesano R. ed e International Agency for Research on Cancer., eds. Age-related factors in carcinogenesis: Proceedings of a symposium. Lyon: International Agency for Research on Cancer, 1985.

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20

Age-Related Factors in Carcinogenesis (International Agency for Research on Cancer). IARC Scientific Publications, 1986.

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21

National military strategy of the United States of America 1995: A strategy of flexible and selective engagement. For sale by the Supt. of Docs, 1995.

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22

Jeff, Abrams, National Cancer Institute (U.S.), National Institutes of Health (U.S.). Office of Research on Women's Health e National Institute of Child Health and Human Development (U.S.), eds. Breast cancer in younger women: Proceedings of a conference held at the National Institutes of Health, Bethesda, Maryland, January 28, 1993. Bethesda, MD: The Institute, 1994.

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23

A, Likhachev, Anisimov Vladimir N, Montesano R, International Agency for Research on Cancer. e Nauchno-issledovatelʹskiĭ institut onkologii im. N.N. Petrova., eds. Age-related factors in carcinogenesis: Proceedings of symposium organized by the IARC and the N.N. Petrov Research Institute of Oncology, held in Leningrad, 7-9 December 1983. Lyon: International Agency for Research on Cancer, 1985.

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24

S, Burger Stasia, New Jersey. Cancer Epidemiology Services. e New Jersey. Cancer Surveillance Program., eds. Cancer among older adults in New Jersey, 1994-1998. Trenton, NJ: New Jersey Dept. of Health and Senior Services, Cancer Epidemiology Services, Cancer Surveillance Program, 2002.

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25

Gosney, Margot, e Keith M. Bellizzi. Cancer and Aging Handbook: Research and Practice. Wiley & Sons, Incorporated, John, 2012.

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26

Gosney, Margot, e Keith M. Bellizzi. Cancer and Aging Handbook: Research and Practice. Wiley & Sons, Incorporated, John, 2012.

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27

Gosney, Margot, e Keith M. Bellizzi. Cancer and Aging Handbook: Research and Practice. Wiley & Sons, Incorporated, John, 2013.

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28

Sue, Yang Stringner, Warner Huber R e Workshop on the Underlying Molecular, Cellular, and Immunological Factors in Age-Related Cancers (1990 : Annapolis, Md.), eds. The Underlying molecular, cellular, and immunological factors in cancer and aging. New York: Plenum Press, 1993.

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29

Sue, Yang Stringner, e Warner Huber R, eds. The Underlying molecular, cellular, and immunologicalfactors in cancer and aging. New York: Plenum Press, 1993.

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30

Coe, Rodney M., e Terry V. Zenser. Cancer and Aging: Progress in Research and Treatment. Springer Pub Co, 1989.

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31

Wilson, Kathryn M., e Lorelei Mucci. Prostate Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0020.

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Prostate cancer is among the most commonly diagnosed cancers among men, ranking second in cancer globally and first in Western countries. There are marked variations in incidence globally, and its incidence must be interpreted in the context of diagnostic intensity and screening. The uptake of prostate-specific antigen screening since the 1990s has led to dramatic increases in incidence in many countries, resulting in an increased proportion of indolent cancers that would never have come to light clinically in the absence of screening. Risk factors differ when studying prostate cancer overall versus advanced disease. Older age, African ancestry, and family history are established risk factors for prostate cancer. Obesity and smoking are not associated with risk overall, but are associated with increased risk of advanced prostate cancer. Several additional lifestyle factors, medications, and dietary factors are now emerging as risk factors for advanced disease.
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32

Walter, Carolyn A., e Julianne S. Oktay. Breast Cancer in the Life Course: Women's Experiences (Springer Series on Social Work). Springer Pub Co, 1992.

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33

National Institutes of Health Consensus Conference on Breast Cancer Screening for Women Ages 40-49: Proceedings of a conference held at the National Institutes ... (Journal of the National Cancer Institute). National Cancer Institute, 1997.

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34

Interrelationship Among Aging, Cancer and Differentiation (Jerusalem Symposia). Springer, 1985.

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35

Gosney, Margot, e Keith M. Bellizzi. Cancer and Aging Handbook: Research and Practice. Wiley & Sons, Incorporated, John, 2012.

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36

Gosney, Margot, e Keith M. Bellizzi. Cancer and Aging Handbook: Research and Practice. Wiley & Sons, Limited, John, 2013.

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37

Kazak, Anne E., Lori S. Wiener, Andrea Farkas Patenaude, Maryland Pao e Mary Jo Kupst. Pediatric Psycho-Oncology: A Quick Reference on the Psychosocial Dimensions of Cancer Symptom Management. Oxford University Press, Incorporated, 2015.

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38

Richiardi, Lorenzo, Giovenale Moirano e Pagona Lagiou. Testicular Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0021.

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Testicular cancer is highly curable and, though relatively rare, it is the most common cancer among young men. Incidence of testicular cancer has been increasing constantly around the world since the beginning of the twentieth century, but factors responsible for the rise in incidence remain enigmatic. Only few risk factors for testicular cancer are established, including age, ethnic group, cryptorchidism and hypospadias, contralateral testicular cancer, family history, and height. While analytic epidemiologic research has provided numerous etiologic clues, many of them remain tentative. Overwhelming evidence indicates the fundamental importance of environmental factors in the etiology of this enigmatic cancer. Prenatal exposures seem to be instrumental in shaping the risk of testicular cancer, but postnatal exposures acting in childhood, adolescence, and very early adulthood are also important. Testicular cancer has also a strong genetic component that is studied through international collaborations and genome-wide association studies.
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39

Key, Timothy J., e Alison J. Price. Epidemiology of prostate cancer. Editado por James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0058.

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Prostate cancer is the second most common malignancy and the sixth most common cause of cancer death for men worldwide. The highest incidence and mortality rates are in populations that originated in Africa, such as African Americans. Rates are also high in Western countries and generally low in East and South Asia. Incidence rates are increasing in some countries which until recently had low rates, but are not changing much in countries which already have high rates. The only well-established risk factors are increasing age, African ancestry, family history of the disease, and certain genetic factors, none of which is modifiable. Many potential risk factors have been investigated in epidemiological studies and randomized trials. Observational studies have shown that prostate cancer risk is positively associated with the plasma concentration of insulin-like growth factor-I, but is not strongly associated with testosterone or other sex hormones. Studies of nutritional factors suggest that risk may be higher in men with a high intake of animal foods and dairy products, but this relationship is not clear enough to be considered as established. Some studies of other nutritional factors such as fat, lycopene and other carotenoids, vitamin D, vitamin E and selenium have suggested possible associations, but overall do not show any clear relationships. Research on other possible risk factors has shown a small positive association of risk with height, but little association with obesity, smoking or alcohol intake, and evidence on sexual behaviour and sexually transmitted infections is inconclusive. Further research is needed, particularly to determine whether potential risk factors may be related more to aggressive than to indolent prostate cancer.
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40

Use of the Health Belief Model in determining mammography screening practice in older women. 1991.

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41

Tamimi, Rulla, Susan Hankinson e Pagona Lagiou. Breast Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0016.

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Most of the established reproductive risk factors for breast cancer, like age at menarche or parity, are not appropriate for public health intervention. Several lines of evidence, like the associations with birthweight and early exposure to radiation, support an important influence of early-life events on subsequent breast cancer risk. The best established modifiable risk factors for the disease include postmenopausal hormone use, moderate alcohol intake, and adult weight gain. More recently, we have come to appreciate that instead of a single disease, breast cancer is rather a heterogeneous group of subtypes with different etiologies. Yet the wealth of available epidemiologic information can be synthesized into a consistent and testable, albeit still hypothetical, causal model. With our increasing knowledge on the relation between endogenous hormones and breast cancer, and the development of selective estrogen receptor modulators, as well as aromatase inhibitors, chemoprevention will likely become more common in the future.
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42

Attitudes, perceptions and coping skills of long-term breast cancer survivors. 1989.

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43

Attitudes, perceptions and coping skills of long-term breast cancer survivors. 1992.

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44

Capitman, John A., Sarita Bhalotra e Mathilda Ruwe. Cancer And Elders Of Color: Opportunities of Reducing Health Disparities : Evidence Review and Recommendations for Research and Policy. Ashgate Publishing, 2005.

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45

Tworoger, Shelley S., Amy L. Shafrir e Susan E. Hankinson. Ovarian Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0046.

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Worldwide, ovarian cancer is the seventh most commonly diagnosed cancer and the eighth most common cause of death from cancer. In 2012, 239,000 women were diagnosed with ovarian cancer and 152,000 women died of the disease worldwide. In the United States in 2015, an estimated 21,290 women were newly diagnosed with ovarian cancer and 14,180 died from the disease. Both incidence and mortality have decreased over time in the United States, with a 1.6% and 2.1% annual decrease, respectively, from 2003 to 2012. Ovarian cancers can arise from epithelial, germ, or stromal cells, although about 90% are epithelial in origin. Risk factors best confirmed to increase risk of ovarian cancer include age and a family history of ovarian cancer, while parity, oral contraceptive use, and tubal ligation decrease risk. Several etiologic pathways, including hormonal and inflammatory pathways, have garnered substantial support from both epidemiologic and laboratory studies, although many questions remain.
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46

Tangen, Catherine M., Marian L. Neuhouser e Janet L. Stanford. Prostate Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0053.

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Prostate cancer is the most common solid tumor and the second leading cause of cancer-related mortality in American men. Worldwide, prostate cancer ranks second and fifth as a cause of cancer and cancer deaths, respectively. Despite the international burden of disease due to prostate cancer, its etiology is unclear in most cases. Established risk factors include age, race/ancestry, and family history of the disease. Prostate cancer has a strong heritable component, and genome-wide association studies have identified over 110 common risk-associated genetic variants. Family-based sequencing studies have also found rare mutations (e.g., HOXB13) that contribute to prostate cancer susceptibility. Numerous environmental and lifestyle factors (e.g., obesity, diet) have been examined in relation to prostate cancer incidence, but few modifiable exposures have been consistently associated with risk. Some of the variability in results may be related to etiological heterogeneity, with different causes underlying the development of distinct disease subgroups.
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47

Kitahara, Cari M., Arthur B. Schneider e Alina V. Brenner. Thyroid Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0044.

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Thyroid cancer, once considered relatively uncommon in the general population, is now the eighth most commonly diagnosed cancer among women worldwide, and the third most common cancer among women under 45 years of age. The incidence is substantially higher in women than men (3:1 ratio); this differential is highest between ages 15 and 39 and declines with age. Nearly all thyroid cancers derive from the follicular epithelium, and the most common histological type is papillary thyroid cancer (PTC). Incidence of thyroid cancer has been increasing in many countries since the early 1980s. This trend appears to be attributable to a combination of diagnostic changes, improvements in the detection and diagnosis of smaller PTCs, and changes in the prevalence of environmental factors. While less common, the incidence of larger, more advanced-stage PTCs has increased at a similar rate to that of smaller PTCs.
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48

Frisch, Morten. Penile Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0055.

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Penile cancers are rare primary malignancies located on the glans, foreskin, or shaft of the penis, excluding the urethra. The vast majority of penile cancers are epithelial tumors representing histological subtypes of squamous cell carcinoma (SCC). Most penile SCCs are believed to develop through pre-invasive lesions known as penile intraepithelial neoplasia and penile carcinoma in situ. They account for 0.1%–0.3% of all incident cancers (excluding non-melanoma skin cancers) in the United States and other developed countries and up to 1% of all cancers in some countries in sub-Saharan Africa. Penile cancers are rare in men younger than 40 years, and are typically diagnosed among men above age 60. The two most important risk factors are pathological phimosis and infection with high-risk types of human papillomaviruses (HPV), both of which are preventable conditions.
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49

Paraskeva, Nicole, Alex Clarke e Diana Harcourt. Altered Appearance from Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190655617.003.0007.

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Abstract: This chapter delineates various appearance alterations that can result from cancer and its treatment. It focuses on describing the impact of appearance changes involving weight, scarring from surgery, alopecia, ascites, and lymphedema. It also examines the influence of patient-related factors (i.e., gender, age) and treatment-related factors on appearance-related distress. Various models of adjustment to alterations in appearance are presented with particular attention to predisposing factors, intervening cognitive processes, and measurable outcomes identified by large-scale research studies conducted by the Appearance Research Collaboration. Finally, the chapter identifies gaps in knowledge and directions for future research needed to advance the understanding of an individual’s experiences of living with an altered appearance due to cancer.
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50

Carlo, George, e Martin Schram. Cell Phones: Invisible Hazards in the Wireless Age: An Insider's Alarming Discoveries about Cancer and Genetic Damage. Carroll & Graf, 2002.

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