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Bibliografias temáticas / Bone resorption – Molecular aspects / Livros

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Livros sobre o tema "Bone resorption – Molecular aspects"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 1 de fevereiro de 2022

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1

Wackym, Phillip A. Molecular temporal bone pathology, parts III and IV. Philadelphia, PA: Lippincott-Raven, 1998.

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2

Raisz, Lawrence G. (Lawrence Gideon), 1925-, Martin T. John e ScienceDirect (Online service), eds. Principles of bone biology. 3^a ed. Amsterdam: Elsevier, 2008.

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3

Rosen, Vicki. The cellular and molecular basis of bone formation and repair. New York: Springer, 1995.

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4

1957-, Thies Robert Scott, ed. The cellular and molecular basis of bone formation and repair. Austin: R.G. Landes, 1995.

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5

Dietz, Georg. Calcium hydroxide and bone regeneration: Odontological aspects of induced osteogenesis in experiment and clinical practice. München: G. Dietz, 1998.

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6

Cardew, Gail. The molecular basis of skeletogenesis. Chichester: Wiley, 2001.

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7

(Foreword), G. A. Rodan, Felix Bronner (Editor) e Mary C. Farach-Carson (Editor), eds. Bone Formation (Topics in Bone Biology). Springer, 2003.

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8

Gurmit, Singh, e Orr F. William, eds. Bone metastasis and molecular mechanisms: Pathophysiology. Dordrecht: Kluwer Academic, 2004.

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9

Orr, William, e Gurmit Singh. Bone Metastasis and Molecular Mechanisms: Pathophysiology. Springer-Verlag New York Inc., 2004.

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10

Singh, Gurmit. Bone Metastasis and Molecular Mechanisms: Pathophysiology. Springer, 2010.

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11

Masaki, Noda, ed. Cellular and molecular biology of bone. San Diego: Academic Press, 1993.

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12

(Editor), Hong-Wen Deng, Yao-zhong Liu (Editor), Chun-Yuan Guo (Editor) e Di Chen (Editor), eds. Current Topics in Bone Biology. World Scientific Publishing Company, 2005.

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13

Hong-wen, Deng, e Liu Yao-zhong, eds. Current topics in bone biology. Singapore: World Scientific, 2005.

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14

Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1986.

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15

Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1989.

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16

Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1989.

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17

The effects of back exercise on bone metabolism in early postmenopausal women. 1987.

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18

The effects of back exercise on bone metabolism in early postmenopausal women. 1987.

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19

The effects of back exercise on bone metabolism in early postmenopausal women. 1985.

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20

Bone mass, menstrual abnormalities, dietary intake, and body composition in classical ballerinas. 1989.

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21

(Editor), Geoffrey Burnstock, e Timothy R. Arnett (Editor), eds. Nucleotides and Regulation of Bone Cell Function. CRC, 2006.

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22

Bone Metastasis and Molecular Mechanisms: Pathophysiology (Cancer Metastasis - Biology and Treatment). Springer, 2004.

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23

Theis, R. Scott, e Vicki Rosen. Cellular & Molec Basis Bone Format (Molecular Biology Intelligence Unit). Springer, 1996.

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24

Principles of Bone Biology. Academic Press, 2002.

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25

Molecular Biology of Hematopoiesis 5. Springer, 2012.

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26

G, Abraham Nader, e Symposium on the Molecular Biology of Hematopoiesis (9th : 1995 : Genoa, Italy), eds. Molecular biology of hematopoiesis 5. New York: Plenum Press, 1996.

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27

The effect of a weight training program on the bone density of women aged 40-50 years. 1994.

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28

(Editor), John P. Bilezikian, Lawrence G. Raisz (Editor) e Gideon A. Rodan (Editor), eds. Principles of Bone Biology, Second Edition (2 Vol. Set). 2^a ed. Academic Press, 2001.

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29

P, Bilezikian John, Raisz Lawrence G. 1925- e Rodan Gideon A, eds. Principles of bone biology. San Diego: Academic Press, 1996.

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30

The effects of high impact exercise versus low impact exercise on bone density in postmenopausal women. 1990.

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31

(Editor), Joseph A. Buckwalter, Michael G. Ehrlich (Editor), Linda J. Sandell (Editor) e Stephen B. Trippel (Editor), eds. Skeletal Growth and Development: Clinical Issues and Basic Science Advances (The Symposium Series). American Academy of Orthopaedic Surgeons, 1998.

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32

A, Buckwalter Joseph, American Academy of Orthopaedic Surgeons., National Institutes of Health (U.S.) e Orthopaedic Research and Education Foundation., eds. Skeletal growth and development: Clinical issues and basic science advances : workshop, Howey-in-the-Hills, Florida, May 1997. Rosemont, IL: American Academy of Orthopaedic Surgeons, 1998.

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33

Osteoporosis: Methods and Protocols (Methods in Molecular Biology). Humana Press, 2008.

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34

Skeletal Growth Factors. Lippincott Williams & Wilkins, 2000.

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35

Genetics for Orthopedic Surgeons. Remedica Publishing, 2002.

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36

Sheridan, Peter J., e Kenneth Blum. Steroid Receptors and Disease: Cancer, Autoimmune, Bone, and Circulatory Disorders. Marcel Dekker Inc, 1988.

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37

1941-, Sheridan Peter J., Blum Kenneth e Trachtenberg Michael C. 1941-, eds. Steroid receptors and disease: Cancer, autoimmune, bone, and circulatory disorders. New York: Dekker, 1988.

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38

Cytogenetics, FISH and Molecular Testing in Hematologic Malignancies. Informa Healthcare, 2008.

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39

The Molecular Basis of Skeletogenesis No. 232. Wiley, 2001.

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40

Thomas, Ranjeny, e Andrew P. Cope. Pathogenesis of rheumatoid arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0109.

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Resumo:

In depth molecular and cellular analysis of synovial tissue and fluid from patients with rheumatoid arthritis has provided important insights into understanding disease pathogenesis. Advances in the 1980s and 1990s included modern cloning strategies, sensitive and specific assays for inflammatory mediators, production of high-affinity neutralizing monoclonal antibodies, advances in flow cytometry, and gene targeting and transgenic strategies in rodents. In the 21st century, technological platforms offer unparalleled opportunities for systematic and unbiased interrogation of the disease process at a whole-genome level. Here we describe the key molecular and cellular characteristics of the inflamed synovium and how infiltrating cells get there. With this background, we outline current concepts of the different phases of disease, how the first phase of genetic susceptibility evolves into autoimmunity, triggered by the exposome, prior to the onset of clinically apparent inflammatory disease. We then describe the pathways that actively contribute to this early inflammatory phase and document the key effector cells and molecules of the innate and adaptive immune systems that orchestrate and maintain chronic synovial inflammatory responses. We summarize how this inflammatory milieu translates to cartilage destruction and bone resorption in synovial joints, and conclude by reviewing those factors in inflamed synovium that promote immune homeostasis.

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