Livros sobre o tema "Beta-Amyloid peptides"

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1

Indu, Kheterpal, e Wetzel Ronald, eds. Amyloid, prions, and other protein aggregates Part C. Amsterdam: Elsevier/Academic, 2006.

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2

Indu, Kheterpal, e Wetzel Ronald, eds. Amyloid, prions, and other protein aggregates Part B. Amsterdam: Elsevier/Academic, 2006.

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3

C, Dowler Brynn, ed. Endocytosis: Structural components, functions, and pathways. Hauppauge, N.Y: Nova Science Publishers, 2010.

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4

Tanzi, Rudolph E. Decoding darkness: The search for the genetic causes of Alzheimer's disease. Cambridge, Mass: Perseus Pub., 2000.

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5

A, Nixon Ralph, Banner Carl D. B e New York Academy of Sciences., eds. Proteases and protease inhibitors in Alzheimer's disease pathogenesis. New York: NewYork Academy of Sciences, 1992.

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6

B, Banner Carl D., e Nixon Ralph A, eds. Proteases and protease inhibitors in Alzheimer's disease pathogenesis. New York, N.Y: New York Academy of Sciences, 1992.

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7

Wetzel, Ronald, e Indu Kheterpal. Amyloid, Prions, and Other Protein Aggregates, Part C. Elsevier Science & Technology Books, 2006.

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8

(Editor), Ronald Wetzel, e Indu Kheterpal (Editor), eds. Amyloid, Prions, and Other Protein Aggregates, Part C, Volume 413 (Methods in Enzymology). Academic Press, 2006.

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9

Abelson, John N., Ronald Wetzel e Melvin I. Simon. Amyloid, Prions, and Other Protein Aggregates. Elsevier Science & Technology Books, 1999.

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10

(Editor), Ronald Wetzel, e Indu Kheterpal (Editor), eds. Amyloid, Prions, and Other Protein Aggregates, Part B, Volume 412 (Methods in Enzymology). Academic Press, 2006.

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11

(Editor), Ronald Wetzel, e Indu Kheterpal (Editor), eds. Amyloid, Prions, and Other Protein Aggregates, Part B, Volume 412 (Methods in Enzymology). Academic Press, 2006.

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12

The nature and origin of amyloid fibrils. Chichester: Wiley, 1996.

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13

Symposium, CIBA Foundation. The Nature and Origin of Amyloid Fibrils. John Wiley & Sons, 1996.

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14

Alzheimers Disease Insights Into Low Molecular Weight And Cytotoxic Aggregates From In Vitro And Computer Experiments Molecular Basis Of Amyloidbeta Protein Aggregation And Fibril Formation. Imperial College Press, 2011.

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15

(Editor), Robin Harris, e Falk Fahrenholz (Editor), eds. Alzheimer's Disease: Cellular and Molecular Aspects of Amyloid beta (Subcellular Biochemistry). Springer, 2004.

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16

Alzheimer's Disease: A Compendium of Current Theories (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2001.

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17

Saido, Takaomi. A-Beta Metabolism and Alzheimer's Disease. Landes Bioscience, 2003.

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18

Chen, Shiouh-Yi. Neuropathology of beta-amyloid peptide (25-35). 1995.

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19

Safar, Jiri G. Prion Paradigm of Human Neurodegenerative Diseases Caused by Protein Misfolding. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0005.

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Data accumulated from different laboratories argue that a growing number of proteins causing neurodegeneration share certain characteristics with prions. Prion-like particles were produced from synthetic amyloid beta (Aβ‎) peptides of Alzheimer’s disease (AD), from recombinant α‎-synuclein linked to Parkinson’s disease (PD), and from recombinant tau associated with frontotemporal dementias (FTD). Evidence from human prions reveals that variable disease phenotypes, rates of propagation, and targeting of different brain structures are determined by distinct conformers (strains) of pathogenic prion protein. Recent progress in the development of advanced biophysical tools identified the structural characteristics of Aβ‎ in the brain cortex of phenotypically diverse AD patients and thus allowed an investigation of the prion paradigm of AD. The findings of distinctly structured strains of human brain Aβ‎, forming a unique spectrum of oligomeric particles in the cortex of rapidly progressive cases, implicates these structures in variable rates of propagation in the brain.
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20

Huang, Tze-Hsien Jackson. Alzheimer amyloid-[beta] peptide aggregation: Alternate products of fibril formation. 2001.

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21

Abeta Peptide and Alzheimer's Disease: Celebrating a Century of Research. Springer, 2006.

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22

Tanzi, Rudolph E., Rudolph Tanzi e Ann B. Parson. Decoding Darkness: The Search for the Genetic Causes of Alzheimer's Disease. Basic Books, 2001.

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23

Tanzi, Rudolph E., e Ann B. Parson. Decoding Darkness: The Search for the Genetic Causes of Alzheimer's Disease. Basic Books, 2008.

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24

Alzheimer amyloid-[beta] peptide aggregation: A potential in vivo model of ealry aggregate formation. Ottawa: National Library of Canada, 2002.

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