Literatura científica selecionada sobre o tema "Autophagic lysosome reformation (ALR)"
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Artigos de revistas sobre o assunto "Autophagic lysosome reformation (ALR)"
Zhang, Lu, Yu Fang, Xuan Cheng, Yajun Lian, Hongliang Xu, Zhaoshu Zeng e Hongcan Zhu. "TRPML1 Participates in the Progression of Alzheimer’s Disease by Regulating the PPARγ/AMPK/Mtor Signalling Pathway". Cellular Physiology and Biochemistry 43, n.º 6 (2017): 2446–56. http://dx.doi.org/10.1159/000484449.
Texto completo da fonteChen, Yang, e Li Yu. "Autophagic lysosome reformation". Experimental Cell Research 319, n.º 2 (janeiro de 2013): 142–46. http://dx.doi.org/10.1016/j.yexcr.2012.09.004.
Texto completo da fonteChen, Yang, e Li Yu. "Recent progress in autophagic lysosome reformation". Traffic 18, n.º 6 (5 de maio de 2017): 358–61. http://dx.doi.org/10.1111/tra.12484.
Texto completo da fonteGan, Qiwen, Xin Wang, Qian Zhang, Qiuyuan Yin, Youli Jian, Yubing Liu, Nan Xuan et al. "The amino acid transporter SLC-36.1 cooperates with PtdIns3P 5-kinase to control phagocytic lysosome reformation". Journal of Cell Biology 218, n.º 8 (24 de junho de 2019): 2619–37. http://dx.doi.org/10.1083/jcb.201901074.
Texto completo da fonteRong, Yueguang, Mei Liu, Liang Ma, Wanqing Du, Hanshuo Zhang, Yuan Tian, Zhen Cao et al. "Clathrin and phosphatidylinositol-4,5-bisphosphate regulate autophagic lysosome reformation". Nature Cell Biology 14, n.º 9 (12 de agosto de 2012): 924–34. http://dx.doi.org/10.1038/ncb2557.
Texto completo da fonteChang, Jaerak, Seongju Lee e Craig Blackstone. "Spastic paraplegia proteins spastizin and spatacsin mediate autophagic lysosome reformation". Journal of Clinical Investigation 124, n.º 12 (3 de novembro de 2014): 5249–62. http://dx.doi.org/10.1172/jci77598.
Texto completo da fonteRong, Y., C. K. McPhee, S. Deng, L. Huang, L. Chen, M. Liu, K. Tracy, E. H. Baehrecke, L. Yu e M. J. Lenardo. "Spinster is required for autophagic lysosome reformation and mTOR reactivation following starvation". Proceedings of the National Academy of Sciences 108, n.º 19 (25 de abril de 2011): 7826–31. http://dx.doi.org/10.1073/pnas.1013800108.
Texto completo da fonteMagalhaes, Joana, Matthew E. Gegg, Anna Migdalska-Richards, Mary K. Doherty, Phillip D. Whitfield e Anthony H. V. Schapira. "Autophagic lysosome reformation dysfunction in glucocerebrosidase deficient cells: relevance to Parkinson disease". Human Molecular Genetics 25, n.º 16 (4 de julho de 2016): 3432–45. http://dx.doi.org/10.1093/hmg/ddw185.
Texto completo da fonteLiu, Xu, e Daniel J. Klionsky. "Regulation of autophagic lysosome reformation by kinesin 1, clathrin and phosphatidylinositol-4,5-bisphosphate". Autophagy 14, n.º 1 (21 de dezembro de 2017): 1–2. http://dx.doi.org/10.1080/15548627.2017.1386821.
Texto completo da fonteSharma, Prashant, Jenny Serra-Vinardell, Wendy J. Introne e May Christine V. Malicdan. "Role of lysosomal trafficking regulator in autophagic lysosome reformation in neurons: a disease perspective". Neural Regeneration Research 19, n.º 5 (22 de setembro de 2023): 957–58. http://dx.doi.org/10.4103/1673-5374.385298.
Texto completo da fonteTeses / dissertações sobre o assunto "Autophagic lysosome reformation (ALR)"
Pietri, David. "Structure and function of the C9ORF72-SMCR8-WDR41 complex and its implication for Amyotrophic Lateral Sclerosis (ALS)". Electronic Thesis or Diss., Strasbourg, 2023. http://www.theses.fr/2023STRAJ087.
Texto completo da fonteAmyotrophic lateral sclerosis (ALS or Charcot disease) is the third most common neurodegenerative disease. The main genetic cause of ALS is an expansion of GGGGCC repeats in the C9ORF72 gene which protein forms a complex with the SMCR8 and WDR41 proteins. To better understand its molecular functions, solving its structure was a main goal of my thesis. In parallel, we discovered that C9ORF72 regulates a newly described mechanism of biogenesis of newly-formed lysosomes, called autophagic lysosome reformation (ALR). This process has been extensively investigated during my thesis, in order to better understand its regulation, particularly for the regeneration of lysosomes in basal conditions and amino acid deprivation. My work reveals a new partner of the C9ORF72 complex as a novel function in lysosome biogenesis. These results could thus explain the dysfunction of lysosomes and neurodegeneration observed in ALS, which open new therapeutic ways for this devastating disease
Capítulos de livros sobre o assunto "Autophagic lysosome reformation (ALR)"
Chen, Yang, Qian Peter Su e Li Yu. "Studying Autophagic Lysosome Reformation in Cells and by an In Vitro Reconstitution System". In Methods in Molecular Biology, 163–72. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8873-0_9.
Texto completo da fonteMahapatra, Kewal Kumar, e Sujit Kumar Bhutia. "Autophagic lysosome reformation: The beginning from the end". In Autophagy Processes and Mechanisms, 153–62. Elsevier, 2024. http://dx.doi.org/10.1016/b978-0-323-90142-0.00009-8.
Texto completo da fonte