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Bibliografias temáticas / Arthritis Molecular aspects

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Literatura científica selecionada sobre o tema "Arthritis Molecular aspects"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 31 de janeiro de 2023

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Artigos de revistas sobre o assunto "Arthritis Molecular aspects"

1

Gross, D. M., e B. T. Huber. "Cellular and molecular aspects of Lyme arthritis". Cellular and Molecular Life Sciences 57, n.º 11 (outubro de 2000): 1562–69. http://dx.doi.org/10.1007/pl00000641.

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2

Chetina, E. V., e E. P. Sharapova. "Rheumatic pain management: molecular aspects". Modern Rheumatology Journal 14, n.º 1 (22 de março de 2020): 93–100. http://dx.doi.org/10.14412/1996-7012-2020-1-93-100.

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Rheumatic diseases (RDs), including osteoarthritis and rheumatoid arthritis, are non-infectious slowly progressive incurable inflammatory diseases that lead to prolonged disability due to damage to the musculoskeletal system. Pain is a dominant symptom at any stage of these diseases, is directly related to joint functioning, and determines the quality of life in patients. Moreover, despite the significant successes of studying the role of inflammation and regulation of autoimmune processes, the pathogenetic mechanisms for the development and maintenance of pain in RDs are little investigated. The nociceptive mechanisms due to inflammation and/or joint structural impairment are involved in the development of rheumatic pain. In addition, the latter is also associated with impaired signaling in the nervous system and with psychological problems in patients.At the present stage, pain treatment includes non-pharmacological interventions, as well as the use of certain pharmacological agents, in particular opioids and narcotic drugs. However, despite significant successes in the design of drugs that relieve pain, at present, a significant proportion of patients with RDs still experience pain after therapy. When designing novel drugs for the treatment of pain, it is necessary to take into account the molecular mechanisms of its development in RDs. This review considers the features of the manifestations of pain, its molecular markers and mechanisms at different stages of the disease in patients with the two most common RDs, such as rheumatoid arthritis and osteoarthritis.

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3

Neeck, Gunther, Rainer Renkawitz e Martin Eggert. "Molecular aspects of glucocorticoid hormone action in rheumatoid arthritis". Cytokines, Cellular & Molecular Therapy 7, n.º 2 (janeiro de 2002): 61–69. http://dx.doi.org/10.1080/13684730412331302081.

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4

Kobayashi, Shu, Shigeki Momohara, Naoyuki Kamatani e Hiroshi Okamoto. "Molecular aspects of rheumatoid arthritis: role of environmental factors". FEBS Journal 275, n.º 18 (25 de julho de 2008): 4456–62. http://dx.doi.org/10.1111/j.1742-4658.2008.06581.x.

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5

Okamoto, Hiroshi, Thomas P. Cujec, Hisashi Yamanaka e Naoyuki Kamatani. "Molecular aspects of rheumatoid arthritis: role of transcription factors". FEBS Journal 275, n.º 18 (25 de julho de 2008): 4463–70. http://dx.doi.org/10.1111/j.1742-4658.2008.06582.x.

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6

García-Hernández, Mariana H., Roberto González-Amaro e Diana Patricia Portales-Pérez. "Specific therapy to regulate inflammation in rheumatoid arthritis: molecular aspects". Immunotherapy 6, n.º 5 (maio de 2014): 623–36. http://dx.doi.org/10.2217/imt.14.26.

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7

Lotz, M., e J. Roudier. "Epstein-Barr virus and rheumatoid arthritis: cellular and molecular aspects". Rheumatology International 9, n.º 3-5 (novembro de 1989): 147–52. http://dx.doi.org/10.1007/bf00271872.

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8

Sacre, Sandra M., Evangelos Andreakos, Peter Taylor, Marc Feldmann e Brian M. Foxwell. "Molecular therapeutic targets in rheumatoid arthritis". Expert Reviews in Molecular Medicine 7, n.º 16 (24 de agosto de 2005): 1–20. http://dx.doi.org/10.1017/s1462399405009488.

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In an attempt to combat the pain and damage generated by rheumatoid arthritis (RA), new drugs are being developed to target molecular aspects of the disease process. Recently, a major development has been the use of biologicals (antibodies and soluble receptors) that neutralise the activity of tumour necrosis factor α (TNF-α) and interleukin 1 (IL-1), both of which are involved in disease progression. An increase in our understanding of cell and molecular biology has resulted in the identification and investigation of potential new targets, and also the refinement and improvement of current therapeutic modalities. This review describes therapies that are approved, in clinical trials or under pre-clinical investigation at the laboratory level, particularly focusing on cytokines, although other therapeutic targets of interest are mentioned.

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9

RULLI, NESTOR E., JULIAN MELTON, ANJA WILMES, GARY EWART e SURESH MAHALINGAM. "The Molecular and Cellular Aspects of Arthritis Due to Alphavirus Infections". Annals of the New York Academy of Sciences 1102, n.º 1 (abril de 2007): 96–108. http://dx.doi.org/10.1196/annals.1408.007.

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10

Okamoto, Hiroshi. "Molecular aspects of rheumatoid arthritis: chemokines, environmental factors and transcription factors". FEBS Journal 275, n.º 18 (25 de julho de 2008): 4447. http://dx.doi.org/10.1111/j.1742-4658.2008.06579.x.

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Teses / dissertações sobre o assunto "Arthritis Molecular aspects"

1

Law, Wai-han, e 羅慧嫺. "Molecular mechanisms of arsenic trioxide in an in vitro model of rheumatoid arthritis synoviocyte". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41508531.

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2

Connolly, Ashley Rex. "Cytokine gene expression in a rat model of polyarthritis /". Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phc75238.pdf.

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3

Olive, Colleen. "Molecular characterisation of the T-cell receptors expressed in rheumatoid arthritis patients". Phd thesis, 1993. http://hdl.handle.net/1885/143110.

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4

Brasted, Melissa. "Effector CD4Ê T lymphocytes in the prodrome of polyarthritis". 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phb8239.pdf.

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5

Connolly, Ashley Rex. "Cytokine gene expression in a rat model of polyarthritis / by Ashley Rex Connolly". Thesis, 1998. http://hdl.handle.net/2440/19176.

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Bibliography: leaves 199-233.
xvii, 233 leaves : ill. ; 30 cm.
Describes the development of a method used for quantification of cytokine mRNA expression and its application to measuring changes in cytokine expression in the synovial tissue and lymph notes draining the hind feet of rats with adjuvant arthritis (AA).
Thesis (Ph.D.)--University of Adelaide, Dept. of Medicine, 1998

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6

Roberts-Thomson, Peter J. "Low molecular weight IgM in health and disease / by Peter John Roberts-Thomson". 1987. http://hdl.handle.net/2440/38410.

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x, 156 leaves :
Title page, contents and abstract only. The complete thesis in print form is available from the University Library.
Thesis (M.D.)--University of Adelaide, Dept. of Medicine, 1988

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7

Roberts-Thomson, Peter J. "Low molecular weight IgM in health and disease / by Peter John Roberts-Thomson". Thesis, 1987. http://hdl.handle.net/2440/38410.

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Livros sobre o assunto "Arthritis Molecular aspects"

1

Brewer, Jonathan Douglas. The coronary cure: A modified Mediterranean diet for the prevention of atherosclerosis, hypertension, obesity, diabetes, arthritis, and kidney stones : molecular mechanisms and practical guidelines : nutrition 101. [United States?]: J.D. Brewer, 2004.

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2

Tsokos, George C. Principles of Molecular Rheumatology (Current Molecular Medicine). Humana Press, 2000.

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3

P, Cope Andrew, ed. Arthritis research: Methods and protocols. Totowa, N.J: Humana Press, 2007.

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4

Cope, Andrew P. Arthritis Research: Volume 1: Methods and Protocols (Methods in Molecular Medicine). Humana Press, 2007.

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5

Cope, Andrew P. Arthritis Research: Volume 2: Methods and Protocols (Methods in Molecular Medicine). Humana Press, 2007.

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6

Arthritis research: Methods and protocols. Totowa, NJ: Humana Press, 2007.

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7

Cope, Andrew P. Arthritis Research : Volume 1: Methods and Protocols. Humana Press, 2010.

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8

Cope, Andrew P. Arthritis Research : Volume 2: Methods and Protocols. Humana Press, 2010.

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Capítulos de livros sobre o assunto "Arthritis Molecular aspects"

1

Gay, S., e R. E. Gay. "Pathology of Rheumatoid Arthritis: Molecular and Inflammatory Aspects". In Symposium in Immunology VIII, 199–203. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59947-7_13.

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2

Winchester, Robert. "MAPPING SUSCEPTIBILITY TO RHEUMATOID ARTHRITIS INTO A CONFORMATIONALLY EQUIVALENT REGION OF MHC CLASS II MOLECULES ENCODED BY DIFFERENT ALLELES". In The Molecular Aspects of Autoimmunity, 241–63. Elsevier, 1990. http://dx.doi.org/10.1016/b978-0-12-249040-8.50021-3.

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3

Ege, Ferhat. "Pathogenesis, Pathology and Genetics of Osteoarthritis". In Rheumatoid Arthritis [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99238.

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Osteoarthritis (OA) is a condition with high prevalence worldwide. OA affects not only the articular cartilage, but the entire joint, including the subchondral bone, ligaments, capsule, synovial membrane and the periarticular muscles. Despite the fact that the risks associated with OA increase with age, it is not a part of the natural aging process. It typically involves the knee, hip, spine, hand and foot joints. Several factors play an important role in the pathogenesis of OA, including biomechanical factors, proinflammatory mediators and proteases. On the other hand, it was mostly the results of the studies conducted on the genetic, genomic and epigenetic aspects of OA, from among many of its underlying etiological factors, which shed light on the molecular processes involved in the etiopathogenesis of OA. As the mechanisms that cause joint tissue damage in OA come to light, the treatment of OA will go beyond just providing symptomatic relief. Consequentially, new treatments will emerge that will either slow or completely stop the progression of OA.

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