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1

Insel, Thomas R. "From Animal Models to Model Animals". Biological Psychiatry 62, n.º 12 (dezembro de 2007): 1337–39. http://dx.doi.org/10.1016/j.biopsych.2007.10.001.

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2

HR, Siddique. "Animal Models in Cancer Chemoprevention". International Journal of Zoology and Animal Biology 2, n.º 5 (2019): 1–5. http://dx.doi.org/10.23880/izab-16000171.

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3

Plaa, Gabriel L. "Animal Models". Drug Safety 5, Supplement 1 (1990): 40–45. http://dx.doi.org/10.2165/00002018-199000051-00007.

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4

Small, D. L., e A. M. Buchan. "Animal models". British Medical Bulletin 56, n.º 2 (1 de janeiro de 2000): 307–17. http://dx.doi.org/10.1258/0007142001903238.

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5

Goetze, Jens P., e Andrew Krentz. "Animal models". Cardiovascular Endocrinology 3, n.º 1 (março de 2014): 1. http://dx.doi.org/10.1097/xce.0000000000000023.

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6

BARNES, DONALD J. "Animal models". Nature 329, n.º 6141 (outubro de 1987): 666. http://dx.doi.org/10.1038/329666c0.

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7

Lomberk, Gwen. "Animal models". Pancreatology 6, n.º 5 (outubro de 2006): 427–28. http://dx.doi.org/10.1159/000094559.

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8

Olivier, Berend. "Animal models". European Psychiatry 13, S4 (1998): 182s. http://dx.doi.org/10.1016/s0924-9338(99)80182-5.

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9

Moyer, Paula. "ANIMAL MODELS". Neurology Today 4, n.º 1 (janeiro de 2004): 14. http://dx.doi.org/10.1097/00132985-200401000-00008.

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10

Saloman, Jami L., Kathryn M. Albers, Zobeida Cruz-Monserrate, Brian M. Davis, Mouad Edderkaoui, Guido Eibl, Ariel Y. Epouhe et al. "Animal Models". Pancreas 48, n.º 6 (julho de 2019): 759–79. http://dx.doi.org/10.1097/mpa.0000000000001335.

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11

&NA;. "Animal Models". Psychiatric Genetics 5, Supplement (agosto de 1995): 105. http://dx.doi.org/10.1097/00041444-199508001-00031.

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12

Wekerle, Hartmut, Kimikazu Kojima, Joseli Lannes-Vieira, Hans Lassmann e Christopher Linington. "Animal models". Annals of Neurology 36, S1 (1994): S47—S53. http://dx.doi.org/10.1002/ana.410360714.

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13

Karol, Meryl H., Yvon Cormier, Kelley J. Donham, Susanna Von Essen, Ulrich F. Gruber, Monica Lundholm, Hal B. Richerson e Moira Chan-Yeung. "Animal models". American Journal of Industrial Medicine 25, n.º 1 (janeiro de 1994): 135–38. http://dx.doi.org/10.1002/ajim.4700250137.

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14

Moon, Richard C. "Animal models". Journal of Cellular Biochemistry 53, S17F (1993): 82. http://dx.doi.org/10.1002/jcb.240531011.

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15

Richelle, MarcN. "Animal models: Models of what?" Behavioural Processes 29, n.º 1-2 (abril de 1993): 114. http://dx.doi.org/10.1016/0376-6357(93)90032-m.

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16

Bates, Jason H. T., Mercedes Rincon e Charles G. Irvin. "Animal models of asthma". American Journal of Physiology-Lung Cellular and Molecular Physiology 297, n.º 3 (setembro de 2009): L401—L410. http://dx.doi.org/10.1152/ajplung.00027.2009.

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Studies in animal models form the basis for much of our current understanding of the pathophysiology of asthma, and are central to the preclinical development of drug therapies. No animal model completely recapitulates all features of the human disease, however. Research has focused primarily on ways to generate allergic inflammation by sensitizing and challenging animals with a variety of foreign proteins, leading to an increased understanding of the immunological factors that mediate the inflammatory response and its physiological expression in the form of airways hyperresponsiveness. Animal models of exaggerated airway narrowing are also lending support to the notion that asthma may represent an abnormality of the airway smooth muscle. The mouse is now the species of choice for asthma research involving animals. This presents practical challenges for physiological study because the mouse is so small, but modern imaging methodologies, coupled with the forced oscillation technique for measuring lung mechanics, have allowed the asthma phenotype in mice to be precisely characterized.
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17

Longo, Umile Giuseppe, Rocco Papalia, Sergio De Salvatore, Riccardo Picozzi, Antonio Sarubbi e Vincenzo Denaro. "Induced Models of Osteoarthritis in Animal Models: A Systematic Review". Biology 12, n.º 2 (10 de fevereiro de 2023): 283. http://dx.doi.org/10.3390/biology12020283.

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The most common induction methods for OA are mechanical, surgical and chemical. However, there is not a gold standard in the choice of OA animal models, as different animals and induction methods are helpful in different contexts. Reporting the latest evidence and results in the literature could help researchers worldwide to define the most appropriate indication for OA animal-model development. This review aims to better define the most appropriate animal model for various OA conditions. The research was conducted on the following literature databases: Medline, Embase, Cinahl, Scopus, Web of Science and Google Scholar. Studies reporting cases of OA in animal models and their induction from January 2010 to July 2021 were included in the study and reviewed by two authors. The literature search retrieved 1621 articles, of which 36 met the selection criteria and were included in this review. The selected studies included 1472 animals. Of all the studies selected, 8 included information about the chemical induction of OA, 19 were focused on mechanical induction, and 9 on surgical induction. Nevertheless, it is noteworthy that several induction models, mechanical, surgical and chemical, have been proven suitable for the induction of OA in animals.
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18

Zhan, Xianbao, Fan Wang, Yan Bi e Baoan Ji. "Animal models of gastrointestinal and liver diseases. Animal models of acute and chronic pancreatitis". American Journal of Physiology-Gastrointestinal and Liver Physiology 311, n.º 3 (1 de setembro de 2016): G343—G355. http://dx.doi.org/10.1152/ajpgi.00372.2015.

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Animal models of pancreatitis are useful for elucidating the pathogenesis of pancreatitis and developing and testing novel interventions. In this review, we aim to summarize the most commonly used animal models, overview their pathophysiology, and discuss their strengths and limitations. We will also briefly describe common animal study procedures and refer readers to more detailed protocols in the literature. Although animal models include pigs, dogs, opossums, and other animals, we will mainly focus on rodent models because of their popularity. Autoimmune pancreatitis and genetically engineered animal models will be reviewed elsewhere.
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19

Mbakam, C. Happi, J. Rousseau, G. Tremblay e J. Tremblay. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S77—S78. http://dx.doi.org/10.1016/j.nmd.2021.07.119.

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20

Egorova, T., A. Polikarpova, I. Savchenko, S. Vassilieva, Y. Ivanova, V. Skopenkova, M. Dzhenkova et al. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S73. http://dx.doi.org/10.1016/j.nmd.2021.07.104.

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21

Dubuisson, N., M. Abou-Samra, M. Davis, L. Noel, C. Selvais e S. Brichard. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S76. http://dx.doi.org/10.1016/j.nmd.2021.07.113.

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22

Hong, A. Vu, N. Bourg-Alibert, P. Sanatine, J. Poupiot, K. Charton, E. Gicquel, M. Spinazzi, I. Richard e D. Israeli. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S77. http://dx.doi.org/10.1016/j.nmd.2021.07.118.

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23

Lambert, M., Y. Zhang, J. Spinazzola, J. Widrick, J. Conner e L. Kunkel. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S78. http://dx.doi.org/10.1016/j.nmd.2021.07.120.

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24

Abou-Samra, M., A. Marino, C. Selvais, N. Dubuisson, L. Noel, C. Beauloye, S. Horman e S. Brichard. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S76—S77. http://dx.doi.org/10.1016/j.nmd.2021.07.116.

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25

Yavas, A., M. van Putten, E. Niks e A. Aartsma-Rus. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S75—S76. http://dx.doi.org/10.1016/j.nmd.2021.07.112.

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26

Mantuano, P., B. Boccanegra, F. Sanarica, E. Conte, A. Mele, M. De Bellis, O. Cappellari e A. De Luca. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S73. http://dx.doi.org/10.1016/j.nmd.2021.07.105.

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27

Kreher, N., X. Li, M. Kheirabadi, K. Kamer, P. Dougherty, W. Lian, C. Waters et al. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S74. http://dx.doi.org/10.1016/j.nmd.2021.07.106.

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28

Engelbeen, S., C. Tanganyika-de Winter, D. Van De Vijver, M. Holierhoek, A. Yavas, S. Kooijman, A. Aartsma-Rus e M. van Putten. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S73. http://dx.doi.org/10.1016/j.nmd.2021.07.103.

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29

Schneider, A., S. Jirka, C. Tanganyika-de Winter, H. Mei, J. Boom e A. Aartsma-Rus. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S75. http://dx.doi.org/10.1016/j.nmd.2021.07.111.

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30

Creisméas, A., C. Gazaille, A. Bourdon, A. Lafoux, M. Allais, V. Le Razavet, M. Ledevin et al. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S76. http://dx.doi.org/10.1016/j.nmd.2021.07.114.

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31

van Putten, M., C. Tanganyika-de Winter, K. Putker, S. Engelbeen, D. Van de Vijver, M. Verhaeg, M. Overzier e A. Aartsma-Rus. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S74. http://dx.doi.org/10.1016/j.nmd.2021.07.107.

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32

Mantuano, P., B. Boccanegra, E. Bresciani, F. Sanarica, A. Mele, M. De Bellis, O. Cappellari et al. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S77. http://dx.doi.org/10.1016/j.nmd.2021.07.117.

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33

Morin, A., O. Petrova, M. Petkova, T. Tensorer, T. Manoliu, I. Richard, L. Garcia et al. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S72—S73. http://dx.doi.org/10.1016/j.nmd.2021.07.102.

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34

Kim, S., N. Buss, C. Qiao, H. Patel, L. Yang, K. Elliott, R. Qian, L. Ye, M. Fiscella e O. Danos. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S76. http://dx.doi.org/10.1016/j.nmd.2021.07.115.

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35

Polikarpova, A., I. Galkin, D. Korshunova, V. Skopenkova, M. Dzhenkova, D. Tsvirkun, A. Shmidt et al. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S74. http://dx.doi.org/10.1016/j.nmd.2021.07.108.

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36

Hong, A. Vu, F. Amor, G. Corre, M. Sanson, L. Suel, S. Blaie, L. Servais, T. Voit, I. Richard e D. Israeli. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S74—S75. http://dx.doi.org/10.1016/j.nmd.2021.07.109.

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37

Lindsay, A., A. Trewin, P. Della Gatta, C. Laird e A. Russell. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S75. http://dx.doi.org/10.1016/j.nmd.2021.07.110.

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38

Peterson, E., R. Potter, D. Griffin, S. Lewis, E. Pozgai, A. Meadows e L. Rodino-Klapac. "DMD – ANIMAL MODELS". Neuromuscular Disorders 31 (outubro de 2021): S78. http://dx.doi.org/10.1016/j.nmd.2021.07.121.

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39

Cole, P., X. Bofill e C. Dulsat. "Animal models snapshot". Drugs of the Future 44, n.º 1 (2019): 29. http://dx.doi.org/10.1358/dof.2019.44.1.2939527.

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40

Chapman, J. L., D. K. Nichols, M. J. Martinez e J. W. Raymond. "Animal Models ofOrthopoxvirusInfection". Veterinary Pathology 47, n.º 5 (3 de agosto de 2010): 852–70. http://dx.doi.org/10.1177/0300985810378649.

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41

Atanasova, Nina. "Validating Animal Models". THEORIA. An International Journal for Theory, History and Foundations of Science 30, n.º 2 (20 de junho de 2015): 163. http://dx.doi.org/10.1387/theoria.12761.

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In this paper, I respond to the challenge raised against contemporary experimental neurobiology according to which the field is in a state of crisis because of the multiple experimental protocols employed in different laboratories and strengthening their reliability that presumably preclude the validity of neurobiological knowledge. I provide an alternative account of experimentation in neurobiology which makes sense of its experimental practices. I argue that maintaining a multiplicity of experimental protocols and strengthening their reliability are well justified and they foster rather than preclude the validity of neurobiological knowledge. Thus, their presence indicates thriving rather than crisis of experimental neurobiology.
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42

Hogan, Quinn. "Animal pain models". Regional Anesthesia and Pain Medicine 27, n.º 4 (julho de 2002): 385–401. http://dx.doi.org/10.1097/00115550-200207000-00009.

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43

Millikan, C. "Animal stroke models." Stroke 23, n.º 6 (junho de 1992): 795–97. http://dx.doi.org/10.1161/01.str.23.6.795.

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44

Pérez, J., M. Ware, G. Bennett e Y. Shir. "Animal pain models". Journal of Pain 5, n.º 3 (abril de 2004): S16. http://dx.doi.org/10.1016/j.jpain.2004.02.031.

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45

Smith, S., M. O'Reilly, G. Plourde e J. Mogil. "Animal pain models". Journal of Pain 5, n.º 3 (abril de 2004): S16. http://dx.doi.org/10.1016/j.jpain.2004.02.032.

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46

Smith, V., C. Beyer e M. Brandt. "Animal pain models". Journal of Pain 5, n.º 3 (abril de 2004): S17. http://dx.doi.org/10.1016/j.jpain.2004.02.033.

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47

Pérez, J., M. Ware, G. >. Bennett e Y. Shir. "Animal pain models". Journal of Pain 5, n.º 3 (abril de 2004): S17. http://dx.doi.org/10.1016/j.jpain.2004.02.034.

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48

Robinson, P., K. Smith, A. Loescher, F. Boissonade, S. Atkins e M. Ferguson. "Animal pain models". Journal of Pain 5, n.º 3 (abril de 2004): S17. http://dx.doi.org/10.1016/j.jpain.2004.02.035.

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49

Soignier, R., P. Nolan, D. Paul e H. Gould. "Animal pain models". Journal of Pain 5, n.º 3 (abril de 2004): S17. http://dx.doi.org/10.1016/j.jpain.2004.02.036.

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50

HOGAN, Q. "Animal pain models". Regional Anesthesia and Pain Medicine 27, n.º 4 (julho de 2002): 385–401. http://dx.doi.org/10.1053/rapm.2002.33630.

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