Teses / dissertações sobre o tema "Aid to injured"
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Schneiders, Anthony G., e n/a. "The influence of a back-support harness on the three dimensional kinematics and electromyography of the trunk in sheep shearers : implications for injury prevention". University of Otago. School of Physiotherapy, 2005. http://adt.otago.ac.nz./public/adt-NZDU20060803.160831.
Texto completo da fonteCole, Elaine. "Characterising factors predictive of infection in severely injured patients". Thesis, Queen Mary, University of London, 2015. http://qmro.qmul.ac.uk/xmlui/handle/123456789/7975.
Texto completo da fonteFranklin, Paula. "First aid when and where you need it, development of first aid self-care, patient education for web and mobile devices /". Online version of thesis, 2008. http://hdl.handle.net/1850/7903.
Texto completo da fonteDidier, Elizabeth. "Being Prepared for Show Livestock Injuries and Illnesses". College of Agriculture and Life Sciences, University of Arizona (Tucson, AZ), 2005. http://hdl.handle.net/10150/144742.
Texto completo da fonteArizona youth livestock quality assurance and food safety: Trainers reference. Arizona youth livestock quality assurance and food safety: Youth manual. Feeding management for show lambs. Feeding management for show steers. Swine nutrition for show animals.
Illness or injury to a show animal may be preventable by following a few guidelines. Providing a clean and safe environment and properly feeding, watering, and vaccinating animals will help to reduce the risks of experiencing illnesses and injuries. Owners should also learn how to identify signs of health problems, such as sudden changes in behavior or appearance, and prepare a first aid kit for use in the event of an emergency. Also, being familiar with emergency treatment guidelines will help owners protect themselves, prevent further injury to the animal, and properly administer care to the animal if appropriate.
Шкатула, Юрій Васильович, Юрий Васильевич Шкатула e Yurii Vasylovych Shkatula. "Urgent medical aid to children with polyorganic and polysystemic injuries at the prehospital stage". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27525.
Texto completo da fonteHarish, Varun. "A Comprehensive Review of the Assessment of and Early Intervention for Burn Injuries in New South Wales, Australia with Recommendations for Clinical Practice". Thesis, The University of Sydney, 2018. http://hdl.handle.net/2123/20428.
Texto completo da fonteKiroff, George Kosta. "Oesophageal mucosal injury by acid and bile salt /". Title page, table of contents and summary only, 1986. http://web4.library.adelaide.edu.au/theses/09MS/09msk59.pdf.
Texto completo da fonteThe experimental work described was performed in the Department of Surgery of the University of Adelaide during 1982-1983. Typescript (photocopy). Includes bibliographical references (leaves 191-211).
Reyes, Leila. "Involvement of inflammatory oxidants in cardiac ischaemia/reperfusion injury". Thesis, The University of Sydney, 2018. http://hdl.handle.net/2123/19667.
Texto completo da fonteLEHMAN, JUSTIN EUGENE. "THE PERCEIVED CONFIDENCE OF RURAL, NORTHWEST OHIO HIGH SCHOOL COACHES IN THEIR ABILITY TO HANDLE VARIOUS INJURIES AND ILLNESSES INVOLVING STUDENT ATHLETES". University of Cincinnati / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1154980798.
Texto completo da fonteArmstrong, Bridget Sarah. "Head and neck position sense in whiplash patients and healthy individuals and the effect of the "chin tuck" action this thesis is submitted to the Auckland University of Technology for the degree of Master of Health Science, February 2003". Full thesis. Abstract, 2003. http://puka2.aut.ac.nz/ait/theses/ArmstrongB.pdf.
Texto completo da fonteKitamura, Jiro. "Chronic Lung Injury by Constitutive Expression of Activation-Induced Cytidine Deaminase Leads to Focal Mucous Cell Metaplasia and Cancer". Kyoto University, 2015. http://hdl.handle.net/2433/200440.
Texto completo da fonteCrack, Peter, Moses Zhang, Maria Morganti-Kossmann, Andrew Morris, Jonathan Wojciak, Jonathan Fleming, Ila Karve et al. "Anti-lysophosphatidic acid antibodies improve traumatic brain injury outcomes". BioMed Central, 2014. http://hdl.handle.net/10150/610194.
Texto completo da fonteSaunders, Royal Duane. "Arachidonic acid and lipid metabolism following spinal cord injury /". The Ohio State University, 1985. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487260859496213.
Texto completo da fonteCRACCO, Laura. "Fatty acid-binding proteins as markers of brain injury". Doctoral thesis, Università degli Studi di Verona, 2009. http://hdl.handle.net/11562/337348.
Texto completo da fonteFatty acid–binding proteins (FABPs) are abundant intracellular proteins that bind long-chain fatty acids with high affinity. 9 different FABPs, with tissuespecific distribution, have been identified so far. The primary role of all the FABP family members is regulation of fatty acid uptake and intracellular transport. Heart-type FABP (H-FABP) is considered one of the most sensible and efficient markers of heart and brain damage, but to date the cross-reactivity limits its specific use. Brain-type FABP (B-FABP) might be considered as a good marker of brain damage, being expressed only in the nervous tissue. The aim of this work was to generate a diagnostic reagent specific for human B-FABP, not cross-reacting with H-FABP. The work has been articulated in two parts: in the first one, recombinant protein B-FABP was expressed in E. coli cells and purified by two subsequent chromatographies. The protein was then utilized as immunogen in New Zealand rabbits and BalbC mice, obtaining two antisera and 8 different hybridoma cells. The antibodies in the rabbit serum and in culture supernatant of hybridoma cells displayed different degrees of affinity for B-FABP and H-FABP; only one hybridoma cell was able to produce antibodies specific and selective for brain-type FABP, but the antibody low-titer level and the activity decrease after subsequent subcloning steps affected its application on B-FABP detection in biological fluids. To overcome the limits encountered in the first part of work, a computer-assisted approach on B-FABP was employed in order to identify the potentially most antigenic regions of the protein. Two synthetic peptides were produced with the selected sequences and inoculated in rabbits. Two polyclonal antibodies were obtained, pAb 2979/2980 and pAb 2981/2982. They showed specific and selective reactivity for human B-FABP, without crossreactions with H-FABP. Taken together, our experimental approach was effective for the generation of specific α-B-FABP antibodies; these results suggest the antibodies obtained could be utilized in ELISA and immunoblot analyses in order to value B-FABP as marker of neurological disorders with ischemic, infective and degenerative etiology.
Potts, Geoffrey. "Biomechanic analysis of 'heavy-load eccentric calf muscle' exercise used in the rehabilitation of achilles tendinosis a dissertation submitted in partial fulfilment for the degree of Master of Health Science, Auckland University of Technology, January 2005". Full thesis. Abstract, 2005. http://puka2.aut.ac.nz/ait/theses/PottsG.pdf.
Texto completo da fonteSturrock, James Lee. "A Behavioral Modification Analysis of the Effects of Multimedia First Aid Training on Injuries in an Industrial Setting". Thesis, North Texas State University, 1987. https://digital.library.unt.edu/ark:/67531/metadc331593/.
Texto completo da fonteLópez, Serrano Clara. "Role of lysophosphatidic acid receptors in spinal cord injury physiopathology". Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/458682.
Texto completo da fonteThe spinal cord is an extremely vital part of the central nervous system (CNS) and, although it is well protected by the spinal column, it can be damaged, resulting in serious consequences. Spinal cord injury (SCI) leads to a disruption of the neuronal networks that are involved in many physiological functions. Since CNS axons of adult mammals do not regenerate following the lesion, and dead neurons and glial cells are not successfully replaced, this results in an irreversible functional loss in patients suffering from SCI. The pathophysiology of SCI involves two degenerative stages, known as primary and secondary injury. The first one results from the direct mechanical trauma to the spinal cord, directly causing cell death, damage to axons, and loss of myelin. This is followed by a secondary wave of tissue degeneration that can extend for several weeks, in which inflammation plays a crucial role. Although regeneration of damaged axons and replacement of lost neurons and glial cells are important goals for the restoration of the injured spinal cord, minimizing secondary damage to axons, neuronal cell bodies, myelin and glial cells that follows the initial trauma is likely to be more easily amenable to treatment. Since inflammation is a major contribution to secondary damage in SCI, targeting the detrimental actions of this physiological response could result in the development of novel approaches for the treatment of this pathology. Lysophosphatidic acid (LPA) is an extracellular bioactive lipid with many physiological functions. It signals through six known G-protein coupled receptors (LPA1-6), which are classified into two families: Endothelial differentiation family gene (Edg) LPA receptors (LPA1-3) and Non-Edg family gene LPA receptors (LPA4-6). LPA synthesis is carried out by two different pathways: (i) by the action of the enzyme named autotaxin (ATX), which is the main responsible in synthesis of this lipid in plasma, and (ii) by action of the phospholipase A2 (PLA2) family enzymes, which are the main route of LPA synthesis in tissues. LPA is a key trigger of secondary damage after SCI, since its increased levels in the spinal cord parenchyma following injury leads to demyelination. Indeed, the lack of LPA1 and LPA2 signalling after SCI enhances functional recovery and myelin sparing. In this thesis, we show that activation of microglial LPA1 and LPA2 leads to oligodendrocyte cell death. We reveal that the cytotoxic actions underlying by microglial cells stimulated with LPA are mediated by the release of purines and the subsequent activation of P2X7 in oligodendrocytes. We also show that, unlike LPA1 and LPA2, LPA4 and LPA5 receptors do not contribute to SCI physiopathology. In the present thesis, we also show that pharmacological inhibition of ATX does not have any effect in functional outcomes and secondary tissue damage after SCI, suggesting that this enzyme is unlikely to be involved in the production of LPA in the spinal cord parenchyma after injury. We also demonstrate that combinatory targeting of LPA1 and LPA2 does not results in additive effects in SCI. Overall, the results shown here suggest that pharmacological inhibition of LPA1, and preferably LPA2, may open a new therapeutic avenue for the treatment of SCI.
King, P. "Branched chain amino/keto acid supplementation following severe burn injury". Thesis, University of Salford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.376875.
Texto completo da fonteJames, Masheika. "Vitamin A and retinoic acid in neonatal hyperoxic lung injury /". Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2009r/james.pdf.
Texto completo da fonteEsson, Ken. "A cost analysis of accidents and injuries in the open cut coalmining industry". Thesis, Federation University Australia, 1992. http://researchonline.federation.edu.au/vital/access/HandleResolver/1959.17/97254.
Texto completo da fonteMasters in Applied Science in Occupational Health and Safety
Schizas, Nikos. "Neuroprotection in the Injured Spinal Cord : Novel Strategies using Immunomodulation, Stem cell Transplantation and Hyaluronic acid Hydrogel carriers". Doctoral thesis, Uppsala universitet, Ortopedi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-251477.
Texto completo da fonteSalmon, Elizabeth. "DEVELOPMENT OF AN EEG BRAIN-MACHINE INTERFACE TO AID IN RECOVERY OF MOTOR FUNCTION AFTER NEUROLOGICAL INJURY". UKnowledge, 2013. http://uknowledge.uky.edu/cbme_etds/8.
Texto completo da fonteMcDevitt, Jane K. "N-METHYL-D-ASPARTIC ACID RECEPTOR SUBUNIT NR2A REPEAT". Diss., Temple University Libraries, 2013. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/216597.
Texto completo da fontePh.D.
During a concussion, mechanical forces cause neuron cell strain that initiates dysfunction through the indiscriminate movement of ions through protein channels. Extracellular glutamate binds with cell membrane proteins (e.g. NR2A), which exacerbates the Ca2+ ion influx and prolongs neuron dysfunction. Genetic variation may be a factor in regulating glutamate binding and therefore cell recovery time. The NR2A subunit of NMDA contains a variable (GT)n nucleotide tandem repeat (VNTR) within GRIN2A promoter region. This VNTR has been shown to regulate transcription levels in a length dependent manner, where longer repeat decreases transcription of the NR2A subunit. The purpose of this study was to determine the association of the GRIN2A VNTR and recovery (days) as well as concussion severity scores within concussed athletes. The independent variable was VNTR (long allele vs. short allele). The primary dependent variable, recovery time, was defined as injury date to return to play (RTP) clearance date as determined by the physician. Participant RTP time was categorized as normal ( 20 days). Secondary dependent variables were assessed at the initial evaluation and included vestibular ocular score, Balance Error Scoring System (BESS) score, and Immediate Post Concussion and Cognitive Testing (ImPACT) module scores. All 51 participants were athletes, comprised of 38 males and 13 females with a mean age of 18.69 6.65. Participants were evaluated at a university concussion center. The standardized concussion evaluation consisted of cranial nerve, vestibular ocular tests, balance (Balance Error Scoring System), signs and symptoms (s/s), and neurocognitive (ImPACT) testing. Each participant was genotyped via saliva sample for the GRIN2A (GT)n repeat polymorphism (rs3219790). Data analysis consisted of descriptive and inferential statistics. Chi-squares were used to assess the association between VNTR (long allele versus short allele) and concussion recovery (prolonged versus normal). Regression analyses were used to estimate extent to which non-genetic factors and genotype contributed to concussion recovery group assignment. One-way ANOVAs were used to identify any significant differences in vestibular ocular, BESS, and ImPACT module scores between long and short alleles. Primary potential non-genetic contributing factors were age, race, sex, concussion history, dizziness at time of injury, history of migraines, and history of ADHD. The alpha level was set at p ≤ .05. There were no differences between demographic or health history within the VNTR or recovery groups. There was a significant association (x2 = 4.01, p = 0.045) between the VNTR group (dominant model: LL versus SS + LS) and recovery, where the chance of prolonged recovery was 4.3 times greater for carriers of the homozygous long allele. There were no differences in concussion severity scores between VNTR group and concussion severity scores. This was the first study to investigate the association of the (GT)n VNTR within GRIN2A. We established a DNA collection, estimation, and genotyping protocol of the (GT)n VNTR for 96 samples and demonstrated accuracy of this genotyping method. Clinically, athletes carrying the long allele genotype may be predisposed to prolonged recovery following a concussive injury.
Temple University--Theses
Li, Ruifu. "A Novel Thiolated Hyaluronic acid Hydrogel for Spinal Cord Injury Repair". Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31410.
Texto completo da fonteEuler, Mia von. "Experimental spinal cord injuries : a histopathological, neurological, and pharmacological study in the rat /". Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3461-4/.
Texto completo da fonteTimko, Tamara C. "Effects of first aid training on seeking adult assistance in response to simulated injuries by young children with disabilities /". The Ohio State University, 1996. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487940308434287.
Texto completo da fonteMüller, Christian. "α-Lipoic Acid Attenuates Ischemia Reperfusion Injury of the Rat Liver". Diss., lmu, 2002. http://nbn-resolving.de/urn:nbn:de:bvb:19-2394.
Texto completo da fonteWeight, Simon C. "The role of nitric acid in renal warm ischaemia reperfusion injury". Thesis, University of Leicester, 1998. http://hdl.handle.net/2381/29558.
Texto completo da fonteNeese, Steven L. "The neuroprotective effects of Z-bisdehydrodoisynolic acid following traumatic brain injury /". Available to subscribers only, 2007. http://proquest.umi.com/pqdweb?did=1342727221&sid=4&Fmt=2&clientId=1509&RQT=309&VName=PQD.
Texto completo da fonteJames, Bryce. "(+/-)-Z-bisdehydrodoisynolic acid and specific enantiomers : effects on traumatic brain injury /". Available to subscribers only, 2007. http://proquest.umi.com/pqdweb?did=1459903541&sid=1&Fmt=2&clientId=1509&RQT=309&VName=PQD.
Texto completo da fonteHumphries, Duncan Charles. "Investigating the cerebral/pulmonary axis following traumatic brain injury in a preclinical model". Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/19511.
Texto completo da fonteReveruzzi, Bianca. "The protective mechanisms of first aid training within a school-based program for early adolescents". Thesis, Queensland University of Technology, 2015. https://eprints.qut.edu.au/82759/1/Bianca_Reveruzzi_Thesis.pdf.
Texto completo da fonteStapleton, Sally C. "A preliminary investigation into the use of mobile phones as a memory aid for individuals with traumatic brain injury". Thesis, University of East Anglia, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.396732.
Texto completo da fonteYin, Terry. "Loss of acid sensing ion channel-1a and bicarbonate administration attenuate the severity of traumatic brain injury". Thesis, University of Iowa, 2013. https://ir.uiowa.edu/etd/2423.
Texto completo da fonteSantos, Nogueira Eva. "Spinal cord injury: Role of endothelial differentiation gene family lysophosphatidic acid receptors". Doctoral thesis, Universitat Autònoma de Barcelona, 2014. http://hdl.handle.net/10803/285416.
Texto completo da fonteSecondary tissue damage that occurs after spinal cord injury (SCI) contributes significantly to permanent functional disabilities. Although regeneration of damaged axons and replacement of lost neurons are important goals to repair the injured spinal cord, the secondary damage to axons, neurons, myelin and glial cells that follows the initial trauma is likely to be more easily amenable to treatment. Therefore, preventing or minimizing such secondary damage after SCI is expected to substantially reduce functional deficits. The inflammatory response that occurs after SCI strongly contributes to secondary injury. A number of mechanisms underlie the recruitment of leukocytes from the peripheral circulation, and the activation of these cells and endogenous microglia and astrocytes within the injured spinal cord. However, the molecules that trigger these responses are not completely known. Lysophosphatidic acid (LPA) is a potent, biologically active lipid mediator that regulates many physiological functions such as of cellular Ca2+ homeostasis, cytoskeleton remodeling, proliferation and survival, adhesion and migration and inflammation. LPA exerts all these functions by signaling via 6 G-protein coupled receptors known as LPARs (LPA1-6). Recent studies highlight the involvement of LPA in the development of many human diseases, such as atherosclerosis, cancer and pulmonary fibrosis. However, few studies have assessed so far whether LPA contributes to nervous system pathologies. Several in vitro observations reveal that LPA activates astrocytes and microglial cells, causes neuronal cell death, and promotes axonal retraction. In vivo studies also show that LPA is involved in the etiology of fetal hydrocephalus, as well as the development of neuropathic pain after sciatic nerve injury and cerebral ischemia. However, whether LPA is involved in the pathophysiology of SCI is still unknown. We currently have novel unpublished data demonstrating that LPA levels rapidly increased in the contused spinal cord. We have also clear evidences indicating that administration of LPA into the uninjured spinal cords triggers glial cell activation and demyelination, and leads to motor impairments. Altogether, our preliminary results strongly suggest that the increased levels of LPA that occurs after SCI may play an important role in triggering secondary damage and functional impairments. The present thesis aimed at assessing which of endothelial differentiation gene family LPA receptors (LPA1-3) exert such harmful effects in SCI. We found that LPA1 and LPA2 contribute to tissue damage and functional impairments after SCI, whereas LPA3 has a neutral effect.. Moreover, we reveal the mechanisms underlying neuronal cell death and demyelination triggered by LPA1 and LPA2. Since major pharmaceutical companies are interested in the development of LPA receptor agonists and antagonists for the treatment of several human diseases, such treatments have a high likelihood of progressing rapidly to the clinic.
Mallek, Jennifer de Toledo. "Hyaluronic acid-olfactory ensheathing cell compositions for spinal cord injury nerve regeneration". [Gainesville, Fla.] : University of Florida, 2006. http://purl.fcla.edu/fcla/etd/UFE0015880.
Texto completo da fonteChaykovska, Lyubov [Verfasser]. "Role of 20-Hydroxyeikosatetraenoic acid in experimental acute kidney injury / Lyubov Chaykovska". Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2010. http://d-nb.info/1025353870/34.
Texto completo da fonteHuebenthal, Jan. "Injury & Resistance: Centering HIV/AIDS Histories in Times of Queer Equality". W&M ScholarWorks, 2019. https://scholarworks.wm.edu/etd/1563898925.
Texto completo da fonteCarter-Allison, Samantha Natalie. "Diagnosis threat and injury beliefs after mid traumatic brain injury". Thesis, King's College London (University of London), 2015. https://kclpure.kcl.ac.uk/portal/en/theses/diagnosis-threat-and-injury-beliefs-after-mid-traumatic-brain-injury(c6ba3d52-13d9-46ea-aeee-d34ed2e43943).html.
Texto completo da fonteFerguson, Scott. "Smartphone technology : everyday prompts for those with prospective memory difficulties following brain injury". Thesis, University of Hertfordshire, 2013. http://hdl.handle.net/2299/19859.
Texto completo da fonteMoldrich, Randal Xavier Joseph 1975. "Functional roles of group II metabotropic glutamate receptors in injury and epilepsy". Monash University, Dept. of Pharmacology, 2002. http://arrow.monash.edu.au/hdl/1959.1/7710.
Texto completo da fonteBadion, Yu. "Duration of prehospital assistance to victims with traumatic injuries in rural areas". Thesis, Sumy State University, 2016. http://essuir.sumdu.edu.ua/handle/123456789/45942.
Texto completo da fonteMcGeorge, Nicolette M. "The effect of training, aim pattern and target type on the ergonomics and efficiency of handheld scanners /". Online version of thesis, 2009. http://hdl.handle.net/1850/10643.
Texto completo da fonteAngus, Ruth. "In vivo and in vitro studies on docosahexaenoic acid in traumatic brain injury". Thesis, Queen Mary, University of London, 2017. http://qmro.qmul.ac.uk/xmlui/handle/123456789/30628.
Texto completo da fonteLi, Guangbi. "Podocyte Dedifferentiation and Glomerular Injury Mediated by Lysosome Dysfunction: Role of Acid Ceramidase". VCU Scholars Compass, 2017. https://scholarscompass.vcu.edu/etd/5169.
Texto completo da fonteFahy, Katherine Erin. "Thermal Burn Injury Induced Microvesicle Particle Release". Wright State University / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=wright149383031006972.
Texto completo da fonteWang, Hongrui. "Developing Novel Methods to Mitigate Freezing Injury in Grapevines". The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1564743163557437.
Texto completo da fonteGouda, Mazen M. "Axon Initial Segment Integrity in Aging and Traumatic Brain Injury". VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5993.
Texto completo da fonteHawes, Carol V. "The impact of ozone on the physiology and growth of beech". Thesis, Lancaster University, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267192.
Texto completo da fonteSapiro, Jessica M., e Jessica M. Sapiro. "Molecular Mechanisms Associated with All-Trans-Retinoic Acid-Mediated Cytoprotection against Renal Cell Injury". Diss., The University of Arizona, 2017. http://hdl.handle.net/10150/625452.
Texto completo da fonte