Gotowa bibliografia na temat „Vitamine D 24-hydroxylase”
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Artykuły w czasopismach na temat "Vitamine D 24-hydroxylase"
Shanmugasundaram, R., i R. K. Selvaraj. "Vitamin D-1α-hydroxylase and vitamin D-24-hydroxylase mRNA studies in chickens". Poultry Science 91, nr 8 (sierpień 2012): 1819–24. http://dx.doi.org/10.3382/ps.2011-02129.
Pełny tekst źródłaRahmaniyan, Mehrdad, Kennerly Patrick i Norman H. Bell. "Characterization of recombinant CYP2C11: a vitamin D 25-hydroxylase and 24-hydroxylase". American Journal of Physiology-Endocrinology and Metabolism 288, nr 4 (kwiecień 2005): E753—E760. http://dx.doi.org/10.1152/ajpendo.00201.2004.
Pełny tekst źródłaRen, Songyang, Lisa Nguyen, Shaoxing Wu, Carlos Encinas, John S. Adams i Martin Hewison. "Alternative Splicing of Vitamin D-24-Hydroxylase". Journal of Biological Chemistry 280, nr 21 (23.03.2005): 20604–11. http://dx.doi.org/10.1074/jbc.m414522200.
Pełny tekst źródłaWarner, M., i A. Tenenhouse. "Regulation of renal vitamin D hydroxylase activity in vitamin D deficient rats". Canadian Journal of Physiology and Pharmacology 63, nr 8 (1.08.1985): 978–82. http://dx.doi.org/10.1139/y85-161.
Pełny tekst źródłaAkeno, N., A. Matsunuma, T. Maeda, T. Kawane i N. Horiuchi. "Regulation of vitamin D-1alpha-hydroxylase and -24-hydroxylase expression by dexamethasone in mouse kidney". Journal of Endocrinology 164, nr 3 (1.03.2000): 339–48. http://dx.doi.org/10.1677/joe.0.1640339.
Pełny tekst źródłaAgu, Chinyere, Ei Cho, Christine Resta i Rakhlin Luba. "LBSAT130 Hypercalcemia In Pregnancy Due To CYP24A1 Mutation". Journal of the Endocrine Society 6, Supplement_1 (1.11.2022): A149. http://dx.doi.org/10.1210/jendso/bvac150.303.
Pełny tekst źródłaLee, Sang R., Mi-Young Park, Hyun Yang, Geun-Shik Lee, Beum-Soo An, Bae-kuen Park, Eui-Bae Jeung i Eui-Ju Hong. "5α-dihydrotestosterone reduces renal Cyp24a1 expression via suppression of progesterone receptor". Journal of Molecular Endocrinology 60, nr 2 (luty 2018): 159–70. http://dx.doi.org/10.1530/jme-17-0187.
Pełny tekst źródłaMata-Greenwood, Eugenia, Hans C. A. Westenburg, Stacy Zamudio, Nicholas P. Illsley i Lubo Zhang. "Decreased Vitamin D Levels and Altered Placental Vitamin D Gene Expression at High Altitude: Role of Genetic Ancestry". International Journal of Molecular Sciences 24, nr 4 (8.02.2023): 3389. http://dx.doi.org/10.3390/ijms24043389.
Pełny tekst źródłaBuffenstein, R., I. N. Sergeev i J. M. Pettifor. "Vitamin D hydroxylases and their regulation in a naturally vitamin D-deficient subterranean mammal, the naked mole rat (Heterocephalus glaber)". Journal of Endocrinology 138, nr 1 (lipiec 1993): 59–64. http://dx.doi.org/10.1677/joe.0.1380059.
Pełny tekst źródłaAgic, Admir, Hong Xu, Christopher Altgassen, Frank Noack, Monika M. Wolfler, Klaus Diedrich, Michael Friedrich, Robert N. Taylor i Daniela Hornung. "Relative Expression of 1,25-Dihydroxyvitamin D3 Receptor, Vitamin D 1α-Hydroxylase, Vitamin D 24-Hydroxylase, and Vitamin D 25-Hydroxylase in Endometriosis and Gynecologic Cancers". Reproductive Sciences 14, nr 5 (lipiec 2007): 486–97. http://dx.doi.org/10.1177/1933719107304565.
Pełny tekst źródłaRozprawy doktorskie na temat "Vitamine D 24-hydroxylase"
Boutinet, Stéphane. "Rôle du calcium intracellulaire dans la compliance de l'artère carotide de rat : effets d'un inhibiteur de l'enzyme de conversion de l'angiotensine 1". Nancy 1, 1995. http://docnum.univ-lorraine.fr/public/SCD_T_1995_0449_BOUTINET.pdf.
Pełny tekst źródłaBoutinet, Stéphane. "Rôle du calcium intracellulaire dans la compliance de l'artère carotide de rat : effets d'un inhibiteur de l'enzyme de conversion de l'angiotensine 1". Nancy 1, 1995. http://docnum.univ-lorraine.fr/public/SCD_T_1995_0449_BOUTINET.pdf.
Pełny tekst źródłaRoy, Stéphane. "Regulation of 1,25 dihydroxyvitamin D3-24-hydroxylase gene expression". Thesis, McGill University, 1997. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=34441.
Pełny tekst źródłaThe fourth study addresses the mechanism whereby EB 1089, an analogue of 1,25-(OH)$ sb2$D$ sb3,$ is less calcemic than the vitamin D hormone, while being more potent in its antiproliferative action. We demonstrate that: (i) EB 1089 has a 50-fold lower affinity than 1,25-(OH)$ sb2$D$ sb3$ for the vitamin D catabolic enzyme, 24-hydroxylase; and (ii) EB 1089 and 1,25-(OH)$ sb2$D$ sb3$ exhibit tissue-specific differences in vitamin D receptor-mediated responses in vivo that may be ascribed, at least in part, to differences in binding affinities for the vitamin D receptor.
Agić, Admir [Verfasser]. "Relative expression of 1,25-dihydroxyvitamin D3-receptor, vitamin D 1alpha-hydroxylase, vitamin D 24-hydroxylase and vitamin D 25-hydroxylase in endometriosis and gynecologic cancers / eingereicht von Admir Agic". 2008. http://d-nb.info/988454319/34.
Pełny tekst źródłaPöppelmeier, Olaf. "Funktionelle Analyse des Polymorphismus im Promotor des 24-Hydroxylase-Gens". Doctoral thesis, 2006. https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-23977.
Pełny tekst źródłaFunctional analysis of the polymorphism in the promotor of the 24-Hydroxylase gene Aim: A functional characterisation of the Polymorphism in the promotor of the 24-hydroxylase (CYP24) gene. The enzyme CYP24 initiates the degradation of the active vitamin D metabolite 1,25(OH)D3 as well as catalysing the production of 24,25(OH)D3, an active metabolite bone metabolism. If the polymorphism influenced the activity of CYP24, local or systemic levels of vitamin D could be altered or bone metabolism disturbed by varying 24,25(OH)D3 levels. The polymorphism could a factor in the pathogenesis of osteoporosis or other diseases of the skeletal system. Methods: Four different alleles of the polymorphism in the promotor of the CYP24 gene (WT, +A, +2A and +C5AC) as well as controls were amplified using PCR and cloned into a luciferase assay vector (pgl3-basic). Using electroporation the vector was transfected into two different cell systems, h-fob cells (human Osteoblasts) and T/C28a2 cells (human Chondrocytes). The cells were cultivated with and without the influence of vitamin D. The promotor activity was quantified using the luciferase assay. Results: By comparison with the controls a specific promotor activity could be demonstrated. For the allele +A a two fold increase and for the allele +2A a three fold increase in promotor activity compared to the wild type promotor was measured (p<0,001). The allele +C5AC showed no significant influence on the promotor activity. Under influence of vitamin D promotor activity was increase at least three times as high as in the basic setting. Discussion: Since the activity of the enzyme CYP24 is controlled mainly by the rate of transcription, the polymorphism in the promotor of the gene could influence CYP24 activity in vivo. The result could be a local or systemic decrease of 1,25(OH)D3 levels or increase of 24,25(OH)D3 levels. The polymorphism might therefore be relevant in the pathogenesis of osteoporosis, prostate cancer or other diseases associated with decreased levels of vitamin D
Zierold, Claudia. "The 24-hydroxylase promoter identification and characterization of its vitamin D response elements /". 1995. http://catalog.hathitrust.org/api/volumes/oclc/34654762.html.
Pełny tekst źródłaIsmail, Rohaizah. "Chicken vitamin D₃ 24-hydroxylase cDNA cloning and mRNA regulation by 1,25-dihydroxyvitamin Dr₃ and parathyroidectomy /". 1994. http://catalog.hathitrust.org/api/volumes/oclc/32033525.html.
Pełny tekst źródłaTan, Cheng Ta Joseph. "Regulation of the 24 - hydroxylase gene promoter by 1,25 - dihydroxyvitamin D3 and chemotherapeutics drugs". Thesis, 2005. http://hdl.handle.net/2440/37863.
Pełny tekst źródłaThesis (Ph.D.)--University of Adelaide, School of Paediatrics and Reproductive Health, 2005.
Tan, Cheng Ta Joseph. "Regulation of the 24 - hydroxylase gene promoter by 1,25 - dihydroxyvitamin D3 and chemotherapeutics drugs". 2005. http://hdl.handle.net/2440/37863.
Pełny tekst źródłaThesis (Ph.D.)--School of Paediatrics and Reproductive Health, 2005.
Części książek na temat "Vitamine D 24-hydroxylase"
Kharb, Simmi. "Role of Vitamin D in Preeclampsia". W Preeclampsia. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.100139.
Pełny tekst źródłaOMDAHL, JOHN, i BRIAN MAY. "The 25-Hydroxyvitamin D 24-Hydroxylase". W Vitamin D, 85–104. Elsevier, 2005. http://dx.doi.org/10.1016/b978-012252687-9/50009-7.
Pełny tekst źródłaVeldurthy, Vaishali, Ran Wei, Megan Campbell, Kamil Lupicki, Puneet Dhawan i Sylvia Christakos. "25-Hydroxyvitamin D3 24-Hydroxylase". W Vitamin D Hormone, 137–50. Elsevier, 2016. http://dx.doi.org/10.1016/bs.vh.2015.10.005.
Pełny tekst źródła"PURIFICATION OF 25-HYDROXYVITAMIN D3 24-HYDROXYLASE FROM RAT KIDNEY MITOCHONDRIA". W Vitamin D, 257–58. De Gruyter, 1991. http://dx.doi.org/10.1515/9783110850345-071.
Pełny tekst źródła"In Vivo Regulation of Rat Intestinal 24-Hydroxylase (I-24-OHase): Potential New Role of Calcitonin". W Vitamin D, 170–71. De Gruyter, 1994. http://dx.doi.org/10.1515/9783110882513-059.
Pełny tekst źródła"Differences of the Regulation of 24-Hydroxylase Gene Expression in the Kidney and Intestine". W Vitamin D, 113–21. De Gruyter, 1994. http://dx.doi.org/10.1515/9783110882513-042.
Pełny tekst źródła"Regulation of 24-Hydroxylase mRNA Expression by 1a,25(OH)2D3 and its Fluorinated Analogues". W Vitamin D, 155–56. De Gruyter, 1994. http://dx.doi.org/10.1515/9783110882513-056.
Pełny tekst źródła"EVIDENCE THAT RAT KIDNEY 25-HYDROXYVIΤΑΜIN D-24-HYDROXYLASE CAN EXIST IN AN INACTIVE STATE." W Vitamin D, 196–97. De Gruyter, 1988. http://dx.doi.org/10.1515/9783110846713.196.
Pełny tekst źródła"Influence of Extracellular Calcium in the Kinetic Behaviour of 1-Hydroxylase (1-OHase) and 24-Hydroxylase (24-OHase) in Walker Carcinosarcoma 256 Cells (WS) and in the Renal Proximal Tube Phenotype Cells (LLC-PK1)". W Vitamin D, 149–50. De Gruyter, 1994. http://dx.doi.org/10.1515/9783110882513-053.
Pełny tekst źródła"Protein Kinase C Up-Regulates 1 a,25-Dihydroxyvitamin D3-Induced Expression of 24-Hydroxylase Gene". W Vitamin D, 133–34. De Gruyter, 1994. http://dx.doi.org/10.1515/9783110882513-045.
Pełny tekst źródłaStreszczenia konferencji na temat "Vitamine D 24-hydroxylase"
Luo, Wei, Adam R. Karpf, Kristin K. Deeb, Josephia R. Muindi, Carl D. Morrison, Donald L. Trump i Candace S. Johnson. "Abstract 4944: Regulation of vitamin D 24-hydroxylase gene expression in human prostate cancer by epigenetic mechanisms". W Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4944.
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