Artykuły w czasopismach na temat „TREK-1 potassium channel”
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Pike, Ashley, Yin Dong, Alexandra Mackenzie, Conor McClenaghan, Shubhashish Mukhopadhyay, Nicola Burgess-Brown, Stephen Tucker i Elisabeth Carpenter. "Structures of the human two-pore domain potassium channels TREK-1 and TREK-2". Acta Crystallographica Section A Foundations and Advances 70, a1 (5.08.2014): C1489. http://dx.doi.org/10.1107/s2053273314085106.
Pełny tekst źródłaLevitz, Joshua, Perrine Royal, Yannick Comoglio, Brigitte Wdziekonski, Sébastien Schaub, Daniel M. Clemens, Ehud Y. Isacoff i Guillaume Sandoz. "Heterodimerization within the TREK channel subfamily produces a diverse family of highly regulated potassium channels". Proceedings of the National Academy of Sciences 113, nr 15 (28.03.2016): 4194–99. http://dx.doi.org/10.1073/pnas.1522459113.
Pełny tekst źródłaBrenner, Tanja, i Kevin M. O'Shaughnessy. "Both TASK-3 and TREK-1 two-pore loop K channels are expressed in H295R cells and modulate their membrane potential and aldosterone secretion". American Journal of Physiology-Endocrinology and Metabolism 295, nr 6 (grudzień 2008): E1480—E1486. http://dx.doi.org/10.1152/ajpendo.90652.2008.
Pełny tekst źródłaBlin, Sandy, Ismail Ben Soussia, Eun-Jin Kim, Frédéric Brau, Dawon Kang, Florian Lesage i Delphine Bichet. "Mixing and matching TREK/TRAAK subunits generate heterodimeric K2P channels with unique properties". Proceedings of the National Academy of Sciences 113, nr 15 (28.03.2016): 4200–4205. http://dx.doi.org/10.1073/pnas.1522748113.
Pełny tekst źródłaRichter, Trevor A., Gennady A. Dvoryanchikov, Nirupa Chaudhari i Stephen D. Roper. "Acid-Sensitive Two-Pore Domain Potassium (K2P) Channels in Mouse Taste Buds". Journal of Neurophysiology 92, nr 3 (wrzesień 2004): 1928–36. http://dx.doi.org/10.1152/jn.00273.2004.
Pełny tekst źródłaWiedmann, Felix, Daniel Schlund, Francisco Faustino, Manuel Kraft, Antonius Ratte, Dierk Thomas, Hugo A. Katus i Constanze Schmidt. "N-Glycosylation of TREK-1/hK2P2.1 Two-Pore-Domain Potassium (K2P) Channels". International Journal of Molecular Sciences 20, nr 20 (20.10.2019): 5193. http://dx.doi.org/10.3390/ijms20205193.
Pełny tekst źródłaPineda, Ricardo H., Balachandar Nedumaran, Joseph Hypolite, Xiao-Qing Pan, Shandra Wilson, Randall B. Meacham i Anna P. Malykhina. "Altered expression and modulation of the two-pore-domain (K2P) mechanogated potassium channel TREK-1 in overactive human detrusor". American Journal of Physiology-Renal Physiology 313, nr 2 (1.08.2017): F535—F546. http://dx.doi.org/10.1152/ajprenal.00638.2016.
Pełny tekst źródłaNamiranian, Khodadad, Eric E. Lloyd, Randy F. Crossland, Sean P. Marrelli, George E. Taffet, Anilkumar K. Reddy, Craig J. Hartley i Robert M. Bryan. "Cerebrovascular responses in mice deficient in the potassium channel, TREK-1". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 299, nr 2 (sierpień 2010): R461—R469. http://dx.doi.org/10.1152/ajpregu.00057.2010.
Pełny tekst źródłaYeliashov, S. I., B. R. Sharopov i Ya M. Shuba. "ROLE OF POTASSIUM CHANNEL TREK-1 IN MECHANOSENSITIVITY OF SMOOTH MUSCLE CELLS FROM RAT DETRUSOR". Fiziolohichnyĭ zhurnal 70, nr 2 (5.02.2024): 43–50. http://dx.doi.org/10.15407/fz70.02.035.
Pełny tekst źródłaVallee, Nicolas, Cédric Meckler, Jean-Jacques Risso i Jean-Eric Blatteau. "Neuroprotective role of the TREK-1 channel in decompression sickness". Journal of Applied Physiology 112, nr 7 (1.04.2012): 1191–96. http://dx.doi.org/10.1152/japplphysiol.01100.2011.
Pełny tekst źródłaYamamoto, Y., i K. Taniguchi. "Expression of Tandem P Domain K+ Channel, TREK-1, in the Rat Carotid Body". Journal of Histochemistry & Cytochemistry 54, nr 4 (6.01.2006): 467–72. http://dx.doi.org/10.1369/jhc.5a6755.2005.
Pełny tekst źródłaYang, Jun Ho, Adrian S. Siregar, Eun-Jin Kim, Marie Merci Nyiramana, Eui-Jung Shin, Jaehee Han, Ju-Tae Sohn, Jong Woo Kim i Dawon Kang. "Involvement of TREK-1 Channel in Cell Viability of H9c2 Rat Cardiomyoblasts Affected by Bupivacaine and Lipid Emulsion". Cells 8, nr 5 (14.05.2019): 454. http://dx.doi.org/10.3390/cells8050454.
Pełny tekst źródłaSchwingshackl, Andreas, Bin Teng, Manik Ghosh, Alina Nico West, Patrudu Makena, Vijay Gorantla, Scott E. Sinclair i Christopher M. Waters. "Regulation and function of the two-pore-domain (K2P) potassium channel Trek-1 in alveolar epithelial cells". American Journal of Physiology-Lung Cellular and Molecular Physiology 302, nr 1 (1.01.2012): L93—L102. http://dx.doi.org/10.1152/ajplung.00078.2011.
Pełny tekst źródłaYarishkin, Oleg, Tam T. T. Phuong, Colin A. Bretz, Kenneth W. Olsen, Jackson M. Baumann, Monika Lakk, Alan Crandall, Catherine Heurteaux, Mary E. Hartnett i David Križaj. "TREK-1 channels regulate pressure sensitivity and calcium signaling in trabecular meshwork cells". Journal of General Physiology 150, nr 12 (16.11.2018): 1660–75. http://dx.doi.org/10.1085/jgp.201812179.
Pełny tekst źródłaMagra, Merzesh, Steven Hughes, Alicia J. El Haj i Nicola Maffulli. "VOCCs and TREK-1 ion channel expression in human tenocytes". American Journal of Physiology-Cell Physiology 292, nr 3 (marzec 2007): C1053—C1060. http://dx.doi.org/10.1152/ajpcell.00053.2006.
Pełny tekst źródłaMathie, Alistair, Emma L. Veale, Kevin P. Cunningham, Robyn G. Holden i Paul D. Wright. "Two-Pore Domain Potassium Channels as Drug Targets: Anesthesia and Beyond". Annual Review of Pharmacology and Toxicology 61, nr 1 (6.01.2021): 401–20. http://dx.doi.org/10.1146/annurev-pharmtox-030920-111536.
Pełny tekst źródłaLesage, Florian, i Michel Lazdunski. "Molecular and functional properties of two-pore-domain potassium channels". American Journal of Physiology-Renal Physiology 279, nr 5 (1.11.2000): F793—F801. http://dx.doi.org/10.1152/ajprenal.2000.279.5.f793.
Pełny tekst źródłaBryan, Robert M., Junping You, Sharon C. Phillips, Jon J. Andresen, Eric E. Lloyd, Paul A. Rogers, Stuart E. Dryer i Sean P. Marrelli. "Evidence for two-pore domain potassium channels in rat cerebral arteries". American Journal of Physiology-Heart and Circulatory Physiology 291, nr 2 (sierpień 2006): H770—H780. http://dx.doi.org/10.1152/ajpheart.01377.2005.
Pełny tekst źródłaUllrich, Jessica, Carsten Ohlhoff, Srujan Kumar Dondapati, Anne Zemella i Stefan Kubick. "Evaluation of the Ion Channel Assembly in a Eukaryotic Cell-Free System Focusing on Two-Pore Domain Potassium Channels K2P". International Journal of Molecular Sciences 24, nr 7 (27.03.2023): 6299. http://dx.doi.org/10.3390/ijms24076299.
Pełny tekst źródłaAfzali, Ali M., Tobias Ruck, Alexander M. Herrmann, Janette Iking, Claudia Sommer, Christoph Kleinschnitz, Corinna Preuβe i in. "The potassium channels TASK2 and TREK1 regulate functional differentiation of murine skeletal muscle cells". American Journal of Physiology-Cell Physiology 311, nr 4 (1.10.2016): C583—C595. http://dx.doi.org/10.1152/ajpcell.00363.2015.
Pełny tekst źródłaSchwingshackl, Andreas, Bin Teng, Manik Ghosh, Keng Gat Lim, Gabor Tigyi, Damodaran Narayanan, Jonathan H. Jaggar i Christopher M. Waters. "Regulation of interleukin-6 secretion by the two-pore-domain potassium channel Trek-1 in alveolar epithelial cells". American Journal of Physiology-Lung Cellular and Molecular Physiology 304, nr 4 (15.02.2013): L276—L286. http://dx.doi.org/10.1152/ajplung.00299.2012.
Pełny tekst źródłaLolicato, Marco, Andrew M. Natale, Fayal Abderemane-Ali, David Crottès, Sara Capponi, Ramona Duman, Armin Wagner, John M. Rosenberg, Michael Grabe i Daniel L. Minor. "K2P channel C-type gating involves asymmetric selectivity filter order-disorder transitions". Science Advances 6, nr 44 (październik 2020): eabc9174. http://dx.doi.org/10.1126/sciadv.abc9174.
Pełny tekst źródłaNamiranian, Khodadad, Christa D. Brink, Jerry Clay Goodman, Claudia S. Robertson i Robert M. Bryan. "Traumatic Brain Injury in Mice Lacking the K Channel, TREK-1". Journal of Cerebral Blood Flow & Metabolism 31, nr 3 (15.12.2010): e1-e6. http://dx.doi.org/10.1038/jcbfm.2010.223.
Pełny tekst źródłaZhang, Yunna, Jiafeng Fu, Yang Han, Dandan Feng, Shaojie Yue, Yan Zhou i Ziqiang Luo. "Two-Pore-Domain Potassium Channel TREK–1 Mediates Pulmonary Fibrosis through Macrophage M2 Polarization and by Direct Promotion of Fibroblast Differentiation". Biomedicines 11, nr 5 (26.04.2023): 1279. http://dx.doi.org/10.3390/biomedicines11051279.
Pełny tekst źródłaLengyel, Miklós, Gábor Czirják i Péter Enyedi. "Formation of Functional Heterodimers by TREK-1 and TREK-2 Two-pore Domain Potassium Channel Subunits". Journal of Biological Chemistry 291, nr 26 (28.04.2016): 13649–61. http://dx.doi.org/10.1074/jbc.m116.719039.
Pełny tekst źródłaBlondeau, Nicolas, Olivier Pétrault, Stella Manta, Valérie Giordanengo, Pierre Gounon, Régis Bordet, Michel Lazdunski i Catherine Heurteaux. "Polyunsaturated Fatty Acids Are Cerebral Vasodilators via the TREK-1 Potassium Channel". Circulation Research 101, nr 2 (20.07.2007): 176–84. http://dx.doi.org/10.1161/circresaha.107.154443.
Pełny tekst źródłaXIANTAOLI, V. DYACHENKO, M. ZUZARTE, C. PUTZKE, R. PREISIGMULLER, G. ISENBERG i J. DAUT. "The stretch-activated potassium channel TREK-1 in rat cardiac ventricular muscle". Cardiovascular Research 69, nr 1 (styczeń 2006): 86–97. http://dx.doi.org/10.1016/j.cardiores.2005.08.018.
Pełny tekst źródłaWang, Ying, Zhijie Fu, Zhiyong Ma, Na Li i Hong Shang. "Bepridil, a class IV antiarrhythmic agent, can block the TREK-1 potassium channel". Annals of Translational Medicine 9, nr 14 (lipiec 2021): 1123. http://dx.doi.org/10.21037/atm-20-7971.
Pełny tekst źródłaMonaghan, Kevin, Salah A. Baker, Laura Dwyer, William C. Hatton, Kyung Sik Park, Kenton M. Sanders i Sang Don Koh. "The stretch-dependent potassium channel TREK-1 and its function in murine myometrium". Journal of Physiology 589, nr 5 (25.02.2011): 1221–33. http://dx.doi.org/10.1113/jphysiol.2010.203869.
Pełny tekst źródłaEnyeart, John J., i Judith A. Enyeart. "Human adrenal glomerulosa cells express K2P and GIRK potassium channels that are inhibited by ANG II and ACTH". American Journal of Physiology-Cell Physiology 321, nr 1 (1.07.2021): C158—C175. http://dx.doi.org/10.1152/ajpcell.00118.2021.
Pełny tekst źródłaVivier, Delphine, Khalil Bennis, Florian Lesage i Sylvie Ducki. "Perspectives on the Two-Pore Domain Potassium Channel TREK-1 (TWIK-Related K+Channel 1). A Novel Therapeutic Target?" Journal of Medicinal Chemistry 59, nr 11 (14.12.2015): 5149–57. http://dx.doi.org/10.1021/acs.jmedchem.5b00671.
Pełny tekst źródłaKim, Ajung, Hyun-Gug Jung, Yeong-Eun Kim, Seung-Chan Kim, Jae-Yong Park, Seok-Geun Lee i Eun Mi Hwang. "The Knockdown of TREK-1 in Hippocampal Neurons Attenuate Lipopolysaccharide-Induced Depressive-Like Behavior in Mice". International Journal of Molecular Sciences 20, nr 23 (24.11.2019): 5902. http://dx.doi.org/10.3390/ijms20235902.
Pełny tekst źródłaBorsotto, M., A. Djillani, C. Devader, C. Heurteaux i J. Mazella. "The Mini-Spadin, an efficient alternate to Spadin in the depression treatment". European Psychiatry 33, S1 (marzec 2016): S407. http://dx.doi.org/10.1016/j.eurpsy.2016.01.1470.
Pełny tekst źródłaAbraham, Dennis M., Teresa E. Lee, Lewis J. Watson, Lan Mao, Gurangad Chandok, Hong-Gang Wang, Stephan Frangakis i in. "The two-pore domain potassium channel TREK-1 mediates cardiac fibrosis and diastolic dysfunction". Journal of Clinical Investigation 128, nr 11 (2.10.2018): 4843–55. http://dx.doi.org/10.1172/jci95945.
Pełny tekst źródłaHeurteaux, Catherine, Guillaume Lucas, Nicolas Guy, Malika El Yacoubi, Susanne Thümmler, Xiao-Dong Peng, Florence Noble i in. "Deletion of the background potassium channel TREK-1 results in a depression-resistant phenotype". Nature Neuroscience 9, nr 9 (13.08.2006): 1134–41. http://dx.doi.org/10.1038/nn1749.
Pełny tekst źródłaJi, Xin-cai, Wan-hong Zhao, Dong-xu Cao, Qiao-qiao Shi i Xiao-liang Wang. "Novel neuroprotectant chiral 3-n-butylphthalide inhibits tandem-pore-domain potassium channel TREK-1". Acta Pharmacologica Sinica 32, nr 2 (luty 2011): 182–87. http://dx.doi.org/10.1038/aps.2010.210.
Pełny tekst źródłaZhiyong, M. A., Zhijie Fu, Na Li, Shanying Huang, Li Li, Yun Zhang i Zhijie Fu. "GW29-e1702 Bepridil a class IV antiarrhythmic agent can block TREK-1 potassium channel". Journal of the American College of Cardiology 72, nr 16 (październik 2018): C55. http://dx.doi.org/10.1016/j.jacc.2018.08.201.
Pełny tekst źródłaWu, Xiao, Ronghua Tang, Yang Liu, Jingjiao Song, Zhiyuan Yu, Wei Wang i Minjie Xie. "Small RNA interference-mediated gene silencing of TREK-1 potassium channel in cultured astrocytes". Journal of Huazhong University of Science and Technology [Medical Sciences] 32, nr 6 (grudzień 2012): 849–55. http://dx.doi.org/10.1007/s11596-012-1046-y.
Pełny tekst źródłaZhao, Ke-Qing, Guoxiang Xiong, Morgan Wilber, Noam A. Cohen i James L. Kreindler. "A role for two-pore K+ channels in modulating Na+ absorption and Cl− secretion in normal human bronchial epithelial cells". American Journal of Physiology-Lung Cellular and Molecular Physiology 302, nr 1 (1.01.2012): L4—L12. http://dx.doi.org/10.1152/ajplung.00102.2011.
Pełny tekst źródłaWiedmann, Felix, Norbert Frey i Constanze Schmidt. "Two-Pore-Domain Potassium (K2P-) Channels: Cardiac Expression Patterns and Disease-Specific Remodelling Processes". Cells 10, nr 11 (27.10.2021): 2914. http://dx.doi.org/10.3390/cells10112914.
Pełny tekst źródłaMuller-Delp, J. M. "The role of TREK-1 in cerebrovascular regulation remains elusive: focus on “Cerebrovascular responses in mice deficient in the potassium channel, TREK-1”". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 299, nr 2 (sierpień 2010): R459—R460. http://dx.doi.org/10.1152/ajpregu.00277.2010.
Pełny tekst źródłaFang, Yongkang, Xiaojiang Huang, Yue Wan, Hao Tian, Yeye Tian, Wei Wang, Suiqiang Zhu i Minjie Xie. "Deficiency of TREK-1 potassium channel exacerbates secondary injury following spinal cord injury in mice". Journal of Neurochemistry 141, nr 2 (15.03.2017): 236–46. http://dx.doi.org/10.1111/jnc.13980.
Pełny tekst źródłaYin, Zongzhi, Yun Li, Wenzhu He, Dan Li, Hongyan Li, Yuanyuan Yang, Bing Shen, Xi Wang, Yunxia Cao i Raouf A. Khalil. "Progesterone inhibits contraction and increases TREK-1 potassium channel expression in late pregnant rat uterus". Oncotarget 9, nr 1 (7.12.2017): 651–61. http://dx.doi.org/10.18632/oncotarget.23084.
Pełny tekst źródłaSharma, Pankaj, Stephen Hughes, Alicia El Haj i Nicola Maffulli. "Expression of the Two Pore Domain Potassium Channel TREK-1 in Human Intervertebral Disc Cells". Current Stem Cell Research & Therapy 7, nr 4 (1.05.2012): 266–71. http://dx.doi.org/10.2174/157488812800793072.
Pełny tekst źródłaMaingret, François, Amanda J. Patel, Florian Lesage, Michel Lazdunski i Eric Honoré. "Mechano- or Acid Stimulation, Two Interactive Modes of Activation of the TREK-1 Potassium Channel". Journal of Biological Chemistry 274, nr 38 (17.09.1999): 26691–96. http://dx.doi.org/10.1074/jbc.274.38.26691.
Pełny tekst źródłaSchwingshackl, Andreas, Bin Teng, Patrudu Makena, Manik Ghosh, Scott E. Sinclair, Charlean Luellen, Louisa Balasz i in. "Deficiency of the Two-Pore-Domain Potassium Channel TREK-1 Promotes Hyperoxia-Induced Lung Injury". Critical Care Medicine 42, nr 11 (listopad 2014): e692-e701. http://dx.doi.org/10.1097/ccm.0000000000000603.
Pełny tekst źródłaStebe, Sabrina, Katharina Schellig, Florian Lesage, Heinz Breer i Joerg Fleischer. "The Thermosensitive Potassium Channel TREK-1 Contributes to Coolness-Evoked Responses of Grueneberg Ganglion Neurons". Cellular and Molecular Neurobiology 34, nr 1 (8.10.2013): 113–22. http://dx.doi.org/10.1007/s10571-013-9992-x.
Pełny tekst źródłaMeadows, H. J., C. D. Benham, W. Cairns, I. Gloger, C. Jennings, A. D. Medhurst, P. Murdock i C. G. Chapman. "Cloning, localisation and functional expression of the human orthologue of the TREK-1 potassium channel". Pfl�gers Archiv European Journal of Physiology 439, nr 6 (23.03.2000): 714–22. http://dx.doi.org/10.1007/s004240050997.
Pełny tekst źródłaMeadows, H. J., C. D. Benham, W. Cairns, I. Gloger, C. Jennings, A. D. Medhurst, P. Murdock i C. G. Chapman. "Cloning, localisation and functional expression of the human orthologue of the TREK-1 potassium channel". Pflügers Archiv - European Journal of Physiology 439, nr 6 (24.01.2000): 714–22. http://dx.doi.org/10.1007/s004249900235.
Pełny tekst źródłaRinné, Susanne, Vijay Renigunta, Günter Schlichthörl, Marylou Zuzarte, Stefan Bittner, Sven G. Meuth, Niels Decher, Jürgen Daut i Regina Preisig-Müller. "A splice variant of the two-pore domain potassium channel TREK-1 with only one pore domain reduces the surface expression of full-length TREK-1 channels". Pflügers Archiv - European Journal of Physiology 466, nr 8 (7.11.2013): 1559–70. http://dx.doi.org/10.1007/s00424-013-1384-z.
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