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Książki na temat „Thiamine deficiency”

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Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 20 lutego 2023

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Sprawdź 17 najlepszych książek naukowych na temat „Thiamine deficiency”.

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1

McCandless, David W. Thiamine deficiency and associated clinical disorders. New York: Humana Press, 2010.

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2

McCandless, David W. Thiamine Deficiency and Associated Clinical Disorders. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-311-4.

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3

Beriberi, white rice, and vitamin B: A disease, a cause, and a cure. Berkeley, CA: University of California Press, 2000.

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4

Carpenter, Kenneth J. Beriberi, white rice, and vitamin B: A disease, a cause, and a cure. Berkeley, CA: University of California Press, 2000.

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5

Carpenter, Kenneth J. Beriberi, white rice, and vitamin B: A disease, a cause, and a cure. Berkeley, CA: University of California Press, 2000.

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6

McCandless, David W. Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2012.

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7

Marrs, Chandler, i Derrick Lonsdale. Thiamine Deficiency Disease, Dysautonomia, and High Calorie Malnutrition. Elsevier Science & Technology Books, 2017.

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8

Marrs, Chandler, i Derrick Lonsdale. Thiamine Deficiency Disease, Dysautonomia, and High Calorie Malnutrition. Elsevier Science & Technology Books, 2017.

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9

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng i Anne Bardsley. Vitamin B1 (thiamine) in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0007.

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Vitamin B1 (thiamine) is involved in nervous system and muscle function and is essential for carbohydrate metabolism. Deficiency is common in Asia, where diets are often high in thiamine-depleted polished rice and can be low in other food sources. Pregnancy imposes an increasing requirement for thiamine over the course of gestation, and deficiency can lead to widespread metabolic disturbances affecting the placenta and fetus. Nutritional deficiency for thiamine is rare in people who consume a moderately varied diet that contains whole grains. However, excessive vomiting in pregnancy can cause thiamine depletion, in which case antenatal vitamins containing thiamine and other B vitamins may be beneficial.
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10

Janssen, Mirian C. H., i Shamima Rahman. Pyruvate Dehydrogenase Complex Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0006.

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Pyruvate dehydrogenase complex (PDHc) deficiency usually first manifests at a young age and is rarely diagnosed in adulthood. The clinical picture varies from neonatal death with overwhelming lactic acidosis to a relatively benign course early in life. The three main presentations are congenital lactic acidosis, Leigh syndrome, and episodic ataxia. Treatment consists of a ketogenic diet and cofactor supplementation with thiamine. Successful therapy is rare.
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11

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson i Nathaniel M. Robbins. A 40-Year-Old Female with Balance Problems and Numbness After Bariatric Surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0011.

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A number of nutritional causes of peripheral neuropathy have been identified. Patients who have gastrointestinal disease or who have undergone bariatric surgery are at risk, although frequently the specific cause is not ascertained. Vitamin B12 and thiamine deficiencies are the most common causes in bariatric surgery patients. The classic clinical picture of Vitamin B12 deficiency is a myelopathy with a concomitant peripheral neuropathy. Thiamine deficiency is the classic triad of mental status changes, opthalmoplegia, and ataxia. Copper deficiency is often unrecognized. Its presentation can be similar to Vitamin B12 deficiency. It is important that bariatric surgery patients receive continual follow up of their nutritional status and adherence to vitamin supplementation. Alcohol may cause a peripheral neuropathy through nutritional deficiency or perhaps direct toxic effects. Other deficiency states are discussed.
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12

Zenuk, Cecli. Prevalence of thiamine deficiency in congestive heart failure patients receiving long-term furosemide therapy. 2000.

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13

Ramrakha, Punit, i Jonathan Hill, red. Cardiovascular disease in less-developed countries. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199643219.003.0014.

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Burden of cardiovascular disease in less-developed countries 654Infectious disease and the heart 656Diphtheria 657HIV and the cardiovascular system 658Chagas’ disease and the heart 660Cardiovascular syphilis 662Beri-beri (thiamine deficiency) 664Tetanus 666Keshan disease 667Restrictive cardiomyopathy 667Endomyocardial fibrosis ...
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14

Carpenter, Kenneth J. Beriberi, White Rice and Vitamin B: A Disease, a Cause, and a Cure. Diane Pub Co, 2000.

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15

Silva, Aminda De, J. A. Saunders i M. A. Stroud. Vitamin deficiencies. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0333.

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Vitamins are organic compounds required by the body in small amounts to perform specific cellular functions. Nine vitamins (thiamine (vitamin B1), riboflavin (vitamin B2), pyridoxine (vitamin B6), cyanocobalamin (vitamin B12), niacin (nicotinic acid; vitamin B3), pantothenic acid (vitamin B5), biotin (vitamin B7; vitamin H), folic acid (folate; vitamin B9), and ascorbic acid (vitamin C)) are water soluble, while four (vitamins A, D, E, and K) are fat soluble. The importance of vitamins was first appreciated through recognition of their clinical deficiency state. However, this approach has led to the concept that the primary purpose of a vitamin is to prevent the associated clinical deficiency state and, consequently, unless patients exhibit signs of a specific clinical deficiency state, they are thought to be replete in the corresponding vitamin. This is a misunderstanding. In reality, most vitamins have many different functions which are incompletely understood, and impaired biochemical function and even functional problems affecting metabolic, immunological, or cognitive status can occur with marginal vitamin depletion long before overt clinical deficiency becomes evident. A high index of suspicion is thus essential in all patients who have malnutrition or malabsorption, to ensure that levels that might compromise health, resistance to disease, and recovery from injury or illness are not left untreated.
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16

Shangari, Nandita. Interdependent toxicity mechanisms of oxidative stress, thiamin deficiency and alpha-oxoaldehydes. 2006.

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17

Hanninen, Stacy Ann. The prevalence of thiamin deficiency in hospitalized patients with congestive heart failure. 2004.

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