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Artykuły w czasopismach na temat "Thérapie de réseau"
Nisse, Martine. "Les maltraitances : Thérapie familiale de réseau". Imaginaire & Inconscient 16, nr 2 (2005): 33. http://dx.doi.org/10.3917/imin.016.0033.
Pełny tekst źródłaBlondeau, Hélène, Anne-Christine Frankard i Eliane Pirard. "Le réseau, partenaire et ressource de la thérapie". Thérapie Familiale 22, nr 4 (2001): 371. http://dx.doi.org/10.3917/tf.014.0371.
Pełny tekst źródłaHoltzmann, J. "Nouvelles perspectives de prise en charge – apports des nouvelles technologies". European Psychiatry 30, S2 (listopad 2015): S38—S39. http://dx.doi.org/10.1016/j.eurpsy.2015.09.110.
Pełny tekst źródłaGrasset, François, i Michel Ducret. "Thérapie institutionnelle, approche écosystémique et réadaptation psycho-sociale". Santé mentale au Québec 18, nr 2 (11.09.2007): 155–81. http://dx.doi.org/10.7202/032276ar.
Pełny tekst źródłaFaretta, Elisa, i Cristina Civilotti. "Thérapie EMDR et psycho-oncologie : un pont entre le corps et l'esprit". Journal of EMDR Practice and Research 11, nr 4 (2017): 102E—117E. http://dx.doi.org/10.1891/1933-3196.11.4.102.
Pełny tekst źródłaMoreau, Maurice. "L’approche structurelle familiale en service social : le résultat d’un itinéraire critique". Travailler le social, nr 7 (3.02.2016): 159–71. http://dx.doi.org/10.7202/1035024ar.
Pełny tekst źródłaHeller, Geneviève. "La doctoresse Charlotte Olivier (1865—1945) et la prise en charge des tuberculeux indigents à Lausanne". Gesnerus 48, nr 3-4 (25.11.1991): 463–76. http://dx.doi.org/10.1163/22977953-0480304018.
Pełny tekst źródłaRichieri, R. M., D. Ducasse, O. Doumy i J. Holtzmann. "Psychothérapies et dépression". European Psychiatry 30, S2 (listopad 2015): S37. http://dx.doi.org/10.1016/j.eurpsy.2015.09.107.
Pełny tekst źródłaAmétépé, L. "Association EMDR France – Indication de l’EMDR dans le traitement des révélations tardives de violences sexuelles". European Psychiatry 30, S2 (listopad 2015): S95. http://dx.doi.org/10.1016/j.eurpsy.2015.09.404.
Pełny tekst źródłaFerrara, Marianna. "Olga Fröbe-Kapteyn’s Ashram : The Great Mother and the Personal History of Eastern Religions". ASDIWAL. Revue genevoise d'anthropologie et d'histoire des religions 16, nr 1 (2021): 79–94. http://dx.doi.org/10.3406/asdi.2021.1205.
Pełny tekst źródłaRozprawy doktorskie na temat "Thérapie de réseau"
Henri, Orianne. "Impact de l'insuffisance cardiaque sur les réseau et transport lymphatiques cardiaques et thérapie pro-lymphangiogénique". Rouen, 2016. http://www.theses.fr/2016ROUES046.
Pełny tekst źródłaThe pathophysiology of myocardial infarction (MI) includes blood capillary rarefaction, inflammation, and cardiac fibrosis and dysfunction, contributing to heart failure (HF). Studies have demonstrated that cardiac edema occurs leading directly to cardiac fibrosis and amplifying the cardiac dysfunction. Using a HF model based on MI in rats, we demonstrate an insufficient cardiac lymphatic transport capacity post-MI in part due to insufficient lymphangiogenic response and deleterious lymphatic remodeling contributing to formation of chronic cardiac edema and inflammation, beyond the infarct scar. In the laboratory, an angiogenic therapy based on targeted and sustained release of angiogenic growth factors via biopolymers was demonstrated as sufficient to limit the capillary depletion and to improve cardiac function post-MI. Here, we used polymers loaded with a mutated form of VEGFC, essential lymphangiogenic growth factor, to determine whether selective therapeutic lymphangiogenesis may show benefit in HF. We found that this treatment accelerated the endogenous lymphangiogenesis process during the early weeks post-MI and treated animals showed prevention superficial cardiac lymphatics remodeling post-MI. The therapy led to improved cardiac lymphatic drainage and accelerated resolution of cardiac edema and inflammation and impacted on cardiac remodeling by preventing the installation of fibrosis, and improved heart function. Our results show for the first time that a targeted VEGF-C therapy improves lymphatic transport and cardiac function post-MI and suggests that therapeutic lymphangiogenesis, alongside therapeutic angiogenesis, may be considered to treat HF in patients
Garcia, Marie-Paule. "Caractérisation du réseau veineux en imagerie scanner 4D : contribution à la planification de thérapie par resynchronisation cardiaque". Phd thesis, Université Rennes 1, 2011. http://tel.archives-ouvertes.fr/tel-00590975.
Pełny tekst źródłaTurcotte, Linda. "Thérapie brève auprès de travailleurs du réseau de la santé et des services sociaux en épuisement professionnel". Mémoire, Université de Sherbrooke, 2006. http://hdl.handle.net/11143/5536.
Pełny tekst źródłaGagnon, Séverine. "Evaluation et prise en charge pluridisciplinaire du patient lombalgique chronique au sein du Réseau Nord - Pas de Calais du Dos (RENODOS)". Lille 2, 2008. http://www.theses.fr/2008LIL2S036.
Pełny tekst źródłaRuel, Annie. "Le rôle du réseau relationnel dans la décision d'entreprendre une thérapie : perspectives des usagers du Centre de réadaptation Ubald-Villeneuve orientés vers le programme intensif". Thesis, Université Laval, 2009. http://www.theses.ulaval.ca/2009/26192/26192.pdf.
Pełny tekst źródłaBastelica, Thomas. "Approche en réseau des interrelations entre cognition et facteurs associés : vers une perspective intégrative". Electronic Thesis or Diss., Lyon 1, 2024. http://www.theses.fr/2024LYO10331.
Pełny tekst źródłaAlterations in cognitive functions are encountered in many neurological or psychiatric conditions, or during the natural aging process. These dysfunctions significantly impair individuals' autonomy, quality of life, and psychosocial functioning. Therefore, their management occupies a central place. This thesis aims to explore factors associated with cognitive alterations, including psychological variables, such as personality, as well as biological factors related to immuno-inflammatory processes. Although previous work has highlighted associations between these various factors and cognitive processes, studying their interactions remains difficult to grasp with conventional statistical approaches in psychology. The network approach is a statistical tool that captures the interrelationships between components of a complex system. This new tool has been the subject of growing interest in psychiatric research in recent years. This thesis work relies on a network approach and has a dual objective. The first, of a clinical nature, consists of deepening the understanding of factors influencing cognitive functions, and examining how cognitive processes are linked to individuals' psychosocial functioning and quality of life. The second objective, methodological, is based on the use of a Bayesian statistical inference method. In this context, we propose a method allowing the consideration of results from previous network analysis studies (by specifying the prior distribution). Three studies were conducted. A first study, in elderly patients free of neurodegenerative disease, showed close links between personality traits and certain cognitive processes. However, no relationship between cognition and inflammation was observed. In a second study, carried out in a large cohort of patients living with bipolar disorders, no relationship between cognition and inflammation was found. The Bayesian approach allowed to confirm these results by providing strong evidence in favor of the absence of association. Finally, a third study conducted in patients living with schizophrenia spectrum disorder showed that certain cognitive functions are more closely associated than others with the level of functioning. Moreover, we showed that taking into account previous studies allowed for more reliable and robust inferences. In conclusion, this thesis work shows the importance of considering complex interactions between different factors (psychological or biological), the level of functioning and quality of life, and cognitive alterations. Our work also highlights the value of Bayesian methods for exploring these interrelationships. A better understanding of the mechanisms underlying cognitive dysfunctions could potentially open new therapeutic avenues, and thus improve patients' functioning and quality of life
Beganton, Benoît. "Caractérisation des réseaux d’interactions protéiques associés aux mutations oncogéniques principales retrouvées dans le cancer du poumon non à petites cellules". Thesis, Montpellier, 2017. http://www.theses.fr/2017MONTT157/document.
Pełny tekst źródłaLung cancer is the leading cause of cancer-related death in France and in the world. It is a cancer of poor prognosis, diagnosed at the late stage III or IV, with a 5-years survival of 6% and 1%, respectively. This cancer encompass several histological types, and among them adenocarcinoma account for 40% of the diagnosis. Genetic sequencing of stage IV tumors highlights redundant mutations, and generally exclusives from each other’s, of KRAS, EGFR, BRAF and ALK genes. The identification of these mutations enable, within companion test, to make eligible patients for targeted therapies when molecules are available.Even though these targeted therapies represent a true revolution in patient’s care, the majority of them cannot benefit from these treatments because their tumors do not harbor activating mutations that are targetable (e.g. absence of EGFR, BRAF and ALK mutation, presence of KRAS mutation). Additionally, when they can be treated using an oral molecule, the benefit observed is unfortunately poor in terms of period of time, and all the patients will escape from the treatment. This is for example the case with EGFR mutations.To better understand the molecular mechanisms associated with the resistance events observed in the clinic, and to propose new therapeutics for patient not-eligible for targeted therapies, I studied at the proteome level, the impact these mutations on protein networks, using the BioID technology (proximity-dependent biotinylation identification). More particularly, I have been interested in the activating mutation of EGFR, KRAS, BRAF and ALK. Considering that proteins from the RAS family (HRAS, NRAS and KRAS) are mutated in around 30% of cancers, I have been also interested in the protein network of these proteins to highlight interaction specific to the KRAS isoform.During my thesis, I showed that the protein networks characterized using BioID are much more dense compared to those identified with the more conventional technic of AP-MS (Affinity-purification and mass spectrometry), and that they enable to identify interactors specifically deregulated upon activating mutation. Additionally, the HEK293 cell model (Human Embryonic Kidney) and BEAS2B cell model (non-cancerous lung cell line) showed a high overlapping degree of the interactors identified, suggesting that the strategy used is relevant to identify interactors specific to mutations. This thesis enabled to identify several interactors specific to the mutant KRAS, EGFR, BRAF, NRAS and EML4-ALk fusion. Thirteen interactors specific to the mutated-KRAS have been functionally validated in lung-cancer cell lines models. Finally, using BioID data I have been able to propose a model of EGFR resistance to targeted therapies. This model shows that CBL and IGH2R might be the EGFR partners responsible for therapeutic escape.Altogether, this thesis propose new perspective to determine resistance mechanisms and to identify new therapeutic targets for KRAS-mutated patients
Decalf, Jérémie. "Les cellules dendritiques plasmacytoïdes au cours de l'infection chronique par le virus de l'hépatite C : de l'immunologie fondamentale à l'application dans le développement de nouvelles thérapies". Paris 6, 2008. http://www.theses.fr/2008PA066030.
Pełny tekst źródłaLouçã, Mathilde. "Functional impacts of Huntingtin lowering on the synaptic maturation and activity of neuronal networks derived from human induced pluripotent stem cells". Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL054.
Pełny tekst źródłaHuntington's disease (HD) is a neurodegenerative disorder caused by a mutation in the Huntingtin gene (HTT). Reducing the expression of mutant HTT is an obvious therapeutic approach explored in patients. However, targeting mutant HTT often leads to a simultaneous reduction in non-mutant HTT. The consequences of losing this protein on neuronal health remain poorly understood.My doctoral work addresses this question using in vitro models of human neuronal networks differentiated from induced pluripotent stem cells. My research demonstrates that HTT loss induces developmental and homeostatic abnormalities in these networks. My results suggest that therapies targeting both mutant and non-mutant HTT indiscriminately could compromise the health of targeted neuronal circuits
Vernon, Megane. "Caractérisation des cibles de microARNs tueurs et des réseaux de régulation associés dans les cancers de l'ovaire". Thesis, Normandie, 2018. http://www.theses.fr/2018NORMC419.
Pełny tekst źródłaImproving the personalized management of ovarian cancer requires the development of new therapeutic approaches and tools able to predict treatment response and recurrence. The study of cellular and circulating miRNAs represents a promising field of investigation in oncology, and they could quickly constitute new diagnostic, prognostic and even therapeutic tools. Following a high-throughput functional screening of a miRNA library on ovarian cancer lines, we identified several miRNAs of interest. While the use of miRNAs as therapeutic agents is not currently possible, the characterization of the targets «step-by-step»of one of these candidates, the miR-3622b-5p, with its broad anti-tumoral activity (pro-apoptic, sensibilisation to cisplatin and anti-invasive) on a panel of ovarian cancer lines, made it possible to reinforce the interest of the approach which we develop which consists in reproducing the effect of miRNAs by targeting the determinants of their action using inhibitory molecules, potentially usable in the clinic. In parallel, in order to globally identify the targets (their direct and indirect aspect next to a miRNA then becoming secondary) and associated networks of the most promising miRNAs, identified during the screening, and initially based on the first laboratory-identified apoptomiR, the miR-491-5p, to provide a proof of concept, an evaluation of the methodological approach combining data from multi-omic strategies led to the conclusion that it is useful to combine the trancriptomic and proteomic analyses to identify new targets/pathways related to its pro-apoptotic action and thus propose relevant and innovative pharmacological associations for ovarian cancers. In addition, we have identified a serum miRNA signature capable of identifying patients who may be resistant to platinum-based chemotherapy and potentially PARP inhibitors, prior to any chemotherapy and at the time of recurrence before the second line of treatment. In summary, these results offer great promise and place miRNAs at the heart of tomorrow's precision medicine in ovarian cancer
Książki na temat "Thérapie de réseau"
Guay, Jérôme. Thérapie brève et intervention de réseau: Une approche intégrée. Montréal, Qué: Presses de l'Université de Montréal, 1992.
Znajdź pełny tekst źródłaLacroix, Jean-Luc. L' individu, sa famille et son réseau: Les thérapies familiales systémiques. Paris: Editions ESF, 1990.
Znajdź pełny tekst źródłaLiz, King, Beak Dick i Centre for Fun & Families., red. Promoting positive parenting of teenagers. Aldershot, Hampshire, England: Arena, 1998.
Znajdź pełny tekst źródłaSystemscentered Theory And Practice The Contribution Of Yvonne Agazarian. Karnac Books, 2011.
Znajdź pełny tekst źródłaNeville, David. Promoting Positive Parenting of Teenagers. Taylor & Francis Group, 2017.
Znajdź pełny tekst źródłaNeville, David, i Liz King. Promoting Positive Parenting of Teenagers. Taylor & Francis Group, 2017.
Znajdź pełny tekst źródłaNeville, David, i Liz King. Promoting Positive Parenting of Teenagers. Taylor & Francis Group, 2017.
Znajdź pełny tekst źródłaArt Therapist's Guide to Social Media: Connection, Community, and Creativity. Taylor & Francis Group, 2017.
Znajdź pełny tekst źródłaMiller, Gretchen M. Art Therapist's Guide to Social Media: Connection, Community, and Creativity. Taylor & Francis Group, 2017.
Znajdź pełny tekst źródłaMiller, Gretchen M. Art Therapist's Guide to Social Media: Connection, Community, and Creativity. Taylor & Francis Group, 2017.
Znajdź pełny tekst źródłaCzęści książek na temat "Thérapie de réseau"
Goldbeter, Edith. "Cahiers critiques de thérapie familiale et de pratiques de réseaux (Journal)". W Encyclopedia of Couple and Family Therapy, 377–78. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-49425-8_956.
Pełny tekst źródłaGoldbeter, Edith. "Cahiers critiques de thérapie familiale et de pratiques de réseaux (Journal)". W Encyclopedia of Couple and Family Therapy, 1–2. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-15877-8_956-1.
Pełny tekst źródła"Les psychothérapies individuelles indiquées dans les troubles psychiques post-traumatiques de guerre". W Médecine et Armées Vol. 46 No.1, 91–96. Editions des archives contemporaines, 2018. http://dx.doi.org/10.17184/eac.7373.
Pełny tekst źródłaCottencin, Olivier. "Thérapies brèves en psychiatrie de liaison et application au travail en réseau". W Interventions et Thérapies Brèves : 12 Stratégies Concrètes, 229–48. Elsevier, 2020. http://dx.doi.org/10.1016/b978-2-294-76812-5.00008-1.
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