Gotowe bibliografie tematyczne / Tendon healing

Gotowa bibliografia na temat „Tendon healing”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 1 lutego 2022

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Artykuły w czasopismach na temat "Tendon healing"

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Hope, Matthew, i Terry S. Saxby. "Tendon Healing". Foot and Ankle Clinics 12, nr 4 (grudzień 2007): 553–67. http://dx.doi.org/10.1016/j.fcl.2007.07.003.

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Al-Qattan, Mohammad M., Jeffrey C. Posnick, Kant Y. Lin i Paul Thorner. "Fetal Tendon Healing". Plastic and Reconstructive Surgery 92, nr 6 (listopad 1993): 1155–60. http://dx.doi.org/10.1097/00006534-199311000-00024.

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Al-Qattan, Mohammad M., Jeffrey C. Posnick, Kant Y. Lin, Paul Thorner i John A. I. Grossman. "Fetal Tendon Healing". Plastic and Reconstructive Surgery 92, nr 6 (listopad 1993): 1161. http://dx.doi.org/10.1097/00006534-199311000-00025.

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MANSKE, P. "Flexor tendon healing". Journal of Hand Surgery: Journal of the British Society for Surgery of the Hand 13, nr 3 (sierpień 1988): 237–45. http://dx.doi.org/10.1016/0266-7681(88)90077-0.

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Manske, Paul R., Richard H. Gelberman i Peggy A. Lesker. "Flexor Tendon Healing". Hand Clinics 1, nr 1 (luty 1985): 25–34. http://dx.doi.org/10.1016/s0749-0712(21)01329-9.

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Chartier, Christian, Hassan ElHawary, Aslan Baradaran, Joshua Vorstenbosch, Liqin Xu i Johnny Ionut Efanov. "Tendon: Principles of Healing and Repair". Seminars in Plastic Surgery 35, nr 03 (15.07.2021): 211–15. http://dx.doi.org/10.1055/s-0041-1731632.

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AbstractTendon stores, releases, and dissipates energy to efficiently transmit contractile forces from muscle to bone. Tendon injury is exceedingly common, with the spectrum ranging from chronic tendinopathy to acute tendon rupture. Tendon generally develops according to three main steps: collagen fibrillogenesis, linear growth, and lateral growth. In the setting of injury, it also repairs and regenerates in three overlapping steps (inflammation, proliferation, and remodeling) with tendon-specific durations. Acute injury to the flexor and extensor tendons of the hand are of particular clinical importance to plastic surgeons, with tendon-specific treatment guided by the general principle of minimum protective immobilization followed by hand therapy to overcome potential adhesions. Thorough knowledge of the underlying biomechanical principles of tendon healing is required to provide optimal care to patients presenting with tendon injury.
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Hayashi, M., C. Zhao, K. N. An i P. C. Amadio. "The effects of growth and differentiation factor 5 on bone marrow stromal cell transplants in an in vitro tendon healing model". Journal of Hand Surgery (European Volume) 36, nr 4 (maj 2011): 271–79. http://dx.doi.org/10.1177/1753193410394521.

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The effects of growth differentiation factor-5 (GDF-5) and bone marrow stromal cells (BMSCs) on tendon healing were investigated under in vitro tissue culture conditions. BMSCs and GDF-5 placed in a collagen gel were interpositioned between the cut ends of dog flexor digitorum profundus tendons. The tendons were randomly assigned into four groups: 1) repaired tendon without gel; 2) repaired tendon with BMSC-seeded gel; 3) repaired tendon with GDF-5 gel without cells; and 4) repaired tendon with GDF-5 treated BMSC-seeded gel. At 2 and 4 weeks, the maximal strength of repaired tendons with GDF-5 treated BMSCs-seeded gel was significantly higher than in tendons without gel interposition. However, neither BMSCs nor GDF-5 alone significantly increased the maximal strength of healing tendons at 2 or 4 weeks. These results suggest that the combination of BMSCs and GDF-5 accelerates tendon healing, but either BMSCs or GDF-5 alone are not effective in this model.
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Müller, Sebastian A., Nicholas P. Quirk, Julia A. Müller-Lebschi, Patricia E. Heisterbach, Lutz Dürselen, Martin Majewski i Christopher H. Evans. "Response of the Injured Tendon to Growth Factors in the Presence or Absence of the Paratenon". American Journal of Sports Medicine 47, nr 2 (14.12.2018): 462–67. http://dx.doi.org/10.1177/0363546518814534.

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Background: The paratenon is important for Achilles tendon healing. There is much interest in the use of exogenous growth factors (GFs) as potential agents for accelerating the healing of damaged Achilles tendons. Purpose/Hypothesis: The present study used a rat model to study the responses of the injured Achilles tendon to GFs in the presence or absence of the paratenon. The hypothesis was that responses of the injured tendon to GFs would be lower in the absence of a paratenon. Study Design: Controlled laboratory study. Methods: A 4-mm defect was created in the right Achilles tendon of 60 skeletally mature rats, which were treated with a validated combination of GFs (bFGF, BMP-12, and TGF-β1). Animals were randomly assigned to the intact paratenon (IP) group or resected paratenon (RP) group. Healing was studied anatomically, mechanically, and histologically after 1, 2, and 4 weeks. Results: IP tendons showed improved healing compared with RP tendons. IP tendons were significantly stronger (32.2 N and 48.9 N, respectively) than RP tendons (20.1 N and 31.1 N, respectively) after 1 and 2 weeks. IP tendons did not elongate as much as RP tendons and had greater cross-sectional areas (18.0 mm2, 14.4 mm2, and 16.4 mm2, respectively) after 1, 2, and 4 weeks compared with RP tendons (10.5 mm2, 8.4 mm2, and 11.9 mm2, respectively). On histology, earlier collagen deposition and parallel orientation of fibrils were found for IP tendons. Conclusion: The paratenon is essential for efficient Achilles tendon healing. Healing with GFs in this Achilles tendon defect model was superior in the presence of the paratenon. Clinical Relevance: Biological approaches to tendon engineering using GFs are in vogue and have been shown to improve healing of the rat Achilles tendon, most likely by inducing progenitor cells located within the paratenon. Clinically, resection or incision of the paratenon has been proposed for wound closure. Our data demonstrate the fundamental importance of the paratenon, which therefore should be preserved during Achilles tendon repair, especially if augmented with products such as platelet-rich plasma or autologous conditioned serum that are rich in GFs.
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Freedman, Benjamin R., Ashley B. Rodriguez, Cody D. Hillin, Stephanie N. Weiss, Biao Han, Lin Han i Louis J. Soslowsky. "Tendon healing affects the multiscale mechanical, structural and compositional response of tendon to quasi-static tensile loading". Journal of The Royal Society Interface 15, nr 139 (luty 2018): 20170880. http://dx.doi.org/10.1098/rsif.2017.0880.

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Tendon experiences a variety of multiscale changes to its extracellular matrix during mechanical loading at the fascicle, fibre and fibril levels. For example, tensile loading of tendon increases its stiffness, with organization of collagen fibres, and increases cell strain in the direction of loading. Although applied macroscale strains correlate to cell and nuclear strains in uninjured tendon, the multiscale response during tendon healing remains unknown and may affect cell mechanosensing and response. Therefore, this study evaluated multiscale structure–function mechanisms in response to quasi-static tensile loading in uninjured and healing tendons. We found that tendon healing affected the macroscale mechanical and structural response to mechanical loading, evidenced by decreases in strain stiffening and collagen fibre realignment. At the micro- and nanoscales, healing resulted in increased collagen fibre disorganization, nuclear disorganization, decreased change in nuclear aspect ratio with loading, and decreased indentation modulus compared to uninjured tendons. Taken together, this work supports a new concept of nuclear strain transfer attenuation during tendon healing and identifies several multiscale properties that may contribute. Our work also provides benchmarks for the biomechanical microenvironments that tendon cells may experience following cell delivery therapies.
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Yu, Tung-Yang, Jong-Hwei S. Pang, Li-Ping Lin, Ju-Wen Cheng, Shih-Jung Liu i Wen-Chung Tsai. "Platelet-Rich Plasma Releasate Promotes Early Healing in Tendon After Acute Injury". Orthopaedic Journal of Sports Medicine 9, nr 4 (1.04.2021): 232596712199037. http://dx.doi.org/10.1177/2325967121990377.

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Background: Acute tendon injury can limit motion and thereby inhibit tendon healing. Positive results have been found after the use of platelet-rich plasma (PRP) to treat tendon injury; however, the early effects of PRP on tendon regeneration are not known. Purpose/Hypothesis: The purpose of this study was to evaluate the effects of PRP releasate (PRPr) on the early stages of tendon healing in a rat partial tenotomy model. It was hypothesized that PRPr can promote early healing of an Achilles tendon in rats. Study Design: Controlled laboratory study. Methods: PRP was prepared by a 2-step method of manual platelet concentration from 10 rats. PRPr was isolated from the clotted preparation after activation by thrombin and was applied to an Achilles tendon on 1 side of 30 rats on the second day after partial tenotomy, with normal saline used as the control on the other side. Achilles tendon samples were harvested 5 and 10 days after tenotomy. At each time point, 15 Achilles tendon samples were obtained, of which 5 samples were evaluated by Masson trichrome staining, apoptosis, and cell proliferation, while the other 10 samples were tested for tensile strength using a material testing machine. Results: Compared with saline-treated control tendons, the PRPr-treated tendons showed increased collagen synthesis near the cut edge and fewer apoptotic cells ( P = .01). An immunohistochemical analysis revealed more Ki-67–positive cells but fewer cluster of differentiation (CD) 68+ (ED1+) macrophages in PRPr tendons compared with saline-treated tendons ( P < .01). Tendons treated with PRPr also showed higher ultimate tensile strength than those treated with saline ( P = .03). Conclusion: PRPr treatment promotes tissue recovery in the early phase of tendon healing by stimulating tendon cell proliferation and collagen production while inhibiting cell apoptosis and CD68+ (ED1+) macrophage infiltration. Clinical Relevance: These findings suggest that with PRPr treatment, higher loads can be applied to the healing tendon at an earlier time, which can help the patient resume activity earlier.
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Rozprawy doktorskie na temat "Tendon healing"

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Blomgran, Parmis. "Inflammation and tendon healing". Doctoral thesis, Linköpings universitet, Avdelningen för Kirurgi, Ortopedi och Onkologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-142349.

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Tendons heal through three different overlapping phases; the inflammatory, proliferative and remodeling phase. Many studies have investigated what factors influence healing of tendons. However, little was known about inflammation and the immune cells present during Achilles tendon healing by the time this thesis started. We developed a flow cytometry method for our rat model of tendon healing, which enabled us to study different leukocyte subpopulations during Achilles tendon healing. The general aim of this thesis was to understand more about inflammation and the immune cell populations present during tendon healing and how the immune cell composition changes during normal tendon healing. Moreover, we investigated how different factors that are known to influence tendon healing affected the composition of the immune cell population. First, we described the immune cells during the time course of tendon healing focusing on different subpopulations of macrophages and T cells. Then, we studied how these cells were influenced by reduced mechanical loading. Mechanical loading prolonged the presence of M1 macrophages and delayed the switch to regulatory T cells and M2 macrophages compared to reduced mechanical loading. Next, the effect of nonsteroidal anti-inflammatory drugs (NSAIDs) on the leukocyte composition revealed that, even though NSAIDs influence the mechanical properties of healing tendon, this effect was not mediated via changes in the leukocyte sub-populations during early and mid-time tendon healing. Further, the effect of corticosteroids during the inflammatory and remodeling phases of tendon healing was an improved healing of tendons and a reduction of CD8a T cells when corticosteroid was administered after the inflammatory phase. Lastly, we investigated if impairment of tendon healing by NSAIDs was related to mechanotransduction or microdamage during mechanical loading and showed that NSAIDs impair tendon healing by reducing the response to microdamage. In conclusion, these studies show that inflammation plays an important role during Achilles tendon healing, and factors that influence healing can also alter the presence or polarization of immune cell populations.
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Murrell, George Anthony Calvert St George Clinical School UNSW. "Nitric oxide and tendon healing". Awarded by:University of New South Wales. St George Clinical School, 2006. http://handle.unsw.edu.au/1959.4/31887.

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Nitric oxide is a small free radical generated by family of enzymes, the nitric oxide synthases. In a series of experiments performed over the last 15 years we showed that nitric oxide is induced by all three isoforms of nitric oxide synthase during tendon healing and that it plays a crucial beneficial role in restoring tendon function. In normal tendon we found very little nitric oxide synthase activity while in injured rat and human tendons nitric oxide synthase activity was expressed in healing fibroblasts in a temporal fashion. In healing rat Achilles tendon fibroblasts the first isoform to be expressed was endothelial nitric oxide synthase (eNOS), followed by inducible nitric oxide synthase (iNOS), and then brain or neuronal nitric oxide synthase (bNOS). Systemic inhibition of nitric oxide synthase activity decreased the cross sectional area and mechanical properties of the healing rodent Achilles tendons. Addition of nitric oxide via NO-flurbiprofen or NO-paracetamol enhanced rat Achilles tendon healing. Addition of nitric oxide to cultured human tendon cells via chemical means and via adenoviral transfection enhanced collagen synthesis, suggesting that one mechanism for the beneficial of nitric oxide on tendon healing might be via matrix synthesis. The final part of the work involved three randomized, double-blind clinical trials which evaluated the efficacy of nitric oxide donation via a patch in the management of the tendinopathy. In all three clinical trials there was a significant positive beneficial effect of nitric oxide donation to the clinical symptoms and function of patients with Achilles tendinopathy, tennis elbow and Achilles tendonitis.
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Molloy, Timothy John St George Clinical School UNSW. "Gene expression in healing tendon". Awarded by:University of New South Wales. St George Clinical School, 2006. http://handle.unsw.edu.au/1959.4/23939.

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Tendon injury is painful and often debilitating, and is a one of the most prevalent soft tissue injuries encountered in the clinic. While common, the underlying molecular and genetic processes of tendon damage and repair remain poorly understood. The work described herein used genome-wide expression analyses to investigate tendon injury and healing from three perspectives. The first identified novel gene expression in tendon fibroblasts following their stimulation with nitric oxide (NO). Of particular relevance to tendon healing was the observation that stimulated fibroblasts express a number of extracellular matrix (ECM) genes in response to NO in a dose-dependent manner, and that NO significantly affects cellular adhesion, a critical process during tendon repair. These findings will be of use when optimising dosages of NO delivery in future work investigating NO as potential treatment for tendon injuries. The second study examined gene expression in an acute tendon injury model in the rat at 1, 7, and 21 days post injury, roughly representing the inflammation, proliferation, and remodelling phase of wound repair. Several novel genes and pathways were found to be differentially expressed at each stage of healing. Of particular interest were the presence of a significant number of genes related to glutamate signaling, a method of cellular communication that has not previously been shown to exist in tendon. Also upregulated were a number of genes which have previously only been associated with embryonic development. Finally, gene expression in a supraspinatus tendinopathy model in the rat was investigated. Several genetic pathways were identified in tendinopathic tendons which have not previously been associated with the disease, and, analogous to the acute injury model study, glutamate signaling gene overexpression was also prevalent. Further in vitro studies showed that the expression of these genes in tendon fibroblasts were stimulated by glutamate treatment, which in turn upregulated pro-apoptotic pathways causing cell death. This may prove to be an important causative factor in the tendon degeneration seen in tendinopathy, in which apoptosis has been identified as playing a significant role. The results of these studies contribute to a better understanding of the aetiology of several extremely common pathologies of this soft tissue, and may help to develop more targeted therapies for increasing the efficacy of tendon healing in future.
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Berglund, Maria. "Biomolecular Aspects of Flexor Tendon Healing". Doctoral thesis, Uppsala universitet, Handkirurgi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-120304.

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Flexor tendon injuries in zone II of the hand (i.e. between the distal volar crease and the distal interphalangeal joint) can be costly for both the afflicted individual and society because of the high cost of a long rehabilitation period, complicated by tendon ruptures or scarring with adhesion formation, causing impaired range of motion. The aim of the present thesis was to characterize more fully the deep flexor tendon, the tendon sheath and their response to injury in a rabbit model in order to find potential targets to improve the outcome of repair. The intrasynovial rabbit deep flexor tendon differed from the extrasynovial peroneus tendon in the expression of collagens and transforming growth factor-β1 gene expression. Differences were also found in collagen III and proteoglycans between regions of the flexor tendon subjected to either compressive or tensile load. After laceration and subsequent repair of the flexor tendon, a shift in collagen gene expression from type I to type III occurred. Proteoglycans were generally increased with the notable exception of decorin, a potential inhibitor of the profibrotic transforming growth factor-β1 which was markedly increased during the first two weeks after repair in tendon tissue but remained unaltered in the sheaths. Both vascular endothelial growth factor and basic fibroblast growth factor mRNA levels remained essentially unaltered, whereas insulin-like growth factor-1 increased later in the healing process, suggesting potential beneficial effects of exogenous addition, increasing tendon strength through stimulating tenocyte proliferation and collagen synthesis. Matrix metalloproteinase-13 mRNA levels increased and remained high in both tendon and sheath, whereas there was only a transient increase of matrix metalloproteinase-3 mRNA in tendon. We could also demonstrate a significant increase of the proportion of myofibroblasts, mast cells and neuropeptide containing nerve fibers in the healing tendon tissue, all components of the profibrotic myofibroblast-mast cell-neuropeptide pathway.
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Schepull, Thorsten. "Stiffness of the healing human Achilles tendon". Doctoral thesis, Linköpings universitet, Institutionen för klinisk och experimentell medicin, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-91727.

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Achilles tendon ruptures in humans are followed by a long period of immobilisation, rehabilitation and limitations of physical activity and sometimes work also. This prolonged period probably leaves a marginal for improvement in the management of this injury. Animal studies have shown that there are several possibilities to influence and improve tendon healing. The aim of this thesis was to find a way to examine the mechanical properties of the healing human Achilles tendon and, by using that method, to gain a better understanding of the tissue properties and healing process in these tendons. It was also our aim to use our knowledge from animal studies in an attempt to improve tendon healing in humans. We developed a radiological method using radiostereophotogrammetric analysis (RSA) and computed tomography (CT), which enabled us to measure the stiffness of the healing Achilles tendon. The results of these measurements, as early as 7 weeks after injury correlated with the late clinical results in all studies. Clinical results were measured using a heel-raise test comparing the injured with the non-injured tendon. We could not find a significant difference in stiffness between patients treated surgically or non-surgically. Neither could we demonstrate that platelet-rich plasma improved the mechanical properties of the healing tendon or the clinical outcome, within the limits of the statistical power. In contrast, patients following a specific training programme with early tension loading of the tendon twice a day developed stiffer tendon tissue later in the healing process. Since RSA is unsuitable for routine clinical use, we evaluated the possibility to use radiodensity findings from CT as a proxy for healing and its correlation to mechanical and clinical results. Density and mechanical properties correlated strongly when analysing all time points together, but only weakly at each particular point in time. Density may still be useful in describing mechanical properties at a later stage of repair, but this remains to be seen. In conclusion, these studies show that early mechanical properties correlate to late clinical outcome and that the early use of daily tension loading sessions leads to an improvement in the mechanical properties of the tendon tissue.
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Brooks, Jonathan Peter. "The biology of the tendon in development and healing". Thesis, University of Manchester, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.488287.

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The biology of tendons has been extensively investigated previously. Many studies have looked into the development and healing of tendons. An overall picture of the events in tendon development and healing, in terms of recently recognised functional molecules is missing, particularly in a single model. The recent understanding of the importance of metalloproteinases and other enzymes in ECM degradation suggests they may be of importance in tendon healing and development. Another unexplored area with relevance to wound healing and development is the functional integrin molecules. This thesis looked at the spatial and temporal localisation of a wide range of functional molecules and at cell proliferation. The subject groups were tendon development, from the limb bud to juvenile animal, and a defined reproducible adult healing model, with time points to six months. The study was then extended to fetal tendon healing. The results were analysed qualitatively using immunohistology and the findings were supported by histological and ultrastructural analysis, which reproduced the findings of previous studies and allowed novel observations. Adult tendon healing was accessible to quantitative analysis, which added further weight to the results. Tendon wounds in the adult model were noted to be asymmetrical in terms of tissue fibrillation, and immunohistology on opposite sides of the wound. Observations of integrin and cytoskeletal patterning in the wound, suggested mechanical load may have modulated this effect. A further study looked at a defined set of wound models. These modified the mechanical load and tissue perfusion of the wound to reproduce the effects in symmetrical wounds. The results showed that the spatial and temporal localisation of collagens, growth factors, enzymes and integrins and cell proliferation were modulated over a six month healing period. This was supported by quantitative analysis. Similar, but different effects were noted in development and fetal healing. Collagen types had a similar pattern in the three models, although the results demonstrated profound differences in timing and events surrounding new matrix production. The results of the wound modulation study using a cell proliferation marker, immunohistology, Western blots and zymograms of these wounds, were analysed and found to show marked differences in morphology, enzyme production and cell proliferation. The results excluded growth factors and tissue perfusion as the cause when these factors were controlled for. Significant (p
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Stevenson, John Howard. "An investigation into the effect of ultrasound on repaired tendons". Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335315.

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Harrison, R. "The elucidation and strategic modification of flexor tendon healing mechanisms". Thesis, University College London (University of London), 2005. http://discovery.ucl.ac.uk/1445576/.

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Introduction and aims: 16,000 flexor tendon repair operations are performed annually in the UK these are frequently complicated by adhesions, causing significant morbidity. The uncertainty over which cells are responsible for the tendon healing mechanisms persists. We aimed to develop a novel experimental model to elucidate the possible role and migratory response of synovial sheath fibroblasts during tendon healing and to determine the effects of differing environment and stimuli on the cellular processes in the healing tendon. 0.1.2 Materials and methods: Rat synovial sheath cells were labelled with a lipophilic tracer dye, and an injury made in the adjacent flexor tendon. Tendons were then harvested at 1,3,5 and 7 days and frozen sectioned. The location of the labelled fibroblasts was determined using ultra-violet microscopy. In a separate experimental series, rat flexor tendons received different injury type / mobilisation or immobilisation / and TGF-pl or saline control application. Tendons were harvested at 7 and 14 days, fixed, sectioned and HandE stained. Cell densities in the injury region were determined. 0.1.3 Results: By 24 hours, labelled synovial fibroblasts were observed to have migrated from the sheath to the zone of injury, with numbers increasing at 3 and 5 days, but diminishing by day 7. In the second experimental model, tendons injured with a superficial scrape, or immobilised post-injury showed a significant increase in relative cellularity in the region of the injury. 0.1.4 Discussion: These results suggest that synovial cells are involved in the early stages of tendon healing, and migrate from the synovial sheath into the healing tendon. Fibroblast density, in the rat model, is modulated by type of injury, immobilisation, but not TGF-p 1 application. This may correlate clinically with collagen deposition and adhesion formation.
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Virchenko, Olena. "Stimulation of tendon repair by platelet concentrate, CDMP-2 and mechanical loading in animal models". Doctoral thesis, Linköping : Univ, 2007. http://www.bibl.liu.se/liupubl/disp/disp2007/med1005s.pdf.

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Kinneberg, Kirsten R. C. "Tissue Engineering Strategies to Improve Tendon Healing and Insertion Site Integration". University of Cincinnati / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1307106075.

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Książki na temat "Tendon healing"

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Society for the Study of Native Arts and Sciences., red. Tendon and ligament healing: A new approach through manual therapy. Berkeley, Calif: North Atlantic Books, 1999.

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Barber, Victoria Lynette. An evaluation of tendon healing using magnetic resonance imaging and a standard physical assessment. [New Haven: s.n.], 1990.

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Kusler, Ruth Weil. Tender hands: Ruth's story of healing. Fargo: Germans from Russia Heritage Collection, North Dakota State University Libraries, 1998.

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The tender touch of God. Eugene, Or: Harvest House Publishers, 1996.

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Tending inner gardens: The healing art of feminist psychotherapy. New York: Haworth Press, 1995.

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Healing the ravaged soul: Tending the spiritual wounds of child sexual abuse. U.K: Lutterworth Press, 2016.

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Barry, Parker. Memorial: A ministry of healing. Chattanooga, TN: Parker Hood Press, 1997.

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Salter, Robert Bruce. Continuous passive motion (CPM): A biological concept for the healing and regeneration of articular cartilage, ligaments, and tendons : from origination to research to clinical applications. Baltimore: Williams & Wilkins, 1993.

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Harrison, Mark. Wound healing. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198765875.003.0057.

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This chapter describes the pathology of wound healing as it applies to Emergency Medicine, and in particular the Primary FRCEM examination. The chapter outlines the key details of haemostasis, inflammation, reconstruction, epithelialization, and maturation, and the specific tissues affected, including skin, tendon, peripheral nerve, bone, myocardium, and brain. This chapter is laid out exactly following the RCEM syllabus, to allow easy reference and consolidation of learning.
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Weintraub, William. Tendon and Ligament Healing. A new approach through manual therapy. Thieme, Stuttgart, 2001.

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Części książek na temat "Tendon healing"

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Pauyo, Thierry, Elmar Herbst i Freddie H. Fu. "Tendon Healing". W Muscle and Tendon Injuries, 45–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-54184-5_4.

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Löhr, J. F., Hirotaka Sano i H. K. Uhthoff. "Rotator Cuff Tendon Healing". W Schulterinstabilität — Rotatorenmanschette, 202–5. Heidelberg: Steinkopff, 1999. http://dx.doi.org/10.1007/978-3-642-58711-5_18.

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Oliva, Francesco, Stefano Gatti, Giuseppe Porcellini, Nicholas R. Forsyth i Nicola Maffulli. "Growth Factors and Tendon Healing". W Rotator Cuff Tear, 53–64. Basel: KARGER, 2011. http://dx.doi.org/10.1159/000328878.

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Bisciotti, Gian Nicola, i Piero Volpi. "Healing Processes of the Tendon". W The Lower Limb Tendinopathies, 21–40. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33234-5_2.

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Volpi, Piero, i Gian Nicola Bisciotti. "Healing Processes in Tendon Tissue". W Muscle Injury in the Athlete, 53–72. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-16158-3_3.

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Ackermann, Paul W. "Biologics in Tendon Healing: PRP/Fibrin/Stem Cells". W The Achilles Tendon, 135–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-54074-9_23.

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Vogel, Laura A., Andreas Voss i Augustus D. Mazzocca. "Healing of the Rotator Cuff Tendon". W Rotator Cuff Across the Life Span, 19–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-58729-4_3.

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Ahmed, Aisha Siddiqah. "Does Diabetes Mellitus Affect Tendon Healing?" W Metabolic Influences on Risk for Tendon Disorders, 179–84. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33943-6_16.

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Domeij-Arverud, Erica, i Paul W. Ackermann. "Deep Venous Thrombosis and Tendon Healing". W Metabolic Influences on Risk for Tendon Disorders, 221–28. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33943-6_21.

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Akintunde, Akinjide R., Daniele E. Schiavazzi i Kristin S. Miller. "Mathematical Model of Age-Specific Tendon Healing". W Lecture Notes in Computational Vision and Biomechanics, 288–96. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43195-2_23.

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Streszczenia konferencji na temat "Tendon healing"

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Carvalho, P. T. C., Cheila O. C. Batista i C. Fabíola. "Low level laser therapy in healing tendon". W Laser Florence 2004, redaktorzy Leonardo Longo, Khalil A. Khatri, Mihail-Lucian Pascu i Wilhelm R. Waidelich. SPIE, 2005. http://dx.doi.org/10.1117/12.660046.

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Gilday, Steven D., Chris Casstevens, Jason T. Shearn i David L. Butler. "Analysis of Regional Strain Patterns Following Surgical Disruption of the Enthesis in a Murine Model of Patellar Tendon Injury". W ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14569.

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Tendon injuries are common yet often fail to heal naturally, especially in cases in which the native tendon-to-bone insertion site is disrupted. Surgical tendon repairs are often limited by the inability of the ruptured tendon to functionally attach back to the underlying bone. For patients with tendon injuries, poor tendon-to-bone integration prolongs recovery time and increases the risk of re-rupture. Improvements in tendon repair will require a more complete understanding of both the biological and mechanical phenomena that occur during natural tendon-to-bone healing. Mechanical studies of tendon repair often utilize larger animal models such as rabbits or canines, but these animals lack many of the genetic and biological tools that are available in the mouse. Thus, the objective of this study was to analyze the biomechanical outcomes of natural tendon-to-bone healing following surgical disruption of the enthesis in a murine model of patellar tendon injury. In particular, this study attempted to define the regional (insertion site versus midsubstance) strain patterns present in normal tendon and compare these patterns to those seen at various stages of healing following a central-third patellar tendon avulsion injury. We hypothesized that 1) murine patellar tendon avulsions would exhibit inferior structural properties compared to contralateral shams and normal controls and 2) insertional strains would greatly exceed midsubstance strains in the healing tendons, resulting in failure initiation at the tendon-bone junction.
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Riggin, Corinne N., Joseph J. Sarver, Benjamin R. Freedman, Stephen J. Thomas i Louis J. Soslowsky. "Analysis of Collagen Fiber Organization in Mouse Achilles Tendon Using High-Frequency Ultrasound Imaging". W ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14472.

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Achilles tendon ruptures are traumatic injuries that frequently occur in active individuals and result in significant medical expense. Common techniques for assessing outcomes of surgical repair and rehabilitation rely heavily on patient-based measures of pain and function. While these measures can provide evidence for recovery of functional performance, they do not directly assess tendon healing which, if insufficient, can lead to re-rupture. The clinical evaluation of collagen organization following Achilles tendon injury may provide a more accurate measure of healing than traditional, functional performance tests. It has been shown that changes in collagen organization precede and correlate with changes in mechanical properties in tendons [1–3] and that load and injury effect collagen organization [4–6]. Ultimately, if collagen organization could be quantified in vivo, it would represent a powerful, diagnostic tool to measure the progression of tendon healing, as well as to monitor damage accumulation due to injury.
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Dyment, Nathaniel A., Namdar Kazemi, Lindsey E. Aschbacher-Smith, Nicolas J. Barthelery, Keith Kenter, Cynthia Gooch, Jason T. Shearn, Christopher Wylie i David L. Butler. "The Relationships Among Spatiotemporal Gene Expression, Histology, and Biomechanics Following Full-Length Injury in the Murine Patellar Tendon". W ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53622.

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Tendon and ligament injuries present a considerable socioeconomic impact as close to 50% of the 32 million musculoskeletal injuries in the US per year include these structures [1]. The inadequate healing in these tissues requires novel treatment modalities. Improving tendon tissue engineering dictates that we better understand the process of natural adult tendon healing. Type-I (Col1) and Type-II (Col2) collagens are important structural proteins in tendon as Col1 is the main collagen type found in the tendon midsubstance while Col2 is expressed at the insertion into bone during development, growth, and healing [2–3]. Expression of Col1 and Col2 has typically been analyzed via qPCR, western blotting, and immunohistochemistry (IHC) during healing. However, the temporal expression of these genes is still poorly understood on a cell-by-cell basis. Our lab has previously studied patellar tendon (PT) healing in NZW rabbits [4]. While the NZW rabbit allows for controlled injuries and accurate biomechanical assessment of healing, it lacks the genetic power that is offered in the mouse. Therefore, pOBCo13.6GFPtpz (Col1) and pCol2ECFP (Col2) double transgenic (DT) reporter mice were created to track spatiotemporal gene expression. Thus, the objectives of this study were to monitor changes in: 1) spatiotemporal Col1 and Col2 gene expression patterns, 2) tissue morphology, and 3) healing biomechanics following a full-length, central PT injury in Col1/Col2 DT mice and to compare these natural healing results to contralateral surgical shams and normal PT in age-matched controls.
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Abramowitch, Steven D., Matthew B. Fisher, Sinan Karaoglu i Savio L. Y. Woo. "The Mechanical and Viscoelastic Properties of the Healing Rabbit Patellar Tendon". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176183.

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Central third bone-patellar tendon-bone (BPTB) autografts are commonly used for anterior cruciate ligament (ACL) reconstructions. Following surgery, complications arise at the donor site, including extension deficits and anterior knee pain [1]. These complications are partially caused by inadequate healing of the patellar tendon (PT) as well as adhesions in the anterior interval. Recent clinical data have suggested these are contributing factors in the early development of osteoarthrosis following ACL reconstruction [2]. Thus, it is necessary to understand the changes in mechanical and viscoelastic behavior in the healing PT.
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Ansorge, H. L., J. E. Hsu, L. Edelstein, D. E. Birk i L. J. Soslowsky. "Injury During Early Neonatal Development Leads to a Faster Repair Response When Compared to Later Injury in a Mouse Achilles Tendon". W ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19232.

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During neonatal development, tendons undergo a well orchestrated process whereby extensive structural and compositional changes occur in synchrony to produce a normal tissue [1,2]. Similarly, during the repair response to injury, structural and compositional changes occur, but in this case, a mechanically inferior tendon is produced. As a result, the process of development has been postulated as a potential paradigm through which improved tissue healing may occur. By examining injury at distinctly different stages of development, we will obtain vital information into the structure-function relationships in tendon. Although the mouse is an intriguing model system due to the availability of assays and genetically altered animals, due to the small size and fragile nature of neonatal tendons, neonatal tendon injury has not been evaluated. Therefore, the objective of this study is to evaluate the differential healing response in neonatal tendon at two distinct stages of development. We hypothesized that when normalized, maximum stress and modulus will be significantly higher in early neonatal injury when compared to later neonatal injury. Also, when normalized, maximum stress, modulus and percent relaxation will be significantly increased over time in early neonatal injury but will remain low in later neonatal injury.
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Thomopoulos, S., R. Das, H. M. Kim, D. Zeltser, K. Kousari i L. Galatz. "The Role of the Loading Environment on the Developing Tendon-to-Bone Insertion". W ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206837.

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A transition zone forms at the attachment of tendon to bone during post-natal development [1]. We previously showed that this transitional tissue reduces stress concentrations at the insertion and hence reduces the risk for failure [2]. During tendon-to-bone healing, on the other hand, a transition zone does not develop at the interface [3]. Unlike development, inferior scar tissue fills the repair site and the repair is prone to rupture at the attachment. Understanding the development of the insertion will allow us to develop solutions to augment tendon-to-bone healing.
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Komolafe, Oluseeni A., i Todd C. Doehring. "Nonlinear Elastic Behavior of Achilles Tendon at the Fascicle Scale". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176880.

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Parallel collagen fibers such as ligaments and tendons are composed of fiber bundles, or fascicles, enclosed in a sheath of reticular membrane. In the Achilles tendon, these fascicles can be long, extending from the gastro-soleus unit to the calcaneal insertion site (Fig. 1). Although the overall functional behavior of the whole tendon is well established[1], there is little information detailing properties of individual fascicles or their interactions. Knowledge of the structural and biomechanical properties at the “mesostructural” scale (i.e. fascicle-scale) is critical to understanding tissue pathologies; in particular the processes involved in injury and healing, and the development of improved computational models and functional tissue engineered constructs.
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Chokalingam, Kumar, Chris Frede, Jane L. Florer, Richard J. Wenstrup i David L. Butler. "Col1 and Col2 Gene Expression During the Development of Murine Knee Joints". W ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192596.

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Recently, tissue engineers have turned to developmental biology to try and improve the repair of tendons and ligaments [1]. The premise is that understanding normal growth and development of tendons and ligaments might facilitate in later repair. Understanding these similarities in extracellular matrix gene expression and growth factor expression might allow tissue engineers to develop treatments to augment adult tendon and ligament healing.
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Thomopoulos, Stavros, Alistair Kent, Victor Birman, Rosalina Das, Brigitte Wopenka, Jill Pasteris i Guy Genin. "Mineral Composition at the Tendon-to-Bone Insertion and Its Role in Stress Transfer". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176387.

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Musculoskeletal injuries are a common cause of pain and disability, and result in significant health care costs. Rotator cuff injuries, which make up the majority of soft tissue injuries to the upper extremity, commonly require surgical repair to the humeral head. At the rotator cuff, for example, the recurrence of tears to surgically repaired tendons has been reported to be as high as 94% [1]. The most dramatic feature of the failed tendon-to-bone healing scenarios is the lack of a transition zone between the tendon and bone that existed before the injury [2].
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