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Frittoli, Emanuela, Andrea Palamidessi, Alessandro Pizzigoni, Letizia Lanzetti, Massimiliano Garrè, Flavia Troglio, Albino Troilo i in. "The Primate-specific Protein TBC1D3 Is Required for Optimal Macropinocytosis in a Novel ARF6-dependent Pathway". Molecular Biology of the Cell 19, nr 4 (kwiecień 2008): 1304–16. http://dx.doi.org/10.1091/mbc.e07-06-0594.
Pełny tekst źródłaTobias, Irene S., Kara K. Lazauskas, Jeremy Siu, Pablo B. Costa, Jared W. Coburn i Andrew J. Galpin. "Sex and fiber type independently influence AMPK, TBC1D1, and TBC1D4 at rest and during recovery from high-intensity exercise in humans". Journal of Applied Physiology 128, nr 2 (1.02.2020): 350–61. http://dx.doi.org/10.1152/japplphysiol.00704.2019.
Pełny tekst źródłaPenisson, Maxime, Mingyue Jin, Shengming Wang, Shinji Hirotsune, Fiona Francis i Richard Belvindrah. "Lis1 mutation prevents basal radial glia-like cell production in the mouse". Human Molecular Genetics 31, nr 6 (12.10.2021): 942–57. http://dx.doi.org/10.1093/hmg/ddab295.
Pełny tekst źródłaEspelage, Lena, Hadi Al-Hasani i Alexandra Chadt. "RabGAPs in skeletal muscle function and exercise". Journal of Molecular Endocrinology 64, nr 1 (styczeń 2020): R1—R19. http://dx.doi.org/10.1530/jme-19-0143.
Pełny tekst źródłaMikłosz, Agnieszka, Bartłomiej Łukaszuk, Elżbieta Supruniuk, Kamil Grubczak, Marcin Moniuszko, Barbara Choromańska, Piotr Myśliwiec i Adrian Chabowski. "Does TBC1D4 (AS160) or TBC1D1 Deficiency Affect the Expression of Fatty Acid Handling Proteins in the Adipocytes Differentiated from Human Adipose-Derived Mesenchymal Stem Cells (ADMSCs) Obtained from Subcutaneous and Visceral Fat Depots?" Cells 10, nr 6 (16.06.2021): 1515. http://dx.doi.org/10.3390/cells10061515.
Pełny tekst źródłaZhou, Qiong L., Zhen Y. Jiang, John Holik, Anil Chawla, G. Nana Hagan, John Leszyk i Michael P. Czech. "Akt substrate TBC1D1 regulates GLUT1 expression through the mTOR pathway in 3T3-L1 adipocytes". Biochemical Journal 411, nr 3 (14.04.2008): 647–55. http://dx.doi.org/10.1042/bj20071084.
Pełny tekst źródłaMafakheri, Samaneh, Alexandra Chadt i Hadi Al-Hasani. "Regulation of RabGAPs involved in insulin action". Biochemical Society Transactions 46, nr 3 (21.05.2018): 683–90. http://dx.doi.org/10.1042/bst20170479.
Pełny tekst źródłaQin, Shu, Robert A. Dorschner, Irene Masini, Ophelia Lavoie‐Gagne, Philip D. Stahl, Todd W. Costantini, Andrew Baird i Brian P. Eliceiri. "TBC1D3 regulates the payload and biological activity of extracellular vesicles that mediate tissue repair". FASEB Journal 33, nr 5 (4.02.2019): 6129–39. http://dx.doi.org/10.1096/fj.201802388r.
Pełny tekst źródłaShen, Y., L. Zhang, H. Zhao i C. L. Shen. "TC-1 mediate the TBC1D3 oncogene induced migration of MCF-7 breast cancer cells". Annals of Oncology 29 (listopad 2018): ix19. http://dx.doi.org/10.1093/annonc/mdy428.017.
Pełny tekst źródłaKong, Chen, Jeffrey J. Lange, Dmitri Samovski, Xiong Su, Jialiu Liu, Sinju Sundaresan i Philip D. Stahl. "Ubiquitination and degradation of the hominoid-specific oncoprotein TBC1D3 is regulated by protein palmitoylation". Biochemical and Biophysical Research Communications 434, nr 2 (maj 2013): 388–93. http://dx.doi.org/10.1016/j.bbrc.2013.04.001.
Pełny tekst źródłaZhang, Pei, Lei Zhu i Xiaodong Pan. "A comprehensive analysis of the oncogenic and prognostic role of TBC1Ds in human hepatocellular carcinoma". PeerJ 12 (14.05.2024): e17362. http://dx.doi.org/10.7717/peerj.17362.
Pełny tekst źródłaRoach, William G., Jose A. Chavez, Cristinel P. Mîinea i Gustav E. Lienhard. "Substrate specificity and effect on GLUT4 translocation of the Rab GTPase-activating protein Tbc1d1". Biochemical Journal 403, nr 2 (26.03.2007): 353–58. http://dx.doi.org/10.1042/bj20061798.
Pełny tekst źródłaHodzic, Didier, Chen Kong, Marisa J. Wainszelbaum, Audra J. Charron, Xiong Su i Philip D. Stahl. "TBC1D3, a hominoid oncoprotein, is encoded by a cluster of paralogues located on chromosome 17q12". Genomics 88, nr 6 (grudzień 2006): 731–36. http://dx.doi.org/10.1016/j.ygeno.2006.05.009.
Pełny tekst źródłaKong, Chen, Dmitri Samovski, Priya Srikanth, Marisa J. Wainszelbaum, Audra J. Charron, Jialiu Liu, Jeffrey J. Lange i in. "Ubiquitination and Degradation of the Hominoid-Specific Oncoprotein TBC1D3 Is Mediated by CUL7 E3 Ligase". PLoS ONE 7, nr 9 (27.09.2012): e46485. http://dx.doi.org/10.1371/journal.pone.0046485.
Pełny tekst źródłaHe, Ze, Tian Tian, Dan Guo, Huijuan Wu, Yang Chen, Yongchen Zhang, Qing Wan i in. "Cytoplasmic Retention of a Nucleocytoplasmic Protein TBC1D3 by Microtubule Network Is Required for Enhanced EGFR Signaling". PLoS ONE 9, nr 4 (8.04.2014): e94134. http://dx.doi.org/10.1371/journal.pone.0094134.
Pełny tekst źródłaJessen, Niels, Ding An, Aina S. Lihn, Jonas Nygren, Michael F. Hirshman, Anders Thorell i Laurie J. Goodyear. "Exercise increases TBC1D1 phosphorylation in human skeletal muscle". American Journal of Physiology-Endocrinology and Metabolism 301, nr 1 (lipiec 2011): E164—E171. http://dx.doi.org/10.1152/ajpendo.00042.2011.
Pełny tekst źródłaWang, Bei, Dandan Chen i Haiying Hua. "TBC1D3 family is a prognostic biomarker and correlates with immune infiltration in kidney renal clear cell carcinoma". Molecular Therapy - Oncolytics 22 (wrzesień 2021): 528–38. http://dx.doi.org/10.1016/j.omto.2021.06.014.
Pełny tekst źródłaZhou, Zhou, Franziska Menzel, Tim Benninghoff, Alexandra Chadt, Chen Du, Geoffrey D. Holman i Hadi Al-Hasani. "Rab28 is a TBC1D1/TBC1D4 substrate involved in GLUT4 trafficking". FEBS Letters 591, nr 1 (20.12.2016): 88–96. http://dx.doi.org/10.1002/1873-3468.12509.
Pełny tekst źródłaMcMillin, Shawna L., Erin C. Stanley, Luke A. Weyrauch, Jeffrey J. Brault, Barbara B. Kahn i Carol A. Witczak. "Insulin Resistance Is Not Sustained Following Denervation in Glycolytic Skeletal Muscle". International Journal of Molecular Sciences 22, nr 9 (6.05.2021): 4913. http://dx.doi.org/10.3390/ijms22094913.
Pełny tekst źródłaHatakeyama, Hiroyasu, Taisuke Morino, Takuya Ishii i Makoto Kanzaki. "Cooperative actions of Tbc1d1 and AS160/Tbc1d4 in GLUT4-trafficking activities". Journal of Biological Chemistry 294, nr 4 (27.11.2018): 1161–72. http://dx.doi.org/10.1074/jbc.ra118.004614.
Pełny tekst źródłaWainszelbaum, Marisa J., Jialu Liu, Chen Kong, Priya Srikanth, Dmitri Samovski, Xiong Su i Philip D. Stahl. "TBC1D3, a Hominoid-Specific Gene, Delays IRS-1 Degradation and Promotes Insulin Signaling by Modulating p70 S6 Kinase Activity". PLoS ONE 7, nr 2 (13.02.2012): e31225. http://dx.doi.org/10.1371/journal.pone.0031225.
Pełny tekst źródłaMann, Gagandeep, Michael C. Riddell i Olasunkanmi A. J. Adegoke. "Effects of Acute Muscle Contraction on the Key Molecules in Insulin and Akt Signaling in Skeletal Muscle in Health and in Insulin Resistant States". Diabetology 3, nr 3 (28.07.2022): 423–46. http://dx.doi.org/10.3390/diabetology3030032.
Pełny tekst źródłaKothari, Charu, Alisson Clemenceau, Geneviève Ouellette, Kaoutar Ennour-Idrissi, Annick Michaud, René C.-Gaudreault, Caroline Diorio i Francine Durocher. "TBC1D9: An Important Modulator of Tumorigenesis in Breast Cancer". Cancers 13, nr 14 (16.07.2021): 3557. http://dx.doi.org/10.3390/cancers13143557.
Pełny tekst źródłaZhao, H. "18P TC-1 is required for TBC1D3-induced Wnt/beta-catenin accumulation and cell migration in MCF-7 breast cancer cells". Annals of Oncology 27 (grudzień 2016): ix5. http://dx.doi.org/10.1016/s0923-7534(21)00180-0.
Pełny tekst źródłaLipsey, Crystal C., Adriana Harbuzariu, Robert W. Robey, Lyn M. Huff, Michael M. Gottesman i Ruben R. Gonzalez-Perez. "Leptin Signaling Affects Survival and Chemoresistance of Estrogen Receptor Negative Breast Cancer". International Journal of Molecular Sciences 21, nr 11 (27.05.2020): 3794. http://dx.doi.org/10.3390/ijms21113794.
Pełny tekst źródłaSakamoto, Kei, i Geoffrey D. Holman. "Emerging role for AS160/TBC1D4 and TBC1D1 in the regulation of GLUT4 traffic". American Journal of Physiology-Endocrinology and Metabolism 295, nr 1 (lipiec 2008): E29—E37. http://dx.doi.org/10.1152/ajpendo.90331.2008.
Pełny tekst źródłaHenriques, Andreia F. A., Paulo Matos, Ana Sofia Carvalho, Mikel Azkargorta, Felix Elortza, Rune Matthiesen i Peter Jordan. "WNK1 phosphorylation sites in TBC1D1 and TBC1D4 modulate cell surface expression of GLUT1". Archives of Biochemistry and Biophysics 679 (styczeń 2020): 108223. http://dx.doi.org/10.1016/j.abb.2019.108223.
Pełny tekst źródłaWang, Bei, Huzi Zhao, Lei Zhao, Yongchen Zhang, Qing Wan, Yong Shen, Xiaodong Bu, Meiling Wan i Chuanlu Shen. "Up-regulation of OLR1 expression by TBC1D3 through activation of TNFα/NF-κB pathway promotes the migration of human breast cancer cells". Cancer Letters 408 (listopad 2017): 60–70. http://dx.doi.org/10.1016/j.canlet.2017.08.021.
Pełny tekst źródłaCartee, Gregory D. "Let's get real about the regulation of TBC1D1 and TBC1D4 phosphorylation in skeletal muscle". Journal of Physiology 592, nr 2 (styczeń 2014): 253–54. http://dx.doi.org/10.1113/jphysiol.2013.269092.
Pełny tekst źródłaZhao, Huzi, Lina Zhang, Yongchen Zhang, Lei Zhao, Qing Wan, Bei Wang, Xiaodong Bu, Meiling Wan i Chuanlu Shen. "Calmodulin promotes matrix metalloproteinase 9 production and cell migration by inhibiting the ubiquitination and degradation of TBC1D3 oncoprotein in human breast cancer cells". Oncotarget 8, nr 22 (31.03.2017): 36383–98. http://dx.doi.org/10.18632/oncotarget.16756.
Pełny tekst źródłaPark, Sang-Youn, i Soon-Jong Kim. "TBC1D1 and TBC1D4 (AS160) RabGAP Domains are Characterized as Monomers in Solution by Analytical Ultracentrifugation". Bulletin of the Korean Chemical Society 32, nr 6 (20.06.2011): 2125–28. http://dx.doi.org/10.5012/bkcs.2011.32.6.2125.
Pełny tekst źródłaCastorena, Carlos M., James G. MacKrell, Makoto Kanzaki, Jonathan S. Bogan i Gregory D. Cartee. "GLUT4, TBC1D1, TBC1D4, TUG and RUVBL2: Relationships with Each Other and Rat Muscle Fiber Type". Medicine & Science in Sports & Exercise 42 (październik 2010): 12–13. http://dx.doi.org/10.1249/01.mss.0000389501.47832.4f.
Pełny tekst źródłaCartee, Gregory D. "Roles of TBC1D1 and TBC1D4 in insulin- and exercise-stimulated glucose transport of skeletal muscle". Diabetologia 58, nr 1 (4.10.2014): 19–30. http://dx.doi.org/10.1007/s00125-014-3395-5.
Pełny tekst źródłaSchnurr, Theresia M., Emil Jørsboe, Alexandra Chadt, Inger K. Dahl-Petersen, Jonas M. Kristensen, Jørgen F. P. Wojtaszewski, Christian Springer i in. "Physical activity attenuates postprandial hyperglycaemia in homozygous TBC1D4 loss-of-function mutation carriers". Diabetologia 64, nr 8 (29.04.2021): 1795–804. http://dx.doi.org/10.1007/s00125-021-05461-z.
Pełny tekst źródłaGunsilius, Harald, Horst Borrmann, Arndt Simon i Werner Urland. "Zur Polymorphie von TbCI3/ Polymorphism of TbCl3". Zeitschrift für Naturforschung B 43, nr 8 (1.08.1988): 1023–28. http://dx.doi.org/10.1515/znb-1988-0819.
Pełny tekst źródłaZhang, Jianxian, Yan Xue, Hengling Gao, Yunxi Yu, Huabin Cheng, Xukun Lv i Ke Ke. "circZC3HAV1 Regulates TBC1D9 to Affect the Biological Behavior of Colorectal Cancer Cells". BioMed Research International 2022 (16.09.2022): 1–17. http://dx.doi.org/10.1155/2022/7386946.
Pełny tekst źródłaChang, Wen-Lin, Lina Cui, Yanli Gu, Minghua Li, Qian Ma, Zeng Zhang, Jing Ye, Fangting Zhang, Jing Yu i Yaoting Gui. "TBC1D20 deficiency induces Sertoli cell apoptosis by triggering irreversible endoplasmic reticulum stress in mice". Molecular Human Reproduction 25, nr 12 (21.10.2019): 773–86. http://dx.doi.org/10.1093/molehr/gaz057.
Pełny tekst źródłaZhou, Z., F. Menzel, T. Benninghoff, A. Chadt, C. Du, GD Holman i H. Al-Hasani. "Rab28 ist ein neu beschriebenes Substrat für TBC1D1/TBC1D4 und beteiligt an der regulierten Translokation von GLUT4". Diabetologie und Stoffwechsel 12, S 01 (5.05.2017): S1—S84. http://dx.doi.org/10.1055/s-0037-1601642.
Pełny tekst źródłaTreebak, Jonas T., Christian Pehmøller, Jonas M. Kristensen, Rasmus Kjøbsted, Jesper B. Birk, Peter Schjerling, Erik A. Richter, Laurie J. Goodyear i Jørgen F. P. Wojtaszewski. "Acute exercise and physiological insulin induce distinct phosphorylation signatures on TBC1D1 and TBC1D4 proteins in human skeletal muscle". Journal of Physiology 592, nr 2 (23.12.2013): 351–75. http://dx.doi.org/10.1113/jphysiol.2013.266338.
Pełny tekst źródłaFukuda, Mitsunori. "TBC proteins: GAPs for mammalian small GTPase Rab?" Bioscience Reports 31, nr 3 (14.01.2011): 159–68. http://dx.doi.org/10.1042/bsr20100112.
Pełny tekst źródłaPeifer-Weiß, Leon, Hadi Al-Hasani i Alexandra Chadt. "AMPK and Beyond: The Signaling Network Controlling RabGAPs and Contraction-Mediated Glucose Uptake in Skeletal Muscle". International Journal of Molecular Sciences 25, nr 3 (5.02.2024): 1910. http://dx.doi.org/10.3390/ijms25031910.
Pełny tekst źródłaHargett, Stefan R., Natalie N. Walker i Susanna R. Keller. "Rab GAPs AS160 and Tbc1d1 play nonredundant roles in the regulation of glucose and energy homeostasis in mice". American Journal of Physiology-Endocrinology and Metabolism 310, nr 4 (15.02.2016): E276—E288. http://dx.doi.org/10.1152/ajpendo.00342.2015.
Pełny tekst źródłaChadt, Alexandra, Anja Immisch, Christian de Wendt, Christian Springer, Zhou Zhou, Torben Stermann, Geoffrey D. Holman i in. "Deletion of Both Rab-GTPase–Activating Proteins TBC1D1 and TBC1D4 in Mice Eliminates Insulin- and AICAR-Stimulated Glucose Transport". Diabetes 64, nr 3 (23.09.2014): 746–59. http://dx.doi.org/10.2337/db14-0368.
Pełny tekst źródłaPark, Sang-Youn, Wanzhu Jin, Ju Rang Woo i Steven E. Shoelson. "Crystal Structures of Human TBC1D1 and TBC1D4 (AS160) RabGTPase-activating Protein (RabGAP) Domains Reveal Critical Elements for GLUT4 Translocation". Journal of Biological Chemistry 286, nr 20 (23.03.2011): 18130–38. http://dx.doi.org/10.1074/jbc.m110.217323.
Pełny tekst źródłaDi Chiara, Marianna, Bob Glaudemans, Dominique Loffing-Cueni, Alex Odermatt, Hadi Al-Hasani, Olivier Devuyst, Nourdine Faresse i Johannes Loffing. "Rab-GAP TBC1D4 (AS160) is dispensable for the renal control of sodium and water homeostasis but regulates GLUT4 in mouse kidney". American Journal of Physiology-Renal Physiology 309, nr 9 (1.11.2015): F779—F790. http://dx.doi.org/10.1152/ajprenal.00139.2015.
Pełny tekst źródłaHargett, Stefan R., Natalie N. Walker, Syed S. Hussain, Kyle L. Hoehn i Susanna R. Keller. "Deletion of the Rab GAP Tbc1d1 modifies glucose, lipid, and energy homeostasis in mice". American Journal of Physiology-Endocrinology and Metabolism 309, nr 3 (1.08.2015): E233—E245. http://dx.doi.org/10.1152/ajpendo.00007.2015.
Pełny tekst źródłaPehmøller, Christian, Jonas T. Treebak, Jesper B. Birk, Shuai Chen, Carol MacKintosh, D. Grahame Hardie, Erik A. Richter i Jørgen F. P. Wojtaszewski. "Genetic disruption of AMPK signaling abolishes both contraction- and insulin-stimulated TBC1D1 phosphorylation and 14-3-3 binding in mouse skeletal muscle". American Journal of Physiology-Endocrinology and Metabolism 297, nr 3 (wrzesień 2009): E665—E675. http://dx.doi.org/10.1152/ajpendo.00115.2009.
Pełny tekst źródłaVichaiwong, Kanokwan, Suneet Purohit, Ding An, Taro Toyoda, Niels Jessen, Michael F. Hirshman i Laurie J. Goodyear. "Contraction regulates site-specific phosphorylation of TBC1D1 in skeletal muscle". Biochemical Journal 431, nr 2 (28.09.2010): 311–20. http://dx.doi.org/10.1042/bj20101100.
Pełny tekst źródłaGutierrez, Jorge A., Christian M. Shannon, Shaun A. Nguyen, Ted A. Meyer i Paul R. Lambert. "Comparison of Transcutaneous and Percutaneous Implantable Hearing Devices for the Management of Congenital Aural Atresia: A Systematic Review and Meta-Analysis". Otology & Neurotology 45, nr 1 (26.11.2023): 1–10. http://dx.doi.org/10.1097/mao.0000000000004061.
Pełny tekst źródłaMafakheri, Samaneh, Ralf R. Flörke, Sibylle Kanngießer, Sonja Hartwig, Lena Espelage, Christian De Wendt, Tina Schönberger i in. "AKT and AMP-activated protein kinase regulate TBC1D1 through phosphorylation and its interaction with the cytosolic tail of insulin-regulated aminopeptidase IRAP". Journal of Biological Chemistry 293, nr 46 (1.10.2018): 17853–62. http://dx.doi.org/10.1074/jbc.ra118.005040.
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