Gotowe bibliografie tematyczne / Susceptibilité au stress

Gotowa bibliografia na temat „Susceptibilité au stress”

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Data publikacji: 20 lipca 2024

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Artykuły w czasopismach na temat "Susceptibilité au stress"

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Tiret, Laurence. "Gene-environment interaction: a central concept in multifactorial diseases". Proceedings of the Nutrition Society 61, nr 4 (listopad 2002): 457–63. http://dx.doi.org/10.1079/pns2002178.

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RésuméA la différence des mutations génétiques rares mais séveères qui causent les maladies monogéniques, les facteurs génétiques qui modulent la susceptibilité individuelle aux maladies multifactorielles (maladies cardiovasculaires, cancer, diabeète) sont des formes fréquentes, fonctionnellement différentes, des ge`nes (polymorphismes) qui ont généralement un effet modeste au niveau individuel, mais en raison de leur grande fréquence dans la population, peuvent être associés à un risque attribuable élevé. Les facteurs environnementaux peuvent révéler ou faciliter l'expression phénotypique de ces ge`nes de susceptibilité. En effet, dans le cas des maladies communes, les effets génétiques peuvent etre considérablement amplifiés en présence de facteurs déclenchants. On sait maintenant que la plupart des geènes de susceptibilité aux maladies communes n'ont pas un rôle étiologique primaire dans la prédisposition a` la maladie mais agissent plutot comme des modificateurs dans la réponse a` des facteurs exoge`nes tels que le stress, l'environnement, la maladie, la prise de médicament, le régime alimentaire. Une meilleure caractérisation des interactions entre facteurs génétiques et environnementaux constitue un élément clé dans la compréhension de la pathogéneèse des maladies multifactorielles. Cet article présente trois exemples d'interaction gène-environnement dans le domaine des maladies coronariennes.
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Benajiba, N., J. De Leiris, B. Lyan, A. Derouiche, N. Mokhtar i H. Aguenaou. "Effet de l’huile d’argan sur la contractilité de l’aorte : susceptibilité au stress oxydatif". Oléagineux, Corps gras, Lipides 13, nr 1 (styczeń 2006): 76–80. http://dx.doi.org/10.1051/ocl.2006.0076.

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Chakroun Feki, Nozha, Patrice Therond, Pierre Jouannet i Jacques Auger. "Composition lipidique des spermatozoides humains et susceptibilité au stress oxydant avant et après migration dans le mucus cervical". Andrologie 13, nr 4 (grudzień 2003): 381–92. http://dx.doi.org/10.1007/bf03035205.

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Auxéméry, Y. "L’état de stress post-traumatique comme conséquence de l’interaction entre une susceptibilité génétique individuelle, un évènement traumatogène et un contexte social". L'Encéphale 38, nr 5 (październik 2012): 373–80. http://dx.doi.org/10.1016/j.encep.2011.12.003.

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Pospichal, A., D. Pokorova, T. Vesely i V. Piackova. "Susceptibility of the topmouth gudgeon (Pseudorasbora parva) to CyHV-3 under no-stress and stress conditions". Veterinární Medicína 63, No. 5 (29.05.2018): 229–39. http://dx.doi.org/10.17221/88/2017-vetmed.

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Cyprinid herpesvirus 3 (CyHV-3), also known as koi herpesvirus, is the causative agent of the highly contagious koi herpesvirus disease, which is restricted to koi and common carp and causes significant losses in both fish stock. Some experimental investigations have shown that other cyprinid or non-cyprinid species may be asymptomatically susceptible to this virus and might play roles as potential carriers of CyHV-3 or might contribute to persistence of this virus in environment. Therefore, it seems important to verify not only the susceptibility of other cyprinid or non-cyprinid species, but also their ability to transmit CyHV-3 infection to susceptible species. Our previous investigation of the susceptibility of the topmouth gudgeon (Pseudorasbora parva) did not reveal the presence of CyHV-3 DNA in the tissues of this species after cohabitation with infected koi. Consequently, we changed the experimental conditions and applied two stress factors (removal of skin mucus and scaring) which would presumably mimic the stress most commonly encountered in the wild. Both experiments (without and with stress factors) consisted of primary and secondary challenges. In both the no-stress and stress experiments, the first challenge was focused only on testing the susceptibility of the topmouth gudgeon to the virus. With the secondary challenge, we investigated potential viral transmission from the topmouth gudgeon to healthy naive koi after exposure to stress factors. All fish (dead, surviving and sacrificed) were tested for the presence of CyHV-3 DNA using nested PCR (no-stress experiment) and real-time PCR (stress experiment). After the primary challenge of the no-stress experiment, PCR did not reveal the presence of CyHV-3 DNA in any specimen of cohabitated topmouth gudgeon, but all specimens of dead koi were CyHV-3 DNA-positive. PCR of fish tissues subjected to the secondary challenge did not show the transfer of virus to naive fish. After exposure to stress (removal of skin mucus), qPCR revealed four out of five samples (80%) of topmouth gudgeon to be positive for CyHV-3 DNA. Two out of five samples (40%) of topmouth gudgeon treated by scaring were found to be positive for the presence of viral DNA. Real-time PCR after the secondary challenge did not reveal any viral DNA positivity in specimens of topmouth gudgeon from groups previously exposed to stress. The stress experiments show that removal of skin mucus might potentially lead to susceptibility of topmouth gudgeon to CyHV-3 infection, but the transmission of the virus to koi carp was not observed.
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Paré, William P. "Prior stress and susceptibility to stress ulcer". Physiology & Behavior 36, nr 6 (styczeń 1986): 1155–59. http://dx.doi.org/10.1016/0031-9384(86)90493-2.

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Salim, Samina, Hesong Liu i Fatin Atrooz. "Early life stress, stress-resilience/susceptibility and oxidative stress". Free Radical Biology and Medicine 120 (maj 2018): S165. http://dx.doi.org/10.1016/j.freeradbiomed.2018.04.542.

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Majlingová, A., M. Závacká i D. Kliment. "An assessment of hucava mountain stream catchment susceptibility to flooding". Journal of Forest Science 58, No. 12 (11.12.2012): 553–59. http://dx.doi.org/10.17221/75/2011-jfs.

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The paper deals with an assessment of the Hucava mountain stream catchment susceptibility to flooding. The model catchment of the Hucava mountain stream is located in the Polana Protected Landscape Area – Biosphere Reserve, situated in the central part of Slovakia. The assessment of overall susceptibility of the model area to flooding is based on the multi-criteria evaluation of environmental factors, which crucially affect the hydrological cycle. These are represented by the geology, soil types, slope, forest type group, degree of ecological stability, exploitation of non-forest landscape and potential runoff in the model area. The methodology is based on the synthesis and subsequent processing of data in the GIS environment. The result is represented by the specification of categories (degrees of  flood risk)  to classify the model area to five degrees of overall susceptibility to flood as well as by the creation of maps representing the spatial distribution of different categories in the Hucava catchment.  
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Ebner, Karl, i Nicolas Singewald. "Individual differences in stress susceptibility and stress inhibitory mechanisms". Current Opinion in Behavioral Sciences 14 (kwiecień 2017): 54–64. http://dx.doi.org/10.1016/j.cobeha.2016.11.016.

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Pohl, Tobias, i Hanna Hörnberg. "SOCIAL STRESS DURING ADOLESCENCE: NEUROIMMUNOLOGICAL SIGNATURES OF STRESS SUSCEPTIBILITY". IBRO Neuroscience Reports 15 (październik 2023): S47. http://dx.doi.org/10.1016/j.ibneur.2023.08.2148.

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Rozprawy doktorskie na temat "Susceptibilité au stress"

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Merlot, Elodie. "Modulation de la production de cytokines par l'environnement social et susceptibilité aux infections". Paris, Institut national d'agronomie de Paris Grignon, 2003. https://pastel.archives-ouvertes.fr/pastel-00000790.

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L'environnement social contribue au développement et à l'expression de maladies. En effet, dans les espèces sociales, la susceptibilité aux infections dépend de la position sociale mais les bases endocriniennes et immunitaires de ces différences au sein du groupe sont ignorées. De plus, une modification de la structure sociale engendre un stress important dont les conséquences immunitaires sont encore sujettes à controverse. Ce travail de thèse a pour objectifs (1) de décrire l'influence du statut social sur le fonctionnement des systèmes endocrinien et immunitaire, (2) de préciser les effets du stress social sur la production de cytokines et la susceptibilité aux infections et (3) de rechercher des facteurs à l'origine de la variabilité des conséquences immunitaires du stress social. Une étude introductive sur le porcelet mise à part, un modèle murin a été utilisé. Les résultats montrent une influence du statut social. En absence de stress, les dominants présentent des niveaux de base de corticostérone et une réponse à la tuberculine supérieurs aux dominés. Suite à une procédure de défaite sociale chronique, les dominants sont plus affectés que les dominés. La défaite sociale accroît la réactivité inflammatoire mais ne modifie pas l'équilibre de la production de cytokines de type1 et 2 et n'affecte pas la réponse à une infection mycobactérienne. Les conséquences immunitaires de la défaite sociale ne sont observées que lorsque le stress est associé à des combats et à des blessures. Ces travaux montrent enfin que la réponse au stress dépend de l'histoire sociale de l'individu, en particulier de son statut social, et son histoire immunitaire récente
In social species, social environment contributes to the development and expression of diseases. Indeed, the social position in a group modulates susceptibility to infections. However, the endocrine and immune mechanisms involved in these differences are not known. Furthermore, instability in the social organisation generates a severe stress of which immune consequences are still controversial. The purposes of this thesis are (1) to describe the influence of social status on the endocrine and immune systems, (2) to specify the effects of social stress on cytokine production and susceptibility to infections and (3) to look for possible sources of variability in the immune consequences of social stress. Excepted a single study on piglets, experiments were performed in mice. Results show an influence of social status. In the absence of stress, dominants show higher basal corticosterone levels and specific response to tuberculin than subordinates. After social defeat, dominants are more affected. Social defeat increases inflammatory reactivity but does not clearly modify the balance between Th1 and Th2 cytokines production and does not affect the development of specific immunity against a mycobacterial infection. Immune effects of social defeat can be observed only when the stress involves significant fights and injuries. Our work also shows that the response to stress depends on individual social experience, in particular on social status, and on the recent immune experience of the individual
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Blein, Sophie. "Étude de la variabilité du génome mitochondrial comme facteur de susceptibilité au cancer du sein". Thesis, Lyon 1, 2014. http://www.theses.fr/2014LYO10240/document.

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Une large part de la composante génétique du risque de cancer du sein est encore inexpliquée. J'ai ainsi étudié dans quelle mesure les variants observés sur le génome mitochondrial pourraient en partie expliquer ce risque. En effet la mitochondrie, en tant que source d'énergie cellulaire, est un organite impliqué dans la synthèse des espèces oxygénées réactives ou radicaux libres, éléments contribuant à l'instabilité génomique et au développement tumoral. Un premier axe de recherche m'a conduit à étudier une interaction potentielle entre des variants du génome mitochondrial et du génome nucléaire, en conjonction avec la consommation d'alcool. J'ai ensuite analysé les haplogroupes mitochondriaux peuvent être considérés en tant que potentiels modificateurs de l'association entre le risque de cancer du sein et les mutations causales portées par les gènes BRCA1 et BRCA2. L'haplogroupe T1a1 a été identifié comme modificateur du risque conféré par les mutations pathogènes localisées sur le gène BRCA2. Enfin, j'ai caractérisé par séquençage à haut débit le génome mitochondrial de 436 femmes ayant un cancer du sein et de forts antécédents familiaux, mais n'étant porteuses d'aucune mutation causale sur BRCA1 et BRCA2. Plusieurs variants ont été prédits comme dommageables. Deux gènes en particulier MT-ATP6 et MT-CYB, sont spécifiquement enrichis à la fois en nombre de variants portés, et de par le nombre d'individus porteurs de ces variants dans notre étude. L'ensemble du travail réalisé a ainsi contribué à enrichir les connaissances sur les potentielles associations entre les variations du génome mitochondrial et le risque du cancer du sein
A large part of the genetic component of breast cancer risk (BCR) is still unexplained. Therefore I studied if variants of the mitochondrial genome (mtDNA) might explain a part of this risk. In fact, mitochondria is the main source of reactive oxygen species (ROS), which contribute to genomic instability and tumor development. As a first axis of research, I studied potential interactions between some nuclear and mitochondrial variants, in conjugation to alcohol consumption. Despite the large dimensions of our dataset, the lack of statistical significant interaction in our data might reveal that former published results that show such interactions were not robust. I also studied if mitochondrial haplogroups could be considered as modificators of known association between BCR and pathogenic mutations in the BRCA1/2 genes. I identified haplogroup T1a1 such as modificator for individuals carrying a mutated BRCA2. Finally, I characterized by NGS mitochondrial genome of women diagnosed for a familial breast cancer, but tested negative for known pathogenic BRCA1/2 mutations. Several variants were identified as potentially damaging. Two genes, MT-ATP6 and MT-CYB are specifically enriched both in terms of distinct variants and in the number of individuals carrying these variants. They are both essential structural components of the mitochondrial respiratory chain, the main ROS production source in the cell. All these analyses contribute to enrich the knowledge about associations between BCR and variability of mtDNA, by integrating questions linked to interactions between genomic variants, environmental exposure, and effect modifications related to mitochondrial haplogroups
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Rakotobé, Malalaniaina. "Bases neuro-développementales de la susceptibilité à l’anxiété : apports de l’étude du système habénulo-interpédonculaire". Electronic Thesis or Diss., Université Côte d'Azur, 2021. http://www.theses.fr/2021COAZ6002.

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Les circuits de récompense du cerveau sont des réseaux neuronaux qui modulent positivement ou négativement la motivation à agir. Ils sont essentiels à la survie en sélectionnant les comportements adaptés à la situation, comme fuir les dangers ou rechercher de la nourriture. Leurs dysfonctionnements peuvent entraîner des troubles de l’humeur et des addictions. Bien que ces troubles psychiatriques se manifestent chez l’adulte, selon l’hypothèse neuro-développementale ils peuvent avoir une origine plus précoce. En effet, il existe des périodes particulières du développement appelées périodes critiques, durant lesquelles les facteurs génétiques et environnementaux influencent la maturation des circuits neuronaux. Un environnement stressant et/ou un défaut d’expression de gènes développementaux durant ces périodes, peuvent conduire à la malformation de ces circuits neuronaux, dont le système de récompense, et ainsi créer une prédisposition à des maladies psychiatriques. Le système habénulo-interpédonculaire (HIPS), constitué de l’habénula médiane (MHb) et de sa principale cible, le noyau interpédonculaire (IPN), émerge comme un composant clé de ce système de récompense. Il est impliqué dans des pathologies comme l’addiction, l’anxiété et la dépression. Notre équipe a récemment montré que le facteur de transcription Otx2 est essentiel pour la formation du HIPS. Son expression est maintenue dans le HIPS tout au long de la vie, fortement dans les derniers neurones générés dans la MHb (neurones Otx2High) et dans les sous-noyaux latéraux et le sous-noyau central de l’IPN caudal, suggérant qu’il pourrait aussi jouer un rôle dans la maturation et le fonctionnement du HIPS. Dans l’aire tegmentale ventrale (VTA), un autre acteur du système de récompense, un stress chronique subi pendant l’enfance diminue le niveau d’expression d’Otx2 et prédispose à la dépression et à l’anxiété. Otx2 pourrait jouer un rôle similaire dans la réponse du HIPS au stress pendant des périodes critiques du développement. Ce travail de thèse vise à mieux comprendre comment des facteurs génétiques et environnementaux affectant le développement du HIPS peuvent conduire à l’apparition de troubles psychiatriques. La première partie de la thèse montre que les neurones Otx2High de la MHb projettent sur les neurones Otx2+ de l’IPN, révélant l’existence d’un sous-circuit Otx2 dépendant dans le HIPS. L’activation chémo-génétique de la MHb à l’aide d’un DREADD a confirmé que les neurones Otx2High de la MHb sont capables de moduler l’activité des neurones Otx2+ de l’IPN et démontre qu’ils forment un sous-circuit au sein du HIPS, marqué par l’expression d’Otx2 et dénommé HIPOPS (Habenulo-InterPeduncular-Otx2-Positive-System).Dans la deuxième partie de ce projet, la période critique du développement du HIPS sensible au stress a été recherchée. L’utilisation des marqueurs d’activation neuronale Fos et Egr1 a permis d’identifier une période préadolescente durant laquelle le HIPS est plus réactif à un stress chronique. De manière intéressante, ce stress chronique engage particulièrement l’activité du HIPOPS, soulignant l’implication de ce sous-circuit dans la réponse au stress. Dans la troisième partie, un modèle de stress chronique « à deux coups » a été développé, et révèle que ce stress chronique subi durant la préadolescente prédispose à l’anxiété mais pas à la dépression à long-terme. Dans une dernière partie, les effets d’une délétion conditionnelle d’Otx2 dans le HIPS sur ces comportements ont été étudiés. Cette étude a permis de montrer que la délétion d’Otx2 dans la MHb protège des effets délétères du stress chronique préadolescente sur l’anxiété. Donc, l’interaction entre environnement (le stress) et gènes (Otx2) à la préadolescence détermine la susceptibilité à développer une anxiété chronique à long-terme et qu’un des supports cellulaires de cette susceptibilité est le HIPOPS, un circuit logé au cœur du HIPS, identifié par l’expression d’Otx2
Reward circuits of the brain are neuronal networks that modulate positively or negatively the motivation to act according to the situation. They are essential for the survival of the subject and of species by selecting the appropriate behaviors, such as fleeing the danger or searching for food. Dysfunctions of these circuits can lead to mood disorders and addictions. Although these psychiatric disorders can emerge in adulthood, according to the neurodevelopmental hypothesis they can find their origins at a much earlier stage. Indeed, there are periods of development, called critical periods of development, during which genetic and environmental factors influence the maturation of neuronal circuits. A stressful environment and/or defaults in the expression of developmental genes during these critical periods can lead to the malformation of neuronal circuits, including the reward circuits, creating a predisposition to psychiatric disorders. The habenulo-interpeduncular system (HIPS), composed of the medial habenula (MHb) and its main target, the interpeduncular nucleus (IPN), emerges as a key component of the reward circuits. It is notably involved in disorders such as addiction, anxiety and depression. Our team has recently identified the Otx2 transcription factor as an essential genetic factor for the formation of the MHb and IPN. Otx2 expression is maintained in the HIPS throughout life, strongly in the last neurons generated in the MHb (Otx2High neurons) and in neurons of the lateral and central subnucleus of the caudal IPN, suggesting that it might also play a role in the maturation and functioning of the HIPS. We already know that in the ventral tegmental area (VTA), another actor of the reward circuits, a chronic stress, endured during a specific period of childhood decreases the expression level of Otx2 and predisposes to depression and anxiety. It is thus possible that Otx2 might play a similar function in the HIPS response to stress during periods of development. This thesis work aimed to better understand how genetic and environmental factors affecting the development of the HIPS could lead to the development of psychiatric disorders.The first part of this thesis shows that Otx2High neurons of the MHb project on the Otx2+ neurons of the IPN, revealing an Otx2-dependant sub-circuit in the HIPS. The MHb was then chemogenetically activated with the use of DREADD to verify that Otx2High neurons of the MHb are indeed able to modulate the activity of the Otx2+ neurons of the IPN and confirmed the idea that these neurons constitute a sub-circuit of the HIPS, labelled with the expression of Otx2, that we will call the HIPOPS (Habenulo-InterPeduncular-Otx2-Positive-System).In the second part of this project, a critical period of development during which the HIPS is more sensitive to stress has been searched. By using Fos and Egr1, two neuronal markers of activity, we identified a preadolescent period during which the HIPS is more responsive to chronic stress. Interestingly, that chronic stress involves more particularly the activity of the HIPOPS, emphasizing the implication of that sub-circuit in the stress response.In the third part, a “2-hit” stress protocol of chronic stress has been developed and revealed that a chronic stress endured during preadolescence predisposes to anxiety but not to depression in the long-term. In the last part, the effects of Otx2 conditional deletion in the HIPS has been assessed. This study shows that Otx2 deletion in the MHb protects from the deleterious effects of preadolescent chronic stress on anxiety symptoms. This work, therefore, demonstrates that the interaction between environment (stress) and genes (Otx2) during preadolescence determines the susceptibility to develop chronic anxiety in the long term and that one of the cellular support of that susceptibility is the HIPOPS, a circuit lodged inside the HIPS, recognizable by Otx2 expression
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Moulin, Sophie. "Rôle de l'hypoxia-inducible factor-1 dans la susceptibilité myocardique à l'ischémie-reperfusion induite par l'hypoxie intermittente". Thesis, Université Grenoble Alpes (ComUE), 2018. http://www.theses.fr/2018GREAV034/document.

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Le syndrome d’apnées obstructives du sommeil (SAOS) est un problème de santé publique majeur qui est considéré comme un facteur indépendant de risque de survenue d’un infarctus du myocarde (IM). Les altérations cardiovasculaires associées au SAOS sont principalement dues à l’hypoxie intermittente (HI) chronique. En particulier, l’HI induit l’activation du facteur de transcription hypoxia-inducible factor-1 (HIF-1), susceptible d’être impliqué dans la vulnérabilité accrue du myocarde à l’ischémie-reperfusion. L’objectif de cette thèse était d’étudier le rôle de HIF-1 dans les mécanismes induits par l’HI et impliqués dans l’augmentation de la taille de l’infarctus suite à une ischémie-reperfusion. Ces travaux ont mis en évidence deux nouveaux effets délétères de l’HI, à savoir l’induction d’un stress du réticulum endoplasmique (RE) et d’altérations mitochondriales. A travers, l’inhibition génétique et/ou pharmacologique de HIF-1, nous avons montré que HIF-1 apparaît comme un acteur primordial dans l’ensemble des mécanismes délétères de l’HI, incluant ceux découverts lors de cette thèse. De plus, HIF-1 joue un rôle majeur dans l’augmentation de la taille de l’IM induite par l’HI chronique. Parallèlement, son activation myocardique est corrélée à l’index d’apnées-hypopnées chez des patients apnéiques atteints d’une maladie coronarienne (comparativement aux non-apnéiques). Par conséquent, l’activation de HIF-1 pourrait être utilisée comme marqueur diagnostic du SAOS chez les patients à risque cardiovasculaire. HIF-1 pourrait également représenter une cible pour le développement de nouvelles thérapies complémentaires ou substitutives aux traitements actuels
Obstructive sleep apnea syndrome (OSAS) is a major public health problem that is considered an independent risk factor for the occurrence of myocardial infarction (MI). The cardiovascular alterations associated with OSA are mainly due to the chronic intermittent hypoxia (IH). In particular, activation by IH, the hypoxia-inducible factor-1 (HIF-1) transcription factor likely contributes to enhance myocardial vulnerability to ischemia-reperfusion injury. The aim of this thesis was to study the role of HIF-1 in the mechanisms involved in the increase in MI induced by chronic IH. This work has highlighted two new deleterious consequences of IH exposure, namely endoplasmic reticulum (ER) stress and mitochondrial alterations. Through genetic and/or pharmacological inhibition of HIF-1, we have shown that HIF-1 appears to be a primordial actor in all the deleterious mechanisms of IH, including those discovered during this thesis. HIF-1 also appears to play a major role in the IH-induced increase in MI size. In parallel, its myocardial activation is correlated with the apnea-hypopnea index in apnoeic, compared to non-apnoeic, patients with coronary heart disease. Therefore, HIF-1 activation could serve as a diagnostic marker of OSA in patients with cardiovascular risk. HIF 1 could also be a target for new therapeutic approaches, in complement or replacement of standard treatments
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Le, guernic Antoine. "Effets sublétaux d'une contamination métallique liée à des rejets miniers uranifères sur l'épinoche à trois épines (Gasterosteus aculeatus L.). Implication dans la susceptibilité envers un stress biologique". Thesis, Reims, 2015. http://www.theses.fr/2015REIMS027.

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L’extraction de l’uranium a eu pour conséquence une remobilisation de cet actinide au niveau des écosystèmes avoisinants les mines. L’utilisation de sels métalliques lors de la réhabilitation des sites miniers et la présence naturelle de métaux ont accentué les niveaux de la contamination métallique dans les hydrosystèmes soumis aux rejets miniers uranifères.Des expériences in situ ont été conduites au niveau de deux anciens sites miniers uranifères français. L’encagement de l’épinoche à trois épines a été employé pour connaître les effets sublétaux de ce mélange métallique, ainsi que sur la susceptibilité de ce poisson envers un stress biologique.Cette pollution, caractérisée par de plus importantes concentrations métalliques (notamment en uranium), a entraîné un stress oxydant chez l’épinoche visible sur plusieurs biomarqueurs, ainsi que d’autres effets dépendants du site d’exposition.La contamination polymétallique a occasionné une augmentation de la susceptibilité des épinoches au stress biologique, en empêchant leurs réponses phagocytaire antioxydante à ce stress. Ces travaux ont permis de renforcer l’intérêt de la technique d’encagement lors d’une étude environnementale, et celui des immunomarqueurs au sein d’une approche multi-biomarqueurs
Uranium extraction has resulted in a remobilisation of this actinide into mine surrounding ecosystems. Uses of metal salts during mining site rehabilitation, and the natural presence of metals have increased the metal contamination in hydrosystems submitted to mine tailings.In situ experiments were conducted in two former French uranium mining sites. Three-spined stickleback caging was used to determine the sublethal effects of this metal mixture on this freshwater fish, as well as its effects on fish susceptibility to a sudden biological stress.This pollution, characterised by higher metal concentrations (especially for uranium), has led to an oxidative stress in sticklebacks visible through several biomarkers, and other effects dependent on the study site. The polymetallic contamination has modified the stickleback responses to the biological stress, by preventing their phagocytic and antioxidant responses. This work has reinforced the interest of the caging technique during environmental studies and that of immunomarkers in a multi-biomarker approach
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Pujol, Marine. "Variabilité comportementale en réponse au stress social : influence du variant allélique VGLUT3-p.T8I sur l'acétylcholine et la dopamine dans le noyau accumbens". Electronic Thesis or Diss., Sorbonne université, 2024. http://www.theses.fr/2024SORUS098.

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Le stress représente un facteur de risque majeur dans le développement des troubles psychiatriques et des troubles du sommeil. La diversité des réponses émotionnelles au stress résulte de l'influence conjointe de facteurs environnementaux et génétiques. Le noyau accumbens (NAc) joue un rôle central dans la susceptibilité au stress et récemment, son implication dans la régulation du sommeil a été mise en évidence. Les adaptations comportementales liées à la résilience ou la susceptibilité au stress sont modulées par les transmissions dopaminergique et cholinergique au sein du NAc. L'acétylcholine (ACh) provient principalement des interneurones cholinergiques (CINs), qui communiquent à la fois avec du glutamate (Glut) et de l'ACh, respectivement grâce au transporteur vésiculaire du glutamate de type 3 (VGLUT3) et au transporteur vésiculaire d'acétylcholine (VAChT). L'équipe a récemment montré que le variant allélique p.T8I-VGLUT3 entraine une diminution de la transmission cholinergique dans le striatum, sans affecter le transport du Glut. Ainsi, l'objectif de ma thèse a été de caractériser le rôle du variant allélique p.T8I dans la réponse au stress en caractérisant le comportement et le sommeil ainsi que les transmissions d'ACh et de DA associées dans le NAc d'un modèle murin. Pour cela, j'ai évalué le comportement social, de type anxieux et dépressif, ainsi que les cycles veille-sommeil des souris VGLUT3T8I/T8I et témoins mâles et femelles avant et après un stress chronique de défaite sociale (CSDS). Afin d'étudier le rôle de l'ACh et de la DA dans ces comportements, j'ai enregistré leur dynamique de libération par photométrie de fibre à l'aide de biosenseurs fluorescents dans ce modèle murin.Les données obtenues montrent que le variant p.T8I est associé à un évitement social accru dépendant de la cible sociale, et de niveaux élevés d'anxiété, accompagnés d'une architecture du sommeil distincte en réaction au stress. De plus, ces comportements sont corrélés à une libération réduite d'ACh en réponse au stress chez les mâles. Ces travaux permettent ainsi de mettre en évidence la relation entre le variant p.T8I, les transmissions d'ACh et de DA, et la réponse au stress chronique social, impliquée dans la susceptibilité au stress
Stress is a risk factor for the development of psychiatric conditions such as generalized anxiety and major depressive disorder which are often associated with sleep disturbances. The behavioral adaptation to stress is influenced by a combination of environmental and genetic factors, leading to significant variability among individuals. The nucleus accumbens (NAc) plays a critical role in stress susceptibility and sleep regulation. In the NAc, the behavioral adaptations associated with resilience or susceptibility to stress are modulated by dopamine (DA) and acetylcholine (ACh) signals. The latter is predominantly released by cholinergic interneurons (CINs). Notably, CINs have the ability to signal with both ACh and glutamate through the expression of vesicular acetylcholine transporter (VAChT) and vesicular glutamate transporter 3 (VGLUT3). Our lab recently showed that the VGLUT3-p.T8I allelic variant results in a reduced ACh release from CINs without affecting glutamate release.The objective of my PhD was to elucidate the role of the p.T8I variant in shaping behavioral and sleep responses to stress as well as the stress-associated neuronal ACh and DA dynamics in the NAc. To address these questions, I took advantage of a mouse line carrying the p.T8I variant (VGLUT3T8I/T8I) to study social, anxiety- and depressive-like behaviors as well as sleep-wake patterns in male and female mice both before and after chronic social defeat stress (CSDS) exposure. Then, I measured in vivo ACh and DA dynamics in the NAc using fiber photometry coupled with fluorescent biosensors to assess their release in response to stress. The findings indicate that the p.T8I variant is associated with enhanced social cue discrimination characterized by high target-dependent social avoidance, and elevated anxiety-like levels, accompanied by distinct acute stress-induced sleep changes. Moreover, these behaviors are associated with reduced stress-induced ACh release in the NAc in male mice. Together, these findings reveal the intricate relationship between the p.T8I variant, diminished cholinergic transmission within the NAc and susceptibility to stress
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Etain, Bruno. "Contribution à l'étude des facteurs génétiques et environnementaux de susceptibilité aux troubles bipolaires : études du trouble bipolaire à début précoce et des traumatismes affectifs de l’enfance". Thesis, Paris Est, 2009. http://www.theses.fr/2009PEST0048.

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Les troubles bipolaires (TB) sont des maladies psychiatriques dont le déterminisme complexe fait intervenir des facteurs génétiques et environnementaux de susceptibilité. Les efforts d’identification des facteurs génétiques ont produit des résultats discordants et les facteurs environnementaux restent mal connus. Notre équipe a contribué à identifier une forme à début précoce des TB (TB-DP), son caractère fortement familial en faisant un candidat pour faciliter l’identification de gènes de susceptibilité. Ainsi, nous avons réalisé un criblage systématique du génome dans le TB-DP suggérant une liaison avec les régions chromosomiques 2p21, 2q14, 3p14, 5q33, 7q36, 10q23, 16q23 et 20p12. Nous avons montré des associations entre le TB-DP et le gène SNAP25 (rôle dans les mécanismes d’exocytose, région 20p12) et entre les TB et le gène codant l’ASMT (rôle dans la synthèse de la mélatonine). Enfin, une étude d’association pangénomique suggère une association entre le TB-DP et deux gènes de la voie du phosphatidyl-inositol (PLEKHA5 et PLCXD3). Concernant les facteurs environnementaux, les traumatismes affectifs subis dans l’enfance (principalement les abus émotionnels) sont associés aux TB, influencent deux dimensions constitutives des TB (labilité affective et intensité des affects) et interagissent avec le 5HTTLPR pour moduler l’âge de début des troubles. Ces travaux illustrent la pertinence de se focaliser sur le TB-DP pour identifier des gènes de susceptibilité, la nécessité d’explorer plus finement les facteurs environnementaux (notamment les stress précoces) et de considérer les interactions gène-environnement afin de mieux appréhender le déterminisme complexe des TB
Bipolar disorders (BD) are psychiatric diseases with a complex determinism in which genetic and environmental susceptibility factors are involved. Attempts to identify genetic factors have produced conflicting results and environmental factors remain unknown. Early-onset bipolar disorder (EO-BD) is a clinical entity that is characterized by a strong familial aggregation ; a specific focus on this subtype might facilitate the identification of susceptibility genes. A genome-wide scan in EO-BD has suggested eight regions of linkage (chromosomal regions 2p21, 2q14, 3p14, 5q33, 7q36, 10q23, 16q23 and 20p12). We have demonstrated an association between EO-BD and the SNAP25 gene (located at 20p12 and involved in exocytosis). We have demonstrated an association between BD and the ASMT gene (involved in the synthesis of melatonin). Finally, a genome-wide association study has suggested the involvement of two phosphatidyl-inositol pathway related genes in the susceptibility to EO-BD (PLEKHA5 et PLCXD3). Concerning environmental susceptibility factors, childhood affective traumatic events (mainly emotional abuse) are associated with BD, might influence two core dimensions of BD (affective lability and affect intensity) and might interact with the serotonin transporter genelinked polymorphic region to modulate the age of onset of the disorder. These studies illustrate the relevance of focusing on the early onset subgroup of the disease to identify susceptibility genes, the need to further explore early stressors as environmental factors associated with BD and to investigate the complex relationships between these two kinds of susceptibility factors
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Jones, C. A. "A study of stress susceptibility in pigs". Thesis, University of Bristol, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.372043.

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Thomas, Gladys Ruth. "Susceptibility of Brugia malayi to oxidative and nitrosative stress". Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267808.

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鍾慧虹 i Wai-hung Angela Chung. "Cognitive vulnerability, stress, and postpartum depressive symptomatology: a prospective study in HongKong". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B37101055.

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Książki na temat "Susceptibilité au stress"

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Gillis, A. R. Cultural susceptibility to crowding: An empirical analysis. Toronto: Centre for Urban and Community Studies, University of Toronto, 1986.

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Lyle, Fred F. Stress-corrosion cracking susceptibility of weldments in duplex stainless steels. St. Louis, Missouri: Materials Technology Institute of the Chemical Process Industries, 1989.

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Madra, Moneek. Interactions between the BDNF (Val66Met) variant and stress as a model of susceptibility to Anorexia Nervosa. [New York, N.Y.?]: [publisher not identified], 2015.

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International Berzelius Symposium. (6th 1985 Malmö, Sweden). Social support--health and disease. Redaktorzy Isacsson Sven-Olof, Janzon Lars i Sweden. Delegationen för social forskning. Stockholm, Sweden: Almqvist & Wiksell International, 1986.

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J, Sholar Clyde. Evaluation of the drought susceptibility of water supplies used in the Kentucky River Basin in 1988. Louisville, Ky. (2301 Bradley Ave., Louisville 40217): U.S. Dept. of the Interior, U.S. Geological Survey, 1991.

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Sholar, Clyde J. Evaluation of the drought susceptibility of water supplies used in the Kentucky River Basin in 1988. Louisville, Ky. (2301 Bradley Ave., Louisville 40217): U.S. Dept. of the Interior, U.S. Geological Survey, 1991.

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B, Ludvigsen J., European Association for Animal Production. Commission on Animal Management and Health., European Association for Animal Production. Commission on Pig Production. i European Association for Animal Production. Meeting, red. Stress susceptibility and meat quality in pigs: Proceeding of Commis[s]ion on Animal Management and Health and Commission on Pig Production joint session, Halkidiki, Greece, 30. Sept.-5. Oct. 1985. [Rome?]: European Association for Animal Production, 1985.

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L, Power Michael, i Schulkin Jay, red. Birth, distress, and disease: Placental-brain interactions. Cambridge: Cambridge University Press, 2005.

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Tsai, Jack, Natalie Jones, Robert H. Pietrzak, Ilan Harpaz-Rotem i Steven M. Southwick. Susceptibility, Resilience, and Trajectories. Redaktorzy Frederick J. Stoddard, David M. Benedek, Mohammed R. Milad i Robert J. Ursano. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457136.003.0019.

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Nearly everyone experiences a highly stressful or traumatic event during their lifetime. However, individual responses to such events vary widely from person to person. Some people respond with symptoms of anxiety, depression, acute stress, or posttraumatic stress disorder, yet others experience minimal or no psychiatric symptoms after trauma. What makes one person more susceptible and another more resilient to the negative effects of trauma? What are the different adaptive trajectories of trauma survivors and what determines their trajectory? These are some of the questions that are examined in this chapter, which focuses on what is currently known about resilience to stress. The chapter is divided into five sections: definition, prevalence, and measurement of resilience; longitudinal studies on trajectories after trauma exposures; research on factors that are predictive of resilience and different trajectories; interventions that have been developed to increase resilience; and discussion about future directions for research on resilience.
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Who Gets Ptsd?: Issues of Posttraumatic Stress Vulnerability. Charles C. Thomas Publisher, 2006.

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Części książek na temat "Susceptibilité au stress"

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Miroshnychenko, Mykola, Oleksandr Kruglov, Pavlo Nazarok i Stanislav Kovalenko. "Identification of the Structure of Soil Cover by Magnetic Susceptibility". W Soils Under Stress, 57–68. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68394-8_6.

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Mohanty, Prasanna, Vladimir D. Kreslavski, Vyacheslav V. Klimov, Dmitry A. Los, Mamoru Mimuro, Robert Carpentier i Suleyman I. Allakhverdiev. "Heat Stress: Susceptibility, Recovery and Regulation". W Photosynthesis, 251–74. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1579-0_12.

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Laudenslager, Mark L. "Psychosocial Stress and Susceptibility to Infectious Disease". W Viruses, Immunity, and Mental Disorders, 391–402. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1799-9_38.

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Nguyen, Chi Cong, Phuoc Vo, Viet Long Doan, Quang Binh Nguyen, Tien Cuong Nguyen i Quoc Dinh Nguyen. "Assessment of the Effects of Rainfall Frequency on Landslide Susceptibility Mapping Using AHP Method: A Case Study for a Mountainous Region in Central Vietnam". W Progress in Landslide Research and Technology, Volume 1 Issue 2, 2022, 87–98. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-18471-0_7.

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AbstractVietnam’s mountainous regions often encounter landslides, frequently resulting in fatalities, infrastructure damage, and landscape destruction. A landslide susceptibility map is an effective tool for mitigating disaster impacts on hazard-prone areas. This study investigates the applicability of the Analytic Hierarchy Process to produce a landslide susceptibility index. Eight major impact factors are analyzed using SAGA, a GIS-based toolkit, including slopes, aspect, land use, soil type, elevation, distance to road, distance to stream, and antecedent rainfall. Four landslide susceptibility maps are produced corresponding to frequency scenarios of 3-day antecedent rainfall data which is taken from Regional Frequency Analysis (RFA). We assess the modeling performances using Area Under the Curve (AUC) index and the results show that the AHP model has good performance. The findings demonstrate a significant influence of rainfall antecedent conditions on the susceptibility map of landslides in this study area.
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Haycock, J. W., G. Falkous i D. Mantle. "Differential susceptibility of skeletal muscle proteins to free radical-induced oxidative damage in vitro". W Oxidative Stress in Skeletal Muscle, 157–76. Basel: Birkhäuser Basel, 1998. http://dx.doi.org/10.1007/978-3-0348-8958-2_10.

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Jiles, D. C., i P. Garikepati. "Detection of Stress in Steels from Differential Magnetic Susceptibility". W Nondestructive Characterization of Materials, 524–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-84003-6_62.

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Kathirvel, Paramasivam, i Alan L. Archibald. "The Halothane Gene, Leanness and Stress Susceptibility in Pigs". W Animal Models — Disorders of Eating Behaviour and Body Composition, 173–90. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9662-6_10.

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Erzagian, Egy, Wahyu Wilopo i Teuku Faisal Fathani. "Landslide Susceptibility Zonation Using GIS-Based Frequency Ratio Approach in the Kulon Progo Mountains Area, Indonesia". W Progress in Landslide Research and Technology, 115–26. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-44296-4_3.

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AbstractLandslides cause many casualties, environmental damage, property losses, and psychological impacts. Landslides frequently occur in the Kulon Progo Mountains area of Indonesia and are especially triggered by high-intensity rainfall between November and March. Research on landslide susceptibility in the Kulon Progo Mountains area can be a relevant tool to prevent or reduce the risk of landslide potential. Therefore, this research aims to develop a landslide susceptibility map using the frequency ratio (FR) method. Controlling factors, namely, elevation, slope, aspect, lithology, lineament density, distance from streams, distance from roads, land use, and rainfall, were combined with landslide data to develop a landslide susceptibility map by GIS software. Seven hundred and forty-four landslide data points were acquired from field surveys, Google Earth image interpretation, and the Regional Disaster Management Agency (BPBD) of the Kulon Progo, Purworejo, and Magelang regencies. Landslide data were randomly selected for map modeling (80%) and validation (20%). The FR analysis shows that the research area can be classified into four landslide susceptibility zones based on the LSI value: low (LSI 4.05–7.85), moderate (LSI 7.86–9.85), high (LSI 9.86–12.81), and very high (LSI 12.82–19.46). The landslide susceptibility map was validated using the area under the curve (AUC) method, resulting in a success rate of 80.1% and a predictive rate of 81.3%.
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Magiri, Royford, Kaampwe Muzandu, George Gitau, Kennedy Choongo i Paul Iji. "Impact of Climate Change on Animal Health, Emerging and Re-emerging Diseases in Africa". W African Handbook of Climate Change Adaptation, 1835–51. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-45106-6_19.

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AbstractThe threat of climate change and global warming is gaining worldwide recognition. The African continent, because of its size, diversity, and its new status as a “hub” of livestock production, need to gear up to mitigate the possible impacts of climate change on animal health. The aim of this review article is to summarize the current state of knowledge regarding the influence of climate and climate change on the health of food-producing animals. Depending on its intensity and duration, heat stress may directly affect livestock health by causing metabolic disruptions, oxidative stress, and immune suppression, causing increased disease susceptibility, and death. Animal health could also be affected by emergence and re-emergence of vector- and non-vector-borne pathogens that are highly dependent on climatic conditions. The response to these challenges requires community participation in the adaptation of animal production systems to new environments and strengthening the efficiency of veterinary services delivery combined with well-coordinated public health services, since many emerging human diseases are zoonotic.
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Stephenson, Kale J., Yugo Ashida, Jeremy T. Busby i Gary S. Was. "Stress Corrosion Crack Initiation Susceptibility of Irradiated Austenitic Stainless Steels". W Proceedings of the 15th International Conference on Environmental Degradation of Materials in Nuclear Power Systems — Water Reactors, 1187–201. Cham: Springer International Publishing, 2011. http://dx.doi.org/10.1007/978-3-319-48760-1_72.

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Streszczenia konferencji na temat "Susceptibilité au stress"

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Jindal, Rajni, Chahat Kumar, Gaurav Jawla i Harshit Goyal. "Predicting Susceptibility to Covid Stress Using Data Mining". W 2022 International Conference on Emerging Smart Computing and Informatics (ESCI). IEEE, 2022. http://dx.doi.org/10.1109/esci53509.2022.9758247.

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Bayles, Robert A., R. K. Singh Raman, Steven P. Knight i Jy-An Wang. "Evaluating Stress-Corrosion Cracking Susceptibility Using a Torsion Test". W ASME 2005 Pressure Vessels and Piping Conference. ASMEDC, 2005. http://dx.doi.org/10.1115/pvp2005-71782.

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A torsion test has been devised that provides for plane strain constraint in small specimens during fracture toughness testing. This method has been extended for stress-corrosion cracking and a simple torsion load frame has been built to provide for step loading of the specimens. This paper describes using the torsion technique to measure KISCC for aluminum alloy 7075 having two thermo-mechanical treatments.
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Fernandes, Carlos, i Osimar Simiao. "Methodology development for pipeline stress corrosion cracking susceptibility evaluation". W Rio Pipeline Conference and Exhibition. IBP, 2023. http://dx.doi.org/10.48072/2447-2069.rpc.2023.187.

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"Distinguishing Effect of Buffing Operation on Surface Residual Stress Distribution and Susceptibility of 304L SS and 321 SS Welds to Chloride Induced SCC". W Residual Stresses 2018. Materials Research Forum LLC, 2018. http://dx.doi.org/10.21741/9781945291890-22.

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Rund, C. O. "Development of a Miniature Portable X-Ray Instrument for the Non-Destructive Measurement of Stresses". W ASME/JSME 2004 Pressure Vessels and Piping Conference. ASMEDC, 2004. http://dx.doi.org/10.1115/pvp2004-2825.

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Residual surface stresses have been shown to play a critical role in the failure of metallic components through fatigue, stress corrosion cracking, and corrosion fatigue. A method of measuring these residual stresses remotely and nondestructively would improve the ability to assess the potential failure susceptibility of components in service.
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Kuzyk, M. G., K. D. Singer, H. E. Zahn i L. A. King. "Controlling the Second Order Nonlinear Optical Tensor Properties of Poled Films With Stress". W Nonlinear Optical Properties of Materials. Washington, D.C.: Optica Publishing Group, 1988. http://dx.doi.org/10.1364/nlopm.1988.wa3.

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The large electronic second order nonlinear optical susceptibilities of organic molecules can be built into bulk systems by imparting polar orientational order to an ensemble of nonlinear optical dopants. The noncentrosymmetric orientational order required for second order nonlinear optical processes in noncrystalline materials, such as molecule-doped liquid crystals and polymer glasses, has been demonstrated using electric field poling.[1] [2] [3] [4] The relationship between the second order molecular tensor susceptibility and the bulk tensor susceptibility of a polymer doped with optically nonlinear molecules has been recently calculated in the mean field approximation for poled materials under the influence of other bulk ordering forces [5] such as uniaxial stress. [6] Also, it has been previously shown that optical second harmonic generation can be a sensitive probe of molecular order.[7] In this paper, the tensor properties of the second order susceptibility of films poled under uniaxial stress in the direction of the poling field are determined. It is shown that both the magnitude and tensor properties of the second order susceptibility can be continuously varied with poling field and stress.
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Chen, Bo, Michael W. Spindler, David J. Smith i Peter E. J. Flewitt. "Effect of Thermo-Mechanical History on Reheat Cracking in 316H Austenitic Stainless Steel Weldments". W ASME 2010 Pressure Vessels and Piping Division/K-PVP Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/pvp2010-25088.

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Reheat cracking has been observed in the heat affected zone of the 316H austenitic stainless steel thick section weldments during service at a temperature of ∼500°C. This has been attributed to the creep dominated relaxation of the highly triaxial residual stresses. Here the role of thermo-mechanical variables that contribute to the susceptibility of thick section 316H austenitic stainless steel weldments is briefly reviewed. The influence of the plastic strain, carbide precipitation and impurity element segregation on the subsequent creep deformation behaviour and the susceptibility to creep cavitation damage is discussed. A systematically designed experiment which includes these parameters has been undertaken for a 316H austenitic stainless steel. In addition, residual stress profiles have been introduced into cylindrical pre-treated specimens and the relaxation of these profiles with heat treatment has been measured by neutron diffraction. The experimental results are considered with respect to the effect of the microstructure on subsequent creep deformation and stress relaxation. The susceptibility to intergranular brittle fracture is discussed and an attempt is made to correlate the microstructure and stress state factors encountered in the HAZ with the accumulation of the creep cavitation associated with reheat cracking.
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"Early life stress leads to enhanced susceptibility to social stress in adult male C57BL/6 mice". W SYSTEMS BIOLOGY AND BIOINFORMATICS. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/sbb-2019-16.

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Ziomek-Moroz, M., i J. Flis. "Susceptibility of Carbon Steel to Stress Corrosion Cracking in Sodium Hydroxide". W ASME 2005 Pressure Vessels and Piping Conference. ASMEDC, 2005. http://dx.doi.org/10.1115/pvp2005-71783.

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Stress corrosion cracking susceptibility of carbon steel and decarburized steel was studied in 8.5 M sodium hydroxide at 100 °C. Potentiodynamic experiments were performed to determine the potential values to be applied in slow strain rate (ssr) experiments. Optical and scanning electron microcopy were used to investigate the surfaces of corroded samples. Severe intergranular stress corrosion cracking was observed on the carbon steel samples in comparison to the decarburized steel samples.
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Mizutani, T., A. Sato, A. Watanabe, Y. Hamakawa, K. Uemasu, N. Tanabe, S. Sato i T. Hirai. "Susceptibility to Oxidative Stress Characterizes Phenotypes in Murine Model of BPD". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5488.

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Raporty organizacyjne na temat "Susceptibilité au stress"

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Christman. L51577 Prediction of SCC Susceptibility Based on Mechanical Properties of Line Pipe Steels. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), sierpień 1988. http://dx.doi.org/10.55274/r0010278.

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If a relationship between the deformation properties of a line pipe steel and its stress-corrosion cracking resistance can be established, then steels may be selected or designed for improved stress-corrosion resistance, based on their mechanical properties. Benefit: In this research program three line pipe steels, removed from long-term service, were examined to determine if there is a correlation between their mechanical properties and stress-corrosion cracking resistance. The hypothesis was that the steel with the greatest tendency for strain hardening, under cyclic and monotonic stress conditions would also have the highest threshold stress for stress-corrosion crack initiation. This hypothesis was verified by the laboratory experiments, which showed the steel with the greatest tendency for strain hardening to have the highest resistance to stress-corrosion. Two other steels, with distinctly lower resistance to plastic deformation, had lower threshold stresses for stress-corrosion. This observation is consistent with the present concept of stress-corrosion crack growth, which holds that crack tip dissolution, and hence crack propagation, occurs because localized plastic deformation ruptures passive films or prevents film formation resulting in crack growth. Result: The cyclic strain behavior of these three steels is consistent with their monotonic stress-strain curves. Both Steels A and B showed a point of extreme strain as the cyclic stress was increased. Their monotonic stress-strain curves both showed well pronounced yield points above which a considerable strain accompanied a small stress increment (low strain hardening). For both steels the rapid increase in cyclic strain occurred at approximately the elevated temperature yield point (\45 ksi for Steel A
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Colleen Shelton-Davis. Fuel Canister Stress Corrosion Cracking Susceptibility Experimental Results. Office of Scientific and Technical Information (OSTI), marzec 2003. http://dx.doi.org/10.2172/911541.

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Leis. L51838 Cyclic Stress Strain Behavior and SCC Susceptibility of Line Pipe Steels. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), marzec 2001. http://dx.doi.org/10.55274/r0010355.

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This report presents results of a preliminary evaluation of a mechanical property that has the potential to correlate with susceptibility to Stress-corrosion cracking (SCC). This mechanical property measures the evolution of the microplastic response of line pipe steels, which underlies high pH SCC, and is believed by many to be essential for near-neutral SCC. If such a property can be proven to reflect susceptibility to SCC, operators could better contain maintenance costs and specify steels for new pipelines that are inherently resistant to SCC. Limited experimentation using X60 line-pipe steel indicated that the onset, duration, and intensity of microplasticity correlated with electrochemical indicators of SCC for the high-pH cracking environment. These results show sensitivity sufficient to detect the incidence and magnitude of plastic strain that evolves in load-controlled cycling under conditions typical of pipeline service, as well as in histories where such behavior has been exhausted by prior strain hardening. Thus, these data indicate that the protocol evaluated herein is potentially capable of identifying susceptible steels, and has sensitivity of the order needed to discriminate susceptibility between steels. Benefit: While the concept and related experimental techniques hold promise as a screening tool for susceptibility, some experimental issues must be resolved before its use can be conclusively established. These and other related aspects are outlined in the discussion section, and the recommendations section of the report.
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Koch, Beavers i Berry. L51491 Effect of Temperature on Stress-Corrosion Cracking of Pre-Cracked Line-Pipe Steel. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), luty 1985. http://dx.doi.org/10.55274/r0010078.

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Research conducted in the Stress-Corrosion-Cracking Phase of the NG-18 program has demonstrated that a number of factors influence the rate of stress-corrosion cracking. These factors are the susceptibility of the steel, the stress level, and loading amplitude, the chemical environment in contact with the steel surface, the electrochemical potential established on the steel surface, and the temperature of the steel/environment system. The temperature, as was pointed out by Fessler et al., has an effect on crack growth rate, the width of the potential range where stress-corrosion cracking occurs, and the range of strain rates within which stress-corrosion cracking is possible. It was shown that each of these effects can act to decrease the susceptibility of line pipe steel to stress-corrosion cracking. Although the beneficial effects of lowering the temperature on the stress-corrosion cracking behavior of line pipe steels in carbonate/bicarbonate solutions was shown, no data are available concerning the effect on initiation of stress-corrosion cracks. The objective of this study was to determine the effect of temperature on the KISCC and the stress-corrosion crack velocity of line pipe steels. This study was initiated to determine whether lowering the temperature of the environment could prevent a stress-corrosion crack from initiating or could stop existing and propagating cracks.
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Rahman, M. M., i K. N. Strafford. USA09-01 Appraisal and Assessment of Factors Involved in the Maintenance of the Integrity of Pipelines. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), wrzesień 1992. http://dx.doi.org/10.55274/r0011770.

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The experimental work reported in this thesis was aimed at investigating the relative importance of surface-finish and condition on: (1) susceptibility to stress corrosion cracking, and (2) FBE coating performance for X-65 pipeline steel.
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Subramanian, K. STRESS CORROSION CRACKING SUSCEPTIBILITY OF HIGH LEVEL WASTE TANKS DURING SLUDGE MASS REDUCTION. Office of Scientific and Technical Information (OSTI), październik 2007. http://dx.doi.org/10.2172/920661.

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Psaila-Dombrowski, M. J., W. A. Van Der Sluys i B. P. Miglin. GRI-97-0001 Investigation of Pipeline Stress Corrosion Cracking Under Controlled Chemistry Conditions. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), sierpień 1997. http://dx.doi.org/10.55274/r0012043.

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Stress corrosion cracking failures of high-pressure gas transmission pipelines have occurred. Although such failures are infrequent, there is a concern about their potentially catastrophic nature. The susceptibility of a material to this failure is controlled by crack growth kinetics which is governed by the composition of the water at the crack tip, the material composition, temperature, and stress/strain conditions. Hence, there is a need to investigate these parameters to begin to understand and develop predictive capabilities to avoid this phenomenon.
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Denys i Lefevre. L52229 SCC Avoidance in Ethanol Pipelines. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), marzec 2005. http://dx.doi.org/10.55274/r0011363.

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The objectives of the present work were (1) to identify and document a potent stress corrosion cracking environment for carbon steels in fuel-grade ethanol, together with a test protocol for laboratory studies, and (2) to evaluate the SCC susceptibility of pipeline steel Base Metal and welds compared with that of a tank steel known to be susceptible.
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Teysseyre, Sebastien. Irradiation Programs and Test Plans to Assess High-Fluence Irradiation Assisted Stress Corrosion Cracking Susceptibility. Office of Scientific and Technical Information (OSTI), marzec 2015. http://dx.doi.org/10.2172/1177229.

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Bruce i Li. L51666 Surface Preparation and Coatings on the Susceptibility of Line Pipe to Stress-Corrosion Cracking. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), luty 1992. http://dx.doi.org/10.55274/r0010315.

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Within the last decade, a number of gas pipeline companies throughout the world have identified the presence of Stress-Corrosion Cracking (SCC) on their pipelines. While the cause of the SCC resulted from a multitude of factors, there appeared to be a commonality relating to pipeline coatings. Although SCC has occurred under polyethylene/PVC tapes and enamel coatings, not a single incident has been recorded where SCC has occurred on a fusion-bonded-epoxy coated line. The three major factors that were thought to account for the difference in the SCC performance of the various coatings were; (1) resistance of a coating to disbonding, (2) the ability to pass current through the coating and thereby protect the disbonded regions and (3) the surface preparation of the line pipe prior to coating application. These factors were addressed in this three-year program for PRCI. This report examines the relationship between coatings and surface preparation and the likelihood of subsequent stress corrosion cracking. Standard Cathodic disbondment tests, measurements of potential gradients beneath disbonded coating, electrochemical measurements of current passages through coatings and actual SCC tests are described and results reported.
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