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Artykuły w czasopismach na temat "Stimulation cellulaire"
Carmen Martínez, Maria, Corinne Kunzelmann i Jean-Marie Freyssinet. "Remodelage de la membrane plasmique et stimulation cellulaire". médecine/sciences 20, nr 2 (luty 2004): 189–95. http://dx.doi.org/10.1051/medsci/2004202189.
Pełny tekst źródłaVallée, Alexandre. "Activation de la glycolyse aérobie par la voie canonique WNT/β-caténine". médecine/sciences 34, nr 4 (kwiecień 2018): 326–30. http://dx.doi.org/10.1051/medsci/20183404013.
Pełny tekst źródłaMollard, Patrice. "L’hypophyse dévoilée : du couplage stimulation-sécrétion aux réseaux cellulaires câblant la glande". Biologie Aujourd’hui 216, nr 3-4 (2022): 83–87. http://dx.doi.org/10.1051/jbio/2022021.
Pełny tekst źródłaSEYER, P., S. GRANDEMANGE, L. PESSEMESSE, F. CASAS, G. CABELLO i C. WRUTNIAK-CABELLO. "L’activité mitochondriale est un régulateur majeur de la différenciation des myoblastes et de l’expression des isoformes de myosine". INRAE Productions Animales 19, nr 4 (13.09.2006): 279–86. http://dx.doi.org/10.20870/productions-animales.2006.19.4.3495.
Pełny tekst źródłaGruffat, Henri, i Evelyne Manet. "Co-infection EBV/KSHV". médecine/sciences 34, nr 1 (styczeń 2018): 79–82. http://dx.doi.org/10.1051/medsci/20183401017.
Pełny tekst źródłaKahn, A. "MAP-kinase, un intégrateur possible des différentes voies de stimulation de la division cellulaire". médecine/sciences 6, nr 4 (1990): 392. http://dx.doi.org/10.4267/10608/4154.
Pełny tekst źródłaDumond, Aurore, Luc Demange i Gilles Pagès. "Les neuropilines". médecine/sciences 36, nr 5 (maj 2020): 487–96. http://dx.doi.org/10.1051/medsci/2020080.
Pełny tekst źródłaGur, Serap, i Wayne J. G. Hellstrom. "Activation of P2Y1 and P2Y2 nucleotide receptors by adenosine 5′-triphosphate analogues augmented nerve-mediated relaxation of human corpus cavernosum". Canadian Urological Association Journal 3, nr 4 (1.05.2013): 314. http://dx.doi.org/10.5489/cuaj.1127.
Pełny tekst źródłaLucas, P. J., C. V. Bare i R. E. Gress. "The human anti-murine xenogeneic cytotoxic response. II. Activated murine antigen-presenting cells directly stimulate human T helper cells." Journal of Immunology 154, nr 8 (15.04.1995): 3761–70. http://dx.doi.org/10.4049/jimmunol.154.8.3761.
Pełny tekst źródłaLin, James C. A., Jimmy K. Li i Walter H. Chang. "Signal Transduction Pathway of Ultrasound Stimulation on Osteoblasts(Cellular & Tissue Engineering)". Proceedings of the Asian Pacific Conference on Biomechanics : emerging science and technology in biomechanics 2004.1 (2004): 87–88. http://dx.doi.org/10.1299/jsmeapbio.2004.1.87.
Pełny tekst źródłaRozprawy doktorskie na temat "Stimulation cellulaire"
Bidouba-Sanvany, Doll-Spencerh. "Simulation de l'interaction thermo-acousto-fluidique en puits : application à la stimulation cellulaire". Electronic Thesis or Diss., Université de Lorraine, 2023. http://www.theses.fr/2023LORR0279.
Pełny tekst źródłaSurface acoustic waves (SAW) are currently being used to study their interaction with biological particles to perform studies of cell proliferation, growth, death and migration. The effect of shear stress on cells has been shown to play an important role in cell modification, providing further details on their function and potential changes. Stamp et al. present an ultrasound approach to dynamic stimulation and tissue healing of SaOs-2 osteoblastic cells using surface acoustic waves (SAW) on a chip. These studies are performed without unwanted side effects such as increased substrate temperature or nutrient flux associated with SAW. These results demonstrate that the dynamic mechanical and electrical stimulation induced by surface acoustic waves directly promotes cell growth and thus rapid tissue regeneration. S. Brugger presents studies on in vitro wound healing and cell growth under the influence of radiofrequency (rf) cell stimuli. These stimuli are generated either by piezoactive surface acoustic waves (SAW) or by electric fields generated by microelectrodes. His results show that vibratory stimulation of cells based on surface acoustic waves can be an alternative to conventional ultrasound treatment. Shear stresses acting on the cells are responsible for the various changes observed after acoustic wave stimulation, but it is impossible to determine these stresses experimentally, so a simulation approach is necessary. The goal of this thesis is to develop a simulation that will allow us to understand the phenomenon of Rayleigh/liquid interaction in a microchannel or droplet and to quantify the stresses exerted on biological particles during surface acoustic wave stimulation, as well as to carry out an optimization study. The study investigated the effects of different frequencies on blood cells (circulating) and bone cells (adherent) using a vertical probe immersed in a well with a frequency of 20 MHz and 40 MHz, respectively. Additionally, the research took into consideration the liquid heating
GUFFLET, ESCHWEGE VALERIE. "Les anticorps antiphospholipides : lien physiopathologique entre la stimulation cellulaire et les reponses procoagulantes et immunitaires". Université Louis Pasteur (Strasbourg) (1971-2008), 1998. http://www.theses.fr/1998STR13064.
Pełny tekst źródłaBernard, Isabelle. "Phénomènes d'activation cellulaire et plasmatique de l'hémostase lors des traitements de stimulation pour fécondation in vitro". Montpellier 1, 1994. http://www.theses.fr/1994MON11125.
Pełny tekst źródłaXia, Rong. "Étude des effets de la stimulation électrique à haute fréquence dans un modèle cellulaire in vitro". Université Joseph Fourier (Grenoble), 2005. https://tel.archives-ouvertes.fr/tel-00009865.
Pełny tekst źródłaA system for electrical stimulation on cells lines in vitro was optimized for studying cellular and molecular mechanism of high frequency electrical stimulation. Two cells lines (GH3 and PC12) were analysed at the transcriptomic and proteomic level as well as for the impact on hormonal secretion and neurotransmitter release. We compared PRL secretion of GH3 cell by HFS, LFS and dopamine as well as catecolamine (DA, AD and NA) secretion of PC12 cell treated by HFS, LFS and 6-OHDA. Proteomic synthesis of stimulated cells were analysed by 35S methionine incorporation and by SELDI-TOF-MS. At last we have investigated modifications of gene expression of stimulated cells using DNA nylon microarray based on long oligonucleotides and radioactive detection. First experiment demonstrated that HFS induced a significant decrease of the amount of prolactin to a level comparable to the level obtained with dopamine, one well known inhibitor of prolactin. Similarly, the amount of catecholamines detected in the cell culture medium was significantly down regulated. Transcriptomic data manifested the existence of characteristic expression profiles for high frequency electrical stimulation. About 100 discriminatory genes were individualized upon HFS involved in the proteomic synthesis, calcium signalisation and cellular energetic. We confirmed the impact of HFS on protein synthesis with SELDI-TOF technique and methionine incorporation. One original HFS mechanism has been validated, involving protein synthesis neutralisation in the reacted inactivation of neuronal structures. Implication of calcium signalisation and PKC pathway were also suggested
Carras, Sylvain. "Rôles de la stimulation chronique du TCR et de la reprogrammation cellulaire dans les lymphomes T périphériques". Thesis, Lyon, 2018. http://www.theses.fr/2018LYSE1322/document.
Pełny tekst źródłaPeripheral T-cell lymphomas (PTCL) are rare non Hodgkin malignant lymphomas emerging from mature T or NK cells. PTCL are highly heterogeneous and mainly misunderstood. As several evidences pointed the potential role of TCR chronic stimulation in human T-cell lymphomagenesis, we developed a murine model based on chronic TCR stimulation to address this question. In this model, transfer of p53-/- T-cells into T-cell deficient mice (CD3e-/-) triggered PTCL development in 60% of cases with a median survival of 230 days while transfer of wt T-cells in CD3e-/- mice did not lead to PTCL development. These PTCL exhibited an effector-memory phenotype CD62LLo-CD44hi-CD122lo-CD25lo associated with a dramatic downregulation of TCR pathway genes expression consistent with a chronic TCR stimulation highlighting it’s implication in lymphomagenesis. The analysis of these PTCL revealed that a large majority of cases (80%) do not depend anymore on TCR stimulation for their growth and survival and that they acquire innate-like features with expression of inhibitory NKR (NKiR) and activating NK receptors (NKaR) as well as the adaptor proteins DAP12 or FceRIg. Expression of these receptors is associated with the expression of SYK and PLC?2, which are classical key effectors downstream of NKaR. We show that these NKaR are functional and can mediate TCR-independent activation in mPTCL and that this signaling is involved in cell survival/proliferation as in vivo blockade of NKG2D and NKp46 delays PTCL development in PTCL transplantation experiments. In parallel, we studied NKR, Syk and PLCg2 expression in human PTCL and found that some entities express a large range of these receptors as well as Syk and PLCg2, suggesting similar lymphomagenesis mechanisms in some human PTCL
Gustave-Dit-Duflo, Sylvie. "Adaptation et restauration des fonctions vestibulaires : Analyses comportementale et cellulaire après lésion ou stimulation vestibulaire chez l'animal". Montpellier 2, 1998. http://www.theses.fr/1998MON20128.
Pełny tekst źródłaDehez, Stéphanie. "Stimulation des voies JNK et P38MAPK par les peptides gastriniques : mécanismes d'activation et rôle dans la prolifération cellulaire". Toulouse 3, 2001. http://www.theses.fr/2001TOU30093.
Pełny tekst źródłaZanin, Carole. "Ecotaxie des cellules de la lignée B spécifiques d'antigènes microbiens après stimulation par voie orale". Nancy 1, 1994. http://www.theses.fr/1994NAN10010.
Pełny tekst źródłaParaskevopoulos, Yannis. "Utilisation des enveloppes cellulaires de levure pour la stimulation de la fermentation malolactique : interprétation de leur mode d'action". Bordeaux 2, 1988. http://www.theses.fr/1988BOR20029.
Pełny tekst źródłaDing, Can. "The influence of Notch over-stimulation on muscle stem cell quiescence versus proliferation, and on muscle regeneration". Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066399/document.
Pełny tekst źródłaMuscle stem cell transplantation possesses great potential for long-term repair of dys-trophic muscle. However expansion of muscle stem cells ex vivo significantly reduces their engraftment efficiency since the myogenic potential is dramatically lost in culture. The Notch signaling pathway has emerged as a major regulator of muscle stem cells (MuSCs) and it has recently been discovered that high Notch activity is crucial for maintaining stemness in MuSCs. This feature might be exploited and developed into a novel therapeutic approach.Murine MuSCs were freshly isolated and seeded on culture vessels coated with Dll1-Fc, which fused Delta-like-1 extracellular domain with human Fc, to activate Notch sig-naling and with human IgG as a control. The rAAV gene delivery system was em-ployed to express Dll1 in murine muscles. P3 mice were treated with AAV for 3 weeks and 6 weeks to investigate the effect of Dll1 during postnatal development. To investi-gate the regeneration process, AAV were injected into mdx muscles whereas wild-type mice were used as control.Higher potential stemness (marked by Pax7 positivity) was observed in MuSCs grow-ing on a Dll1-Fc surface as compared to their counterparts on the control surface, while their proliferation rate was reduced. During postnatal development, overstimulation of Notch signaling by Dll1 on the mus-cle fibers was able to enlarge the Pax7+ cell pool, while also resulting in decreased muscle mass and smaller muscle fibers without affecting the accretion of myonuclei into the fiber. In quiescent (wild-type) MuSCs, overstimulation of Notch signaling did not have any discernible effect. Overexpression of Dll1 in mdx muscle decreased the muscle mass and enlarged the muscle stem cell pool, while muscle regeneration re-mained unaffected. By investigating Notch stimulation in MuSCs both in vitro and in vivo, we demonstrate that high Notch activity preserves stemness via inhibition of MuSCs proliferation and myogenic differentiation. Our findings point out that the Dll1 molecule, as a canonical Notch ligand, might have a therapeutic potential in cell-based therapies against muscu-lar dystrophies
Książki na temat "Stimulation cellulaire"
1945-, Dexter T. Michael, Garland John M. 1942- i Testa Nydia G. 1938-, red. Colony-stimulating factors: Molecular and cellular biology. New York: M. Dekker, 1990.
Znajdź pełny tekst źródła1942-, Garland John M., Quesenberry Peter J i Hilton Douglas J. 1964-, red. Colony-stimulating factors: Molecular and cellular biology. Wyd. 2. New York: M. Dekker, 1997.
Znajdź pełny tekst źródłaGregory, Bock, i Goode Jamie, red. The molecular basis of cellular defence mechanisms. Chichester: Wiley, 1997.
Znajdź pełny tekst źródłaChevanne, Marta, i Riccardo Caldini. Immagini di Istopatologia. Florence: Firenze University Press, 2007. http://dx.doi.org/10.36253/978-88-5518-023-8.
Pełny tekst źródłaPoljsak, Borut, Irina Milisav, Thomas N. Seyfried, Dominic P. D'Agostino i Angela M. Poff. Reduction of Sporadic Malignancies by Stimulation of Cellular Repair Systems and by Targeting Cellular Energy Metabolism. Nova Science Publishers, Incorporated, 2017.
Znajdź pełny tekst źródłaGarland, John M. Colony-Stimulating Factors: Molecular and Cellular Biology, Second Edition,. Taylor & Francis Group, 2020.
Znajdź pełny tekst źródłaGarland, John M. Colony-Stimulating Factors: Molecular and Cellular Biology, Second Edition,. Taylor & Francis Group, 2020.
Znajdź pełny tekst źródłaGarland, John M. Colony-Stimulating Factors: Molecular and Cellular Biology, Second Edition,. Taylor & Francis Group, 2020.
Znajdź pełny tekst źródłaGarland, John M. Colony-Stimulating Factors: Molecular and Cellular Biology, Second Edition,. Taylor & Francis Group, 2020.
Znajdź pełny tekst źródłaGuo, Liang. Principles of Electrical Neural Interfacing: A Quantitative Approach to Cellular Recording and Stimulation. Springer International Publishing AG, 2022.
Znajdź pełny tekst źródłaCzęści książek na temat "Stimulation cellulaire"
Carlstrom, Lucas P. "Chapter 22 Molecular and Cellular Neuromodulation for Central Nervous System Injury and Regeneration". W Deep Brain Stimulation, 415–54. Pan Stanford Publishing Pte. Ltd. Penthouse Level, Suntec Tower 3 8 Temasek Boulevard Singapore 038988: Pan Stanford Publishing, 2016. http://dx.doi.org/10.1201/9781315364759-23.
Pełny tekst źródłaWatanabe, A. M., F. Green i Z. Ahmad. "Studies on the cellular mechanisms of action of positive and negative inotropic agents". W Inotropic Stimulation and Myocardial Energetics, 19–22. Heidelberg: Steinkopff, 1989. http://dx.doi.org/10.1007/978-3-662-07908-9_3.
Pełny tekst źródłade la Higuera, Luis, Martín López-García, Grant Lythe i Carmen Molina-París. "IL-2 Stimulation of Regulatory T Cells: A Stochastic and Algorithmic Approach". W Modeling Cellular Systems, 81–105. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-45833-5_4.
Pełny tekst źródłaAlsner, J., E. Kjeldsen, J. Q. Svejstrup, K. Christiansen i O. Westergaard. "Stimulation of Human DNA Topoisomerase I By Protein Kinase C". W Cellular Regulation by Protein Phosphorylation, 429–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75142-4_52.
Pełny tekst źródłaNissen, P. "Stimulation of Somatic Embryogenesis in Carrot by Ethylene". W Cellular and Molecular Aspects of the Plant Hormone Ethylene, 359–64. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-017-1003-9_80.
Pełny tekst źródłaMeyer-Hermann, Michael, Frido Erler i Gerhard Soff. "Presynaptic Calcium Dynamics of Neurons in Answer to Various Stimulation Protocols". W Function and Regulation of Cellular Systems, 63–71. Basel: Birkhäuser Basel, 2004. http://dx.doi.org/10.1007/978-3-0348-7895-1_7.
Pełny tekst źródłaKather, H. "Beta Adrenergic Receptor Mediated Stimulation of Adenine Nucleotide Catabolism and Purine Release in Human Adipocytes". W Purines in Cellular Signaling, 120–25. New York, NY: Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3400-5_20.
Pełny tekst źródłaRavingerová, Tanya, Nigel J. Pyne i James R. Parratt. "Ischaemic preconditioning in the rat heart: The role of G-proteins and adrenergic stimulation". W Cellular Interactions in Cardiac Pathophysiology, 123–28. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-2005-4_16.
Pełny tekst źródłaHaneda, Takashi, i Paul J. McDermott. "Stimulation of ribosomal RNA synthesis during hypertrophic growth of cultured heart cells by phorbol ester". W Molecular Mechanisms of Cellular Growth, 169–77. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3886-8_21.
Pełny tekst źródłaDartsch, Peter C., i Eberhard Betz. "Cellular and Cytoskeletal Response of Vascular Cells to Mechanical Stimulation". W Medical Textiles for Implantation, 193–218. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75802-7_14.
Pełny tekst źródłaStreszczenia konferencji na temat "Stimulation cellulaire"
Steward, Robert L., Chao-Min Cheng i Philip R. LeDuc. "Probing Dynamic Responses of the Extracellular Matrix to Coupled Mechanical and Chemical Inputs". W ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19206.
Pełny tekst źródłaMcGarry, Patrick, Robert M. McMeeking, Anthony G. Evans i Vikram S. Deshpande. "Modeling the Active Response of Cells to Mechanical Stimulation". W ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193071.
Pełny tekst źródłaAl-Shamasi, Al-Anood, Meram Elsayed, Nabeel Abdulrahman, Jensa Joseph i Fatima Mraiche. "The Cardiovascular benefits of Empagliflozin, a Sodium Glucose Cotransporter Inhibitor: Is NHE1 a viable target?" W Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0228.
Pełny tekst źródłaTaylor, Rebecca, Oscar Abilez, Feng Cao, Joe Wu, Chengpei Xu, Christopher Zarins i Beth Pruitt. "Pulsatile Pressure System for Cellular Mechanical Stimulation". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176413.
Pełny tekst źródłaTakeda, Shota, Yoshihiro Tomita i Eiji Nakamachi. "Cellular Automaton and Finite Element Hybrid Simulation to Predict Axonal Extension Enhancement of Nerve Cell Under Mechanical Stimulation". W ASME 2018 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/imece2018-86653.
Pełny tekst źródłaCheng, Chao-Min, i Philip R. LeDuc. "Effects of Local Mechanical Stimulation on Cellular Behavior". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176082.
Pełny tekst źródłaSteward, Robert L., Chao-Min Cheng i Philip R. LeDuc. "Probing Nonlinear Cellular Responses to Integrated Mechanical Signals Through Examining Cell Alignment". W ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19205.
Pełny tekst źródłaYang, Jui-Ming, i Philip R. LeDuc. "Three-Dimensional Laminar Flow for Localized Cellular Stimulation". W ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61643.
Pełny tekst źródłaJeon, Jong Heon, Tae Kyung Kim, So Hee Park, Jung Wook Shin i Ok Chan Jeong. "Experimental Study on Cytoplasmic Calcium Oscillation in MG-63 Cells Induced by Pressure-Driven Fluid Flow". W ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-11121.
Pełny tekst źródłaWechsler, Marissa E., Courtney M. Creecy, Christine F. O’Neill i Rena Bizios. "Effects of Electrical Stimulation on Select Functions of Bone Cells". W ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80367.
Pełny tekst źródłaRaporty organizacyjne na temat "Stimulation cellulaire"
Naim, Michael, Andrew Spielman, Shlomo Nir i Ann Noble. Bitter Taste Transduction: Cellular Pathways, Inhibition and Implications for Human Acceptance of Agricultural Food Products. United States Department of Agriculture, luty 2000. http://dx.doi.org/10.32747/2000.7695839.bard.
Pełny tekst źródłaFunkenstein, Bruria, i Cunming Duan. GH-IGF Axis in Sparus aurata: Possible Applications to Genetic Selection. United States Department of Agriculture, listopad 2000. http://dx.doi.org/10.32747/2000.7580665.bard.
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