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Artykuły w czasopismach na temat "STIM1"
WILLIAMS, Richard T., Shehnaaz S. M. MANJI, Nigel J. PARKER, Manuela S. HANCOCK, Leonie van STEKELENBURG, Jean-Pierre EID, Paul V. SENIOR i in. "Identification and characterization of the STIM (stromal interaction molecule) gene family: coding for a novel class of transmembrane proteins". Biochemical Journal 357, nr 3 (25.07.2001): 673–85. http://dx.doi.org/10.1042/bj3570673.
Pełny tekst źródłaCully, Tanya R., Joshua N. Edwards, Oliver Friedrich, D. George Stephenson, Robyn M. Murphy i Bradley S. Launikonis. "Changes in plasma membrane Ca-ATPase and stromal interacting molecule 1 expression levels for Ca2+ signaling in dystrophic mdx mouse muscle". American Journal of Physiology-Cell Physiology 303, nr 5 (1.09.2012): C567—C576. http://dx.doi.org/10.1152/ajpcell.00144.2012.
Pełny tekst źródłaYoshikawa, Soichiro, Masatsugu Oh-hora, Ryota Hashimoto, Toshihisa Nagao, Louis Peters, Mayumi Egawa, Takuya Ohta i in. "Pivotal role of STIM2, but not STIM1, in IL-4 production by IL-3–stimulated murine basophils". Science Signaling 12, nr 576 (9.04.2019): eaav2060. http://dx.doi.org/10.1126/scisignal.aav2060.
Pełny tekst źródłaSkibinska-Kijek*, Anna, Marta Wisniewska, Joanna Gruszczynska-Biegala, Axel Methner i Jacek Kuznicki. "Immunolocalization of STIM1 in the mouse brain". Acta Neurobiologiae Experimentalis 69, nr 4 (31.12.2009): 413–28. http://dx.doi.org/10.55782/ane-2009-1753.
Pełny tekst źródłaSkopin, Anton Yu, Andrey D. Grigoryev, Lyubov N. Glushankova, Alexey V. Shalygin, Guanghui Wang, Viktor G. Kartzev i Elena V. Kaznacheyeva. "A Novel Modulator of STIM2-Dependent Store-Operated Ca2+ Channel Activity". Acta Naturae 13, nr 1 (15.03.2021): 140–46. http://dx.doi.org/10.32607/actanaturae.11269.
Pełny tekst źródłaChung, Steve, MengQi Zhang i Peter Stathopulos. "The 2β Splice Variation Alters the Structure and Function of the Stromal Interaction Molecule Coiled-Coil Domains". International Journal of Molecular Sciences 19, nr 11 (25.10.2018): 3316. http://dx.doi.org/10.3390/ijms19113316.
Pełny tekst źródłaKim, Soo J., Roland R. Roy, Hui Zhong, Hideki Suzuki, Lusine Ambartsumyan, Fadia Haddad, Kenneth M. Baldwin i V. Reggie Edgerton. "Electromechanical stimulation ameliorates inactivity-induced adaptations in the medial gastrocnemius of adult rats". Journal of Applied Physiology 103, nr 1 (lipiec 2007): 195–205. http://dx.doi.org/10.1152/japplphysiol.01427.2006.
Pełny tekst źródłaSkobeleva, Ksenia, Alexey Shalygin, Elena Mikhaylova, Irina Guzhova, Maria Ryazantseva i Elena Kaznacheyeva. "The STIM1/2-Regulated Calcium Homeostasis Is Impaired in Hippocampal Neurons of the 5xFAD Mouse Model of Alzheimer’s Disease". International Journal of Molecular Sciences 23, nr 23 (26.11.2022): 14810. http://dx.doi.org/10.3390/ijms232314810.
Pełny tekst źródłaGarcia-Alvarez, Gisela, Bo Lu, Kenrick An Fu Yap, Loo Chin Wong, Jervis Vermal Thevathasan, Lynette Lim, Fang Ji i in. "STIM2 regulates PKA-dependent phosphorylation and trafficking of AMPARs". Molecular Biology of the Cell 26, nr 6 (15.03.2015): 1141–59. http://dx.doi.org/10.1091/mbc.e14-07-1222.
Pełny tekst źródłaWalsh, Ciara M., Michael Chvanov, Lee P. Haynes, Ole H. Petersen, Alexei V. Tepikin i Robert D. Burgoyne. "Role of phosphoinositides in STIM1 dynamics and store-operated calcium entry". Biochemical Journal 425, nr 1 (14.12.2009): 159–68. http://dx.doi.org/10.1042/bj20090884.
Pełny tekst źródłaRozprawy doktorskie na temat "STIM1"
Walsh, Ciara. "The regulation of STIM1 translocation to the plasma membrane". Thesis, University of Liverpool, 2010. http://livrepository.liverpool.ac.uk/1482/.
Pełny tekst źródłaTroupes, Constantine. "The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction". Diss., Temple University Libraries, 2016. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/403786.
Pełny tekst źródłaPh.D.
Increases in cardiac afterload caused by disease conditions results in remodeling of heart structure by hypertrophy and alterations in the molecular regulation of contractile performance. These adaptations can be regulated by various Ca2+-dependent signaling processes. STIM1 is an important regulator of Ca2+ signaling in different cell types by sensing endoplasmic reticular Ca2+ levels and coupling to plasma membrane Orai channels. The role of STIM1 in heart is not well understood, given the robust Ca2+ regulatory machinery present within cardiac myocytes. Previous reports indicate that STIM1 may play a role in regulation of cardiac hypertrophy. The goal of this work is to understand how STIM1 can affect contractile Ca2+ regulation in normal and diseased myocytes. We induced cardiac hypertrophy by slow progressive pressure overload in adult cats. Isolated adult feline ventricular myocytes (AFMs) exhibited increased STIM1 expression and activity, which correlated with altered Ca2+ handling. Use of BTP2 to block Orai channels resulted in a reduction of action potential (AP) duration and diastolic spark rate of hypertrophied myocytes, without affecting myocytes from sham-operated animals. Overexpressed STIM1 in cultured AFMs caused persistent Ca2+ influx that resulted in increased diastolic spark rates and prolonged APs, similar to myocytes from banded animals. STIM1 mediated Ca2+ influx could load the sarcoplasmic reticulum and activated CaMKII, which increased spark rates and lead to spontaneous APs. Importantly, STIM1 operated by associating with Orai channels because these effects could be blocked with either BTP2 or with a dominant negative Orai construct. Prolonged Ca2+ entry through this pathway eventually causes cell death. In conclusion, the work presented in this thesis establishes a role for STIM1-Orai in contractile Ca2+ regulation.
Temple University--Theses
Gueder, Nahla. "sp²-Iminosugar-glucosidases inhibitor 1-C-octyl-2-oxa-3-oxocastanospermine - induced antiproliferative, apoptotic and necrotic effects in breast cancer cells via targeting GRP78, Stim1 and Orai1". Thesis, Amiens, 2018. http://www.theses.fr/2018AMIE0033/document.
Pełny tekst źródłaAlteration in glycosylation pattern is one of the hallmarks of breast cancer. The levels and the abnormal expressions of glycan were found in breast cancer patients. Glycosylation defect can affect different glycosylated proteins which are implicated in cancerogenesis. Changes in intracellular Ca2+ levels can regulate different cellular processes. SOC channels are implicated in breast cancer proliferation, migration and survival. CO-OCS is a new glycosylation inhibitor with more selectivity toward theα- glucosidases exhibited anti-cancer activities in breast cancer cells without affecting the normal mammary cells. The objective of my thesis is investigating the related molecular mechanisms by which CO-OCS induced its anti-tumour effects.CO-OCS impaired breast cancer migration through decrease β1-integrin expression and the activation of FAK and ERK1/2 signalling pathways. CO-OCS also induced anti-migratory effect via Stim1 protein expression down-regulation leading to inhibition of SOCE. Additionally, CO-OCS affected the expression of both Orai1 and Stim1 proteins leading to anti-proliferative effects and cell cycle arrest in G1 and G2/M phase respectively. Moreover, CO-OCS affected the expression of Stim1 at the protein level without affecting its transcript level. GRP78 implicated in CO-OCS apoptotic death. The expression of Stim1 regulated the apoptosis induced by CO-OCS via modulating GRP78 expression. Orai1 down-regulation promoted CO-OCS necrotic effect. CO-OCS induced ER- calcium depletion due to increase in ER calcium leak via the Translocon; Anisomycin (Translocon inhibitor) decreased the apoptosis induced by CO-OCS. In conclusion, these results show that in breast cancer, by targeting Stim1, Orai1 and GRP78, CO-OCS reduced cell proliferation and induced apoptosis and necrosis cell death. Stim1 favours CO-OCS apoptotic effect while Orai1 protected from necrosis induced by CO-OCS. The inhibition of Translocon decreased CO-OCS apoptotic cell death via restoring the ER calcium homeostasis
Frappier, Maude. "MURC est un partenaire d’interaction de STIM1 impliqué dans la signalisation calcique". Mémoire, Université de Sherbrooke, 2015. http://hdl.handle.net/11143/8332.
Pełny tekst źródłaLur, Gyorgy. "STIM1, Orai1 and store operated calcium entry in pancreatic acinar cells". Thesis, University of Liverpool, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539501.
Pełny tekst źródłaSaliba, Youakim. "Identification des partenaires de STIM1 dans le cœur normal et hypertrophié". Paris 6, 2012. http://www.theses.fr/2012PA066541.
Pełny tekst źródłaWe previously showed an important role of STIM1 in cardiac hypertrophy; however, the identity of the channels responsible for the STIM1 dependent pro-hypertrophic Ca2+ entry in the rat ventricular myocytes remains to be elucidated. In this study we developed a new method of myocardial non viral gene delivery, by using the combination of ultrasound energy (USE), liposomes and high pressure injections to the rat heart. Due to its simplicity, low toxicity and low immunogenicity, this method produced sufficient number of transfected cells to perform biochemical experiments and single cell physiological measurements. We then characterized the expression profile of TRPCs and ORAIs proteins in both normal and hypertrophied ventricular myocytes, and found an upregulation of TRPC1 in hypertrophied cells. We further used the new gene delivery method to identify and screen for the STIM1 associated channel candidates via RNA interference. We identified TRPC5 as a non-selective Ca2+ channel that operates constitutively in basal conditions with increased activity in cardiac hypertrophy, as well as ORAI3 that functions in both modes: SOCE and constitutive basal Ca2+ entry in concordance with TRPC5. We developed an efficient non-viral cardiac gene delivery which we used to elucidate TRPC5 and ORAI3 as new voltage independent STIM1 regulated Ca2+ channels in the ventricular rat myocytes
Premsler, Thomas [Verfasser], Albert [Akademischer Betreuer] Sickmann i Jan Georg [Gutachter] Hengstler. "Mass spectrometry based interaction study of the STIM1 and VASP proteins : (Massenspektrometrie-basierte Interaktionsstudie der Proteine STIM1 und VASP) / Thomas Premsler. Betreuer: Albert Sickmann. Gutachter: Jan Georg Hengstler". Dortmund : Universitätsbibliothek Dortmund, 2012. http://d-nb.info/1110892373/34.
Pełny tekst źródłaCao, Hang [Verfasser]. "Effect of STIM1/2 and of Ceritinib on Platelet Function / Hang Cao". Tübingen : Universitätsbibliothek Tübingen, 2021. http://d-nb.info/1234450763/34.
Pełny tekst źródłaMukherjee, Sreya. "Applications of Molecular Modelling and Structure Based Drug Design in Drug Discovery". Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6331.
Pełny tekst źródłaMaues, de Paula André. "Pour une meilleure compréhension de la myopathie à agrégats tubulaires". Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM5056.
Pełny tekst źródłaMyopathy with tubular aggregates (MTA) is a rare disease characterized by the presence of tubular aggregates in muscle biopsy. We found 15 cases of MAT in our registry including 13987 muscle biopsies performed over 35 years. Among them, there was a family of three patients that did not fit any of the previously described clinical groups of MTA, since they were asymptomatic with isolated hyperCKemia and positive contracture test. Our works revealed heterozygous mutations in the gene STIM1. These mutations localized in the gene portion that codes for the intraluminal EF-hand domain, leads to a constitutive activation of STIM1 with SOCE mecanism enhancement and consequent increase of the Ca2+ entry which was demonstrated using transfected myoblasts. We also revealed the mutation p.Arg304Trp in the coding sequencing of the CC1 domain of STIM1, as the cause of Stormorken syndrome. This fact increases the phenotypical spectra of the mutations for this gene.The results of our proteomic analysis show that the proteostasis in MTA is disturbed, because the proteome profile is different in total muscle of the patients and in their tubular aggregates when compared to controls. The enriched proteins belong to the biological pathways linked to ionic homeostasis, membrane systems of the triads and the exosome, and to the mitochondrial metabolism.Our works bring perspectives for the continuation of our studies, in order to better understand the physiopathology of the myopathy with tubular aggregates and propose efficacious therapeutic solutions
Książki na temat "STIM1"
Stimm, Thomas. Thomas Stimm: Keramik 1987-2001. Wien: Kunst Bundeskanzleramt, 2002.
Znajdź pełny tekst źródłaHanson, Sten. Det praktiska tonsätteriets historia: Föreningen svenska tonsättare genom 75 år. [Stockholm]: Edition Reimers, 1993.
Znajdź pełny tekst źródłaPredi, Renzo. Metodi di stima delle strutture familiari. Bologna: CLUEB, 1991.
Znajdź pełny tekst źródłaI metodi di stima nella progettazione. Roma: Gangemi, 1985.
Znajdź pełny tekst źródłaHeusser, Pierre. Stimm- und Wahlrecht für Ausländerinnen und Ausländer. Zürich: Schulthess, 2001.
Znajdź pełny tekst źródłaSonderforschungsbereich 447 "Kulturen des Performativen.", red. Stimm-Welten: Philosophische, medientheoretische und ästhetische Perspektiven. Bielefeld: Transcript, 2009.
Znajdź pełny tekst źródłaAmodio, Fabia Peschitz. No, se non hai stima di me--. Pasian di Prato (Udine): Campanotto, 2004.
Znajdź pełny tekst źródłaStimm, Oswald. Oswald Stimm: Der Tänzer in der Zeit. Gumpoldskirchen: D.E.A. Verlag, 2016.
Znajdź pełny tekst źródłaPatent, copyright & trademark / by Richard Stim. Berkeley, CA: Nolo, 2010.
Znajdź pełny tekst źródłaM, Bonnor G., i Pacific Forestry Centre, red. A guide to the STIM growth model. Victoria, B.C: Pacific Forestry Centre, 1995.
Znajdź pełny tekst źródłaCzęści książek na temat "STIM1"
Jabbari, Parnian, i Nima Rezaei. "STIM1 Deficiency". W Genetic Syndromes, 1–4. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-66816-1_100-1.
Pełny tekst źródłaFrischauf, Irene, Marc Fahrner, Isaac Jardín i Christoph Romanin. "The STIM1: Orai Interaction". W Advances in Experimental Medicine and Biology, 25–46. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26974-0_2.
Pełny tekst źródłaChoi, Seok, Jozsef Maleth, Archana Jha, Kyu Pil Lee, Min Seuk Kim, Insuk So, Malini Ahuja i Shmuel Muallem. "The TRPCs–STIM1–Orai Interaction". W Handbook of Experimental Pharmacology, 1035–54. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-05161-1_13.
Pełny tekst źródłaYee, Christina. "Calcium Channel Defects (STIM1 and ORAI1)". W Encyclopedia of Medical Immunology, 86–91. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4614-8678-7_176.
Pełny tekst źródłaYee, Christina. "Calcium Channel Defects (STIM1 and ORAI1)". W Encyclopedia of Medical Immunology, 1–6. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-9209-2_176-1.
Pełny tekst źródłaBodnar, Dora, Woo Young Chung, Dongki Yang, Jeong Hee Hong, Archana Jha i Shmuel Muallem. "STIM-TRP Pathways and Microdomain Organization: Ca2+ Influx Channels: The Orai-STIM1-TRPC Complexes". W Store-Operated Ca²⁺ Entry (SOCE) Pathways, 139–57. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57732-6_8.
Pełny tekst źródłaWoo, Jin Seok, Sonal Srikanth i Yousang Gwack. "Modulation of Orai1 and STIM1 by Cellular Factors". W Calcium Entry Channels in Non-Excitable Cells, 73–92. Boca Raton : Taylor & Francis, 2017. | Series: Methods in signal transduction series: CRC Press, 2017. http://dx.doi.org/10.1201/9781315152592-4.
Pełny tekst źródłaWang, Wen-An, i Nicolas Demaurex. "Proteins Interacting with STIM1 and Store-Operated Ca2+ Entry". W Cellular Biology of the Endoplasmic Reticulum, 51–97. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-67696-4_4.
Pełny tekst źródłaNg, Lih Chyuan, Judith A. Airey i Joseph R. Hume. "The Contribution of TRPC1 and STIM1 to Capacitative Ca2+ Entry in Pulmonary Artery". W Advances in Experimental Medicine and Biology, 123–35. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-500-2_8.
Pełny tekst źródłaZhou, Yandong, Youjun Wang i Donald L. Gill. "Assessing the Molecular Nature of the STIM1/Orai1 Coupling Interface Using FRET Approaches". W Calcium Entry Channels in Non-Excitable Cells, 127–44. Boca Raton : Taylor & Francis, 2017. | Series: Methods in signal transduction series: CRC Press, 2017. http://dx.doi.org/10.1201/9781315152592-7.
Pełny tekst źródłaStreszczenia konferencji na temat "STIM1"
Hubrack, Satanay, Ethel Adap, Stefan Feske i Khaled Machaca. "Role Of Stim1 And Orai1 In Mammalian Oocyte Activation". W Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0176.
Pełny tekst źródłaAravamudan, Bharathi, Justin Drawz, Michael A. Thompson, Christina Pabelick, Y. S. Prakash i Gary Sieck. "Inflammation-Induced STIM1 Aggregation In Human Airway Smooth Muscle Cells". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2146.
Pełny tekst źródłaBabicheva, A., R. Powers, P. P. Jain, M. Xiong, A. Makino i J. X. J. Yuan. "STIM1 Contributes to Endothelial-to-Mesenchymal Transition in Pulmonary Hypertension". W American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a7136.
Pełny tekst źródłaLatour, Simon, Isabelle Mahouche, Floriane Cherrier, Jean-Philippe Merlio, Sandrine Poglio i Laurence Bresson Bepoldin. "Abstract 1881: STIM1 and Orai1 control non-Hodgkin lymphoma cells migration". W Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1881.
Pełny tekst źródłaYang, Shengyu, Jianwei Sun i Huifang He. "Abstract 4317: Stim1 and Orai1 are critical regulators of melanoma invasion and anoikis". W Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4317.
Pełny tekst źródłaLi, Yongsheng. "Abstract A198: A novel regulatory circuit involving STIM1 and HIF-1 mediates hypoxia-driven hepatocarcinogenesis". W Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics; November 5-9, 2015; Boston, MA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1535-7163.targ-15-a198.
Pełny tekst źródłaLi, Yongsheng. "Abstract 5149: A novel regulatory circuit involving STIM1 and HIF-1 mediates hypoxia-driven hepatocarcinogenesis". W Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5149.
Pełny tekst źródłaUmemura, Masanari, Erdene Baljinnyam, Lai-Hua Xie, Stefan Feske, Martha Nowycky i Kosaku Iwatsubo. "Abstract 5259: Role of Orai1 and STIM1 in store-operated Ca2+ entry and cell migration in melanoma". W Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-5259.
Pełny tekst źródłaPradyumna Kulkarni, Rashmi, Nancy Nader, Ethel Alcantara-adap, Maya Dib i Khaled Machaca. "To Be Or Not To Be: Mechanisms Of Regulation Of Stim1 By Its 3'utr In Breast Cancer". W Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0199.
Pełny tekst źródłaIto, Satoru, Nobukazu Suganuma, Hiromichi Aso, Masashi Kondo, Mitsuo Sato i Yoshinori Hasegawa. "STIM1 Regulates Ca2+ Influx And Migration Induced By Platelet-Derived Growth Factor In Human Airway Smooth Muscle Cells". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3575.
Pełny tekst źródłaRaporty organizacyjne na temat "STIM1"
Savard, Annie, Alexandre Cavalcante i Daniela Caprioara. L’enseignement des mathématiques dans les écoles secondaires du Québec: L’alignement entre les enseignants, les concepts mathématiques des programmes ministériels et les concepts mathématiques utilisés dans les emplois STIM. CIRANO, 2022. http://dx.doi.org/10.54932/mldf5092.
Pełny tekst źródłaDeutsch-Heng, Mikhael, Benoit Dostie i Geneviève Dufour. Documenter l’évolution de la demande des compétences liée aux STIM. CIRANO, styczeń 2022. http://dx.doi.org/10.54932/hajn9336.
Pełny tekst źródłaBelzil, Christian, Jorgen Hansen i Julie Pernaudet. Les déterminants cognitifs et non-cognitifs du choix de filière et leur impact sur la phase initiale du cycle professionnel. CIRANO, maj 2024. http://dx.doi.org/10.54932/zmct9599.
Pełny tekst źródłaDufour, Genevieve, Nathalie de Marcellis-Warin i Molivann Panot. Améliorer les compétences en mathématiques au Québec: Cinq recommandations tirées d’En avant math ! CIRANO, luty 2023. http://dx.doi.org/10.54932/dlcb6893.
Pełny tekst źródłaHaigh, Susan, i Mary Lee Kennedy. Observations on Research Libraries’ Alignment with Institutional STEM Priorities / Observations quant à l’alignement des bibliothèques de recherche sur les priorités institutionnelles en STIM. Association of Research Libraries and Canadian Association of Research Libraries, maj 2023. http://dx.doi.org/10.29242/report.stem2023.
Pełny tekst źródłaFrancesca, Crotta. Docenti di oggi e di domani in Ticino: Stime previsionali del fabbisogno di docenti nelle scuole dell’infanzia, elementari, medie e medie superiori entro il 2022/2023. Scuola universitaria professionale della Svizzera italiana, 2019. http://dx.doi.org/10.33683/ins.19.11232.
Pełny tekst źródłaHaeck, Catherine, Robert Lacroix i Richard E. Tremblay. S’attaquer à la sous-scolarisation des hommes, sans nuire au succès des femmes. CIRANO, marzec 2023. http://dx.doi.org/10.54932/tqny8179.
Pełny tekst źródłaArias, Karla, David López, Segundo Camino-Mogro, Mariana Weiss, Dylan Walsh, Livia Gouvea i Michelle Carvalho Metanias Hallack. Green Transition and Gender Bias: An Analysis of Renewable Energy Generation Companies in Latin America. Redaktor Amanda Beaujon Marin. Inter-American Development Bank, wrzesień 2022. http://dx.doi.org/10.18235/0004461.
Pełny tekst źródła