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Artykuły w czasopismach na temat „Sodium in the body”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 1 sierpnia 2024

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1

Simpson, F. O. "SODIUM INTAKE, BODY SODIUM, AND SODIUM EXCRETION." Lancet 332, no. 8601 (July 1988): 25–29. http://dx.doi.org/10.1016/s0140-6736(88)92954-6.

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2

Skøtt, Ole. "Body sodium and volume homeostasis." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 285, no. 1 (July 2003): R14—R18. http://dx.doi.org/10.1152/ajpregu.00100.2003.

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3

Simpson, F. O. "Sodium Intake, Sodium Handling and Body Sodium in Rats with Spontaneous Genetic Hypertension." Japanese Heart Journal 34, no. 4 (1993): 472–73. http://dx.doi.org/10.1536/ihj.34.472.

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4

Bie, Peter. "Mechanisms of sodium balance: total body sodium, surrogate variables, and renal sodium excretion." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 315, no. 5 (November 1, 2018): R945—R962. http://dx.doi.org/10.1152/ajpregu.00363.2017.

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The classical concepts of human sodium balance include 1) a total pool of Na+ of ≈4,200 mmol (total body sodium, TBS) distributed primarily in the extracellular fluid (ECV) and bone, 2) intake variations of 0.03 to ≈6 mmol·kg body mass−1·day−1, 3) asymptotic transitions between steady states with a halftime (T½) of 21 h, 4) changes in TBS driven by sodium intake measuring ≈1.3 day [ΔTBS/Δ(Na+ intake/day)], 5) adjustment of Na+ excretion to match any diet thus providing metabolic steady state, and 6) regulation of TBS via controlled excretion (90–95% renal) mediated by surrogate variables. The

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5

Martin, Kylie, Sven-Jean Tan, and Nigel D. Toussaint. "Total Body Sodium Balance in Chronic Kidney Disease." International Journal of Nephrology 2021 (September 22, 2021): 1–10. http://dx.doi.org/10.1155/2021/7562357.

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Excess sodium intake is a leading but modifiable risk factor for mortality, with implications on hypertension, inflammation, cardiovascular disease, and chronic kidney disease (CKD). This review will focus mainly on the limitations of current measurement methods of sodium balance particularly in patients with CKD who have complex sodium physiology. The suboptimal accuracy of sodium intake and excretion measurement is seemingly more marked with the evolving understanding of tissue (skin and muscle) sodium. Tissue sodium represents an extrarenal influence on sodium homeostasis with demonstrated

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6

Kitada, Kento, and Akira Nishiyama. "Revisiting blood pressure and body fluid status." Clinical Science 137, no. 9 (May 2023): 755–67. http://dx.doi.org/10.1042/cs20220500.

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Abstract Homeostasis of body fluid is a key component for maintaining health. An imbalance of body sodium and water causes various pathological states, such as dehydration, volume overload, hypertension, cardiovascular and renal diseases, and metabolic disorders. Conventional concepts regarding physiology and pathophysiology of body sodium and water balance have been established by several assumptions. These assumptions are that the kidneys are the master regulator of body sodium and water content, and that sodium moves inside the body in parallel with water. However, recent clinical and basic

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7

Titze, Jens, Natalia Rakova, Christoph Kopp, Anke Dahlmann, Jonathan Jantsch, and Friedrich C. Luft. "Balancing wobbles in the body sodium." Nephrology Dialysis Transplantation 31, no. 7 (September 25, 2015): 1078–81. http://dx.doi.org/10.1093/ndt/gfv343.

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Cross, W. G., and H. Ing. "Sodium Activation in the Human Body." Radiation Protection Dosimetry 10, no. 1-4 (January 1, 1985): 265–76. http://dx.doi.org/10.1093/rpd/10.1-4.265.

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9

Cross, W. G., and H. Ing. "Sodium Activation in the Human Body." Radiation Protection Dosimetry 10, no. 1-4 (January 1, 1985): 265–76. http://dx.doi.org/10.1093/oxfordjournals.rpd.a079428.

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10

Vieweg, W. V. R., and L. S. Godleski. "Psychosis, Body Weight and Plasma Sodium." British Journal of Psychiatry 153, no. 1 (July 1988): 122–23. http://dx.doi.org/10.1192/bjp.153.1.122b.

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11

Heer, Martina. "Sodium Regulation in the Human Body." Current Sports Medicine Reports 7, Suppl. 1 (July 2008): S3—S6. http://dx.doi.org/10.1249/jsr.0b013e31817f2241.

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12

Stefanidis, I., S. Stiller, V. Ikonomov, and H. Mann. "Sodium and body fluid homeostasis in profiling hemodialysis treatment." International Journal of Artificial Organs 25, no. 5 (May 2002): 421–28. http://dx.doi.org/10.1177/039139880202500512.

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Acute adverse side-effects of hemodialysis such as hypotension, muscle cramps, osmotic imbalance and thirst are induced by the interference with fluid and electrolyte balance occurring during treatment. Changes in osmolarity due to alterations of plasma sodium concentration during hemodialysis strongly influence fluid distribution between extracellular and intracellular fluid volume. Increased sodium dialysate concentration induces fluid shift from the intracellular to the extracellular compartment. This shift leads to a more efficient ultrafiltration by increasing plasma refilling volume but

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13

Palacios, Cristina, Karin Wigertz, and Connie M. Weaver. "Comparison of 24-Hour Whole Body versus Patch Tests for Estimating Body Surface Electrolyte Losses." International Journal of Sport Nutrition and Exercise Metabolism 13, no. 4 (December 2003): 479–88. http://dx.doi.org/10.1123/ijsnem.13.4.479.

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Purpose:To compare dermal electrolyte loss between whole body and regional patch methods in women during 24-h.Methods:Dermal loss was collected in 6 healthy women mean age 27 ± 4 years, while consuming 936 mg/d sodium, 1764 mg/d potassium, 696 mg/d calcium, and 152 mg/d magnesium. Twenty-four hour whole body dermal loss was collected using cotton suits by a washdown procedure. Twenty-four hour patch loss was collected from 8 patches placed on the legs, arms, and back.Results:Dermal loss from whole body was 108 ± 110 mg/d sodium, 133 ± 87 mg/d potassium, 103 ± 22 mg/d calcium, and 35 ± 13 mg/d

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14

Kim, Ji-Hong. "Water and Sodium Balance of Body Fluid." Journal of the Korean Society of Pediatric Nephrology 14, no. 2 (2010): 111. http://dx.doi.org/10.3339/jkspn.2010.14.2.111.

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15

Munteanu, Constantin, and Alexandru Iliuta. "The role of sodium in the body." Balneo Research Journal 2, no. 2 (May 1, 2011): 70–74. http://dx.doi.org/10.12680/balneo.2011.1015.

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16

Staessen, Jan, Robert Fagard, Paul Lijnen, and Antoon Amery. "Body weight, sodium intake and blood pressure." Journal of Hypertension 7, Supplement 1 (February 1989): S19—S23. http://dx.doi.org/10.1097/00004872-198902001-00006.

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17

Whelton, Paul K. "Body Weight, Sodium, Potassium, and Blood Pressure." Journal of Clinical Hypertension 17, no. 12 (August 29, 2015): 926–28. http://dx.doi.org/10.1111/jch.12653.

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18

Trevisan, Maurizio, Pasquale Strazzullo, Franco Paulo Cappuccio, Eduardo Farinaro, Fabrizio Jossa, Vittorio Krogh, Roberto Iacone, and Mario Mancini. "Sodium-lithium countertransport and body fat distribution." Life Sciences 51, no. 9 (January 1992): 687–93. http://dx.doi.org/10.1016/0024-3205(92)90242-h.

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19

Bangerter, Neal K., Joshua D. Kaggie, Meredith D. Taylor, and J. Rock Hadley. "Sodium MRI radiofrequency coils for body imaging." NMR in Biomedicine 29, no. 2 (September 29, 2015): 107–18. http://dx.doi.org/10.1002/nbm.3392.

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20

Boicelli, C. A., and A. M. Giuliani. "Sodium ion distribution in the vitreous body." Magma: Magnetic Resonance Materials in Physics, Biology, and Medicine 4, no. 3-4 (September 1996): 241–45. http://dx.doi.org/10.1007/bf01772012.

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21

Frisbie, Malcolm Pratt, and William A. Dunson. "Seasonal aspects of sodium, potassium, and water balance in the predaceous diving beetle Dytiscus verticalis." Canadian Journal of Zoology 66, no. 7 (July 1, 1988): 1553–61. http://dx.doi.org/10.1139/z88-227.

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A previous study showed that food availability i the laboratory can dramatically affect sodium balance of the predaceous diving beetle Dytiscus verticalis. Because this species inhabits small ponds that dry up in the summer, it seemed likely that wild beetles would undergo annual changes in ody ion content. Seasonally collected predaceous diving beetles were analyzed for dry body mass, body water, body sodium and potassium, hemolymph sodium concentration, and hemolymph osmolality. Beetles varied in all characteristics over a 16-month period, and variation was seasonally cyclic for body water,

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22

Lopes‐Menezes, V. C., R. C. Dos‐Santos, V. Felintro, L. R. N. Monteiro, B. Paes‐Leme, D. Lustrino, E. A. Casartelli, L. Vivas, A. S. Mecawi, and L. C. Reis. "Acute body sodium depletion induces skin sodium mobilization in female Wistar rats." Experimental Physiology 104, no. 12 (October 24, 2019): 1754–61. http://dx.doi.org/10.1113/ep087998.

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23

Kapusta, D. R., and J. C. Obih. "Role of endogenous central opioid mechanisms in maintenance of body sodium balance." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 268, no. 3 (March 1, 1995): R723—R730. http://dx.doi.org/10.1152/ajpregu.1995.268.3.r723.

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The role of endogenous central opioids in the regulation of renal function was studied in Sprague-Dawley rats. In metabolism studies, changes in sodium balance were examined during normal dietary sodium intake (days 1-7; Na+ of 174 meq/kg) and sodium restriction (days 8-14; Na+ of 4.0 meq/kg). The influence of endogenous central opioids was investigated by repeating the protocol in the same rats during intracerebroventricular infusion of the opioid antagonist naltrexone methylbromide (NMBR). Intracerebroventricular NMBR did not alter sodium balance in rats fed normal sodium chow. In contrast,

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24

Herlitz, Hans, Olof Jonsson, and Bengt-Åke Bengtsson. "Effect of Recombinant Human Growth Hormone on Cellular Sodium Metabolism." Clinical Science 86, no. 3 (March 1, 1994): 233–37. http://dx.doi.org/10.1042/cs0860233.

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1. The effect of treatment with recombinant human growth hormone on urinary sodium excretion, total body water, the renin-angiotensin system and erythrocyte sodium metabolism was investigated in 16 adults with growth hormone deficiency. 2. Total body water was determined by isotopic dilution, and erythrocyte electrolyte contents were analysed using flame photometry. The rate of sodium influx and the efflux rate constant of sodium were calculated from values of 22Na in erythrocytes in vitro. 3. One week of treatment with recombinant human growth hormone caused a decrease in urinary sodium excre

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25

Sun, Yijuan, David Mills, Todd S. Ing, Joseph I. Shapiro, and Antonios H. Tzamaloukas. "Body Sodium, Potassium and Water in Peritoneal Dialysis-Associated Hyponatremia." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 34, no. 3 (May 2014): 253–59. http://dx.doi.org/10.3747/pdi.2012.00201.

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Objective This report presents a method quantitatively analyzing abnormalities of body water and monovalent cations (sodium plus potassium) in patients on peritoneal dialysis (PD) with true hyponatremia. Methods It is well known that in the face of euglycemia serum sodium concentration is determined by the ratio between the sum of total body sodium plus total body potassium on the one hand and total body water on the other. We developed balance equations that enabled us to calculate excesses or deficits, relative to the state of eunatremia and dry weight, in terms of volumes of water and volum

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26

Siegler, Jason C., Amelia J. Carr, William T. Jardine, Lilia Convit, Rebecca Cross, Dale Chapman, Louise M. Burke, and Megan Ross. "The Hyperhydration Potential of Sodium Bicarbonate and Sodium Citrate." International Journal of Sport Nutrition and Exercise Metabolism 32, no. 2 (March 1, 2022): 74–81. http://dx.doi.org/10.1123/ijsnem.2021-0179.

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Buffering agents have not been comprehensively profiled in terms of their capacity to influence water retention prior to exercise. The purpose of this investigation was to profile the fluid retention characteristics of sodium bicarbonate (BIC) and sodium citrate (CIT) to determine the efficacy of these buffering mediums as hyperhydrating agents. Nineteen volunteers (13 males and six females; age = 28.3 ± 4.9 years) completed three trials (randomized and cross-over design). For each trial, a baseline measurement of body mass, capillary blood, and urine was collected prior to ingestion of their

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27

Simpson, F. O., and Janet M. Ledingham. "Surfeit and Deficit of Sodium: Evidence from Studies of Body Sodium in Rats." Nephron 54, no. 1 (1990): 61–69. http://dx.doi.org/10.1159/000185811.

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28

Twigg, LE, RJ Mead, and DR King. "Metabolism of Fluoroacetate in the Skink (Tiliqua rugosa) and the Rat (Rattus norvegicus)." Australian Journal of Biological Sciences 39, no. 1 (1986): 1. http://dx.doi.org/10.1071/bi9860001.

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Administration of 100 mg sodium fiuoroacetate (compound 1080) per kilogram body weight to T. rugosa resulted in a 3 �4-fold increase in plasma citrate levels 48 h after dosing while administration of 3 mg sodium fiuoroacetate per kilogram body weight to R. norvegicus produced a fivefold increase in plasma citrate levels within 4 h. Administration of 300 mg sodium fiuoroacetate per kilogram body weight reduced the oxygen consumption of the skink by between 2�5 and 11 % while in the rat, 2 mg sodium fiuoroacetate per kilogram body weight reduced oxygen consumption by between 28 and 57%.

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29

Stumpf, Marcelo Tempel, Vivian Fischer, Giovani Jacob Kolling, Maira Balbinotti Zanela, Maria Edi Rocha Ribeiro, and Alexandre Süsenbach de Abreu. "Metabolic attributes, yield and stability of milk in Jersey cows fed diets containing sodium citrate and sodium bicarbonate." Pesquisa Agropecuária Brasileira 48, no. 5 (May 2013): 564–67. http://dx.doi.org/10.1590/s0100-204x2013000500014.

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The objective of this work was to evaluate the inclusion of sodium citrate and sodium bicarbonate in the diet of lactating Jersey cows, and its effects on the metabolic attributes, productivity and stability of milk. We evaluated urinary pH, levels of glucose and urea in blood, body weight, body condition score, milk yield, milk stability (ethanol test), and milk physicochemical properties of 17 cows fed diets containing sodium citrate (100 g per cow per day), sodium bicarbonate (40 g per cow per day) or no additives. Assessments were made at the 28th and 44th days. Supply of sodium citrate or

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30

Mohamed, Ahmed, and Andrew Davenport. "Sodium loss, extracellular volume overload and hypertension in peritoneal dialysis patients treated by automated peritoneal dialysis cyclers." International Journal of Artificial Organs 43, no. 1 (August 12, 2019): 17–24. http://dx.doi.org/10.1177/0391398819864368.

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Introduction: Achieving sodium balance is important for peritoneal dialysis patients, as sodium excess may lead to hypertension and extracellular water expansion. We wished to determine whether greater sodium removal had adverse consequences. Methods: We calculated 24-h urinary and peritoneal sodium losses in peritoneal dialysis patients treated by automated cyclers, when attending for peritoneal membrane and bioimpedance assessments. Results: We reviewed 439 peritoneal dialysis patients, 56.7% male, average age 54.6 years, median sodium loss 110 (68–155) mmol/day. Sodium loss was strongly ass

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31

&NA;, &NA;. "Sodium, potassium, body mass, alcohol and blood pressure." Journal of Hypertension 6, no. 4 (December 1988): S584–586. http://dx.doi.org/10.1097/00004872-198812040-00183.

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32

Beretta-Piccoli, Carlo. "Body Sodium in Normal Subjects Predisposed to Hypertension." Journal of Cardiovascular Pharmacology 16 (1990): S52—S55. http://dx.doi.org/10.1097/00005344-199000167-00017.

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Beretta-Piccoli, Carlo. "Body Sodium in Normal Subjects Predisposed to Hypertension." Journal of Cardiovascular Pharmacology 16 (1990): S52—S55. http://dx.doi.org/10.1097/00005344-199006167-00017.

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34

Shinar, Hadassah, and Gil Navon. "Sodium-23 NMR relaxation times in body fluids." Magnetic Resonance in Medicine 3, no. 6 (December 1986): 927–34. http://dx.doi.org/10.1002/mrm.1910030613.

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35

Adrogué, Horacio J., Sreedhar Mandayam, Hocine Tighiouart, and Nicolaos E. Madias. "Osmotic and Nonosmotic Sodium Storage during Acute Hypertonic Sodium Loading." American Journal of Nephrology 50, no. 1 (2019): 11–18. http://dx.doi.org/10.1159/000501190.

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Background: The Edelman equation has long guided the expected response of plasma [Na+] to changes in sodium, potassium, and water balance, but recent short-term studies challenged its validity. Plasma [Na+] following hypertonic NaCl infusion in individuals on low-sodium diet fell short of the Edelman predictions supposedly because sodium restriction caused progressive osmotic inactivation of 50% of retained sodium. Here, we examine the validity of this challenge. Methods: We evaluated baseline total body water (TBW) and Na+ space following acute hypertonic NaHCO3 infusion in dogs with variable

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36

Laurén, Darrel Jon, and D. G. McDonald. "Acclimation to Copper by Rainbow Trout, Salmo gairdneri: Physiology." Canadian Journal of Fisheries and Aquatic Sciences 44, no. 1 (January 1, 1987): 99–104. http://dx.doi.org/10.1139/f87-012.

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Juvenile rainbow trout (Salmo gairdneri) were exposed to 55 μg copper∙L−1 for 28 d and then transferred to uncontaminated water for 7 d. Whole body sodium concentration and sodium uptake (Jin) were measured at weekly intervals; Jin was measured at various Na+ concentrations and kinetic parameters estimated. After 24 h of copper exposure, the maximum rate of sodium uptake (Jmax) was inhibited by 55%, the affinity for sodium (Km) reduced by 49%, and whole body Na+ decreased by about 12.5%. After 7 d of exposure, whole body Na+ had returned to control values, but Jmax was still inhibited by 41%.

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37

Takamata, A., G. W. Mack, C. M. Gillen, and E. R. Nadel. "Sodium appetite, thirst, and body fluid regulation in humans during rehydration without sodium replacement." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 266, no. 5 (May 1, 1994): R1493—R1502. http://dx.doi.org/10.1152/ajpregu.1994.266.5.r1493.

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After a 7-h H2O and Na+ depletion period (DP), produced by intermittent light exercise (8 bouts) at 35 degrees C, we examined thirst and taste palatability responses to 10 different NaCl solutions during 23 h of rehydration (RH) at 25 degrees C. During DP, net H2O and Na+ loss were 27.2 +/- 2.9 ml/kg and 3.29 +/- 0.45 meq/kg, respectively. Plasma osmolality (POsm) and plasma Na+ concentration ([Na+]p) increased significantly during DP by 3.4 +/- 1.2 mosmol/kgH2O and 3.0 +/- 1.0 meq/kgH2O, respectively. Plasma volume (PV) decreased by 6.5 +/- 1.9%. Thirst rating, renal fractional reabsorption o

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38

Seeliger, Erdmann, Katrin Lohmann, Benno Nafz, Pontus B. Persson, and H. Wolfgang Reinhardt. "Pressure-dependent renin release: effects of sodium intake and changes of total body sodium." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 277, no. 2 (August 1, 1999): R548—R555. http://dx.doi.org/10.1152/ajpregu.1999.277.2.r548.

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The impact of sodium intake and changes in total body sodium (TBS) for the setting of pressure-dependent renin release (PDRR) was studied in freely moving dogs. An aortic cuff allowed servo control of renal perfusion pressure (RPP) at preset values. Protocols were 1) high sodium intake (HSI), 2) low sodium intake (LSI), 3) TBS moderately increased (+3.1 mmol Na/kg body wt) by 20% reduction of RPP for 2–4 days, 4) large increase of TBS (+8.2) by combining protocol 3 with aldosterone infusion, and 5) TBS reduced (−3.1) by peritoneal dialyses. Twenty-four-hour time courses of arterial plasma reni

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Segar, Jeffrey L., Connie C. Grobe, and Justin L. Grobe. "Fetal storage of osmotically inactive sodium." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 318, no. 3 (March 1, 2020): R512—R514. http://dx.doi.org/10.1152/ajpregu.00336.2019.

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Work in adult humans and animals suggest sodium (Na) is stored in tissue reservoirs without commensurate water retention. These stores may protect from water loss, regulate immune function, and participate in blood pressure regulation. A role for such stores early in life, during which total body Na sufficiency is vital for optimal growth, has not been explored. Using data from previously published literature, we calculated total body stores of Na, potassium (K), and chloride (Cl) during fetal development (24–40 wk gestation) using two methods 1) based on the distribution of body water mass wi

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40

Marlita, Jeli, Yuyun Febriani, Risma Hayatun Nufus, Muhlisun Azim, and Baiq Maylinda Gemantari. "ANALYSIS OF NaOCl CONTENT OF HAND & BODY LOTION AND FACE WASH CREAM BY IODOMETRIC TITRATION METHOD." Medical Sains : Jurnal Ilmiah Kefarmasian 9, no. 2 (June 6, 2024): 417–22. http://dx.doi.org/10.37874/ms.v9i2.1242.

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Cosmetics are needed by the public, such as hand and body lotions and face brightening creams, and there are many misuses of hazardous chemicals in cosmetics, including lotions and face brightening creams. Sodium hypochlorite, a hazardous chemical, can cause skin damage, such as irritation, rash, hypersensitivity, and burns. The purpose of this study was to determine the presence of sodium hypochlorite compounds and sodium hypochlorite in hand and body lotion cosmetics and face brightening cream. The method used in this research was qualitative analysis with color reaction and quantitative ana

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Tremblay, Angelo, Marie-Pascale Gagné, Louis Pérusse, Catherine Fortier, Véronique Provencher, Ronan Corcuff, Sonia Pomerleau, Nicoletta Foti, and Vicky Drapeau. "Sodium and Human Health: What Can Be Done to Improve Sodium Balance beyond Food Processing?" Nutrients 16, no. 8 (April 18, 2024): 1199. http://dx.doi.org/10.3390/nu16081199.

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Sodium plays a key role in the regulation of water balance and is also important in food formulation due to its contribution to the taste and use in the preservation of many foods. Excessive intake of any essential nutrient is problematic and this seems to be particularly the case for sodium since a high intake makes it the nutrient most strongly associated with mortality. Sodium intake has been the object of recommendations by public health agencies such as the WHO and this has resulted in efforts by the food industry to reduce the sodium content of packaged foods, although there is still roo

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42

Reinhardt, H. Wolfgang, and Erdmann Seeliger. "Toward an Integrative Concept of Control of Total Body Sodium." Physiology 15, no. 6 (December 2000): 319–25. http://dx.doi.org/10.1152/physiologyonline.2000.15.6.319.

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Total body sodium (TBSodium) is a major determinant of body water and arterial pressure. Several observations, in particular that of a “sodium memory,” indicate that TBSodium is a controlled variable. Various regulatory elements are involved, e.g., the renin-angiotensin-aldosterone system, atrial receptors, and renal arterial pressure. Balance studies in dogs provide new insights into their contributions to TBSodium control.

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White, B. D., G. L. Edwards, and R. J. Martin. "Interaction of type I and type II corticosteroid receptor stimulation on carcass energy and carcass water." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 270, no. 5 (May 1, 1996): R1099—R1108. http://dx.doi.org/10.1152/ajpregu.1996.270.5.r1099.

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The effects of chronic type I and type II corticosteroid receptor stimulation were examined in adrenalectomized Sprague-Dawley rats to quantify the relative contribution of body energy and body water changes to changes in body weight. Adrenalectomy caused a decrease in both body energy and water. Aldosterone (type I agonist) treatment increased body weight gain and returned energy accretion to the level of sham-operated animals. However, most of the change in body weight (72%) was attributable to a change in body water. The aldosterone-induced increase in body weight gain and carcass water wer

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Yanwittayakul, Krittin, Tararat Khaokhiew, Woraphan Chaisriratanakul, Win Bunjongpru, and Sira Srinives. "Fabrication of an ISFET Sensor for the Detection of Sodium Ions in Body Plasma." Key Engineering Materials 824 (October 2019): 190–96. http://dx.doi.org/10.4028/www.scientific.net/kem.824.190.

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Sodium chloride, commonly known as table salt, is widely used as essential seasoning in food, snacks and deserts worldwide. However, excessive consumption of table salt is a major cause of various health issues, involving high blood pressure, liver cirrhosis, kidney disease, and heart failure. This research aims at creating a portable, low-powered, efficient sensor for detection of sodium ions in body plasma for medical diagnosis purpose. The device was fabricated on a platform of Ion-Sensitive Field-Effect Transistor (ISFET) modified with sodium ionophore (sodium recognizing element), entrapp

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Sokolař, Radomír, and Lucie Vodová. "Sodium Hexametaphosphate as Deflocculation Agent for Calcium Aluminate Cements in Porcelain Body." Advanced Materials Research 897 (February 2014): 30–33. http://dx.doi.org/10.4028/www.scientific.net/amr.897.30.

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Influence of typical ceramic deflocculant – sodium hexametaphosphate – on the rheological properties (viscosity) of calcium aluminate cement paste, porcelain raw materials mixture (casting slip), and fired porcelain body respectively, was determined. It was used two different typed of calcium aluminate cements (from two sources – producers - Istra, Almatis) with different content of Al2O3 (40 % and 70 %). Sodium hexametaphosphate decreases of water content needed to prepare slip casting with constant viscosity. Deflocculant increases the modulus of rupture MOR of dried green body and its bulk

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Simpson, F. O. "The Control of Body Sodium in Relation to Hypertension." Journal of Cardiovascular Pharmacology 16 (1990): S27—S30. http://dx.doi.org/10.1097/00005344-199000167-00010.

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Simpson, F. O. "The Control of Body Sodium in Relation to Hypertension." Journal of Cardiovascular Pharmacology 16 (1990): S27—S30. http://dx.doi.org/10.1097/00005344-199006167-00010.

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Alderman, M. H., H. W. Cohen, and J. Fang. "DIET SODIUM, BODY MASS INDEX, EXERCISE AND CARDIOVASCULAR EVENTS." Journal of Hypertension 22, Suppl. 1 (February 2004): S5. http://dx.doi.org/10.1097/00004872-200402001-00005.

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Geraci, J. P., K. L. Jackson, and M. S. Mariano. "Fluid and Sodium Loss in Whole-Body-Irradiated Rats." Radiation Research 111, no. 3 (September 1987): 518. http://dx.doi.org/10.2307/3576937.

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Baker, Lindsay B., Ryan P. Nuccio, Adam J. Reimel, Shyretha Brown, Corey T. Ungaro, Peter JD De Chavez, and Kelly A. Barnes. "Cross-validation Of Whole Body Sweat Sodium Prediction Equations." Medicine & Science in Sports & Exercise 52, no. 7S (July 2020): 969. http://dx.doi.org/10.1249/01.mss.0000686096.35530.dc.

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