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Artykuły w czasopismach na temat "Sleep architecture"

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Deatherage, Joseph R., R. David Roden i Kenneth Zouhary. "Normal Sleep Architecture". Seminars in Orthodontics 15, nr 2 (czerwiec 2009): 86–87. http://dx.doi.org/10.1053/j.sodo.2009.01.002.

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Wang, David, i Harry Teichtahl. "Opioids, sleep architecture and sleep-disordered breathing". Sleep Medicine Reviews 11, nr 1 (luty 2007): 35–46. http://dx.doi.org/10.1016/j.smrv.2006.03.006.

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Hoffstein, V., J. H. Mateika i S. Mateika. "Snoring and Sleep Architecture". American Review of Respiratory Disease 143, nr 1 (styczeń 1991): 92–96. http://dx.doi.org/10.1164/ajrccm/143.1.92.

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Lorrain, D., D. Bélisle i I. Viens. "Subjective sleep quality and sleep architecture in aging". Sleep Medicine 64 (grudzień 2019): S231—S232. http://dx.doi.org/10.1016/j.sleep.2019.11.648.

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Palinkas, Marcelo, Marisa Semprini, João Espir Filho, Graziela de Luca Canto, Isabela Hallak Regalo, César Bataglion, Laíse Angélica Mendes Rodrigues, Selma Siéssere i Simone Cecilio Hallak Regalo. "Nocturnal sleep architecture is altered by sleep bruxism". Archives of Oral Biology 81 (wrzesień 2017): 56–60. http://dx.doi.org/10.1016/j.archoralbio.2017.04.025.

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Hachinski, Vladimir C., Mortimer Mamelak i John W. Norris. "Clinical Recovery and Sleep Architecture Degradation". Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 17, nr 3 (sierpień 1990): 332–35. http://dx.doi.org/10.1017/s0317167100030699.

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ABSTRACT:We achieved a unique and timely recording of cerebral activity in a 70 year old woman immediately pre- and post-stroke, while studying the effect of acute cerebral infarction on sleep-electroencephalogram (EEG) patterns. Normal patterns, except for increased wakefulness, were recorded during two pre-infarct polysomnograms. Immediately following cerebral infarction increased delta activity was recorded from the infarcted hemisphere only. Initially, REM sleep could not be recorded from either side; however, on the third post infarct day REM sleep returned. Background EEG levels from both hemispheres became progressively slower, flatter and simpler. In addition, sleep spindles and the distinctive saw-tooth wave forms of sleep almost disappeared. At one year post-stroke sleep-EEG rhythm recordings from both hemispheres became more similar except for persisting delta activity from the left hemisphere. Unexpected deterioration of sleep-EEG pattern recordings from the undamaged hemisphere taken during the patient's clinical recovery remains unexplained. Serial sleep recording may facilitate the study of brain recovery, activity and reorganization following stroke.
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Deshaies-Rugama, A. S., H. Blais, Z. Sekerovic, M. Massicotte, J. Carrier, C. Thompson, M. Nigam, A. Desautels, J. Montplaisir i N. Gosselin. "Sleep architecture in idiopathic hypersomnia". Sleep Medicine 100 (grudzień 2022): S104. http://dx.doi.org/10.1016/j.sleep.2022.05.290.

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Jafari, Behrouz. "Sleep Architecture and Blood Pressure". Sleep Medicine Clinics 12, nr 2 (czerwiec 2017): 161–66. http://dx.doi.org/10.1016/j.jsmc.2017.02.003.

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Walsh, B. Timothy. "Sleep Architecture in Eating Disorders". Archives of General Psychiatry 47, nr 9 (1.09.1990): 880. http://dx.doi.org/10.1001/archpsyc.1990.01810210088018.

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Chan, Martin, Tracy C. H. Wong, Aidan Weichard, Gillian M. Nixon, Lisa M. Walter i Rosemary S. C. Horne. "Sleep macro-architecture and micro-architecture in children born preterm with sleep disordered breathing". Pediatric Research 87, nr 4 (13.06.2019): 703–10. http://dx.doi.org/10.1038/s41390-019-0453-1.

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Rozprawy doktorskie na temat "Sleep architecture"

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Dingwall, Kylie. "Effects of medication on the sleep architecture of patients with obstructive sleep apnea syndrome (OSAS) /". [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19264.pdf.

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Ajwad, Asma'a A. "SLEEP AND THERMOREGULATION: A STUDY OF THE EFFECT OF AMBIENT TEMPERATURE MANIPULATION ON MOUSE SLEEP ARCHITECTURE". UKnowledge, 2018. https://uknowledge.uky.edu/cbme_etds/54.

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Good quality sleep is essential for mental and physical health. Inadequate sleep impacts memory consolidation, learning and cognition, immune function, autonomic regulation, physical performance, and other vital functions. In many neurological disorders that are associated with sleep problems such as epilepsy and Alzheimer’s disease, changes in brain circuitry affect sleep-wake regulation mechanisms; this is reflected in anomalous sleep-wake architecture and usually accompanied by poor sleep depth. Thus, over many years, many approaches have been tried in humans and animal models with the goal of improving sleep quality. Unfortunately, each of those approaches comes with limitations or side effects. Thus, there is a need for a natural, safe, and low cost approach that overcomes many limitations to improve sleep and eventually the lives of individuals with sleep problems. Environmental temperature is one of the most important factors that affect sleep in humans and other animals. Studies have shown that the part of the brain governing thermoregulation is also involved in sleep-wake regulation. Even a mild change in environmental temperature can produce a significant effect on sleep. Thus, a better understanding of the sleep-thermoregulation interaction could lead to novel ways for treating many sleep disorders. As a first step on the translational pathway, experiments in animal models of disease conditions with disordered sleep are needed for investigating sleep–thermoregulation interactions and for devising and validating related approaches to enhance sleep quality before conducting them on humans. This dissertation explores and assesses the effect of changes in ambient temperature on sleep-wake architecture in control mice and epileptic mice, the latter from a model of temporal lobe epilepsy as an example of a disease model with disordered sleep. Then, based on the results of temperature effects on sleep in control and epileptic mice, different strategies are proposed and tested to modulate sleep through ambient temperature regulation in closed loop to improve sleep depth and regulate the timing of the sleep-wake cycle. The results presented in this dissertation demonstrate the feasibility of sleep enhancement and regulation of its timing and duration through manipulation of ambient temperature using closed-loop control systems. Similar approaches could foreseeably be used as more natural means for enhancing deep sleep in patients with epilepsy, Alzheimer’s, or Parkinson’s disease in which poor sleep is common and associated with adverse outcomes.
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Kallushi, Abi Elena. "Healing Architecture for Troubled Nightowls: Restoring Natural Rhythms in Nocturnal U Street". Thesis, Virginia Tech, 2016. http://hdl.handle.net/10919/73776.

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This thesis is an exploration of architecture as an active participant in human life, not just as a stage. When architecture is considered a participant it contributes to the improvement of our quality of life as well as aiding  the healing process when we fall ill. As more people are diagnosed with anxiety and sleep disorders, the thesis considered questions of how the places in which we carry out our day-to-day lives improve the prognosis and, further, can architecture be used as a powerful tool for healing? It is possible that our disconnection from nature is partially responsible for our disrupted sleep patterns and misplaced anxieties. Perhaps natural elements and rhythms are too absent in our daily urban lives. But as more of us move into cities and our urban centers become denser, designers must find clever ways to help city owls reset their circadian rhythm. Architecture can help reestablish that bond with nature. This thesis explores an architecture of healing by proposing a program and design that follows the day and night circadian rhythm of our bodies, which in turn follow the sun and other natural phenomena. Located in a tricky triangular site in one of the busiest nighttime neighborhoods of Washington DC, a wellness center and sleep clinic would allow city dwellers to find a peaceful oasis for healing. In parallel, this thesis is also a study of collage as a design tool, as well as designing from details and the human scale.
Master of Architecture
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Johnson, Pamela Lesley. "Sleep and Breathing at High Altitude". University of Sydney, 2008. http://hdl.handle.net/2123/3531.

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Doctor of Philosphy (PhD)
This thesis describes the work carried out during four treks, each over 10-11 days, from 1400m to 5000m in the Nepal Himalaya and further work performed during several two-night sojourns at the Barcroft Laboratory at 3800m on White Mountain in California, USA. Nineteen volunteers were studied during the treks in Nepal and seven volunteers were studied at White Mountain. All subjects were normal, healthy individuals who had not travelled to altitudes higher than 1000m in the previous twelve months. The aims of this research were to examine the effects on sleep, and the ventilatory patterns during sleep, of incremental increases in altitude by employing portable polysomnography to measure and record physiological signals. A further aim of this research was to examine the relationship between the ventilatory responses to hypoxia and hypercapnia, measured at sea level, and the development of periodic breathing during sleep at high altitude. In the final part of this thesis the possibility of preventing and treating Acute Mountain Sickness with non-invasive positive pressure ventilation while sleeping at high altitude was tested. Chapter 1 describes the background information on sleep, and breathing during sleep, at high altitudes. Most of these studies were performed in hypobaric chambers to simulate various high altitudes. One study measured sleep at high altitude after trekking, but there are no studies which systematically measure sleep and breathing throughout the whole trek. Breathing during sleep at high altitude and the physiological elements of the control of breathing (under normal/sea level conditions and under the hypobaric, hypoxic conditions present at high altitude) are described in this Chapter. The occurrence of Acute Mountain Sickness (AMS) in subjects who travel form near sea level to altitudes above 3000m is common but its pathophysiology not well understood. The background research into AMS and its treatment and prevention are also covered in Chapter 1. Chapter 2 describes the equipment and methods used in this research, including the polysomnographic equipment used to record sleep and breathing at sea level and the high altitude locations, the portable blood gas analyser used in Nepal and the equipment and methodology used to measure each individual’s ventilatory response to hypoxia and hypercapnia at sea level before ascent to the high altitude locations. Chapter 3 reports the findings on the changes to sleep at high altitude, with particular focus on changes in the amounts of total sleep, the duration of each sleep stage and its percentage of total sleep, and the number and causes of arousals from sleep that occurred during sleep at increasing altitudes. The lightest stage of sleep, Stage 1 non-rapid eye movement (NREM) sleep, was increased, as expected with increases in altitude, while the deeper stages of sleep (Stages 3 and 4 NREM sleep, also called slow wave sleep), were decreased. The increase in Stage 1 NREM in this research is in agreement with all previous findings. However, slow wave sleep, although decreased, was present in most of our subjects at all altitudes in Nepal; this finding is in contrast to most previous work, which has found a very marked reduction, even absence, of slow wave sleep at high altitude. Surprisingly, unlike experimental animal studies of chronic hypoxia, REM sleep was well maintained at all altitudes. Stage 2 NREM and REM sleep, total sleep time, sleep efficiency and spontaneous arousals were maintained at near sea level values. The total arousal index was increased with increasing altitude and this was due to the increasing severity of periodic breathing as altitude increased. An interesting finding of this research was that fewer than half the periodic breathing apneas and hypopneas resulted in arousal from sleep. There was a minor degree of upper airway obstruction in some subjects at sea level but this was almost resolved by 3500m. Chapter 4 reports the findings on the effects on breathing during sleep of the progressive increase of altitude, in particular the occurrence of periodic breathing. This Chapter also reports the results of changes to arterial blood gases as subjects ascended to higher altitudes. As expected, arterial blood gases were markedly altered at even the lowest altitude in Nepal (1400m) and this change became more pronounced at each new, higher altitude. Most subjects developed periodic breathing at high altitude but there was a wide variability between subjects as well as variability in the degree of periodic breathing that individual subjects developed at different altitudes. Some subjects developed periodic breathing at even the lowest altitude and this increased with increasing altitude; other subjects developed periodic breathing at one or two altitudes, while four subjects did not develop periodic breathing at any altitude. Ventilatory responses to hypoxia and hypercapnia, measured at sea level before departure to high altitude, was not significantly related to the development of periodic breathing when the group was analysed as a whole. However, when the subjects were grouped according to the steepness of their ventilatory response slopes, there was a pattern of higher amounts of periodic breathing in subjects with steeper ventilatory responses. Chapter 5 reports the findings of an experimental study carried out in the University of California, San Diego, Barcroft Laboratory on White Mountain in California. Seven subjects drove from sea level to 3800m in one day and stayed at this altitude for two nights. On one of the nights the subjects slept using a non-invasive positive pressure device via a face mask and this was found to significantly improve the sleeping oxyhemoglobin saturation. The use of the device was also found to eliminate the symptoms of Acute Mountain Sickness, as measured by the Lake Louise scoring system. This finding appears to confirm the hypothesis that lower oxygen saturation, particularly during sleep, is strongly correlated to the development of Acute Mountain Sickness and may represent a new treatment and prevention strategy for this very common high altitude disorder.
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Sedgwick, Philip Martin. "Sleep in a naturalistic environment and the influence of the calendar week". Thesis, St George's, University of London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.313277.

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Krenzke, Shaun R. "Housing for empowerment : more than just a place to eat, sleep, and watch TV". Virtual Press, 2004. http://liblink.bsu.edu/uhtbin/catkey/1305456.

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I began this investigation by asking a question. What is a possible design solutionthat can enable people who live-in or seek-out affordable housing to inhabit a structure that is more than a shelter, but a place they are proud to return to, bring friends to, and live in?The first portion of this thesis documents the need for affordable housing in the United States. Franklin Roosevelt, in 1944 before United States Congress, listed one of the economic rights of every citizen to be, "the right of every family to a decent home." Less money is being spent building new affordable housing or maintaining existing housing than at any other time in our history. The need for affordable housing continues to grow while the amount of available units continues to decline. There will always be a need for affordable housing in the United States. Some people will move out, but there will be new people with a need. I believe housing should be more than merely shelter. The rundown big box affordable housing we are all familiar with does not empower the people who occupy it to live their lives or easily better themselves. They are isolated in location and by negative connotation. There are a growing number of architects who have taken on the challenge to help people to better themselves, when they are unable to themselves. The four architectural precedents that are documented in the second portion of this thesis have dedicated their lives and abilities to creating better affordable housing that aids in allowing citizens, reguardless of race, ethnicity, or income (economic status), to benefit from their physical environment. Examples of each architectural firm's work are presented. I examine the design and participatory processes that enabled the architects to empower the people who live in their well-designed affordable housing.The final portion of this thesis focuses on stating and justifying seventeen design principles to enable people to create decent affordable housing based on the research and interviews. These principles investigate the ideas of being human, giving the sense of ownership to the people who live in affordable housing, being contextually respectful to one's neighborhood and community, being environmentally friendly, being modifiable or changeable for the different people who live in it over time, and respecting each family's specific story and enabling them to express their life and lifestyle. This thesis expresses the design principles of housing for empowerment.
Department of Architecture
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Stewart, Fiona Anne. "The evolution of shelter : ecology and ethology of chimpanzee nest building". Thesis, University of Cambridge, 2011. https://www.repository.cam.ac.uk/handle/1810/241033.

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Human beings of all cultures build some form of shelter, and the global distribution of Homo sapiens depends on this basic trait. All great apes (chimpanzee, bonobo, gorilla, and orangutan) build analogous structures (called nests or beds) at least once a day throughout their adult lives, which suggests that this elementary technology was present before the hominid lines separated. This thesis investigates the variability and function of specifically wild chimpanzee shelters. I compared characteristics of chimpanzee nests, nesting trees, nest shape, and architecture in two savanna-dwelling populations on opposite sides of Africa: Fongoli, Senegal, and Issa, Tanzania. Savanna habitats are the most extreme habitats in which chimpanzees survive today, and may represent a similar environment to that in which early hominins evolved in the Plio-Pleistocene (Chapter 2). Investigating variation in nest-building within and between these two extreme habitats made it possible to tackle hypotheses of the shelter function of nests (Chapter 3).The influence of environment, specifically the role of protection from disease vectors and fluctuating temperatures, was assessed through a novel experiment in which I slept overnight in arboreal chimpanzee nests and on the bare earth (Chapter 4). To assess whether or not nests serve as an anti-predation function, I compared nesting in Issa, where predators are abundant, to Fongoli, where they are absent (Chapter 5). I provided further support for the thermoregulatory function of nests by showing that chimpanzees build more insulating nests in adverse weather conditions (Chapter 6).Nest-building is a learned behaviour, but its ontogeny is little known. I investigated social sources of variation in nest building in Fongoli to examine whether sex and age differences exist in nest building duration, nest position, shape and architecture (Chapter 7). Finally, ecosystem engineering is a consequence of animal construction, from ants to humans. I investigated use-wear traces around nests to assess niche construction of nest- building. I showed that chimpanzees repeatedly re-used these specific nest-spots within trees, which are pre-fabricated for future building through repeated pruning and shaping of these structures (Chapter 8).Nest building in great apes may be the foundation of constructivity in hominids. This thesis describes proximate functions and influences on nest-building variation in wild chimpanzees that help to model the evolution of shelter in hominids.
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Buechel, Heather M. "CHANGES IN SLEEP ARCHITECTURE AND COGNITION WITH AGE AND PSYCHOSOCIAL STRESS: A STUDY IN FISCHER 344 RATS". UKnowledge, 2013. http://uknowledge.uky.edu/pharmacol_etds/4.

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Changes in both sleep architecture and cognition are common with age. Typically these changes have a negative connotation: sleep fragmentation, insomnia, and deep sleep loss as well as forgetfulness, lack of focus, and even dementia and Alzheimer’s disease. Research has shown that psychosocial stressors, such as isolation from family and friends or loss of a loved one can also have significant negative effects on sleep architecture and cognitive capabilities. This leaves the elderly in a particularly vulnerable situation: suffering from cognitive decline and sleep dysregulation already, and more likely to respond negatively to psychosocial stressors. Taking all of these factors into account, it’s surprising that little research has been done to elucidate the mechanisms behind aged subjects’ enhanced vulnerability to new onset psychosocial stress. Our lab embarked on a series of studies to test the effects of age and psychosocial stress on sleep architecture and cognition. Our first study measured sleep stages in young adult and aged F344 rats during their resting and active periods. Animals were behaviorally characterized on the Morris water maze and gene expression profiles of their parietal cortices were taken. We confirmed previous studies that found impaired cognition and decreased resting deep sleep with age. However, it was increased active deep sleep that correlated best with poor cognitive performance. In the second study rats were subjected to immobilization (restraint stress) immediately preceding their final water maze task. Hippocampi were prepared for synaptic electrophysiology and trunk blood was taken for corticosterone measurement after post-stress sleep architecture data was collected. Young subjects responded to acute stress with decreased cognition, elevated CORT levels and altered sleep architecture. In contrast, stressed aged subjects were statistically indistinguishable from control aged subjects, suggesting that aged rats are less responsive to an acute psychosocial stress event. Together, these studies suggest that alleviating sleep dysregulation could therapeutically benefit cognition psychosocial stress resilience.
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Henry, Michelle. "Associations between sleep architecture, cortisol concentrations, cognitive performance, and quality of life in patients with Addison's disease". Doctoral thesis, Faculty of Humanities, 2019. http://hdl.handle.net/11427/30377.

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Recent literature in the neurosciences suggests that there are mechanistic relations between sleep disruption and cognitive (particularly memory) deficits, and that varying concentrations of the hormone cortisol may play a particularly important role in mediating those relations. Because patients with Addison’s disease (AD) experience consistent and predictable periods of sub- and supra-physiological cortisol concentrations (due to lifelong glucocorticoid replacement therapy), and because they frequently report disrupted sleep and poor memory, those presenting with that endocrinological disorder form an ideal population to use in studies testing hypotheses about the ways in which (a) disrupted sleep is related to impaired consolidation of previously learned material (and, hence, poor performance on tests assessing memory for that material), and (b) cortisol concentrations may mediate this relationship between sleep and memory. This dissertation presents four studies that, together, tested those hypotheses. Study 1 (n = 60 per group) found that patients with AD self-reported significantly more disturbed sleep and poorer cognition and quality of life compared to matched healthy controls. Importantly, our analyses suggested that disrupted sleep, and not AD per se, accounted most strongly for the reported cognitive impairment. Study 2 (n = 35 per group) found that patients had significantly poorer objectively-measured declarative memory performance compared to matched healthy controls, but that other domains of cognition were relatively unimpaired. Study 3 (n = 10 per group) suggested that matched healthy controls retained significantly more declarative information than patients. Importantly, while controls retained significantly more declarative information when a period of sleep, rather than waking, separated learning from recall, patients derived no such benefit. Study 4 (n = 7 per group) suggested that, relative to matched healthy controls, patients had different patterns of night-time cortisol secretion, accompanied by significantly reduced slow-wave sleep. Together, these four studies suggest that, despite being on replacement medication, patients with AD still experience disrupted sleep and memory deficits. These disruptions and deficits may be related to the failure of replacement regimens to restore a normal circadian rhythm of cortisol secretion. This pattern of results provides support for existing theoretical frameworks which posit that (in AD and other neuroendocrine, neurological, or psychiatric disorders) disrupted sleep is an important biological mechanism that underlies, at least partially, the memory impairments that patients frequently report experiencing. With specific regard to patients with AD, the findings presented here suggest that future initiatives aimed at improving patients’ cognitive performance (and, indeed, their overall quality of life) should prioritise optimizing sleep. More generally, this dissertation advances our understanding of sleep as a critical biological process essential for cognitive well-being.
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Facer-Childs, Elise Rose. "'Citius, Altius, Fortius' : the impact of circadian phenotype and sleep on the brain's intrinsic functional architecture, well-being & performance". Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8300/.

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A major challenge facing our rapidly developing ‘round the clock’ society is in understanding how disruptions to sleep and endogenously driven biological clocks influence many aspects of our lives. This thesis combines the fields of chronobiology, sleep and neuroimaging to investigate the impact of Circadian Phenotype and time of day on the brain’s intrinsic architecture (using functional MRI), well-being and performance. Brain function changes during wakefulness and sleep, as well as in a range of neurological and psychiatric disorders. The results show, for the first time, clear differences in intrinsic functional architecture between Early and Late Circadian Phenotypes (ECP/LCP), as well as variations depending on the time of day. In general, ECPs have higher functional connectivity than LCPs to the majority of regions identified, and these differences can predict better outcomes in cognitive and physical performance measures. Performance also shows significant diurnal variations within Circadian Phenotypes. A wider investigation in the LCP group showed that a phase advance in a real world setting using non-pharmacological interventions has a positive impact on mental well-being and performance. In summary, this thesis supports the need to consider both Circadian Phenotype and time of day in neuroimaging and performance research. It also highlights that chronic disruptions often associated with LCPs could have intrinsic neural origins. Furthermore, it provides a novel intervention strategy for LCPs to enhance well-being and performance during a societal constrained day. These findings could have significant implications in clinical, research and real world settings.
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Książki na temat "Sleep architecture"

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Chiang, Rayleigh Ping-Ying. Introduction to Modern Sleep Technology. Dordrecht: Springer Netherlands, 2012.

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Madaan, Vishal. Psychotropics and Sleep Architecture. Taylor & Francis Group, 2019.

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Press, Mirako. Eat Sleep Architecture Repeat: Genkouyoushi Notebook. Independently Published, 2019.

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Groenendal, Nathan. Eat Sleep Studio Repeat: Architecture Sketch Book. Independently Published, 2019.

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Press, Mirako. Eat Sleep Architecture Repeat: Appointment Book 2 Columns. Independently Published, 2018.

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Press, Mirako. Eat Sleep Architecture Repeat: Appointment Book 4 Columns. Independently Published, 2019.

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Carrión, Victor G., John A. Turner i Carl F. Weems. Sleep. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190201968.003.0005.

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The architecture of healthy sleep rests upon a network of several interacting neurochemical systems, an arrangement that is easily disrupted by the experience of traumatic stress. As a result, sleep may be among the most susceptible of behaviors to have a negative impact as a result of trauma. Sleep disturbances, or “parasomnias,” such as nightmares, sleepwalking, and insomnia are one of the most prominent hallmarks of PTSD, and the study of these sleep-specific symptoms can provide a window into the underlying pathology of the disorder. The current chapter reviews the preclinical animal literature that has informed our understanding of the brain structures that are involved in the development of these parasomnias. In reviewing adult and child studies of disrupted sleep in PTSD, a distinction is made between the subjective and objective assessment of sleep quality, with a call made for an emphasis on objective measurements in future research.
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Press, Minkyo. Sleep All Day Architecture All Night: 3 Column Ledger. Independently Published, 2018.

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Press, Minkyo. Sleep All Day Architecture All Night: Unruled Composition Book. Independently Published, 2018.

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Press, Minkyo. Sleep All Day Architecture All Night: 4 Column Ledger. Independently Published, 2018.

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Części książek na temat "Sleep architecture"

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Insana, Salvatore. "Sleep Architecture". W Encyclopedia of Behavioral Medicine, 1803–5. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_842.

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Guion, Kim, i Kristin T. Avis. "Sleep Architecture". W Encyclopedia of Child Behavior and Development, 1372–74. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_2664.

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Insana, Salvatore. "Sleep Architecture". W Encyclopedia of Behavioral Medicine, 2056–58. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_842.

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Herdegen, James J. "Chronic Disease and Sleep Architecture". W Sleep and Sleep Disorders, 163–69. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/0-387-27682-3_18.

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Häuplik-Meusburger, Sandra. "Human Activity Sleep". W Architecture for Astronauts, 97–128. Vienna: Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0667-9_4.

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Au, Chun-Ting. "Sleep Physiology and Architecture". W Paediatric Sleep Disorders, 9–16. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-5791-8_2.

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Karmakar, Nemai Chandra, Yang Yang i Abdur Rahim. "Wireless On-Body Sensor Architecture". W Microwave Sleep Apnoea Monitoring, 79–99. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-6901-7_6.

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Dorffner, Georg, Martin Vitr i Peter Anderer. "The Effects of Aging on Sleep Architecture in Healthy Subjects". W Advances in Experimental Medicine and Biology, 93–100. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-08939-3_13.

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Tonchev, Krasimir, Georgi Tsenov, Valeri Mladenov, Agata Manolova i Vladimir Poulkov. "Personalized and Intelligent Sleep Lifestyle Reasoner with Web Application for Improving Quality of Sleep Part of AAL Architecture". W Lecture Notes of the Institute for Computer Sciences, Social Informatics and Telecommunications Engineering, 107–12. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-74935-8_15.

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Perantoni, E., P. Steiropoulos, D. Filos, N. Maglaveras, K. Nikolaou i I. Chouvarda. "Association Between SpO2 Signal Characteristics and Sleep Architecture with Insulin Resistance in Patients with Obstructive Sleep Apnea Syndrome". W Precision Medicine Powered by pHealth and Connected Health, 119–23. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-7419-6_21.

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Streszczenia konferencji na temat "Sleep architecture"

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Liu, Juan, Qin Li, Yi Xin i Xiao Lu. "Obstructive Sleep Apnea Detection Using Sleep Architecture". W 2020 IEEE International Conference on Mechatronics and Automation (ICMA). IEEE, 2020. http://dx.doi.org/10.1109/icma49215.2020.9233529.

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Barnes, Maree, i Danielle Wilson. "Sleep Architecture And Sleep Disordered Breathing In Pregnancy". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6529.

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Traxdorf, M., P. Krauss, K. Tziridis i H. Schulze. "Architecture of sleep: automated sleep stage analysis based on spatial EEG patterns". W Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640976.

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Lee, Hoyoung, Jing Liu i Sunyoung Han. "Practical Multicast Security Architecture Using Sleep State". W 22nd International Conference on Advanced Information Networking and Applications - Workshops (aina workshops 2008). IEEE, 2008. http://dx.doi.org/10.1109/waina.2008.191.

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Tataraidze, Alexander, Lyudmila Korostovtseva, Lesya Anishchenko, Mikhail Bochkarev i Yurii Sviryaev. "Sleep architecture measurement based on cardiorespiratory parameters". W 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7591477.

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Zambrano Chacon, M. D. L. A., J. Herrero Huertas, M. F. Troncoso Acevedo, M. J. Rodríguez Guzmán, C. P. Esteban Amarilla, L. Núñez García, L. De La Dueña Muñóz, C. López Chang i M. J. Rodríguez Nieto. "Sleep-tracking device compared to polysomnography for respiratory events and sleep architecture analysis". W ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2729.

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Li, Z. H., Z. Q. Yu i W. L. Zhang. "A Sleep Monitoring System Based on Cloud Architecture". W 2015 International Conference on Artificial Intelligence and Industrial Engineering. Paris, France: Atlantis Press, 2015. http://dx.doi.org/10.2991/aiie-15.2015.30.

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Jia, Ziyu, Youfang Lin, Jing Wang, Xuehui Wang, Peiyi Xie i Yingbin Zhang. "SalientSleepNet: Multimodal Salient Wave Detection Network for Sleep Staging". W Thirtieth International Joint Conference on Artificial Intelligence {IJCAI-21}. California: International Joint Conferences on Artificial Intelligence Organization, 2021. http://dx.doi.org/10.24963/ijcai.2021/360.

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Sleep staging is fundamental for sleep assessment and disease diagnosis. Although previous attempts to classify sleep stages have achieved high classification performance, several challenges remain open: 1) How to effectively extract salient waves in multimodal sleep data; 2) How to capture the multi-scale transition rules among sleep stages; 3) How to adaptively seize the key role of specific modality for sleep staging. To address these challenges, we propose SalientSleepNet, a multimodal salient wave detection network for sleep staging. Specifically, SalientSleepNet is a temporal fully convolutional network based on the $U^2$-Net architecture that is originally proposed for salient object detection in computer vision. It is mainly composed of two independent $U^2$-like streams to extract the salient features from multimodal data, respectively. Meanwhile, the multi-scale extraction module is designed to capture multi-scale transition rules among sleep stages. Besides, the multimodal attention module is proposed to adaptively capture valuable information from multimodal data for the specific sleep stage. Experiments on the two datasets demonstrate that SalientSleepNet outperforms the state-of-the-art baselines. It is worth noting that this model has the least amount of parameters compared with the existing deep neural network models.
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Selvaraj, Nandakumar. "Screening of sleep architecture using a disposable patch sensor". W 2017 IEEE EMBS International Conference on Biomedical & Health Informatics (BHI). IEEE, 2017. http://dx.doi.org/10.1109/bhi.2017.7897226.

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Wang, Yanwen, Hainan Chen, Xiaoling Wu i Lei Shu. "Improving WSNs sleep scheduling mechanism with SDN-like architecture". W the 14th International Conference. New York, New York, USA: ACM Press, 2015. http://dx.doi.org/10.1145/2737095.2742011.

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Raporty organizacyjne na temat "Sleep architecture"

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Basis, Najwa, i Tamar Shochat. Associations between religion and sleep: A systematic review of observational studies in the adult population. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, lipiec 2022. http://dx.doi.org/10.37766/inplasy2022.7.0057.

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Review question / Objective: The aim of this systematic review is to investigate observational studies on the association between religion and sleep in the adult population. To this end, the proposed systematic review will address the following question: What is the role religion plays in shaping an individual's sleep health? Condition being studied: Sleep is a fundamental biological process increasingly recognized as a critical indicator of development and overall health. Generally, insufficient sleep is associated with depressed mood, daytime fatigue, poor daytime functioning and daytime sleepiness, increased risk of cancer, cardiovascular problems, diabetes, and the cause of the higher risk of mortality. Furthermore, changes in sleep architecture and quality have been related to cognitive deterioration, including dementia and Alzheimer's disease. Here we will identify the role of religion in elements of sleep health, to include sleep duration and sleep quality, and associated health outcomes in the adult population.
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Thangavel, Kalavathi Devi, Sakthivel Palaniappan i Sathish Kumar Shanmugam. Performance Analysis of VLSI Architecture of Viterbi Decoder in WLAN Using the Sleepy Keeper Technique. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, sierpień 2020. http://dx.doi.org/10.7546/crabs.2020.08.11.

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