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Książki na temat „Skin Blood-vessels”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 31 stycznia 2023

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Sprawdź 27 najlepszych książek naukowych na temat „Skin Blood-vessels”.

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1

Salmon, Michel. Arteries of the skin. Redaktorzy Taylor G. Ian i Tempest Michael N. London: Churchill Livingstone, 1988.

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2

Ian, Taylor G., i Tempest Michael N, red. Arteries of the skin. London: Churchill Livingstone, 1988.

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3

Enzo, Berardesca, Elsner Peter 1955- i Maibach Howard I, red. Bioengineering of the skin: Cutaneous blood flow and erythema. Boca Raton: CRC Press, 1995.

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4

H, Lamberty B. George, red. The arterial anatomy of skin flaps. Edinburgh: Churchill Livingstone, 1986.

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5

Cormack, George C. The arterial anatomy of skin flaps. Wyd. 2. Edinburgh: Churchill Livingstone, 1994.

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6

Cormack, George C. The arterial anatomy of skin flaps. Edinburgh: Churchill Livingstone, 1986.

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7

The three-edged sword: Being ill in America. Lanham, MD: University Press of America, 1992.

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8

T, Tan Oon, red. Management and treatment of benign cutaneous vascular lesions. Philadelphia: Lea & Febiger, 1992.

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9

Sangüeza, Omar P. Pathology of vascular skin lesions: Clinicopathological correlations. Totowa, N.J: Humana Press, 2003.

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10

National Conference on the Diabetic Foot (1st 1986 Malvern, Worcestershire). The foot in diabetes: Proceedings of the 1st National Conference on the Diabetic Foot, Malvern, May 1986. Chichester: Wiley, 1987.

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11

Dix, Jay. Handbook for death scene investigators. Boca Raton, Fla: CRC Press, 1999.

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12

Sangueza, MD Omar P., i MD Luis Requena. Pathology of Vascular Skin Lesions: Clinicopathologic Correlations. Humana Press, 2010.

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13

Case of laceration of the intestine with rupture of the mesenteric artery without a skin wound. [S.l: s.n., 1985.

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14

(Editor), Enzo Berardesca, Peter Elsner (Editor) i Howard I. Maibach (Editor), red. Bioengineering of the Skin: Cutaneous Blood Flow and Erythema, Volume II. Wyd. 2. CRC, 1994.

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15

Sangüeza, Omar P., i Luis Requena. Pathology of Vascular Skin Lesions (Current Clinical Pathology). Humana Press, 2003.

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16

Soon, Christine. Cutaneous vasculitis. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0247.

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Vasculitis is inflammation of the blood vessels. It can affect large vessels, medium-sized vessels, or small vessels. Cutaneous vasculitis is inflammation of the blood vessels of the skin. This can include the capillaries, the venules, and the arterioles.

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17

Leonard, Barry. Prevent Diabetes Problems: Eyes, Teeth and Gums, Kidneys, Feet and Skin, Heart, and Blood Vessels, and Nervous System. Diane Pub Co, 2002.

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18

Coleridge Smith, P. D. 1953-, red. Microcirculation in venous disease. Austin: R.G. Landes, 1994.

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19

Microcirculation in venous disease. Wyd. 2. Austin, Tex., U.S.A: Landes Bioscience, 1998.

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20

Luqmani, Raashid. Vasculitis. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0272.

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The vasculitides are a heterogeneous group of disorders that can range from mild inflammation of blood vessels in the skin, to organ- and life-threatening diseases. The term ‘vasculitis’ is a pathological description of blood vessel wall inflammation which leads to ischaemia and infarction of the target organs. Definitions and classifications of the primary vasculitides are mainly based on the predominant calibre of the blood vessels involved but incorporate clinical, pathological, and laboratory features. The secondary vasculitides usually occur in the context of other connective tissue diseases and are not discussed further in this section. Goodpasture’s disease is not usually included in the primary vasculitides, but has compatible clinical features of pulmonary capillaritis and glomerulonephritis.

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21

(Editor), Andrew J. Boulton, A. J. Boulton (Editor) i J. D. Ward (Editor), red. The Foot in Diabetes: Proceedings of the 1st National Conference on the Diabetic Foot, Malvern, May 1986 (Wiley Medical Publication). John Wiley & Sons, 1987.

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22

Taylor, Jennie, i Patrick Y. Wen. Meningomas. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0130.

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Meningiomas are the most common primary brain tumor diagnosed in adults. Arising from the arachnoid (meningothelial) cells of the inner layer of the dura, they are often slow growing, but can lead to significant morbidity. They can invade through the outer layer of dura into overlying bone or skin, or into critical bordering structures such as the cavernous sinus or orbits, or encase cerebral blood vessels. These limitations can make surgical resection difficult if not impossible in some circumstance. However, they rarely metastasize outside the central nervous system (CNS), with the lungs being the most common site seen with higher-grade tumors.

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23

Price, Susan. Genetic bone and joint disease. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0276.

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Genetic conditions affecting the skeleton and supporting structures are individually rare and heterogeneous. This chapter presents an approach to assessing patients with suspected skeletal dysplasia, osteogenesis imperfecta, Marfan syndrome, and Ehlers–Danlos syndrome. Skeletal dysplasias are caused by abnormalities of bone growth and modelling; the commonest non-lethal type is achondroplasia, with an incidence of 1/10 000 to 1/30 000. The typical presentation of osteogenesis imperfecta is with multiple fractures, sometimes prenatally. There may be associated short stature, bone deformity, dentogenesis imperfecta, blue sclera, and hearing loss. Most patients with osteogenesis imperfecta have mutations in COL1A1 or COL1A2. Marfan syndrome is a connective tissue disease with a pattern of symptoms related to the presence of fibrillin in tissues. Typically, affected individuals are of tall, thin stature, with long fingers and toes (arachnodactyly), a pectus deformity, and scoliosis. Between 66% and 91% of individuals with Marfan syndrome have a mutation in fibrillin-1 (FBN1; locus: 15q21). All forms of Ehlers–Danlos syndrome present with variable thinning and fragility of skin, leading to easy bruising and poor scar formation. There is skin and joint laxity. In severe forms, blood vessels and internal organs are affected.

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24

Dix, Jay. Handbook for Death Scene Investigators. Taylor & Francis Group, 2017.

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25

Dix, Jay. Handbook for Death Scene Investigators. Taylor & Francis Group, 2017.

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26

Dix, Jay. Handbook for Death Scene Investigators. Taylor & Francis Group, 2017.

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27

Dix, Jay, i Mary Fran Ernst. Handbook for Death Scene Investigators. Taylor & Francis Group, 2010.

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