Gotowe bibliografie tematyczne / Sin1

Gotowa bibliografia na temat „Sin1”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 20 lutego 2023

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Artykuły w czasopismach na temat "Sin1"

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McCarthy, Nicola. "An original SIN1". Nature Reviews Cancer 13, nr 12 (22.11.2013): 823. http://dx.doi.org/10.1038/nrc3641.

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Kruger, W., i I. Herskowitz. "A negative regulator of HO transcription, SIN1 (SPT2), is a nonspecific DNA-binding protein related to HMG1". Molecular and Cellular Biology 11, nr 8 (sierpień 1991): 4135–46. http://dx.doi.org/10.1128/mcb.11.8.4135-4146.1991.

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The SIN1 gene was initially identified because mutations in SIN1 bypass the need for SWI1 to activate transcription of the yeast HO gene. We show here that transcription of HO in swi1 sin1 cells efficiently utilizes the normal start site. We have cloned SIN1 and found that it is identical to the previously identified gene SPT2, mutations in which allow transcription from certain mutated regulatory regions. The predicted SIN1/SPT2 protein has a distinctive amino acid composition (45% charged residues, 25% basic and 20% acidic) and has similarity to the mammalian HMG1 protein, a nonhistone component of chromatin. We show that SIN1 is concentrated in the nucleus and binds to DNA with little or no sequence specificity in vitro. It thus exhibits properties of an HMG protein. Addition of random DNA segments to a test promoter alters regulation by SIN1 in a manner similar to addition of a segment from the HO upstream region. Functional analysis of certain SIN1 mutations suggests that SIN1 may be part of a multiprotein complex. On the basis of these results, we propose that SIN1 is a nonhistone component of chromatin which creates the proper context for transcription. Because sin1 mutants exhibit increased loss of chromosome III, SIN1 may also play a role in fidelity of chromosome segregation.
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Kruger, W., i I. Herskowitz. "A negative regulator of HO transcription, SIN1 (SPT2), is a nonspecific DNA-binding protein related to HMG1." Molecular and Cellular Biology 11, nr 8 (sierpień 1991): 4135–46. http://dx.doi.org/10.1128/mcb.11.8.4135.

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The SIN1 gene was initially identified because mutations in SIN1 bypass the need for SWI1 to activate transcription of the yeast HO gene. We show here that transcription of HO in swi1 sin1 cells efficiently utilizes the normal start site. We have cloned SIN1 and found that it is identical to the previously identified gene SPT2, mutations in which allow transcription from certain mutated regulatory regions. The predicted SIN1/SPT2 protein has a distinctive amino acid composition (45% charged residues, 25% basic and 20% acidic) and has similarity to the mammalian HMG1 protein, a nonhistone component of chromatin. We show that SIN1 is concentrated in the nucleus and binds to DNA with little or no sequence specificity in vitro. It thus exhibits properties of an HMG protein. Addition of random DNA segments to a test promoter alters regulation by SIN1 in a manner similar to addition of a segment from the HO upstream region. Functional analysis of certain SIN1 mutations suggests that SIN1 may be part of a multiprotein complex. On the basis of these results, we propose that SIN1 is a nonhistone component of chromatin which creates the proper context for transcription. Because sin1 mutants exhibit increased loss of chromosome III, SIN1 may also play a role in fidelity of chromosome segregation.
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Xu, Yanyan, Xue Chen i Junling Liu. "The Critical Roles of SIN1 in Platelet Activation and Myocardial Infarction". Blood 128, nr 22 (2.12.2016): 3715. http://dx.doi.org/10.1182/blood.v128.22.3715.3715.

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Abstract Mammalian stress-activated protein kinase interacting protein 1 (SIN1) is an essential subunit of the mTORC2 complex, which regulates Akt activation by phosphorylation of Akt at Ser473 residue. Despite the function of Akt in platelet activation and thrombosis was well studied, the role of SIN1 in platelet activation and thrombosis remains unknown. In this study, we observed that megakaryocyte/platelet specific SIN1 deficiency caused 30% reduction of platelet counts in peripheral blood probably by blockage of megakaryocyte differentiation and enhancing platelet apoptosis, suggesting that SIN1 had an important role in thrombopoiesis. More importantly, SIN1 deficiency caused a defect in platelet aggregation in response to low level of thrombin, U46619, ADP and collagen. SIN1 deficiency also exhibited diminished ability of platelet to spread on immobilized fibrinogen and the decreased rate of clot retraction in platelet-rich plasma containing SIN1 deficient platelets. mTORC2 complex analysis revealed that the expression levels of Rictor, another mTORC2 component, were significantly diminished in SIN1 deficient platelet. And SIN1 deficiency attenuated agonist-induced phosphorylation of Akt at Ser473, Thr308 and Thr450, and Gsk3β at Ser9 in platelet. SIN1 could be phosphorylated at Thr86, which correlated with the phosphorylation of Akt at Ser473 in activated platelets. Further study demonstrated that the phosphorylation levels of SIN1 at Thr86 and Akt at Ser473 and Thr450, but not at Thr308 were enhanced in the platelets collected from ST-segment elevation myocardial infarction (STEMI) patients, indicating that SIN1 activation correlated with myocardial infarction process. A mouse model of chronic myocardial infarction (MI) was performed and the results demonstrated that platelet-specific SIN1 deficient mice had less platelet activation, reduced MI size, and improved post-MI heart function. In conclusion, SIN1 plays critical roles in platelet activation, MI and post-MI heart failure, therefore serves as a target for therapeutic intervention in the thrombosis and myocardial infarction. Disclosures No relevant conflicts of interest to declare.
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Ray, A., J. D. Lang, T. Golden i S. Ray. "SHORT INTEGUMENT (SIN1), a gene required for ovule development in Arabidopsis, also controls flowering time". Development 122, nr 9 (1.09.1996): 2631–38. http://dx.doi.org/10.1242/dev.122.9.2631.

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The short integument (sin1) mutation causes a female-specific infertility, and a defect in the control of time to flowering in Arabidopsis. Female sterility of Sin- plants is due to abnormal ovule integument development and aberrant differentiation of the megagametophyte in a subset of ovules. An additional defect of sin1 mutants is the production of an increased number of vegetative leaf and inflorescence primordia leading to delayed flowering. The delayed flowering phenotype of sin1-1 is not due to a defect in the perception of day length periodicity or in gibberellic acid metabolism. Phenotypes of double mutant combinations of sin1 with terminalflower (tfl1) indicate that SIN1 activity is required for precocious floral induction typical in a tfl1 mutant. Unexpectedly, sin1-1 tfl1-1 plants do not make pollen, thus revealing a novel role for TFL1 in the anther. Early flowers of sin1-1 ap1-1 double mutants are transformed to long inflorescence-like shoots. A genetic model for the role of SIN1 in flowering time control is proposed.
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Zhou, Yang. "Exploring the emergence of the postverbal sin1 先 in Cantonese". Language and Linguistics / 語言暨語言學 19, nr 2 (15.03.2018): 333–75. http://dx.doi.org/10.1075/lali.00012.zho.

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Abstract Sin1 先 as a function word in contemporary Cantonese encodes a number of grammatical and pragmatic meanings. As its most prominent feature in syntax, it predominantly occurs in the postverbal position while indicating the meaning of ‘first’. This paper explores the emergence of the postverbal sin1 先 ‘first’ in Cantonese. We first examine the word order typology on the element for ‘first’ in the languages and dialects of southern coastal China. In this linguistic area, the postverbal elements for ‘first’ in Chinese dialects are contact-induced by Tai-Kadai and Hmong-Mien languages; whereas sin1 先 ‘first’ in the mainstream Cantonese shows a stronger tendency to be placed in the postverbal position than its counterparts in other Chinese dialects. We then discuss the word order and semantic changes of sin1 先 from 1820s to 1960s based on Cantonese historical materials. Besides the pressure of language contact, the formation of the postverbal sin1 先 ‘first’ has been further triggered by the semantic motivation to formally differentiate the ‘precedent-subsequent’ polysemy within sin1 先 itself. In short, the emergence of the postverbal sin1 先 ‘first’ in Cantonese has been a two-stage process, dually driven by external and internal causes, respectively.
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Cameron, Angus J. M., Mark D. Linch, Adrian T. Saurin, Cristina Escribano i Peter J. Parker. "mTORC2 targets AGC kinases through Sin1-dependent recruitment". Biochemical Journal 439, nr 2 (28.09.2011): 287–97. http://dx.doi.org/10.1042/bj20110678.

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The protein kinase TOR (target of rapamycin) is a key regulator of cell growth and metabolism with significant clinical relevance. In mammals, TOR signals through two distinct multi-protein complexes, mTORC1 and mTORC2 (mammalian TOR complex 1 and 2 respectively), the subunits of which appear to define the operational pathways. Rapamycin selectively targets mTORC1 function, and the emergence of specific ATP-competitive kinase inhibitors has enabled assessment of dual mTORC1 and mTORC2 blockade. Little is known, however, of the molecular action of mTORC2 components or the relative importance of targeting this pathway. In the present study, we have identified the mTORC2 subunit Sin1 as a direct binding partner of the PKC (protein kinase C) ϵ kinase domain and map the interaction to the central highly conserved region of Sin1. Exploiting the conformational dependence for PKC phosphorylation, we demonstrate that mTORC2 is essential for acute priming of PKC. Inducible expression of Sin1 mutants, lacking the PKC-interaction domain, displaces endogenous Sin1 from mTORC2 and disrupts PKC phosphorylation. PKB (protein kinase B)/Akt phosphorylation is also suppressed by these Sin1 mutants, but not the mTORC1 substrate p70S6K (S6 kinase), providing evidence that Sin1 serves as a selectivity adaptor for the recruitment of mTORC2 targets. This inducible selective mTORC2 intervention is used to demonstrate a key role for mTORC2 in cell proliferation in three-dimensional culture.
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Castel, Pau, Srisathiyanarayanan Dharmaiah, Matthew J. Sale, Simon Messing, Gabrielle Rizzuto, Antonio Cuevas-Navarro, Alice Cheng i in. "RAS interaction with Sin1 is dispensable for mTORC2 assembly and activity". Proceedings of the National Academy of Sciences 118, nr 33 (11.08.2021): e2103261118. http://dx.doi.org/10.1073/pnas.2103261118.

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RAS proteins are molecular switches that interact with effector proteins when bound to guanosine triphosphate, stimulating downstream signaling in response to multiple stimuli. Although several canonical downstream effectors have been extensively studied and tested as potential targets for RAS-driven cancers, many of these remain poorly characterized. In this study, we undertook a biochemical and structural approach to further study the role of Sin1 as a RAS effector. Sin1 interacted predominantly with KRAS isoform 4A in cells through an atypical RAS-binding domain that we have characterized by X-ray crystallography. Despite the essential role of Sin1 in the assembly and activity of mTORC2, we find that the interaction with RAS is not required for these functions. Cells and mice expressing a mutant of Sin1 that is unable to bind RAS are proficient for activation and assembly of mTORC2. Our results suggest that Sin1 is a bona fide RAS effector that regulates downstream signaling in an mTORC2-independent manner.
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Pérez-Martín, José, i Alexander D. Johnson. "The C-Terminal Domain of Sin1 Interacts with the SWI-SNF Complex in Yeast". Molecular and Cellular Biology 18, nr 7 (1.07.1998): 4157–64. http://dx.doi.org/10.1128/mcb.18.7.4157.

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ABSTRACT In the yeast Saccharomyces cerevisiae, the SWI-SNF complex has been proposed to antagonize the repressive effects of chromatin by disrupting nucleosomes. The SIN genes were identified as suppressors of defects in the SWI-SNF complex, and theSIN1 gene encodes an HMG1-like protein that has been proposed to be a component of chromatin. Specific mutations (sin mutations) in both histone H3 and H4 genes produce the same phenotypic effects as do mutations in the SIN1 gene. In this study, we demonstrate that Sin1 and the H3 and H4 histones interact genetically and that the C terminus of Sin1 physically associates with components of the SWI-SNF complex. In addition, we demonstrate that this interaction is blocked in the full-length Sin1 protein by the N-terminal half of the protein. Based on these and additional results, we propose that Sin1 acts as a regulatable bridge between the SWI-SNF complex and the nucleosome.
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Zdetsis, Aristides D. "Fluxional and aromatic behavior in small magic silicon clusters: A full ab initio study of Sin, Sin1−, Sin2−, and Sin1+, n=6, 10 clusters". Journal of Chemical Physics 127, nr 1 (7.07.2007): 014314. http://dx.doi.org/10.1063/1.2746030.

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Rozprawy doktorskie na temat "Sin1"

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Cloonan, Nicole, i N/A. "Sin1 and Sin1 Isoforms: An Investigation into the Biological Significance of a Novel Human Protein Family". Griffith University. School of Biomolecular and Biomedical Science, 2006. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20071102.150237.

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Stress activated protein kinase (SAPK) interacting protein 1 (Sin1) is a member of a recently characterized gene family, conserved from yeast to humans. The gene copy number is strictly conserved (one Sin1 gene per genome), and the protein may be expressed ubiquitously in mammalian tissues. The Sin1 family has been implicated in several different signal transduction pathways. Originally identified as a partial cDNA and candidate Ras inhibitor, recent functional studies have revealed interactions with an interferon (IFN) receptor subunit (IFNAR2), and the SAPK JNK. Interactions have also been described between the yeast orthologues and the phosphatidylinositol kinase TOR2. Collectively, these data suggest that Sin1 has an important cellular role, and this study has investigated possible functions for this protein. As human Sin1 proteins have no paralogues within the genome, secondary structure homology was used to identify major domains within the protein. Four major domains within human Sin1 were deduced: an N-terminal domain containing a functional nuclear localization signal, a functional nuclear export signal, and a coiledcoil region; the conserved region in the middle that is likely to be a ubiquitin-like β-grasp protein binding domain; a Ras binding domain; and a pleckstrin homology-like domain that targets Sin1 to the plasma membrane and lipid rafts in vivo. Full and partial length EGFP constructs were used to examine the localization of human Sin1, and several isoforms derived from alternative splicing. All isoforms localized to the nucleus and nucleolus. Beyond this, Sin1α and Sin1ϒ had cytoplasmic staining, while Sin1 and Sin1β were also found at the plasma membrane and lipid rafts. Both the N-terminal domain and the conserved region in the middle were found to contribute to nuclear localization. Comparative genomic analysis between human, mouse, rat, dog, and chicken Sin1 genes revealed a number of conserved intronic regions, and the putative functions of these were predicted. Additionally, a putative promoter module within a CpG island and encompassing the transcription start site was predicted in all species. The human CpG island was found to have promoter activity in HEK293 cells. Using bioinformatics, genes that may be co-regulated with Sin1 were identified. These genes contained the Sin1 promoter module, and were found to co-express in large scale gene expression studies. Most of these genes were directly involved in the cellular response to pathogen infection, suggesting a conserved role for Sin1 in this pathway. Key biochemical functions of the Sin1 proteins were also identified, including the ability of Sin1 proteins to form dimers, and the ability of over-expressed Sin1 to induce apoptosis (mediated through the conserved region in the middle). Additionally, endogenous Sin1 protein levels were found to change following serum deprivation and hypoosmotic stress. Together, these studies have provided significant insight into the cellular role of Sin1, suggesting a role in inducing apoptosis as part of the IFN response to viral infection. The biological significance of the Sin1 proteins is discussed in the context of their predicted functions and the evolution of the protein family.
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Cloonan, Nicole. "Sin1 and Sin1 Isoforms: An Investigation into the Biological Significance of a Novel Human Protein Family". Thesis, Griffith University, 2006. http://hdl.handle.net/10072/367210.

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Stress activated protein kinase (SAPK) interacting protein 1 (Sin1) is a member of a recently characterized gene family, conserved from yeast to humans. The gene copy number is strictly conserved (one Sin1 gene per genome), and the protein may be expressed ubiquitously in mammalian tissues. The Sin1 family has been implicated in several different signal transduction pathways. Originally identified as a partial cDNA and candidate Ras inhibitor, recent functional studies have revealed interactions with an interferon (IFN) receptor subunit (IFNAR2), and the SAPK JNK. Interactions have also been described between the yeast orthologues and the phosphatidylinositol kinase TOR2. Collectively, these data suggest that Sin1 has an important cellular role, and this study has investigated possible functions for this protein. As human Sin1 proteins have no paralogues within the genome, secondary structure homology was used to identify major domains within the protein. Four major domains within human Sin1 were deduced: an N-terminal domain containing a functional nuclear localization signal, a functional nuclear export signal, and a coiledcoil region; the conserved region in the middle that is likely to be a ubiquitin-like β-grasp protein binding domain; a Ras binding domain; and a pleckstrin homology-like domain that targets Sin1 to the plasma membrane and lipid rafts in vivo. Full and partial length EGFP constructs were used to examine the localization of human Sin1, and several isoforms derived from alternative splicing. All isoforms localized to the nucleus and nucleolus. Beyond this, Sin1α and Sin1ϒ had cytoplasmic staining, while Sin1 and Sin1β were also found at the plasma membrane and lipid rafts. Both the N-terminal domain and the conserved region in the middle were found to contribute to nuclear localization. Comparative genomic analysis between human, mouse, rat, dog, and chicken Sin1 genes revealed a number of conserved intronic regions, and the putative functions of these were predicted. Additionally, a putative promoter module within a CpG island and encompassing the transcription start site was predicted in all species. The human CpG island was found to have promoter activity in HEK293 cells. Using bioinformatics, genes that may be co-regulated with Sin1 were identified. These genes contained the Sin1 promoter module, and were found to co-express in large scale gene expression studies. Most of these genes were directly involved in the cellular response to pathogen infection, suggesting a conserved role for Sin1 in this pathway. Key biochemical functions of the Sin1 proteins were also identified, including the ability of Sin1 proteins to form dimers, and the ability of over-expressed Sin1 to induce apoptosis (mediated through the conserved region in the middle). Additionally, endogenous Sin1 protein levels were found to change following serum deprivation and hypoosmotic stress. Together, these studies have provided significant insight into the cellular role of Sin1, suggesting a role in inducing apoptosis as part of the IFN response to viral infection. The biological significance of the Sin1 proteins is discussed in the context of their predicted functions and the evolution of the protein family.
Thesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Biomolecular and Biomedical Sciences
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Paramo, Sanchez Blanca Estela. "The role of Sin1 in cell survival". Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-sin1-in-cell-survival(10b70b16-0e06-405f-a89e-caaae8f5974d).html.

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Cancer and neurodegeneration are detrimental conditions associated with an inappropriate regulation of cell survival and cell death, causing compromised cells to evade death or excessive death of healthy neurons. The mammalian target of rapamycin complex 2 (mTORC2) has been implicated in the regulation of cell survival by phosphorylating the protein kinase Akt. This is dependent upon the scaffold protein Sin1, a core component of mTORC2. The requirement of Sin1 in cell survival, and in particular in neuronal survival, has not been established due to the early embryonic lethality of mice with a targeted deletion of the Sin1 gene. To circumvent this issue, a novel conditional mouse knockout model was established. The role of Sin1 in regulating cell survival was evaluated in fibroblasts and cortical neurons. The loss of Sin1 significantly affected the phosphorylation and activity of Akt in fibroblasts and caused a reduction in cell survival by potentially inducing premature senescence. In contrast, the loss of Sin1 caused an increase in caspase-independent cell death in cortical neurons. Gene-expression analysis of Sin1 knockout cortical neurons demonstrated an important down-regulation of transcription factors, cytoskeletal proteins and components of signalling pathways involved in neuronal survival, aiding to uncover the mechanism by which Sin1 promotes neuronal survival. Taken together, the results presented in this study show a key role of the scaffold protein Sin1 in regulating neuronal survival.
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Mavroudis-Chocholis, Orestis. "Characterization of the stress-dependent phosphorylation of Sin1". Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.509385.

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Marafie, Sulaiman. "TRIM7, a novel binding protein of the mTORC2 component Sin1". Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/trim7-a-novel-binding-protein-of-the-mtorc2-component-sin1(c344b542-0706-4ec0-be06-ce6683cee52e).html.

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TRIM7 is a member of the TRIM (tripartite motif-containing) protein superfamily. This family has been implicated in many disorders such as genetic diseases, neurological diseases, and cancers. Little is known about the function of TRIM7 except that it interacts with glycogenin and may regulate glycogen biosynthesis. Recently, a yeast two-hybrid protein-protein interaction screen revealed the binding of TRIM7 to Sin1, a protein found in a complex with the mammalian target of rapamycin (mTOR) protein kinase. mTOR can form two complexes, mTORC1 and mTORC2, which are important for cell growth, differentiation, and survival. Sin1 is a core component of mTORC2 and is critical for mTORC2 stability and activity. It was confirmed by co-immunoprecipitation that TRIM7 associates with Sin1 and mTOR in cultured mammalian cells. Furthermore, it was demonstrated that TRIM7 is a phosphoprotein, although it was not directly targeted by mTOR in vitro. Similar to some other TRIM family proteins, it was demonstrated that TRIM7 has a ubiquitin E3 ligase function allowing it to autoubiquitinate both in vitro and in cells. The autoubiquitination of TRIM7 was dependent on its RING domain. Further characterization of TRIM7 indicated that it can both homo-oligomerise as well as hetero-oligomerise with other members of its sub-class of TRIM proteins and that it co-localises with them into discrete cytoplasmic loci. To determine the cellular function of TRIM7, a stable cell line expressing an shRNA directed against TRIM7 was generated. Successful knock down of TRIM7 was achieved and this led to an increase in the protein levels of components of the mTORC2 complex, including Sin1. This coincided with an increase in cell proliferation. In conclusion, this research identifies a novel role for TRIM7 as a ubiquitin ligase involved in regulating cell proliferation and provides a potential link between TRIM7 and the mTOR pathway, a major transducer of proliferative and cell survival signals.
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Clark, Lorna Evelyn. "The role of Sin1 in mammalian stress-responsive protein kinase signalling". Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495740.

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Cells respond to changes in their extra- and intracellular environments through the activation of signa ng pamways. Regulatory proteins function to introduce effidency and specifity to falling networks. Sin1 is an evolutionary conserved protein, which could function as a regulator of cell signalling.
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Thebault, Philippe. "Le facteur d'élongation SPT2/SIN1 est impliqué dans l'assemblage du nucléosome couplé à la transcription". Thesis, Université Laval, 2009. http://www.theses.ulaval.ca/2009/26726/26726.pdf.

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Wang, Shu-Zong. "Cloning and characterization of SAPK interacting protein 1 (SIN1), a type 1 IFN receptor subunit 2 (IFNAR2)-interacting protein /". Free to MU Campus, others may purchase, 2004. http://wwwlib.umi.com/cr/mo/fullcit?p3137761.

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Eriksson, Susanne. "Högbegåvade ungdomars upplevelser av sina liv och sin vardag". Thesis, Stockholms universitet, Psykologiska institutionen, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-145433.

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Högbegåvade barn och ungdomar har uppmärksammats mer i Sverige de senaste åren. Litteraturen presenterar divergerande uppfattningar kring om hög begåvning främst är en skyddsfaktor eller en sårbarhetsfaktor. Syftet med föreliggande studie var att undersöka högbegåvade ungdomars upplevelser av sina liv och sin vardag. Tio högbegåvade tonåringar intervjuades och materialet bearbetades med induktiv tematisk analys med deduktiva inslag som resulterade i ett övergripande huvudtema högbegåvade tonåringars energiflöde i en modell som kan utläsas ”Jag har stora energikällor och om jag får utlopp för energin i dessa genom intellektuell stimulans och gemenskap bidrar detta till god psykisk hälsa vilket i sin tur ger mer energi. Om energin hindras av t.ex. brist på intellektuell stimulans och gemenskap bidrar det till psykisk ohälsa och minskad energi.” Resultatet diskuteras utifrån betydelse för frigörande av potential och förbyggande av psykisk ohälsa.
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Glacet, Jean-Yves. "Propriétés électroniques des interfaces SiN/Si et SiNx/GalnAs". Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb37597926x.

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Książki na temat "Sin1"

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Seven deadly sins: A very partial list. Cambridge, Mass: Harvard University Press, 2008.

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Hoag, Tami. Night sins: Guilty as sin. London: Orion, 2003.

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Hoag, Tami. Night sins: Guilty as sin. New York: Bantam Books, 2008.

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Hoag, Tami. Night sins: Guilty as sin. New York: Bantam Books, 2008.

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Hoag, Tami. Night sins and guilty as sin. [Place of publication not identified]: Orion Books Ltd., 2006.

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Houziaux, Alain. Ces péchés capitaux-- si capiteux. Paris: Lethielleux groupe DDB, 2011.

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Nichiren kyōgaku ni okeru tsumi no kenkyū. Kyōto-shi: Heirakuji Shoten, 1999.

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Weiss, Heipe. Das Lexikon der Sünde. Frankfurt am Main: Eichborn, 1989.

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Nanthavongdūangsī, Vīangkham. Sin kap mǣying Lāo =: Sinh and Lao women. [Vientiane]: Hatthakam Phǣng Mai, 2006.

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Shivʻat ha-ḥaṭaʼim: Reshimah ḥelḳit. Tel-Aviv: Sifre ʻAliyat ha-gag, 2007.

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Części książek na temat "Sin1"

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Asendorpf, Jens B. "Sinn der Vielfalt: Unterschiede sind menschlich". W Persönlichkeit: was uns ausmacht und warum, 341–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-56106-5_19.

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Henriksen, Jan-Olav. "From Sin to Sins and Back". W Theological Anthropology in the Anthropocene, 211–17. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-21058-7_16.

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Schiffers, Maximilian. "Praktische Implikationen und Bausteine moderner Interessenvermittlung". W NGOs als besondere Akteure der Interessenvermittlung, 103–13. Wiesbaden: Springer Fachmedien Wiesbaden, 2021. http://dx.doi.org/10.1007/978-3-658-34851-9_6.

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ZusammenfassungDas nachfolgende Kapitel stellt praktische Implikationen für die moderne Interessenvermittlung vor, die sich als Verknüpfung aus den Ergebnissen der empirischen Analyse und den theoretischen Überlegungen ableiten. Die verschiedenen Aspekte sind zu Bausteinen aggregiert, die jeweils die gesamte Bandbreite der aufgezeigten Handlungslogiken innerhalb eines Aspekts darstellen. Die Bausteine ergeben nur in der Zusammenschau Sinn.
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Wittpahl, Volker. "Wie Klima funktioniert und warum sich die Atmosphäre erwärmt". W Klima, 32–52. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-62195-0_2.

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ZusammenfassungKlima ist im engeren Sinn definiert als das durchschnittliche Wetter bzw. als die statistische Beschreibung relevanter Größen mittels der Ermittlung von Durchschnitt und Variabilität über Zeitspannen im Bereich von Monaten, Jahren oder gar von Millionen von Jahren. Die Weltorganisation für Meteorologie (WMO)10 definiert den Zeitraum zur Mittelung einer Variable mit 30 Jahren; die wesentlichen Variablen sind Temperatur, Niederschlag und Wind. In einem weiter gefassten Sinn ist Klima der Zustand des Klimasystems einschließlich einer statistischen Beschreibung wie zum Beispiel mittlere Jahrestemperatur und -niederschlag aber auch die Eintrittswahrscheinlichkeit bzw. Häufigkeit von Ereignissen wie die mittlere Dauer von Dürren, Sturmhäufigkeit und Starkniederschlagen.
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"Financial Data Prediction by Artificial Sine and Cosine Trigonometric Higher Order Neural Networks". W Emerging Capabilities and Applications of Artificial Higher Order Neural Networks, 262–301. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-3563-9.ch006.

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This chapter develops two new nonlinear artificial higher order neural network models. They are sine and sine higher order neural networks (SIN-HONN) and cosine and cosine higher order neural networks (COS-HONN). Financial data prediction using SIN-HONN and COS-HONN models are tested. Results show that SIN-HONN and COS-HONN models are good models for some sine feature only or cosine feature only financial data simulation and prediction compared with polynomial higher order neural network (PHONN) and trigonometric higher order neural network (THONN) models.
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Zhang, Ming. "Artificial Sine and Cosine Trigonometric Higher Order Neural Networks for Financial Data Prediction". W Advances in Computational Intelligence and Robotics, 208–36. IGI Global, 2016. http://dx.doi.org/10.4018/978-1-5225-0063-6.ch009.

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This chapter develops two new nonlinear artificial higher order neural network models. They are Sine and Sine Higher Order Neural Networks (SIN-HONN) and Cosine and Cosine Higher Order Neural Networks (COS-HONN). Financial data prediction using SIN-HONN and COS-HONN models are tested. Results show that SIN-HONN and COS-HONN models are good models for financial data prediction compare with Polynomial Higher Order Neural Network (PHONN) and Trigonometric Higher Order Neural Network (THONN) models.
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Zhang, Ming. "Ultra High Frequency SINC and Trigonometric Higher Order Neural Networks for Data Classification". W Advances in Computational Intelligence and Robotics, 113–53. IGI Global, 2016. http://dx.doi.org/10.4018/978-1-5225-0063-6.ch005.

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This chapter develops a new nonlinear model, Ultra high frequency SINC and Trigonometric Higher Order Neural Networks (UNT-HONN), for Data Classification. UNT-HONN includes Ultra high frequency siNc and Sine Higher Order Neural Networks (UNS-HONN) and Ultra high frequency siNc and Cosine Higher Order Neural Networks (UNC-HONN). Data classification using UNS-HONN and UNC-HONN models are tested. Results show that UNS-HONN and UNC-HONN models are better than other Polynomial Higher Order Neural Network (PHONN) and Trigonometric Higher Order Neural Network (THONN) models, since UNS-HONN and UNC-HONN models can classify the data with error approaching 0.0000%.
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Siker, Jeffrey. "Sin in the Gospel of John and the Johannine Epistles". W Sin in the New Testament, 87–107. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190465735.003.0006.

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In the Gospel of John, Jesus is the lamb of God whose sacrificial death will provide the ultimate forgiveness of sin. John equates sin with the failure of people to believe that Jesus is God’s son and messiah sent to save the world. By identifying Jesus as the lamb of God who takes away the sins of the world, John’s Gospel conflates the meaning of Passover (celebrating freedom from slavery with the sacrifice of the paschal lamb) with the meaning of Yom Kippur (celebrating freedom from sin with the ritual of the scape goat who bares away sin). The story of the healing of the man born blind (John 9) also makes it clear that the religious leaders who oppose Jesus are spiritually blind and remain in their sins. By contrast, the man born blind not only recovers his physical sight, but has gained salvific spiritual sight through his growing recognition of Jesus as God’s son.
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Abraham, William J. "Human Action in the Fall". W Divine Agency and Divine Action, Volume III, 171–85. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198786528.003.0013.

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The Christian tradition takes moral evil with radical seriousness. The tradition is divided on how to read Genesis 3 as seen in the work of Augustine and Symeon the New Theologian. We need several angles of vision to do justice to the fall and to human sin: a theological reading of Genesis 1–11, the many concepts used to describe sin, canonical lists of sins, hierarchies of sin, and the morphology of temptation. These enable us to develop a rich description of sin which does not undermine the goodness of creation. The deepest level of sin is represented by demon possession.
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"I MISSED THE CRUSH OF FISHES". W Sing Sing Sing, 45–46. New York University Press, 2020. http://dx.doi.org/10.18574/nyu/9780814763285.003.0018.

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Streszczenia konferencji na temat "Sin1"

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Niu, Meng-Nian, i Eun Sok Kim. "Residual Stress Effect on Performance of Diaphragm-Based MEMS Pressure Transducers". W ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-1102.

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Abstract We experimentally and theoretically confirm that residual stress within a diaphragm is critical in limiting the performance of diaphragm-based piezoelectric microphones even if the stress is low (around 50 MPa). We have fabricated and studied microphones with Al/parylene/ZnO/SiN2/poly-Si/SiN1 (from top to bottom) diaphragm. As the SiN1 supporting layer is removed layer by layer from the backside with CF4 plasma (in an RIE system), we measure both the sensitivity and center displacement of the microphone before and after each RIE etching of the SiN1 from the microphone diaphragm, and find the sensitivity increasing about 5–16 times with the best sensitivity reaching 11 μV/μbar from a mere 0.6 μV/μbar. The center displacement increases very moderately as the SiN1 layer thickness decreases from 0.8 to 0.2 μm. However, the center displacement starts to increase greatly as the SiN1 layer thickness goes below 0.2 μm, which compares with our theoretical analysis well. In the case of the SiN1 layer having compressive residual stress, the compressive stress can enhance the microphone sensitivity and center displacement to a certain extent.
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Niu, Meng-Nian, Fred J. von Preissig i Eun Sok Kim. "Partially Etched Holes for Residual Stress Release in Diaphragm-Based Pressure Sensors". W ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-1096.

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Abstract We have designed and fabricated various diaphragm-based piezoelectric microphones with film-layer structure SiN1/poly-Si/SiN2/ZnO/parylene/Al/parylene (from bottom to top) that contain various square “holes” uniformly distributed over the whole diaphragm with various hole ratios (defined to be the ratio between the total area of the holes and the whole diaphragm area). The holes (which are formed by etching out all the layers except SiN2 and top parylene in the diaphragm) are to release the residual stress in the diaphragm. We have fabricated and analyzed various microphones with and without the holes, and have observed that the microphones with the holes have significantly larger acoustic sensitivities and diaphragm displacements than those without any hole. The best microphone sensitivity of the hole-array microphones has been over 12 times greater than that of the no-hole microphones. Also, we have observed that the center displacement of the diaphragm is dependent on the hole ratio but not on the hole size. We have corroborated the experimental results with FEM analysis.
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Wang, Qing, Jiang Zhu i Hsin-Sheng Yang. "Abstract LB-032: Tumor suppressor Pdcd4 attenuates Sin1 translation to inhibit invasion in colon carcinoma". W Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-lb-032.

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Kwak, Chong Hoon, Ho Hyung Suh i El-Hang Lee. "Conical diffraction in two-dimensional thin phase gratings". W The European Conference on Lasers and Electro-Optics. Washington, D.C.: Optica Publishing Group, 1994. http://dx.doi.org/10.1364/cleo_europe.1994.cthi66.

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The diffraction of electromagnetic waves from a periodic structure has been intensively investigated both in classical and conical (off-plane) diffraction mountings.1−3 In this paper, we present the multiple diffraction rings of a two-dimensional planar phase grating (so-called Dammann grating4) for the conical diffraction mounting and propose a simple method for measuring the unknown period of the grating as a possible application. Consider a unit amplitude plane wave incident on a two-dimensional (cross) surface-relief grating at a spherical angle (θ1,ϕ1), as illustrated in Fig. 1: where the components of the incident wave vector k1 are (αo, — ßo, —γoo) = kon1 (sinθ,cosϕ1, —sinθ1,sinϕ1, —cosθ1, ko is the wave vector in vacuum, and ni is the refractive index of regions 1 and 3.
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Dai, Hui-Ju, i Lian-Zhong Li. "The (G'/G)-Expansion Method for the Sine-Gordon Equation, Sinh-Gordon Equation and Liouville Equaiton". W 3rd Annual International Conference on Advanced Material Engineering (AME 2017). Paris, France: Atlantis Press, 2017. http://dx.doi.org/10.2991/ame-17.2017.32.

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Wilson, C., T. Mertens, S. Collum, W. Bi, A. Guha, R. Thandavarayan, K. Rajagopal, S. S. Jyothula i H. Karmouty-Quintana. "Deletion of Alveolar Epithelial Type II Sine Oculis Homeobox Homolog 1 (Six1) Attenuates Established Pulmonary Fibrosis". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5400.

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Wang, Yang. "Analysis on the Evolution of Online Public Opinion on Sina Weibo Platform “Sino-US Trade Dispute”". W 2020 4th International Seminar on Education, Management and Social Sciences (ISEMSS 2020). Paris, France: Atlantis Press, 2020. http://dx.doi.org/10.2991/assehr.k.200826.217.

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Alkhazaleh, Anas, Mohamed Younes El-Saghir Selim, Fadi Alnaimat i Bobby Mathew. "Thermo-Hydraulic Performance of Heat Sinks With Microchannel Embedded With Pin-fins". W ASME 2021 Heat Transfer Summer Conference collocated with the ASME 2021 15th International Conference on Energy Sustainability. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/ht2021-62804.

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Abstract In this work, an investigation of the heat sink performance employing sinusoidal microchannels embedded with pin fins was conducted. The effect of the sine wave frequency, the pin fins’ diameter, and the hydraulic diameter of the microchannel are studied. The results are quantified in terms of thermal resistance and pressure drop. The study was done using Reynolds numbers varying from 250 to 2000. As Reynolds number increases, the heat sink’s thermal resistance decreased while the pressure drop increased accordingly for all scenarios. The sinusoidal microchannels showed better performance — lower thermal resistance — but with the cost of higher pressure drop compared to the straight microchannel heat sink. The heat sink’s performance was improved by increasing the frequency, diameter of pin fins, and hydraulic diameter; however, this reduction in thermal resistance was associated with an increase in pressure drop. The reduction in thermal resistance of the different configurations of the sinusoidal microchannels was between 17% and 69% compared to the straight microchannel heat sink.
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Hosseini, Mohammad, Yu Jiang, Poliang Wu, Richard B. Berlin i Lui Sha. "SINk". W Middleware '15: 16th International Middleware Conference. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2834965.2834967.

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Ahn, June, Michael Gubbels, Jinyoung Kim i Johnny Wu. "SINQ". W the 2012 ACM annual conference extended abstracts. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2212776.2223756.

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Raporty organizacyjne na temat "Sin1"

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von Speyr, Adrienne. Vom Sinn der Krankheit. Saint John Publications, 2022. http://dx.doi.org/10.56154/sd.

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Kroh, Julia, i Carsten Schultz. Urbane Innovationen sind machbar! Universitatsbibliothek Kiel, listopad 2022. http://dx.doi.org/10.38071/2022-00441-9.

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Um den zukünftigen Anforderungen ökologischer, ökonomischer und sozialer Nachhaltigkeit gerecht zur werden, müssen sich die technischen und sozialen Strukturen in Städten transformieren. Dazu ist die Entwicklung und Umsetzung von technologischen und sozialen Innovationen, sogenannten urbanen Innovationen, in einem Ökosystem mit vielen und diversen Akteuren notwendig. Etablierte Ansätze des Innovations- und des Projektmanagements sind aufgrund der komplexen Akteursstruktur nur partiell übertragbar. Ziel dieser Forschung ist es am Beispiel einer umfassenden empirischen Analyse von 107 Innovationsprojekten, die die Steigerung von Energieeffizienz und Reduktion von Kohlenstoffdioxidemissionen in bestehenden Quartieren in kleinen, mittleren und großen Städten in Deutschland zum Ziel hatten, zu zeigen, ob und wie Managementansätze in urbanen Innovationsprozessen angepasst werden müssen. Unsere Forschung zeigt, dass sich die untersuchten Innovationsprojekte größtenteils mit der energetischen Sanierung des Gebäudebestands und weniger mit der energieeffizienten Nutzung von Energie (d.h. der Entwicklung von Smart City Lösungen) beschäftigen. Weiterhin zeigt sich, dass der Erfolg von urbanen Innovationsprojekten nicht eindimensional messbar ist. Neben der Einhaltung von klassischen Projektzielen (Einhaltung von Budget-, Zeit und Inhaltszielen), sind Neuartigkeit und Umsetzungserfolg erstrebenswert. Unsere Forschung zeigt weiterhin, dass die Zusammenarbeit mit skeptischen Akteuren in allen Prozessphasen wesentlich für den Erfolg ist. Gerade die Einbindung von skeptischen Akteuren führt zu neuartigeren Lösungsalternativen und in gewissem Maße zu höherem Umsetzungserfolg.
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Ressler, Aaron R. Advancing Sino-U.S. Space Cooperation. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2009. http://dx.doi.org/10.21236/ada539619.

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Bayarsaikhan, B. D. Prospects of Sino-Russian Relations. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2003. http://dx.doi.org/10.21236/ada414583.

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Mehmedagic, I., i J. Krug. Heat Sink Design and Optimization. Fort Belvoir, VA: Defense Technical Information Center, grudzień 2015. http://dx.doi.org/10.21236/ad1000573.

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Viloria-de-la-Hoz, Joaquín. CERETE : municipio agrícola del Sinú. Bogotá, Colombia: Banco de la República, luty 2002. http://dx.doi.org/10.32468/dtseru.26.

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Hines, James. Taxing Consumption and Other Sins. Cambridge, MA: National Bureau of Economic Research, grudzień 2006. http://dx.doi.org/10.3386/w12730.

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Lewis, Donald C. The Sino-Soviet Military Rapprochement. Fort Belvoir, VA: Defense Technical Information Center, luty 1991. http://dx.doi.org/10.21236/ada233476.

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Lockwood, Benjamin, i Dmitry Taubinsky. Regressive Sin Taxes. Cambridge, MA: National Bureau of Economic Research, styczeń 2017. http://dx.doi.org/10.3386/w23085.

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Paik, Keun-Wook. Sino-Russian Gas and Oil Cooperation. Oxford Institute for Energy Studies, kwiecień 2015. http://dx.doi.org/10.26889/9781784670290.

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