Gotowe bibliografie tematyczne / Responses

Gotowa bibliografia na temat „Responses”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 19 lutego 2023

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Artykuły w czasopismach na temat "Responses"

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Kindellan, Michael, i Joshua Kotin. "Response to Responses". Modernist Cultures 12, nr 3 (listopad 2017): 388–90. http://dx.doi.org/10.3366/mod.2017.0183.

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SANDERS, MICHAEL, VEERLE SNIJDERS i MICHAEL HALLSWORTH. "Response to responses". Behavioural Public Policy 2, nr 2 (14.08.2018): 263–69. http://dx.doi.org/10.1017/bpp.2018.14.

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Ho, Engseng. "Response to Responses". Journal of Asian Studies 76, nr 4 (27.09.2017): 961–62. http://dx.doi.org/10.1017/s0021911817000912.

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Chesterman, Andrew. "Response to the responses". Translation Studies 7, nr 3 (20.06.2014): 349–52. http://dx.doi.org/10.1080/14781700.2014.927330.

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Noble, Marianne. "Response to the Responses". Yale Journal of Criticism 12, nr 1 (1999): 161–67. http://dx.doi.org/10.1353/yale.1999.0010.

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Morris, Eva. "Response to the Responses". Humanity & Society 33, nr 4 (listopad 2009): 389. http://dx.doi.org/10.1177/016059760903300410.

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VandeCreek, Larry. "Response to the Responses". Journal of Health Care Chaplaincy 8, nr 1-2 (wrzesień 1998): 137–38. http://dx.doi.org/10.1300/j080v08n01_14.

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Barlowe, J. "Response to the Responses". American Literary History 9, nr 2 (1.02.1997): 238–43. http://dx.doi.org/10.1093/alh/9.2.238.

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Dirlik, Arif. "Response to the responses". Interventions 1, nr 2 (styczeń 1999): 286–90. http://dx.doi.org/10.1080/13698019900510401.

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Metz, David. "Response to the Responses". Transport Reviews 28, nr 6 (listopad 2008): 713–15. http://dx.doi.org/10.1080/01441640802536027.

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Rozprawy doktorskie na temat "Responses"

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Klein, Entink Rinke. "Statistical models for responses and response times". Enschede : University of Twente [Host], 2009. http://doc.utwente.nl/60452.

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Järvinen, Jaakko Lauri Paivio. "Inverted responses and response recovery in amphibian rod photoreceptors". Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.615722.

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Buske-Kirschbaum, Angelika. "Cortisol Responses to Stress in Allergic Children: Interaction with the Immune Response". Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135731.

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Allergic manifestations are increasingly common in infants and children. Accumulating evidence suggests that the ‘epidemic’ increase of childhood allergy may be associated with environmental factors such as stress. Although the impact of stress on the manifestation and exacerbation of allergy has been demonstrated, the underlying mechanisms of stress-induced exacerbation are still obscure. A growing number of studies have suggested an altered hypothalamus-pituitary-adrenal (HPA) axis function to stress in allergic children. It is speculated that a dysfunctional HPA axis in response to stress may facilitate and/or consolidate immunological aberrations and thus, may increase the risk for allergic sensitization and exacerbation especially under stressful conditions. In the present review the potential impact of a hyporesponsive as well as a hyperresponsive HPA axis on the onset and chronification of childhood allergy is summarized. Moreover, potential factors that may contribute to the development of an aberrant HPA axis responsiveness in allergy are discussed
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich
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Rodgers, Angela. "Macrophage responses and their involvement in generating an immune response against tuberculosis". Thesis, St George's, University of London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406802.

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Buske-Kirschbaum, Angelika. "Cortisol Responses to Stress in Allergic Children: Interaction with the Immune Response". Karger, 2009. https://tud.qucosa.de/id/qucosa%3A27671.

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Allergic manifestations are increasingly common in infants and children. Accumulating evidence suggests that the ‘epidemic’ increase of childhood allergy may be associated with environmental factors such as stress. Although the impact of stress on the manifestation and exacerbation of allergy has been demonstrated, the underlying mechanisms of stress-induced exacerbation are still obscure. A growing number of studies have suggested an altered hypothalamus-pituitary-adrenal (HPA) axis function to stress in allergic children. It is speculated that a dysfunctional HPA axis in response to stress may facilitate and/or consolidate immunological aberrations and thus, may increase the risk for allergic sensitization and exacerbation especially under stressful conditions. In the present review the potential impact of a hyporesponsive as well as a hyperresponsive HPA axis on the onset and chronification of childhood allergy is summarized. Moreover, potential factors that may contribute to the development of an aberrant HPA axis responsiveness in allergy are discussed.
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich.
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Sunter, Nicola. "DNA Damage Responses". Thesis, University of Newcastle Upon Tyne, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.489314.

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The histone H2AX has been established as a reliable biomarker of DNA damage, becoming phosphorylated rapidly following damage in discrete foci which can be correlated with DNA DSB number. In the present study, the phosphorylation of H2AX was used as a marker of DNA DSB damage to compare and contrast the damage induced by ionizing radiation, the topo II poison, etoposide, and the topo II catalytic inhibitor, ICRF-193. To examine the DNA damage numbers at time-points in the 24 hours following exposure to these damaging agents. Topo lIP null cells were used to investigate the contribution of topo II a and Pthese damage responses and the Trapped in Agarose DNA Immunostaining assay was utilised to quantify the numbers of topo II_DNA complexes formed in response to these agents. This study aimed to examine the levels of DNA damage following exposure to these damaging agents and to investigate differences in the complement of proteins associated with DNA damage-induced foci. By using both the y-H2AX and TARDIS assays and protein colocalisation techniques, the studies detailed here presents novel findings on the differing damage responses induced following these three agents.
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Roberts, Emma Margaret. "Contralateral inflammatory responses". Thesis, University of Bath, 2005. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432371.

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Albrechtsen, Justin Scott. "Are intuitive responses more accurate at detecting deception than deliberate responses?" To access this resource online via ProQuest Dissertations and Theses @ UTEP, 2007. http://0-proquest.umi.com.lib.utep.edu/login?COPT=REJTPTU0YmImSU5UPTAmVkVSPTI=&clientId=2515.

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Fitzsimmons, James. "Ecological Responses to Threats in an Evolutionary Context: Bacterial Responses to Antibiotics and Butterfly Species’ Responses to Climate Change". Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/23807.

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Humans are generally having a strong, widespread, and negative impact on nature. Given the many ways we are impacting nature and the many ways nature is responding, it is useful to study responses in an integrative context. My thesis is focused largely (two out of the three data chapters) on butterfly species’ range shifts consistent with modern climate change in Canada. I employed a macroecological approach to my research, drawing on methods and findings from evolutionary biology, phylogenetics, conservation biology, and natural history. I answered three main research questions. First, is there a trade-off between population growth rate (rmax) and carrying capacity (K) at the mutation scale (Chapter 2)? I found rmax and K to not trade off, but in fact to positively co-vary at the mutation scale. This suggests trade-offs between these traits only emerge after selection removes mutants with low resource acquisition rates (i.e., unhealthy genotypes), revealing trade-offs between remaining genotypes with varied resource allocation strategies. Second, did butterfly species shift their northern range boundaries northward over the 1900s, consistent with climate warming (Chapter 3)? Leading a team of collaborators, we found that most butterfly species’ northern range boundaries did indeed shift northward over the 1900s. But range shift rates were slower than those documented in the literature for more recent time periods, likely reflecting the weaker warming experienced in the time period of my study. Third, were species’ rates of range shift related to their phylogeny (Chapter 3) or traits (Chapter 4)? I found no compelling relationships between rates of range shift and phylogeny or traits. If certain traits make some species more successful at northern boundary range expansion than others, their effect was not strong enough to emerge from the background noise inherent in the broad scale data set I used.
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Ghaffari, Emma Louise Marie. "Early growth response genes -2 and -3 are essential for optimal immune responses". Thesis, Brunel University, 2013. http://bura.brunel.ac.uk/handle/2438/8134.

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Early Growth Response Genes (EGR) is a family of four transcription factors containing a unique zinc finger domain. EGR-2 and EGR-3 are important for hindbrain development and myelination. These transcription factors are also necessary for lymphocyte function however, the mechanisms are still unclear. Previous findings have shown that EGR-2cKO mice develop lupus-like autoimmune disease with high levels of pro-inflammatory cytokines despite showing normal T and B cell proliferation after mitogenic stimulation. Therefore we established the CD2-EGR-2-/-EGR-3-/- mouse model to explore the phenotype, susceptibility to autoimmune disease and relevant lymphocyte function. We discovered that CD2-EGR-2-/-EGR-3-/- mice developed severe systemic autoimmune disease and expressed high levels of inflammatory cytokines. More importantly we discovered a novel finding that CD2-EGR-2-/-EGR-3-/- T and B cells had impaired cell proliferation after mitogenic stimulation. Further investigations revealed that the molecular mechanism defected in the T cell receptor signalling pathway is due to a dysfunction in Activator Protein-1 (AP-1). AP-1 is a heterodimeric protein composed of AP-1 family members including Jun, Atf and Fos. Our data shows that EGR-2 and EGR-3 directly bind with the Atf family member Batf, which prevents Batf’s inhibitory function on AP-1 activation. This research demonstrates that EGR-2 and EGR-3 intrinsically regulate chronic inflammation and also positively regulate antigen receptor activation. In conclusion EGR-2 and EGR-3 are essential for providing optimal immune responses, whilst limiting inflammatory immunopathology. We propose that this new model could be used for studying autoimmune disease.
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Książki na temat "Responses"

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Homeric responses. Austin: University of Texas Press, 2003.

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Oslowski, Christine M., red. Stress Responses. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2522-3.

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Great Britain. Parliament. House of Lords. Select Committee on the European Union. Government responses. London: Stationery Office, 2001.

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Andrew, Blowers, i Hinchliffe Steve 1955-, red. Environmental responses. Chichester: Wiley in Association with the Open University, 2003.

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Arts Council of Northern Ireland, red. Institutional responses. Belfast: Arts Council of Northern Ireland, 2011.

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Responses to crime. Oxford: Clarendon, 1993.

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O, Jenkins William, United States. Dept. of Homeland Security i United States. Government Accountability Office, red. National emergency responses. New York: Nova Science, 2009.

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Responses to crime. Oxford: Clarendon Press, 1987.

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John, Adler, red. Responses to Shakespeare. London: Routledge/Thoemmes Press, 1997.

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Hazards and responses. London: CollinsEducational, 1998.

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Części książek na temat "Responses"

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McDowell, John. "Responses". W Studies in German Idealism, 231–58. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98896-2_13.

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Tolba, Mostafa K. "Responses". W Saving Our Planet, 231–41. Dordrecht: Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2278-8_21.

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Brown, John Russell. "Responses". W Shakespeare and the Theatrical Event, 41–62. London: Macmillan Education UK, 2002. http://dx.doi.org/10.1007/978-0-230-62961-5_4.

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Spaaij, Ramón. "Responses". W Understanding Lone Wolf Terrorism, 77–96. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-2981-0_8.

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Pitt, Christopher. "Responses". W The Definitive Guide to AdonisJs, 61–70. Berkeley, CA: Apress, 2018. http://dx.doi.org/10.1007/978-1-4842-3390-0_5.

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Wyatt, Ray. "Responses". W Plan Prediction, 213–42. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46430-5_8.

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Howe, David. "Responses". W Attachment Theory for Social Work Practice, 208–23. London: Macmillan Education UK, 1995. http://dx.doi.org/10.1007/978-1-349-24081-4_16.

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Hall, Matthew, i Jeff Hearn. "Responses". W Revenge Pornography, 27–42. Abingdon, Oxon; New York, NY: Routledge, 2018.: Routledge, 2017. http://dx.doi.org/10.4324/9781315648187-3.

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Barnett, Tony, i Alan Whiteside. "Responses". W AIDS in the Twenty-First Century, 316–46. London: Palgrave Macmillan UK, 2002. http://dx.doi.org/10.1057/9780230599208_13.

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Mislevy, Robert J. "Missing Responses in Item Response Modeling". W Handbook of Item Response Theory, 171–94. Boca Raton, FL: CRC Press, 2015- | Series: Chapman & Hall/CRC Statistics in the Social and Behavioral Sciences.: Chapman and Hall/CRC, 2017. http://dx.doi.org/10.1201/b19166-10.

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Streszczenia konferencji na temat "Responses"

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"Front Matter: Volume 10495". W Biophotonics and Immune Responses XIII, redaktor Wei R. Chen. SPIE, 2018. http://dx.doi.org/10.1117/12.2323042.

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Zhou, Benqing, Meng Wang, Feifan Zhou, Jun Song, Junle Qu i Wei R. Chen. "BSA-modified gold nanorods for combined photothermal therapy and immunotherapy of melanoma". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506097.

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Qi, Shuhong, Lisen Lu i Zhihong Zhang. "ALDH3A2 as a potential molecular marker for nasopharyngeal carcinoma". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506121.

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Gallagher, Kyra A., Austin Doughty, Elivia Layton, Sara Zukerman, Benqing Zhou i Wei R. Chen. "The characterization of graphene-reconstituted low-density lipoproteins and small molecule inhibitor nanoparticles in preparation for use in laser immunotherapy". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506714.

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Chen, Wei R., Jingxuan Yang, Min Li, Feifan Zhou i Meng Wang. "Treatment of pancreatic tumors using combination of laser irradiation and immunological stimulation (Conference Presentation)". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506731.

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Wang, Meng, Lu Wang, Benqing Zhou, Feifan Zhou i Wei Chen. "NIR-triggered photodynamic therapy via antigen-capturing upconversion nanoparticle enhances cancer immunotherapy (Conference Presentation)". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507021.

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Liu, Siyu, Feifan Zhou, Yue Zhao, Liwei Liu, Junle Qu i Wei R. Chen. "Temperature detection by photoacoustic for nanoprobe-mediated photothermal therapy". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507030.

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Chen, Haibin, Dezi Li i Zhifang Li. "Temperature feedback-controlled photothermal treatment based on thermal infrared imager". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507579.

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Zhang, Zhihong, Lisen Lu, Yuan Qian, Sha Qiao i Qingming Luo. "Targeting delivery of nanovaccine/nanoimmunomudulator based on peptide-lipid nanoparticle for immunotherapy in vivo (Conference Presentation)". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507601.

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Wu, Haiwei, Quanyu Zhou, Huan Zhou, Kai Ping, Bobo Gu, Hao He i Xunbin Wei. "Non-invasive monitoring of circulating melanoma cells by in vivo photoacoustic flow cytometry". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507807.

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Raporty organizacyjne na temat "Responses"

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DEFENSE MANPOWER DATA CENTER ARLINGTON VA. 2009 QuickCompass of Sexual Assault Responders: Tabulations of Responses. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2009. http://dx.doi.org/10.21236/ada508930.

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Walrath, James D. Phraselator Questionnaire Responses. Fort Belvoir, VA: Defense Technical Information Center, maj 2009. http://dx.doi.org/10.21236/ada499874.

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Metz, C. OTP Extended Responses. RFC Editor, listopad 1997. http://dx.doi.org/10.17487/rfc2243.

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McManus, P. HTTP Immutable Responses. RFC Editor, wrzesień 2017. http://dx.doi.org/10.17487/rfc8246.

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Cioffi, William G., David G. Burleson, Basil A. Pruitt i Jr. Leukocyte Responses to Injury,. Fort Belvoir, VA: Defense Technical Information Center, listopad 1993. http://dx.doi.org/10.21236/ada277949.

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Barnett, Michael, Greg Buchak i Constantine Yannelis. Epidemic Responses Under Uncertainty. Cambridge, MA: National Bureau of Economic Research, maj 2020. http://dx.doi.org/10.3386/w27289.

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Britt, Anne. G2 Checkpoint Responses in Arabidopsis. Office of Scientific and Technical Information (OSTI), marzec 2013. http://dx.doi.org/10.2172/1116357.

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Hanna, Philip C. Macrophage Responses to B. Anthracis. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2006. http://dx.doi.org/10.21236/ada456287.

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Peterson, Scott N. Macrophage Responses to B. Anthracis. Fort Belvoir, VA: Defense Technical Information Center, listopad 2004. http://dx.doi.org/10.21236/ada428855.

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Research Institute (IFPRI), International Food Policy. Seed demand and supply responses. Washington, DC: International Food Policy Research Institute, 2018. http://dx.doi.org/10.2499/9780896292833_04.

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