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Oliveira, Ana Luísa Araújo. "Adventitious respiratory sounds in children with respiratory infection". Master's thesis, Universidade de Aveiro, 2014. http://hdl.handle.net/10773/13734.
Pełny tekst źródłaBackground: Lower respiratory tract infections (LRTI) are the leading cause of hospital visits in children under 5 years old. Therefore, there is an urgent and unmet need to develop objective, reliable and quick measures for respiratory paediatric assessment. Computerised adventitious respiratory sounds (ARS) have shown to be objective and reliable to assess/monitor respiratory diseases; however its application in children with LRTI is unknown. Aim: To characterise/compare ARS in healthy children and children with LRTI. Methods: A cross-sectional descriptive-comparative study was conducted in three healthcare institutions. Children were diagnosed by the paediatrician as healthy or with a LRTI and grouped according to their age (i.e, 0-2 years old or 3-5 years old). Socio-demographic and anthropometric data, type and severity of LRTI and cardio-respiratory parameters were collected. Respiratory sounds were recorded from the chest with a digital stethoscope following the Computerised Respiratory Sound Analysis guidelines. Wheezes’ location, mean number, type, frequency and occupation rate and crackles’ location, mean number, type, frequency, initial deflection width, two cycle duration, and largest deflection width were analysed per breathing phase. Results: Forty children enrolled in this study: 22 aged 0-2 years old (G1: 11 healthy; G2: 11 with LRTI) and 18 aged 3-5 years old (G3: 9 healthy; G4: 9 with LRTI). Few children, both healthy and with LRTI presented wheezes. In both age ranges, children with LRTI presented a higher percentage of the expiratory phase occupied by wheezes (G1: M 2.15 IQR 1.45 vs. G2: M 4.73 IQR 6.72 p=0.001; G3: M 2.80 IQR 3.27 vs. G4: M 5.17 IQR 15.99 p=0.07). Crackles were found in all children in at least one chest location. In both age ranges, children with LRTI presented more inspiratory crackles (G1: M 0.25 IQR 0.31 vs. G2: M 0.52 IQR 0.70; p<0.001; G3: M 0.50 IQR 0.49 vs. G4: M 0.70 IQR 0.21 p=0.03), especially fine crackles than healthy children (G1: M 0.07 IQR 0.13 vs. G2: M 0.18 IQR 0.42 p=0.001; G3: M 0.11 IQR 0.21 vs. G4: M 0.17 IQR 0.23 p=0.001). Coarse expiratory crackles were the most common type of crackle found in both healthy children (G1: M 0.33 IQR 0.56; G3: M 0.56 IQR 0.99) and children with LRTI (G2: M 0.33 IQR 0.56; G4: M 1.14 IQR 1.38). No differences were found for the remaining parameters. Conclusion: Healthy children and children with LRTI of different ages present ARS (i.e., crackles and wheezes). The occupation rate of wheezes and the mean number of crackles were the parameters that most differed between healthy children and children with LRTI in both age ranges. Therefore these ARS’ parameters may be the best criteria to discriminate the groups.
Enquadramento: As infeções respiratórias do tracto inferior (IRTI) são a principal causa de visitas/admissões hospitalares em crianças com idade inferior a 5 anos. Desta forma, verifica-se uma urgente necessidade de desenvolver medidas de avaliação respiratória pediátricas que sejam objetivas, fiáveis e de rápida aplicação. Os sons respiratórios adventícios (SRA) computorizados têmse revelado objetivos e fiáveis na avaliação/monitorização de doenças respiratórias; contudo a sua aplicação em pediatria é desconhecida. Objetivos: Caracterizar/comparar os SRA em crianças saudáveis e com IRTI. Métodos: Um estudo transversal descritivo-comparativo foi realizado em três instituições de saúde. As crianças foram diagnosticadas pelo pediatra como saudáveis ou com IRTI e agrupadas de acordo com a sua idade (i.e., 0-2 anos ou 3-5 anos). Dados antropométricos, sócio-demográficos, cardio-respiratório e tipo/severidade da IRTI foram recolhidos. Os sons respiratórios foram foram recolhidos no tórax com um estetoscópio digital, de acordo com as orientações internacionais. A localização, número médio, tipo, frequência e taxa de ocupação das sibilâncias e a localização número médio, tipo, frequência, initial deflection width, two cycle duration, e largest deflection width dos fervores foram analizados por fase respiratória. Resultados: Quarenta crianças participaram neste estudo: 22 com idades entre is 0-2 anos (G1: 11 saudáveis; G2: 11 com IRTI) e 18 com idades entre os 3-5 anos (G3: 9 saudáveis; G4: 9 com IRTI). Poucas crianças de ambos os grupos apresentaram sibilâncias. Para ambas as faixas etárias as crianças com IRTI apresentaram uma maior percentagem da expiração ocupada por sibilâncias (G1: M 2.15 IQR 1.45 vs. G2: M 4.73 IQR 6.72 p=0.001; G3: M 2.80 IQR 3.27 vs. G4: M 5.17 IQR 15.99 p=0.07). Todas as crianças apresentaram fervores em pelo menos um local de auscultação. Em ambas as faixas etárias, aqueles com IRTI apresentaram mais fervores inspiratórios (G1: M 0.25 IQR 0.31 vs. G2: M 0.52 IQR 0.70; p<0.001; G3: M 0.50 IQR 0.49 vs. G4: M 0.70 IQR 0.21 p=0.03), especialmente fervores crepitantes , (G1: M 0.07 IQR 0.13 vs. G2: M 0.18 IQR 0.42 p=0.001; G3: M 0.11 IQR 0.21 vs. G4: M 0.17 IQR 0.23 p=0.001). Os fervores expiratórios subcrepitantes foram os mais comuns entre todas as crianças (G1: M 0.33 IQR 0.56; G2: M 0.33 IQR 0.56; G3: M 0.56 IQR 0.99; G4: M 1.14 IQR 1.38).Não foram encontradas diferenças relativamente aos restantes parâmetros avaliados. Conclusão: Crianças saudáveis e com IRTI de diferentes faixas etárias apresentam SRA (i.e., sibilâncias e fervores). A taxa de ocupação das sibilâncias e o número de fervores foram as características que apresentaram mais diferenças entre os participantes saudáveis e os participantes com IRTI. Desta forma, conclui-se que estas características dos SRA poderão constituir os melhores critérios de discriminação entre os grupos.
Del, Valle Mendoza Juana, Tapia Ángela Cornejo, Pablo Weilg, Eduardo Verne, Fuertes Ronald Nazario, Claudia Ugarte, Valle Luis J. del i Toma´ s. Pumarola. "Incidence of Respiratory Viruses in Peruvian Children With Acute Respiratory Infections". John Wiley & Sons, 2015. http://hdl.handle.net/10757/347016.
Pełny tekst źródłaAcute respiratory infections are responsible for high morbi–mortality in Peruvian children. However, the etiological agents are poorly identified. This study, conducted during the pandemic outbreak of H1N1 influenza in 2009, aims to determine the main etiological agents responsible for acute respiratory infections in children from Lima, Peru. Nasopharyngeal swabs collected from 717 children with acute respiratory infections between January 2009 and December 2010 were analyzed by multiplex RT-PCR for 13 respiratory viruses: influenza A, B, and C virus; parainfluenza virus (PIV) 1, 2, 3, and 4; and human respiratory syncytial virus (RSV) A and B, among others. Samples were also tested with direct fluorescent-antibodies (DFA) for six respiratory viruses. RT-PCR and DFA detected respiratory viruses in 240 (33.5%) and 85 (11.9%) cases, respectively. The most common etiological agents were RSV-A (15.3%), followed by influenza A (4.6%), PIV-1 (3.6%), and PIV-2 (1.8%). The viruses identified by DFA corresponded to RSV (5.9%) and influenza A (1.8%). Therefore, respiratory syncytial viruses (RSV) were found to be the most common etiology of acute respiratory infections. The authors suggest that active surveillance be conducted to identify the causative agents and improve clinical management, especially in the context of possible circulation of pandemic viruses
Pruikkonen, H. (Hannele). "Viral infection induced respiratory distress in childhood". Doctoral thesis, Oulun yliopisto, 2015. http://urn.fi/urn:isbn:9789526207919.
Pełny tekst źródłaTiivistelmä Hengitysvaikeus on yleinen oire lapsilla virusten aiheuttamien hengitystieinfektioiden yhteydessä. Kurkunpäätulehdukseen liittyy sisäänhengitysvaikeus. Ilmatiehyttulehdukseen, ahtauttavaan keuhkoputkentulehdukseen ja akuuttiin astmakohtaukseen liittyy uloshengitysvaikeus. Hengitystieinfektioihin liittyvä hengitysvaikeus on yksi yleisimmistä syistä päivystyspoliklinikkakäynteihin ja äkillisiin sairaalahoitojaksoihin lapsipotilailla. Hengitystieinfektioiden taudinkulun tuntemisella ja hengitysvaikeuden vaikeusasteen arvioinnilla on tärkeä merkitys näiden potilaiden hoidon toteuttamisessa. Hengitystieinfektioon liittyvää hengitysvaikeutta on pidetty riskitekijänä astman kehittymiselle. Tämän tutkimuksen tarkoituksena oli selvittää kurkunpäätulehduksen riskitekijöitä ja sairaalahoitoon vaikuttavia tekijöitä hengitystieinfektioon liittyvän uloshengitysvaikeuden hoidossa sekä varhaislapsuudessa sairastetun hengitystieinfektion yhteyttä myöhempään astma- ja allergiasairastavuuteen. Tutkimukseen sisältyi kaksi rekisteriaineistoa ja yksi seurantatutkimusaineisto. Tutkimuksessa todettiin, että kurkunpäätulehduksen uusiutuminen on erittäin tavallista ja sisarusten ja vanhempien sairastama kurkunpäätulehdus on merkittävin riskitekijä kurkunpäätulehdukselle ja sen uusiutumiselle. Alle 6 kuukauden ikäisillä lapsilla ilmatiehyttulehduksen taudinkuva on epävakaa ensimmäisen 5 oirepäivän aikana. Kuume, matala happisaturaatioarvo ja respiratory syncytial -virusinfektio ennustavat osastohoidon ja invasiivisten toimenpiteiden tarvetta ilmatiehyttulehduksen yhteydessä. Yli 6 kuukauden ikäisillä lapsilla happisaturaatioarvo > 93 % ennustaa lievää taudinkuvaa hengitystieinfektioon liittyvän uloshengitysvaikeuden hoidossa. Käyttämällä tätä happisaturaatioarvoa raja-arvona, kun arvioidaan sairaalahoidon tarvetta, voidaan merkittävästi ja turvallisesti vähentää sairaalahoidon tarvetta lasten hengitystieinfektioon liittyvän uloshengitysvaikeuden hoidossa. Alle 6 kuukauden iässä sairastettu respiratory syncytial -virusinfektio on riskitekijä varhaislapsuudessa ilmeneville astmaoireille, mutta tämä riski vähenee iän myötä ja 8 vuoden iässä ei ole havaittavissa eroja astma- ja allergiasairastavuudessa, kun verrataan näitä potilaita muun hengitystieinfektion sairastaneisiin potilaisiin ja terveisiin kontrollipotilaisiin
Hussain, Imran Raza. "The immunobiology of respiratory syncytial virus infection". Thesis, University of Southampton, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289569.
Pełny tekst źródłaWrightson, John M. "Pathogen identification in lower respiratory tract infection". Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:30c757ec-99b7-492e-a12e-ff996581863a.
Pełny tekst źródłaClark, Tristan William. "The role of respiratory virus infection in adults hospitalised with acute respiratory illness". Thesis, University of Leicester, 2013. http://hdl.handle.net/2381/28098.
Pełny tekst źródłaTong, Jie [Verfasser]. "Co-infection of respiratory epithelial cells by respiratory viruses and streptococci / Jie Tong". Hannover : Bibliothek der Tierärztlichen Hochschule Hannover, 2018. http://d-nb.info/1162715758/34.
Pełny tekst źródłaShi, Ting. "Epidemiology of respiratory syncytial virus associated acute lower respiratory infection in young children". Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/23610.
Pełny tekst źródłaBarasheed, Osamah Abdullah A. "Prevention of respiratory viral infection among Hajj pilgrims". Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/23506.
Pełny tekst źródłaKristo, A. (Aila). "Acute rhinosinusitis during upper respiratory infection in children". Doctoral thesis, University of Oulu, 2005. http://urn.fi/urn:isbn:9514278720.
Pełny tekst źródłaManley, Grace C. A. "The roles of DUSPs in respiratory viral infection". Thesis, University of Sheffield, 2018. http://etheses.whiterose.ac.uk/19257/.
Pełny tekst źródłaMize, Maximillion. "Interleukin-17A Worsens Severe Murine Respiratory Mycoplasma Infection". Thesis, University of North Texas Health Science Center at Fort Worth, 2018. http://pqdtopen.proquest.com/#viewpdf?dispub=10843638.
Pełny tekst źródłaThe purpose of these studies was to determine the role of Interleukin-17A (IL-17A) in the immune response to respiratory mycoplasma infection. Serum levels of IL-17A increase in disease-susceptible BALB/c mice, but not disease-resistant C57BL/6 mice, infected with Mycoplasma pulmonis. Increased serum IL-17A was associated with mycoplasma pathology during infection in BALB/c mice, including: the presence of pulmonary neutrophils, progressive weight loss, and the development of inflammatory lung lesions.
Neutralizing the function of IL-17A using monoclonal anti-IL-17A antibodies during mycoplasma infection reduced disease severity in disease-susceptible BALB/c mice, but not disease-resistant C57BL/6 mice. Providing daily intra-peritoneal injections of anti-IL-17A antibodies to BALB/c mice infected with M. pulmonis was effective at reducing weight loss, the prevalence of clinical signs, and the incidence of gross lesions. Histological lesions, characterized by the presence of pulmonary neutrophils, were also lower in infected BALB/c mice receiving anti-IL-17A antibodies daily. Bacterial burden remained unaffected in mice regardless of treatment. Neutralizing IL-17A throughout infection was effective at reducing late mycoplasma pathology, a period influenced by the actions of adaptive immunity and this is supported by a reduction in disease severity when infected BALB/c mice were provided intra-peritoneal injections of anti-IL-17A antibodies only after T-cells infiltrate the lungs.
Pulmonary T-cells, specifically CD4+ T-helper (Th17) cells, were the primary source of IL-17A throughout infection with M. pulmonis in disease-susceptible BALB/c mice. Although Th17 cells increased in the lung after infection, the Th17 response did not reach its peak until the later stages of infection and coincided with when the neutralization of IL-17A started to reduce the severity of disease. IL-17A+ T-cells did not express Retinoic Acid Related (RAR) Orphan Receptor-γt (RORγt), a signature Th17 transcription factor, after infecting BALB/c mice with M. pulmonis and suggests that RORγt is not a suitable marker to identify the IL-17A+ T-cells worsening mycoplasma disease.
The effect of neutralizing IL-17A was mimicked in disease-susceptible BALB/c mice depleted of neutrophils during M. pulmonis infection. Depleting neutrophils in BALB/c mice infected with M. pulmonis abrogated weight loss while reducing the appearance of both clinical signs and gross lesions. IL-17A promotes pathology during disease utilizing various mechanisms, one of which is to mobilize and activate neutrophils; however, the IL-17A failed to worsen mycoplasma disease in the absence of neutrophils during M. pulmonis infection in BALB/c mice. These results suggest that IL-17A relies only upon neutrophil recruitment and activation to exacerbate mycoplasma disease. Supporting this, combining the neutralization of IL-17A with the depletion of neutrophils failed to lessen disease severity beyond what either treatment could achieve alone. These findings underscore IL-17A or neutrophils as targets for inhibition to reduce the severity of disease during mycoplasma infection.
Both IL-4 and IL-17A increase in the lungs of BALB/c mice infected with M. pulmonis and there are Th17 cells that secrete IL-4. In STAT6 KO mice that respond poorly to IL-4 and generate defective Th2-mediate immunity, neutralizing IL-17A also reduced inflammatory damage during M. pulmonis infection. Treating STAT6 KO mice with anti-IL-17A antibodies during M. pulmonis infection reduced weight loss, the prevalence of clinical signs, and incidence of inflammatory lesions. Like wild-type mice, the pathologic effect of IL-17A manifested during the later stages of M. pulmonis infection in STAT6 KO mice and coincided with the activation of adaptive immunity. Neutralizing IL-17A also failed to change mycoplasma numbers during infection in STAT6 KO mice. IL-17A is highlighted as an independent contributor to mycoplasma pathology with no impact on mycoplasma clearance; inhibiting the activation of Th2- and Th17-mediated immune responses could increase resistance by permitting the development of protective responses during infection.
This work emphasizes the importance of IL-17A and Th17 cells as an autonomous immune response worsening neutrophil-mediated pathology during late mycoplasma infection in susceptible mice. Monoclonal antibodies that neutralize the function of IL-17A could reduce the severity of disease during mycoplasma infection in man and animals. Directly targeting neutrophils may also lessen the negative impact IL-17A has on mycoplasma pathology. Vaccines that do not activate IL-17A-mediated immunity could reduce the susceptibility to mycoplasma infection and allow for the development of immune responses that lead to mycoplasma clearance. IL-17A functions to worsen disease severity without impacting mycoplasma clearance, and so IL-17A is identified as a contributor to pathology during infection.
Cline, Troy. "Innate Immune Mechanisms of Controlling Respiratory Virus Infection". The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1262099397.
Pełny tekst źródłaDeacon, Jill. "Antimicrobial nanotherapies for respiratory infection in cystic fibrosis". Thesis, Queen's University Belfast, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.695326.
Pełny tekst źródłaBrown, Helen. "Host responses to respiratory infection in cystic fibrosis". Thesis, University of Bath, 2001. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392017.
Pełny tekst źródłaXie, Xiaojian. "Evaporation and movement of respiratory droplets in indoor environments". Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40987802.
Pełny tekst źródłaAgoti, Charles Nyaigoti. "Genetic diversity of respiratory syncytial virus (RSV) in relation to infection and re-infection". Thesis, Open University, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.664468.
Pełny tekst źródłaGraham, Neil M. H. "Psychosocial factors in the epidemiology of acute respiratory infection /". Title page, contents, thesis synopsis and summary only, 1987. http://web4.library.adelaide.edu.au/theses/09MD/09mdg741.pdf.
Pełny tekst źródłaSnelgrove, Robert. "Manipulation of the myeloid immune compartment during respiratory infection". Thesis, Imperial College London, 2007. http://hdl.handle.net/10044/1/11917.
Pełny tekst źródłaDroniou, Magali Eliane. "Localisation of human respiratory syncytial virus proteins during infection". Thesis, University of Warwick, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487956.
Pełny tekst źródłaAlmond, Elizabeth Jennifer Philippa. "Epstein-Barr virus infection of the lower respiratory tract". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1989. http://hub.hku.hk/bib/B31208484.
Pełny tekst źródłaWeber, Martin Willi. "Infection with the respiratory syncytial virus in the Gambia". Thesis, Open University, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.262711.
Pełny tekst źródłaVan, der Heyde Yolande. "Lower respiratory tract infection in sudden unexpected infant deaths". Master's thesis, University of Cape Town, 2012. http://hdl.handle.net/11427/14391.
Pełny tekst źródłaHardisty, Gareth Rhys. "The effect of on-going and persistent infection on acute respiratory infection with influenza A". Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/22080.
Pełny tekst źródłaSpyer, Moira Jane. "Respiratory syncytial virus host cell receptor interactions". Thesis, University of Reading, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269926.
Pełny tekst źródłaXie, Xiaojian, i 解晓健. "Evaporation and movement of respiratory droplets in indoor environments". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40987802.
Pełny tekst źródłaThomas, Linda D., i n/a. "Pseudomonas aeruginosa : development of a mucosal vaccine for respiratory infection". University of Canberra. Human & Biomedical Sciences, 2001. http://erl.canberra.edu.au./public/adt-AUC20061109.130804.
Pełny tekst źródłaMcGill, Alison Kate. "The role of antigenic variation in respiratory syncytial virus infection". Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391959.
Pełny tekst źródłaSÌarmaÌ„, RaviÌ„ndra. "Immuno-pathogenesis of bovine respiratory syncytial virus infection in lambs". Thesis, University of Liverpool, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316908.
Pełny tekst źródłaMuloiwa, Rudzani. "Epidemiology of pertussis in children hospitalised with respiratory tract infection". Doctoral thesis, Faculty of Health Sciences, 2021. http://hdl.handle.net/11427/33846.
Pełny tekst źródłaStokes, A. "Immune effector mechanisms in equine herpesvirus type-1 infection". Thesis, Open University, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233248.
Pełny tekst źródłaJuntti, H. (Hanna). "Association of respiratory syncytial virus infection with asthma and atopic allergy". Doctoral thesis, University of Oulu, 2008. http://urn.fi/urn:isbn:9789514287947.
Pełny tekst źródłaHayes, Peter John. "The role of macrophages in respiratory syncytial virus infection of mice". Thesis, University of Newcastle Upon Tyne, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.358972.
Pełny tekst źródłaHamal, Giuma Fituri. "Respiratory tract infection in infants and young children with bronchopulmonary dysplasia". Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365909.
Pełny tekst źródłaSorensen, George Edwin Peter. "Host-virus interactions in porcine reproductive and respiratory syndrome virus infection". Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/10040.
Pełny tekst źródłaAllman, Mark. "What influences the prescribing of antibiotics in lower respiratory tract infection?" Thesis, University of Bath, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.687300.
Pełny tekst źródłaMolina, Barrios Ramón Miguel. "Porcine reproductive and respiratory syndrome virus understanding and managing persistent infection /". [Ames, Iowa : Iowa State University], 2008.
Znajdź pełny tekst źródłaSchmidt, Megan Elizabeth. "Assessing T cell responses in respiratory syncytial virus infection and vaccination". Diss., University of Iowa, 2019. https://ir.uiowa.edu/etd/6850.
Pełny tekst źródłaBustami, Mona Ratib. "Reactive oxygen and nitrogen species in cystic fibrosis". Thesis, University of Bath, 2002. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248100.
Pełny tekst źródłaWoensel, Jacobus Bernardus Maria van. "Lower respiratory tract infection caused by respiratory syncytial virus the short-term and long-term efficacy of corticosteroids /". [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2002. http://dare.uva.nl/document/63898.
Pełny tekst źródłaAl, Alwadhi Fahimah Kamil M. R. "Upper respiratory tract infection : implementation of multiple interventions on antibiotic prescribing for patients with upper respiratory tract infection in primary health care settings in United Arab Emirates". Thesis, University of Aberdeen, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.440096.
Pełny tekst źródłaBinns, Sarah Helen. "Studies on the epidemiology of Bordetella bronchiseptica infection in cats". Thesis, University of Liverpool, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243267.
Pełny tekst źródłaOrtiguerra, Ryan Gatdula. "Risk Factors Associated With Severe Acute Respiratory Infections Cases". ScholarWorks, 2016. https://scholarworks.waldenu.edu/dissertations/2749.
Pełny tekst źródłaMulpuru, Sunita. "Does Respiratory Viral Testing in Adult Hospitalized Patients Impact Hospital Resource Utilization and Improve Patient Outcomes?" Thèse, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31165.
Pełny tekst źródłaSaravanos, Gemma Lea. "Unknowns of Severe Acute Respiratory Infection (SARI) in Australian Children: Enhancing understanding of epidemiology to inform disease prevention". Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/27237.
Pełny tekst źródłaWest, Keith Henry. "The effect of vaccination on the response to experimental infection with bovine respiratory syncytial virus infection in calves". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ37923.pdf.
Pełny tekst źródłaChauhan, Anoop Jivan. "Personal exposure to the air pollutant nitrogen dioxide and the risk of lower respiratory disease with upper respiratory infection". Thesis, University of Southampton, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266657.
Pełny tekst źródłaDavies, Bronwen J. "Physical activity and symptoms of upper respiratory tract infection in university students". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ61257.pdf.
Pełny tekst źródłaGerman, Matthew. "Tissue specific pathology associated with micronutrient supplementation during respiratory syncytial virus infection". Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=106408.
Pełny tekst źródłaLes micronutriments sont des plus appréciés comme immunomodulateurs puissants. Longtemps utilisé pour traiter measles virus (MeV), la vitamine A a été récemment montré à agir par le biais de RIG-I pour réguler les interférons de type 1. Des actions similaires sont observés avec certains (canine distemper virus, CDV), mais pas tous les membres du Paramyxoviridae. Cependant, les enfants infectés par des voies respiratoire, respiratory syncycial virus (RSV), un proche cousine de MeV, faire pire lorsqu'il est administré des doses pharmacologiques de vitamine A. Le RSV est connu pour induire une résponse pathologique biaisée au Th2. La vitamine A est également une forte influence pour la Th2 déviation. Notre principal objectif était de développer un modèle animal de la carence en vitamine A et la suffisance dans lesquels nous pourrions évaluer l'impact du statut en vitamine A et de la supplémentation sur l'infection à RSV in vivo. Un tel modèle sera d'une grande utilité pour caractériser le mécanisme d'action des rétinoïdes dans cette infection. Nous avons réussi dans le développement de ce modèle en limitant le rétinol alimentaires grâce à deux générations de souris BALB / c (ie: état de carence) et en introduisant de nouveaux moyens de atteindre un état fiable de vitamine A cohérente supplémentation (ie: les Etats reconstitué & excès). Les données préliminaires utilisant ce modèle suggéré qu'il y avait des différences marquées dans la pathologie du RSV entre les groupes de souris déficientes et suffisante. Comme la situation apparente dans les humains, l'infection dans la vitamine A des souris déficientes en était paradoxalement moins sévère que dans les souris avec une vitamine positifs d'un statut. Ce modèle et les données générées avec elle peut être d'un intérêt particulier dans les régions où les régimes son riches en vitamine A (Amérique du Nord en particulier). Historiquement, très peu d'attention a été accordée aux effets négatifs possibles des micronutriments sur-nutrition. Les données humaines limitées et les données préliminaires de notre nouveau modèle remet en question si oui ou non nous sommes d'amorçage nous-mêmes pour l'infection à VRS plus sévères que se produirait autrement. Les données générées dans ce modèle peut également être très pertinentes pour guider les efforts de la supplémentation dans les régions du monde qui ont actuellement un accès moindre à la vitamine A.
Munywoki, Patrick Kiio. "Transmission of respiratory syncytial virus in households : who acquires infection from whom?" Thesis, Open University, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607465.
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