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Książki na temat „Rape (Plant) – Genetics”

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Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 28 stycznia 2023

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Sprawdź 27 najlepszych książek naukowych na temat „Rape (Plant) – Genetics”.

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1

Engqvist, Gabriele. Biometrical genetics of oilseed rape: Selection among crosses in F3̳. Svalöv: Swedish University of Agricultural Sciences, Dept. of Plant Breeding Research, 1993.

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2

Osipova, G. M. Raps v Sibiri: Morfobiologicheskie, geneticheskie i selekt͡s︡ionnye aspekty. Novosibirsk: Rossiĭskai͡a︡ akademii͡a︡ s.-kh. nauk, Sibirskoe otd-nie, Sibirskiĭ nauchno-issl. in-t kormov, 1998.

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3

Commission of the European Communities. Division Genetics and Biotechnology., red. Molecular biology and crop improvement: A case study of wheat, oilseed rape, and faba beans. Cambridge [Cambridgeshire]: Cambridge University Press, 1986.

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4

Zhongguo Xizang you cai yi chuan zi yuan. Beijing: Ke xue chu ban she, 2009.

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5

Austin, R. B. Molecular biology and crop improvement: A case study of wheat, oilseed rape, and faba beans. Cambridge: Cambridge University Press, 2009.

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6

Sjödahl, Staffan. Studies on the structure, expression and gene regulation of cruciferin, the 12S storage globulin from Brassica napus (oilseed rape). Uppsala: Swedish University of Agricultural Sciences, Dept. of Cell Research, 1994.

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7

Rödin, Joakim. Studies on the structure and expression of cruciferin, the 12S storage globulin from Brassica napus (oilseed rape). Uppsala: Swedish University of Agricultural Sciences, Dept. of Cell Research, 1990.

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8

Boronnikova, S. V. Molekuli︠a︡rno-geneticheskai︠a︡ identifikat︠s︡ii︠a︡ i pasportizat︠s︡ii︠a︡ redkikh i nakhodi︠a︡shchikhsi︠a︡ pod ugrozoĭ ischeznovenii︠a︡ vidov rasteniĭ. Permʹ: Permskiĭ gos. universitet, 2009.

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9

Brunsfeld, Steven J. Preliminary genetic analysis of Cirsium longistylum (Long-styled thistle), a candidate threatened species. Helena, MT: Montana Natural Heritage Program, 1994.

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10

F, Smith James. The genetic diversity of the rare Idaho endemic Allium aaseae Ownbey (Alliaceae) and potential introgression with A. simillimum Henderson: Final report. Boise, Idaho: Bureau of Land Management, Idaho State Office, 1995.

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11

E, Kunin William, i Gaston Kevin J, red. The biology of rarity: Causes and consequences of rare-common differences. London: Chapman & Hall, 1997.

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12

Chalhoub, Boulos, Shengyi Liu i Rod Snowdon. Brassica Napus Genome. Springer International Publishing AG, 2018.

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13

Austin, R. B., R. B. Flavell, I. E. Henson i H. J. B. Lowe. Molecular Biology and Crop Improvement: A Case Study of Wheat, Oilseed Rape and Faba Beans. Cambridge University Press, 2010.

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14

Austin, R. B., R. B. Flavell, I. E. Henson i H. J. B. Lowe. Molecular Biology and Crop Improvement: A Case Study of Wheat, Oilseed Rape and Faba Beans. Cambridge University Press, 2012.

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15

B.D.L. Fitt (Editor), N. Evans (Editor), B. J. Howlett (Editor) i B. M. Cooke (Editor), red. Sustainable strategies for managing Brassica napus (oilseed rape) resistance to Leptosphaeria maculans (phoma stem canker). Springer, 2006.

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16

Fitt, B. D. L., B. J. Howlett, N. Evans i B. M. Cooke. Sustainable Strategies for Managing Brassica Napus (oilseed Rape) Resistance to Leptosphaeria Maculans (phoma Stem Canker). Springer London, Limited, 2006.

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17

A, Falk Donald, Holsinger Kent E i Center for Plant Conservation (Boston, Mass.), red. Genetics and conservation of rare plants. New York: Oxford University Press, 1991.

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18

Holsinger, Kent E., i Donald A. Falk. Genetics and Conservation of Rare Plants. Oxford University Press, Incorporated, 1991.

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19

Holsinger, Kent E., i Donald A. Falk. Genetics and Conservation of Rare Plants. Oxford University Press, 1991.

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20

Brunsfeld, Steven J., Montana Natural Heritage Program i U S Fish and Wildlife Service. Preliminary Genetic Analysis of Cirsium Longistylum (Long-Styled Thistle), a Candidate Threatened Species. Creative Media Partners, LLC, 2018.

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21

Daniels, Catherine Hollinger. The expression of disease resistance response genes in Pisum sativum L. during race-specific resistance and heat shock. 1987.

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22

Wang, Kan, Alfredo Herrera-Estrella i Marc van Montagu. Transformation of Plants and Soil Microorganisms. Cambridge University Press, 2012.

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23

Wang, Kan, Alfredo Herrera-Estrella i Marc van Montagu. Transformation of Plants and Soil Microorganisms. Cambridge University Press, 2010.

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24

(Editor), Kan Wang, Alfredo Herrera-Estrella (Editor) i Marc van Montagu (Editor), red. Transformation of Plants and Soil Microorganisms (Biotechnology Research). Cambridge University Press, 2004.

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25

Nachtrieb, Erik S. RAPD marker identification for confirmation of asymmetric somatic hybrids between Brassica oleracea and B. juncea. 1996.

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26

Ahmed, Ahmed I., Sarah Aldhaheri i Allison Bannick. Inherited Metabolic Diseases (IMDs) and Pregnancy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0030.

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Inherited metabolic diseases (IMDs) are rare genetic disorders: clinically heterogeneous, and they can present at any age. With the expanded newborn screening panels, many of the IMDs have been successfully screened. Early diagnosis and treatment of these conditions have led to improved neurological outcomes and overall survival of these individuals, and now many of them are reaching childbearing age. Despite treatment, the potential presence of preexisting organ involvement may not only impact their fertility potentials but also may impose a higher risk of adverse maternal and fetal outcomes. Pregnancy leads to an extra strain on maternal metabolism; this may result in the manifestation of symptoms of a previously unknown disease or a progression of a known disease. This chapter will address the possible complications of some inherited disorders of metabolism that are associated with maternal or fetal neurological manifestations such as disorders of energy metabolism (eg, mitochondrial disorders, adult onset urea cycle disorders, ornithine transcarbamylase (OTC) deficiency, amino acidopathies, phenylketonuria (PKU), and impaired fatty acid oxidation disorders). We will provide special emphasis on the available potential treatments and plan of care during pregnancy and postpartum periods.
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27

Grant, Warren, i Martin Scott-Brown. Prevention of cancer. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0350.

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In the UK, the four commonest cancers—lung cancer, breast cancer, colon cancer, and prostate cancer—result in around 62 000 deaths every year. Although deaths from cancer have fallen in the UK over the last 20 years, the UK still suffers from higher cancer death rates than many other countries in Western Europe. In 1999, the UK government produced a White Paper called Saving Lives: Our Healthier Nation that outlined a national target to reduce the death rate from cancer by at least 20% in people under 75 by 2010. The subsequent NHS Cancer Plan of 2000 designed a framework by which to achieve this target through effective prevention, screening, and treatment programmes as well as restructuring and developing new diagnostic and treatment facilities. But do we know enough about the biology of the development of cancer for government health policies alone to force dramatic changes in survival? The science behind the causes of cancer tells us that its origin lies in acquired or inherited genetic abnormalities. Inherited gene mutation syndromes and exposure to environmental mutagens cause cancer, largely through abnormalities in DNA repair mechanisms, leading to uncontrolled cell proliferation. Although screening those thought to be at highest risk, and regulating exposure to environmental carcinogens such as tobacco or ionizing radiation, have reduced, and will continue to reduce, cancer deaths, there are many other environmental factors that have been shown to increase the population risk of cancer. These will be outlined in this chapter. However, the available evidence is largely from retrospective and cross-sectional population-based studies and therefore limits the ability to apply this knowledge to the risk of the individual patient who may been seen in clinic. Although we may be able to put him or her into a high-, intermediate-, or low-risk category, the question ‘will I get cancer, doc?’ is one that we cannot answer with certainty. The NHS Cancer Plan of 2000, designed to reduce cancer deaths in this country and to bring UK treatment results in line with those other countries in Europe, focuses on preventing malignancy as part of its comprehensive cancer management strategy. It highlights that the rich are less likely to develop cancer, and will survive longer if they are diagnosed than those who live in poverty. This may reflect available treatment options, but is more likely to be related to the lifestyle of those with regular work, as they may be more health aware. The Cancer Plan, however, suggests that relieving poverty may be more labour intensive and less rewarding than encouraging positive risk-reducing behaviour in all members of the population. Eating well can reduce the risk of developing many cancers, particularly of the stomach and bowel. The Cancer Plan outlines the ‘Five-a-Day’ programme which was rolled out in 2002 and encouraged people to eat at least five portions of fruit and vegetables per day. Obese people are also at higher risk of cancers, in particular endometrial cancer. A good diet and regular exercise not only reduce obesity but are also independent risk-reducing factors. Alcohol misuse is thought to be a major risk factor in around 3% of all cancers, with the highest risk for cancers of the mouth and throat. As part of the Cancer Plan, the Department of Health promotes physical activity and general health programmes, as well as alcohol and smoking programmes, particularly in deprived areas. Focusing on these healthy lifestyle points can potentially reduce an individual lifetime risk of all cancers. However, our knowledge of the biology of four cancers in particular has led to the development of specific life-saving interventions. Outlined in this chapter are details regarding ongoing prevention strategies for carcinomas of the lung, the breast, the bowel, and the cervix.
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