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Gotowe bibliografie tematyczne / Pulmonary disease / Artykuły w czasopismach

Artykuły w czasopismach na temat „Pulmonary disease”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 24 stycznia 2023

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1

Herald, G. Peter Praveen, i H. Krishna Murthy. "EVALUATION OF PULMONARY HYPERTENSION IN CHRONIC OBSTRUCTIVE PULMONARY DISEASE". International Journal of Integrative Medical Sciences 6, nr 1 (20.02.2019): 765–68. http://dx.doi.org/10.16965/ijims.2019.102.

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2

De Palo, Vera A. "Pulmonary Disease". Journal of the American Podiatric Medical Association 94, nr 2 (1.03.2004): 157–67. http://dx.doi.org/10.7547/87507315-94-2-157.

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Although many medical problems are generally managed in concert with a general medical physician, it is important for the podiatric physician to be familiar with some of the major diseases of the lung. Pneumonia, an infectious process within the lung, is the sixth-leading overall cause of death. Antibiotic treatment, oxygen administration, and supportive care are the mainstays of its therapy. Chronic obstructive pulmonary disease presents as a spectrum from chronic bronchitis, with a greater inflammatory component, to emphysema, with a more significant destructive component. Asthma, often a more episodic chronic obstructive disease, is characterized by inflammation of the airways leading to their narrowing. The work of breathing is often increased in these diseases, and treatment is with combination therapies with a focus on smoking cessation. Thromboembolic disease, the occlusion of blood vessels with consequent interruption of blood flow, may occur in a patient with risk factors, especially after surgery. Treatment is with anticoagulation agents or in some cases with thrombolysis. Prophylaxis is key. (J Am Podiatr Med Assoc 94(2): 157-167, 2004)

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3

Ramraje, N. N. "Study of OSA in Chronic Obstructive Pulmonary Disease". Journal of Medical Science And clinical Research 05, nr 05 (12.05.2017): 21712–14. http://dx.doi.org/10.18535/jmscr/v5i5.78.

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4

Roe, Paul G., i J. Gareth Jones. "Pulmonary disease". Current Opinion in Anaesthesiology 4, nr 6 (grudzień 1991): 853–59. http://dx.doi.org/10.1097/00001503-199112000-00019.

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5

Musani, Ali I. "Pulmonary Disease". Medical Clinics of North America 103, nr 3 (maj 2019): xix—xx. http://dx.doi.org/10.1016/j.mcna.2019.02.001.

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6

White, Dorothy A., i Muhammad K. Zaman. "Pulmonary disease". Medical Clinics of North America 76, nr 1 (styczeń 1992): 19–44. http://dx.doi.org/10.1016/s0025-7125(16)30369-8.

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7

Musani, Ali I. "Pulmonary Disease". Medical Clinics of North America 103, nr 3 (maj 2019): i. http://dx.doi.org/10.1016/s0025-7125(19)30020-3.

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8

Walker, Patricia A., i Dorothy A. White. "PULMONARY DISEASE". Medical Clinics of North America 80, nr 6 (listopad 1996): 1337–62. http://dx.doi.org/10.1016/s0025-7125(05)70493-4.

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9

Williams, Ruth. "Pulmonary disease". Nursing Management 18, nr 9 (26.01.2012): 13. http://dx.doi.org/10.7748/nm.18.9.13.s12.

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10

Wiedemann, Herbert P., i Richard A. Matthay. "Cor Pulmonale in Chronic Obstructive Pulmonary Disease". Clinics in Chest Medicine 11, nr 3 (wrzesień 1990): 523–45. http://dx.doi.org/10.1016/s0272-5231(21)00715-2.

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11

Devapriya, Inoka A., i Ravi T. Chandran. "CHRONIC OBSTRUCTIVE PULMONARY DISEASE AND COR PULMONALE". Chest 130, nr 4 (październik 2006): 171S. http://dx.doi.org/10.1378/chest.130.4_meetingabstracts.171s-a.

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12

Morley, Thomas F., Silvio J. Zappasodi, Albert Belli i James C. Giudice. "Pulmonary Vasodilator Therapy for Chronic Obstructive Pulmonary Disease and Cor Pulmonale". Chest 92, nr 1 (lipiec 1987): 71–76. http://dx.doi.org/10.1378/chest.92.1.71.

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13

Liao, Shi-xia, Peng-peng Sun, Yan-hui Gu, Xi-min Rao, Lan-ying Zhang i Yao Ou-Yang. "Autophagy and pulmonary disease". Therapeutic Advances in Respiratory Disease 13 (styczeń 2019): 175346661989053. http://dx.doi.org/10.1177/1753466619890538.

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Autophagy is a process of cell self-renewal that is dependent on the degradation of the cytoplasmic proteins or organelles of lysosomes. Many diseases, such as metabolic diseases, cancer, neurodegenerative diseases, and lung diseases, have been confirmed to be associated with elevated or impaired levels of autophagy. At present, studies have found that autophagy participates in the regulation of chronic obstructive pulmonary disease (COPD), idiopathic pulmonary fibrosis, pulmonary hypertension, acute lung injury, lung cancer, and other pulmonary diseases. Using recent literature on the signal transduction mechanisms of autophagy and the effects of autophagy signalling on lung diseases, this review intends to clarify the mechanisms of lung disease to guide the treatment of related diseases. The reviews of this paper are available via the supplemental material section.

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14

Andreev, V. M. "Diagnosis and treatment of patients with chronic cor pulmonale". Kazan medical journal 76, nr 2 (15.03.1995): 105–8. http://dx.doi.org/10.17816/kazmj97077.

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Cor pulmonale refers to hypertrophy and (or) dilatation of the right ventricle due to diseases that affect the structure or function of the lungs, or both at the same time. Chronic cor pulmonale occurs with lesions of the bronchopulmonary system (chronic bronchitis, bronchial asthma, pulmonary emphysema, pulmonary tuberculosis, etc.), impaired mobility of the chest (kyphoscoliosis, Bechterew's disease, pleural folds, obesity, etc.) and diseases of the pulmonary artery system (repeated thromboembolism, primary pulmonary hypertension, lung resection, etc.). Right ventricular hypertrophy and insufficiency also occur with secondary changes in the lungs in patients with diseases of the left heart (cardiosclerosis of various etiologies, mitral stenosis) and congenital heart defects, but these cases do not apply to cor pulmonale. Most often, cor pulmonale develops in diseases of the bronchopulmonary system.

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15

Carlin, Chris, i Andrew J. Peacock. "Pulmonary vascular disease: pulmonary thromboembolism and pulmonary hypertension". Medicine 36, nr 6 (czerwiec 2008): 331–37. http://dx.doi.org/10.1016/j.mpmed.2008.03.002.

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16

Davies, Rachel, i Luke Howard. "Pulmonary vascular disease: pulmonary thromboembolism and pulmonary hypertension". Medicine 40, nr 4 (kwiecień 2012): 214–20. http://dx.doi.org/10.1016/j.mpmed.2012.01.003.

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17

Davies, Rachel, i Luke Howard. "Pulmonary vascular disease: pulmonary thromboembolism and pulmonary hypertension". Medicine 44, nr 4 (kwiecień 2016): 255–62. http://dx.doi.org/10.1016/j.mpmed.2016.02.006.

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18

Haji, Gulam, Nicola Read i Rachel Davies. "Pulmonary vascular disease: pulmonary thromboembolism and pulmonary hypertension". Medicine 48, nr 4 (kwiecień 2020): 288–93. http://dx.doi.org/10.1016/j.mpmed.2020.01.006.

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19

Newman, Glenn E. "Pulmonary angiography in pulmonary embolic disease". Journal of Thoracic Imaging 4, nr 4 (październik 1989): 28–39. http://dx.doi.org/10.1097/00005382-198910000-00010.

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20

Ghigna, Maria-Rosa, i Peter Dorfmüller. "Pulmonary vascular disease and pulmonary hypertension". Diagnostic Histopathology 25, nr 8 (sierpień 2019): 304–12. http://dx.doi.org/10.1016/j.mpdhp.2019.05.002.

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21

Cheng, Guang-Shing, i Jennifer D. Possick. "Pulmonary Disease in Non-Pulmonary Malignancy". Clinics in Chest Medicine 38, nr 2 (czerwiec 2017): i. http://dx.doi.org/10.1016/s0272-5231(17)30011-4.

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22

Cheng, Guang-Shing, i Jennifer D. Possick. "Pulmonary Disease in Non-Pulmonary Malignancy". Clinics in Chest Medicine 38, nr 2 (czerwiec 2017): xiii—xiv. http://dx.doi.org/10.1016/j.ccm.2017.03.001.

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23

Kawashima, Masahiro, Nobuharu Ohshima i Nobuyuki Kobayashi. "3. Pulmonary Disease". Nihon Naika Gakkai Zasshi 102, nr 10 (2013): 2549–57. http://dx.doi.org/10.2169/naika.102.2549.

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24

Nelson, Kenwyn G., David Griffith i Richard J. Wallace. "Pulmonary Mycobacterial Disease". Clinical Pulmonary Medicine 11, nr 6 (listopad 2004): 355–62. http://dx.doi.org/10.1097/01.cpm.0000145617.71992.50.

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25

Gadkowski, L. Beth, i Jason E. Stout. "Cavitary Pulmonary Disease". Clinical Microbiology Reviews 21, nr 2 (kwiecień 2008): 305–33. http://dx.doi.org/10.1128/cmr.00060-07.

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SUMMARY A pulmonary cavity is a gas-filled area of the lung in the center of a nodule or area of consolidation and may be clinically observed by use of plain chest radiography or computed tomography. Cavities are present in a wide variety of infectious and noninfectious processes. This review discusses the differential diagnosis of pathological processes associated with lung cavities, focusing on infections associated with lung cavities. The goal is to provide the clinician and clinical microbiologist with an overview of the diseases most commonly associated with lung cavities, with attention to the epidemiology and clinical characteristics of the host.

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26

Bone, Roger C., i Michael W. Owens. "PULMONARY DISEASE REVIEWS". Critical Care Medicine 15, nr 11 (listopad 1987): 1077. http://dx.doi.org/10.1097/00003246-198711000-00028.

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27

Goldhaber, Samuel Z. "Thromboembolic pulmonary disease". Current Opinion in Cardiology 5, nr 5 (październik 1990): 677–80. http://dx.doi.org/10.1097/00001573-199010000-00018.

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28

Pineo, Graham F., i Russell D. Hull. "Pulmonary vascular disease". Current Opinion in Pulmonary Medicine 7, nr 5 (wrzesień 2001): 323–25. http://dx.doi.org/10.1097/00063198-200109000-00012.

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29

Pineo, Graham F., i Russell D. Hull. "Pulmonary vascular disease". Current Opinion in Pulmonary Medicine 8, nr 5 (wrzesień 2002): 357–59. http://dx.doi.org/10.1097/00063198-200209000-00002.

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30

Cleutjens, Fiona A. H. M., Daisy J. A. Janssen, Rudolf W. H. M. Ponds, Jeanette B. Dijkstra i Emiel F. M. Wouters. "COgnitive-Pulmonary Disease". BioMed Research International 2014 (2014): 1–8. http://dx.doi.org/10.1155/2014/697825.

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Over the past few decades, chronic obstructive lung disease (COPD) has been considered a disease of the lungs, often caused by smoking. Nowadays, COPD is regarded as a systemic disease. Both physical effects and effects on brains, including impaired psychological and cognitive functioning, have been demonstrated. Patients with COPD may have cognitive impairment, either globally or in single cognitive domains, such as information processing, attention and concentration, memory, executive functioning, and self-control. Possible causes are hypoxemia, hypercapnia, exacerbations, and decreased physical activity. Cognitive impairment in these patients may be related to structural brain abnormalities, such as gray-matter pathologic changes and the loss of white matter integrity which can be induced by smoking. Cognitive impairment can have a negative impact on health and daily life and may be associated with widespread consequences for disease management programs. It is important to assess cognitive functioning in patients with COPD in order to optimize patient-oriented treatment and to reduce personal discomfort, hospital admissions, and mortality. This paper will summarize the current knowledge about cognitive impairment as extrapulmonary feature of COPD. Hereby, the impact of smoking on cognitive functioning and the impact of cognitive impairment on smoking behaviour will be examined.

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31

Barst, Robyn J. "Pulmonary Vascular Disease". Chest 114, nr 1 (lipiec 1998): 8–9. http://dx.doi.org/10.1378/chest.114.1.8.

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32

Burrows, Benjamin, Joseph S. Alpert i Joseph C. Ross. "Pulmonary heart disease". Journal of the American College of Cardiology 10, nr 2 (sierpień 1987): 63A—65A. http://dx.doi.org/10.1016/s0735-1097(87)80452-7.

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33

Bale, Patricia. "Pediatric Pulmonary Disease". Pathology 21, nr 4 (1989): 322. http://dx.doi.org/10.1016/s0031-3025(16)36454-6.

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34

BENYEHUDA, O. "Pulmonary vascular disease". Cardiology Clinics 22, nr 3 (sierpień 2004): xi—xii. http://dx.doi.org/10.1016/s0733-8651(04)00027-x.

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35

CRAWFORD, M. "Pulmonary Vascular Disease". Cardiology Clinics 22, nr 3 (sierpień 2004): ix. http://dx.doi.org/10.1016/s0733-8651(04)00028-1.

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36

Cataño, Juan Carlos, i Diana Karina Agredo. "Hepatic-pulmonary disease". European Journal of Internal Medicine 64 (czerwiec 2019): e5-e6. http://dx.doi.org/10.1016/j.ejim.2018.12.013.

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37

Ben-Yehuda, Ori. "Pulmonary vascular disease". Cardiology Clinics 22, nr 3 (sierpień 2004): xi—xii. http://dx.doi.org/10.1016/j.ccl.2004.04.012.

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38

Stulbarg, Michael S., i James A. Frank. "OBSTRUCTIVE PULMONARY DISEASE". Radiologic Clinics of North America 36, nr 1 (styczeń 1998): 1–13. http://dx.doi.org/10.1016/s0033-8389(05)70004-x.

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39

Pineo, Graham F., i Russell D. Hull. "Pulmonary vascular disease". Current Opinion in Pulmonary Medicine 12, nr 5 (wrzesień 2006): 289–90. http://dx.doi.org/10.1097/01.mcp.0000239541.50381.3c.

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40

Kaur Sandhu, Azmat, Pavneet Kaur Selhi i Ruchita Tyagi. "Pulmonary hydatid disease". IP Journal of Diagnostic Pathology and Oncology 4, nr 4 (15.12.2019): 338–41. http://dx.doi.org/10.18231/j.jdpo.2019.069.

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41

Bell, William R., i John R. Bartholomew. "Pulmonary thromboembolic disease". Current Problems in Cardiology 10, nr 9 (wrzesień 1985): 1–69. http://dx.doi.org/10.1016/0146-2806(85)90031-3.

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42

Regalia, Kirsten, Andrew Shelton, Gerald Berry, George Triadafilopoulos i Christine A. Cartwright. "Pulmonary Crohn’s Disease". Digestive Diseases and Sciences 62, nr 1 (21.12.2015): 64–67. http://dx.doi.org/10.1007/s10620-015-3993-1.

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43

Weissmann, Norbert. "Chronic Obstructive Pulmonary Disease and Pulmonary Vascular Disease. A Comorbidity?" Annals of the American Thoracic Society 15, Supplement_4 (grudzień 2018): S278—S281. http://dx.doi.org/10.1513/annalsats.201808-532mg.

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44

Weitzenblum, Emmanuel, Ari Chaouat, Matthieu Canuet i Romain Kessler. "Pulmonary Hypertension in Chronic Obstructive Pulmonary Disease and Interstitial Lung Diseases". Seminars in Respiratory and Critical Care Medicine 30, nr 04 (24.07.2009): 458–70. http://dx.doi.org/10.1055/s-0029-1233315.

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45

Passowicz-Muszyńska, Ewa, Anna Gostkowska-Malec, Renata Jankowska i Paweł Piesiak. "Chronic obstructive pulmonary disease and cardiovascular diseases". Pneumonologia i Alergologia Polska 78, nr 1 (29.01.2010): 28–32. http://dx.doi.org/10.5603/arm.27751.

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46

Kumar, Priyatham. "Deep Vein Thrombosis and Pulmonary Embolism in Sickle Cell Disease". Biomedical Research and Clinical Reviews 1, nr 5 (4.12.2020): 01–04. http://dx.doi.org/10.31579/2692-9406/024.

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Sickle Cell Disease (SCD) is considered a group of genetic red blood cell (RBC) disorders. Healthy red blood cells (RBC) are round in shape and migrates throughout the body to carry oxygen in the small blood vessels. In SCD, the RBC turns into hard and sticky, and the shape is similar to a C-Shaped tool called "SICKLE." Because of the early death of the sickle cells, a constant shortage of red blood cells arises. Because of the typical shape of the sickle cells, their movement in the blood vessel is not as smooth as normal RBC and get stuck and clog the blood flow leading to anemia. The changes in shape make the cells more easily destroyed, causing anemia. Defective hemoglobin is the primary cause of SCD.

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47

Arabboyevich, Anvar Dalimov, Dilfuzahon Mamarasulova Zakirjanovna i Nodirbek Ilkhomjon Ogli Yakubov. "Clinical, Radiological And Laboratory Predictors Of Postcovid Interstitial Pulmonary Disease". American Journal of Medical Sciences and Pharmaceutical Research 03, nr 08 (31.08.2021): 26–36. http://dx.doi.org/10.37547/tajmspr/volume03issue08-06.

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The Fergana Valley model was used to study the risk of postcovid interstitial lung disease in patients who have had COVID-19 associated pneumonia with 50% or more of the pulmonary parenchyma affected. Predictors of the formation of postcovid pulmonary fibrosis were determined and a risk assessment scale was developed. It was found that the use of ultrasound scanners in the early postcovid period is informative and is not inferior in terms of predicting fibrosis by serial MSCT.

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48

Adedeji, Moses O., Julio Cespedes, Kay Allen, Charu Subramony i Michael D. Hughson. "Pulmonary Thrombotic Arteriopathy in Patients With Sickle Cell Disease". Archives of Pathology & Laboratory Medicine 125, nr 11 (1.11.2001): 1436–41. http://dx.doi.org/10.5858/2001-125-1436-ptaipw.

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Abstract Background.—Shortened life expectancy due to pulmonary hypertension (PH) is seen in 5% to 10% of patients with sickle cell disease. The principal factors suspected of causing PH are pulmonary thromboemboli (PE) and in situ arterial thrombosis. Objective.—To investigate the possible role that PE or in situ arterial thrombosis play in the development of PH in sickle cell disease. Methods.—Autopsies of 12 patients with sickle cell disease were correlated with clinical data from medical records. Results.—Right ventricular hypertrophy was present in 9 of 12 patients. Six patients with right ventricular hypertrophy had thrombi in large elastic pulmonary arteries. All patients with elastic artery thrombi had fresh or organized thrombi in small muscular pulmonary arteries. Hypertensive small arterial changes were present in 5 of these 6 patients. Six patients showed no thrombi in elastic arteries. Among these 6 patients, 3 had right ventricular hypertrophy and recent and organized thrombi, as well as hypertensive changes in small arteries. One of these 3 patients demonstrated plexiform-like lesions and fibrinoid necrosis of small arteries. Three patients without right ventricular hypertrophy had pneumonia or pulmonary edema with no identifiable pulmonary artery pathology. Conclusions.—Arterial thrombosis with PH and cor pulmonale was regarded as the cause of death among most of these patients. Elastic artery thrombi are pulmonary thromboemboli, but pulmonary thromboemboli are always associated with widespread thrombosis of small arteries. Widespread thrombosis of small arteries alone was associated with PH in some cases. This finding suggests that pulmonary thromboemboli may be a late complication of PH and cor pulmonale and that an in situ thrombotic arteriopathy underlies the development of PH in most patients with sickle cell disease.

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49

Tee, Augustine KH. "Chronic Obstructive Pulmonary Disease (COPD): “Not a Cigarette Only Pulmonary Disease”". Annals of the Academy of Medicine, Singapore 46, nr 11 (15.11.2017): 415–16. http://dx.doi.org/10.47102/annals-acadmedsg.v46n11p415.

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50

Foster, Steven, i Henry M. Thomas. "Pulmonary Rehabilitation in Lung Disease Other Than Chronic Obstructive Pulmonary Disease". American Review of Respiratory Disease 141, nr 3 (marzec 1990): 601–4. http://dx.doi.org/10.1164/ajrccm/141.3.601.

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