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Gotowe bibliografie tematyczne / Pulmonary disease / Książki

Książki na temat „Pulmonary disease”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 24 stycznia 2023

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1

Farver, Carol, Subha Ghosh, Thomas Gildea i Charles D. Sturgis. Pulmonary Disease. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47598-7.

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2

Thomas, Stocker J., i Society for Pediatric Pathology (U.S.). Meeting, red. Pediatric pulmonary disease. New York: Hemisphere Pub. Corp., 1989.

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3

A, Stockley Robert, red. Chronic obstructive pulmonary disease. Malden, Mass: Blackwell Pub., 2005.

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4

Cytopathology of pulmonary disease. Basel, Switzerland: Karger, 1988.

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5

Currie, Graeme P. Chronic obstructive pulmonary disease. Oxford: Oxford University Press, 2009.

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6

Nakamura, Hiroyuki, i Kazutetsu Aoshiba, red. Chronic Obstructive Pulmonary Disease. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-0839-9.

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7

Nici, Linda, i Richard ZuWallack, red. Chronic Obstructive Pulmonary Disease. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-673-3.

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8

Stockley, Robert A., Stephen I. Rennard, Klaus Rabe i Bartolome Celli, red. Chronic Obstructive Pulmonary Disease. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470755976.

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9

Calverley, P. M. A., i N. B. Pride, red. Chronic Obstructive Pulmonary Disease. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9.

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10

Pokorski, Mieczyslaw, red. Pulmonary Dysfunction and Disease. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42010-3.

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11

Celli, Bartolome R., i Stephen I. Rennard. Chronic obstructive pulmonary disease. Philadelphia, Pennsylvania: Saunders, an imprint of Elsevier, Inc., 2012.

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12

A, Calverley P. M., red. Chronic obstructive pulmonary disease. Wyd. 2. London: Arnold, 2003.

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13

1948-, Ambrosino N., i Goldstein Roger, red. Pulmonary rehabilitation. London: Hodder Arnold, 2005.

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14

Highland, Kristin B. Pulmonary manifestations of rheumatic disease. Philadelphia, Pa: Saunders, 2010.

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15

Dumont, Robert, i Youngran Chung, red. Nutrition in Pediatric Pulmonary Disease. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8474-5.

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16

McCormack, Francis X., Ralph J. Panos i Bruce C. Trapnell, red. Molecular Basis of Pulmonary Disease. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-59745-384-4.

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17

Pulmonary Vascular Disease. Saunders, 2006.

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18

Pulmonary Heart Disease. Springer, 2013.

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19

Shure. Pulmonary Vascular Disease. B.C. Decker, 2006.

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20

Rubin, L. J. Pulmonary Heart Disease. Springer London, Limited, 2012.

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21

Jess, Mandel, i Taichman Darren, red. Pulmonary vascular disease. Philadelphia, Pa: Saunders Elsevier, 2006.

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22

Rubin, L. J. Pulmonary Heart Disease. Springer, 2011.

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23

Pulmonary Vascular Disease. Elsevier, 2006. http://dx.doi.org/10.1016/b978-1-4160-2246-6.x5001-9.

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24

Katritsis, Demosthenes G., Bernard J. Gersh i A. John Camm. Pulmonary valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0396_update_002.

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25

Pulmonary Disease Reviews. John Wiley & Sons Inc, 1987.

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26

Crapo, James D., Jeffrey L. Glassroth, Talmadge E. King i Joel B. Karlinsky. Baum's Textbook of Pulmonary Diseases (Textbook of Pulmonary Disease-(Baum)). Wyd. 7. Lippincott Williams & Wilkins, 2003.

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27

Yearbook of Pulmonary Disease 1998 (Year Book of Pulmonary Disease). Mosby-Year Book, 1998.

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28

Scharloo, Margreet, Maarten J. Fischer, Esther Van Den Ende i Adrian A. Kapstein. Pulmonary Rehabilitation in Chronic Obstructive Pulmonary Disease. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780199733989.013.0018.

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29

Miller, Owen I., i Werner Budts. Heart valve disease: pulmonary valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0038.

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Streszczenie:

Congenital abnormalities of the pulmonary valve (PV) are common either as a single lesion or in the context of more complex congenital lesions where abnormalities of the PV play a major role in the cardiac physiology. Transthoracic echocardiographic (TTE) imaging of the PV is relatively straightforward in the normally connected heart due to its anterior position close to common sonographic windows. Imaging of the abnormally positioned PV requires modifications to standard projections and may be better demonstrated by a transoesophageal (TOE) or three-dimensional (3D) echocardiographic approach. Standard 3D TTE may offer advantages in surgical planning for an abnormally positioned pulmonary valve in complex congenital anatomy and 3D TOE may add value to the demonstration of abnormalities of the subpulmonary right ventricular outflow tract.

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30

Year Book of Pulmonary Diseases 2011 Year Book of Pulmonary Disease. Mosby, 2011.

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31

1932-, Petty Thomas L., red. Chronic obstructive pulmonary disease. Wyd. 2. New York: Dekker, 1985.

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32

Wilford Hall USAF Medical Center. Pulmonary Disease Service, red. Manual of pulmonary disease. [San Antonio, Tex.?]: Pulmonary Disease Service, 1988.

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33

Rennard, Stephen I., Robert A. Stockley, Klaus Rabe i Bartolome Celli. Chronic Obstructive Pulmonary Disease. Wiley & Sons, Incorporated, John, 2008.

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34

National Heart, Lung, and Blood Institute. Division of Lung Diseases i National Heart, Lung, and Blood Institute. Office of Prevention, Education, and Control, red. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: National Institutes of Health, National Heart, Lung, and Blood Institute, 1993.

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35

S, Niederman Michael, red. Pulmonary disease in pregnancy. Philadelphia: Saunders, 1992.

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36

1939-, Hodgkin John E., red. Chronic obstructive pulmonary disease. Philadelphia: W. B. Saunders, 1990.

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37

National Heart, Lung, and Blood Institute, red. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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38

J, Saldana Mario, red. Pathology of pulmonary disease. Philadelphia: J.B. Lippincott Co., 1994.

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39

D, Cooper J. Allen, red. Drug-induced pulmonary disease. Philadelphia: W. B. Saunders, 1990.

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40

Rennard, Stephen I., Robert A. Stockley, Klaus Rabe i Bartolome Celli. Chronic Obstructive Pulmonary Disease. Wiley & Sons, Incorporated, John, 2008.

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41

National Heart, Lung, and Blood Institute., red. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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42

Pokorski, Mieczyslaw. Pulmonary Dysfunction and Disease. Springer, 2018.

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43

Pediatric Pulmonary Heart Disease. Lippincott Williams & Wilkins, 1990.

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44

Rennard, S., P. M. A. Calverley, N. Pride i W. Macnee. Chronic Obstructive Pulmonary Disease. Taylor & Francis Group, 2003.

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45

Pokorski, Mieczyslaw. Pulmonary Dysfunction and Disease. Springer London, Limited, 2016.

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46

Pride, N. B., i P. M. A. Calverley. Chronic Obstructive Pulmonary Disease. Springer London, Limited, 2013.

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47

Pokorski, Mieczyslaw. Pulmonary Dysfunction and Disease. Springer, 2016.

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48

Mills, Gary H. Pulmonary disease and anaesthesia. Redaktor Philip M. Hopkins. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0082.

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Respiratory adverse events are the commonest complications after anaesthesia and have profound implications for the recovery of the patient and their subsequent health. Outcome prediction related to respiratory disease and complications is vital when determining the risk:benefit balance of surgery and providing informed consent. Surgery produces an inflammatory response and pain, which affects the respiratory system. Anaesthesia produces atelectasis, decreases the drive to breathe, and causes muscle weakness. As the respiratory system ages, closing capacity increases and airway closure becomes an increasing issue, resulting in atelectasis. Increasing comorbidity and polypharmacy reduces the patient’s ability to eliminate drugs. The proportion of major operations on older frailer patients is rising and postoperative recovery becomes more complicated and the demand for critical care rises. At the same time, the population is becoming more obese, producing rapid decreases in end-expiratory lung volume on induction, together with a high incidence of sleep-disordered breathing. Despite this, many high-risk patients are not accurately identified preoperatively, and of those that are admitted to critical care, some are discharged and then readmitted to the intensive care unit with complications. Respiratory diseases may lead to increases in pulmonary vascular resistance and increased load on the right heart. Some lung diseases are primarily fibrotic or obstructive. Some are inflammatory, autoimmune, or vasculitic. Other diseases relate to the drive to breathe, the nerve supply to, or the respiratory muscles themselves. The range of types of respiratory disease is wide and the physiological consequences of respiratory support are complex. Research continues into the best modes of respiratory support in theatre and in the postoperative period and how best to protect the normal lung. It is therefore essential to understand the effects of surgery and anaesthesia and how this impacts existing respiratory disease, and the way this affects the balance between load on the respiratory system and its capacity to cope.

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49

Davey, Patrick, Sherif Gonem, Salman Siddiqui i David Sprigings. Chronic obstructive pulmonary disease. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0134.

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The Global Initiative for Chronic Lung Disease (GOLD) states that ‘chronic obstructive pulmonary disease (COPD), a common preventable and treatable disease, is characterised by persistent airflow limitation that is usually progressive and is associated with an enhanced chronic inflammatory response in the airways and the lung to noxious particles and gases. Exacerbations and comorbidities contribute to the overall severity in individual patients.’

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50

Barnes, Peter J., red. Chronic Obstructive Pulmonary Disease. CRC Press, 2005. http://dx.doi.org/10.1201/b14103.

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