Gotowa bibliografia na temat „Pulmonary arterial hyperreactivity”
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Artykuły w czasopismach na temat "Pulmonary arterial hyperreactivity"
Cardouat, G., M. Douard, L. Roubenne, C. Bouchet, Z. Kmecová, P. Robillard, C. Guignabert i in. "NGF induces pulmonary arterial hyperreactivity through Connexin 43 increased expression". Revue des Maladies Respiratoires 38, nr 6 (czerwiec 2021): 582. http://dx.doi.org/10.1016/j.rmr.2021.02.029.
Pełny tekst źródłaCardouat, G., M. Douard, L. Roubenne, S. Kmecova, P. Robillard, C. Guignabert, L. Tu i in. "NGF induces pulmonary arterial hyperreactivity through Connexin 43 increased expression". Archives of Cardiovascular Diseases Supplements 12, nr 2-4 (październik 2020): 213. http://dx.doi.org/10.1016/j.acvdsp.2020.03.036.
Pełny tekst źródłaBouchet, Clement, Guillaume Cardouat, Matthieu Douard, Paul Robillard, Roger Marthan, Christelle Guibert i Véronique Freund-Michel. "Short-term mechanisms activated by NGF to induce pulmonary arterial hyperreactivity". Archives of Cardiovascular Diseases Supplements 14, nr 2 (czerwiec 2022): 157–58. http://dx.doi.org/10.1016/j.acvdsp.2022.04.147.
Pełny tekst źródłaBouchet, C., G. Cardouart, M. Douard, Z. Kmecova, P. Robillard, F. Delcambre, R. Marthan, P. Berger, C. Guibert i V. Freund-Michel. "Short-term mechanisms activated by NGF to induce pulmonary arterial hyperreactivity". Revue des Maladies Respiratoires 40, nr 2 (luty 2023): 129. http://dx.doi.org/10.1016/j.rmr.2022.11.041.
Pełny tekst źródłaHinton, M., L. Mellow, A. J. Halayko, A. Gutsol i S. Dakshinamurti. "Hypoxia induces hypersensitivity and hyperreactivity to thromboxane receptor agonist in neonatal pulmonary arterial myocytes". American Journal of Physiology-Lung Cellular and Molecular Physiology 290, nr 2 (luty 2006): L375—L384. http://dx.doi.org/10.1152/ajplung.00307.2005.
Pełny tekst źródłaPeták, Ferenc, Tibor Z. Janosi, Carole Myers, Fabienne Fontao i Walid Habre. "Impact of elevated pulmonary blood flow and capillary pressure on lung responsiveness". Journal of Applied Physiology 107, nr 3 (wrzesień 2009): 780–86. http://dx.doi.org/10.1152/japplphysiol.00157.2009.
Pełny tekst źródłaSmith, Kelly J., George B. Mallory, Steven H. Abman i Dunbar D. Ivy. "AIRWAY HYPERREACTIVITY AFTER ILOPROST INHALATION IN PEDIATRIC PATIENTS WITH PULMONARY ARTERIAL HYPERTENSION". Chest 130, nr 4 (październik 2006): 156S. http://dx.doi.org/10.1378/chest.130.4_meetingabstracts.156s-b.
Pełny tekst źródłaMichel, R. P., i T. S. Hakim. "Increased resistance in postobstructive pulmonary vasculopathy: structure-function relationships". Journal of Applied Physiology 71, nr 2 (1.08.1991): 601–10. http://dx.doi.org/10.1152/jappl.1991.71.2.601.
Pełny tekst źródłaAzamar-Solis, Brizeida, Yahveth Cantero-Fortiz, Juan Carlos Olivares-Gazca, Jesús Mauricio Olivares-Gazca, Gisela Berenice Gómez-Cruz, Iván Murrieta-Álvarez, Guillermo J. Ruiz-Delgado i Guillermo J. Ruiz-Argüelles. "Primary Thrombophilia in Mexico XIII: Localization of the Thrombotic Events in Mexican Mestizos With the Sticky Platelet Syndrome". Clinical and Applied Thrombosis/Hemostasis 25 (1.01.2019): 107602961984170. http://dx.doi.org/10.1177/1076029619841700.
Pełny tekst źródłaOhar, Jill A., Kemberly S. Waller i Thomas E. Dahms. "Platelet-activating Factor Induces Selective Pulmonary Arterial Hyperreactivity in Isolated Perfused Rabbit Lungs". American Review of Respiratory Disease 148, nr 1 (lipiec 1993): 158–63. http://dx.doi.org/10.1164/ajrccm/148.1.158.
Pełny tekst źródłaRozprawy doktorskie na temat "Pulmonary arterial hyperreactivity"
Bouchet, Clement. "Expression et rôle du facteur de croissance des nerfs NGF dans l’hypertension pulmonaire". Electronic Thesis or Diss., Bordeaux, 2024. http://www.theses.fr/2024BORD0092.
Pełny tekst źródłaPulmonary hypertension (PH) is a severe disease characterized by an increase in mean pulmonary arterial pressure over 20mmHg at rest, compared with 14mmHg in healthy individuals. Ultimately, PH leads to right heart failure and death. In the absence of any curative treatment, identification of new therapeutic targets is a major challenge. In this context, a better understanding of PH development and progression is crucial. In our laboratory, one target of interest is the nerve growth factor or NGF, overexpressed in pulmonary arteries both in experimental and human PH, and playing a significant role in three major PH features: pulmonary arterial inflammation, reactivity and remodeling.First, we focused on possible mechanisms activated in PH that may contribute to NGF pulmonary arterial increased expression. We showed that pro-inflammatory cytokines whose expression are increased in PH, such as interleukin-1β (IL-1β) or Tumor necrosis factor-α, as well as oxidative stress can increase NGF expression and secretion by pulmonary arterial endothelial cells - EC- or smooth muscle cells -CML-. We showed that this secretion involved Transforming growth factor β1, which is secreted by both ECs and SMCs, and then acts in an autocrine manner on these cells to stimulate NGF secretion.We then studied the role of NGF in two major PH features: pulmonary arterial hyperreactivity and inflammation.Regarding hyperreactivity first, we showed that a chronic treatment with NGF (24h) led to pulmonary arterial hyperreactivity, by enhancing activity of connexin-43-dependent gap junctions in SMCs. In parallel, we showed that a shorter treatment with NGF (1h) also led to led to pulmonary arterial hyperreactivity, but in a connexin-43-independent manner, with deregulation of intracellular calcium signaling and increased sensitization of the contractile apparatus in SMCs.Regarding the link between NGF and inflammation, our results showed that in vivo, in a rat model of PH, a preventive treatment with anti-NGF blocking antibodies reduced pulmonary arterial monocyte/macrophage infiltration and inflammasome protein expression as well as both circulating and pulmonary levels of IL-1β. In vitro, we showed that NGF is able to attract monocytes, and that IL-1β plays an important role in pulmonary arterial remodeling, inflammation and endothelial dysfunction.In conclusion, this work has highlighted mechanisms contributing to NGF increased expression in PH and confirmed its involvement in the progression of the disease. The overall results obtained further support NGF as a therapeutic target of interest in this disease
Streszczenia konferencji na temat "Pulmonary arterial hyperreactivity"
Cardouat, guillaume, Matthieu Douard, Lukas Roubenne, Zuzana Kmecová, Paul Robillard, Christophe Guignabert, Ly Tu i in. "NGF induces pulmonary arterial hyperreactivity through connexin 43 increased expression". W ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3565.
Pełny tekst źródłaBOUCHET, Clement, Guillaume Cardouat, Matthieu Douart, Paul Robillard, Roger Marthan, Bernard Muller, Christelle Guibert i Veronique Freund-Michel. "Short-term mechanisms activated by NGF to induce pulmonary arterial hyperreactivity". W ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa585.
Pełny tekst źródłaSikarwar, Anurag S., K. T. Santhosh i Shyamala Dakshinamurti. "Inhibition Of Palmitoylation Normalizes Hyperreactivity Of The Pulmonary Arterial Thromboxane Receptor In Hypoxic Pulmonary Hypertension". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5034.
Pełny tekst źródłaDouard, Matthieu, Paul Robillard, Juliette Deweirdt, Isabelle Baudrimont, Mathilde Dubois, Roger Marthan, Jean-Pierre Savineau, Bernard Muller, Christelle Guibert i Veronique Freund-Michel. "Late Breaking Abstract - NGF induces pulmonary arterial hyperreactivity through increased connexin-43 expression: potential role in pulmonary hypertension". W ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.oa3274.
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