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Książki na temat „Protein Based Molecular Diseases”

Autor: Grafiati

Data publikacji: 7 września 2023

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1

Protein chaperones and protection from neurodegenerative diseases. Hoboken: John Wiley & Sons, 2011.

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2

Protein evolution. Oxford [England]: Blackwell Science, 1999.

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3

Henrik, Bohr, i Brunak Søren, red. Protein folds: A distance-based approach. Boca Raton: CRC Press, 1996.

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4

Coleman, Thomas F. Parallel continuation-based global optimization for molecular conformation and protein folding. Ithaca, N.Y: Cornell Theory Center, Cornell University, 1994.

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5

Protein evolution. Wyd. 2. Oxford: Blackwell Science, 2007.

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6

service), SpringerLink (Online, red. Osteoimmunopathology: Evidence-Based Perspectives from Molecular Biology to Systems Biology. New York, NY: Springer Science+Business Media, LLC, 2011.

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7

Crichton, Robert R. Metal-based neurodegeneration: From molecular mechanisms to therapeutic strategies. Wyd. 2. Chichester, West Sussex, U.K: John Wiley & Sons, 2013.

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8

J, Ward Roberta, red. Metal-based neurodegeneration: From molecular mechanisms to therapeutic strategies. Chichester: J. Wiley & Sons, 2006.

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9

Schwarz, Siegfried. Molecules of life & mutations: Understanding diseases by understanding proteins. Basel: Karger, 2002.

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10

Schwarz, Siegfried. Molecules of life & mutations: Understanding diseases by understanding proteins. Basel: Karger, 2002.

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11

Xu, Jiru. Application of PCR and DNA sequencing based molecular diagnosis in infectious diseases. [S.l: The Author], 2003.

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12

Macario, Alberto J. L. The chaperonopathies: Diseases with defective molecular chaperones : an introduction and guide to diseases in which chaperones play an etiologic-pathogenic role. Dordrecht: Springer, 2013.

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13

Expert Workshop on DNA-Based Molecular Diagnostic Techniques : Research Needs for Standardization and Validation of the Detection of Aquatic Animal Pathogens and Diseases (1999 Bangkok, Thailand). DNA-based molecular diagnostic techniques: Research needs for standardization and validation of the detection of aquatic animal pathogens and diseases. Rome: Food and Agriculture Organization of the United Nations, 2000.

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14

1942-, Garland John M., Quesenberry Peter J i Hilton Douglas J. 1964-, red. Colony-stimulating factors: Molecular and cellular biology. Wyd. 2. New York: M. Dekker, 1997.

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15

Jon, Lorsch, red. Translation initiation: Cell biology, high-throughput methods, and chemical-based approaches. San Diego, Calif: Academic Press, 2007.

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16

Grunert, Marcel, Andreas Perrot i Silke Rickert-Sperling. Complex network interactions: cardiovascular systems biology. Redaktorzy José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso i Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0033.

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A large quantity of molecular information on heart development, function, and disease has been generated over recent decades. However, most recent studies have been dominated by reductionistic approaches, and thus many aspects remain unclear, particularly regarding the primary causes of complex cardiovascular diseases such as congenital heart malformations. With the advent of high-throughput technologies, systems-based approaches have developed rapidly in biology and medicine. In the biology of cardiovascular systems complex data within or across different molecular levels of biological systems or pathways can be integrated and combined to identify the causes underlying cardiac diseases, which might not be possible otherwise. This is in agreement with data suggesting that biological molecules in individual regulatory layers, such as transcripts, proteins, and metabolites, act within networksrather than independently of each other. Thus systems biology provides a promising approach to fully addressing the complexities of congenital heart disease.

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17

Ras Superfamily of GTPases (1993). Taylor & Francis Group, 2017.

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18

Lacal, Juan Carlos, i Frank Patrick McCormick. Ras Superfamily of GTPases (1993). Taylor & Francis Group, 2017.

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19

Lacal, Juan Carlos, i Frank Patrick McCormick. Ras Superfamily of GTPases (1993). Taylor & Francis Group, 2017.

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20

Lacal, Juan Carlos, i Frank Patrick McCormick. Ras Superfamily of GTPases (1993). Taylor & Francis Group, 2017.

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21

Septic Shock: Methods and Protocols (Methods in Molecular Medicine). Humana Press, 2000.

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22

Seneci, Pierfausto. Molecular Targets in Protein Misfolding and Neurodegenerative Disease. Elsevier Science & Technology Books, 2018.

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23

Seneci, Pierfausto. Molecular Targets in Protein Misfolding and Neurodegenerative Disease. Elsevier Science & Technology Books, 2014.

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24

Seneci, Pierfausto. Molecular Targets in Protein Misfolding and Neurodegenerative Disease. Elsevier Science & Technology Books, 2014.

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25

Fink, Anthony, i Vladimir N. Uversky. Protein Misfolding, Aggregation and Conformational Diseases : Part B: Molecular Mechanisms of Conformational Diseases. Springer, 2010.

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26

Bohr, Henrik, i Soren Brunak. Protein Folds: A Distance-Based Approach. CRC, 1995.

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27

Protein Dysfunction in Human Genetic Disease (Human Molecular Genetics). Garland Science, 1997.

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28

Nicolau, Dan V. Nanodevices Based on Protein Molecular Motors: An Engineering Approach (Fundamental Biomedical Technologies). Springer, 2008.

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29

Li, Y. Robert. Cardiovascular Diseases: From Molecular Pharmacology to Evidence-Based Therapeutics. Wiley & Sons, Incorporated, John, 2015.

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30

Li, Y. Robert. Cardiovascular Diseases: From Molecular Pharmacology to Evidence-Based Therapeutics. Wiley & Sons, Incorporated, John, 2015.

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31

Li, Y. Robert. Cardiovascular Diseases: From Molecular Pharmacology to Evidence-Based Therapeutics. Wiley & Sons, Incorporated, John, 2015.

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32

Li, Y. Robert. Cardiovascular Diseases: From Molecular Pharmacology to Evidence-Based Therapeutics. Wiley & Sons, Incorporated, John, 2015.

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Cardiovascular Diseases: From Molecular Pharmacology to Evidence-Based Therapeutics. Wiley, 2015.

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34

Zhang, Jiapu. Molecular Dynamics Analyses of Prion Protein Structures: The Resistance to Prion Diseases Down Under. Springer, 2018.

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35

Zhang, Jiapu. Molecular Dynamics Analyses of Prion Protein Structures: The Resistance to Prion Diseases Down Under. Springer, 2018.

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36

Coolen, A. C. C., A. Annibale i E. S. Roberts. Applications of random graphs. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198709893.003.0011.

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This chapter reviews graph generation techniques in the context of applications. The first case study is power grids, where proposed strategies to prevent blackouts have been tested on tailored random graphs. The second case study is in social networks. Applications of random graphs to social networks are extremely wide ranging – the particular aspect looked at here is modelling the spread of disease on a social network – and how a particular construction based on projecting from a bipartite graph successfully captures some of the clustering observed in real social networks. The third case study is on null models of food webs, discussing the specific constraints relevant to this application, and the topological features which may contribute to the stability of an ecosystem. The final case study is taken from molecular biology, discussing the importance of unbiased graph sampling when considering if motifs are over-represented in a protein–protein interaction network.

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37

Plant virology protocols: From viral sequence to protein function. Wyd. 2. Totowa, NJ: H umana Press, 2008.

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38

Conformational diseases - a compendium: Based on the first international workshop on conformational diseases. Jerusalem: Center for the Study of Emerging Diseases, 2001.

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39

Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Incorporated, John, 2013.

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40

Crichton, Robert, i Roberta Ward. Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Incorporated, John, 2007.

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41

(Editor), Johannes Buchner, i Thomas Kiefhaber (Editor), red. Protein Folding Handbook 5-volume set. Wiley-VCH, 2005.

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42

Tuori, Robert P. Molecular characterization of a protein toxin involved in the Pyrenophora tritici-repentis/wheat interaction. 1998.

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43

Crichton, Robert, i Roberta Ward. Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Incorporated, John, 2006.

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44

Crichton, Robert, i Roberta Ward. Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Incorporated, John, 2013.

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45

Crichton, Robert, i Roberta Ward. Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Incorporated, John, 2013.

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46

Crichton, Robert, i Roberta Ward. Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Limited, John, 2006.

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47

Crichton, Robert, i Roberta Ward. Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Incorporated, John, 2013.

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48

Crichton, Robert, i Roberta Ward. Metal-Based Neurodegeneration: From Molecular Mechanisms to Therapeutic Strategies. Wiley & Sons, Limited, John, 2013.

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49

Chiappelli, Francesco. Osteoimmunopathology: Evidence-Based Perspectives from Molecular Biology to Systems Biology. Springer, 2014.

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50

Seneci, Pierfausto. Chemical Modulators of Protein Misfolding and Neurodegenerative Disease. Elsevier Science & Technology Books, 2015.

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