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Artykuły w czasopismach na temat "Prostate – Cancer – Nutritional aspects"

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Schmid, H. P. "S11 Nutritional aspects of primary prostate cancer prevention". European Journal of Cancer Supplements 8, nr 2 (marzec 2010): 3–4. http://dx.doi.org/10.1016/s1359-6349(10)70747-3.

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Berg, Courtney J., David J. Habibian, Aaron E. Katz, Kaitlin E. Kosinski, Anthony T. Corcoran i Andrew S. Fontes. "Active Holistic Surveillance: The Nutritional Aspect of Delayed Intervention in Prostate Cancer". Journal of Nutrition and Metabolism 2016 (2016): 1–7. http://dx.doi.org/10.1155/2016/2917065.

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Purpose. Active surveillance is an emergent strategy for management of indolent prostate cancer. Our institution’s watchful waiting protocol, Active Holistic Surveillance (AHS), implements close monitoring for disease progression along with various chemopreventive agents and attempts to reduce unnecessary biopsies. Our objective is to report on the treatment rates of men on our AHS protocol as well as determine reasons for progression.Materials/Methods. Low risk and low-intermediate risk patients were enrolled in AHS at Winthrop University Hospital between February 2002 and August 2015. Our IRB-approved study analyzed survival rate, discontinuation rates, and definitive treatments for patients in our AHS cohort.Results. 235 patients met inclusion criteria. Median age and follow-up for the cohort were 66 (44–88) years and 42 (3–166) months, respectively. The overall survival for the cohort was 99.6% and the disease specific survival was 100%. A total of 27 (11.5%) patients discontinued AHS.Conclusion. The incorporation of chemopreventive agents in our AHS protocol has allowed patients to prolong definitive treatment for many years. Longer follow-up and additional studies are necessary to further validate the effectiveness of AHS.
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Omenukor, K. "Cancer Awareness Campaign and Screening". Journal of Global Oncology 4, Supplement 2 (1.10.2018): 142s. http://dx.doi.org/10.1200/jgo.18.73900.

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Background and context: Colorectal cancer is the 3rd leading cause of cancer-related mortalities, which can be prevented by early screening. However, inadequate knowledge regarding the importance of early screening contributes to low cancer screening rates in the population. Aims: A collaborative initiative between David Omenukor Foundation and Fight Colorectal Cancer Organization strives to cancer awareness and screening in the population. Strategy: The David Omenukor Foundation organized a 5-km WALK-A-THON in Mesquite, Texas, on March 10, 2018, as part of the activities to observe the March Colorectal Cancer Awareness Month. During the event, participants received free screening for colorectal, breast, and prostate cancers. Free cholesterol, diabetes, and blood pressure testing were also done because of the impact of these comorbidities on health outcomes. Education experts on cancer were available to teach aspects of healthy diets and exercise. Two cancer patients and a survivor also shared their experiences. Program/Policy process: The program seeks to increase cancer awareness among populations and promote the culture of early and regular screening. Outcomes: Free colorectal and breast cancer screenings were provided to 270 people. About 60 people received free prostate-specific antigen (PSA) test, whereas 135 people received fecal occult blood testing. Similarly, 75 women received mammogram testing. The total number of patients who received colorectal cancer screening increased from 50 on 11th March 2017 to 135 on March 11th, 2018. Impact: The foundation created awareness of all forms of cancer and emphasized the value of early screening as the most effective to avoid the cancer scourge. The participants benefitted from nutritional advice as one strategy for reducing the risk of colorectal cancer. The event indicated that the campaign on early screening for detection was beginning to catch up. Regular interactive events and screenings increase knowledge of cancer and reduce disparities in cancer screening in the community.
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Burden, Sorrel, Jana Sremanakova, Debra Jones i Chris Todd. "Dietary interventions for cancer survivors". Proceedings of the Nutrition Society 78, nr 1 (19.12.2018): 135–45. http://dx.doi.org/10.1017/s0029665118002690.

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This review evaluates evidence on dietary interventions for cancer survivors giving an overview of people's views and preferences for service attributes and provides a narrative review. After cancer, people often want to change their diet and there is a plethora of evidence why dietary optimisation would be beneficial. However, cancer survivors have different preferences about attributes of services including: place, person and communication mode. Randomised control trials have been reviewed to provide a narrative summary of evidence of dietary interventions. Most studies were on survivors of breast cancer, with a few on colorectal, prostate and gynaecological survivors. Telephone interventions were the most frequently reported means of providing advice and dietitians were most likely to communicate advice. Dietary assessment methods used were FFQ, food diaries and 24-h recalls. Dietary interventions were shown to increase intake of fruit and vegetables, dietary fibre, and improve diet quality in some studies but with contradictory findings in others. Telephone advice increased fruit and vegetable intake primarily in women with breast cancer and at some time points in people after colorectal cancer, but findings were inconsistent. Findings from mail interventions were contradictory, although diet quality improved in some studies. Web-based and group sessions had limited benefits. There is some evidence that dietary interventions improve diet quality and some aspects of nutritional intake in cancer survivors. However, due to contradictory findings between studies and cancer sites, short term follow-up and surrogate endpoints it is difficult to decipher the evidence base.
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Uzunlulu, Mehmet, Ozge Telci Caklili i Aytekin Oguz. "Association between Metabolic Syndrome and Cancer". Annals of Nutrition and Metabolism 68, nr 3 (2016): 173–79. http://dx.doi.org/10.1159/000443743.

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Growing data show the association of metabolic syndrome (MetS) or its components with cancer development and cancer-related mortality. It is suggested that in MetS and cancer association, insulin resistance and insulin-like growth factor 1 system play a key role, especially adipokines secreted from visceral adipocytes, free fatty acids and aromatase activity contribute to this process. It is also reported that MetS has a link with colorectal, breast, endometrial, pancreas, primary liver and, although controversial, prostate cancer. Although every component of MetS is known to have an association with cancer development, it is still debated whether the effects of these components are additive or synergistic. On the other hand, in the association between MetS and cancer, the role of antidiabetic and antihypertensive treatments including thiazolidinedione, insulin, angiotensin receptor blockers is also suggested. The primary approach in MetS-cancer relation is to prevent risk factors. Life style changes including weight loss and a healthy diet are known to decrease cancer risk in normal population. It is postulated that an insulin-sensitizing agent, metformin, has cancer-preventing effects on diabetic patients. This review discusses the relationship between MetS and cancer from different aspects and examines this relationship in some of the cancers suggested to be linked with MetS.
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Shivappa, Nitin, James R. Hébert, Faezeh Askari, Mehdi Kardoust Parizi i Bahram Rashidkhani. "Increased Inflammatory Potential of Diet is Associated with Increased Risk of Prostate Cancer in Iranian Men". International Journal for Vitamin and Nutrition Research 86, nr 5-6 (październik 2016): 161–68. http://dx.doi.org/10.1024/0300-9831/a000395.

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Purpose: Various aspects of diet, including specific foods and nutrients, have been implicated to play a role in modulating inflammation and in the etiology of prostate cancer. Studies examining this association have been conducted primarily in Western countries; but none in Middle Eastern Countries. Method: We examined the association between the dietary inflammatory index (DII) and prostate cancer in an ageand BMI-matched case-control study among 40-78 year-old Iranian males. A total of 50 incident cases and 100 controls attending the same hospital as the cases during the same time period were recruited. The DII is a literature-derived population-based dietary index developed to determine the inflammatory potential of individuals' diets and was computed based on dietary intake assessed using a previously validated semi-quantitative food frequency questionnaire (FFQ) that was expanded to assess diet and cancer in the Iranian population. Logistic regression was used to estimate odds ratios, with DII score fit as continuous and as a dichotomous variable. Results: Multivariable-adjusted analyses revealed that men with higher DII score (>0.23) to be at higher risk of prostate cancer [odds ratio (OR) = 3.96; 95% CI =1.29–12.16, p-value = 0.02)] compared to men with lower DII scores (≤0.23). Conclusion: These data suggest a pro-inflammatory diet, as indicated by increasing DII score, may be a risk factor for prostate cancer in Iranian men..
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Rodríguez-García, Carmen, Cristina Sánchez-Quesada i José J. Gaforio. "Dietary Flavonoids as Cancer Chemopreventive Agents: An Updated Review of Human Studies". Antioxidants 8, nr 5 (18.05.2019): 137. http://dx.doi.org/10.3390/antiox8050137.

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Over the past few years, interest in health research has increased, making improved health a global goal for 2030. The purpose of such research is to ensure healthy lives and promote wellbeing across individuals of all ages. It has been shown that nutrition plays a key role in the prevention of some chronic diseases such as obesity, cardiovascular disease, diabetes, and cancer. One of the aspects that characterises a healthy diet is a high intake of vegetables and fruits, as both are flavonoid-rich foods. Flavonoids are one of the main subclasses of dietary polyphenols and possess strong antioxidant activity and anti-carcinogenic properties. Moreover, some population-based studies have described a relationship between cancer risk and dietary flavonoid intake. In this context, the goal of this review was to provide an updated evaluation of the association between the risk of different types of cancers and dietary flavonoid intake. We analysed all relevant epidemiological studies from January 2008 to March 2019 using the PUBMED and Web of Science databases. In summary, this review concludes that dietary flavonoid intake is associated with a reduced risk of different types of cancer, such as gastric, breast, prostate, and colorectal cancers.
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Rinaldi, J. C., S. A. A. Santos, K. T. Colombelli, L. Birch, G. S. Prins, L. A. Justulin i S. L. Felisbino. "Maternal protein malnutrition: effects on prostate development and adult disease". Journal of Developmental Origins of Health and Disease 9, nr 4 (27.03.2018): 361–72. http://dx.doi.org/10.1017/s2040174418000168.

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AbstractWell-controlled intrauterine development is an essential condition for many aspects of normal adult physiology and health. This process is disrupted by poor maternal nutrition status during pregnancy. Indeed, physiological adaptations occur in the fetus to ensure nutrient supply to the most vital organs at the expense of the others, leading to irreversible consequences in tissue formation and differentiation. Evidence indicates that maternal undernutrition in early life promotes changes in key hormones, such as glucocorticoids, growth hormones, insulin-like growth factors, estrogens and androgens, during fetal development. These alterations can directly or indirectly affect hormone release, hormone receptor expression/distribution, cellular function or tissue organization, and impair tissue growth, differentiation and maturation to exert profound long-term effects on the offspring. Within the male reproductive system, maternal protein malnutrition alters development, structure, and function of the gonads, testes and prostate gland. Consequently, these changes impair the reproductive capacity of the male offspring. Further, permanent alterations in the prostate gland occur at the molecular and cellular level and thereby affect the onset of late life diseases such as prostatitis, hyperplasia and even prostate cancer. This review assembles current thoughts on the concepts and mechanisms behind the developmental origins of health and disease as they relate to protein malnutrition, and highlights the effects of maternal protein malnutrition on rat prostate development and homeostasis. Such insights on developmental trajectories of adult-onset prostate disease may help provide a foundation for future studies in this field.
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Gould, Kathy. "Nutritional Aspects of Cancer Rehabilitation". Rehabilitation Oncology 13, nr 3 (1995): 12–21. http://dx.doi.org/10.1097/01893697-199513030-00010.

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Shaw, Clare. "Nutritional aspects of advanced cancer". Palliative Medicine 6, nr 2 (kwiecień 1992): 105–10. http://dx.doi.org/10.1177/026921639200600204.

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Rozprawy doktorskie na temat "Prostate – Cancer – Nutritional aspects"

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Ambrosini, Gina L. "Dietary risk factors for prostate cancer and benign prostatic hyperplasia". University of Western Australia. School of Population Health, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0135.

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[Truncated abstract] This thesis examines the potential role of dietary intake in the development of two common conditions affecting the prostate gland; prostate cancer and benign prostatic hyperplasia (BPH). Diet is of interest as a potential risk factor for prostate cancer because of geographical variations in prostate cancer incidence and increased prostate cancer risks associated with migration from Asian to western countries. Some geographical variation has been suggested for BPH, but this is less certain. However, both prostate cancer and BPH have potential links with diet through their positive associations with sex hormone levels, metabolic syndrome, increased insulin levels and chronic inflammation. In addition, zinc is an essential dietary micronutrient required for semen production in the prostate gland. The original work for this thesis is presented in six manuscripts of which, four have been published in peer-reviewed journals (at the time of thesis completion). BPH investigated in this thesis is defined as surgically-treated BPH. The following hypotheses were investigated. Regarding foods, nutrients and the risk of prostate cancer and BPH: 1. Increasing intakes of fruits, vegetables and zinc are inversely associated with the risk of prostate cancer and BPH 2. Increasing intakes of total fat and calcium are positively associated with the risk of prostate cancer and BPH. 3. Dietary patterns characterised by high meat, processed meat, calcium and fat content are positively associated with the risk of prostate cancer and BPH. 4. Dietary patterns characterised by high fruit and vegetable and low meat content are inversely associated with the risk of prostate cancer and BPH. v Regarding methodological issues related to the study of diet-disease relationships: 5. Dietary patterns (overall diet) elicited from principal components analysis yield stronger diet-disease associations than when studying isolated nutrients. 6. Remotely recalled dietary intake is reliable enough to be used in studies of chronic disease with long latency periods, such as prostate cancer and BPH. Methods: Data from two studies was used to address the hypotheses above. ... Based on the literature reviewed and the original work for this thesis, the most important dietary risk factors for prostate cancer and BPH appear to be those common to western style diets, i.e. diets high in red meat, processed meat, refined grains, dairy products, and low in fruit and vegetables. This type of diet is likely to result in marginal intakes of antioxidants and fibre, excess intakes of fat and possibly, moderate intakes of carcinogens associated with processed meat and meat cooked at high temperatures. These dietary factors have been linked with biomarkers of inflammation, and they support the hypotheses that chronic inflammation is involved in the development of both prostate cancer and BPH. In addition, this work builds on evidence that zinc is an important factor in prostate health. There is scope for more investigation into the reliability of dietary patterns and the use of nutrient patterns as an alternative to focussing on single food components. Further studies on the reliability of remote dietary intake would also be useful. Because of the latency of chronic disease, it can be theorised that remote dietary recall may uncover more robust diet-disease relationships.
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Haseen, Farhana. "Nutritional factors and lifestyle in prostate cancer patients". Thesis, Queen's University Belfast, 2011. https://kclpure.kcl.ac.uk/portal/en/theses/nutritional-factors-and-lifestyle-in-prostate-cancer-patients(3e845f58-4e1b-4f5c-a5cc-605d084b9868).html.

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Prostate cancer is the most common male cancer in the UK. A healthy diet and lifestyle may have the potential to impact upon cancer progression, treatment related side effects, and comorbidities as well as on quality of life in prostate cancer patients. Nutrition and lifestyle behaviours are therefore important aspects to consider in the treatment of prostate cancer patients. The broad aims of this thesis were to explore the importance of health behaviours in prostate cancer patients and to identify and test a suitable intervention specifically for those who are treated with androgen deprivation therapy (ADT). To achieve these aims this thesis has documented the findings of the following studies investigating nutritional and life-style behaviours in prostate cancer patients. 1) Using the cross-sectional population-based data from California Health Interview Survey (CHIS), diet and health behaviours in men with prostate cancer were compared to men with cancer other than prostate and to non-cancer controls. No differences were noted between prostate cancer patients and controls with respect to their dietary intake and physical activity levels. 2) A systematic review was conducted to determine the effects of ADT on body composition in prostate cancer patients. Significant increases in fat and declines in lean mass were observed in prostate cancer patients treated with ADT. 3) A systematic review of the literature related to lycopene intake and prostate cancer progression suggested that evidence available to date was insufficient to draw any firm conclusions regarding the benefits of lycopene supplementation in prostate cancer patients. 4) Preliminary analysis of a RCT of a 6-month dietary and physical activity intervention in prostate cancer patients treated with ADT showed that the intervention group had significant improvements in their body composition compared with controls. Physical activity, functional capacity and dietary quality also improved in the intervention group compared with controls. Recruitment to this trial is ongoing.
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Price, Alison Jane. "Nutritional and hormonal biomarkers in prostate cancer epidemiology". Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:8d96d746-7c87-4133-b873-e9a8426da953.

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Evidence from international comparisons and migrant studies suggest that environmental factors, such as a Western diet, may be important in prostate cancer development, possibly through effects on hormone and growth factor secretion and metabolism. However, despite considerable research, convincing associations between diet and risk for prostate cancer have not been established. Random and systematic measurement error in dietary assessment using traditional survey methods may contribute to inconsistent findings, particularly as they may not capture adequately specific nutritional constituents of the diet that may be associated with risk, such as fatty acids or vitamins. Validated biomarkers of nutritional factors and hormonal activity, as used in this thesis, provide more precise, objective and integrated measures of exposure, with the capacity to clarify potential mechanisms in the causal pathway of prostate cancer development. Nutritional and hormonal biomarkers investigated for their potential role in the development of prostate cancer include: folate and vitamin B12, which are essential for DNA methylation, repair and synthesis; phytanic acid, obtained predominantly from ruminant fat intake and associated with an enzyme (α-Methylacyl-Coenzyme A Racemase (AMACR)) that is consistently over-expressed in prostate cancer tissue; and insulin-like growth factor (IGF-I), a growth factor influenced by diet and involved in the regulation of cell proliferation, differentiation, and apoptosis. All work presented in this thesis is from the European Prospective Investigation into Cancer and Nutrition (EPIC) study of 500,000 European men and women, using prospectively collected diet and lifestyle data and biological samples. The large number of prostate cancer cases diagnosed during long-term follow-up of EPIC participants enabled investigation of heterogeneity in risk for prostate cancer by time from recruitment to diagnosis (of particular importance for a disease with a long pre-clinical phase) and cancer characteristics such as disease grade and stage. Plasma phytanic acid concentration was highly correlated with dietary intake of fat from dairy products (r = 0.46) and beef (r = 0.30); capturing differences between countries in consumption of fat from these foods. Although phytanic acid is a useful biomarker of ruminant fat consumption, there was little evidence to support the hypothesis that the association between dairy products and prostate cancer risk (as suggested by previous work in EPIC and other studies) is mediated by phytanic acid (OR for doubling in concentration 1.05; 95%CI 0.91 – 1.21; P trend = 0.53). There was strong evidence for an association between higher circulating IGF-I concentration and risk for prostate cancer (OR for highest versus lowest fourth 1.69; 95% CI: 1.35, 2.13; P trend = 0.0002). Furthermore, the positive association observed among men diagnosed with advanced stage disease and among men diagnosed more than seven years after blood collection, supports the hypothesis that high IGF-I concentration is associated with clinically significant prostate cancer many years before diagnosis. There was no evidence of an association between prostate cancer risk and dietary folate or vitamin B12 intake, or between circulating levels of folate (OR for doubling in concentration 1.05; 95%CI 0.95 – 1.15; P trend = 0.33) or vitamin B12 (1.05; 95%CI 0.92 – 1.21; P trend = 0.47) and only limited evidence for an increased risk associated with elevated vitamin B12 in a meta-analysis of six prospective studies, that included the present study. All of these analyses were based on a blood sample taken at one point in time, with the assumption that this reflects the ‘true’ underlying concentration over the long-term. The poor to modest reliability estimates (intra-class correlation coefficients ranging from 0.18 to 0.48) for circulating concentrations of folate, IGF-I, phytanic acid and vitamin B12 taken in samples approximately six years apart in a sub-sample of participants from EPIC Oxford, show that estimates of usual concentrations based on a single blood measurement weaken the ability to detect associations with disease risk. Where small effect sizes are anticipated, this may bias associations toward the null. In conclusion, there is convincing evidence that IGF-I is an important and potentially modifiable risk factor for prostate cancer many years before diagnosis. However, there is little evidence for an association between biomarkers of folate, vitamin B12 and phytanic acid concentrations and risk for prostate cancer. Future studies should, where possible, incorporate multiple blood samples taken several years apart to better characterise long term relationships between biomarkers of nutritional and hormonal exposure and disease risk and pool individual participant data from multiple prospective studies to strengthen the power to detect modest associations.
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Ahouandjinou, Theodora Vignon. "Facteurs nutritionnels associés à la présence de lésions précancéreuses de la prostate (PIN) chez des hommes ayant une hypertrophie bénigne de la protestate". Master's thesis, Université Laval, 2008. http://hdl.handle.net/20.500.11794/19946.

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La néoplasie intra-épithéliale prostatique, un des précurseurs possibles du cancer de la prostate, pourrait donner des renseignements sur les causes de ce dernier dans la mesure où ils cohabitent souvent. Dans une étude transversale, réalisée chez 510 hommes traités chirurgicalement pour une hypertrophie bénigne de la prostate, nous avons testé l'hypothèse que les facteurs de risque souvent associés au cancer de la prostate pouvaient être aussi associés à la présence de la néoplasie intra-épithéliale prostatique. La consommation alimentaire au cours de l'année précédant la chirurgie a été documentée par un questionnaire alimentaire détaillé administré par une diététiste. Le tissu prostatique prélevé à la chirurgie a été examiné par un seul pathologiste. Seuls les patients ayant une hypertrophie bénigne et aucune évidence de cancer ont été retenus pour l'étude. La présence de lésions précancéreuses de la prostate (PIN) a été observée chez 81 participants. La majorité des analyses a porté sur les relations entre les facteurs nutritionnels suspectés de jouer un rôle dans le cancer de la prostate et la présence de PIN. Les rapports de cotes ajustés (RC) et leurs intervalles de confiance (IC à 95%) ont été estimés à l'aide de la régression logistique pour les différentes variables des apports alimentaires. Les résultats sont pour la majorité non statistiquement significatifs, seule une importante consommation de carottes avec un RC, comparant le 3ème au premier tercile, de 1,84 (IC à 95% =1,00-3,40), et d'alpha-carotène avec un RC = 1,90 (IC à 95% =1,04-3,45) sont statistiquement significatifs. En regard du nombre d'associations évaluées ces résultats ne suggèrent aucune association entre les facteurs nutritionnels associés au cancer de la prostate et la présence de PIN.
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Miller, Elizabeth C. "Studies of nutritional support for prostate cancer prevention and therapy". Connect to resource, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1124140836.

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Trotter, James Marshall. "Nutrition and cancer : studies on nutritional abnormalities, nutritional support and protein metabolism in malnourished cancer patients /". Title page, contents and abstract only, 1987. http://web4.library.adelaide.edu.au/theses/09MD/09mdt858.pdf.

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Allen, Naomi E. "Nutritional and genetic determinants of hormone levels in relation to prostate cancer risk". Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365882.

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徐慧恩 i Wai-yan Tsui. "Determination of PTEN mutations in prostate cancer in Chinese". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31969951.

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Kwan, Pak-shing, i 關百誠. "Roles of Daxx in mitosis and prostate carcinogenesis". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43085337.

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Zucchero, Renee A. "Marital adjustment of older adult couples with breast cancer, prostate cancer, and couples without cancer". Virtual Press, 1998. http://liblink.bsu.edu/uhtbin/catkey/1117099.

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The purpose of this study was to explore the marital adjustment of older adult couples with breast cancer, prostate cancer, and couples who have experienced neither. Participants were 64 couples in which at least one of the spouses was over 55 years of age, including 19 breast cancer couples, 20 prostate cancer couples, 25 couples who had experienced neither of these cancers. Most participants were young-old, Protestant, Caucasians from a high socioeconomic class. The breast cancer and prostate cancer participants had completed treatment an average of 39.5 months prior to participation. The methodology was a mail survey. Participants completed a demographic questionnaire, the Marital Satisfaction Questionnaire for Older Adults (MSQFOP) (Haynes et al., 1992), Primary Communication Inventory (PCI) (Navran, 1967), Miller Social Intimacy Scale (MSIS) (Miller & Lefcourt, 1982), and the Index of Sexual Satisfaction (ISS) (Hudson et al., 1981).There were no differences in the amount of discordance between the couples groups' level of marital satisfaction, communication, intimacy, and sexual satisfaction. In addition, there were no differences in the level of marital satisfaction, communication, intimacy, and sexual satisfaction between the participant groups. There was a significantly greater correlation between the prostate cancer couples' scores on the ISS than the correlation between the breast cancer couples' scores and the scores of the couples who had not experienced breast cancer or prostate cancer.The level of marital satisfaction, communication, intimacy, and sexual satisfaction reported was similar to that of the normative samples. There was no difference between the marital adjustment of the cancer couples and older couples who had experienced neither type of cancer. These results are good news for breast and prostate cancer survivors, and professionals. Older adults may be better able to incorporate the experience of cancer into their lives or are better prepared for chronic illness through anticipatory socialization. The high degree of agreement between the prostate cancer spouses on the ISS may be related to the sexual dysfunction that frequently accompanies treatment for this cancer. Future research should be qualitative and longitudinal and continue to explore the psychosocial implications of prostate cancer.
Center for Gerontology
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Książki na temat "Prostate – Cancer – Nutritional aspects"

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Sgarlat, Steck Sara, i Myers Rose Sgarlat, red. Eating your way to better health: The prostate forum nutrition guide. Charlottesville: Rivanna Health Publications, Inc., 1999.

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Brennan, Rosemary Anne Theresa. Nutritional aspects of cancer. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1986.

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S, Ross Jeffrey. Molecular oncology of prostate cancer. Sudbury, MA: Jones and Bartlett Publishers, 2007.

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Schmidt, Gerhard. Cancer and nutrition. Spring Valley, N.Y: Anthroposophic Press, 1986.

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Strogat, Lew. Cancer: Cause-prevention-treatment. New York: Literary Frontier Publishers, 1991.

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Passwater, Richard A. Cancer prevention and nutritional therapies. New Canaan, Conn: Keats Pub., 1993.

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Somer, Elizabeth. Cancer and nutrition. Danbury, CT: Grolier Educational, 1988.

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Eat to beat cancer. Los Angeles, CA: Renaissance Books, 1998.

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Ireland. Food Safety Advisory Committee. Diet and cancer (updated version). Dublin: Stationery Office, 1994.

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Coleman, Vernon. Power over cancer. Barnstaple: European Medical Journal, 1996.

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Części książek na temat "Prostate – Cancer – Nutritional aspects"

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Schmid, Hans-Peter, Claus Fischer, Daniel S. Engeler, Marcelo L. Bendhack i Bernd J. Schmitz-Dräger. "Nutritional Aspects of Primary Prostate Cancer Prevention". W Clinical Cancer Prevention, 101–7. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-10858-7_8.

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Venugopal, Smrruthi V., Michael R. Freeman, Stephen F. Freedland i Shafiq A. Khan. "Cholesterol and Prostate Cancer". W Nutritional Oncology, 209–28. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9780429317385-10.

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Hofmann, R. "Anatomy: Surgical Aspects". W Prostate Cancer, 127–35. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-56321-8_12.

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Bonkhoff, H. "Morphogenetic Aspects of Prostate Cancer". W Prostate Cancer, 3–12. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-56321-8_1.

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Parr, Kathryn. "Nutritional Management In Prostate Cancer". W Nutrition and Cancer, 363–78. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118788707.ch18.

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Griffiths, K., M. E. Harper, C. L. Eaton, A. Turkes i W. B. Peeling. "Endocrine Aspects of Prostate Cancer". W Prostate Cancer 2000, 21–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79178-9_5.

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Blackledge, George R. P. "Clinical Aspects of Casodex". W Antiandrogens in Prostate Cancer, 89–94. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-45745-6_8.

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Ernstoff, Marc S. "Clinical Aspects of Prostate Cancer". W Atlas of the Prostate, 95–113. London: Current Medicine Group, 2003. http://dx.doi.org/10.1007/978-1-4615-6505-5_8.

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Pinto, Peter. "Surgical Aspects of Prostate Cancer". W Cancer Consult: Expertise for Clinical Practice, 747–53. Oxford, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118589199.ch115.

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Galustian, Christine, Oussama Elhage, Richard Smith i Prokar Dasgupta. "Immunologic Aspects of Prostate Cancer". W Prostate Cancer: A Comprehensive Perspective, 65–72. London: Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-2864-9_5.

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Streszczenia konferencji na temat "Prostate – Cancer – Nutritional aspects"

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Gabriele, D., G. Cattari, C. Fiorito, M. T. Carchedi i E. Garibaldi. "Data collection for models validation: Application to prostate cancer — Clinical aspects". W 2014 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2014. http://dx.doi.org/10.1109/bhi.2014.6864290.

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Pasquier, D., N. Betrouni, M. Vermandel, T. Lacornerie, E. Lartigau i J. Rousseau. "MRI alone simulation for conformal radiation therapy of prostate cancer: technical aspects". W Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.260341.

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Nahlawi, Layan, Farhad Imani, Mena Gaed, Jose A. Gomez, Madeleine Moussa, Eli Gibson, Aaron Fenster i in. "Using Hidden Markov Models to capture temporal aspects of ultrasound data in prostate cancer". W 2015 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2015. http://dx.doi.org/10.1109/bibm.2015.7359725.

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