Rozprawy doktorskie na temat „Proliferation”
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Cheng, Wai. "The relationship between peroxisome proliferator-activated receptors (PPARs) and cell proliferation /". View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36433937.
Pełny tekst źródłaCheng, Wai, i 鄭蔚. "The relationship between peroxisome proliferator-activated receptors (PPARs) and cell proliferation". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B45010614.
Pełny tekst źródłaJemec, Barbara. "Proliferation and action of an anti-proliferative agent in Dupuytren's fibroblast cultures". Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343601.
Pełny tekst źródłaVeerapen, Pierce Elaine. "Proliferation in astrocytomas". Thesis, University of Greenwich, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245348.
Pełny tekst źródłaGreen, Daniel M. "Monitoring technology proliferation: an open source methodology for generating proliferation intelligence". Thesis, Monterey, California. Naval Postgraduate School, 1993. http://hdl.handle.net/10945/39686.
Pełny tekst źródłaThis thesis develops a methodology to monitor technology proliferation. It is designed to provide proliferation intelligence on specific threat technologies and can be used to augment export controls or enhance counter proliferation initiatives. A high-te
Miller, Nicholas L. (Nicholas LeSuer). "Hegemony and nuclear proliferation". Thesis, Massachusetts Institute of Technology, 2014. http://hdl.handle.net/1721.1/95553.
Pełny tekst źródłaCataloged from PDF version of thesis.
Includes bibliographical references (pages 277-292).
Contrary to longstanding of predictions of nuclear tipping points, the number of states interested in nuclear weapons has sharply declined in recent decades. In contrast to existing explanations, this dissertation argues that the decline is largely attributable to US nonproliferation policies, in particular the threat of sanctions that was instituted in the late 1970s. By credibly threatening to cut off economic and military support to countries pursuing nuclear weapons, I argue that this threat of sanctions deters states within the US sphere of influence from proliferating, reducing the overall rate of proliferation and also explaining why recent nuclear aspirants have exclusively been "rogue" states outside the US sphere of influence. Because states that depend on the United States have been deterred from proliferating in recent decades, the observed success rate of sanctions should be low, since they will generally be targeted at states that do not rely on US resources. This dissertation also offers a theory of the sources of US nonproliferation policy, arguing that fears of nuclear domino effects are necessary to explain (1) why US policy strengthened so dramatically in the wake of Chinese and Indian nuclear tests in the 1960s and 1970s, and (2) why the US abandoned a selective nonproliferation policy and decided to enforce nonproliferation across the board. To test these two arguments, this dissertation employs a mix of quantitative and qualitative methods. First, I draw on archival documents to show that fears of nuclear domino effects motivated US nonproliferation policy advances in the 1960s and 1970s, and that this motivation was prominent in individual cases of nonproliferation. Second, I show quantitatively that states dependent on the United States have been less likely to pursue nuclear weapons since sanctions policies were instituted in the late 1970s, that observed cases of sanctions have been largely ineffective, and that the deterrent effect of sanctions largely accounts for the temporal decline in proliferation. Case studies of US policy toward Pakistan and Taiwan demonstrate that a credible threat of sanctions can arrest ongoing nuclear programs when the proliferator is dependent on the United States and underestimated the likelihood of sanctions.
by Nicholas L. Miller.
Ph. D.
Li, Xiaoling. "Peroxisome proliferation and division". Available to US Hopkins community, 2002. http://wwwlib.umi.com/dissertations/dlnow/3080712.
Pełny tekst źródłaRenda, Guido. "Resisting Nuclear Proliferation Through Design : a Systems Approach to Nuclear Proliferation Resistance Assessment". Thesis, University of Bristol, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520167.
Pełny tekst źródłaDu, Zhenjian Cheung H. Tak. "Febrile condition and lymphocyte proliferation". Normal, Ill. Illinois State University, 1991. http://wwwlib.umi.com/cr/ilstu/fullcit?p9203029.
Pełny tekst źródłaTitle from title page screen, viewed December 8, 2005. Dissertation Committee: H. Tak Cheung (chair), Herman E. Brockman, Lynne A. Lucher, Anthony J. Otsuka, Alan J. Katz. Includes bibliographical references (leaves 99-110) and abstract. Also available in print.
Ashagbley, Anthony J. "Ethanolamine requirement and cell proliferation". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq23203.pdf.
Pełny tekst źródłaFalk, Anna. "Stem cells : proliferation, differentiation, migration /". Stockholm, 2005. http://diss.kib.ki.se/2006/91-7140-497-X/.
Pełny tekst źródłaFrankenstein, William. "Computational Models of Nuclear Proliferation". Research Showcase @ CMU, 2016. http://repository.cmu.edu/dissertations/782.
Pełny tekst źródłaHooper, Nigel I. "Methylglyoxal, glyoxalases and cell proliferation". Thesis, Aston University, 1987. http://publications.aston.ac.uk/12548/.
Pełny tekst źródłaHutley, Louise Joyce. "Human preadipocyte proliferation and differentiation". Thesis, Queensland University of Technology, 2002. https://eprints.qut.edu.au/37117/6/37117_Digitised_Thesis.pdf.
Pełny tekst źródłaReed, Brian K. "Models for proliferation interdiction response analysis". Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 1994. http://handle.dtic.mil/100.2/ADA288645.
Pełny tekst źródłaThesis advisor(s): David Morton, Peter Lavoy. "September 1994." Includes bibliographical references. Also available online.
Nichols, Patti J. "Understanding China's nuclear non-proliferation policy". Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 1999. http://handle.dtic.mil/100.2/ADA366857.
Pełny tekst źródła"June 1999". Thesis advisor(s): Rodney Kennedy-Minott, Denny Roy. Includes bibliographical references (p. 103-108). Also available online.
McDermott, Paul B. "Proliferation mechanisms in Methanothermobacter thermautotrophicus DeltaH". Thesis, University of York, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.485452.
Pełny tekst źródłaPetersen, Cecilia. "Paracrine regulation of Sertoli cell proliferation /". Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-443-7/.
Pełny tekst źródłaGan, Lisha. "Corneal cellular proliferation and wound healing /". Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4505-5/.
Pełny tekst źródłaYamak, Fatimah Abir. "GATA4 Partners in Cardiac Cell Proliferation". Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/23802.
Pełny tekst źródłaÅkesson, Lovisa. "Identifikation av proteinmarkörer för cellulär proliferation". Thesis, KTH, Skolan för bioteknologi (BIO), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-170359.
Pełny tekst źródłaCheung, Man-keung, i 張文強. "FBI-1 and choriocarcinoma cell proliferation". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193565.
Pełny tekst źródłapublished_or_final_version
Pathology
Master
Master of Medical Sciences
Bell, Alexander. "The molecular basis of peroxisome proliferation". Thesis, University of Nottingham, 1998. http://eprints.nottingham.ac.uk/10395/.
Pełny tekst źródłaAdenrele, Festus Oladipo Ayinla. "Proliferation of nuclear weapons in Africa". Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337530.
Pełny tekst źródłaBraut-Hegghammer, Malfrid. "Nuclear entrepreneurs : drivers of nuclear proliferation". Thesis, London School of Economics and Political Science (University of London), 2009. http://etheses.lse.ac.uk/2760/.
Pełny tekst źródłaDunphy, Elizabeth Louise. "TAF1 HAT activity in cell proliferation /". Thesis, Connect to this title online; UW restricted, 2003. http://hdl.handle.net/1773/6250.
Pełny tekst źródłaFiorentini, Enrico. "Avoiding non-proliferation atrophy: the effectiveness of multilateral cooperation, regime dynamics and the case of nuclear non-proliferation". Doctoral thesis, Università degli studi di Trento, 2018. https://hdl.handle.net/11572/367756.
Pełny tekst źródłaFiorentini, Enrico. "Avoiding non-proliferation atrophy: the effectiveness of multilateral cooperation, regime dynamics and the case of nuclear non-proliferation". Doctoral thesis, University of Trento, 2018. http://eprints-phd.biblio.unitn.it/3085/1/PhD_Thesis_EnricoFiorentini.pdf.
Pełny tekst źródłaLafenêtre, Pauline. "Regulation der Proliferation im Hippocampus adulter Mäuse". [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=979107407.
Pełny tekst źródłaBarretta, Michael A. "Nuclear proliferation and the stability-instability paradox". Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 1995. http://handle.dtic.mil/100.2/ADA304388.
Pełny tekst źródłaTalay, Brian J. "Preventing ballistic missile proliferation : lessons from Iraq /". Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 1996. http://handle.dtic.mil/100.2/ADA326672.
Pełny tekst źródłaThesis advisor(s): Peter R. Lavoy. "December 1996." Includes bibliographical references (p. 97-100), Also available online.
Roshan, Amit. "Stochasticity and order : studies of keratinocyte proliferation". Thesis, University of Cambridge, 2014. https://www.repository.cam.ac.uk/handle/1810/252966.
Pełny tekst źródłaCurrie, Ian Stewart. "Proliferation and maturation in developing human liver". Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/27856.
Pełny tekst źródłaWalker, John Ronald. "British attitudes to nuclear proliferation, 1952-1982". Thesis, University of Edinburgh, 1987. http://hdl.handle.net/1842/27024.
Pełny tekst źródłaSutton, Selina Kaye. "How does mitochondrial heteroplasmy affect cell proliferation?" Thesis, University of Canterbury. Biological Sciences, 2006. http://hdl.handle.net/10092/1306.
Pełny tekst źródłaDorward, Ann M. "Hydrogen peroxide production and autocrine proliferation control". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ66202.pdf.
Pełny tekst źródłaWang, Yanling. "cAMP-Regulated Cell Proliferation in Brown Preadipocytes". Doctoral thesis, Stockholms universitet, Institutionen för molekylär biovetenskap, Wenner-Grens institut, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-88393.
Pełny tekst źródłaAt the time of the doctoral defense, the following papers were unpublished and had a status as follows: Paper 2: Submitted. Paper 3: Manuscript. Paper 4: Manuscript.
Sohi, Jasloveleen. "Investigation of factors regulating parathyroid cell proliferation". Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=55530.
Pełny tekst źródłaIn summary, my studies have shown that parathyroid cells respond to selected growth factors. This proliferative response involves increased expression of c-myc, c-fos, c-jun and PTHrP. 1,25-(OH)$ rm sb2D sb3$ inhibits the expression or c-myc, and cell proliferation is inhited. The differentiated parathyroid cell expresses high levels of CgA and PTH. However, during proliferation these high levels are not sustained.
Whitworth, Emma Jane. "Adrenocortical proliferation and signal transduction 'in vitro'". Thesis, Queen Mary, University of London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.415058.
Pełny tekst źródłaWoodman, Anthony C. "Studies on the control of hepatocyte proliferation". Thesis, Imperial College London, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364568.
Pełny tekst źródłaKranc, Kamil. "The role of Cited2 in cellular proliferation". Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398233.
Pełny tekst źródłaKuffer, Christian. "Proliferation and arrest of human tetraploid cells". Diss., Ludwig-Maximilians-Universität München, 2015. http://nbn-resolving.de/urn:nbn:de:bvb:19-182558.
Pełny tekst źródłaErroneously arising tetraploid mammalian cells are chromosomally unstable and may facilitate cell transformation. An increasing body of evidence suggests that the propagation of mammalian tetraploid cells is limited by a p53-dependent arrest, however, the triggers of this arrest have thus far not been identified. To elucidate the timing and causes of this arrest, time-lapse live cell imaging was performed to track the fate of individual cells immediately after tetraploidization. Newly formed tetraploid cells can progress through one cell cycle, but the majority of cells arrest or die in the subsequent G1 stage, with the fate of these tetraploid cells determined by the preceding mitosis. Daughter cells arising from defective mitosis accumulated p53 in the nucleus, which led to irreversible cell cycle arrest or death. Furthermore this p53 accumulation coincides and correlates with an increase of the oxidative DNA damage marker 8-OHdG, suggesting an increase in reactive oxygen species (ROS), but does not coincide with the phosphorylation of H2AX (γ-H2AX), a marker for canonical DNA damage. Using RNA interference and chemical genetics, several p53 activating kinases were tested for their contribution to the cell cycle arrest of tetraploid cells. Of the tested kinases, only ATM was shown to play a role in the activation of p53 after defects in mitosis. ATM kinase is a DNA damage-responsive kinase, however, it has been shown that increased ROS levels activate ATM in a non-canonical way. To gain further insights into arrest of tetraploid cells, an unbiased genome-wide esiRNA screen was performed to analyze cell proliferation after induced tetraploidization. Using FUCCI cell cycle probes, combined with DNA content cell cycle profiling, allowed an image-based assay to examine tetraploid and diploid cells side-by-side. This novel approach enabled us to screen for genes that specifically restricts or enhances cell proliferation after tetraploidization, if inhibited by esiRNA mediated knockdown. From the primary screen we identified 1159 genes that decreased and 431 genes that increased the cell proliferation after tetraploidization, if knocked down by esiRNA. From the 431 genes that increased proliferation upon knockdown, 374 were selected and subjected to a re-screen. Of these 374 genes, we were able to confirm the results for 158 of the genes. A bioinformatics analysis of the 158 genes for which the phenotype were confirmed by the re-screen revealed a significant enrichment of genes involved in DNA replication, the canonical Wnt signaling pathway and in pathways linked to cancer. Among the latter, CCDC6 is particularly interesting, because its gene product is a target of the ATM kinase and an upstream regulator of the tumor suppressor 14-3-3σ. Moreover, by comparing the results of the primary screen with the data of the “Project Archilles”, which measured the proliferation in genome wide pooled-shRNA screens for 108 cancer cell lines, 18 genes were identified that are essential for the proliferation of cells after tetraploidization, as well as for the proliferation of cancer cell lines that derive from cancer types with a high incidence for chromosomal instability (CIN). Taken together, the presented data builds an excellent resource not only for elucidating how the arrest after tetraploidization is mediated, but also to identify novel potential therapeutic targets against tumors with CIN, which are frequently resistant to many of today’s anti-cancer therapies.
Wilshusen, John D. "Do U.S. security commitments discourage nuclear proliferation?" Monterey, California. Naval Postgraduate School, 1997. http://hdl.handle.net/10945/8275.
Pełny tekst źródłaU.S. policy makers claim that nuclear weapons are needed to guarantee security commitments and discourage the international spread of nuclear weapons. This thesis evaluates the link between security guarantees and efforts to prevent nuclear proliferation. It draws three conclusions based on case studies of the use of conventional security commitments and nuclear security guarantees to prevent nuclear weapons development in South Korea and Taiwan. First, nuclear security guarantees alone are not sufficient to prevent proliferation. Second, strong conventional commitments made credible by visible presence of forces are sufficient to prevent nuclear proliferation when the direct security threat is conventional. Third, when the security threat being faced includes nuclear weapons, nuclear proliferation prevention requires both a nuclear security guarantee and a physically evident conventional military guarantee. Two implications for security policy follow from these findings. First, nuclear weapons are necessary in the modem security environment. Second, nuclear security guarantees are not credible without the stationing of conventional forces
Dixon, Mark. "Signal transduction mechanisms involved in hepatocyte proliferation". Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245710.
Pełny tekst źródłaLivingstone, D. "Modelling cell proliferation in a structured tissue". Thesis, University of Reading, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.379764.
Pełny tekst źródłaWebster, Lynne. "Hypoxia and proliferation in murine tumour models". Thesis, University College London (University of London), 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336415.
Pełny tekst źródłaGoodlad, J. R. "Germinal centre cell proliferation in murine spleens". Thesis, University of Aberdeen, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241447.
Pełny tekst źródłaWoodrow, Sarah Louise. "Mechanisms of human papillomavirus-induced benign proliferation". Thesis, University of London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405572.
Pełny tekst źródłaNoordin, Liza. "Molecular mechanisms of cell proliferation in endometriosis". Thesis, University of Strathclyde, 2011. http://oleg.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=16808.
Pełny tekst źródłaMitchell, Angela M. "Hepatic peroxisome proliferation : mechanisms and species differences". Thesis, University of Surrey, 1985. http://epubs.surrey.ac.uk/847808/.
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