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Artykuły w czasopismach na temat "Prise alimentaire – Régulation"
Luquet, Serge. "Régulation de la prise alimentaire". Nutrition Clinique et Métabolisme 22, nr 2 (czerwiec 2008): 52–58. http://dx.doi.org/10.1016/j.nupar.2008.04.011.
Pełny tekst źródłaDisse, E. "Régulation neuro-hormonale de la prise alimentaire". Annales d'Endocrinologie 78, nr 4 (wrzesień 2017): 204. http://dx.doi.org/10.1016/j.ando.2017.07.750.
Pełny tekst źródłaMithieux, G. "Régulation de la prise alimentaire par les protéines". Médecine des Maladies Métaboliques 7, nr 1 (luty 2013): 35–40. http://dx.doi.org/10.1016/s1957-2557(13)70490-5.
Pełny tekst źródłaPénicaud, Luc, Alexandre Benani, Xavier Fioramonti, Thierry Alquier, Élise Guillod, Anne Lorsignol i Corinne Leloup. "Détecteurs de glucose et régulation de la prise alimentaire". Cahiers de Nutrition et de Diététique 42, nr 3 (maj 2007): 134–38. http://dx.doi.org/10.1016/s0007-9960(07)88755-4.
Pełny tekst źródłaLuquet, Serge. "Du nouveau dans la régulation de la prise alimentaire ?" médecine/sciences 24, nr 8-9 (sierpień 2008): 680–82. http://dx.doi.org/10.1051/medsci/20082489680.
Pełny tekst źródłaBerthoz, S. "Concept d’alimentation émotionnelle : mesure et données expérimentales". European Psychiatry 30, S2 (listopad 2015): S29—S30. http://dx.doi.org/10.1016/j.eurpsy.2015.09.088.
Pełny tekst źródłaAndreelli, F., i H. Mosbah. "IRM fonctionnelle cérébrale et régulation de la prise alimentaire chez l’homme". Médecine des Maladies Métaboliques 8, nr 1 (luty 2014): 21–27. http://dx.doi.org/10.1016/s1957-2557(14)70678-9.
Pełny tekst źródłaDupont, Mariève, Vicky Drapeau, Caroline Senécal, Annette Gallant, Émilie Pérusse-Lachance i Angelo Tremblay. "Régulation de la prise alimentaire consécutive à un travail mental exigeant." Canadian Journal of Behavioural Science / Revue canadienne des sciences du comportement 49, nr 1 (styczeń 2017): 18–31. http://dx.doi.org/10.1037/cbs0000061.
Pełny tekst źródłaBOUVAREL, I., S. TESSERAUD i C. LETERRIER. "L’ingestion chez le poulet de chair : n’oublions pas les régulations à court terme". INRAE Productions Animales 23, nr 5 (19.12.2010): 391–404. http://dx.doi.org/10.20870/productions-animales.2010.23.5.3317.
Pełny tekst źródłaBallon, N., S. Berthoz, P. Brunault i N. Rigal. "L’alimentation est-elle soluble dans la psychologie et la psychiatrie ? Cas de l’alimentation émotionnelle et de l’addiction à l’alimentation". European Psychiatry 30, S2 (listopad 2015): S29. http://dx.doi.org/10.1016/j.eurpsy.2015.09.087.
Pełny tekst źródłaRozprawy doktorskie na temat "Prise alimentaire – Régulation"
Félix, Bernadette. "Régulation nerveuse de la prise alimentaire chez l'oie de race landaise". Paris 6, 1990. http://www.theses.fr/1990PA066501.
Pełny tekst źródłaSentissi, El Idrissi Othman. "Régulation neurobiologique centrale et périphérique de la prise alimentaire chez les schizophrènes traités par antipsychotiques : aspects cliniques et biologiques". Paris 6, 2009. http://www.theses.fr/2009PA066107.
Pełny tekst źródłaVinera, Jennifer. "Rôle des récepteurs périphériques aux endocannabinoïdes dans la régulation de la prise alimentaire". Thesis, Lyon, 2018. http://www.theses.fr/2018LYSE1339.
Pełny tekst źródłaRoux, Julien. "Brain-derived neurotrophic factor (BDNF) et système endocannabinoïde au niveau du complexe vagual dorsal (CVD) : implication dans le contrôle dela prise alimentaire". Aix-Marseille 3, 2009. http://www.theses.fr/2009AIX30065.
Pełny tekst źródłaBDNF/TrkB signaling and the endocannabinoid system play respectively an anorexigenic and orexigenic role in the central control of food intake. The ventromedical hypothalamus has been identified as a site wher BDNF plays its anorexigenic role, as a downstream effector of melanocortinergic signaling. .
Schoukroun, Florian. "Rôle du noyau tegmental rostromédian dans la prise alimentaire". Electronic Thesis or Diss., Strasbourg, 2023. http://www.theses.fr/2023STRAJ140.
Pełny tekst źródłaThe rostromedial tegmental nucleus (RMTg) is a brain structure projecting to the ventral tegmental area (VTA) and the lateral hypothalamus (LH), two key regulatory centers for feeding behavior. The function of the RMTg remains mostly unknown, and it is plausible that this region plays a role in food intake dysregulation as observed in obesity and binge-eating disorders. We firstly studied the role of the RMTg-VTA and RMTg-LH pathways in food intake regulation through a projection-specific lesional approach followed by a 6-week exposure to an obesogenic diet. Then, we analyzed and compared the adaptations of the endocannabinoid system (ECS), an endogenous system regulating food intake, within reward-related brain regions in obesity and binge-eating disorder models. Our results indicate that the RMTg-VTA and RMTg-LH pathways respectively modulate high-fat and standard food consumption. The molecular analysis revealed distinct dysregulation of ECS expression in obesity and binge-eating disorder, particularly within the RMTg. These findings underscore the involvement of the RMTg and the ECS in pathologies characterized by dysregulated food intake
Coupé, Bérengère. "Impact de la nutrition périnatale sur la programmation du comportement alimentaire : de l'ontogenèse des réseaux hypothalamiques à la régulation de la prise alimentaire". Nantes, 2009. http://www.theses.fr/2009NANT2103.
Pełny tekst źródłaIn neonatology, low birth weight babies (small for gestational age) who suffered of intrauterine growth restriction (IUGR) are fed with protein enriched milk to ensure a catch-up growth and a better verbal intelligence quotient development. However, a catch-up growth after IUGR is correlated with metabolic syndrome associated diseases and with an energy balance regulation impair at adulthood. This observation is known as “foetal and/or postnatal programming”. The mechanisms at the origin of programming are still unknown. We hypothesized that food behaviour alterations could be associate to metabolic disease at adulthood and take place during early postnatal period when hypothalamus ontogeny occurs. The aim of this study was to determine, using IUGR experimental model in rodents induce by protein restriction during foetal life, the impact of a rapid catch-up growth (IUGR pups were nursed by ad-libitum-fed dams or artificial reared with protein enriched milk) on the ontogeny and the regulation of appetite hypothalamic networks. We demonstrated a positive effect of rapid catch-up growth after IUGR on establishment of hypothalamic pathways and on appetite regulation at weaning. However, food behaviour and metabolic parameters during a refeeding period were impaired in adult IUGR rats. These alterations seem to be programmed in utero. Our results indicate an alteration of food behavior which could be an important factor in the way to develop metabolic disorders
Atgié, Claude. "Prise alimentaire et niveau de l'activité adrénergique périphérique chez un rongeur hibernant, le Lérot Eliomys quercinus L". Toulouse 3, 1990. http://www.theses.fr/1990TOU30059.
Pełny tekst źródłaRomer, Michael. "Sensations hédoniques impliquées dans le contrôle de la prise alimentaire chez l'homme : alliesthésie alimentaire et Rassasiement sensoriel spécifique". Phd thesis, Université de Bourgogne, 2011. http://tel.archives-ouvertes.fr/tel-00728852.
Pełny tekst źródłaBariohay, Bruno. "Implication du Brain-Derived Neurotrophic Factor (BDNF) au niveau du Complexe Vagal Dorsal (CVD) dans le contrôle de la prise alimentaire". Aix-Marseille 3, 2007. http://www.theses.fr/2007AIX30018.
Pełny tekst źródłaBDNF has been implicated as an anorexigenic factor in the central regulation of food intake. In the present work, we identify the DVC as a key site where BDNF plays its anorexigenic role, downstream of melanocortinergic signalling. In the adult rat, we show that central application of exogenous BDNF at the DVC level induces anorexia and body weight loss, whereas endogenous BDNF protein content in the DVC is modulated in a manner consistent with an anorexigenic role, during the dark/light cycle, as well as following experimental paradigms affecting nutritional status or implying leptin, CCK or ghrelin treatments. In addition, we show that 4th ventricle administration of agonists or antagonists for type 3/4 melanocortinergic receptors (MC3/4R) induces respectively an increase and a decrease in BDNF protein content, whereas the orexigenic effects of the MC3/4R antagonist are blocked by a co-treatment with exogenous BDNF
Baraboi, Elena-Dana. "Glucagon-like peptide 1 et peptide YY : activation neuronal et contrôle de la prise alimentaire chez le rat". Thesis, Université Laval, 2010. http://www.theses.ulaval.ca/2010/27813/27813.pdf.
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