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Gotowe bibliografie tematyczne / Presenile dementia / Artykuły w czasopismach

Artykuły w czasopismach na temat „Presenile dementia”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 3 marca 2023

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1

NAKAMURA, SHIGENOBU. "Senile Dementia and Presenile Dementia". Tohoku Journal of Experimental Medicine 161, Supplement (1990): 49–60. http://dx.doi.org/10.1620/tjem.161.supplement_49.

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2

Pinhorn, A. "Presenile dementia. Dementia classification misleading." BMJ 306, nr 6888 (15.05.1993): 1343–44. http://dx.doi.org/10.1136/bmj.306.6888.1343-a.

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3

Järpe, Sven. "PRESENILE DEMENTIA AND HYDROCEPHALUS". Acta Neurologica Scandinavica 46, S43 (29.01.2009): 89. http://dx.doi.org/10.1111/j.1600-0404.1970.tb02167.x.

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4

Lying-Tunell, Ulla, Doris Bergquist, Gerhard Böhmer, Hans Olov Malmlund, Olle Marions i Berndt Söderborg. "STUDIES IN PRESENILE DEMENTIA". Acta Neurologica Scandinavica 46, S43 (29.01.2009): 90–92. http://dx.doi.org/10.1111/j.1600-0404.1970.tb02168.x.

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5

Treves, T., A. D. Korczyn, N. Zilber, E. Kahana, Y. Leibowitz, M. Alter i B. S. Schoenberg. "Presenile dementia in Israel". Alzheimer Disease & Associated Disorders 1, nr 1 (1987): 44. http://dx.doi.org/10.1097/00002093-198701000-00011.

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6

Whalley, L., G. McGonigal, B. MacLennan, C. McQuade, J. Starr i B. Thomas. "Presenile dementia: Authors' reply". BMJ 306, nr 6888 (15.05.1993): 1343–44. http://dx.doi.org/10.1136/bmj.306.6888.1343-c.

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7

Treves, T., A. D. Korczyn, N. Zilber, E. Kahana, Y. Leibowitz, M. Alter i B. S. Schoenberg. "Presenile Dementia in Israel". Archives of Neurology 43, nr 1 (1.01.1986): 26–29. http://dx.doi.org/10.1001/archneur.1986.00520010022014.

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8

Wright, Christine E., i Paul L. Furlong. "Visual Evoked Potentials in Elderly Patients with Primary or Multi-Infarct Dementia". British Journal of Psychiatry 152, nr 5 (maj 1988): 679–82. http://dx.doi.org/10.1192/bjp.152.5.679.

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Flash and pattern-reversal visual evoked potentials (VEP) were recorded in 35 elderly patients with dementia, and 19 controls of equivalent age. Dementia produced a slowing of the major positive (P2) component of the flash VEP but did not affect the latency of the flash P1 component or the P100 pattern-reversal component. This unusual type of abnormality was found in both primary and multi-infarct types of dementia, and has previously been found in primary presenile dementia. The results show that the VEP can be used for the diagnosis of multi-infarct, and primary presenile and senile dementias.

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9

Ernst, B., M. A. Dalby i A. Dalby. "APHASIC DISTURBANCES IN PRESENILE DEMENTIA". Acta Neurologica Scandinavica 46, S43 (29.01.2009): 99–100. http://dx.doi.org/10.1111/j.1600-0404.1970.tb02173.x.

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10

Tonkonogy, Joseph, i Gary S. Moak. "Alois Alzheimer on Presenile Dementia". Topics in geriatrics 1, nr 4 (październik 1988): 199–206. http://dx.doi.org/10.1177/089198878800100403.

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11

Derderian, Sarkis S., Krishnan R. Rajagopal i Bahman Jabbari. "Respiratory Control in Presenile Dementia". American Review of Respiratory Disease 137, nr 1 (styczeń 1988): 158–61. http://dx.doi.org/10.1164/ajrccm/137.1.158.

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12

Schmutzhard, Erich, Mikael Carlsson i Bo-Eric Malmvall. "BORRELIA INFECTION AND PRESENILE DEMENTIA". Lancet 330, nr 8569 (listopad 1987): 1214. http://dx.doi.org/10.1016/s0140-6736(87)91354-7.

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13

Abalan, François, i Jean Michel Delile. "B12 deficiency in presenile dementia". Biological Psychiatry 20, nr 11 (listopad 1985): 1251. http://dx.doi.org/10.1016/0006-3223(85)90187-8.

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14

Newens, A. J., D. P. Forster, D. W. Kay i J. Edwardson. "Presenile dementia. Gender difference unlikely." BMJ 306, nr 6888 (15.05.1993): 1343–44. http://dx.doi.org/10.1136/bmj.306.6888.1343-b.

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15

Goh, Kah Kheng, Yi-Hang Chiu i Winston W. Shen. "Hashimoto’s encephalopathy mimicking presenile dementia". General Hospital Psychiatry 36, nr 3 (maj 2014): 360.e9–360.e11. http://dx.doi.org/10.1016/j.genhosppsych.2014.01.006.

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16

Koo, E. M., S. J. DeArmond, J. Hart, B. Gordon, D. J. Selkoe i D. L. Price. "CEREBELLAR PLAQUES IN PRESENILE DEMENTIA". Journal of Neuropathology and Experimental Neurology 46, nr 3 (maj 1987): 338. http://dx.doi.org/10.1097/00005072-198705000-00026.

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17

Harding, Graham F. A., Christine E. Wright i Arnold Orwin. "Primary Presenile Dementia: The Use of the Visual Evoked Potential as a Diagnostic Indicator". British Journal of Psychiatry 147, nr 5 (listopad 1985): 532–39. http://dx.doi.org/10.1192/bjp.147.5.532.

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The use of the flash and pattern reversal visual evoked potential (VEP) in the diagnosis of primary presenile dementia was investigated. The results from 20 patients with primary presenile dementia were compared with those from a control group of normals of equivalent age and from a control group of 20 patients with cortical atrophy but no dementia. Presenile dementia caused a slowing of the major positive (P2) component of the VEP to flash stimulation. However, the VEP to pattern reversal stimulation (P100) was of normal latency. The difference between these two latencies characterises this unusual combination of results and is found to be a more specific diagnostic indicator of primary presenile dementia than the EEG or CT scan.

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18

Gunner-Svensson, Finn, Poul Gertz Andersson, Knud Jensen i K. A. Lorentzen. "PRESENILE DEMENTIA (ALZHEIMER'S AND PICK'S DISEASES)". Acta Neurologica Scandinavica 46, S43 (29.01.2009): 77. http://dx.doi.org/10.1111/j.1600-0404.1970.tb02159.x.

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19

Ernst, B., A. Dalby i M. A. Dalby. "GNOSTIC-PRAXIC DISTURBANCES IN PRESENILE DEMENTIA". Acta Neurologica Scandinavica 46, S43 (29.01.2009): 101–2. http://dx.doi.org/10.1111/j.1600-0404.1970.tb02174.x.

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20

Passant, Ulla, Jovanka Ostojic, Susanne Froelich Fabre, Lars Gustafson, Lars Lannfelt, Elna-Marie Larsson, Karin Nilsson, Ingmar Rosén i Christina Elfgren. "Familial Presenile Dementia with Bitemporal Atrophy". Dementia and Geriatric Cognitive Disorders 17, nr 4 (2004): 287–92. http://dx.doi.org/10.1159/000077156.

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21

Waltregny, Alain, Alhakam Abdul Maula i Jean-Marie Brucher. "Stereotactic Brain Biopsies in Presenile Dementia". Stereotactic and Functional Neurosurgery 50, nr 1-6 (1987): 218–22. http://dx.doi.org/10.1159/000100712.

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22

Mitsuyama, Yoshio. "Presenile Dementia with Motor Neuron Disease". Dementia and Geriatric Cognitive Disorders 4, nr 3-4 (1993): 137–42. http://dx.doi.org/10.1159/000107312.

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23

French, L. "Presenile dementia among the mentally retarded". Archives of Clinical Neuropsychology 1, nr 3 (1.01.1986): 255. http://dx.doi.org/10.1093/arclin/1.3.255.

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24

Delany, Norma, i Henry Rosenvinge. "Presenile dementia: Sufferers, carers and services". International Journal of Geriatric Psychiatry 10, nr 7 (lipiec 1995): 597–601. http://dx.doi.org/10.1002/gps.930100710.

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25

Torvik, Ansgar. "ASPECTS OF THE PATHOLOGY OF PRESENILE DEMENTIA". Acta Neurologica Scandinavica 46, S43 (29.01.2009): 19–31. http://dx.doi.org/10.1111/j.1600-0404.1970.tb02154.x.

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26

Johannesson, G., B. Hagberg, L. Gustafson i D. H. Ingvar. "EEG and cognitive impairment in presenile dementia". Acta Neurologica Scandinavica 59, nr 5 (29.01.2009): 225–40. http://dx.doi.org/10.1111/j.1600-0404.1979.tb02933.x.

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27

TSUNODA, Ayami, Shuji IRITANI i Norio OZAKI. "Presenile dementia diagnosed as posterior cortical atrophy". Psychogeriatrics 11, nr 3 (15.06.2011): 171–76. http://dx.doi.org/10.1111/j.1479-8301.2011.00366.x.

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28

Mitsuyama, Y. "14 Presenile dementia with motor neuron disease". Neurobiology of Aging 17, nr 4 (styczeń 1996): S4. http://dx.doi.org/10.1016/s0197-4580(96)80016-9.

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29

Ghetti, B., M. R. Farlow, R. A. Crowther, M. Goedert i M. G. Spillantini. "HEREDITARY MULTIPLE SYSTEM DEGENERATION WITH PRESENILE DEMENTIA". Journal of Neuropathology and Experimental Neurology 55, nr 5 (maj 1996): 608. http://dx.doi.org/10.1097/00005072-199605000-00026.

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30

Fujihara, Satomi, Sonia M. D. Brucki, Maria Sheila G. Rocha, Alzira A. Carvalho i Ana C. Piccolo. "Prevalence of presenile dementia in a tertiary outpatient clinic". Arquivos de Neuro-Psiquiatria 62, nr 3a (wrzesień 2004): 592–95. http://dx.doi.org/10.1590/s0004-282x2004000400005.

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There are very few reports about prevalence of presenile dementia in Brazil. We reviewed files of patients evaluated with early onset of cognitive impairment in our institution. Among 141 patients (61% males) there was no difference between gender by age at onset or at first evaluation. We have observed an increasing number of patients after 50 years. The most frequent causes were: vascular dementia (36.9%), Alzheimer's disease (20.3%) and traumatic brain injury (9.2%). There was difference among dementia type by age of onset and first evaluation, educational level and length of dementia. These results may be compared with those from other neurologic services in order to replicate or confirm these results.

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31

Shibayama, Hiroto, Hiroshi Kobayashi, Shoji Iwase, Minoru Nakagawa, Yasuo Marui, Yuhei Kayukawa, Hiromu Iwata i Toru Takeuchi. "Unusual Cases of Presenile Dementia with Fahr's Syndrome". Psychiatry and Clinical Neurosciences 40, nr 1 (marzec 1986): 85–100. http://dx.doi.org/10.1111/j.1440-1819.1986.tb01615.x.

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32

Kamp, P. E., W. A. den Hartog Jager, J. Maathuis, P. A. de Groot, J. M. de Jong i P. A. Bolhuis. "Brain gangliosides in the presenile dementia of Pick." Journal of Neurology, Neurosurgery & Psychiatry 49, nr 8 (1.08.1986): 881–85. http://dx.doi.org/10.1136/jnnp.49.8.881.

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33

Carlsson, Mikael, i Bo-Eric Malmvall. "BORRELIA INFECTION AS A CAUSE OF PRESENILE DEMENTIA". Lancet 330, nr 8562 (październik 1987): 798. http://dx.doi.org/10.1016/s0140-6736(87)92528-1.

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34

Helme, T. "Presenile dementia. Risk to women inflated by artefact." BMJ 306, nr 6888 (15.05.1993): 1343–44. http://dx.doi.org/10.1136/bmj.306.6888.1343.

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35

Newens, A. J., D. P. Forster i D. W. Kay. "Death certification after a diagnosis of presenile dementia." Journal of Epidemiology & Community Health 47, nr 4 (1.08.1993): 293–97. http://dx.doi.org/10.1136/jech.47.4.293.

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36

Dark, Frances. "A Family with Autosomal Dominant, Non-Alzheimer's Presenile Dementia". Australian & New Zealand Journal of Psychiatry 31, nr 1 (luty 1997): 139–44. http://dx.doi.org/10.3109/00048679709073812.

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Objective:A rare family pedigree is described with a multigenerational history of an early onset, non-Alzheimer's dementia consistent with autosomal dominant inheritance. Information on five generations, with 26 suspected or proven cases of dementia, are presented. Method:Previous work on the family was collated and verified. The pedigree was updated. Thirty-three family members agreed to be interviewed for the present study. Standardised clinical information was obtained using the Cambridge Mental Disorders of the Elderly Examination (CAMDEX) National Adult Reading Test (NART), vocabulary and digit substitution subscales of the Wechster adult intelligence scale — revised edition (WARS-R). Abbot samples were taken for biochemical and genetic analysis. Results:Fifteen males and 11 females have been affected. The age of onset of dementia in those for whom data were available (n = 12) ranged from 39 to 64 years with a mean of 53 years. The duration of illness ranged from 4 to 14 years and the age at death ranged from 49 to 69 years (mean 62 years). Autopsy data exist for nine cases. In one case the neuropathology was consistent with Alzheimer's disease. In two cases the diagnosis of Pick's disease was made on the basis of frontal or frontal/temporal lobe atrophy without Pick bodies or cells. One case diagnosed as Pick's disease had frontal/temporal lobe atrophy with cells resembling Pick's bodies. In the five remaining cases there were no distinctive neuropathological features to differentiate the type of dementia. Conclusions:The importance of recognising familial dementia, collating information on multiple generations and prospectively collecting standardised data is discussed.

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37

Renvoize, E. B., R. H. S. Mindham, M. Stewart, R. McDonald i D. R. D. Wallace. "Identical Twins Discordant for Presenile Dementia of the Alzheimer Type". British Journal of Psychiatry 149, nr 4 (październik 1986): 509–12. http://dx.doi.org/10.1192/bjp.149.4.509.

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In genetically proven identical female twins, discordant for presenile dementia of the Alzheimer type, the affected twin began to dement at the age of 49, and died 15 years later; the diagnosis was confirmed at post-mortem. The surviving twin remains clinically unaffected 20 years after the onset of dementia in her sister. Environmental aetiological factors are postulated to account for this discordance.

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38

Swearer, Joan M., i David A. Drachman. "Caretaker Obstreperous Behavior Rating Scale". International Psychogeriatrics 8, S3 (maj 1997): 321–24. http://dx.doi.org/10.1017/s1041610297003554.

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Although Alzheimer's original description of the dementing disorder that bears his name emphasized the prominence of troublesome and disruptive behaviors, a systematic investigation of behavioral disturbances of dementia did not begin in earnest until the 1980s. At that time, as the neuropathologic identity of presenile Alzheimer's disease and late-onset “senile dementia” was recognized, the redefinition of Alzheimer's disease abruptly increased the number of patients diagnosed with this condition. Physicians and other medical personnel working with Alzheimer's disease patients recognized both the importance of abnormal behaviors in this now large patient population and the need to describe, classify, and quantify these behaviors.

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39

McGonigal, G., B. Thomas, C. McQuade, J. M. Starr, W. J. MacLennan i L. J. Whalley. "Epidemiology of Alzheimer's presenile dementia in Scotland, 1974-88." BMJ 306, nr 6879 (13.03.1993): 680–83. http://dx.doi.org/10.1136/bmj.306.6879.680.

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40

Müller, G., R. A. Richter, S. Weisbrod i F. Klingberg. "Labyrinth Learning Impairment in Presenile Dementia: 1 Year Later". Gerontology 37, nr 6 (1991): 349–50. http://dx.doi.org/10.1159/000213284.

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41

Neary, D., J. S. Snowden, D. M. Bowen, N. R. Sims, D. M. Mann, J. S. Benton, B. Northen, P. O. Yates i A. N. Davison. "Neuropsychological syndromes in presenile dementia due to cerebral atrophy." Journal of Neurology, Neurosurgery & Psychiatry 49, nr 2 (1.02.1986): 163–74. http://dx.doi.org/10.1136/jnnp.49.2.163.

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42

Mendez, Mario F., i Anne Lipton. "Emergent Neuroleptic Hypersensitivity as a Herald of Presenile Dementia". Journal of Neuropsychiatry and Clinical Neurosciences 13, nr 3 (sierpień 2001): 347–56. http://dx.doi.org/10.1176/jnp.13.3.347.

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43

Kosaka, K. "Diffuse neurofibrillary tangles with calcification: a new presenile dementia." Journal of Neurology, Neurosurgery & Psychiatry 57, nr 5 (1.05.1994): 594–96. http://dx.doi.org/10.1136/jnnp.57.5.594.

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44

Greicius, M. D. "Presenile dementia syndromes: an update on taxonomy and diagnosis". Journal of Neurology, Neurosurgery & Psychiatry 72, nr 6 (1.06.2002): 691–700. http://dx.doi.org/10.1136/jnnp.72.6.691.

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45

Woodburn, K. J., i E. Johnstone. "Presenile dementia in Scotland: A clinical and genetic analysis". European Psychiatry 11 (styczeń 1996): 269s. http://dx.doi.org/10.1016/0924-9338(96)88810-9.

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46

Modrego, P. J., J. Mojonero, M. Serrano i N. Fayed. "Fahr’s syndrome presenting with pure and progressive presenile dementia". Neurological Sciences 26, nr 5 (grudzień 2005): 367–69. http://dx.doi.org/10.1007/s10072-005-0493-7.

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47

Kamp, P. E., W. A. den Hartog Jager, J. M. B. V. de Jong i P. A. Bolhuis. "Cerebral gangliosides in pick's presenile dementia: thin-layer chromatography". Clinical Neurology and Neurosurgery 87, nr 1 (styczeń 1985): 76. http://dx.doi.org/10.1016/0303-8467(85)90093-9.

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48

Filipović, Saša R., i Vladimir S. Kostić. "Utility of auditory P300 in detection of presenile dementia". Journal of the Neurological Sciences 131, nr 2 (sierpień 1995): 150–55. http://dx.doi.org/10.1016/0022-510x(95)00093-h.

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49

Farlow, M. R., J. Murrell i B. Ghetti. "1-12-18 Multi-systems tauopathy and presenile dementia". Journal of the Neurological Sciences 150 (wrzesień 1997): S20. http://dx.doi.org/10.1016/s0022-510x(97)84920-9.

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50

Starr, J. M., B. M. Thomas i L. J. Whalley. "Familial or sporadic clusters of presenile dementia in Scotland". Psychiatric Genetics 7, nr 4 (1997): 141–46. http://dx.doi.org/10.1097/00041444-199700740-00001.

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