Gotowa bibliografia na temat „Post-partum breast cancer”
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Artykuły w czasopismach na temat "Post-partum breast cancer"
Zimovjanova, M., P. Tesarova, B. Konopasek, J. Barkmanova, P. Brabec, J. Novotny, D. Pavlista i L. B. Petruzelka. "Pregnancy-associated breast cancer (PABC): Czech Young Breast Cancer Study Group Project 35." Journal of Clinical Oncology 29, nr 27_suppl (20.09.2011): 265. http://dx.doi.org/10.1200/jco.2011.29.27_suppl.265.
Pełny tekst źródłaZhang, Zhenzhen, Shangyuan Ye, Sarah M. Bernhardt, Heidi D. Nelson, Ellen M. Velie, Virginia F. Borges, Emma R. Woodward, D. Gareth R. Evans i Pepper J. Schedin. "Postpartum Breast Cancer and Survival in Women With Germline BRCA Pathogenic Variants". JAMA Network Open 7, nr 4 (19.04.2024): e247421. http://dx.doi.org/10.1001/jamanetworkopen.2024.7421.
Pełny tekst źródłaOktaviani, Ika, Sri Widiyas i Hellen Anggranis. "ANALISIS IBU POSTPARTUM DENGAN BENDUNGAN ASI LITERATURE VIEW". Prosiding Simposium Nasional Multidisiplin (SinaMu) 4 (6.02.2023): 310. http://dx.doi.org/10.31000/sinamu.v4i1.7891.
Pełny tekst źródłaLue, Jaida, Derek Radisky, Mark Sherman, Amy Degnim, Stacey Windam, Morian Curtis i Melody Stallings-Mann. "396 Unraveling the Immunological Basis of Lobular Involution in Breast Cancer Development". Journal of Clinical and Translational Science 8, s1 (kwiecień 2024): 118. http://dx.doi.org/10.1017/cts.2024.346.
Pełny tekst źródłaMenes, Tehillah S., Tal Sella i Gabriel Chodick. "Time to cancer diagnosis in young women presenting to surgeons with breast-related symptoms: A population-based cohort study." Journal of Clinical Oncology 37, nr 15_suppl (20.05.2019): e13099-e13099. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e13099.
Pełny tekst źródłaPanchal, Hiral. "A Study to Identify Problems of Lactation Among Postnatal Mothers During Early Post-Partum Period at Selected Hospitals of Bardoli Taluka of Surat District, Gujarat." Nursing Journal of India CXII, nr 06 (2021): 283–88. http://dx.doi.org/10.48029/nji.2021.cxii607.
Pełny tekst źródłaFaccio, Flavia, Chiara Ionio, Eleonora Mascheroni, Fedro Peccatori, Giulia Ongaro, Elena Cattaneo, Camilla Pisoni i in. "Risk factors in pregnant women with an oncological diagnosis and their impact in the post-partum period." Journal of Clinical Oncology 37, nr 15_suppl (20.05.2019): e23166-e23166. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e23166.
Pełny tekst źródłaAltshuler, Ellery, Sarah Wheeler i Karen Daily. "Bilateral primary breast Burkitt’s lymphoma in pregnancy". BMJ Case Reports 16, nr 1 (styczeń 2023): e251896. http://dx.doi.org/10.1136/bcr-2022-251896.
Pełny tekst źródłaPoorvu, Philip D., Yue Zheng, Tal Sella, Shoshana M. Rosenberg, Kathryn Jean Ruddy, Shari I. Gelber, Rulla M. Tamimi i in. "Diagnostic and treatment delays in young women with breast cancer." Journal of Clinical Oncology 37, nr 15_suppl (20.05.2019): 6575. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.6575.
Pełny tekst źródłaVan Der Hock, Sarah, Alekhya Chintamani, Gabriella Bulloch, Ishith Seth i Nita Dhupar. "Pregnancy-associated breast cancer: a case report and literature review". International Journal of Reproduction, Contraception, Obstetrics and Gynecology 12, nr 10 (28.09.2023): 3177–80. http://dx.doi.org/10.18203/2320-1770.ijrcog20232968.
Pełny tekst źródłaRozprawy doktorskie na temat "Post-partum breast cancer"
Charifou, Elsa. "Characterization and impact of cellular senescence during mammary gland involution". Electronic Thesis or Diss., Sorbonne université, 2022. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2022SORUS559V2.pdf.
Pełny tekst źródłaCellular senescence is a biological stress response characterized by a stable cell cycle arrest. Nonetheless, cells remain metabolically active and acquire a senescence-associated secretory phenotype (SASP), a complex secretome composed of cytokines, chemokines, growth factors, and extracellular matrix remodeling modulators. Senescence is associated with various pathological processes, such as tumorigenesis and aging. However, it is unknown when, where and how senescence contributes to physiological processes. To answer this question, we took advantage of the mammary gland (MG), an organ with remarkable plasticity throughout postnatal development. The MG involution is one of the major mammalian cell death and tissue remodeling events, when milk-producing epithelial cells are removed, and the MG returns to its pre-gestation state, resting for further pregnancy. During my Ph.D., we showed that senescence was transiently induced during the irreversible phase of involution. The senescent program occurred specifically in the alveolar milk-producing luminal cells and correlated with the expression of the cell cycle inhibitor p16. In parallel, we established a novel organoid system to mimic MG gestation, lactation, and involution. In this ex-vivo model, we also highlighted the presence of senescent cells strictly during the involution-like process. To assess the biological impact of senescence in vivo, we used a teat sealing method to uncouple the reversible and irreversible phases of involution. We unveiled a close association between the withdrawal of lactogenic hormones occurring in the second phase of involution and the induction of the senescence program. To further define the physiological roles of senescence during involution, we treated mice with ABT-263, a senolytic compound inducing apoptosis of senescent cells. Interestingly, we observed an impaired tissue remodeling upon senescence elimination, with larger remaining alveolar structures and delayed adipocyte refilling. Moreover, in organoids from transgenic p16-3MR mice, we successfully removed senescent cells with ganciclovir and delayed the involution-like process. Taken together, both in vivo and ex-vivo models suggest an essential role of senescence in modulating the tissue remodeling phase of MG involution. Importantly, the involution process is intimately associated with postpartum breast cancer (PPBC), a cancer diagnosed within 10 years following delivery with a poor prognosis. Investigating how senescence impacts the microenvironment during the involution process might provide major insights to understand PPBC
Streszczenia konferencji na temat "Post-partum breast cancer"
Ogony, Joshua, DereK C. Radisky, Amy C. Degnim, Marlene Frost, Tanya L. Hoskin, Michael G. Heckman, Launia J. White i Mark E. Sherman. "Abstract 4635: Analysis of post-partum breast tissue to understand early onset breast cancer". W Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-4635.
Pełny tekst źródłaStanford, Jamie C., Barbara FIngleton, Phil Owen i Rebecca S. Cook. "Abstract B097: Innate immune response to cell death in the post-partum mammary gland increases malignancy of parity-associated mammary tumors". W Abstracts: AACR Special Conference on Advances in Breast Cancer Research: Genetics, Biology, and Clinical Applications - October 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1557-3125.advbc-b097.
Pełny tekst źródłaCook, Rebecca S., Shelton Earp i Jamie Stanford. "Abstract P4-04-02: Cell death and efferocytosis generate a pro-metastatic landscape during mammary gland involution that increase dissemination of post-partum breast cancers". W Thirty-Seventh Annual CTRC-AACR San Antonio Breast Cancer Symposium; December 9-13, 2014; San Antonio, TX. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.sabcs14-p4-04-02.
Pełny tekst źródłaBorges, VF, ES Callihan, S. Jindal, T. Lyons, E. Manthey, D. Gao i PJ Schedin. "P2-01-04: The Post-Partum Diagnosis of Pregnancy Associated Breast Cancer Confers an Increased Risk for Metastasis without Increased Incidence of Poorer Prognosis Biologic Subtype." W Abstracts: Thirty-Fourth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 6‐10, 2011; San Antonio, TX. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/0008-5472.sabcs11-p2-01-04.
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