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Karnati, Srikanth. "Functional characterization of peroxisomes and pathological consequences of peroxisomal dysfunction in the lung". Giessen : VVB Lauferweiler, 2009. http://d-nb.info/100020572X/04.
Pełny tekst źródłaCastro, Ines Gomes Oliveira. "Tail-anchored proteins at peroxisomes : identification of MIRO1 as a novel peroxisomal motility factor". Thesis, University of Exeter, 2016. http://hdl.handle.net/10871/24657.
Pełny tekst źródłaLiu, Xiaoxi. "BEYOND PEROXISOME: ABCD2 MODIFIES PPARα SIGNALING AND IDENTIFIES A SUBCLASS OF PEROXISOMES IN MOUSE ADIPOSE TISSUE". UKnowledge, 2014. http://uknowledge.uky.edu/pharmacy_etds/41.
Pełny tekst źródłaAboushadi, Nahla Mohamed. "Role of peroxisomes in isoprenoid biosynthesis /". Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2000. http://wwwlib.umi.com/cr/ucsd/fullcit?p9963646.
Pełny tekst źródłaKarnati, Srikanth [Verfasser]. "Functional characterization of peroxisomes and pathological consequences of peroxisomal dysfunction in the lung / by Srikanth Karnati". Giessen : VVB Laufersweiler, 2009. http://d-nb.info/999686674/34.
Pełny tekst źródłaBottger, Gina. "Receptor-mediated import of proteins into peroxisomes". [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2001. http://dare.uva.nl/document/60502.
Pełny tekst źródłaVries, Bart de. "The biology of peroxisomes in Hansenula polymorpha". [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2008. http://irs.ub.rug.nl/ppn.
Pełny tekst źródłaAnderson, I. W. "Permeability of leaf peroxisomes to photorespiratory metabolites". Thesis, University of Oxford, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.379882.
Pełny tekst źródłaDrago, Ilaria. "Ca2+ homeostasis in mammalian and plant peroxisomes". Doctoral thesis, Università degli studi di Padova, 2009. http://hdl.handle.net/11577/3426033.
Pełny tekst źródłaI perossisomi sono degli organelli intracellulari circondati da una singola membrana coinvolti nell’eliminazione di specie reattive dell’ossigeno, ?- e ?-ossidazione di acidi grassi, biosintesi di eteri di fosfolipidi e in altre reazioni metaboliche. Sebbene studi recenti abbiano elucidato i meccanismi alla base della formazione, della fusione- fissione e dell’importo di proteine nella matrice dei perossisomi, le informazioni riguardanti il ruolo dei perossisomi nel signalling cellulare sono scarse e, fino a poco tempo fa, quelle riguardanti il possibile ruolo dei perossisomi nel signalling cellulare del Ca2+ erano totalmente assenti. Il signalling del Ca2+ è alla base di un ampio numero di funzioni cellulari sia fisiologiche che patologiche e mentre il ruolo di compartimenti subcellulari come il reticolo endoplasmico, i mitocondri, il nucleo e l’apparato di Golgi nelle dinamiche intracellulari del Ca2+ è stato ampiamente studiato negli ultimi decenni, i perossisomi sono rimasti nella “zona d’ombra” di questo scenario. Infine, c’è stato ultimamente un rinnovato interesse circa le funzioni dei perossisomi grazie alla scoperta di un certo numero di malattie umane (chiamate “disordini dei perossisomi”) dovute a mutazioni di proteine perossisomiali. Per tutte queste ragioni, ho deciso di investigare se, e come, i perossisomi rivestono un qualche ruolo nell’omeostasi intracellulare del Ca2+. A questo scopo ho indirizzato alla matrice dei perossisomi una sonda per il Ca2+ geneticamente codificata e basata su FRET e ho potuto dimostrare che la concentrazione di Ca2+ nei perossisomi di cellule vive in condizioni di riposo è molto simile a quella citosolica mentre aumenti della concentrazione di Ca2+ (causati sia da mobilizzazione di Ca2+ dai depositi intracellulari che da influsso attraverso canali per il Ca2+ situati nella membrana plasmatica) sono solitamente seguiti da un lento aumento della concentrazione di Ca2+ nella matrice perossisomiale. Mi sono inoltre occupata della caratterizzazione del meccanismo che sta alla base dell’entrata di Ca2+ nei perossisomi e sono arrivata alla conclusione che questo fenomeno non è dovuto alla presenza di una pompa dipendente da ATP, né di un potenziale di membrana o di un gradiente di H+ o Na+. La membrana dei perossisomi sembra costituire una barriera che previene l’entrata di Ca2+ nel caso di aumenti brevi nel tempo, mentre nel caso di aumenti prolungati della concentrazione di Ca2+ nel citosol permette una lenta equilibrazione della concentrazione di Ca2+ nella matrice perossisomiale con l’ambiente citosolico. I perossisomi sembrano quindi costituire un nuovo sistema-tampone per il Ca2+del citosol, sebbene il loro meccanismo di influsso ed efflusso per il Ca2+ è totalmente differente da quello di ogni altro organello cellulare. La seconda parte del mio lavoro si è poi concentrata sullo studio dei possibili ruoli fisiologici del fenomeno dell’entrata di Ca2+ nei perossisomi. In letteratura non sono al momento riportati degli enzimi localizzati nei perossisomi delle cellule di mammifero che siano regolati da Ca2+; al contrario, alcuni enzimi localizzati nei perossisomi delle piante sembrano essere regolati da Ca2+. Di questi, quello che più mi è sembrato interessante è un’isoforma di un enzima deputato all’eliminazione di H2O2, la catalasi. L’attività di Cat3 è infatti riportata essere attivata in vitro da Ca2+ e calmodulina. La sonda per il Ca2+ utilizzata per lo studio dei perossisomi in cellule di mammifero è stata quindi indirizzata ai perossisomi di cellule vegetali e ha permesso di dimostrare che il fenomeno dell’entrata di Ca2+ nei perossisomi è molto simile, sia per ampiezza che per cinetica, tra perossisomi di mammifero e di pianta. L’iperpolarizzazione della membrana plasmatica ha dimostrato essere uno stimolo ripetibile che causa un prolungato aumento della concentrazione di Ca2+ nei perossisomi (e nel citosol) di pianta ed è quindi stato scelto per verificare se un aumento di Ca2+ nei perossisomi possa in qualche modo influenzare l’eliminazione di H2O2. Esperimenti preliminari effettuati in piante di Arabidopsis che esprimono stabilmente una sonda per H2O2 geneticamente codificata indicano che l’eliminazione di H2O2 è notevolmente accelerata in seguito all’entrata di Ca2+; questo correla con il livello di Cat3 espressa nei perossisomi.
Smith, Jennifer Joy. "Maintenance of peroxisomes in the yeast Yarrowia lipolytica". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0008/NQ59670.pdf.
Pełny tekst źródłaTappia, Paramjit Singh. "Analysis of peroxisomes from mammalian intestine and liver". Thesis, University of Wolverhampton, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306242.
Pełny tekst źródłaGuedes, Mariana da Rocha Soares. "Mitochondria and peroxisomes : role within cellular antiviral defense". Master's thesis, Universidade de Aveiro, 2014. http://hdl.handle.net/10773/12960.
Pełny tekst źródłaThe present paper presents a review and compilation of all the scientifically relevant bibliography to date, regarding the antiviral signalling pathways implicated in the cellular innate immune system in humans. Emphasizing the mitochondrial antiviral signalling adaptor (MAVS), this paper explores the special features of the signal transduction pathways and their components in two specific organelles: mitochondria and peroxisomes. These pathways, ultimately, result in the expression of interferon-stimulated genes (ISGs), which are primarily responsible for fighting against viral replication, viral particle assembly and virion release within the cell. In this paper, several proposals for further investigation are also presented, since there is still a lot to learn about the role of peroxisomes in the antiviral innate immune responses.
O presente trabalho propõe-se a rever e compilar toda a bibliografia cientificamente relevante até à data, no que respeita as vias de sinalização antivirais implicadas na imunidade celular inata em células humanas. Com ênfase na proteína adaptadora MAVS, este trabalho explora as particularidades das vias de transdução de sinal e respetivos intervenientes em dois organelos celulares específicos: mitocôndrias e peroxissomas. Estas vias, em última instância, resultam na expressão de genes estimulados por interferões (ISGs), principais responsáveis pelo combate celular eficaz contra a replicação viral, montagem de partículas virais e libertação de viriões na célula infetada. Neste trabalho são ainda apresentadas propostas para investigações futuras, uma vez que ainda muito pouco se sabe sobre o papel dos peroxissomas nas respostas imunitárias inatas contra infeções virais.
Parshyna, Iryna. "Autophagic degradation of peroxisomes in the alkane-assimilating yeast Yarrowia lipolytica". Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2006. http://nbn-resolving.de/urn:nbn:de:swb:14-1164836301920-35300.
Pełny tekst źródłaXie, Weiqiao Hope Lila W. "Isolation and characterization of a gene required for peroxisome biogenesis". Oregon Health & Science University, 1993. http://content.ohsu.edu/u?/etd,234.
Pełny tekst źródłaMolecular Biology
This thesis describes the cloning and analysis of PER6, a gene required for peroxisome biogenesis in Pichia pastoris. The gene was cloned by functional complementation of a per6 P. pastoris mutant strain that was one of a number of peroxisome-deficient mutants isolated in this laboratory. The complementing activity was localized to a small DNA fragment by subcloning and Northern filter hybridization analysis and the DNA sequence of the fragment was determined. The sequence revealed a 1296-bp open reading frame which potentially encodes a 432-amino acid protein of 49 kD. The gene was transcribed into a message of 1.4 kilobases that was constitutively expressed but induced several-fold in cells growing on methanol. A mutant strain with a deletion of a large portion of the open reading frame was constructed and used to genetically demonstrate that the cloned gene was identical to the defective gene in the originally isolated per6 mutant. The predicted amino acid sequence of the PER6 product revealed several interesting features, including a significant regional similarity to PAF-1, a gene known to be defective in some patients with Zellweger syndrome, a lethal human genetic disease caused by peroxisome deficiency. Finally, the PER6 product was produced in E. coli and purified to serve as antigen for antibody production.
Xie, Weiqiao. "Isolation and characterization of a gene required for peroxisome biogenesis /". Full text open access at:, 1993. http://content.ohsu.edu/u?/etd,234.
Pełny tekst źródłaKlein, André Theodoor Jan. "Pex5p, a guide for import of proteins into peroxisomes". [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2003. http://dare.uva.nl/document/69923.
Pełny tekst źródłaBoateng, Eistine [Verfasser]. "The Role of Peroxisomes in Pulmonary Fibrosis / Eistine Boateng". Gießen : Universitätsbibliothek, 2019. http://d-nb.info/1199264539/34.
Pełny tekst źródłaAlves, Sandra Manuela Carvalho. "Exploring the role of peroxisomes in Helicobacter pylori infection". Master's thesis, Universidade de Aveiro, 2015. http://hdl.handle.net/10773/14216.
Pełny tekst źródłaThe Helicobacter pylori is a gram-negative microaerophilic bacterium. Humans are the principal reservoir and it has been estimated that about 50% of the world population is infected by this bacterium, even though only about 20% of the patients present symptoms. The H. pylori colonizes the human gastric mucosa and is associated to several gastrointestinal diseases, such chronic gastritis, peptic ulceration and gastric cancer. Peroxisomes are membrane-enclosed subcellular organelles, which can be virtually found in all eukaryotic cells. They are involved in several metabolic reactions, with emphasis for the fatty acid oxidation, lipid biosynthesis and hydrogen peroxide detoxification. It is increasingly accepted that peroxisomes are more than simple metabolic organelles within cells. Previous studies of viral infections have demonstrated that peroxisomes are involved in the cellular innate immune response. It has also been suggested that all bacterial and viral pathogens may be exposed to peroxisomal recognition. Thus, it becomes important to better study the role of the peroxisomes in response to infections. In the present work we have analysed the possible peroxisomal alterations in morphology, number and enzymatic function in infected cells with the main aim of exploring a possible role for peroxisomes in H. pylori infection. The obtained results suggest that H. pylori infection does not affect significantly the peroxisomal morphology, number or localization. However, the results obtained suggested that H. pylori infection affects the amount of peroxisomal catalase, probably due to an increase of ROS in the cellular environment, as a consequence of the bacterial infection.
Helicobacter pylori é uma bactéria gram-negativa microaerofílica. Os seres humanos são o seu principal reservatório e estima-se que cerca de 50% da população mundial encontra-se infetada, embora apenas cerca de 20% dos pacientes apresentem sintomas. H. pylori coloniza a mucosa gástrica humana e está associada a várias doenças gastrointestinais, como gastrite crónica, úlcera péptica e cancro gástrico. Os peroxissomas são organelos de membrana simples, que se encontram virtualmente em todas as células eucarióticas. Nestes ocorrem várias reações metabólicas, com destaque para a oxidação dos ácidos gordos, a biossíntese de lípidos e a desintoxicação do peróxido de hidrogénio. É cada vez mais aceite que os peroxissomas desempenham muito mais do que simples funções metabólicas. Em estudos anteriores foi demonstrado que os peroxissomas estão envolvidos na resposta imune em infeções virais. Para além disso, foi sugerido que tanto os patogénios bacterianos como os virais podem ser expostos ao reconhecimento peroxissomal. Assim, torna-se importante estudar melhor o papel dos peroxissomas em resposta a infeções. No presente trabalho foram analisadas possíveis alterações peroxisomais relativamente à sua morfologia, número e função enzimática, em células infetadas, com o principal objetivo de explorar o possível papel dos peroxissomas na infeção por H. pylori. Os resultados obtidos sugerem que a infeção por H. pylori não afeta significativamente a morfologia, número ou localização dos peroxissomas. No entanto, os resultados sugerem que a infeção por H. pylori afeta a quantidade de catalase peroxissomal, provavelmente devido a um aumento das EROs no meio celular, resultante da infeção bacteriana.
Maxwell, Megan Amanda, i n/a. "PEX1 Mutations in Australasian Patients with Disorders of Peroxisome Biogenesis". Griffith University. School of Biomolecular and Biomedical Science, 2004. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20040219.100649.
Pełny tekst źródłaMaxwell, Megan Amanda. "PEX1 Mutations in Australasian Patients with Disorders of Peroxisome Biogenesis". Thesis, Griffith University, 2004. http://hdl.handle.net/10072/366184.
Pełny tekst źródłaThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Biomolecular and Biomedical Sciences
Full Text
Ferreira, Ana Rita Filgueiras. "Hepatitis C virus and peroxisomes : evasion from the cellular antiviral response". Master's thesis, Universidade de Aveiro, 2015. http://hdl.handle.net/10773/14348.
Pełny tekst źródłaHepatitis C virus (HCV) causes the most prevalent viral infection worldwide. Upon infection, the HCV genome is detected by the RIG-I-MAVS signalling pathway leading to the production of direct antiviral effectors. NS3/4A protease is the main inhibitor of innate immunity against HCV and it was found to inhibit the mitochondrial signalling protein (MAVS). MAVS was recently found to localize at peroxisomes coordinating with mitochondria the activation of effective antiviral response. Peroxisomal MAVS is responsible for inducing a rapid but short termed antiviral response that is IFNindependent, contrary to mitochondrial MAVS which is associated with the activation of an IFN-dependent antiviral response with delayed kinetics. With this work we aimed at evaluating the effect of NS3/4A over the peroxisomal– MAVS pathway. Our results showed that the MAVS localizing exclusively at peroxisomes is targeted by the HCV NS3/4A protease. We also show that the MAVS cleavage by NS3/4A impaired the antiviral response mediated by peroxisomal-MAVS. These results reaffirm the importance of peroxisomes for viral-host interaction and in antiviral defences. Further studies are proposed in order to better understand the role of this organelle in innate immunity. These may lead to the improvement of therapy against HCV infection.
O vírus da hepatite C (VHC) provoca a infeção viral mais prevalente em todo o mundo. Após infeção, o genoma do VHC é detetado pela via de sinalização RIGI- MAVS levando à produção de efetores diretos da resposta antiviral. A protease NS3/4A é o principal inibidor da resposta imune produzido pelo VHC e foi descrito como inibidor da proteína MAVS. A proteína MAVS foi recentemente localizada nos peroxissomas que, juntamente com a mitocôndria, coordenam a resposta antiviral. A MAVS peroxisomal é responsável pela indução de uma resposta antiviral rápida mas de curta duração que é independente de interferões, mas pelo contrário, a MAVS mitocondrial está associada a uma ativação da resposta antiviral que é dependente de interferões mas que se caracteriza por uma cinética retardada. O nosso objetivo com este trabalho consistiu em avaliar o efeito da NS3/4A na via de sinalização coordenada pelos peroxissomas. Os nossos resultados mostram que a MAVS localizada nos peroxissomas é alvo da protease NS3/4A do VHC. Também mostramos que a clivagem da proteína MAVS pela NS3/4A inibe a resposta antiviral mediada pela MAVS peroxissomal. Estes resultados reafirmam a importância dos peroxissomas na interação vírushospedeiro e na defesa antiviral. Futuros estudos são aconselhados para que se compreenda a função dos peroxissomas na imunidade inata. Estes podem levar a uma melhoria na terapia da infeção pelo VHC.
Zwacka, Ralf Michael. "The human homologue of the murine glomerulosclerosis gene Mpv17". Thesis, Open University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.262628.
Pełny tekst źródłaMunck, Joanne Marie. "Molecular characterisation of the maintenance of peroxisomes in saccharoyces cerevisiae". Thesis, University of Sheffield, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.489055.
Pełny tekst źródłaSmith, Steven Andrew. "The role of Peroxisome proliferator-activated receptors in the rat brain /". St. Lucia, Qld, 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17982.pdf.
Pełny tekst źródłaFan, Wei [Verfasser]. "The role of peroxisomes in osteoblast differentiation and functions / Wei Fan". Gießen : Universitätsbibliothek, 2015. http://d-nb.info/1077170742/34.
Pełny tekst źródłaSoliman, Kareem [Verfasser], Jutta [Akademischer Betreuer] Gärtner, Silvio [Gutachter] Rizzoli i Stefan [Gutachter] Jakobs. "Characterization of peroxisomes and peroxisome deficient cell lines by super-resolution microscopy and biochemical methods / Kareem Soliman ; Gutachter: Silvio Rizzoli, Stefan Jakobs ; Betreuer: Jutta Gärtner". Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2016. http://d-nb.info/1140641921/34.
Pełny tekst źródłaEl-Merhie, Natalia [Verfasser]. "The role of peroxisomes in chronic obstructive pulmonary disease / Natalia El-Merhie". Gießen : Universitätsbibliothek, 2017. http://d-nb.info/1147680590/34.
Pełny tekst źródłaFredericks, Ernst. "Molecular signaling in colorectal carcinogenesis : the roles and relationships of beta-catenin, PPARgamma and COX-2". Thesis, Nelson Mandela Metropolitan University, 2013. http://hdl.handle.net/10948/d1021014.
Pełny tekst źródłaDe, Mora Kim Stephen. "Foundational technologies in synthetic biology : promoter measurement and peroxisome engineering". Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/7870.
Pełny tekst źródłaJohnson, Monique A. "Novel genetic selections for peroxisome biogenesis mutants (pex) and the isolation and characterization of PEX14 and Pex14p in Pichia Pastoris /". Full text open access at:, 2000. http://content.ohsu.edu/u?/etd,15.
Pełny tekst źródłaOzeki, Katharine. "Pichia pastoris Rppa09976 is a peroxisomal membrane-associated ACBP domain-containing protein, delivered to peroxisomes from the ER, and is required for their selective degradation". Diss., [La Jolla] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p1465084.
Pełny tekst źródłaTitle from first page of PDF file (viewed June 19, 2009). Available via ProQuest Digital Dissertations. Includes bibliographical references.
Delille, Hannah Katharina [Verfasser], i Ralf [Akademischer Betreuer] Jacob. "Biogenesis of peroxisomes in mammalian cells : Characterization of the Pex11 proteins and their role in peroxisomal growth and division / Hannah Katharina Delille. Betreuer: Ralf Jacob". Marburg : Philipps-Universität Marburg, 2011. http://d-nb.info/1013075285/34.
Pełny tekst źródłaLogan, Heather Elizabeth. "Phosphatidylcholine turnover and protein kinase C in human fibroblasts with mutations affecting peroxisomes". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/mq24866.pdf.
Pełny tekst źródłaWood, Adrian J. "Subcellular analysis of normal and pathological gastrointestinal tissue with specific reference to peroxisomes". Thesis, University of Wolverhampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386569.
Pełny tekst źródłaResende, Albina Dolores Cardoso da Silva Castro. "Seasonal and toxicological study of brown trout (Salmo trutta) kidney and liver peroxisomes". Doctoral thesis, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/7260.
Pełny tekst źródłaResende, Albina Dolores Cardoso da Silva Castro. "Seasonal and toxicological study of brown trout (Salmo trutta) kidney and liver peroxisomes". Tese, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/7260.
Pełny tekst źródłaChakraborty, Anirban [Verfasser], Ralf [Gutachter] Erdmann i Christian [Gutachter] Herrmann. "New insights into de novo biogenesis of peroxisomes and into peroxisome proliferation in accharomyces cerevisiae / Anirban Chakraborty ; Gutachter: Ralf Erdmann, Christian Herrmann ; Fakultät für Chemie und Biochemie". Bochum : Ruhr-Universität Bochum, 2018. http://d-nb.info/1191481360/34.
Pełny tekst źródłaChauhan, Dushyant. "Protein import into peroxisomes and oxidative stress: a study to elucidate the potential functional role of the conserved cysteine in Pex5p". Thesis, Högskolan i Skövde, Institutionen för vård och natur, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-5888.
Pełny tekst źródłaMiyata, Kenji Sean. "The molecular mechanism of transcriptional activation by the peroxisome proliferator activated-receptor (alpha) /". *McMaster only, 1999.
Znajdź pełny tekst źródłaGuo, Guanlun. "Stereo-selective binding of enantiomeric ligands in PPAR[gamma] : a molecular modeling study". HKBU Institutional Repository, 2013. http://repository.hkbu.edu.hk/etd_ra/1516.
Pełny tekst źródłaBabujee, Lavanya. "Proteome studies on leaf peroxisomes from Spinacia oleracea L. and Arabidopsis thaliana (L.) Heynh". Doctoral thesis, [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=972079734.
Pełny tekst źródłaVasko, Radovan [Verfasser], Hassan [Akademischer Betreuer] Dihazi i Michael [Akademischer Betreuer] Thumm. "Peroxisomes and Kidney Damage / Radovan Vasko. Gutachter: Hassan Dihazi ; Michael Thumm. Betreuer: Hassan Dihazi". Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2014. http://d-nb.info/1064404030/34.
Pełny tekst źródłaBonilla-Martinez, Hermelinda del Rocio [Verfasser]. "Peroxisomes in endocrine pancreatic islets, possible protectors against lipotoxicity? / Hermelinda del Rocio Bonilla-Martinez". Gießen : Universitätsbibliothek, 2019. http://d-nb.info/1185976922/34.
Pełny tekst źródłaAlkuwayti, Mayyadah. "The role of peroxisomes in sterol biosynthesis by the cellular slime mould Dictyostelium discoideum". Thesis, University of Sheffield, 2014. http://etheses.whiterose.ac.uk/7085/.
Pełny tekst źródłaNenicu, Anca [Verfasser]. "Influence of peroxisomes on development, maturation and adult functions of the testis / Anca Nenicu". Giessen : VVB Laufersweiler, 2010. http://d-nb.info/1008505536/34.
Pełny tekst źródłaCheng, Wai. "The relationship between peroxisome proliferator-activated receptors (PPARs) and cell proliferation /". View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36433937.
Pełny tekst źródłaWestin, Maria A. K. "Acyl-CoA thioesterases - auxiliary enzymes in peroxisomal lipid metabolism /". Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-241-5/.
Pełny tekst źródłaChing, Steven LK. "Arabidopsis glyoxylate reductase 1 is localized in the cytosol and not peroxisomes in plant cells". Thesis, Journal of Intergrative Plant Biology, 2012. http://hdl.handle.net/10214/3330.
Pełny tekst źródłaNSERC
Behari, Richa. "Establishment and characterisation of an in vitro system for import of proteins into plant peroxisomes". Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283921.
Pełny tekst źródłaWang, Yunhong [Verfasser], i Christian [Akademischer Betreuer] Schultz. "Subcellular distributions of peroxisomes and endoplasmic reticulum in hippocampal neurons / Yunhong Wang ; Betreuer: Christian Schultz". Heidelberg : Universitätsbibliothek Heidelberg, 2019. http://d-nb.info/1185170901/34.
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