Gotowa bibliografia na temat „Pathologies pulmonaires”
Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych
Spis treści
Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Pathologies pulmonaires”.
Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.
Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.
Artykuły w czasopismach na temat "Pathologies pulmonaires"
Bourguignon, Chloé, Charlotte Vernisse, Joffrey Mianné, Mathieu Fieldès, Engi Ahmed, Aurélie Petit, Isabelle Vachier i in. "Les organoïdes pulmonaires". médecine/sciences 36, nr 4 (kwiecień 2020): 382–88. http://dx.doi.org/10.1051/medsci/2020056.
Pełny tekst źródłaAder, F., S. Nseir, B. Guery i I. Tillie-Leblond. "Aspergillose pulmonaire aiguë invasive et pathologies pulmonaires chroniques". Revue des Maladies Respiratoires 23, nr 3 (czerwiec 2006): 11–20. http://dx.doi.org/10.1016/s0761-8425(06)71583-2.
Pełny tekst źródłaBonay, Marcel, Françoise Camus i Catherine Bancal. "Pathologies pulmonaires et sport". EMC - Pneumologie 1, nr 1 (styczeń 2004): 1–9. http://dx.doi.org/10.1016/s1155-195x(02)00070-1.
Pełny tekst źródłaBoutros, J., i S. Leroy. "Pathologies pulmonaires infiltratives diffuses". Revue des Maladies Respiratoires Actualités 12, nr 2 (październik 2020): 298–303. http://dx.doi.org/10.1016/s1877-1203(20)30127-0.
Pełny tekst źródłaMassin, Nicle, i Marie-Nathalie Kolopp-Sarda. "Pathologies pulmonaires d'origine immuno-allergique en milieu professionnel". Revue Française des Laboratoires 2004, nr 361 (marzec 2004): 39–46. http://dx.doi.org/10.1016/s0338-9898(04)90093-5.
Pełny tekst źródłaDeslée, G. "Macrolides à faible dose dans les pathologies pulmonaires". Revue des Maladies Respiratoires Actualités 5, nr 1 (2013): 54–59. http://dx.doi.org/10.1016/s1877-1203(13)70359-8.
Pełny tekst źródłaBarras, Guillemette, Yann Michel, Noémie Wagner i Louis Loutan. "Pathologies [b]pulmonaires[/b] au retour de voyage". Revue Médicale Suisse 8, nr 340 (2012): 1000–1005. http://dx.doi.org/10.53738/revmed.2012.8.340.1000.
Pełny tekst źródłaKambouchner, M., i J. F. Bernaudin. "Les lymphatiques dans les pathologies pulmonaires acquises non tumorales". Revue de Pneumologie Clinique 69, nr 3 (czerwiec 2013): 170–74. http://dx.doi.org/10.1016/j.pneumo.2012.12.008.
Pełny tekst źródłaBoyer, L., L. Savale, J. Boczkowski i S. Adnot. "Sénescence cellulaire et pathologies pulmonaires : exemple de la BPCO". Revue des Maladies Respiratoires 31, nr 10 (grudzień 2014): 893–902. http://dx.doi.org/10.1016/j.rmr.2014.07.015.
Pełny tekst źródłaLacombe, C., L. Monnier-Cholley, M. Lewin, L. Arrivé i J. M. Tubiana. "THO12 Imagerie des pathologies pulmonaires chez le patient HIV". Journal de Radiologie 87, nr 10 (październik 2006): 1561. http://dx.doi.org/10.1016/s0221-0363(06)87994-1.
Pełny tekst źródłaRozprawy doktorskie na temat "Pathologies pulmonaires"
Couffin, Rozenn Lagarde André. "Inter-relations pathologie générale et pathologie odonto-stomatologique pathologies pulmonaires /". [S.l.] : [s.n.], 2004. http://theses.univ-nantes.fr/thesemed/CDcouffin.pdf.
Pełny tekst źródłaFlamein, Florence. "Anomalies génétiques d'ABCA3 dans les détresses respiratoires néonatales sévères et les pathologies alvéolo-interstitielles de l'enfant". Paris 6, 2011. http://www.theses.fr/2011PA066290.
Pełny tekst źródłaMontigaud, Yoann. "Modèles précliniques ex vivo pour l'étude de la délivrance pulmonaire d'aérosols dans le traitement de pathologies pulmonaires". Thesis, Lyon, 2020. http://www.theses.fr/2020LYSEM028.
Pełny tekst źródłaPulmonary delivery seem to be a preferential choice for the treamt of respiratory diseases. However, optimal targeting should be reached to increase efficacy and decrase the risk of side effects. Thus, research is needed to improve aerosol delivery devices. However, ethical restrictions related to human experiment are not in agreement with the previous statement. Therefore, preclinical model are needed but could lack of relevance or generated date could be hard to extrpolate. The present work aimed to develop a panel of preclinical ex vivo respiratory models to systematise knowledge to facilitate the clinical transfer of aerosol technologies. For each developed ex vivo model, the aerosol deposition pattern was assessed and compared to human and/or animal data to ensure the extrapolability of the results and to position the model among the available preclinical models. Applications, such as the optimal position of a nebuliser during invasive mechanical ventilation or the deposition profile of electronic cigarette aerosol, were performed. The developed ex vivo models showed comparability with patients deposition profile of aersosol, as well as their utility as a new preclinical tool fitting 3R guidelines to complete exisiting preclinical models in aerosol therapy
Merrien, Florence Caroff Nathalie. "Pathologies pulmonaires liées aux endotoxines bactériennes le modèle de la byssinose /". [S.l.] : [s.n.], 2004. http://theses.univ-nantes.fr/thesemed/.pdf.
Pełny tekst źródłaButeau, Marion. "Analyse d'images radiographiques du thorax pour l'aide au diagnostic des pathologies pulmonaires". Rennes 1, 1998. http://www.theses.fr/1998REN10011.
Pełny tekst źródłaKryza, Thomas. "Etude de l'implication de la kallicréine 12 dans le remodelage tissulaire associé aux pathologies pulmonaires". Thesis, Tours, 2013. http://www.theses.fr/2013TOUR3314.
Pełny tekst źródłaIn this thesis, we studied the involvement of the serine protease kallikrein-12 (KLK12), in lung carcinogenesis. The KLK12 is known to be overexpressed in non-small cell lung cancer but its role in carcinogenesis is not clearly established. Our work shows that it possesses a proangiogenic effect on pulmonary endothelial cells. Indeed, KLK12 stimulates lung endothelial cells migration notably by modulating the extracellular matrix architecture. On the other hand, KLK12 regulates the bioavailability of growth factors associated with angiogenesis such as plateletderived growth factor-B. In addition, our work has identified a possible regulation of the expression of KLK12 by hypoxia, which would explain its overexpression in lung tumors and would reinforce its involvement in angiogenesis. The confirmation in vivo of these mechanisms could help consider KLK12 as a therapeutic target for regulating tumor angiogenesis
GOUALIN, POUSSET PASCALE. "Interet du diagnostic echographique dans la prise en charge des pathologies pleuro-pulmonaires du foetus". Angers, 1993. http://www.theses.fr/1993ANGE1054.
Pełny tekst źródłaAndrault, Pierre-Marie. "Rôle des cathepsines à cystéine dans la régulation du peptide antimicrobien LL-37 lors de pathologies inflammatoire chroniques pulmonaires". Thesis, Tours, 2015. http://www.theses.fr/2015TOUR4035/document.
Pełny tekst źródłaDuring chronic inflammatory lung diseases like cystic fibrosis or COPD, proteases/antiproteases imbalance leads to pulmonary tissue degradation and compromise antimicrobial barrier. Cysteine cathepsins are involved in the proteolytic inactivation of several lung antimicrobial peptides (AMPs) such as SLPI, lactoferrin and β- defensins -2 and -3 during emphysema or cystic fibrosis. During this thesis, we studied the ability of cathepsins B, K, L and S to degrade LL-37, which is an important AMP in lung immunity. Only cathepsins K and S degrade readily LL-37 and inactivate its antimicrobial property. Conversely, LL-37 is a competitive inhibitor of cathepsin L. Beside, lung expression of human cathepsin S is significantly increased in smokers with or without COPD compared to non-smokers. Cigarette smoke that is a major source of oxidative stress significantly increases the expression and activity of cathepsin S. Despite an unfavorable oxidative environment, cathepsin S retains its proteolytic activity toward LL-37 and thus could participate to COPD exacerbation
Lemjabbar, Alaoui Hassan. "Implication de la gélatinase B (92 kDa) au cours des pathologies pulmonaires humaines : fibrose interstitielle diffuse et etat de mal asthmatique". Paris 12, 1998. http://www.theses.fr/1998PA120028.
Pełny tekst źródłaGoven, Delphine. "Régulation de l’hème oxygénase-1 dans les macrophages au cours des pathologies pulmonaires liées à l’exposition de la fumée de cigarette". Thesis, Paris Est, 2009. http://www.theses.fr/2009PEST0051.
Pełny tekst źródłaChronic cigarette smoking, a source of oxidants, is an important risk factor for lung emphysema and primary spontaneous pneumothorax development. Alveolar macrophages are mainly involved in lung inflammation observed in these pathologies through the production of metalloproteases and reactive oxygen species resulting to protease/anti-protease and oxidant/anti-oxidant imbalances. Heme oxygenase-1 (HO-1), mainly expressed in macrophages, is a key enzyme in pulmonary anti-oxidant defences. Therefore, the first aim of our studies was to investigate the expression and cellular localisation of HO-1 and its potential regulators (Nrf2, Keap1, Bach1 and HIF-1a) in alveolar macrophages from smoking related lung emphysema and primary spontaneous pneumothorax. Regulation pathways involved in expression of these proteins were assessed in vitro in macrophage cell line THP-1 exposed or not to cigarette smoke condensate and with or without hypoxia-reoxygenation mimicking parts of events induced by atelectasia-reexpansion during recurrent pneumothorax constitution and treatment. In these studies, we showed an altered expression of Nrf2/Keap1- Bach1 pathway associated with a reduced expression of anti-oxidants enzymes, like HO-1, in alveolar macrophages from smoking related lung emphysema patients, despite an important oxidative stress. These alterations might be related to cigarette smoke condensate activated ERK1/2 and JNK MAPKinases as observed in THP-1 cells. Furthermore, we showed that HO- 1 system induction was mediated by HIF-1a instead of Nrf2 pathway in alveolar macrophages from smoking related recurrent primary spontaneous pneumothorax. These findings may contribute to a better knowledge of the pathophysiology of lung emphysema and could provide new therapeutic approaches based on preservation and/or restoration of Nrf2/Keap1-Bach1 equilibrium. Our results also suggest that the pathophysiology of primary spontaneous pneumothorax could be different in smokers and non smokers. Spontaneous pneumothorax in smokers is associated with lung oxidative stress and the orchestrated induction of HO-1 probably via HIF-1a. These results provide a new link between oxidative stress and hypoxia/reoxygenation
Książki na temat "Pathologies pulmonaires"
Herbert, Spencer. Pathology of the lung. Wyd. 4. Oxford [Oxfordshire]: Pergamon Press, 1985.
Znajdź pełny tekst źródłaChanussot, Jean-Claude. Kinésithérapie respiratoire : Pathologie pulmonaire. Editions Masson, 1997.
Znajdź pełny tekst źródłaCzęści książek na temat "Pathologies pulmonaires"
Lacombe, P., M. El Hajjam i S. Binsse. "Malformations artérioveineuses pulmonaires: Du diagnostic au traitement". W Thérapeutiques endovasculaires des pathologies veineuses, 277–84. Paris: Springer Paris, 2013. http://dx.doi.org/10.1007/978-2-8178-0291-6_19.
Pełny tekst źródła"Pathologies cardio-pulmonaires". W Pathologies maternelles et grossesse, 223–72. Elsevier, 2014. http://dx.doi.org/10.1016/b978-2-294-71330-9.00009-x.
Pełny tekst źródłaBenachi, Alexandra. "Pathologies pulmonaires et diaphragme". W Conduites pratiques en médecine foetale, 87–100. Elsevier, 2010. http://dx.doi.org/10.1016/b978-2-294-70626-4.00005-9.
Pełny tekst źródłaLaurent, François, Ilyes Benlala i Gaël Dournes. "Pathologie pulmonaire professionnelle". W Imagerie des Pneumopathies Interstitielles Diffuses (PID), 171–82. Elsevier, 2023. http://dx.doi.org/10.1016/b978-2-294-78190-2.00015-7.
Pełny tekst źródła"PATHOLOGIE TUMORALE DU PARENCHYME PULMONAIRE". W Imagerie Thoracique, 277–451. Elsevier, 2013. http://dx.doi.org/10.1016/b978-2-294-71321-7.50014-8.
Pełny tekst źródła"TROISIÈME CHAPITRE La pathologie pulmonaire". W Apprenez à respirer à vos enfants, 23–38. EDP Sciences, 2015. http://dx.doi.org/10.1051/978-2-7598-1897-6.c007.
Pełny tekst źródłaStreszczenia konferencji na temat "Pathologies pulmonaires"
Lafont, J., J. H. Catherine, M. Lejeune, U. Ordioni, R. Lan i F. Campana. "Manifestations buccales de la sclérose tubéreuse de Bourneville". W 66ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2020. http://dx.doi.org/10.1051/sfco/20206603014.
Pełny tekst źródła