Artykuły w czasopismach na temat „Pathological hypertrophy”
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Li, Wei-ming, Yi-fan Zhao, Guo-fu Zhu, Wen-hui Peng, Meng-yun Zhu, Xue-jing Yu, Wei Chen, Da-chun Xu i Ya-wei Xu. "Dual specific phosphatase 12 ameliorates cardiac hypertrophy in response to pressure overload". Clinical Science 131, nr 2 (23.12.2016): 141–54. http://dx.doi.org/10.1042/cs20160664.
Pełny tekst źródłaYu, Qing, Wenxin Kou, Xu Xu, Shunping Zhou, Peipei Luan, Xiaopeng Xu, Hailing Li i in. "FNDC5/Irisin inhibits pathological cardiac hypertrophy". Clinical Science 133, nr 5 (1.03.2019): 611–27. http://dx.doi.org/10.1042/cs20190016.
Pełny tekst źródłaKang, Peter M., Patrick Yue, Zhilin Liu, Oleg Tarnavski, Natalya Bodyak i Seigo Izumo. "Alterations in apoptosis regulatory factors during hypertrophy and heart failure". American Journal of Physiology-Heart and Circulatory Physiology 287, nr 1 (lipiec 2004): H72—H80. http://dx.doi.org/10.1152/ajpheart.00556.2003.
Pełny tekst źródłaLi, Peng-Long, Hui Liu, Guo-Peng Chen, Ling Li, Hong-Jie Shi, Hong-Yu Nie, Zhen Liu i in. "STEAP3 (Six-Transmembrane Epithelial Antigen of Prostate 3) Inhibits Pathological Cardiac Hypertrophy". Hypertension 76, nr 4 (październik 2020): 1219–30. http://dx.doi.org/10.1161/hypertensionaha.120.14752.
Pełny tekst źródłaJACOB, R., M. VOGT i H. RUPP. "Physiological and pathological hypertrophy*". Journal of Molecular and Cellular Cardiology 18 (1986): 35. http://dx.doi.org/10.1016/s0022-2828(86)80135-3.
Pełny tekst źródłaTanaka, M., H. Fujiwara i C. Kawai. "Pathological features of hypertrophic cardiomyopathy without asymmetrical septal hypertrophy." Heart 56, nr 3 (1.09.1986): 294–97. http://dx.doi.org/10.1136/hrt.56.3.294.
Pełny tekst źródłaHu, Chengyun, Feibiao Dai, Jiawu Wang, Lai Jiang, Di Wang, Jie Gao, Jun Huang i in. "Peroxiredoxin-5 Knockdown Accelerates Pressure Overload-Induced Cardiac Hypertrophy in Mice". Oxidative Medicine and Cellular Longevity 2022 (29.01.2022): 1–12. http://dx.doi.org/10.1155/2022/5067544.
Pełny tekst źródłaLu, Dan, Jizheng Wang, Jing Li, Feifei Guan, Xu Zhang, Wei Dong, Ning Liu, Shan Gao i Lianfeng Zhang. "Meox1 accelerates myocardial hypertrophic decompensation through Gata4". Cardiovascular Research 114, nr 2 (16.11.2017): 300–311. http://dx.doi.org/10.1093/cvr/cvx222.
Pełny tekst źródłaGao, Si, Xue-ping Liu, Li-hua Wei, Jing Lu i Peiqing Liu. "Upregulation of α-enolase protects cardiomyocytes from phenylephrine-induced hypertrophy". Canadian Journal of Physiology and Pharmacology 96, nr 4 (kwiecień 2018): 352–58. http://dx.doi.org/10.1139/cjpp-2017-0282.
Pełny tekst źródłaLuckey, Stephen W., Chris D. Haines, John P. Konhilas, Elizabeth D. Luczak, Antke Messmer-Kratzsch i Leslie A. Leinwand. "Cyclin D2 is a critical mediator of exercise-induced cardiac hypertrophy". Experimental Biology and Medicine 242, nr 18 (13.09.2017): 1820–30. http://dx.doi.org/10.1177/1535370217731503.
Pełny tekst źródłaZeitz, Michael J., i James W. Smyth. "Translating Translation to Mechanisms of Cardiac Hypertrophy". Journal of Cardiovascular Development and Disease 7, nr 1 (10.03.2020): 9. http://dx.doi.org/10.3390/jcdd7010009.
Pełny tekst źródłaYuan, Yonggang, Wanzhong Peng, Yongxing Liu i Zesheng Xu. "Palmatine attenuates isoproterenol-induced pathological hypertrophy via selectively inhibiting HDAC2 in rats". International Journal of Immunopathology and Pharmacology 30, nr 4 (22.11.2017): 406–12. http://dx.doi.org/10.1177/0394632017742225.
Pełny tekst źródłaJoseph, Jacob, Lija Joseph, Nawal S. Shekhawat, Sulochana Devi, Junru Wang, Russell B. Melchert, Martin Hauer-Jensen i Richard H. Kennedy. "Hyperhomocysteinemia leads to pathological ventricular hypertrophy in normotensive rats". American Journal of Physiology-Heart and Circulatory Physiology 285, nr 2 (sierpień 2003): H679—H686. http://dx.doi.org/10.1152/ajpheart.00145.2003.
Pełny tekst źródłaZhou, Ning, Shaunrick Stoll i Hongyu Qiu. "VCP represses pathological cardiac hypertrophy". Aging 9, nr 12 (26.12.2017): 2469–70. http://dx.doi.org/10.18632/aging.101357.
Pełny tekst źródłaShimizu, Ippei, i Tohru Minamino. "Physiological and pathological cardiac hypertrophy". Journal of Molecular and Cellular Cardiology 97 (sierpień 2016): 245–62. http://dx.doi.org/10.1016/j.yjmcc.2016.06.001.
Pełny tekst źródłaKavazis, Andreas N. "Pathological vs. physiological cardiac hypertrophy". Journal of Physiology 593, nr 17 (1.09.2015): 3767. http://dx.doi.org/10.1113/jp271161.
Pełny tekst źródłaHarrison, Brooke C., Charles R. Roberts, David B. Hood, Meghan Sweeney, Jody M. Gould, Erik W. Bush i Timothy A. McKinsey. "The CRM1 Nuclear Export Receptor Controls Pathological Cardiac Gene Expression". Molecular and Cellular Biology 24, nr 24 (15.12.2004): 10636–49. http://dx.doi.org/10.1128/mcb.24.24.10636-10649.2004.
Pełny tekst źródłaLiao, Hai-han, Nan Zhang, Yan-yan Meng, Hong Feng, Jing-jing Yang, Wen-jin Li, Si Chen, Hai-ming Wu, Wei Deng i Qi-zhu Tang. "Myricetin Alleviates Pathological Cardiac Hypertrophy via TRAF6/TAK1/MAPK and Nrf2 Signaling Pathway". Oxidative Medicine and Cellular Longevity 2019 (6.12.2019): 1–14. http://dx.doi.org/10.1155/2019/6304058.
Pełny tekst źródłaYang, Jin, Xuhui Feng, Qiong Zhou, Wei Cheng, Ching Shang, Pei Han, Chiou-Hong Lin, Huei-Sheng Vincent Chen, Thomas Quertermous i Ching-Pin Chang. "Pathological Ace2-to-Ace enzyme switch in the stressed heart is transcriptionally controlled by the endothelial Brg1–FoxM1 complex". Proceedings of the National Academy of Sciences 113, nr 38 (6.09.2016): E5628—E5635. http://dx.doi.org/10.1073/pnas.1525078113.
Pełny tekst źródłaBrown, R. Dale, S. Kelly Ambler, Min Li, Timothy M. Sullivan, Lauren N. Henry, Joseph T. Crossno, Carlin S. Long, Timothy P. Garrington i Kurt R. Stenmark. "MAP kinase kinase kinase-2 (MEKK2) regulates hypertrophic remodeling of the right ventricle in hypoxia-induced pulmonary hypertension". American Journal of Physiology-Heart and Circulatory Physiology 304, nr 2 (15.01.2013): H269—H281. http://dx.doi.org/10.1152/ajpheart.00158.2012.
Pełny tekst źródłaBuss, Sebastian J., Johannes H. Riffel, Pratima Malekar, Marco Hagenmueller, Christina Asel, Min Zhang, Celine Weiss, Hugo A. Katus i Stefan E. Hardt. "Chronic Akt blockade aggravates pathological hypertrophy and inhibits physiological hypertrophy". American Journal of Physiology-Heart and Circulatory Physiology 302, nr 2 (styczeń 2012): H420—H430. http://dx.doi.org/10.1152/ajpheart.00211.2011.
Pełny tekst źródłaDeng, Yawen, Zhitong Li, Xiangbo An, Rui Fan, Yao Wang, Jiatian Li, Xiaolei Yang, Jiawei Liao i Yunlong Xia. "Hyperhomocysteinemia Promotes Cardiac Hypertrophy in Hypertension". Oxidative Medicine and Cellular Longevity 2022 (22.08.2022): 1–16. http://dx.doi.org/10.1155/2022/1486157.
Pełny tekst źródłaKong, Sek Won, Natalya Bodyak, Patrick Yue, Zhilin Liu, Jeffrey Brown, Seigo Izumo i Peter M. Kang. "Genetic expression profiles during physiological and pathological cardiac hypertrophy and heart failure in rats". Physiological Genomics 21, nr 1 (21.03.2005): 34–42. http://dx.doi.org/10.1152/physiolgenomics.00226.2004.
Pełny tekst źródłaAgo, Tetsuro, i Junichi Sadoshima. "From Contractile Enhancement to Pathological Hypertrophy". Journal of the American College of Cardiology 66, nr 3 (lipiec 2015): 273–77. http://dx.doi.org/10.1016/j.jacc.2015.05.058.
Pełny tekst źródłaWehbe, Nadine, Suzanne Nasser, Gianfranco Pintus, Adnan Badran, Ali Eid i Elias Baydoun. "MicroRNAs in Cardiac Hypertrophy". International Journal of Molecular Sciences 20, nr 19 (23.09.2019): 4714. http://dx.doi.org/10.3390/ijms20194714.
Pełny tekst źródłaXie, Xin, Hai-Lian Bi, Song Lai, Yun-Long Zhang, Nan Li, Hua-Jun Cao, Ling Han, Hong-Xia Wang i Hui-Hua Li. "The immunoproteasome catalytic β5i subunit regulates cardiac hypertrophy by targeting the autophagy protein ATG5 for degradation". Science Advances 5, nr 5 (maj 2019): eaau0495. http://dx.doi.org/10.1126/sciadv.aau0495.
Pełny tekst źródłaWatson, Peter A., Jane E. B. Reusch, Sylvia A. McCune, Leslie A. Leinwand, Stephen W. Luckey, John P. Konhilas, David A. Brown i in. "Restoration of CREB function is linked to completion and stabilization of adaptive cardiac hypertrophy in response to exercise". American Journal of Physiology-Heart and Circulatory Physiology 293, nr 1 (lipiec 2007): H246—H259. http://dx.doi.org/10.1152/ajpheart.00734.2006.
Pełny tekst źródłaOldfield, Christopher J., Todd A. Duhamel i Naranjan S. Dhalla. "Mechanisms for the transition from physiological to pathological cardiac hypertrophy". Canadian Journal of Physiology and Pharmacology 98, nr 2 (luty 2020): 74–84. http://dx.doi.org/10.1139/cjpp-2019-0566.
Pełny tekst źródłaŢarcă, Elena, Elena Cojocaru, Alina Costina Luca, Laura Mihaela Trandafir, Solange Tamara Roşu, Valentin Munteanu, Viorel Țarcă, Cristian Constantin Budacu i Claudia Florida Costea. "Unusual Case of Masseter Muscle Hypertrophy in Adolescence—Case Report and Literature Overview". Diagnostics 12, nr 2 (16.02.2022): 505. http://dx.doi.org/10.3390/diagnostics12020505.
Pełny tekst źródłaHaines, Christopher D., Pamela A. Harvey i Leslie A. Leinwand. "Estrogens Mediate Cardiac Hypertrophy in a Stimulus-Dependent Manner". Endocrinology 153, nr 9 (1.09.2012): 4480–90. http://dx.doi.org/10.1210/en.2012-1353.
Pełny tekst źródłaQian, Yanxia, Mingming Zhang, Ningtian Zhou, Xiaohan Xu, Jiahui Zhang, Qiang Ding i Junhong Wang. "A long noncoding RNA CHAIR protects the heart from pathological stress". Clinical Science 134, nr 13 (lipiec 2020): 1843–57. http://dx.doi.org/10.1042/cs20200149.
Pełny tekst źródłaChen, Lijuan, Jia Huang, Yanxiao Ji, Xiaojing Zhang, Pixiao Wang, Keqiong Deng, Xi Jiang, Genshan Ma i Hongliang Li. "Tripartite motif 32 prevents pathological cardiac hypertrophy". Clinical Science 130, nr 10 (1.04.2016): 813–28. http://dx.doi.org/10.1042/cs20150619.
Pełny tekst źródłaAbibillaev, Damirbek, i Fuat Kocyigit. "Athletic heart adaptation, pathological hypertrophy and sudden cardiac death". Heart, Vessels and Transplantation 4, Issue 2 (27.05.2020): 55. http://dx.doi.org/10.24969/hvt.2020.199.
Pełny tekst źródłaGrund, Andrea, Malgorzata Szaroszyk, Janina K. Döppner, Mona Malek Mohammadi, Badder Kattih, Mortimer Korf-Klingebiel, Anna Gigina i in. "A gene therapeutic approach to inhibit calcium and integrin binding protein 1 ameliorates maladaptive remodelling in pressure overload". Cardiovascular Research 115, nr 1 (20.06.2018): 71–82. http://dx.doi.org/10.1093/cvr/cvy154.
Pełny tekst źródłaSu, Dongmei, Sun Jing, Lina Guan, Qian Li, Huiling Zhang, Xiaobo Gao i Xu Ma. "Role of Nodal–PITX2C signaling pathway in glucose-induced cardiomyocyte hypertrophy". Biochemistry and Cell Biology 92, nr 3 (czerwiec 2014): 183–90. http://dx.doi.org/10.1139/bcb-2013-0124.
Pełny tekst źródłaBi, Hai-Lian, Xiao-Li Zhang, Yun-Long Zhang, Xin Xie, Yun-Long Xia, Jie Du i Hui-Hua Li. "The deubiquitinase UCHL1 regulates cardiac hypertrophy by stabilizing epidermal growth factor receptor". Science Advances 6, nr 16 (kwiecień 2020): eaax4826. http://dx.doi.org/10.1126/sciadv.aax4826.
Pełny tekst źródłaIemitsu, Motoyuki, Takashi Miyauchi, Seiji Maeda, Satoshi Sakai, Tsutomu Kobayashi, Nobuharu Fujii, Hitoshi Miyazaki, Mitsuo Matsuda i Iwao Yamaguchi. "Physiological and pathological cardiac hypertrophy induce different molecular phenotypes in the rat". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 281, nr 6 (1.12.2001): R2029—R2036. http://dx.doi.org/10.1152/ajpregu.2001.281.6.r2029.
Pełny tekst źródłaGrieco, Teresa, Vito Gomes, Alfredo Rossi, Carmen Cantisani, Maria Elisabetta Greco, Giovanni Rossi, Alvise Sernicola i Giovanni Pellacani. "The Pathological Culprit of Neuropathic Skin Pain in Long COVID-19 Patients: A Case Series". Journal of Clinical Medicine 11, nr 15 (31.07.2022): 4474. http://dx.doi.org/10.3390/jcm11154474.
Pełny tekst źródłaNosenko, N. M., D. V. Shchehlov, M. Yu Mamonova i Ya E. Kudelskyi. "Left ventricular hypertrophy: differential diagnosis". Endovascular Neuroradiology 30, nr 4 (11.03.2020): 49–58. http://dx.doi.org/10.26683/2304-9359-2019-4(30)-49-58.
Pełny tekst źródłaYalçin, Fatih, Nagehan Kucukler, Oscar Cingolani, Blaid Mbiyangandu, Lars Sorensen, Aurelio Pinherio, M. Roselle Abraham i Theodore P. Abraham. "Evolution of ventricular hypertrophy and myocardial mechanics in physiological and pathological hypertrophy". Journal of Applied Physiology 126, nr 2 (1.02.2019): 354–62. http://dx.doi.org/10.1152/japplphysiol.00199.2016.
Pełny tekst źródłaGesmundo, Iacopo, Michele Miragoli, Pierluigi Carullo, Letizia Trovato, Veronica Larcher, Elisa Di Pasquale, Mara Brancaccio i in. "Growth hormone-releasing hormone attenuates cardiac hypertrophy and improves heart function in pressure overload-induced heart failure". Proceedings of the National Academy of Sciences 114, nr 45 (25.10.2017): 12033–38. http://dx.doi.org/10.1073/pnas.1712612114.
Pełny tekst źródłaPeng, Shi, Xiao-feng Lu, Yi-ding Qi, Jing Li, Juan Xu, Tian-you Yuan, Xiao-yu Wu i in. "LCZ696 Ameliorates Oxidative Stress and Pressure Overload-Induced Pathological Cardiac Remodeling by Regulating the Sirt3/MnSOD Pathway". Oxidative Medicine and Cellular Longevity 2020 (18.09.2020): 1–15. http://dx.doi.org/10.1155/2020/9815039.
Pełny tekst źródłaGallo, Simona, Annapia Vitacolonna, Alessandro Bonzano, Paolo Comoglio i Tiziana Crepaldi. "ERK: A Key Player in the Pathophysiology of Cardiac Hypertrophy". International Journal of Molecular Sciences 20, nr 9 (1.05.2019): 2164. http://dx.doi.org/10.3390/ijms20092164.
Pełny tekst źródłaDobrzyn, Pawel, Aleksandra Pyrkowska, Monika K. Duda, Tomasz Bednarski, Michal Maczewski, Jozef Langfort i Agnieszka Dobrzyn. "Expression of lipogenic genes is upregulated in the heart with exercise training-induced but not pressure overload-induced left ventricular hypertrophy". American Journal of Physiology-Endocrinology and Metabolism 304, nr 12 (15.06.2013): E1348—E1358. http://dx.doi.org/10.1152/ajpendo.00603.2012.
Pełny tekst źródłaLi, Haobo, Lena E. Trager, Xiaojun Liu, Margaret H. Hastings, Chunyang Xiao, Justin Guerra, Samantha To i in. "lncExACT1 and DCHS2 Regulate Physiological and Pathological Cardiac Growth". Circulation 145, nr 16 (19.04.2022): 1218–33. http://dx.doi.org/10.1161/circulationaha.121.056850.
Pełny tekst źródłaPiven, O. O. "Signalling function of β-catenin is important at early stages of adult heart pathological hypertrophy". Visnik ukrains'kogo tovaristva genetikiv i selekcioneriv 14, nr 1 (20.06.2016): 44–51. http://dx.doi.org/10.7124/visnyk.utgis.14.1.543.
Pełny tekst źródłaRen, Zongna, Peng Yu, Dandan Li, Zheng Li, Yingnan Liao, Yin Wang, Bingying Zhou i Li Wang. "Single-Cell Reconstruction of Progression Trajectory Reveals Intervention Principles in Pathological Cardiac Hypertrophy". Circulation 141, nr 21 (26.05.2020): 1704–19. http://dx.doi.org/10.1161/circulationaha.119.043053.
Pełny tekst źródłaLiu, Yaoqiu, Yahui Shen, Jingai Zhu, Ming Liu, Xing Li, Yumei Chen, Xiangqing Kong, Guixian Song i Lingmei Qian. "Cardiac-Specific PID1 Overexpression Enhances Pressure Overload-Induced Cardiac Hypertrophy in Mice". Cellular Physiology and Biochemistry 35, nr 5 (2015): 1975–85. http://dx.doi.org/10.1159/000374005.
Pełny tekst źródłaLi, Jin, Zhao Sha, Xiaolan Zhu, Wanru Xu, Weilin Yuan, Tingting Yang, Bing Jin i in. "Targeting miR-30d reverses pathological cardiac hypertrophy". eBioMedicine 81 (lipiec 2022): 104108. http://dx.doi.org/10.1016/j.ebiom.2022.104108.
Pełny tekst źródłaLi, Zhenhua, Jian Wang i Xiao Yang. "Functions of Autophagy in Pathological Cardiac Hypertrophy". International Journal of Biological Sciences 11, nr 6 (2015): 672–78. http://dx.doi.org/10.7150/ijbs.11883.
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