Gotowe bibliografie tematyczne / Pathogen

Gotowa bibliografia na temat „Pathogen”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 25 maja 2024

Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych

Wybierz rodzaj źródła:

Spis treści

Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Pathogen”.

Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.

Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.

Artykuły w czasopismach na temat "Pathogen"

1

Gvozdyak, R. I. "«Pathogen-1» Experiment Aggression of pathogenic bacteria in microgravity". Kosmìčna nauka ì tehnologìâ 6, nr 4 (30.07.2000): 111. http://dx.doi.org/10.15407/knit2000.04.119.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Conceição, Cinara Souza da, Barbara Victor Souza, Jessica Manya Bittencourt Dias Vieira i Janaína dos Santos Nascimento. "Pathogen killing pathogen: antimicrobial substance from Acinetobacter active against foodborne pathogens". Journal of Infection in Developing Countries 12, nr 05 (31.05.2018): 297–304. http://dx.doi.org/10.3855/jidc.9894.

Pełny tekst źródła
Streszczenie:
Introduction: Antimicrobial substances (AMS) produced by bacteria may reduce or prevent the growth of pathogenic and spoilage microorganisms in food. In this study, 16 isolates of Acinetobacter baumannii/calcoaceticus (ABC) complex, previously obtained from reconstituted infant milk formula (IMF) samples and the preparation and distribution utensils from the nursery of a public hospital, were used to screen for AMS production. Methodology: Antimicrobial substance production and spectrum of activity assays were performed by agar-spot assay. Optimization of growth conditions for AMS production was also evaluated. Results: Three (17.6%) isolates, namely JE3, JE4, and JE6, produced AMS against the principal indicator strain Salmonella enterica subsp. enterica serotype Typhi ATCC 19214. JE6 was also able to inhibit strains of Klebsiella pneumoniae, Proteus vulgaris, and Bacillus cereus, a Gram-positive bacteria. Remarkably, JE6 was able to inhibit all the tested resistant and multidrug-resistant (MDR) strains of the ABC complex and Shigella dysenteriae associated with IMF and utensils, indicating a potentially valuable application. AMS produced by JE6 does not appear to be affected by proteolytic enzymes and the producer strain showed specific immunity to its own AMS. Conclusion: This study highlights AMS produced by Acinetobacter with applications against MDR spoilage and foodborne pathogens - some of them, infectious disease causing agents - which, to our knowledge, has not been previously described.
Style APA, Harvard, Vancouver, ISO itp.
3

Sánchez-Vallet, Andrea, Simone Fouché, Isabelle Fudal, Fanny E. Hartmann, Jessica L. Soyer, Aurélien Tellier i Daniel Croll. "The Genome Biology of Effector Gene Evolution in Filamentous Plant Pathogens". Annual Review of Phytopathology 56, nr 1 (25.08.2018): 21–40. http://dx.doi.org/10.1146/annurev-phyto-080516-035303.

Pełny tekst źródła
Streszczenie:
Filamentous pathogens, including fungi and oomycetes, pose major threats to global food security. Crop pathogens cause damage by secreting effectors that manipulate the host to the pathogen's advantage. Genes encoding such effectors are among the most rapidly evolving genes in pathogen genomes. Here, we review how the major characteristics of the emergence, function, and regulation of effector genes are tightly linked to the genomic compartments where these genes are located in pathogen genomes. The presence of repetitive elements in these compartments is associated with elevated rates of point mutations and sequence rearrangements with a major impact on effector diversification. The expression of many effectors converges on an epigenetic control mediated by the presence of repetitive elements. Population genomics analyses showed that rapidly evolving pathogens show high rates of turnover at effector loci and display a mosaic in effector presence-absence polymorphism among strains. We conclude that effective pathogen containment strategies require a thorough understanding of the effector genome biology and the pathogen's potential for rapid adaptation.
Style APA, Harvard, Vancouver, ISO itp.
4

Cunze, Sarah, Judith Kochmann, Lisa K. Koch, Korbinian J. Q. Hasselmann i Sven Klimpel. "Leishmaniasis in Eurasia and Africa: geographical distribution of vector species and pathogens". Royal Society Open Science 6, nr 5 (maj 2019): 190334. http://dx.doi.org/10.1098/rsos.190334.

Pełny tekst źródła
Streszczenie:
Leishmaniasis is a vector-borne disease with a broad global occurrence and an increasing number of recorded cases; however, it is still one of the world's most neglected diseases. We here provide climatic suitability maps generated by means of an ecological niche modelling approach for 32 Phlebotomus vector species with proven or suspected vector competence for five Leishmania pathogens occurring in Eurasia and Africa. A GIS-based spatial overlay analysis was then used to compare the distributional patterns of vectors and pathogens to help evaluate the vector species–pathogen relationship currently found in the literature. Based on this single factor of vector incrimination, that is, co-occurrence of both vector and pathogen, most of the pathogens occurred with at least one of the associated vector species. In the case of L. donovani , only a not yet confirmed vector species, P. rodhaini, could explain the occurrence of the pathogen in regions of Africa. Phlebotomus alexandri and P. longiductus on the other hand, proven vector species of L. donovani, do not seem to qualify as vectors for the pathogen. Their distribution is restricted to northern latitudes and does not match the pathogen's distribution, which lies in southern latitudes. Other more locally confined mismatches were discussed for each pathogen species. The comparative geographical GIS-overlay of vector species and pathogens functions as a first indication that testing and re-evaluation of some pathogen–vector relationships might be worthwhile to improve risk assessments of leishmaniasis.
Style APA, Harvard, Vancouver, ISO itp.
5

Mazarei, Mitra, Irina Teplova, M. Hajimorad i C. Stewart. "Pathogen Phytosensing: Plants to Report Plant Pathogens". Sensors 8, nr 4 (14.04.2008): 2628–41. http://dx.doi.org/10.3390/s8042628.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Lepper, Simone, i Sylvia Münter. "Spotlight on pathogens: ‘Imaging Host-Pathogen Interactions’". Cellular Microbiology 11, nr 6 (czerwiec 2009): 855–62. http://dx.doi.org/10.1111/j.1462-5822.2009.01321.x.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Fonville, Judith M. "Expected Effect of Deleterious Mutations on Within-Host Adaptation of Pathogens". Journal of Virology 89, nr 18 (24.06.2015): 9242–51. http://dx.doi.org/10.1128/jvi.00832-15.

Pełny tekst źródła
Streszczenie:
ABSTRACTAdaptation is a common theme in both pathogen emergence, for example, in zoonotic cross-species transmission, and pathogen control, where adaptation might limit the effect of the immune response and antiviral treatment. When such evolution requires deleterious intermediate mutations, fitness ridges and valleys arise in the pathogen's fitness landscape. The effect of deleterious intermediate mutations on within-host pathogen adaptation is examined with deterministic calculations, appropriate for pathogens replicating in large populations with high error rates. The effect of deleterious intermediate mutations on pathogen adaptation is smaller than their name might suggest: when two mutations are required and each individual single mutation is fully deleterious, the pathogen can jump across the fitness valley by obtaining two mutations at once, leading to a proportion of adapted mutants that is 20-fold lower than that in the situation where the fitness of all mutants is neutral. The negative effects of deleterious intermediates are typically substantially smaller and outweighed by the fitness advantages of the adapted mutant. Moreover, requiring a specific mutation order has a substantially smaller effect on pathogen adaptation than the effect of all intermediates being deleterious. These results can be rationalized when the number of routes of mutation available to the pathogen is calculated, providing a simple approach to estimate the effect of deleterious mutations. The calculations discussed here are applicable when the effect of deleterious mutations on the within-host adaptation of pathogens is assessed, for example, in the context of zoonotic emergence, antigenic escape, and drug resistance.IMPORTANCEAdaptation is critical for pathogens after zoonotic transmission into a new host species or to achieve antigenic immune escape and drug resistance. Using a deterministic approach, the effects of deleterious intermediate mutations on pathogen adaptation were calculated while avoiding commonly made simplifications that do not apply to large pathogen populations replicating with high mutation rates. Perhaps unexpectedly, pathogen adaptation does not halt when the intermediate mutations are fully deleterious. The negative effects of deleterious mutations are substantially outweighed by the fitness gains of adaptation. To gain an understanding of the effect of deleterious mutations on pathogen adaptation, a simple approach that counts the number of routes available to the pathogen with and without deleterious intermediate mutations is introduced. This methodology enables a straightforward calculation of the proportion of the pathogen population that will cross a fitness valley or traverse a fitness ridge, without reverting to more complicated mathematical models.
Style APA, Harvard, Vancouver, ISO itp.
8

Singer, Merrill. "Pathogen-pathogen interaction". Virulence 1, nr 1 (styczeń 2010): 10–18. http://dx.doi.org/10.4161/viru.1.1.9933.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

Chen, Huanhuan, Guangshuo Zhang, Guiling Ding, Jiaxing Huang, Hong Zhang, Mayra C. Vidal, Richard T. Corlett, Cong Liu i Jiandong An. "Interspecific Host Variation and Biotic Interactions Drive Pathogen Community Assembly in Chinese Bumblebees". Insects 14, nr 11 (17.11.2023): 887. http://dx.doi.org/10.3390/insects14110887.

Pełny tekst źródła
Streszczenie:
Bumblebees have been considered one of the most important pollinators on the planet. However, recent reports of bumblebee decline have raised concern about a significant threat to ecosystem stability. Infectious diseases caused by multiple pathogen infections have been increasingly recognized as an important mechanism behind this decline worldwide. Understanding the determining factors that influence the assembly and composition of pathogen communities among bumblebees can provide important implications for predicting infectious disease dynamics and making effective conservation policies. Here, we study the relative importance of biotic interactions versus interspecific host resistance in shaping the pathogen community composition of bumblebees in China. We first conducted a comprehensive survey of 13 pathogens from 22 bumblebee species across China. We then applied joint species distribution modeling to assess the determinants of pathogen community composition and examine the presence and strength of pathogen–pathogen associations. We found that host species explained most of the variations in pathogen occurrences and composition, suggesting that host specificity was the most important variable in predicting pathogen occurrences and community composition in bumblebees. Moreover, we detected both positive and negative associations among pathogens, indicating the role of competition and facilitation among pathogens in determining pathogen community assembly. Our research demonstrates the power of a pluralistic framework integrating field survey of bumblebee pathogens with community ecology frameworks to understand the underlying mechanisms of pathogen community assembly.
Style APA, Harvard, Vancouver, ISO itp.
10

Lin, Borong, Xue Qing, Jinling Liao i Kan Zhuo. "Role of Protein Glycosylation in Host-Pathogen Interaction". Cells 9, nr 4 (20.04.2020): 1022. http://dx.doi.org/10.3390/cells9041022.

Pełny tekst źródła
Streszczenie:
Host-pathogen interactions are fundamental to our understanding of infectious diseases. Protein glycosylation is one kind of common post-translational modification, forming glycoproteins and modulating numerous important biological processes. It also occurs in host-pathogen interaction, affecting host resistance or pathogen virulence often because glycans regulate protein conformation, activity, and stability, etc. This review summarizes various roles of different glycoproteins during the interaction, which include: host glycoproteins prevent pathogens as barriers; pathogen glycoproteins promote pathogens to attack host proteins as weapons; pathogens glycosylate proteins of the host to enhance virulence; and hosts sense pathogen glycoproteins to induce resistance. In addition, this review also intends to summarize the roles of lectin (a class of protein entangled with glycoprotein) in host-pathogen interactions, including bacterial adhesins, viral lectins or host lectins. Although these studies show the importance of protein glycosylation in host-pathogen interaction, much remains to be discovered about the interaction mechanism.
Style APA, Harvard, Vancouver, ISO itp.
Więcej źródeł

Rozprawy doktorskie na temat "Pathogen"

1

Panagoda, Gehan Joseph. "Pathogenic features of Candida parapsilosis : an emerging fungal pathogen /". Thesis, Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B20377770.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Hovhannisyan, Hrant 1992. "Comparative transcriptomics of host-pathogen interactions and hybridization in Candida pathogens". Doctoral thesis, Universitat Pompeu Fabra, 2020. http://hdl.handle.net/10803/670316.

Pełny tekst źródła
Streszczenie:
Candida pathogenic yeasts represent a global healthcare problem. They comprise phylogenetically diverse species, including newly emerged pathogens. How human-Candida interactions vary across species, and what processes underlie the emergence of novel pathogens are poorly understood. Current thesis addresses these issues using comparative transcriptomics and bioinformatics. We established the global patterns of host-pathogen interactions between human host and the main Candida species, providing novel mechanistic insights into their interplay. We also explored lncRNAs of these pathogens, assessing their implications in infection. Further, we designed and validated a pan-Candida RNA enrichment approach, opening new possibilities for studying host-pathogen interactions in vivo. Then, we assessed the impact of hybridization on transcriptomes of hybrid yeasts, exploring the links between hybridization and virulence emergence. We also developed a new bioinformatics tool facilitating the research in the field. Altogether, results of this thesis expand our knowledge on relevant aspects of human-Candida interactions and yeast evolution.
Las levaduras patógenas Candida representan un problema de salud global. Este grupo de levaduras, comprenden especies filogenéticamente diversas, e incluye patógenos emergidos recientemente. La forma en que las interacciones entre humanos y Candida varían de una especie a otra y qué procesos subyacen a la aparición de nuevos patógenos son poco conocidos. La tesis actual aborda estos problemas utilizando una aproximación de transcriptómica comparativa y bioinformática. Establecimos los patrones globales de las interacciones huésped-patógeno entre el huésped humano y las principales especies de Candida, proporcionando nuevas ideas mecanicistas sobre su interacción. También exploramos los lncRNA de estos patógenos, evaluando sus implicaciones en la infección. Además, diseñamos y validamos un enfoque de enriquecimiento de ARN pan-Candida, abriendo nuevas posibilidades para estudiar las interacciones huésped-patógeno in vivo. Luego, evaluamos el impacto de la hibridación en los transcriptomas de levaduras híbridas, explorando los vínculos entre la hibridación y la aparición de virulencia. En su conjunto, los resultados de esta tesis amplían nuestro conocimiento sobre aspectos relevantes de las interacciones humano-Candida y la evolución de las levaduras.
Style APA, Harvard, Vancouver, ISO itp.
3

Gibson, Josie. "In vivo imaging and analysis of host-pathogen interactions of intracellular pathogens". Thesis, University of Sheffield, 2017. http://etheses.whiterose.ac.uk/19047/.

Pełny tekst źródła
Streszczenie:
Cellular and extracellular host-pathogen interactions are important in the progression of infection. Extracellular survival and growth can be significant for pathogen dissemination. Equally, intracellular pathways, such as autophagy, can be employed in host cell defence. Autophagy is a cellular self-degradation process that recycles cellular components through lysosomal degradation. Primarily for regulating starvation and housekeeping pathways, autophagy is also important for degradation of invading intracellular pathogens. Selective autophagy receptors can also target pathogens for autophagic degradation. However, some intracellular pathogens are able to subvert or block host cell autophagy. Cryptococcus neoformans and Staphylococcus aureus represent fungal and bacterial pathogens which can reside either intracellularly or extracellularly. The role of host cell autophagy in C. neoformans and S. aureus infection is unclear. Infection of zebrafish with C. neoformans or S. aureus enabled in vivo imaging of host-pathogen interactions, to examine infection growth dynamics and dissemination, in addition to cellular level imaging of pathogen interactions with host cell autophagy components. Cryptococcal infection can cause cryptococcal meningitis, frequently associated with cerebral infarcts. Analysis of cryptococcal proliferation during infection suggested that formation of intravascular cryptococcal masses precedes invasion of surrounding tissue. Vessel integrity analysis highlighted cryptococcal-mediated vascular damage, potentially providing a route for tissue dissemination. Vasculature damage may explain the origin of cortical infarcts in disease. Autophagy mutant zebrafish were generated to analyse host autophagy in pathogen infection. Characterisation of mutant larvae revealed a clear survival and growth defect, indicating autophagy is required for larval development. Autophagy mutants were subsequently used to analyse the role of autophagy during infection through analysis of pathogenic burden and pathogen association with autophagosome marker LC3-II. LC3II recruitment to pathogens was reduced in autophagy mutant neutrophils. Additionally, selective autophagy receptor p62 was recruited to S. aureus and C. neoformans within neutrophils, highlighting the involvement of host cell autophagy during infection.
Style APA, Harvard, Vancouver, ISO itp.
4

Yan, Ling. "Phagocyte-pathogen interactions". [Lincoln, Neb. : University of Nebraska-Lincoln], 2004. http://www.unl.edu/libr/Dissertations/2004/YanDis.pdf.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Feldmann, Friederike. "Implication of extraintestinal pathogenic Escherichia coli siderophore receptors in host pathogen interaction". kostenfrei, 2008. http://mediatum2.ub.tum.de/doc/649951/649951.pdf.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Kärkkäinen, Riikka M. "Production of DNA aptamers with specificity for bacterial food pathogens". Thesis, University of Chester, 2012. http://hdl.handle.net/10034/620695.

Pełny tekst źródła
Streszczenie:
Aptamers are biomolecular ligands composed of nucleic acids. They can be selected to bind specifically to a range of target molecules and subsequently exploited in a fashion analogous to more traditional biomolecules such as antibodies. In this study a method for selecting new aptamers which specifically bind whole live bacterial cells is described. A non-pathogenic strain of Escherichia coli K12 was used to develop the method. A DNA library containing 100 bases long random nucleotide sequences was produced and the aptamer selection process was repeated nine times. An enzyme-linked technique was first used to detect bound aptamers thereafter fluorimetry and fluorescence microscopy methods were used for the detection. The aptamers were cloned and sequenced and the cloned aptamers produced with fluorescent labels. The E. coli K12-binding aptamers were used to demonstrate the detection of the bacterial cells in a complex food matrix, namely probiotic yogurt, by using fluorescence based detection method. The aptamer selection method with some modifications was also used to select aptamers with specificity for the food pathogens E. coli O157, Listeria monocytogenes, L. innocua, S. typhimurium and S. enteritidis. The aptamers against E. coli O157 and S. typhimurium were cloned and the sequences and the binding properties of these aptamers were analysed. The use of E. coli K12 as a target organism and the aptamer sequences presented in this study, have not previously been published in scientific literature. This is also the first report where the aptamers have been used in detection of live bacterial cells in yogurt.
Style APA, Harvard, Vancouver, ISO itp.
7

Ahmad, Sarah. "Identification of pathogen-specific protein-encoding genes from microbial pathogens based on bioinformatic analysis". Thesis, University of Exeter, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425246.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Almiman, Bandar F. "Molecular genetic and genomic characterization of an emerging mycotoxigenic pathogen Fusarium proliferatum". Thesis, University of Bedfordshire, 2018. http://hdl.handle.net/10547/622835.

Pełny tekst źródła
Streszczenie:
This aim of this research was to elucidate the genotypic diversity of the mycotoxigenic species Fusarium proliferatum associated with diverse hosts and distributed in wide geographic locations to gain new insights into the biology of this emerging pathogen. This study developed a novel molecular genetic marker FG1056. Multilocus typing of F. proliferatum isolates (52) using F. verticillioides (2) and F. oxysporum (3) as references was carried out with FG1056 and a set of known genetic markers (ITS, TEF1, CAL and FUM1). This distinguished up to 10 genetic groups, 2 clusters and 23 haplotypes among the F. proliferatum isolates. FG1056 marker showed the highest number of SNPs (169), informative sites (89) and haplotypes (23) relative to other markers used and was comparable to the multi locus typing. Varying patterns of relationships were observed between isolates represented in the genetic groups and their host and geographic origin. Considerable biological variability was recorded among the F. proliferatum isolates in morphology, growth, sporulation and most notably fumonisin production (up to 140-fold differences) with reference to variable temperature, water activity and duration. De novo genome assemblies with the size ranging from 43.96 - 50 Mb have been developed for four diverse F. proliferatum isolates. In silico analysis led to the identification of 12,980 genes common to all isolates and up to 134 genes potentially unique to an isolate. Using these resources, FUM gene cluster (~45.3 Kb) was identified for the first time in F. proliferatum. Order and orientation of the 16 FUM genes and the complete flanking genes (MSF1 and ZCB1 at 5’; ANK1 and GAT1 at 3’) have been determined. This study has provided new insights into the genetic and biological diversity of F. proliferatum and also developed new genetic and genomic resources, which will serve as a solid platform for further research particularly to understand the regulation of fumonisins production in the laboratory and in the field.
Style APA, Harvard, Vancouver, ISO itp.
9

Berriri, Souha. "Identification of constitutively active forms of Arabidopsis MAP Kinases : brings more evidence on MPK4 function in plant immunity". Thesis, Evry-Val d'Essonne, 2012. http://www.theses.fr/2011EVRY0024.

Pełny tekst źródła
Streszczenie:
La phosphorylation/déphosphorylation des protéines est un mécanisme de signalisation intracellulaire commun. Parmi les kinases végétales, les Mitogen-Activated Protein Kinases (MAPKs) sont impliquées dans de nombreux processus biologiques importants, comme la réponse aux stress biotiques et abiotiques, le développement et la dynamique du cytosquelette. Chez Arabidopsis thaliana et ce malgré de nombreux efforts, les fonctions des kinases impliquées dans les cascades MAPK restent peu inconnues. L'activation des kinases en utilisant des mutations mimant la phosphorylation des sites normalement phosphorylés est une approchequi a fait ses preuves dans le cas de MAP2Ks et a largement contribué à élucider leurs fonctions. Cette stratégie s’est révélée impossible dans le cas des MAPKs, puisque les résidus à muter restent encore à identifier. Pour contourner ce problème, nous avons adapté un crible basé sur la complémentation fonctionnelle d’un mutant MAPK de levure avec des formes aléatoirement mutées de MPK6d’Arabidopsis dans le but d'identifier des mutants présentant une activité constitutive. Nous en avons identifiés plusieurs et avons montré que ces formes constitutivement actives (CA) de MPK6 sont actives sans phosphorylation par les MAP2Ks. Par ailleurs, les mutations des résidus équivalents dans d'autres MAPKs les rendent également hyperactives, ce qui indique que cette stratégie peut être utilisée comme approche générale pour activer les MAPKs afin d’en comprendre les fonctions. L’étude des interactions protéine-protéine et l’analyse des profils dephosphorylation indiquent que les MAPKs CA conservent leur spécificité envers leurs substrats et interacteurs. Comme preuve de concept, nous avons généré des formes actives du MPK4. La MPK4 CA exprimée sous son propre promoteur a parfaitement complémenté le mutant mpk4. La caractérisation des lignées exprimant MPK4 CA confirme le rôle négatif de cette kinase dans les réponses de défense aux pathogènes des plantes que ce soit dans la PTI (PAMP Triggered Immunity) ou dans la ETI (Effector Triggered Immunity). Globalement, ce travail permettra de fournir des informations directes sur les cibles des MAPKs et devrait contribuer à la compréhension globale de la transduction du signal chez les plantes
Protein phosphorylations and dephosphorylations are common events occurring duringintracellular signaling processes. Among plant kinases, Mitogen-Activated Protein Kinases (MAPKs) are involved in signaling of many important biological processes, including biotic and abiotic stresses, development and cytoskeleton organization. Despite an abundant literature on MAPKs, the exact roles and direct targets of many Arabidopsis thaliana MAPKs are not clear yet. The activation of kinases using phospho-mimicking mutations of the phosphorylated residues was a successful approach in the case of MAP2Ks, helping to elucidate their functions. This strategy failed in the case of MAPKs since the necessary residues to mutate remain unclear. To bypass this problem, we adapted a screen based on the functional complementation of a MAPK yeast mutant with randomly mutated Arabidopsis MPK6 in order to identify the ones mutants showing constitutive activity. We identified several clones and showed that these constitutively active (CA) of MPK6 candidates are indeed active without phosphorylation by MAP2Ks. Interestingly, mutations of the equivalent residues in other MAPKs triggered constitutive activity as well, indicating that this strategy may be used as a general approach to activate MAPKs and identify their functions. Interaction and phosphorylation assays indicatedthat CA MAPKs retain their substrate and interactor specificity. As proof-of-concept, we generated active versions of MPK4. CA MPK4 expressed under itsown promoter successfully complements mpk4 mutant plants. Characterization of CA MPK4 lines further confirmed the negative role of MPK4 in plant pathogen defense responses and its implication in both PTI (PAMP Triggered Immunity) and ETI (Effector Triggered Immunity). Overall, the work will help to provide direct information on all MAPK targets and should be an important contribution to the overall understanding of signal transduction in plants
Style APA, Harvard, Vancouver, ISO itp.
10

Su, Fan. "Modifications physiologiques induites par Burkholderia phytofirmans chez Arabidopsis thaliana. Applications à la protection contre les stress biotique et abiotique". Thesis, Reims, 2015. http://www.theses.fr/2015REIMS032.

Pełny tekst źródła
Streszczenie:
La PGPR Burkholderia phytofirmans PsJN (Bp) stimule la croissance de diverses plantes et les protège également contre certains stress environnementaux. L’objectif des travaux a été d’approfondir les connaissances sur l’interaction Bp-plante, en se focalisant sur l’aspect physiologique et métabolique des feuilles d’Arabidopsis thaliana. Nous avons également déterminé les mécanismes impliqués dans la réponse des feuilles suite à l’inoculation de cette bactérie lors d’un stress abiotique (froid) ou biotique (Pseudomonas syringae pv. tomato DC3000, Pst).Nos résultats montrent que l’induction de la promotion de croissance d’A. thaliana par Bp pourrait être liée à l’accumulation des teneurs en métabolites primaires (acides aminés, glucides solubles et vitamines) et la variation du niveau des hormones dans les feuilles. La physiologie et le métabolisme des feuilles sont modifiés localement et de façon distale par la colonisation épi- et endophytique de Bp. De plus, les modifications des taux de métabolites sont plus marquées après une interaction plante-bactéries relativement longue. Par ailleurs, l’inoculation de Bp peut réduire les dommages sur l’activité photosynthétique dus au froid par une limitation non-stomatique de la photosynthèse et l’accumulation de pigments photosynthétiques. Enfin, la présence de Bp entraîne localement un retard dans le développement initial de Pst. Cependant, l’inoculation par Bp ne protège pas l’appareil photosynthétique lors d’une attaque par Pst. Ces travaux soulignent donc que le temps de présence et la localisation d’une PGPR dans une plante influencent la physiologie, le métabolisme et la tolérance aux stress de cette même plante
Endophytic PGPR Burkholderia phytofirmans PsJN (Bp) promotes growth of various plants and triggers protection against several environmental stresses. To get more insights into the interaction between plant and Bp, we focused on leaf physiological and metabolic aspects of Arabidopsis thaliana. We also determined the mechanisms involved in the defense of leaves after inoculation of the bacteria followed by an abiotic (cold) or a biotic (Pseudomonas syringae pv. tomato DC3000, Pst) stress. Our results show that the induction of growth promotion of A. thaliana by Bp could be related to the accumulation of primary metabolite levels (amino acids, soluble carbohydrates and vitamins) and to the variation of hormone levels in the leaves. Leaf physiology and metabolism are changed locally and distally by Bp epi- and endophytic colonization. In addition, changes in metabolite levels are more pronounced after a relatively long interaction between plant and bacteria.Moreover, Bp inoculation can also reduce cold injury on the photosynthetic activity by a non-stomatal limitation of photosynthesis and accumulation of photosynthetic pigments. Finally, the local presence of Bp causes a delay in the development of Pst, but only in the early stages of the infection. However, the inoculation with Bp does not protect the photosynthetic apparatus during Pst attack.Thus, our results emphasize that the time of presence of a PGPR and his location in the plant could influence the plant physiology and stress tolerance
Style APA, Harvard, Vancouver, ISO itp.
Więcej źródeł

Książki na temat "Pathogen"

1

Shaw, Karen Joy, red. Pathogen Genomics. Totowa, NJ: Humana Press, 2002. http://dx.doi.org/10.1007/978-1-59259-172-5.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Pamela, Ronald C. Plant-Pathogen Interactions. New Jersey: Humana Press, 2006. http://dx.doi.org/10.1385/1592599664.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
3

Russell, David G., i Siamon Gordon, red. Phagocyte-Pathogen Interactions. Washington, DC, USA: ASM Press, 2009. http://dx.doi.org/10.1128/9781555816650.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
4

Birch, Paul, John T. Jones i Jorunn I. B. Bos, red. Plant-Pathogen Interactions. Totowa, NJ: Humana Press, 2014. http://dx.doi.org/10.1007/978-1-62703-986-4.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Unden, Gottfried, Eckhard Thines i Anja Schüffler, red. Host - Pathogen Interaction. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2016. http://dx.doi.org/10.1002/9783527682386.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Rupp, Steffen, i Kai Sohn, red. Host-Pathogen Interactions. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-204-5.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Medina, Carlos, i Francisco Javier López-Baena, red. Host-Pathogen Interactions. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7604-1.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Nick, Talbot, red. Plant-pathogen interactions. Oxford: Blackwell Pub, 2004.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

1943-, Zwilling Bruce S., i Eisenstein Toby K, red. Macrophage-pathogen interactions. New York: M. Dekker, 1994.

Znajdź pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
10

Medina, Carlos, i Francisco Javier López-Baena, red. Host-Pathogen Interactions. New York, NY: Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3617-6.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
Więcej źródeł

Części książek na temat "Pathogen"

1

Gooch, Jan W. "Pathogen, Pathogene". W Encyclopedic Dictionary of Polymers, 520. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_8468.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Frank, J. Howard, J. Howard Frank, Michael C. Thomas, Allan A. Yousten, F. William Howard, Robin M. Giblin-davis, John B. Heppner i in. "Pathogen". W Encyclopedia of Entomology, 2758–59. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_2798.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
3

Bährle-Rapp, Marina. "pathogen". W Springer Lexikon Kosmetik und Körperpflege, 403. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_7473.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
4

Nahler, Gerhard. "pathogen". W Dictionary of Pharmaceutical Medicine, 131. Vienna: Springer Vienna, 2009. http://dx.doi.org/10.1007/978-3-211-89836-9_1006.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Singh Saharan, Govind, Naresh Mehta i Prabhu Dayal Meena. "Pathogen". W Alternaria Diseases of Crucifers: Biology, Ecology and Disease Management, 53–86. Singapore: Springer Singapore, 2015. http://dx.doi.org/10.1007/978-981-10-0021-8_3.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Baker, Julien S., Fergal Grace, Lon Kilgore, David J. Smith, Stephen R. Norris, Andrew W. Gardner, Robert Ringseis i in. "Pathogen". W Encyclopedia of Exercise Medicine in Health and Disease, 690. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2840.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Gooch, Jan W. "Pathogen". W Encyclopedic Dictionary of Polymers, 913. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14437.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Tudor, Jacqueline Hill. "Pathogen". W Encyclopedia of Animal Cognition and Behavior, 1–4. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-47829-6_1649-1.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

Tudor, Jacqueline Hill. "Pathogen". W Encyclopedia of Animal Cognition and Behavior, 5087–91. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-319-55065-7_1649.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
10

Kendig, Amy E., S. Luke Flory, Erica M. Goss, Robert D. Holt, Keith Clay, Philip F. Harmon, Brett R. Lane, Ashish Adhikari i Christopher M. Wojan. "The role of pathogens in plant invasions." W Plant invasions: the role of biotic interactions, 208–25. Wallingford: CABI, 2020. http://dx.doi.org/10.1079/9781789242171.0208.

Pełny tekst źródła
Streszczenie:
Abstract Plant-pathogen interactions occur throughout the process of plant invasion: pathogens can acutely influence plant survival and reproduction, while the large densities and spatial distributions of invasive plant species can influence pathogen communities. However, interactions between invasive plants and pathogens are often overlooked during the early stages of invasion. As with introductions of invasive plants, the introduction of agricultural crops to new areas can also generate novel host-pathogen interactions. The close monitoring of agricultural plants and resulting insights can inform hypotheses for invasive plants where research on pathogen interactions is lacking. This chapter reviews the known and hypothesized effects of pathogens on the invasion process and the effects of plant invasion on pathogens and infectious disease dynamics throughout the process of invasion. Initially, pathogens may inhibit the transport of potentially invasive plants. After arrival in a new range, pathogens can facilitate or inhibit establishment success of introduced plants depending on their relative impacts on the introduced plants and resident species. As invasive plants spread, they may encounter novel pathogens and alter the abundance and geographic range of pathogens. Pathogens can mediate interactions between invasive plants and resident species and may influence the long-term impacts of invasive plants on ecosystems. As invasive plants shift the composition of pathogen communities, resident species could be subject to higher disease risk. We highlight gaps in invasion biology research by providing examples from the agricultural literature and propose topics that have received little attention from either field.
Style APA, Harvard, Vancouver, ISO itp.

Streszczenia konferencji na temat "Pathogen"

1

Crucean, Stefan, Tatiana Scerbacova i Leonid Volosciuc. "The use of Trichoderma species against the main mycotic pathogens of walnut". W Scientific International Symposium "Plant Protection – Achievements and Perspectives". Institute of Genetics, Physiology and Plant Protection, Republic of Moldova, 2023. http://dx.doi.org/10.53040/ppap2023.20.

Pełny tekst źródła
Streszczenie:
Walnut plantations suffer from the impact caused by a number of pathogenic mycotic microorganisms. The most common pathogen fungus is Alternaria alternata and Fusarium poae. In the framework of the research, we aimed to determine the antagonistic activity of the strain Trichoderma harzianum CNMN - FD - 16 against the identified pathogens. Thus, on the 10th day the inhibition of the pathogen A. alternata by T. harzianum is 74%. On the same day, the inhibition of F. poae by T. harzianum is 73%. Trichoderma harzianum suppressed the colonies of A. alternata of 83% - this means 4 points, and the suppression of the colonies of F. poae was 69% - registering 3 points.
Style APA, Harvard, Vancouver, ISO itp.
2

Tay, Li-Lin, Ping-Ji Huang i Lai-Kwan Chau. "Plasmonic Sensors for Pathogen Detection". W Applied Industrial Spectroscopy. Washington, D.C.: Optica Publishing Group, 2022. http://dx.doi.org/10.1364/ais.2022.jm2e.5.

Pełny tekst źródła
Streszczenie:
Plasmonic surface enhanced Raman scattering (SERS) probes were synthesized with different Raman reporter molecules and conjugated with pathogen specific antibodies to provide a rapid screening platform for the detection of pathogenic bacterium, S. aureus and S. Typhimurium.
Style APA, Harvard, Vancouver, ISO itp.
3

Roberts, Anthony, Richard McClatchey, Saad Liaquat, Nigel Edwards i Mike Wray. "POSTER: Introducing pathogen". W the 2013 ACM SIGSAC conference. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2508859.2512518.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
4

Prešern, Andreja. "Pathogen-Plant Interactions in Plant Membrane Perforation". W Socratic Lectures 8. University of Lubljana Press, 2023. http://dx.doi.org/10.55295/psl.2023.ii14.

Pełny tekst źródła
Streszczenie:
Plants are targets of many pathogens that produce a lot of different effectors to damage plant cells during infection. For plant survival, it is, therefore, crucial to possess an effi-cient immune system, which in contrast to mammalian immunity, consists only of in-nate immunity. Traditionally, plant immunity is divided into two branches, i.e. pat-tern-triggered (PTI) and effector-triggered immunity (ETI), but the accumulating knowledge has shown that the division cannot be strictly maintained. ETI coevolves with pathogen e fector molecules, which can function in many different ways to escape plant immunity and damage plant cells. Among their targets is the plant plasma membrane, which repre-sents an important cell barrier. There are several different tactics to bypass this barrier, e.g. membrane perforation by proteins or peptides, which is an important and ubiqui-tously found mechanism of toxicity as well as self-defense in all kingdoms of life. It can be used to get specific molecules from cells, for signaling, or even to deliver effector mol-ecules into the cytoplasm. The exact knowledge on plant membrane perforation, howev-er, is lacking, and the hidden details still await to be unveiled. Keywords: Plant immune System; Pathogen-Plant Interactions; Plant Membrane Perfora-tion
Style APA, Harvard, Vancouver, ISO itp.
5

Saaski, Elric W. "BioHawk portable pathogen detector". W BiOS, redaktor Philippe M. Fauchet. SPIE, 2009. http://dx.doi.org/10.1117/12.811062.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

BOWN, K. J., J. A. ARCHARD, A. J. CORBITT, R. L. LESLIE, M. J. MURPHY i D. J. SQUIRRELL. "RAPID FOODBORNE PATHOGEN DETECTION". W Bioluminescence and Chemiluminescence - Progress and Current Applications - 12th International Symposium on Bioluminescence (BL) and Chemiluminescence (CL). WORLD SCIENTIFIC, 2002. http://dx.doi.org/10.1142/9789812776624_0079.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Carpi, Giovanna. "The role of pathogen genomics in assessing tick-borne pathogen evolutionary dynamics". W 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.105334.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

Russo, Ashlee N., Mark D. Wewers, Ruairi Fahy, Jennifer Hollyfield i Matthew C. Exline. "Severity Of Illness, Not Pathogen Determines Suppression Of Pathogen Sensors In Sepsis". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5761.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

Pershin, L., M. Ringuette, M. Sain i J. Mostaghimi. "Copper-Embedded Facemasks for the Destruction of Covid-19 and Other Pathogens". W ITSC2022. DVS Media GmbH, 2022. http://dx.doi.org/10.31399/asm.cp.itsc2022p0489.

Pełny tekst źródła
Streszczenie:
Abstract Viruses and microbial pathogens can survive for hours on fabrics. This paper reports that copper-doping of natural and synthetic fabrics inactivates, within minutes, a human COVID surrogate pathogen. The fabric is embedded with copper particles by twin-wire arc thermal spray. The long-lasting fabric surface simultaneously provides good breathability, it is scalable and cost-effective. Virucidal activity is not affected by repeated washing of the fabric. Importantly, copper-embedded material will provide effective protection against all classes of pathogens, regardless of their mutation rates and infection strategies. It also can provide protection against all classes of pathogens, regardless of their mutation rates in industrial and residential filters.
Style APA, Harvard, Vancouver, ISO itp.
10

Gemmell, M., T. Hsu, SH Berry, I. Mukhopadhya, R. Hansen, M. Michaud, EM El-Omar i in. "PWE-026 Elucidating the pathogenic potential of the intestinal pathogen campylobacter showae". W British Society of Gastroenterology, Annual General Meeting, 19–22 June 2017, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2017. http://dx.doi.org/10.1136/gutjnl-2017-314472.271.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.

Raporty organizacyjne na temat "Pathogen"

1

Schoeniger, Joseph. Predictive Pathogen Biology: Genome-Based Prediction of Pathogenic Potential and Countermeasures Targets. Office of Scientific and Technical Information (OSTI), styczeń 2018. http://dx.doi.org/10.2172/1733280.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
2

Langlois, R. G., A. Wang, B. Colston, D. Masquelier, L. Jones, K. S. Venkateswaran, S. Nasarabadi, S. Brown, A. Ramponi i F. P. Milanovich. Autonomous pathogen detection system 2001. Office of Scientific and Technical Information (OSTI), styczeń 2001. http://dx.doi.org/10.2172/15006176.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
3

Langlois, R. G., S. Brown, L. Burris, B. Colston, L. Jones, T. Makarewicz, R. Mariella i in. APDS: Autonomous Pathogen Detection System. Office of Scientific and Technical Information (OSTI), luty 2002. http://dx.doi.org/10.2172/15006308.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
4

Haggerty, Ryan P. Pathogen Management Kit (Technical Bulletin). Office of Scientific and Technical Information (OSTI), kwiecień 2020. http://dx.doi.org/10.2172/1615565.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
5

Weiss, Ron. Programmed Pathogen Sense and Destroy Circuits. Fort Belvoir, VA: Defense Technical Information Center, luty 2009. http://dx.doi.org/10.21236/ada494507.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
6

Schikora, B., S. Hietala, L. Shi, L. Lee, E. Skowronski i A. Ardans. Hybrid Pathogen DNA Detector:Users? Manual v1.5. Office of Scientific and Technical Information (OSTI), styczeń 2004. http://dx.doi.org/10.2172/15014078.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
7

Sinclair, Michael B., Todd W. Lane, Howland D. T. Jones, Roberto Rebeil, Susan Jeanne Altman, Julie Kaiser, Lucas K. McGrath i Caroline Ann Souza. Exploratory research into pathogen surface interactions. Office of Scientific and Technical Information (OSTI), luty 2006. http://dx.doi.org/10.2172/877739.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
8

KUHNE, WENDY. ENHANCED FILTER MATERIAL FOR PATHOGEN REMOVAL. Office of Scientific and Technical Information (OSTI), październik 2021. http://dx.doi.org/10.2172/1827681.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
9

Barrett, Louise M., Andrew J. Skulan, Anup K. Singh, Eric B. Cummings i Gregory J. Fiechtner. Continuous-Flow Detector for Rapid Pathogen Identification. Office of Scientific and Technical Information (OSTI), wrzesień 2006. http://dx.doi.org/10.2172/1323909.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
10

Slichter, Sherrill J. Pathogen-Reduced, Plasmalyte-Extended Stored Platelets (PREPS). Fort Belvoir, VA: Defense Technical Information Center, październik 2013. http://dx.doi.org/10.21236/ada612437.

Pełny tekst źródła
Style APA, Harvard, Vancouver, ISO itp.
Możesz być także zainteresowany rozszerzonymi bibliografiami na temat „Pathogen” dla poszczególnych typów źródeł:
Artykuły w czasopismach Rozprawy doktorskie Książki
Oferujemy zniżki na wszystkie plany premium dla autorów, których prace zostały uwzględnione w tematycznych zestawieniach literatury. Skontaktuj się z nami, aby uzyskać unikalny kod promocyjny!

Do bibliografii