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Artykuły w czasopismach na temat "'-nucleotidase"
Darvish, A., R. W. Pomerantz, P. G. Zografides i P. J. Metting. "Contribution of cytosolic and membrane-bound 5'-nucleotidases to cardiac adenosine production". American Journal of Physiology-Heart and Circulatory Physiology 271, nr 5 (1.11.1996): H2162—H2167. http://dx.doi.org/10.1152/ajpheart.1996.271.5.h2162.
Pełny tekst źródłaStochaj, U., i H. G. Mannherz. "Affinity labelling of 5′-nucleotidases with 5′-p-fluorosulphonylbenzoyladenosine". Biochemical Journal 266, nr 2 (1.03.1990): 447–51. http://dx.doi.org/10.1042/bj2660447.
Pełny tekst źródłaHeadrick, J. P., i R. J. Willis. "5′-Nucleotidase activity and adenosine formation in stimulated, hypoxic and underperfused rat heart". Biochemical Journal 261, nr 2 (15.07.1989): 541–50. http://dx.doi.org/10.1042/bj2610541.
Pełny tekst źródłaBiswas, Nabanita, Marta Rodriguez-Garcia, Zheng Shen, Sarah G. Crist, Jack E. Bodwell, John V. Fahey i Charles R. Wira. "Effects of Tenofovir on Cytokines and Nucleotidases in HIV-1 Target Cells and the Mucosal Tissue Environment in the Female Reproductive Tract". Antimicrobial Agents and Chemotherapy 58, nr 11 (18.08.2014): 6444–53. http://dx.doi.org/10.1128/aac.03270-14.
Pełny tekst źródłaSkladanowski, A. C., G. B. Sala i A. C. Newby. "Inhibition of IMP-specific cytosolic 5′-nucleotidase and adenosine formation in rat polymorphonuclear leucocytes by 5′-deoxy-5′-isobutylthio derivatives of adenosine and inosine". Biochemical Journal 262, nr 1 (15.08.1989): 203–8. http://dx.doi.org/10.1042/bj2620203.
Pełny tekst źródłaAslam, Nazia, Syeda Fatima, Sofia Khalid, Shahzad Hussain, Mughal Qayum, Khurram Afzal i Muhammad Hassham Hassan Bin Asad. "Anti-5 ′ -Nucleotidases (5 ′ -ND) and Acetylcholinesterase (AChE) Activities of Medicinal Plants to Combat Echis carinatus Venom-Induced Toxicities". BioMed Research International 2021 (4.02.2021): 1–10. http://dx.doi.org/10.1155/2021/6631042.
Pełny tekst źródłaSkladanowski, A. C., R. T. Smolenski, M. Tavenier, J. W. de Jong, M. H. Yacoub i A. M. Seymour. "Soluble forms of 5'-nucleotidase in rat and human heart". American Journal of Physiology-Heart and Circulatory Physiology 270, nr 4 (1.04.1996): H1493—H1500. http://dx.doi.org/10.1152/ajpheart.1996.270.4.h1493.
Pełny tekst źródłaBorowiec, Agnieszka, Katarzyna Lechward, Kinga Tkacz-Stachowska i Andrzej C. Składanowski. "Adenosine as a metabolic regulator of tissue function: production of adenosine by cytoplasmic 5'-nucleotidases." Acta Biochimica Polonica 53, nr 2 (12.06.2006): 269–78. http://dx.doi.org/10.18388/abp.2006_3339.
Pełny tekst źródłaPiec, G., i M. Le Hir. "The soluble ‘low-Km’ 5′-nucleotidase of rat kidney represents solubilized ecto-5′-nucleotidase". Biochemical Journal 273, nr 2 (15.01.1991): 409–13. http://dx.doi.org/10.1042/bj2730409.
Pełny tekst źródłaMoses, G. C., J. F. Tuckerman i A. R. Henderson. "Biological variance of cholinesterase and 5'-nucleotidase in serum of healthy persons." Clinical Chemistry 32, nr 1 (1.01.1986): 175–77. http://dx.doi.org/10.1093/clinchem/32.1.175.
Pełny tekst źródłaRozprawy doktorskie na temat "'-nucleotidase"
SIEGFRIED, GERALDINE. "Modulation de l'activite 5'-nucleotidase tubulaire renale". Paris 7, 1996. http://www.theses.fr/1996PA077134.
Pełny tekst źródłaRoever, Lisa. "Inhibitor Studies for 5’-ecto-nucleotidase (CD73)". Ohio University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1553892946798977.
Pełny tekst źródłaZanin, Rafael Fernandes. "Investigação das ectonucleotidases na diferenciação de macrófagos e na ativação de plaquetas : o papel da homocisteína". reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2010. http://hdl.handle.net/10183/61004.
Pełny tekst źródłaExtracellular nucleotides modulate a variety of biological actions via purinergic receptor activation. These effects are modulated by ectonucleotidases, such as ENTPDases and ecto-5´- NT/CD73, which hydrolyze ATP to adenosine in the extracellular milieu. In the cells of the immune system, the ATP can act as danger signaling whereas adenosine, the ATP breakdown product, serves as a negative feedback mechanism to limit inflammation. Already, in the vasculare system, the ADP is a physiological agonist involved in normal hemostasis and thrombosis. Since, macrophages are key to inflammatory process, that depending on the microenvironmental stimulation exhibit proinflammatory/ defense (classical/M1) and antiinflammatory/reparatory (alternative/M2) phenotype. The objective of this study was investigate the activity and expression of the ectonuclotidases in differential macrophage phenotype and evaluate the homocysteine (Hcy) effects on theses enzymes in macrophages and platelets. . The analysis of differential macrophages in phenotype proinflamatory/ M1 and antiinflamatory/M2 showed the same expression to P1 and P2 purinoreceptors. However, change profile of the ectonucleotidases as E-NTPDase1, E-NTPDase3 and ecto-5’- nucleotidase enzymes in macrophages during phenotypic differentiation were found, suggesting that macrophages must alter the purinergic cascade during macrophages differentiation phenotypic. In the pro-inflamatory/M1 phenotype the ATP hydrolysis decreased, suggesting ATP accumulation. On the other hand, the antiinflamatory/M2 phenotype the enzymes lead to a progressive decrease in nucleotides (ATP) concentrations and an increase the adenosine availability. Already, the macrophages exposed to Hcy present a polarized pro-inflammatory profile (M1) and our findings suggest the involvement of the E-NTPDase3 and ecto-5’-nucleotidase in the inflammatory complications associates to homocysteine. In the Platelets, which are fundamental elements to the thrombogenesis process, the homocysteine decreased ADP hydrolysis. This elevation of ADP around of the platelets due inactivating of ectonucleotidase, probably by the indirect action of Hcy, may be contributing to increase thrombotic risk described in individuals with hyperhomocysteinemia. In addition, the animals that received Hcy treatment potentiate platelet aggregation induced by ADP. In conclusion, in the present study the results reinforce purinergic signaling involvement in inflammatory/thrombosis process and point to development of treatments to inflammatory/thrombotic diseases.
ZEKRI, MUSTAPHA. "Heterogeneite structurale et fonctionnelle de la 5-nucleotidase". Nantes, 1990. http://www.theses.fr/1990NANT2056.
Pełny tekst źródłaBavaresco, Luci. "Estudo do papel da ecto-5'-nucleotidase/CD73 na proliferação de gliomas". reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/12713.
Pełny tekst źródłaMalignant gliomas are the most common and devastating primary tumors in the central nervous system. Despite treatment, patients with these tumors have a poor prognosis. Ecto-5‟-nucleotidase/CD73 (ecto-5‟-NT/CD73) may regulate the extracellular AMP and adenosine levels, which have been described as proliferation factor. The participation of ecto-5‟-NT/CD73 per se has been proposed as a proliferative factor, being involved in the control of cell growth, maturation, differentiation, invasion, migration and metastases processes. In the present study, we evaluate the ecto-5‟-NT/CD73 activity and functions in rat C6 and human U138-MG glioma cell lines proliferation process. Crescent confluences and culture times leads to an increase on ecto-5‟-NT/CD73 activity in both C6 and U138-MG glioma cells. RT-PCR analysis and flow cytometry showed a significant increase on ecto-5‟-NT/CD73 mRNA and protein levels respectively, when compared confluent cultures with subconfluent one in human U138-MG glioma cells. Treatment with 1 M APCP, a competitive ecto-5‟-NT inhibitor, caused a significant reduction in glioma cell proliferation of 20% for U138-MG glioma cell line. In addition, 100 M adenosine increases cell proliferation in 25% and AMP 1m M and 3 mM decrease U138-MG glioma cells proliferation in 29% and 42% respectively. The stable silencement of ecto-5‟-NT/CD73 by RNAi technique reduces cell migration in human U138-MG glioma cell line. Taken together these results suggest the participation of ecto-5‟-NT/CD73 in cell proliferation, being this process dependent of enzymatic activity generating adenosine, a proliferative factor and removing toxic levels of AMP, as well as a function as adhesive molecule.
Knöfel, Thomas. "Röntgenstrukturanalyse der 5'-Nucleotidase aus Escherichia coli mit dinuklearem Metallzentrum". [S.l. : s.n.], 2000. http://www.diss.fu-berlin.de/2000/131/index.html.
Pełny tekst źródłaOsborne, Foy Naomi. "Over-Expression of Ecto-5'-Nucleotidase in Pig Endothelial Cells". Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487200.
Pełny tekst źródłaMcMillen, Lyle, i l. mcmillen@sct gu edu au. "Isolation and Characterisation of the 5'-Nucleotidase from Escherichia coli". Griffith University. School of Biomolecular and Biomedical Science, 2001. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20030226.153545.
Pełny tekst źródła黎錦明 i Kam-ming Lai. "Structure and function of 5'-nucleotidase of the rat brain". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1991. http://hub.hku.hk/bib/B31232280.
Pełny tekst źródłaSaraiva, Antonio Marcos. "Caracterização funcional e estrutural da nucleotidase SurE de Xyllela fastidiosa". [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/316474.
Pełny tekst źródłaTese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia
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Resumo: A linhagem 9a5c da bactéria Xylella fastidiosa foi o primeiro fitopatógeno a ter seu genoma completamente sequenciado, o qual gerdu diversas informações sobre seu metabolismo e patogenicidade. Das orfs codificadas por esta bactéria, destaca-se; no presente trabalho, a XF0703, cuja proteína correlata (com 28,3 kDa) possui similaridade com proteínas SurE de várias outras bactérias. Proteínas SurEs são nucleotidases que desfosforilam diversos nucleosideos monofosforilados para seus respectivos nucleosideos. Tal função é de fundamental importância para manter o pool balanceado dos quatro (deoxi)ribonucleosideos para síntese de DNA e RNA, respectivamente. Este trabalho descreve a clonagem da orfXF0703 no vetor pET29a, a expressão da proteína recombinante (XfSurE) em Escheríchia coli BL21(DE3) e a purificação da mesma por cromatografia de afinidade ao níquel. A análise da estrutura secundária foi feita por dicroísmo circular e realizou-se a determinação do estado oligomérico por cromatografia de gel filtração e espalhamento de luz a baixo ângulo (SAXS), os quais revelaram que a proteína é um tetrâmero. Dados de caracterização funcional indicam que a proteína possui maior atividade em pH neutro na presença do íon manganês como cofator, com uma maior afinidade pelo substrato 3'-AMP (K0,5=0,16 mM). Além disso, ensaios cinéticos mostram que a proteína possui um comportamento alostérico com alta cooperatividade positiva (coeficiente de Hill em torno de 2,6) com todos os quatro substratos naturais testados (3'-AMP, 5'-dAMP, 5'-AMP e 5-GMP). Experimentos com a técnica de SAXS permitiram calcular o raio de giro (32,7 ± 0.2 A), distância máxima intramolecular (100 A) e a simetria do envelope da molécula (222). A estrutura de diversas SurEs homólogas já cristalizadas foram superpostas ao envelope obtido, sendo que StSurE (SurE de Salmonella com maior idenjidade de aminoácidos) mostrou ter o melhor ajuste. No entanto, notou-se que havia espaços vazios no envelope de XfSurE e tais espaços podiam ser preenchidos a partir do afastamento das alças responsáveis pela tetramerização e pela rotação dos f dímeros. Estes movimentos (translação e rotação) podem explicar o comportamento alostérico da proteína, facilitando a entrada de substrato ao sítio catalítico da molécula.
Abstract: The 9a5c strain from bacterium Xylella fastidiosa was the first phytopathogen to have its genome completely sequenced, which revealed a lot of information about its metabolism and its pathogenicity. 'From a variety of orfs encoded by this bacterium, we highlight, in this work, the XF0703, which correlated protein (with 28.3 kDa) has similarity with SurE proteins from several other bacteria. The SurE proteins *are nucleotidases that dephosphorylate various monophosphorylated nucleosides to their respective nucleosides. This function is critical for maintaining the balanced pool of four (deoxy) ribonucleosides for DNA and RNA synthesis. In this work, we describes the cloning of the XF0703 orf into the vector pET29a, the recombinant protein overexpression (XfSurE) in Escherichia coli BL21(DE3) and the protein purification by nickel affinity chromatography. The secondary structure analysis was done by circular dichroism, while oligomeric state determination was achieved by gel filtration chromatography and small-angle X-ray light scattering (SAXS), which showed that the protein is a tetramer. Functional characterization data indicate that the protein has a highest activity at neutral pH in the presence of manganese as a cofactor, with a highest affinity for the 3-AMP substrate (K0,5 = 0,16 mM). Furthermore, kinetic tests showed that the protein has a allosteric behavior with a high positive cooperativity (Hill coefficient around 2.6) for all natural substrates screened (3-AMP, 5'-dAMP, 5'-AMP and 5'-GMP). Experiments with SAXS technique have allowed to calculate the radius of gyration (32.7 ± 0.2 A), maximum intramolecular distance (100 A) and molecule symmetry.
Doutorado
Genetica de Microorganismos
Doutor em Genetica e Biologia Molecular
Książki na temat "'-nucleotidase"
Barber, Ian Stuart. 5'-nucleotidase: It's [sic]Membrane/cytoskeletal associations. Birmingham: Universityof Birmingham, 1989.
Znajdź pełny tekst źródłaMarani, Enrico. Topographic histochemistry of the cerebellum: 5'-nucleotidase, acetylocholinesterase, immunology of FAL. Stuttgart: G. Fischer Verlag, 1986.
Znajdź pełny tekst źródłaCzęści książek na temat "'-nucleotidase"
Schomburg, Dietmar, i Margit Salzmann. "Nucleotidase". W Enzyme Handbook 3, 437–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76463-9_92.
Pełny tekst źródłaSchomburg, Dietmar, i Margit Salzmann. "5’-Nucleotidase". W Enzyme Handbook 3, 309–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76463-9_66.
Pełny tekst źródłaSchomburg, Dietmar, i Margit Salzmann. "3’-Nucleotidase". W Enzyme Handbook 3, 315–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76463-9_67.
Pełny tekst źródłaSchomburg, Dietmar, i Margit Salzmann. "Phosphoadenylate 3’-nucleotidase". W Enzyme Handbook 3, 319–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76463-9_68.
Pełny tekst źródłaFroehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, Jan Rémi, Soheyl Noachtar, Werner J. Heppt, Annette Cryer i in. "5′-Nucleotidase Hyperactivity". W Encyclopedia of Molecular Mechanisms of Disease, 1501–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_4.
Pełny tekst źródłaBraun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber i in. "Pyrimidine 5′ Nucleotidase Deficiency". W Encyclopedia of Molecular Mechanisms of Disease, 1798. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8047.
Pełny tekst źródłaBraun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber i in. "Pyrimidine 5′ Nucleotidase-1 Deficiency". W Encyclopedia of Molecular Mechanisms of Disease, 1798. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8048.
Pełny tekst źródłaKreutzberg, G. W., D. Heymann i M. Reddington. "5′-Nucleotidase in the Nervous System". W Proceedings in Life Sciences, 147–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70664-6_11.
Pełny tekst źródłaMarinello, E., A. Tabucchi, F. Carlucci, P. Galieni i F. Rosi. "Isoenzymes of 5′-Nucleotidase in Human Lymphocytes". W Advances in Experimental Medicine and Biology, 555–58. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5381-6_107.
Pełny tekst źródłaWebster, A. D. B., M. Rowe, S. M. Johnson, G. L. Asherson i A. Harkness. "Ecto 5′-Nucleotidase Deficiency in Primary Hypogammaglobulinaemia". W Ciba Foundation Symposium 68 - Enzyme Defects and Immune Dysfunction, 135–64. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720516.ch9.
Pełny tekst źródłaStreszczenia konferencji na temat "'-nucleotidase"
AL-Tikrity, Ilham, Abeer Alattar, Harith Mustafa i Safaa Sultan. "Biochemical Characterization of Nucleotidase in some Parasites". W Proceedings of the 1st International Multi-Disciplinary Conference Theme: Sustainable Development and Smart Planning, IMDC-SDSP 2020, Cyperspace, 28-30 June 2020. EAI, 2020. http://dx.doi.org/10.4108/eai.28-6-2020.2298245.
Pełny tekst źródłaNguyen, Anna M., Jianhong Zhou i Yuchun Du. "Abstract B05: Ecto-5’-nucleotidase (CD73) confers radioresistance in pancreatic cancer". W Abstracts: AACR Special Conference on Pancreatic Cancer: Advances in Science and Clinical Care; May 12-15, 2016; Orlando, FL. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.panca16-b05.
Pełny tekst źródłaGerhardt, Josefine, Corinna Steinbrech, Florian Fritzsche, Verena Tischler, Michael Müntener, Tullio Sulser, Carsten Stephan, Klaus Jung, Holger Moch i Glen Kristiansen. "Abstract 1772: Calcium activated nucleotidase 1 (CANT1) promotes progression of prostate carcinomas". W Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1772.
Pełny tekst źródłaJordheim, Lars P., Zsuzsanna Marton, Moez Rhimi, Laurent Chaloin, Emeline Cros-Perrial, Corinne Lionne, Suzanne Peyrottes, Nushin Aghajari i Charles Dumontet. "Abstract 3835: Identification and characterization of inhibitors of 5′-nucleotidase cN-II issued from virtual screening". W Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3835.
Pełny tekst źródłaBowser, Jessica L., Michael R. Blackburn, Gregory L. Shipley, Susu Xie i Russell R. Broaddus. "Abstract 3384: Down-regulation of 5’-nucleotidase (CD73)-generated adenosine: A novel mechanism for regulating endometrial cancer metastasis". W Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3384.
Pełny tekst źródłaMatsuyama, Masahiro, Masatoshi Wakui, Makoto Monnai, Tomoko Mizushima, Chiyoko NIshime, Kenji Kawai, Hiroshi Suemizu i in. "Abstract 2366: Reduced ecto-5′-nucleotidase CD73 expression and altered purine nucleotide metabolism in colorectal cancer cells robustly causing liver metastases". W Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2366.
Pełny tekst źródłaZborovsky, AB, BV Zavodovsky, EV Bobicheva, LE Sivordova i NA Fofanova. "THU0055 Role of antibodies to 5’nucleotidase in rheumatoid arthritis, systemic lupus erythematosus, systemic sclerosis, ankylosing spondylarthritis and reactive arthritis patients". W Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.899.
Pełny tekst źródłaFaulkes, Rosemary, Joanne O’Rourke, Owen Cain, Daniel Patten, Alex Wilkinson, Tahir Shah, Christopher Weston i Shishir Shetty. "O02 Deep sequencing of HCC endothelium reveals an active role in immunosuppression and highlights the ecto nucleotidase CD73 as a potential therapeutic target". W Abstracts of the British Association for the Study of the Liver Annual Meeting, 22–24 November 2021. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2021. http://dx.doi.org/10.1136/gutjnl-2021-basl.2.
Pełny tekst źródłaZborovsky, AB, BV Zavodovsky, TA Pankratova, AV Rvachev, OV Bykova i TV Serdukova. "SAT0001 Role of determination of succinate dehydrogenase, myeloperoxidase, na ± k ± atph-ase, 5?-nucleotidase in cells of peripheral blood of ankylosing spondylarthritis patients". W Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.353.
Pełny tekst źródłaGoueli, Said A., i Kevin hsiao. "Abstract 414: Biochemical and cellular monitoring of the activity of the ecto-5’-nucleotidase (CD73), a key cancer modulator using HTS-formatted bioluminescent technology". W Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-414.
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