Rozprawy doktorskie na temat „Non-melanoma skin cancer”
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Ashton, Kevin John, i K. Ashton@griffith edu au. "Genetic Aberrations in Non-Melanoma Skin Cancer". Griffith University. School of Health Science, 2002. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20030818.122305.
Pełny tekst źródłaCarless, Melanie, i n/a. "Molecular Analysis of Non-Melanoma Skin Cancer". Griffith University. School of Health Science, 2004. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20041101.123114.
Pełny tekst źródłaCarless, Melanie. "Molecular Analysis of Non-Melanoma Skin Cancer". Thesis, Griffith University, 2004. http://hdl.handle.net/10072/367527.
Pełny tekst źródłaThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Health Sciences
Full Text
Ashton, Kevin John. "Genetic Aberrations in Non-Melanoma Skin Cancer". Thesis, Griffith University, 2002. http://hdl.handle.net/10072/367012.
Pełny tekst źródłaThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Health Sciences
Full Text
Ramsay, Helen Mary. "Non-melanoma skin cancer in renal transplant recipients". Thesis, University of Birmingham, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408971.
Pełny tekst źródłaUr-Rehman, Ishtiaq. "Genetic change in human non-melanoma skin cancer". Thesis, University of Newcastle Upon Tyne, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318260.
Pełny tekst źródłaJones, Michael Howard. "Chromosomal rearrangement and mutation in non-melanoma skin cancer". Thesis, University of Newcastle Upon Tyne, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287141.
Pełny tekst źródłaBrownlie, Laura. "Differential gene expression studies in non-melanoma skin cancer". Thesis, University of Newcastle Upon Tyne, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323449.
Pełny tekst źródłaBen, Ketah Antsar. "Defining and targeting differentiation of non-melanoma skin cancer". Thesis, Cardiff University, 2014. http://orca.cf.ac.uk/64974/.
Pełny tekst źródłaLi, Xin. "Genetics, Caffeine Consumption, Height and Non-Melanoma Skin Cancer". Thesis, Harvard University, 2016. http://nrs.harvard.edu/urn-3:HUL.InstRepos:27201750.
Pełny tekst źródłaChen, Andrew Chih-Chieh. "Effect of oral nicotinamide on non-melanoma skin cancer and skin barrier function". Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/15831.
Pełny tekst źródłaMladkova, Nikol. "Epigenetic profiling and molecular characterisation of non-melanoma skin cancer". Thesis, Queen Mary, University of London, 2014. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8768.
Pełny tekst źródłaAlshammari, Eid Salem. "∆Np63α Positively Regulates ERK3 Expression in Non-Melanoma Skin Cancer". Wright State University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1556229754222487.
Pełny tekst źródłaFarrell, Andrew William. "The Role of Brm in Non-‐Melanoma Skin Cancer Progression". Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/16438.
Pełny tekst źródłaSandberg, Carin. "Aspects of fluorescence diagnostics and photodynamic therapy in non-melanoma skin cancer /". Göteborg : Department of Dermatology and Venereology, Sahlgrenska University Hospital, Institute of Clinical Sciences, Sahlgrenska Academy University, University of Gothenburg, 2009. http://hdl.handle.net/2077/21192.
Pełny tekst źródłaTehrani, Hamid. "Can the SIAscope aid in the diagnosis of non-melanoma skin cancer?" Thesis, University of East Anglia, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.430601.
Pełny tekst źródłaCasabonne, Delphine. "The seroepidemiology of human-papillomavirus in relation to non-melanoma skin cancer". Thesis, Queen Mary, University of London, 2009. http://qmro.qmul.ac.uk/xmlui/handle/123456789/1411.
Pełny tekst źródłaTaylor, Marlon D., i Marlon D. Taylor. "Sulforaphane Potentiates Non-Melanoma Skin Cancer in UVB-Treated Nrf2 Knockout Mice". Thesis, The University of Arizona, 2016. http://hdl.handle.net/10150/622859.
Pełny tekst źródłaBachelor, Michael A. "Identification of molecular targets for the chemoprevention of non-melanoma skin cancer". Diss., The University of Arizona, 2004. http://hdl.handle.net/10150/280585.
Pełny tekst źródłaJackson, Sarah. "Studies on cutaneous human papillomaviruses and their association with non-melanoma skin cancer". Thesis, Queen Mary, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392129.
Pełny tekst źródłaQuinn, Anthony G. "Chromosome loss and tumour suppressor gene inactivation in human non-melanoma skin cancer". Thesis, University of Newcastle Upon Tyne, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260068.
Pełny tekst źródłaJones, Brad. "Efficacy and action of topical diterpenes from Euphorbia Peplus in non-melanoma skin cancer /". St. Lucia, Qld, 2002. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17268.pdf.
Pełny tekst źródłaSURJANA, Devita. "The Role of Nicotinamide in DNA Repair and Prevention of Non-melanoma Skin Cancer". Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/10552.
Pełny tekst źródłaVeomett, Nicholas James. "ASSAY DEVELOPMENT FOR THE PHARMACOKINETIC STUDY OF A NOVEL NON-MELANOMA SKIN CANCER DRUG". Thesis, The University of Arizona, 2009. http://hdl.handle.net/10150/193019.
Pełny tekst źródłaHarwood, Catherine Anne. "Human papillomavirus and the molecular pathogenesis of non-melanoma skin cancer associated with organ transplantation". Thesis, Queen Mary, University of London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.407957.
Pełny tekst źródłaSullivan, Nicholas James. "Interleukin-6 as a Potential Mediator of Breast Cancer Progression and Non-Melanoma Skin Carcinogenesis". The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1249493495.
Pełny tekst źródłaClouser, Mary Catherine. "Non-melanoma Skin Cancer Prevention: Impact of Non-Steroidal Anti-Inflammatory Drugs, Retinoid Dose Response and Measurement Reliability". Diss., The University of Arizona, 2009. http://hdl.handle.net/10150/195524.
Pełny tekst źródłaLovatt, Tracy J. "Association of ultraviolet radiation and genetic polymorphism with clinical phenotype in non melanoma skin cancer patients". Thesis, Keele University, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.409551.
Pełny tekst źródłaTeegarden, Matthew D. "Determination of Biologically Relevant Vitamin D Metabolites in a Mouse Model of Non-Melanoma Skin Cancer". The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1408121763.
Pełny tekst źródłaHolloway, Amy Frances. "Investigation of the contribution of cutaneous HPV E6 proteins towards the development of non melanoma skin cancer". Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:9369b19d-21f7-44fd-b48a-aa3f3d59e456.
Pełny tekst źródłaBock, Vanessa Leonie. "The Role of Brm, Brg-1, Snail 1 and Snail 2 in the Progression of Non-Melanoma Skin Cancer". Thesis, The University of Sydney, 2008. http://hdl.handle.net/2123/4091.
Pełny tekst źródłaBock, Vanessa Leonie. "The Role of Brm, Brg-1, Snail 1 and Snail 2 in the Progression of Non-Melanoma Skin Cancer". University of Sydney, 2008. http://hdl.handle.net/2123/4091.
Pełny tekst źródłaNon-melanoma skin cancer (NMSC) is the most common human cancer worldwide. Squamous cell carcinoma (SCC) and basal cell carcinoma (BCC) make up almost all NMSC. SCC usually arises from actinic keratosis (AK) as a result of exposure to sunlight. SCC and AK provide a useful clinical model to investigate changes involved in the progression of NMSC. This project examines the expression of Brm, Brg-1, Snail 1 and Snail 2 in the progression of NMSC. Brm and Brg-1 are subunits of the SWI/SNF chromatin-remodelling complex which is involved in regulating the access of cell machinery to DNA by altering the structure of chromatin. It has been suggested that loss of this function is involved in carcinogenesis as the cell is unable to access to DNA normally in order to repair mutations or activate apoptosis. The loss of Brm or Brg-1 has been described in several human cancers. Snail 1 and Snail 2 are zinc-finger transcription factors that are known for their role in epithelial to mesenchymal transition (EMT), a process vital to embryological development. Increased expression of these factors leads to a loss of cell-cell adhesion and a migratory phenotype and has been described in some human cancers. In this project, double-label immunohistochemistry was used to determine the relative expression of these proteins in human SCC, BCC, AK and normal skin. The expression of Snail was unable to be determined due to poor specificity of the antibodies used. The expression of both Brm and Brg-1 proteins was found to be dramatically and consistently decreased in SCC and BCC when compared to normal skin and AK. This loss of Brm and Brg-1 occured as the tumour progressed from benign AK to malignant SCC. This finding suggests that the loss of either Brm or Brg-1 constitutes a key step in carcinogenesis. The results of this study identify Brm and Brg-1 as putative tumour suppressors involved in the progression of non-melanoma skin cancer from benign to malignant.
Albati, Amal Abdulah. "PURIFICATION OF RECOMBINANT δ NP63 α AND CHARACTERIZATION OF PEPTIDE BINDING". Wright State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=wright1447223071.
Pełny tekst źródłaGiampieri, Silvia. "A study on the potential role of cutaneous human papillomaviruses in the development of non-melanoma skin cancer". Thesis, Queen Mary, University of London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.413322.
Pełny tekst źródłaSilva, Raquel Inácia Almeida Brites Pereira da. "Psoríase: evolução farmacoterapêutica e risco acrescido de desenvolvimento de certas neoplasias". Master's thesis, [s.n.], 2014. http://hdl.handle.net/10284/4511.
Pełny tekst źródłaA psoríase é uma doença crónica autoimune e inflamatória, considerada uma das dermatoses mais frequentes na prática clínica. Os doentes que sofrem desta patologia podem manifestar diferentes variantes, nomeadamente, psoríase em placa, inversa, gutata, pustular, eritrodérmica e artrite psoriática. Esta doença apresenta um carácter multifatorial com uma forte componente genética, no entanto, as suas manifestações clínicas podem resultar de outros tipos de fatores tais como, imunológicos e ambientais. A prevalência psoriática, de um modo geral, abrange cerca de 1 a 3% da população mundial, sendo que, em Portugal, existem aproximadamente 250 000 indivíduos diagnosticados com psoríase. Independentemente da abordagem teórica, muitos autores consideram a pele como o primeiro meio de contacto do indivíduo com o meio. Partindo desta posição, pode-se pensar nos prejuízos causados no bem-estar psicológico de indivíduos acometidos por este tipo de doença, uma vez que, os mesmos podem desenvolver comportamentos de ansiedade, depressão, exclusão social e em situações mais graves, podem exibir intensões suicidas. Além do impacto da sua vida social, a vida familiar e a dieta alimentar são, igualmente, afetadas. O tratamento das lesões psoriáticas envolve terapia tópica, sistémica, fototerapia e, mais recentemente, terapia biológica. A escolha farmacoterápica é efetuada de acordo com a gravidade da patologia e tendo em conta que, apenas se fará a manutenção das lesões, dado tratar-se de uma doença sem cura. Com o tempo, o doente pode exibir certas comorbilidades relacionadas com o seu quadro clínico, entre elas, cancro. Os dados bibliográficos publicados até ao ano corrente descrevem vários tipos de cancro, sendo que, os mais comuns são os linfomas e o cancro de pele não-melanoma. Este trabalho consiste num estudo meta-analítico, que pretende desenvolver mais estas temáticas. Psoriasis is chronic, autoimmune and inflammatory disease, considered to be one of the most frequent dermatitis in clinical practice. This condition has several variants that can manifest in patients. These subtypes are: plaque psoriasis, gutata, pustular, eritrodermic, inverse and psoriatic arthritis. Being a disease with multifactorial influences, psoriasis has a strong genetic association, nevertheless, its clinical manifestations may also come from several other types of interactions, such as immunologic and environmental factors. Psoriatic prevalence in the world varies from 1 to 3%. In Portugal there are approximately 250 000 individual diagnosed with this condition. Despite the theoretic approach that may be picked, many authors consider the skin as the first barrier of contact between the individual and the outside world. Taking this into account, it´s possible to imagine the damage caused in the well being of individuals who suffer from this condition, considering that they may develop behaviors of anxiety, depression and social exclusion. In more severe situations, patients may even exhibit suicidal tendencies. Besides the social impact shown, family life and individual diet are equally affected. The treatment for psoriatic lesions entails topical and systemic drugs, phototherapy and, more recently, biologic therapy. Pharmacotherapeutic choices are made based on the condition’s gravity and keeping in mind that the aim is to control the lesions, considering this disease has no cure. As time goes by, individuals can exhibit comorbidities associated with psoriasis. Cancer is one of these conditions. The bibliographic data published until the present year, describes several types of cancer, but the ones most often mentioned are lymphomas and non melanoma skin cancer. This essay is a meta-analytic study aiming to further discuss these issues.
Drew, Benjamin. "Treatment Patterns, Outcomes, and Patient Satisfaction of Epidermal Superficial Non-Melanoma Skin Cancer at an Academic Dermatologic Surgery Center". Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17295900.
Pełny tekst źródłaCamillo, Lara. "Evaluation of vitamin B3, D3 and E as photoprotectors against UVB-induced damages on primary keratinocytes and fibroblasts". Doctoral thesis, Università del Piemonte Orientale, 2022. http://hdl.handle.net/11579/142780.
Pełny tekst źródłaCampbell, Christine. "The role of tumour suppressor gene P53 in non-melanoma skin cancer and in the cellular response to ultraviolet radiation". Thesis, University of Newcastle Upon Tyne, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260075.
Pełny tekst źródłaQuina, Carolina Lopes. "Estudo da morte celular em queratinócitos de animais nocauteados para os genes BIM, PUMA e mutante para as moléculas FAS tratados com quimioterápicos e indutores de carcinogênese". Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-27082015-185715/.
Pełny tekst źródłaThe non-melanoma cancer accounts for 25% of all tumors and is characterized by a neoplasm of keratinocytes. Evidence shows that apoptosis resistance is one of the characteristics for the emergence and development of malignant tumors. The This study investigated what roles the BIM, PUMA and Fas molecules play in keratinocytes when subjected to cell transformations, such as UVB and DMBA, and apoptosis inducers. For this, keratinocytes primary culture was established from neonatal skin to C57BL / 6 WT; PUMA KO; LPR. The cells were treated with AD, VP-16, CHX, UVB and DMBA in vitro. We observed the expression of Bid, Bim, Bax in treatment with UVB. We evaluated tumor development profile in these animals using a model of chemical tumorigenesis in vivo. PUMA-deficient keratinocytes and Fas mutant have similarly behave in chemotherapy treatment, however, Fas molecule plays a more important role in the induction of cell death with the treatment with carcinogens in the development and carcinogenesis.
Burns, Erin Marie. "Men and Women are Not Just From Different Planets: The Role of Sex-Based Differences in the Prevention of Non-Melanoma Skin Cancer". The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1364481982.
Pełny tekst źródłaHira, Akshay. "TIP60 regulation of DNp63a is associated with cisplatin resistance". Wright State University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1566585763492406.
Pełny tekst źródłaBoone, Marc. "High-definition optical coherence tomography: Contribution to the non-invasive near infrared optical imaging techniques of the skin". Doctoral thesis, Universite Libre de Bruxelles, 2016. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/232235.
Pełny tekst źródłaDoctorat en Sciences médicales (Santé Publique)
info:eu-repo/semantics/nonPublished
Leonard, Mary Kathryn. "Regulation of the Transcription and Subcellular Localization of the Tumor Suppressor PTEN by ΔNp63α". Wright State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=wright1353525238.
Pełny tekst źródłaValentine, Ronan. "Biophysical aspects of photodynamic therapy". Thesis, University of St Andrews, 2011. http://hdl.handle.net/10023/2471.
Pełny tekst źródłaStacy, Andrew Jared. "Regulation of ΔNp63α by TIP60 promotes cellular proliferation". Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1596151919161674.
Pełny tekst źródłaSakaram, Suraj. "Delineating ΔNp63α's function in epithelial cells". Wright State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=wright1484411625682248.
Pełny tekst źródłaWestphal, Kathi. "Molekulare Mechanismen kutaner humaner Papillomviren (HPV) während der Hautkarzinogenese". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2009. http://dx.doi.org/10.18452/15998.
Pełny tekst źródłaIn the last years epidemiologic and molecular biological studies accumulated increasing evidence that cutaneous human papillomaviruses are etiologically involved in the formation of non-melanoma skin cancer (NMSC). The presented work aims to identify the underlying molecular mechanisms of the viral proteins E6 and E7 of cutaneous HPV types. The E6 and E7 genes of the different HPV types 1, 4, 5, 8, 20, and RTRX7, which are in vivo associated with cutaneous benign or malignant lesions, were studied. Natural host cells of these viruses, human primary keratinocyts (HPK) of the skin, were infected with recombinant E6 and E7 encoding retroviruses. The following analyses were performed in monolayer culture (non-differentiated keratinocytes) or in organotypic skin culture (induction of keratinocyte differentiation). The expression of E6 and E7 elongated the life span of monolayer HPK and significantly increased the doubling rate. An activation of the telomerase, characteristic for immortalized cells, was only detected in HPV 8 E6 positive cells. In organotypic skin cultures E7 of HPV 1, 4 and 38 induced drastic changes in differentiation and proliferation. Additionally an impairment of the normal cell cycle control in suprabasale HPV 5 E7 and 8 E7 cultures was seen. Hints for a strong invasive potential of E7 infected HPK were proven for HPV 8 E7 and expanded to HPV 4 E7, HPV 38 E7 and RTRX E7. The viral E6 and E7 genes of cutaneous and mucosal HPV types exhibit different molecular mechanisms. The multistep model of carcinogenesis includes a series of fundamental cell transformations necessary for tumorigenesis. Mechanisms for the modulation of cell differentiation and proliferation by cutaneous HPV types 4, 5, 8 and 38 described in this work could potentially contribute to the induction and progression of early stages of squamous cell carcinoma.
Surentheran, Thirunavukarasu. "A putative role for cutaneous human papillomaviruses in the pathogenesis of non-melanoma skin cancers". Thesis, Queen Mary, University of London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.272275.
Pełny tekst źródłaMarwah, Mandeep Kaur. "Development of a novel deformable liposomal formulation for the dermal drug delivery of anticancer agents in the treatment of non-melanoma skin cancers". Thesis, Aston University, 2017. http://publications.aston.ac.uk/37493/.
Pełny tekst źródłaBeiggi, Sara. "Epidemiological study of chronic lymphocytic leukemia (CLL) in the province of Manitoba, Canada". British Journal of Cancer (Nature Group), 2013. http://hdl.handle.net/1993/23508.
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