Gotowa bibliografia na temat „Non invasive positive pressure ventilation”
Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych
Spis treści
Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Non invasive positive pressure ventilation”.
Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.
Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.
Artykuły w czasopismach na temat "Non invasive positive pressure ventilation"
Nieberg, Astrid. "Non-Invasive Positive Pressure Ventilation". Critical care 3, nr 2 (kwiecień 2006): 56–58. http://dx.doi.org/10.1007/bf03063107.
Pełny tekst źródłaPreston, Roland, Frances Kelly i Morag McNulty. "Introducing non-invasive positive pressure ventilation". Nursing Standard 15, nr 26 (14.03.2001): 42–45. http://dx.doi.org/10.7748/ns2001.03.15.26.42.c2997.
Pełny tekst źródłaHaddad, J., A. Mokline, I. Rahmani, H. Oueslati, I. El Jami, K. Brini, K. Bousselmi i A. Messadi. "Non-invasive positive pressure ventilation in burns". Critical Care 14, Suppl 1 (2010): P240. http://dx.doi.org/10.1186/cc8472.
Pełny tekst źródłaPavone, M., E. Verrillo, V. Caldarelli, N. Ullmann i R. Cutrera. "Non-invasive positive pressure ventilation in children". Early Human Development 89 (październik 2013): S25—S31. http://dx.doi.org/10.1016/j.earlhumdev.2013.07.019.
Pełny tekst źródłaMehta, Akshay. "Synopsis on Non-invasive Ventilation in Neonatology". International Journal of Clinical Case Reports and Reviews 7, nr 04 (17.07.2021): 01–06. http://dx.doi.org/10.31579/2690-4861/128.
Pełny tekst źródłaWindisch, Wolfram, Jan Hendrik Storre i Thomas Köhnlein. "Nocturnal non-invasive positive pressure ventilation for COPD". Expert Review of Respiratory Medicine 9, nr 3 (20.04.2015): 295–308. http://dx.doi.org/10.1586/17476348.2015.1035260.
Pełny tekst źródłaAggarwal, Deepak, i Prasanta Raghab Mohapatra. "Non-invasive positive pressure ventilation for severe COPD". Lancet Respiratory Medicine 2, nr 10 (październik 2014): e18-e19. http://dx.doi.org/10.1016/s2213-2600(14)70199-7.
Pełny tekst źródłaLobato, Salvador Díaz, i Sagrario Mayoralas Alises. "Non-invasive positive pressure ventilation for severe COPD". Lancet Respiratory Medicine 2, nr 10 (październik 2014): e17-e18. http://dx.doi.org/10.1016/s2213-2600(14)70200-0.
Pełny tekst źródłaKharasch, Virginia S. "Non-invasive positive pressure ventilation: Principles and applications". Pediatric Pulmonology 34, nr 1 (4.06.2002): 89. http://dx.doi.org/10.1002/ppul.10122.
Pełny tekst źródłaRosemeier, F., i J. H. Cook. "A complication of non-invasive positive pressure ventilation". Anaesthesia 56, nr 4 (kwiecień 2001): 390–91. http://dx.doi.org/10.1046/j.1365-2044.2001.01976-31.x.
Pełny tekst źródłaRozprawy doktorskie na temat "Non invasive positive pressure ventilation"
Lyall, Rebecca Ann. "Respiratory muscle function and non-invasive positive pressure ventilation in motor neurone disease". Thesis, King's College London (University of London), 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417858.
Pełny tekst źródłaOscroft, Nicholas Stephen. "The effects of long-term non-invasive positive pressure ventilation in hypercapnic chronic obstructive pulmonary disease". Thesis, University of London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.542958.
Pełny tekst źródłaMarano, Alexis. "Non-invasive positive pressure ventilation (nppv) its uses, complications, & implications within nursing practice in acute care settings". Honors in the Major Thesis, University of Central Florida, 2012. http://digital.library.ucf.edu/cdm/ref/collection/ETH/id/580.
Pełny tekst źródłaB.S.N.
Bachelors
Nursing
Nursing
Cantin, Danny. "Effet inhibiteur de la ventilation nasale à pression positive intermittente sur les reflux gastro-oesophagiens chez l'agneau nouveau-né". Mémoire, Université de Sherbrooke, 2015. http://hdl.handle.net/11143/6719.
Pełny tekst źródłaJohnson, Pamela Lesley. "Sleep and Breathing at High Altitude". University of Sydney, 2008. http://hdl.handle.net/2123/3531.
Pełny tekst źródłaThis thesis describes the work carried out during four treks, each over 10-11 days, from 1400m to 5000m in the Nepal Himalaya and further work performed during several two-night sojourns at the Barcroft Laboratory at 3800m on White Mountain in California, USA. Nineteen volunteers were studied during the treks in Nepal and seven volunteers were studied at White Mountain. All subjects were normal, healthy individuals who had not travelled to altitudes higher than 1000m in the previous twelve months. The aims of this research were to examine the effects on sleep, and the ventilatory patterns during sleep, of incremental increases in altitude by employing portable polysomnography to measure and record physiological signals. A further aim of this research was to examine the relationship between the ventilatory responses to hypoxia and hypercapnia, measured at sea level, and the development of periodic breathing during sleep at high altitude. In the final part of this thesis the possibility of preventing and treating Acute Mountain Sickness with non-invasive positive pressure ventilation while sleeping at high altitude was tested. Chapter 1 describes the background information on sleep, and breathing during sleep, at high altitudes. Most of these studies were performed in hypobaric chambers to simulate various high altitudes. One study measured sleep at high altitude after trekking, but there are no studies which systematically measure sleep and breathing throughout the whole trek. Breathing during sleep at high altitude and the physiological elements of the control of breathing (under normal/sea level conditions and under the hypobaric, hypoxic conditions present at high altitude) are described in this Chapter. The occurrence of Acute Mountain Sickness (AMS) in subjects who travel form near sea level to altitudes above 3000m is common but its pathophysiology not well understood. The background research into AMS and its treatment and prevention are also covered in Chapter 1. Chapter 2 describes the equipment and methods used in this research, including the polysomnographic equipment used to record sleep and breathing at sea level and the high altitude locations, the portable blood gas analyser used in Nepal and the equipment and methodology used to measure each individual’s ventilatory response to hypoxia and hypercapnia at sea level before ascent to the high altitude locations. Chapter 3 reports the findings on the changes to sleep at high altitude, with particular focus on changes in the amounts of total sleep, the duration of each sleep stage and its percentage of total sleep, and the number and causes of arousals from sleep that occurred during sleep at increasing altitudes. The lightest stage of sleep, Stage 1 non-rapid eye movement (NREM) sleep, was increased, as expected with increases in altitude, while the deeper stages of sleep (Stages 3 and 4 NREM sleep, also called slow wave sleep), were decreased. The increase in Stage 1 NREM in this research is in agreement with all previous findings. However, slow wave sleep, although decreased, was present in most of our subjects at all altitudes in Nepal; this finding is in contrast to most previous work, which has found a very marked reduction, even absence, of slow wave sleep at high altitude. Surprisingly, unlike experimental animal studies of chronic hypoxia, REM sleep was well maintained at all altitudes. Stage 2 NREM and REM sleep, total sleep time, sleep efficiency and spontaneous arousals were maintained at near sea level values. The total arousal index was increased with increasing altitude and this was due to the increasing severity of periodic breathing as altitude increased. An interesting finding of this research was that fewer than half the periodic breathing apneas and hypopneas resulted in arousal from sleep. There was a minor degree of upper airway obstruction in some subjects at sea level but this was almost resolved by 3500m. Chapter 4 reports the findings on the effects on breathing during sleep of the progressive increase of altitude, in particular the occurrence of periodic breathing. This Chapter also reports the results of changes to arterial blood gases as subjects ascended to higher altitudes. As expected, arterial blood gases were markedly altered at even the lowest altitude in Nepal (1400m) and this change became more pronounced at each new, higher altitude. Most subjects developed periodic breathing at high altitude but there was a wide variability between subjects as well as variability in the degree of periodic breathing that individual subjects developed at different altitudes. Some subjects developed periodic breathing at even the lowest altitude and this increased with increasing altitude; other subjects developed periodic breathing at one or two altitudes, while four subjects did not develop periodic breathing at any altitude. Ventilatory responses to hypoxia and hypercapnia, measured at sea level before departure to high altitude, was not significantly related to the development of periodic breathing when the group was analysed as a whole. However, when the subjects were grouped according to the steepness of their ventilatory response slopes, there was a pattern of higher amounts of periodic breathing in subjects with steeper ventilatory responses. Chapter 5 reports the findings of an experimental study carried out in the University of California, San Diego, Barcroft Laboratory on White Mountain in California. Seven subjects drove from sea level to 3800m in one day and stayed at this altitude for two nights. On one of the nights the subjects slept using a non-invasive positive pressure device via a face mask and this was found to significantly improve the sleeping oxyhemoglobin saturation. The use of the device was also found to eliminate the symptoms of Acute Mountain Sickness, as measured by the Lake Louise scoring system. This finding appears to confirm the hypothesis that lower oxygen saturation, particularly during sleep, is strongly correlated to the development of Acute Mountain Sickness and may represent a new treatment and prevention strategy for this very common high altitude disorder.
Patout, Maxime. "Evaluation des techniques pour la prise en charge diagnostique et thérapeutique de l'insuffisance respiratoire chronique A Randomized controlled trial on the effect of needle gauge on the pain and anxiety experienced during radial arterial puncture Long term survival following initiation of home non-invasive ventilation : a European study Neural respiratory drive predicts long-term outcome following admission for exacerbation of COPD : a post hoc analysis Neural respiratory drive and cardiac function in patients with obesity hypoventilation syndrome following initiation of non-invasive ventilation Polysomnography versus limited respiratory monitoring and nurse-led titration to optimise non-invasive ventilation set-up a pilot randomised clinical trial Chronic ventilator service Step-down from non-invasive ventilation to continuous positive airway pressure : a better phenotyping is required AVAPS-AE versus ST mode : a randomized controlled trial in patients with obesity hypoventilation syndrome Technological advances in home non-invasive ventilation monitoring : reliability of data and effect on patient outcomes Efficacy of a home discharge care bundle after acute exacerbation of COPD Prediction of severe acute exacerbation using changes in breathing pattern of COPD patients on home noninvasive ventilation Charasteristics and outcome of patients set up on high-flow oxygen therapy at home Trial of portable continuous positive airway pressure for the management of tracheobronchomalacia". Thesis, Normandie, 2019. http://www.theses.fr/2019NORMR115.
Pełny tekst źródłaSingle-organ respiratory failure defines chronic respiratory failure. Obesity hypoventilation syndrome is the main cause of chronic respiratory failure and occurs in 4 to 5% of obese patients. Chronic respiratory failure is also the end-stage evolution of chronic obstructive pulmonary disease that has a prevalence of 6 to 8% in the adult population. The incidence of these diseases increases so does the incidence of chronic respiratory failure. In this thesis, we will evaluate novel diagnostic and therapeutic modalities that could improve the care of patients with chronic respiratory failure. Regarding diagnostic modalities, we have seen that evaluating the work of breathing with surface parasternal electromyography was an independent prognostic marker in patients with chronic obstructive pulmonary disease. We have also seen that it was a relevant tool to predict the clinicalefficacy and compliance to home non-invasive ventilation. Regarding therapeutic modalities, we have shown that the use of a semi-automatic mode of non-invasive ventilation had the same efficacy of a standard mode with a shorter length of stay for its setup. We have shown the relevance and feasibility of the use of high-flow oxygen therapy in the home setting whilst it was only used in intensive care units. Finally, we have shown the benefits of continuous positive airway pressure during exertion in patients with tracheobronchomalacia. Regarding patients’ follow-up, we have shown that the use of data from built-in software could predict the onset of a severe exacerbation of chronic obstructive pulmonary disease. However, we also show that the implementation of tele-medicine in patients with chronic respiratory failure cannot be included in daily clinical practice yet. In this thesis, we have identified novel physiological tools, novel ways to administer treatments and novel follow-up tools that can improve the management of patients with chronic respiratory failure
Anjos, Carlos Frederico Dantas. "Ventilação mecânica não invasiva com pressão positiva em vias aéreas, em pacientes HIV/AIDS com lesão pulmonar aguda e insuficiência respiratória: estudo de avaliação do melhor valor de PEEP". Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/5/5167/tde-09112011-134108/.
Pełny tekst źródłaINTRODUTION: The acquired immunodeficiency syndrome (AIDS) is a pandemic, and lung diseases are the leading cause of morbidity and mortality and are often associated with respiratory infections, hypoxemia and death. The noninvasive ventilation with positive pressure refers to the provision of mechanical ventilatory assistance without the need for artificial airway invasion, being recognized for improving oxygenation and dyspnea in patients with hipoxemic respiratory failure. Patients with AIDS and hypoxemic respiratory failure often require invasive mechanical ventilation, which is independently associated with mortality. Given the uncertainties about response in oxygenation with PEEP in patients with AIDS with acute hypoxemic respiratory failure and using the rational for progressive pressurization of the airway and its potential benefits on blood oxygenation, we made the hypothesis that increased levels of sequential PEEP up to 15 cmH2O may improve blood oxygenation without affecting the comfort and hemodynamics of the patient. The main objective of this study was to investigate the effects of different sequences of PEEP levels on gas exchange, the sensation of dyspnea and hemodynamics in patients with AIDS and acute hypoxemic respiratory failure. The secondary objective was to assess the time free of invasive mechanical ventilation in 28 days and hospital mortality within 60 days. METHODS: We studied 30 adults patients with HIV/AIDS and acute hypoxemic respiratory failure. All patients received a randomized sequence of noninvasive PEEP (the values used were 5,10 or 15 cmH2O) for twenty minutes. PEEP was delivered via face mask with pressure support (PSV) of 5 cmH2O and FiO2 = 1. A washout period of 20 minutes with spontaneous breathing was allowed between each PEEP trial. Clinical variables and arterial blood gases were recorded after each PEEP step. RESULTS: Analyzing the 30 patients, oxygenation improved linearly with increasing PEEP, however studying the patients randomized according to the initial PEEP, oxygenation was similar regardless of the first randomized PEEP (5,10 or 15 cmH2O), and only the subgroup with initial PEEP = 5 cmH2O further improve the oxygenation when high PEEP were used. The PaCO2 also rose beside the PEEP elevation, especially with a PEEP = 15 cmH2O. The use of PSV = 5 cmH2O was associated with significant and consistent improvement of subjective sensation of dyspnea and respiratory rate with a PEEP from 0 to 15 cmH2O. CONCLUSION: AIDS-patients with hypoxemic respiratory failure improve oxygenation with a progressive sequential elevation of PEEP up to 15 cmH2O, however the elevation of PaCO2 limit the PEEP up to 10 cmH2O. A PSV = 5 cmH2O promotes an improvement of subjective sensation of dyspnea independently from the use of PEEP
Reis, Michel Silva. "Modulação autonômica da freqüência cardíaca de homens saudáveis e pacientes com disfunções cardiorrespiratórias crônicas". Universidade Federal de São Carlos, 2007. https://repositorio.ufscar.br/handle/ufscar/5210.
Pełny tekst źródłaUniversidade Federal de Sao Carlos
The changes on the heart rate sympathetic-vagal balance caused to chronic obstructive pulmonary disease or chronic heart failure, as well as, the hemodynamics change induced by the non-invasive ventilation were unclear. In this context, we proposed to develop two studies. The first study was titled by The heart rate autonomic control in chronic obstructive pulmonary disease and chronic heart failure patients on the rest and during the respiratory sinusal arrhythmia maneuver . The purpose of this study was to evaluate the heart rate (HR) autonomic modulation in chronic obstructive pulmonary disease (COPD) patients as well as chronic heart failure (CHF) patients on the rest as well as during the respiratory sinus arrhythmia maneuver (M-RSA); and to correlation the HR autonomic modulation and seriousness levels of both pathologies. Twenty-seven male volunteers were subdivided in three groups: ten presented COPD (69±9 years); seven presented CHF (62±8 years) and; ten were healthy with 64±5 year-old (control). When resting, the three groups electrocardiography signal was obtained in three conditions: 1) lying position for 15 min; 2) lying position during the M-RSA for 4 min; and 3) sitting position for 15 min. The data was analyzed by the time (RMSSD and SDNN indexes) and the frequency domain, in total power, low frequency, high frequency absolute (ab) and normalized (nu) units and LF/HF ratio. Regarding the M-RSA indexes, the expiratory/inspiratory ratio (E/I) and the inspiratory/expiratory difference (∆IE) were calculated. The main results showed that the CHF and the COPD patients presented lower E/I ratio values (0,03±0,01 vs 0,09±0,04 e 0,04±0,02 vs 0,09±0,04) and ∆I/E values (0,67±0,13 vs 1,09±0,13 e 0,81±0,20 vs 1,09±0,13), when compared to control group. Strong correlations were observed between the forced expiratory volume in the first second (FEV1) and the RMSSD (r=-0,73) and between the FEV1 and the BF absolute (r=-0,71) in the COPD patients. At same, strong correlations were observed between the ejecting fraction and the RMSSD (r=0,83) in CHF patients. Concluding, the results of this study suggest that both, the COPD and the CHF patients, presented parasympathetic activity reduction and there is a relation between the seriousness levels of both pathologies and the HR autonomic activity. The second study was titled to The acute effects of the continuous positive airway pressure (CPAP) in the heart rate autonomic control of chronic obstructive pulmonary disease and chronic heart failure patients . The purpose of this study was to evaluate the acute continuous positive pressure airway over the heart rate (HR) autonomic control and the respiratory variables behavior in COPD as well as CHF patients. Twenty-eight male volunteers were sub-divided in three groups: ten presented COPD (69±9 years); eight presented CHF (62±8 years) and; ten were healthy with 64±5 year-old (control). The electrocardiography signal was obtained for 10 min in the sitting position with spontaneous breath (SB) and following randomly conditions: CPAP Sham, CPAP 5, and CPAP 10 cmH2O. Additionally, the breath rate, the endtidal of carbon dioxide, and the peripheral oxygen saturation were obtained. The HR and it variability data were analyzed by the time and the frequency domain, in according with previous describe. The main results showed that the ETCO2 reduced in all groups during the CPAP application. COPD group were significantly lower values of the RMSSD index in the Sham (1.06), CPAP 5 (1.08), and CPAP 10 (1,01) than SB (1,22). In addition, they presented increased in the LFnu (1.60 vs 1.82) and decreased in the HFab (1.90 vs 1.55) from the SB to CPAP 10. The CHF group RMSSD index and TP increased to SB (1.31 and 2.62) to CPAP 5 (1.44 and 2.87) and the CPAP 10 (1.48 and 2.97), respectively. Concluding, the CPAP caused modification in the HR autonomic control and improvement in the alveolar ventilation of COPD, CHF patients and healthy individuals.
As modificações do balanço simpato-vagal da freqüência cardíaca (FC) provocadas com o curso da doença pulmonar obstrutiva crônica (DPOC) e da insuficiência cardíaca crônica (ICC), bem como, as que ocorrem em função dos ajustes hemodinâmicos induzidos pela aplicação da ventilação não invasiva são bastante contraditórias. Neste contexto, propusemos o desenvolvimento de dois estudos que poderiam contribuir com novas informações. O primeiro intitulado por Controle autonômico da freqüência cardíaca de pacientes com doença pulmonar obstrutiva crônica ou insuficiência cardíaca crônica em repouso e durante a manobra de acentuação arritmia sinusal respiratória teve por objetivos avaliar o controle autonômico da FC de pacientes com DPOC ou ICC em repouso e durante uma manobra de acentuação da arritmia sinusal respiratória (M-ASR), bem como, relacionar a atividade autonômica da FC com a gravidade das patologias. Vinte e sete voluntários do sexo masculino foram subdivididos em três grupos: 10 com DPOC (GD) e 69±9 anos; 7 com ICC (GI) e 62±8 anos; e 10 saudáveis (GC) com 64±5 anos. Em repouso, o sinal eletrocardiográfico foi obtido em três situações: 1) 15 min na posição supina; 2) 4 min durante M-ASR na posição supina; e 3) 15 min na posição sentada. Os dados foram analisados no domínio do tempo (índices RMSSD e SDNN) e da freqüência, pela densidade espectral total (DET), bandas de baixa (BF) e alta freqüências (AF) - absolutas (ab) e normalizadas (un), e a razão BF/AF. Durante M-ASR foram calculadas a razão expiração/inspiração (E/I) e a diferença inspiração/expiração (∆IE). Os principais resultados em logaritmos decimais (média±desvio-padrão) mostraram que os pacientes com ICC e DPOC apresentaram menor razão E/I (0,03±0,01 vs 0,09±0,04 e 0,04±0,02 vs 0,09±0,04) e ∆IE (0,67±0,13 vs 1,09±0,13 e 0,81±0,20 vs 1,09±0,13), respectivamente, comparados ao GC durante a M-ASR. Correlações fortes foram observadas entre volume expiratório forçado no primeiro segundo com o RMSSD (r=-0,73) e com a BF absoluta (r=-0,71) nos pacientes com DPOC; e entre fração de ejeção e o RMSSD (r=0,83) nos pacientes com ICC. Em conclusão, os resultados sugerem que tanto a DPOC como a ICC levam a redução da atividade parassimpática e que a gravidade de ambas está relacionada com o controle autonômico da FC. O segundo estudo com o titulo: Efeitos da aplicação aguda da pressão positiva continua nas vias aéreas sobre o controle autonômico da freqüência cardíaca de pacientes com doença pulmonar obstrutiva crônica ou insuficiência cardíaca crônica , objetivou avaliar o efeito agudo da pressão positiva continua nas vias aéreas (CPAP) sobre o controle autonômico da freqüência cardíaca (FC) e o comportamento de variáveis respiratórias de pacientes com DPOC ou ICC. 28 homens foram subdivididos em três grupos: 10 com DPOC (GD) e 69±9 anos; 8 com ICC (GI) e 62±8 anos; e 10 saudáveis (GC) com 64±5 anos. O sinal eletrocardiográfico foi obtido por 10 min na posição sentada com respiração espontânea (RE) e randomicamente nas condições: CPAP sham, CPAP 5 e CPAP 10 cmH2O. Adicionalmente, foram obtidos os valores da freqüência respiratória, o volume de dióxido de carbono no final da expiração (ETCO2) e a saturação periférica de oxigênio. A FC e sua variabilidade foram analisadas no domínio do tempo e da freqüência, conforme descrito anteriormente. Os principais resultados mostraram que o ETCO2 reduziu em todos os grupos durante a aplicação da CPAP. O GD apresentou menores valores do RMSSD durante a CPAP sham (1,06), 5 (1,08) e 10 (1,01) em comparação a RE (1,22), bem como, aumento da BFun (1,60 vs 1,82) e redução da AFab (1,90 vs 1,55) da RE para a CPAP 10. No GI, o SDNN e a DET aumentaram da condição de RE (1,31 e 2,62) para CPAP 5 (1,44 e 2,87) e 10 (1.48 e 2,97), respectivamente. Os resultados sugerem que a CPAP melhorou a ventilação alveolar e provocou atenuação da atividade simpática sobre a FC de pacientes com ICC, bem como redução do tônus vagal de pacientes com DPOC.
Roos, Kerstin. "Omvårdnadsdokumentation för patienter med kronisk obstruktiv lungsjukdom som behandlas med Non-invasiv ventilation : en journalgranskning". Thesis, Högskolan Kristianstad, Sektionen för hälsa och samhälle, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:hkr:diva-13934.
Pełny tekst źródłaNewnam, Katherine. "A Comparative Effectiveness Study of Continuous Positive Airway Pressure (CPAP) Related Skin Breakdown when using Different Nasal Interfaces in the Extremely Low Birth Weight (ELBW) Neonate". VCU Scholars Compass, 2013. http://scholarscompass.vcu.edu/etd/510.
Pełny tekst źródłaKsiążki na temat "Non invasive positive pressure ventilation"
Christine, Mikelsons, red. Non-invasive respiratory support techniques: Oxygen therapy, non-invasive ventilation, and CPAP. Chichester, West Sussex: Wiley-Blackwell, 2008.
Znajdź pełny tekst źródłaRafferty, Mary Sara. A structural description of the experiences of individuals with severe Chronic Obstructive Pulmonary Disease using domiciliary non-invasive positive pressure ventilation. (s.l: The Author), 2001.
Znajdź pełny tekst źródłaSpoletini, Giulia, i Nicholas S. Hill. Non-invasive positive-pressure ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0090.
Pełny tekst źródłaMasip, Josep, Kenneth Planas i Arantxa Mas. Non-invasive ventilation. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0025.
Pełny tekst źródłaMasip, Josep, Kenneth Planas i Arantxa Mas. Non-invasive ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0025_update_001.
Pełny tekst źródłaMasip, Josep, Kenneth Planas i Arantxa Mas. Non-invasive ventilation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0025_update_002.
Pełny tekst źródłaMasip, Josep, Kenneth Planas i Arantxa Mas. Non-invasive ventilation. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0025_update_003.
Pełny tekst źródłaEsmond, Glenda, i Christine Mikelsons. Non-Invasive Respiratory Support Techniques: Oxygen Therapy, Non-Invasive Ventilation and CPAP. Wiley & Sons, Incorporated, John, 2009.
Znajdź pełny tekst źródłaDabo, Liu. Non-Invasive Positive Pressure Ventilation for Pediatric Sleep-Disordered Breathing. Nova Science Publishers, Incorporated, 2014.
Znajdź pełny tekst źródłaKreit, John W. Noninvasive Mechanical Ventilation. Redaktor John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0016.
Pełny tekst źródłaCzęści książek na temat "Non invasive positive pressure ventilation"
Marik, Paul Ellis. "Non-invasive Positive-Pressure Ventilation". W Handbook of Evidence-Based Critical Care, 175–81. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-5923-2_15.
Pełny tekst źródłaGilbert, Christopher R., i Philippe Haouzi. "Noninvasive Positive Pressure Ventilation in Acute Respiratory Failure". W Nocturnal Non-Invasive Ventilation, 247–69. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7624-6_10.
Pełny tekst źródłaStrollo, Patrick J., i John M. Coleman. "Noninvasive Positive Pressure Ventilation: Systems for Tracking Adherence and Efficacy". W Nocturnal Non-Invasive Ventilation, 209–34. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7624-6_8.
Pełny tekst źródłaBrochard, L., i D. Isabey. "Non-Invasive Positive Pressure Ventilation for Acute Respiratory Failure". W Yearbook of Intensive Care and Emergency Medicine 1994, 554–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-85068-4_51.
Pełny tekst źródłaBauman, Kristy A. "Acute Exacerbation of COPD: Non-invasive Positive Pressure Ventilation". W Evidence-Based Critical Care, 199–204. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-43341-7_22.
Pełny tekst źródłaSandri, Fabrizio, i Gina Ancora. "Continuous Positive Airways Pressure and other Non-Invasive Ventilation Techniques". W Neonatology, 509–21. Milano: Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-1405-3_69.
Pełny tekst źródłaJoho, Shuji. "Noninvasive Positive Pressure Ventilation". W Update of Non-Pharmacological Therapy for Heart Failure, 25–35. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-4843-7_4.
Pełny tekst źródłaBraun, Norma MT. "Negative Pressure Noninvasive Ventilation (NPNIV): History, Rationale, and Application". W Nocturnal Non-Invasive Ventilation, 27–71. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7624-6_2.
Pełny tekst źródłaSandri, Fabrizio, Gina Ancora, Gianluca Lista i Luc J. I. Zimmermann. "Continuous Positive Airways Pressure and Other Non-invasive Respiratory Techniques in Newborns". W Neonatology, 1–22. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-18159-2_209-1.
Pełny tekst źródłaOrtega, Yessika María, Isabel Cristina Muñoz i Alher Mauricio Hernández. "Work of Breathing Dynamics Under Changes of PEEP and Pressure Support in Non-invasive Mechanical Ventilation". W Communications in Computer and Information Science, 408–17. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-00353-1_36.
Pełny tekst źródłaStreszczenia konferencji na temat "Non invasive positive pressure ventilation"
Ma, Xuesong, Jiahua Zhu, Wenpeng Gao i Yili Fu. "A Novel Robot System Assisting Non-invasive Positive Pressure Ventilation". W 2018 IEEE International Conference on Information and Automation (ICIA). IEEE, 2018. http://dx.doi.org/10.1109/icinfa.2018.8812411.
Pełny tekst źródłaPobeha, Pavol, Imrich Mucska, Róbert Vyšehradský, Marta Hájková, Juraj Jakubička, Ivana Paraničová i Pavol Joppa. "Updated guidelines for indication of non-invasive positive pressure ventilation (NIPPV) in Slovakia". W ERS Respiratory Failure and Mechanical Ventilation Conference 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/23120541.rfmvc-2020.09.
Pełny tekst źródłaBaba, Yuya, Fumihiko Takatori, Masayuki Inoue i Isao Matsubara. "A Novel Mainstream Capnometer System for Non-invasive Positive Pressure Ventilation". W 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9175950.
Pełny tekst źródłaObeid, Imad M., Melisa A. Coaker, Luisa F. Bazan i David W. Hudgel. "High Grade Heart Block Therapy With Non-Invasive Positive Pressure Ventilation". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3698.
Pełny tekst źródłaHolcroft, John J., Christoph Lehmann, Stacey Mann, Joseph Dwyer, James Fackler, Margaret Birdson, Kimberly McIltrot i Ronald B. Easley. "Non-Invasive Positive Pressure Ventilation Utilization In Children With Respiratory Failure". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3905.
Pełny tekst źródłaPenuelas, Oscar, Fernando Frutos-Vivar, Andres Esteban, Niall D. Ferguson, Konstatinos Raymondos, Du Bin, Marco Gonzalez i in. "Factor Associated To Failure And Outcome Of Non-Invasive Positive Pressure Ventilation". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3741.
Pełny tekst źródłaJimenez, Manuel J., Nathaniel Marchetti, Nicholas S. Hill, Frank C. Sciurba, Patrick J. Strollo i Gerard J. Criner. "High Intensity Non-Invasive Positive Pressure Ventilation (NPPV) For Stable Hypercapnic COPD Patients". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5784.
Pełny tekst źródłaYasui, Hideki, Tomoyuki Fujisawa, Masato Karayama, Yutaro Nakamura, Naoki Inui, Koushi Yokomura, Takafumi Suda i Kingo Chida. "Usefulness Of Non-Invasive Positive Pressure Ventilation (NPPV) For Acute Exacerbation Of Asthma". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3141.
Pełny tekst źródłaMehta, Bhavin, Rohit Goyal, Roman Reznik, Jazeela Fayaaz, Charles Allred, Larry DiFabrizio, Klaus Lessnau i Murray Rogers. "Hospital Utilization Of Bilevel Non-Invasive Positive Pressure Ventilation Could Become More Cost-effective". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3052.
Pełny tekst źródłaSidhom, SS, A. Ozsancak, P. Alkana, A. Khodabandeh, V. Maheshwari i NS Hill. "Impact of an Educational Intervention on the Utilization of Non-Invasive Positive Pressure Ventilation." W American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3037.
Pełny tekst źródła