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Artykuły w czasopismach na temat "Neuropsychology"

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Caramazza, Alfonso. "Is Cognitive Neuropsychology Possible?" Journal of Cognitive Neuroscience 4, nr 1 (styczeń 1992): 80–95. http://dx.doi.org/10.1162/jocn.1992.4.1.80.

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Cognitive neuropsychology's domain of inquiry concerns the structure of normal perceptual, motor, and cognitive processes. As such, it constitutes a branch of cognitive science. Cognitive neuropsychology differs from other branches of cognitive science only by the type of observation that it uses in developing and evaluating theories of normal cognition. The data used in cognitive neuropsychology are the patterns of performance produced by brain-damged subjects. Because the basic data used in cognitive neuropsychology are the result of a biological manipulation—a brain lesion—these data will be relevant to claims about the functional organization of the brain. Hence, cognitive neuropsychology may also be considered to be a branch of cognitive neuroscience. However, in this paper I will be concerned with an assessment of research programs whose principal or only aim is to constrain theories of normal cognitive functioning through the analysis of acquired disorders of cognition. Following a brief discussion of the basic assumptions that motivate cognitive neuropsychological research, I consider Kosslyn and Van Kleeks (1990) claim that the study of brain-damaged subjects for the purpose of constraining theories of normal cognitive processing cannot lead to meaningful conclusions unless the theories are directly cast in terms of anatomical and physiological facts. I argue that these authors conflated criticisms that may apply to any empirical science with criticisms that may apply specifically to cognitive neuropsychology. Separate consideration of the criticisms specific to cognitive neuropsychology reveals that these are unfounded. The main point of this discussion is to emphasize the pragmatic character of the motivation for using impaired performance to constrain theories of normal cognition. The usefulness of cognitive neuropsychological research is illustrated through specific examples.
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Milberg, William. "A Valid Assessment of Contemporary Neuropsychology?" Journal of the International Neuropsychological Society 5, nr 3 (marzec 1999): 269–70. http://dx.doi.org/10.1017/s1355617799253106.

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This collection of generally well written reviews is sure to elicit a variety of reactions from readers interested in the field of clinical neuropsychology. Many, particularly those new to neuropsychology will likely appreciate this well organized and clearly written compilation of chapters edited by Gerald Goldstein and Theresa M. Incagnoli. The 12 chapters written by some of clinical neuropsychology's plenipotentiaries cover the main assessment systems currently employed in the United States. There are also chapters concerned with the evaluation of such special populations as severely demented older adults and children. In addition there are introductory chapters covering longstanding debates that still occupy some quarters of the neuropsychological community. To this group of readers Contemporary Approaches to Neuropsychological Assessment will serve as an intellectual “county fair” where experts have proudly displayed the best of their “wares”: the test systems and ideas with which they have long been associated. However, there will also be readers who will find this compilation disconcerting. From the perspective of many clinicians who are more familiar with the field, including those who must make a living in the real world of managed care and the increasing disillusionment of traditional medical allies of clinical neuropsychology (e.g., behavioral neurology, the court system, etc.), this volume will appear overly optimistic and in some cases anachronistic.
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Gupta, P. D. "Neuropsychology of Expectant Mothers". Journal of Clinical Surgery and Research 2, nr 3 (29.07.2021): 01–02. http://dx.doi.org/10.31579/2768-2757/017.

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Pregnancy is a special event in woman's life; during this time period and sometimes after childbirth, not only her lifestyle changes but her physiology, endocrinology, and neuropsychology also changes. Because of all these, her body structure and nutritional status along with her likings and dislikings and behavior patterns also changes.
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Mascoop, Susan. "Neuropsychology". Psychiatric Services 50, nr 7 (lipiec 1999): 978a—979. http://dx.doi.org/10.1176/ps.50.7.978a.

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Rabbitt, Patrick M. A. "Neuropsychology". Current Opinion in Psychiatry 3, nr 4 (sierpień 1990): 507–11. http://dx.doi.org/10.1097/00001504-199008000-00019.

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Lovell, Mark. "Neuropsychology". Journal of Neuropsychiatry and Clinical Neurosciences 1, nr 1 (luty 1989): 78–80. http://dx.doi.org/10.1176/jnp.1.1.78.

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Lovell, Mark R. "Neuropsychology". Journal of Neuropsychiatry and Clinical Neurosciences 1, nr 2 (maj 1989): 202–4. http://dx.doi.org/10.1176/jnp.1.2.202.

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Lovell, Mark R. "Neuropsychology". Journal of Neuropsychiatry and Clinical Neurosciences 1, nr 3 (sierpień 1989): 327–29. http://dx.doi.org/10.1176/jnp.1.3.327.

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Lovell, Mark R. "Neuropsychology". Journal of Neuropsychiatry and Clinical Neurosciences 1, nr 4 (listopad 1989): 426–28. http://dx.doi.org/10.1176/jnp.1.4.426.

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Lovell, Mark R. "Neuropsychology". Journal of Neuropsychiatry and Clinical Neurosciences 2, nr 1 (luty 1990): 104–6. http://dx.doi.org/10.1176/jnp.2.1.104.

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Rozprawy doktorskie na temat "Neuropsychology"

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Shanker, Shanti. "Neuropsychology of motivated forgetting". Thesis, Bangor University, 2016. https://research.bangor.ac.uk/portal/en/theses/neuropsychology-of-motivated-forgetting(74254543-1b02-4c4d-b6f8-da577cca45db).html.

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When confronted with an unwelcome reminder, people often inhibit the unwanted memory from awareness, a process that causes forgetting. This suppression-induced forgetting (SIF), also sometimes known as motivated forgetting, can be empirically measured by the Think/No-Think (TNT) task. Chapter 1 reviews the literature on memory inhibition. Imaging work indicates suppressing retrieval engages the right dorsolateral prefrontal cortex (rDLPFC), which in turn may inhibit the retrieval processes within the hippocampus. This thesis, using a range of methods, aims to better understand the neuropsychology of motivated forgetting. Chapter 2 investigates whether the ability to inhibit unwanted memories can be modulated through electrical stimulation. Stimulation methods do not appear to improve inhibition, at least in this cohort, one possible reason for this being the increased perceived thought control ability. Chapter 3 reports the first ever adaptation of the TNT task in patients with unilateral frontal lesions. Pilot testing in Chapter 4 reports the study in patients with unilateral frontal lesions. The results suggested that patients with left frontal lesions showed a robust SIF, compared to those with right-frontal lesions who showed none. Finally, Chapter 5 attempted to identify the structural connectivity underlying inhibitory control of motivated forgetting. The results indicate that the DLPFC is connected to the hippocampus by a subset of the two tracts, namely the anterior thalamic projection connecting the DLPFC to the caudate nucleus, and the fornix. Future directions to expand on the finding of this thesis are discussed in Chapter 6.
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Deakin, J. B. "The neuropsychology of decision-making". Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598466.

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This thesis is concerned with emotional decision-making and impulsivity. In a series of experiments, the processes involved in emotional decision-making were investigated. Techniques used included both established, neuropsychologically validated tasks known to be dependent on frontal lobe function, and an entirely novel paradigm. A probabilistic decision-making and gambling task has been widely used to delineate the differences between pathological groups and normal volunteers, and attempts to distinguish between different components of impulsivity. In a large cohort of healthy volunteers, older volunteers took longer to make poorer decisions but risked less when gambling. This pattern of changes is unlike the patterns demonstrated by any of the pathological groups previously reported. A factor analysis of the results suggest that the variability across healthy volunteers on the four measures of impulsivity provided by the task can be explained by two orthogonal components, only one of these is influenced by age. Delay aversion is an unusual phenomenon in adult humans. A new task was designed so that optimal choice and delay-averse behaviour could be distinguished. In previous delay aversion tasks, volunteers can either pick the best option and be delay tolerant or the less optimal choice and be delay-averse. I predicted that one would be able to detect smaller variations in delay aversion when there was less of a cost to the delay-averse choice. Normal adults chose optimally and did not demonstrate delay aversion. When age-groups were compared it was seen that older adults did not choose optimally, but in fact choose the sub-optimal and most delay-tolerant option. A group of individuals known to have a decision-making deficit are those with frontal variant frontotemporal dementia. It was hypothesised that this was due to a lack of availability of 5-HT which could be corrected by paroxetine. A double-blind randomised controlled trial was used to detect improved performance on this task (and other tasks sensitive to tryptophan depletion) as well as any change in symptoms using a course of paroxetine (an SSRI). Paroxetine did not improve symptoms and in fact impaired test performance. Surprisingly, long term administration of paroxetine produced a pattern of effects on the test battery which was almost indistinguishable from that of tryptophan depletion. Diazepam is known to have receptors distributed throughout the prefrontal cortex but its effects on tests of orbitofrontal function have never before been examined. In contrast to the well-known sedative effects of diazepam, I demonstrate disinhibitory effects on two speeded reaction time tasks. It is shown that diazepam can impair performance on reaction time tasks both by impairing sensitivity and by increasing the bias to respond. Furthermore diazepam impaired performance on tests of frontal lobe function including both tests of planning and decision-making that depend predominantly on dorsolateral and orbitofrontal regions of the PFC respectively. In summary, a variety of factors have been found that impinge on decision-making. It is demonstrated that older adults are less able to make an optimal choice both when deciding between rewards of different probabilities and when deciding between rewards which come after a different delay. Volunteers who take diazepam are more likely to pick the larger reward associated with the less optimal choice than normal volunteers and demonstrate impulsive behaviour in two speeded reaction time tests. Paroxetine had no effect on decision-making in an impaired group of volunteers with frontotemporal dementia but had a paradoxical effect of impairing performance in tasks that are impaired by damage to orbitofrontal cortex and by tryptophan depletion. Possible neuronal substrates for these effects are discussed, as are explanations for the surprising effects of paroxetine and future directions in therapeutics. Finally a tentative neurochemical and anatomical model is detailed that would account for the effects described here.
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Ayre, Gareth Andrew. "The comparative neuropsychology of dementia". Thesis, University of Newcastle Upon Tyne, 1998. http://hdl.handle.net/10443/850.

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On the basis of neuropathological, neurochemical, genetic, and clinical profile studies on patients, distinct forms of dementia, such as dementia with Lewy bodies (DLB), have been distinguished which were originally thought to be Alzheimer's disease (AD). Dementia with Lewy bodies is probably the second most common form of dementia in the elderly. In this thesis, a well characterised and investigated cohort of DLB and AD patients were compared to non-demented elderly controls in order to establish profiles of cognitive decline in these groups. Initially, comprehensively matched experimental groups were compared using the Cambridge Neuropsychological Test Automated Battery (CANTAB). The DLB group was less impaired than the AD group on a test of visual pattern recognition memory. However, the DLB group performed worse on a number of cognitive tests. Comparison of larger, carefully matched, experimental groups using the Cognitive Drug Research Computerised Assessment Battery (CDR) also revealed differences in the profile of cognitive impairment in DLB and AD. The DLB group showed more marked deficits in attentional abilities than the AD group. In particular, the DLB group were unable to sustain attention. Conversely, the DLB group were less impaired on a test of visual secondary recognition memory than the AD group. Further division of the DLB group into cases with and without persistent visual hallucinations revealed distinct patterns of cognitive impairment in these two groups. Generally, DLB cases with persistent visual hallucinations showed greater attentional and spatial working memory deficits than the DLB cases without persistent visual hallucinations. A final study compared decline in cognitive function over 1 year in DLB, AD and control groups. Similar rates of cognitive decline were identified in a number of cognitive domains in AD and DLB groups. In addition, disproportionate decline in the ability to sustain attention was identified in the DLB group. A comparative model relating known neuropsychological, neurochemical, and neuropathological features of DLB and AD was proposed.
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Alderdice, Fiona A. "The neuropsychology of alcohol abuse". Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334532.

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Charlesworth, G. "Neuropsychology of frontal lobe dementia". Thesis, University of East Anglia, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296874.

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Weber, Mareen. "The neuropsychology of sport concussion". Thesis, University of Birmingham, 2010. http://etheses.bham.ac.uk//id/eprint/1049/.

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This thesis presents four empirical chapters that challenge current sport concussion research and practice. Chapter 2 measured sport concussion knowledge in the UK general public using an online survey. It showed high sport concussion awareness, but limited and erroneous understanding. Chapter 3 examined the effect of terminology (i.e., concussion; mild traumatic brain injury, mTBI; minor head injury, mHI) on familiarity, injury outcome expectations and symptom self-report in athletes using a questionnaire. The mTBI terminology was the least familiar, reliably more negative conceptualised, but knowledge was more accurate than the other two. Symptom self-report did not vary with terminology or injury history. Chapter 4 compared the late neuropsychological functioning in self-reported sport-concussed to non-concussed athletes using a comprehensive test battery. Injury self-report was associated with worse memory recall and executive function shifting. Chapter 5 piloted a computerised neuropsychological test battery in athletes using a longitudinal control group design. A single case study showed transient deficits in memory recall and executive function at one to six weeks post-concussion. The overall data suggest that (i) education is needed; (ii) the interchangeable terminology use is inappropriate; (iii) sport concussion assessment should be complemented by memory recall and executive function tests; (iv) case studies might be more appropriate than group comparisons.
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Baker, David Wayne. "Neuropsychology and appropriate modes of instruction /". Access Digital Full Text version, 1989. http://pocketknowledge.tc.columbia.edu/home.php/bybib/10858489.

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Thesis (Ed.D.) -- Teachers College, Columbia University, 1989.
Typescript; issued also on microfilm. Sponsor: A. Harry Passow. Dissertation Committee: James H. Borland III. Bibliography: leaves 192-207.
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Hemberger, Helga Christine. "The neuropsychology of obsessive-compulsive symptoms". Thesis, The University of Sydney, 2007. http://hdl.handle.net/2123/1902.

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Obsessive-compulsive (OC) symptoms occur in a variety of clinical conditions, but the underlying pathogenesis of these symptoms remains elusive. Few neuropsychological investigations have compared idiopathic Obsessive-Compulsive Disorder (OCD) with patient groups where OC symptoms are acquired. The present study investigated the neuropsychological correlates of OC symptoms in OCD and frontotemporal dementia (FTD), a neurodegenerative illness in which OC symptoms are often acquired. Neuroimaging in OCD has consistently implicated the frontal-striatal-thalamic circuit, particularly the orbitofrontal cortex and basal ganglia. These areas overlap considerably with the sites of cerebral pathology found in FTD. OCD has been associated with a number of neuropsychological deficits, with most consistent findings pointing towards impaired executive function (EF), and less commonly reported deficits in visual memory and visuospatial ability. The neuropsychological hallmark of FTD is deficits in EF. However in both OCD and FTD, the relationship between cognitive deficits and OC symptoms remains unclear. Further, the extent to which OC symptoms are comparable between the groups is ambiguous. Part I of the present study compared 19 OCD subjects to 20 age, education and IQ-matched healthy controls on a battery of neuropsychological tests of all major cognitive domains with emphasis on EF. A measure of Theory of Mind (ToM) thought to be sensitive to orbitofrontal function was also administered. OCD subjects performed worse than controls on a measure of visual memory, visuospatial reasoning and on only one measure of EF. OCD symptom subtypes, as measured by the Obsessive-Compulsive Inventory (OCI), were not correlated with any cognitive deficits. No group differences in ToM were found. It is suggested that prior research has overestimated the severity and significance of EF deficits in OCD. Part II of the study compared 9 FTD participants with 10 matched healthy controls on the same neuropsychological test battery and OC symptom measures. In addition, a measure of compulsive behaviours used in neurological populations was administered to carers. While the incidence of OC symptoms was comparable to reports in previous studies (78%), the OCI was not sensitive in the detection of OC symptoms in FTD. The similarities and differences in OC symptoms between the two patient groups are discussed.
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Hemberger, Helga Christine. "The neuropsychology of obsessive-compulsive symptoms". University of Sydney, 2007. http://hdl.handle.net/2123/1902.

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Doctor of Clinical Psychology
Obsessive-compulsive (OC) symptoms occur in a variety of clinical conditions, but the underlying pathogenesis of these symptoms remains elusive. Few neuropsychological investigations have compared idiopathic Obsessive-Compulsive Disorder (OCD) with patient groups where OC symptoms are acquired. The present study investigated the neuropsychological correlates of OC symptoms in OCD and frontotemporal dementia (FTD), a neurodegenerative illness in which OC symptoms are often acquired. Neuroimaging in OCD has consistently implicated the frontal-striatal-thalamic circuit, particularly the orbitofrontal cortex and basal ganglia. These areas overlap considerably with the sites of cerebral pathology found in FTD. OCD has been associated with a number of neuropsychological deficits, with most consistent findings pointing towards impaired executive function (EF), and less commonly reported deficits in visual memory and visuospatial ability. The neuropsychological hallmark of FTD is deficits in EF. However in both OCD and FTD, the relationship between cognitive deficits and OC symptoms remains unclear. Further, the extent to which OC symptoms are comparable between the groups is ambiguous. Part I of the present study compared 19 OCD subjects to 20 age, education and IQ-matched healthy controls on a battery of neuropsychological tests of all major cognitive domains with emphasis on EF. A measure of Theory of Mind (ToM) thought to be sensitive to orbitofrontal function was also administered. OCD subjects performed worse than controls on a measure of visual memory, visuospatial reasoning and on only one measure of EF. OCD symptom subtypes, as measured by the Obsessive-Compulsive Inventory (OCI), were not correlated with any cognitive deficits. No group differences in ToM were found. It is suggested that prior research has overestimated the severity and significance of EF deficits in OCD. Part II of the study compared 9 FTD participants with 10 matched healthy controls on the same neuropsychological test battery and OC symptom measures. In addition, a measure of compulsive behaviours used in neurological populations was administered to carers. While the incidence of OC symptoms was comparable to reports in previous studies (78%), the OCI was not sensitive in the detection of OC symptoms in FTD. The similarities and differences in OC symptoms between the two patient groups are discussed.
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Cardinal, Rudolf N. "Neuropsychology of reinforcement processes in the rat". Thesis, University of Cambridge, 2001. https://www.repository.cam.ac.uk/handle/1810/236348.

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This thesis investigated the role played by regions of the prefrontal cortex and ventral striatum in the control of rats' behaviour by Pavlovian conditioned stimuli, and in their capacity to choose delayed reinforcement. First, the function of the anterior cingulate cortex (ACC) in simple Pavlovian conditioning tasks was addressed. The ACC is a subdivision of prefrontal cortex that has previously been suggested to be critical for the formation of stimulus-reward associations. It was found that lesions of the ACC did not prevent rats from learning a simple conditioned approach response to a conditioned stimulus (CS) predictive of food reward, or from utilizing that CS as a conditioned reinforcer subsequently. Additionally, these subjects successfully acquired a conditioned freezing response to a CS predicting footshock. However, the same animals were impaired at the acquisition of autoshaped behaviour, an impairment that has been demonstrated previously. An autoshaping deficit was also observed when lesions were made following training. The phenomenon of Pavlovian-instrumental transfer was intact in these subjects. The hypothesis was developed that the ACC is not critical for the formation of stimulus-reward associations per se, but is critical when multiple stimuli must be discriminated on the basis of their differential association with reward. In support of this hypothesis, animals with lesions of the ACC were impaired on a version of the conditioned approach task in which a second, neutral stimulus, perceptually similar to the CS, was added; the lesioned subjects exhibited reduced discrimination. Second, the role of the nucleus accumbens (Acb) in Pavlovian-instrumental transfer was investigated. The nucleus accumbens core, together with a larger amygdalar-striatal network of which it is a component, has previously been shown to be necessary for the expression of 'simple' Pavlovian-instrumental transfer. Rats with lesions of the nucleus accumbens core (AcbC) and shell (AcbSh) were tested on a 'response-specific' Pavlovian-instrumental transfer task, in which a Pavlovian CS selectively enhances instrumental responding for the outcome with which the CS was originally paired. AcbC lesions impaired the response specificity of this effect, while AcbSh lesions abolished Pavlovian-instrumental transfer entirely. These results are consistent with some - but not all - previous results in suggesting that the shell provides 'vigour' and the core provides 'direction' for the potentiation of behaviour by Pavlovian CSs. Third, an attempt was made to train rats on a task for assessing preference for delayed reinforcement, using the 'adjusting-delay' paradigm. It was not immediately apparent that the rats reacted to the contingencies operative in this task, and mathematical analysis of their behaviour was conducted to establish whether their behaviour was sensitive to the delay, and what 'molar' features of performance on this task could be explained by delay-independent processes. Fourth, a different delayed reinforcement choice task was developed, modifying a previously published task in which the subject is repeatedly offered a choice, in discrete trials, of a small reward delivered immediately, and a large reward delivered after a delay, with the delays systematically varied by the experimenter. Rats were trained on versions of this task in which the large, delayed reinforcer was or was not explicitly signalled by a cue present during the delay. The behavioural basis of performance on this task was examined, and d-amphetamine, chlordiazepoxide, and alpha-flupenthixol were administered systemically. It was found that the effects of d-amphetamine depended on whether the delayed reinforcer was signalled or unsignalled, increasing preference for signalled delayed reinforcement at some doses, but decreasing preference for unsignalled delayed reinforcement. These results may resolve contradictions in the literature, and are suggested to reflect the known effect of amphetamine to potentiate responding for conditioned reinforcers. Fifth, rats that had been trained on this task (with no explicit signals present during the delay) were given lesions of the ACC, AcbC, or medial prefrontal cortex (mPFC). ACC-lesioned rats were no different from sham-operated controls in their ability to choose a large, delayed reinforcer. Lesions of mPFC reduced the tendency of subjects to shift from one lever to the other during the course of a session, but mPFC-lesioned subjects responded normally to removal of the delays, suggesting a loss of stimulus control. However, rats with lesions of the AcbC were severely impaired on this task, preferring the small, immediate reward, even though they discriminated the reinforcers. Additionally, the effects of intra-Acb amphetamine were assessed using a different version of the delayed reinforcement choice task, and found to have slight but inconsistent effects to reduce preference for the delayed reinforcer, though this effect did not depend on whether the delayed reward was signalled or unsignalled. These results suggest that the AcbC contributes significantly to the rat's ability to choose a delayed reward, a finding that has important implications for the understanding of Acb function. It is suggested that dysfunction of the AcbC may be a key element in the pathology of impulsivity.
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Książki na temat "Neuropsychology"

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Isabelle, Rapin, i Segalowitz Sidney J, red. Handbook of neuropsychology. Amsterdam: Elsevier, 1992.

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Boulton, Alan A., Glen B. Baker i Merrill Hiscock. Neuropsychology. New Jersey: Humana Press, 1990. http://dx.doi.org/10.1385/0896031330.

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Smith, Alastair, i Chris Moulin. Neuropsychology. 1 Oliver's Yard, 55 City Road, London EC1Y 1SP United Kingdom: SAGE Publications Ltd, 2012. http://dx.doi.org/10.4135/9781446262610.

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Bizyuk, Aleksandr. Neuropsychology. ru: INFRA-M Academic Publishing LLC., 2021. http://dx.doi.org/10.12737/1039182.

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The textbook is based on the materials of well-known domestic and foreign manuals on neurology, publications of leading specialists in the field of neuropsychology and related disciplines. It contains basic information on the anatomy of the central nervous system; basic principles and theories that provide an understanding of the laws of the brain; psychological characteristics of symptoms and syndromes that develop in lesions of the cortex and subcortical structures; a brief description of neuropsychological tests and hardware techniques that allow identifying the topic of the lesion. Meets the requirements of the federal state educational standards of higher education of the latest generation. It is intended for students specializing in the field of clinical (medical) special psychology, speech therapy, as well as for students of retraining and advanced training courses in these specialties.
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Goldstein, Gerald, Paul David Nussbaum i Sue R. Beers, red. Neuropsychology. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4899-1950-2.

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A, Boulton A., Baker Glen B. 1947- i Hiscock Merrill, red. Neuropsychology. Clifton, N.J: Humana Press, 1990.

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W, Zaidel Dahlia, red. Neuropsychology. San Diego: Academic Press, 1994.

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Stirling, John D. Introducing neuropsychology. Wyd. 2. Hove, East Sussex: Psychology Press, 2008.

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Stirling, John D. Introducing neuropsychology. Wyd. 2. Hove, East Sussex: Psychology Press, 2008.

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Hodges, Elise K., Jeffrey G. Kuentzel i Julie N. Hook. Pediatric Neuropsychology. New York: Routledge, 2022. http://dx.doi.org/10.4324/9781003349075.

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Części książek na temat "Neuropsychology"

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Eglit, Graham M. L. "Neuropsychology". W Handbook of Intellectual Disabilities, 461–81. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-20843-1_26.

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Stringer, Anthony Y. "Neuropsychology". W Encyclopedia of Clinical Neuropsychology, 2440–44. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_671.

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Holmes, Cooper B. "Neuropsychology". W Your Career in Psychology, 271–82. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781444315929.ch20.

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Hoffman, Richard. "Neuropsychology". W Encyclopedia of Behavioral Medicine, 1496–99. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_267.

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Tiro, Jasmin, Simon J. Craddock Lee, Steven E. Lipshultz, Tracie L. Miller, James D. Wilkinson, Miriam A. Mestre, Barbara Resnick i in. "Neuropsychology". W Encyclopedia of Behavioral Medicine, 1328–31. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_267.

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Stringer, Anthony. "Neuropsychology". W Encyclopedia of Clinical Neuropsychology, 1–5. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_671-2.

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Davies, Roger, i Peter Houghton. "Neuropsychology". W Mastering Psychology, 243–54. London: Macmillan Education UK, 1995. http://dx.doi.org/10.1007/978-1-349-13553-0_15.

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Speh, Kathrin, i Sofia von Humboldt. "Neuropsychology". W Encyclopedia of Gerontology and Population Aging, 1–3. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-69892-2_78-1.

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McLellan, Tracey. "Neuropsychology". W Encyclopedia of Child Behavior and Development, 1014–15. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_1961.

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Hoffman, Richard. "Neuropsychology". W Encyclopedia of Behavioral Medicine, 1–4. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-6439-6_267-2.

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Streszczenia konferencji na temat "Neuropsychology"

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Poláček, Miroslav. "Neuropsychology in Neuromarketing Services". W International Scientific Days 2018. Wolters Kluwer ČR, Prague, 2018. http://dx.doi.org/10.15414/isd2018.s9.14.

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Glozman, J. M., i A. A. Zarechnaya. "PROBLEM OF GIFTNESS IN NEUROPSYCHOLOGY". W ПСИХОЛОГИЯ ТВОРЧЕСТВА И ОДАРЕННОСТИ. Москва: Ассоциация технических университетов, 2021. http://dx.doi.org/10.53677/9785919160472_268_272.

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Solovieva, Yulia, i Luis Quintanar. "Qualitative neuropsychological assessment of children". W 2nd International Neuropsychological Summer School named after A. R. Luria “The World After the Pandemic: Challenges and Prospects for Neuroscience”. Ural University Press, 2020. http://dx.doi.org/10.15826/b978-5-7996-3073-7.7.

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Qualitative assessment is an essential method of clinical neuropsychology. According to conceptual bases of historical and cultural neuropsychology, qualitative assessment should include analysis of the neuropsychological syndrome in each particular case. The paper presents the method for qualitative neuropsychological assessment created for children in Mexico for Spanish.speaking population aged 5–12 with the help of the Scheme for “Brief Neuropsychological Assessment for Children”. The structure and the ways for analysis of clinical data are included. The conclusions mention the possible applications of this instrument.
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REISCHIES, F. M., i B. GEISELMANN. "NEUROPSYCHOLOGY OF DEMENTIA AND DEPRESSION IN OLD AGE". W IX World Congress of Psychiatry. WORLD SCIENTIFIC, 1994. http://dx.doi.org/10.1142/9789814440912_0114.

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Chetitah, Mounsif, Julian Müller, Lorenz Deserno, Maria Waltmann i Sebastian von Mammen. "Gamification Framework for Reinforcement Learning-based Neuropsychology Experiments". W FDG 2023: Foundations of Digital Games 2023. New York, NY, USA: ACM, 2023. http://dx.doi.org/10.1145/3582437.3587190.

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Flax, L. "Algebraic modelling of some cognitive neuropsychology of schizophrenia". W Proceedings of the Third IEEE International Conference on Cognitive Informatics, 2004. IEEE, 2004. http://dx.doi.org/10.1109/coginf.2004.1327468.

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Chupakhina, S. V. "Basic aspects of speech formation: the theory of neuropsychology". W RESEARCH ACTIVITIES AND ACHIEVEMENTS IN PEDAGOGY AND PSYCHOLOGY. Baltija Publishing, 2023. http://dx.doi.org/10.30525/978-9934-26-312-5-46.

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Rotarescu, Violeta. "THE MENTAL MODEL OF PSYCHOLOGY STUDENTS FOR NEUROPSYCHOLOGY COURSE". W SGEM 2014 Scientific Conference on PSYCHOLOGY AND PSYCHIATRY, SOCIOLOGY AND HEALTHCARE, EDUCATION. Stef92 Technology, 2014. http://dx.doi.org/10.5593/sgemsocial2014/b11/s1.062.

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Tan, T. Z., G. S. Ng i S. S. Erdogan. "A Neuropsychology-inspired Learning System for Human Uncertainty Monitoring". W 2006 9th International Conference on Control, Automation, Robotics and Vision. IEEE, 2006. http://dx.doi.org/10.1109/icarcv.2006.345430.

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Markowitsch, Hans. "Autobiographical Memory across the Life Span - Brain Imaging and Neuropsychology". W 9th Conference of the Australasian Society for Cognitive Science. Sydney: Macquarie Centre for Cognitive Science, 2010. http://dx.doi.org/10.5096/ascs200934.

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Raporty organizacyjne na temat "Neuropsychology"

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De Lizo, Caroline V. Cost Effectiveness of Using Low-Bandwidth Video-Teleconferencing in the Neuropsychology Service at Naval Medical Center San Diego. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 1998. http://dx.doi.org/10.21236/ada372276.

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Paul-Brown, Diane. Evaluating and Treating Communication and Cognitive Disorders: Approaches to Referral and Collaboration for Speech-Language Pathology and Clinical Neuropsychology. Rockville, MD: American Speech-Language-Hearing Association, 2003. http://dx.doi.org/10.1044/policy.tr2003-00137.

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Reeves, Dennis L., i Steven L. Taube. Developments in Neuropsychological and Neurophysiological Assessment: An Overview of Progress and Products of the JWGD3 (Joint Working Group on Drug Dependent Degradation) Level I Neuropsychology Task Area Group. Fort Belvoir, VA: Defense Technical Information Center, styczeń 1986. http://dx.doi.org/10.21236/ada201852.

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Ernst, Thomas. Brain Function, Structure, and Neurochemistry After Tamoxifen/Chemotherapy Assessed by Neuropsychologic Testing and 1H Magnetic Resonance Spectroscopy. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2001. http://dx.doi.org/10.21236/ada403373.

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Chlebowski, Rowan. Brain Function, Structure, and Neurochemistry After Tamoxifen/Chemotherapy Assessed by Neuropsychologic Testing and H Magnetic Resonance Spectroscopy. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2002. http://dx.doi.org/10.21236/ada412888.

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Marchildon, Allison, Claire Boine, Andréane Sabourin Laflamme, Dave Anctil, Antoine Boudreau LeBlanc, Sylvain Auclair, Christine Balagué i in. Un an après l’arrivée de ChatGPT: Réflexions de l’Obvia sur les enjeux et pistes d’action possibles face à l’IA générative. Observatoire international sur les impacts sociétaux de l’intelligence artificielle et du numérique, styczeń 2024. http://dx.doi.org/10.61737/zkwz3721.

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Depuis l’automne 2022, avec l’arrivée abrupte de ChatGPT, la version gratuite du modèle de langage de l’entreprise OpenAI, les expérimentations à grande échelle du déploiement de ce système d’intelligence artificielle ont suscité un tsunami de réactions dont on perçoit les conséquences autant dans nos vies personnelles que professionnelles. En raison de l’ampleur des implications de cet événement, qui symbolise un changement hautement disruptif pour nos sociétés, il s’avérait incontournable qu’un groupe de chercheuses et de cherecheurs de l’Obvia s’y penche afin de contribuer à la réflexion sur le sujet et dégager des pistes d’action. Le but du présent document vise par conséquent à apporter des éléments de réflexion sur les différents enjeux que soulève l’émergence soudaine de ces technologies au potentiel révolutionnaire. Le présent document vise plus spécifiquement: • à situer dans son contexte le développement des applications d’IA générative; • à identifier ce qui nous semble être les principaux enjeux transversaux soulevés par l’arrivée de ces technologies dans nos vies et nos sociétés; • à esquisser quelques pistes d’action pour tenter de composer adéquatement avec ces enjeux. Ce document a été rédigé par une équipe multidisciplinaire de chercheuses et de chercheurs membres de l’Obvia. En effet, pour mener une réflexion sur une situation aussi large et complexe, il nous semblait essentiel de réunir des expertes et experts de plusieurs domaines, notamment en éthique appliquée, en neuropsychologie, en droit, en bioéthique et en philosophie. Les différentes thématiques qui émergent de cette réflexion ont été organisées comme suit, mais elles peuvent être découvertes dans l’ordre ou dans le désordre, selon les intérêts de la lectrice ou du lecteur.
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Understanding and supporting children looked after and adopted children - recording. ACAMH, marzec 2024. http://dx.doi.org/10.13056/acamh.26660.

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For delegates only. It is recognised that the children looked after population is often on clinical caseloads, yet there appears to be little discussion around both the impact of their early life experiences on their neuropsychology, and the practical aspects of working with this specific population due to this. This webinar focused on the mental health of children looked after and adopted children.
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