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Artykuły w czasopismach na temat "Neuronal differentiation"
Kawaguchi, Tsutomu, Hiroaki Yokoyama, Masaru Inoue, Akio Ichikura, Masashige Onizuka i Masao Kishikawa. "A Case of Pineocytoma with Neuronal Differentiation". Japanese Journal of Neurosurgery 4, nr 2 (1995): 180–84. http://dx.doi.org/10.7887/jcns.4.180.
Pełny tekst źródłaLéopold, Pierre. "Neuronal Differentiation". Cell 119, nr 1 (październik 2004): 4–5. http://dx.doi.org/10.1016/j.cell.2004.09.024.
Pełny tekst źródłaMiyahara, Hiroaki, Manabu Natsumeda, Junichi Yoshimura, Yukihiko Fujii, Akiyoshi Kakita, Yasushi Iwasaki i Mari Yoshida. "MBRS-32. TOPOISOMERASE II β INDUCES NEURONAL, BUT NOT GLIAL, DIFFERENTIATION IN MEDULLOBLASTOMA". Neuro-Oncology 22, Supplement_3 (1.12.2020): iii404. http://dx.doi.org/10.1093/neuonc/noaa222.546.
Pełny tekst źródłaPérez, María Julia, Tomas Roberto Carden, Paula Ayelen dos Santos Claro, Susana Silberstein, Pablo Martin Páez, Veronica Teresita Cheli, Jorge Correale i Juana M. Pasquini. "Transferrin Enhances Neuronal Differentiation". ASN Neuro 15 (styczeń 2023): 175909142311707. http://dx.doi.org/10.1177/17590914231170703.
Pełny tekst źródłaFUKUSHIMA, Takeo, Masamichi TOMONAGA, Toshio SAWADA i Hiroshi IWASAKI. "Pineocytoma with Neuronal Differentiation". Neurologia medico-chirurgica 30, nr 1 (1990): 63–68. http://dx.doi.org/10.2176/nmc.30.63.
Pełny tekst źródłaTateno, M., W. Ukai, M. Yamamoto, E. HashimotAo, H. Ikeda i T. Saito. "ALCOHOL AND NEURONAL DIFFERENTIATION." Alcoholism: Clinical & Experimental Research 28, Supplement (sierpień 2004): 69A. http://dx.doi.org/10.1097/00000374-200408002-00376.
Pełny tekst źródłaRösner, H., M. Al-Aqtum i H. Rahmann. "Gangliosides and neuronal differentiation". Neurochemistry International 20, nr 3 (kwiecień 1992): 339–51. http://dx.doi.org/10.1016/0197-0186(92)90048-v.
Pełny tekst źródłaLamar, E., C. Kintner i M. Goulding. "Identification of NKL, a novel Gli-Kruppel zinc-finger protein that promotes neuronal differentiation". Development 128, nr 8 (15.04.2001): 1335–46. http://dx.doi.org/10.1242/dev.128.8.1335.
Pełny tekst źródłaPfaender, Stefanie, Karl Föhr, Anne-Kathrin Lutz, Stefan Putz, Kevin Achberger, Leonhard Linta, Stefan Liebau, Tobias M. Boeckers i Andreas M. Grabrucker. "Cellular Zinc Homeostasis Contributes to Neuronal Differentiation in Human Induced Pluripotent Stem Cells". Neural Plasticity 2016 (2016): 1–15. http://dx.doi.org/10.1155/2016/3760702.
Pełny tekst źródłaErin, Nuray, i Özkan Ulusoy. "Differentiation of neuronal from non-neuronal Substance P". Regulatory Peptides 152, nr 1-3 (styczeń 2009): 108–13. http://dx.doi.org/10.1016/j.regpep.2008.10.006.
Pełny tekst źródłaRozprawy doktorskie na temat "Neuronal differentiation"
Rocha, Joana Fernandes da. "Understanding APP-dependent neuronal differentiation". Master's thesis, Universidade de Aveiro, 2011. http://hdl.handle.net/10773/7389.
Pełny tekst źródłaAmyloid Precursor Protein (APP) is a type 1 membrane protein that suffers proteolytic cleavages and has been implicated in roles such as cell adherence, survival, migration and differentiation. Although a role in neuritogenesis has been attributed to APP, some contradictory results have been reported regarding the benefits of knocking-down or overexpressing APP. Further, while the addition of the APP proteolytic sAPP (secreted APP) fragment to the cell medium enhances neuritogenesis, the amount of cellular APP and other APP fragments may be deleterious for this process. Further, preliminary work from the Neuroscience laboratory of the Center for Cell Biology indicated that pAPP (APP phosphorylated at the S655 residue) can potentially be crucial in APPmediated neuronal differentiation, for example by increasing APP cleavage to its biological fragment sAPP or APP binding to specific signal transducers. In this work, the capacity of APP and pAPP to mediate neuronal differentiation was tested, in the initial period of retinoic acid (RA)-induced SH-SY5Y cells differentiation. These neuroblastoma cells are a well documented neuronal-like cell model used in neuronal differentiation studies. Several molecular tools were used, including wild-type and phosphomutants APP-GFP. The evaluation of differentiation included neuritogenic output analysis by bright field and epifluorescence microscopy, using various approaches. Namely scoring the number of differentiated cells and performing morphometric analyses of transfected cells and of the all cellular population. The levels of APP and medium secreted sAPP, and of cytoskeleton-related proteins and posttranslational modifications, such as MAP2, Acetylated Tubulin and Actin were also quantified by Western blot analysis, and related to the morphological parameters. Additionally, the potential role of AICD in APP-mediated neuronal differentiation was inferred from pharmacologic assays, where its generation is inhibited. Together the results obtained show that APP, sAPP and AICD modulate neuritogenesis in a complex and well-ordered manner. While long-term increases in APP can be detrimental to neuronal-like differentiation, in an AICD-dependent manner, short-term increases benefit this process in an APP S655 phosphorylation dependent manner, potentially involving sAPP secretion and specific cytoskeleton rearrangements.
A Proteína Precursora de Amilóide de Alzheimer (PPA) é uma proteína membranar tipo 1 sujeita a processamento proteolítico que tem sido associada a funções como adesão celular, sobrevivência, migração e diferenciação. Apesar de lhe terem sido atribuídas funções na neuritogénese, os dados experimentais obtidos até à data que envolveram modulação dos níveis da PPA revelam-se contraditórios. De facto, enquanto a adição do fragmento PPA secretado (PPAs) ao meio celular favorece a neuritogénese, a quantidade de PPA celular e de outros fragmentos da PPA poderão já não constituir um benefício para este processo. Adicionalmente, dados preliminares do laboratório de Neurociências do Centro de Biologia Celular sugerem que a PPAp (PPA fosforilada na S655) poderá ser fundamental na diferenciação neuronal mediada pela PPA, nomeadamente por aumentar a proteólise da PPA a PPAs ou a ligação da PPA a sinais de transdução específicos. No presente trabalho, avaliou-se a capacidade da PPA e PPAp em mediar o período inicial de diferenciação neuronal induzida por ácido retinóico. Para tal recorreu-se a células de neuroblastoma SH-SY5Y, um modelo celular do tipo neuronal bem estabelecido para estudos de diferenciação. Adicionalmente, várias ferramentas moleculares, como PPA-GFP selvagem e fosfomutantes foram usadas. A avaliação da diferenciação incluiu a análise de vários parâmetros neuritogénicos por microscopia de luz (de campo claro e de fluorescência), nomeadamente monitorização de células diferenciadas e análises morfométricas das células transfectadas e da população geral. Os níveis de PPA e PPAs, e de proteínas relacionadas com citosqueleto e suas modificações pós-traducionais (MAP2, Tubulina Acetilada e Actina) também foram quantificados. Além do mais, a influência do DIP na diferenciação neuronal dependente de PPA foi avaliada usando um composto farmacológico para inibir a sua produção. De um modo geral, os resultados obtidos demonstram que a PPA, PPAs e DIP modulam a neuritogénese de um modo complexo e ordenado. Enquanto a indução de níveis altos de expressão de PPA (48h) podem ser detrimentais para a diferenciação tipo-neuronal, de uma forma dependente de DIP, induções mais breves (24h) beneficiam este processo de um modo dependente da fosforilação na S655, potencialmente envolvendo a secreção de PPA e rearranjos específicos do citosqueleto.
Unsworth, Harriet Christina. "Connexins in neuronal and epidermal differentiation". Thesis, Queen Mary, University of London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.429065.
Pełny tekst źródłaGore, S. "Neuronal differentiation markers in basal cell carcinoma". Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1445574/.
Pełny tekst źródłaHigginbotham, Holden Richard. "Polarity regulation during neuronal migration and differentiation". Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2008. http://wwwlib.umi.com/cr/ucsd/fullcit?p3315121.
Pełny tekst źródłaTitle from first page of PDF file (viewed Aug. 4, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 150-172).
CECI, CLAUDIA. "Effect of nickel exposure on neuronal differentiation". Doctoral thesis, Università degli Studi di Roma "Tor Vergata", 2013. http://hdl.handle.net/2108/203180.
Pełny tekst źródłaHein, Paul. "A role for C/EBP[beta] in neuronal differentiation and neuronal regeneration /". Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=100623.
Pełny tekst źródłaCortical progenitor cell fate involves collaborations between cell-intrinsic factors and extracellular cue-activated signalling pathways. Similarly, the activity of C/EBP family members is regulated by cell-intrinsic factors and by the extrinsically activated Erk 1/2-C/EBP signalling pathway in several differentiating non-neuronal cells. Here we present evidence of an analogous function for C/EBP family members in neurogenesis. The results presented in this thesis show that (1) inhibition of MEK (upstream of Erk 1/2) or inhibition of C/EBP family members blocks cortical progenitor neurogenesis; (2) inhibition of C/EBP family members promotes CNTF-induced astrogenesis; and (3) forced expression of a C/EBPbeta mutant (that is, a mimic of its Erk 1/2-RSK phosphorylated form) enhances the expression of neuron-specific genes such as Talpha1 alpha-tubulin.
Neuronal regeneration involves the re-activation of some development-associated genes such as Talpha1 alpha-tubulin and GAP-43. To further define the role of C/EBP family members in the transcription of the Talpha1 alpha-tubulin gene in neurons during regeneration, we crossed transgenic mice that express the beta-galactosidase gene under the control of the Talpha1 alpha-tubulin minimal promoter (Talpha1MP:nLacZ) with mice that carry a null mutation for either C/EBPbeta or C/EBPdelta. The results of facial nerve crush experiments conducted on these hybrid mice show that C/EBPbeta plays a role in the transcriptional activation of the Talpha1 alpha-tubulin minimal promoter following neuronal injury. Injury-induced mRNA expression for either Talpha1 alpha-tubulin or GAP-43 was not noticeably affected by the absence of C/EBPbeta. This suggests that C/EBPbeta-independent mechanisms also play a role in neuronal regeneration.
Lochter, André. "Control of neuronal differentiation by extracellular matrix constituents /". [S.l.] : [s.n.], 1993. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10325.
Pełny tekst źródłaDe, las Heras Rachel, i n/a. "Neuronal Differentiation: A Study Into Differential Gene Expression". Griffith University. School of Biomolecular and Biomedical Science, 2003. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20040225.161725.
Pełny tekst źródłaMarote, Ana Maria Franco Aveiro. "The effects of piezoelectric polymers on neuronal differentiation". Master's thesis, Universidade de Aveiro, 2013. http://hdl.handle.net/10773/11630.
Pełny tekst źródłaO crescimento de neurites é crucial para o desenvolvimento neuronal, bem como para a plasticidade e reparação na fase adulta. Após uma lesão neuronal, o sucesso da reparação é determinando pelas propriedades plásticas constitutivas dos neurónios afetados e pelo seu potencial de regeneração, que é influenciado por sinais externos físicos (ex.: cicatriz glial) e químicos (ex.: moléculas inibitórias). Recentemente, o desenvolvimento de materiais à nano-escala, que interagem com os sistemas biológicos a nível molecular, prometem revolucionar o tratamento das lesões do Sistema Nervoso Central e Periférico. Os scaffolds de nanomateriais podem suportar e promover o crescimento de neurites e consequentemente, intervir nas complexas interações moleculares que ocorrem a após o dano neuronal, entre as células e o seu ambiente extracelular. Vários estudos têm demonstrado que os materiais piezoeléctricos, que geram carga elétrica em resposta ao stress mecânico, podem ser usados para a preparação de scaffolds eletricamente carregados que devem influenciar o comportamento celular. Este estudo centrou-se nos efeitos dos materiais baseados em PLLA (ácido poli (L – láctico)) sob a forma de filmes, nanofibras orientadas aleatória e alinhadamente, e da sua polarização, na diferenciação neuronal. A linha celular de neuroblastoma (SH-SY5Y) foi utilizada para avaliar o efeito dos materiais-baseados em PLLA na adesão, viabilidade, morfologia celular, bem como na diferenciação tipo-neuronal. A análise proteómica baseada em espectrometria de massa das células cultivadas em nanofibras de PLLA foi também efetuada. Os neurónios corticais embriónicos foram seguidamente utilizados para avaliar os efeitos das nanofibras de PLLA alinhadas e da sua polarização no crescimento de neurites. Nesta análise, descobrimos que os materiais de PLLA parecem inibir parcialmente a proliferação celular, enquanto promovem a diferenciação, alterando os níveis das proteínas que intervêm nestes processos. Ocorrem alterações significativas do citoesqueleto, particularmente ao nível do citoesqueleto de actina, que não induzem mas parecem potenciar o crescimento de neurites sob exposição a um sinal extracelular como o ácido retinóico. Este efeito parece ser particularmente evidente para as nanofibras de PLLA alinhadas, que induzem efeitos intermédios na restruturação do citoesqueleto. Em geral, a polarização das amostras de PLLA tem efeitos benéficos na proliferação celular e potencia o crescimento de neurites, particularmente nos neurónios. Acreditamos que as nanofibras de PLLA alinhadas serão um bom scaffold para regeneração neuronal, uma vez que mimetiza o ambiente mecânico natural das células. Contudo, futuras experiências in vitro e in vivo são necessárias para comprovar a eficácia deste potencial scaffold.
Neuritic growth is crucial for neural development, as well as for adaptation and repair in adulthood. Upon neuronal injury, the successful neuritic regrowth is determined by the constitutive plastic properties of neurons and by their regenerative potential, which is influenced by physical (e.g. glial scar) and chemical (e.g. inhibitory molecules) extrinsic cues. Recently, the development of nanometer-scale materials, which can interact with biological systems at a molecular level, provide hope to revolutionize the treatment of central and peripheral nervous system injuries. Nanomaterial scaffolds can support and promote neuritic outgrowth and consequently, take part in the complex molecular interactions between cells and their extracellular environment after neuronal injury. Several studies have shown that piezoelectric materials, which generate electrical charge in response to mechanical strain, may be used to prepare bioactive electrically charged scaffolds that may influence cell behavior. This study focused on the effects of PLLA (poly-L-lactic acid) – based materials in the form of films, random and aligned nanofibers, and of their polarization, on neuronal-like and neuronal differentiation. The neuroblastoma SH-SY5Y cell line was used to evaluate the effect of PLLA – based materials on cellular adhesion, viability, morphology and neuron-like differentiation. Mass spectrometry-based proteomic analysis of cells grown on PLLA nanofibers was also conducted. Primary embryonic cortical neurons were further used to evaluate the effect of PLLA aligned nanofibers and their polarization on neuritic outgrowth. In this analysis, we found that PLLA materials seem to partially inhibit cell proliferation, while promoting neuronal differentiation, altering the levels of proteins that intervene in these processes. Dramatic cytoskeleton remodeling occurs, particularly at the actin cytoskeleton level, which does not induce but may potentiate neuritic outgrowth upon exposure to an extracellular cue, such as Retinoic Acid. This effect seems to be particularly evident for PLLA aligned nanofibers, which induce intermediate effects in the cytoskeleton remodeling. In general, polarization of the PLLA polymers has beneficial effects on cell proliferation and potentiates the neuritic outgrowth, particularly in neurons. We believe that polarized PLLA aligned nanofibers would be a good scaffold for neuronal regeneration, since it mimics the natural mechanical cell environment and enhances neuritic outgrowth. However, further in vitro and in vivo investigations are required to prove the efficacy of this potential scaffold.
De, las Heras Rachel. "Neuronal Differentiation: A Study Into Differential Gene Expression". Thesis, Griffith University, 2003. http://hdl.handle.net/10072/367735.
Pełny tekst źródłaThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Biomolecular and Biomedical Sciences
Full Text
Książki na temat "Neuronal differentiation"
E, Rodriguez-Boulan, Nelson W. J i Keystone Meeting on Epithelial and Nueronal Cell Polarity and Differentiation (1993 : Tamarron, Colo.), red. Epithelial and neuronal cell polarity and differentiation. Cambridge [England]: Company of Biologists Ltd., 1993.
Znajdź pełny tekst źródłaMarty, Shankland, i Macagno Eduardo R, red. Determinants of neuronal identity. San Diego: Academic Press, 1992.
Znajdź pełny tekst źródłaZheng, Chaogu. Genetic Basis of Neuronal Subtype Differentiation in Caenorhabditis elegans. [New York, N.Y.?]: [publisher not identified], 2015.
Znajdź pełny tekst źródłaKandror, Elena. Modeling the Transcriptional Landscape of in vitro Neuronal Differentiation and ALS Disease. [New York, N.Y.?]: [publisher not identified], 2019.
Znajdź pełny tekst źródłaLoeb, David Mark. The role of Trk and secondary signaling molecules in NGF-mediated neuronal differentiation. [New York]: [Columbia University], 1993.
Znajdź pełny tekst źródłaUlrich, Henning. Perspectives of Stem Cells: From tools for studying mechanisms of neuronal differentiation towards therapy. Dordrecht: Springer Science+Business Media B.V., 2010.
Znajdź pełny tekst źródłaH, Yu Albert C., red. Neuronal-astrocytic interactions: Implications for normal and pathological CNS function. Amsterdam: Elsevier, 1992.
Znajdź pełny tekst źródłaHerrera, Esperanza Meléndez, Bryan V. Phillips-Farfán i Gabriel Gutiérrez Ospina. Endothelial cell plasticity in the normal and injured central nervous system. Boca Raton: CRC Press/Taylor & Francis, 2015.
Znajdź pełny tekst źródłaKevin, Hunt R., red. Cellular and molecular differentiation. Orlando: Academic Press, 1987.
Znajdź pełny tekst źródłaTan, Glenn Christopher. The Dual Role of Notch Signaling During Motor Neuron Differentiation. [New York, N.Y.?]: [publisher not identified], 2012.
Znajdź pełny tekst źródłaCzęści książek na temat "Neuronal differentiation"
Tobet, S. A., i T. O. Fox. "Sex Differences in Neuronal Morphology Influenced Hormonally throughout Life". W Sexual Differentiation, 41–83. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4899-2453-7_2.
Pełny tekst źródłaMagavi, Sanjay S., i Jeffrey D. Macklis. "Immunocytochemical Analysis of Neuronal Differentiation". W Neural Stem Cells, 345–52. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-133-8_26.
Pełny tekst źródłaDatta, Indrani, Debanjana Majumdar, Kavina Ganapathy i Ramesh R. Bhonde. "Stem Cells and Neuronal Differentiation". W Stem Cell Therapy for Organ Failure, 71–101. New Delhi: Springer India, 2014. http://dx.doi.org/10.1007/978-81-322-2110-4_5.
Pełny tekst źródłaDarbinian, Nune. "Cultured Cell Line Models of Neuronal Differentiation: NT2, PC12". W Neuronal Cell Culture, 23–33. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-640-5_3.
Pełny tekst źródłaRoque, Cláudio Gouveia, i Christine E. Holt. "Tctp in Neuronal Circuitry Assembly". W Results and Problems in Cell Differentiation, 201–15. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-67591-6_10.
Pełny tekst źródłaDarbinian, Nune. "Cultured Cell Line Models of Neuronal Differentiation: , , and SK-N-MC". W Neuronal Cell Culture, 25–38. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1437-2_3.
Pełny tekst źródłaAppel, Bruce, i Ajay Chitnis. "Neurogenesis and Specification of Neuronal Identity". W Results and Problems in Cell Differentiation, 237–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-540-46041-1_12.
Pełny tekst źródłaBassell, Gary J., i Robert H. Singer. "Neuronal RNA Localization and the Cytoskeleton". W Results and Problems in Cell Differentiation, 41–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-540-40025-7_3.
Pełny tekst źródłaFeizi, T. "Gangliosides as Autoantigens and Differentiation Antigens". W Gangliosides and Modulation of Neuronal Functions, 409–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71932-5_36.
Pełny tekst źródłaLópez-Sánchez, Noelia, María C. Ovejero-Benito, Lucía Borreguero i José M. Frade. "Control of Neuronal Ploidy During Vertebrate Development". W Results and Problems in Cell Differentiation, 547–63. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19065-0_22.
Pełny tekst źródłaStreszczenia konferencji na temat "Neuronal differentiation"
Previtera, Michelle L., Mason Hui, Malav Desai, Devendra Verma, Rene Schloss i Noshir A. Langrana. "Neuronal Precursor Cell Proliferation on Elastic Substrates". W ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53246.
Pełny tekst źródłaLiu, Chun, Seungik Baek i Christina Chan. "The Complementary Effect of Mechanical and Chemical Stimuli on the Neural Differentiation of Mesenchymal Stem Cells". W ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80131.
Pełny tekst źródłaKim, Jeong Hee, i Ishan Barman. "Quantifying the differentiation of 50B11 dorsal root ganglion cells using quantitative phase imaging". W CLEO: Applications and Technology. Washington, D.C.: Optica Publishing Group, 2023. http://dx.doi.org/10.1364/cleo_at.2023.atu4q.5.
Pełny tekst źródłaKleiman, Ross, Michelle Previtera, Sharan Parikh, Devendra Verma, Rene Schloss i Noshir Langrana. "The Effects of Extracellular Matrix Proteins and Stiffness on Neuronal Cell Adhesion". W ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53596.
Pełny tekst źródłaLi, Lulu, Alexander Davidovich, Jennifer Schloss, Uday Chippada, Rene Schloss, Noshir Langrana i Martin Yarmush. "Control of Neural Lineage Differentiation in an Alginate Encapsulation Microenvironment via Cellular Aggregation". W ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206496.
Pełny tekst źródłaChen, Chi-Shuo, Catherine Le, Sushant Soni, Eric Y.-T. Chen i Wei-Chun Chin. "Silk-carbon nanotube composite for stem cell neuronal differentiation". W 2011 IEEE 4th International Nanoelectronics Conference (INEC). IEEE, 2011. http://dx.doi.org/10.1109/inec.2011.5991795.
Pełny tekst źródłaLi, Lulu, Rene Schloss, Noshir Langrana i Martin Yarmush. "Effects of Encapsulation Microenvironment on Embryonic Stem Cell Differentiation". W ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192587.
Pełny tekst źródłaLiu, Mingli, Koichi Inoue i Zhi-gang Xiong. "Abstract 4625: ASIC1 regulates neuronal differentiation of neuroblastoma through Notch signaling pathway". W Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4625.
Pełny tekst źródłaWagner, Omer, Alexander K. Winkel, Eva Kreysing i Kristian Franze. "Multimodal imaging using combined Optical Fourier Ptychographic Microscopy and Atomic Force Microscopy for biological measures". W CLEO: Applications and Technology. Washington, D.C.: Optica Publishing Group, 2022. http://dx.doi.org/10.1364/cleo_at.2022.atu5i.3.
Pełny tekst źródłaLiu, Mingli, Koichi Inoue, An Zhou i Zhigang Xiong. "Abstract B29: ASIC1 impacts Notch signaling pathway in the neuronal differentiation of neuroblatoma". W Abstracts: Eighth AACR Conference on The Science of Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; November 13-16, 2015; Atlanta, Georgia. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7755.disp15-b29.
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