Gotowe bibliografie tematyczne / Neurological Disorder

Gotowa bibliografia na temat „Neurological Disorder”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 11 września 2023

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Artykuły w czasopismach na temat "Neurological Disorder"

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Popkirov, Stoyan, Jon Stone i Alastair M. Buchan. "Functional Neurological Disorder". Stroke 51, nr 5 (maj 2020): 1629–35. http://dx.doi.org/10.1161/strokeaha.120.029076.

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Pimentel Filho, Lucio Huebra, i Eduardo Genaro Mutarelli. "Diagnostic pitfalls in functional neurological disorders". Arquivos de Neuro-Psiquiatria 80, nr 5 suppl 1 (maj 2022): 324–27. http://dx.doi.org/10.1590/0004-282x-anp-2022-s120.

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ABSTRACT The diagnosis of functional neurological disorders is a major challenge in neurologist practice. Some clinical strategies can facilitate the recognition of functional disorders, but several pitfalls make their diagnosis difficult. Here we highlight the following points of attention during evaluation of patients with functional disorder: not all bizarre behavior is functional; not every event triggered by an emotional factor is a functional disorder; not every topographic incongruity is a functional disorder; patients may present functional and organic symptoms at the same time; psychiatric comorbid condition is not always evident in the history of a functional disorder; problematic communication at the time of diagnosis can compromise treatment and prognosis. In conclusion, we emphasize that special attention to these possible pitfalls facilitate the correct diagnosis and management of functional neurological disorders.
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Poalelungi, Alina, i Bogdan O. Popescu. "Alzheimer's disease - neurological or psychiatric disorder?" Romanian Journal of Neurology 12, nr 1 (31.03.2013): 5–14. http://dx.doi.org/10.37897/rjn.2013.1.1.

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Alzheimer’s disease (AD), a progressive neurodegenerative disorder, is the most common form of dementia in the elderly. The clinical manifestations of Alzheimer’s disease evolve from an initial discrete impairment of recent memory to severe cognitive loss, in time behavioural and psychiatric symptoms becoming obvious and disturbing. The cause of this complex clinical picture is the gradual functional deterioration and eventually loss of all brain cell types, with severe alteration of neuronal networks supporting cognitive processes. The aim of this paper is to examine different features of AD and to formally establish whether it belongs to the neurological or psychiatric group of disorders. A review of key literature in the field was performed for main attributes of AD neuropathology and pathophysiology. In this respect, we have compared AD with classical psychiatric disorders (schizophrenia, bipolar disorder, obsessive compulsive disorder) and with neurological degenerative disorders (AD, Parkinson’s disease, epilepsy, amyotrophic lateral sclerosis, Huntington’s disease). In brief, AD pathogenic mechanisms involve protein aggregation, synapse alteration, oxidative stress, neurotransmitter deficit, intracellular calcium dyshomeostasis and mitochondrial dysfunction, all together finally leading to cell death and brain atrophy. To some extent, some of these features are common for both psychiatric and neurodegenerative disorders. However, from the cellular and molecular pathology perspective, AD seems to be closer to other neurological conditions than to classical psychiatric diseases.
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Das, Priscilla, Nyi Nyi Naing, Nadiah Wan-Arfah, KON Noorjan, Yee Cheng Kueh i Kantha Rasalingam. "ASSESSMENT OF DEPRESSION AND ANXIETY IN NEUROLOGICAL DISORDER PATIENTS AND THEIR RELATIONSHIP WITH QUALITY OF LIFE". Malaysian Journal of Public Health Medicine 21, nr 2 (28.08.2021): 112–23. http://dx.doi.org/10.37268/mjphm/vol.21/no.2/art.804.

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Objectives: To assess the association between major depressive disorder, anxiety disorders and the quality of life of neurological disorder (brain tumour/brain disorder) patients. Methods: This study was conducted at Kuala Lumpur Hospital, Malaysia, a tertiary referral centre hospital for neurological disorder patients. The cross-sectional study design was applied. The Mini-International Neuropsychiatric Interview and European Organization for Research and Treatment of Cancer Quality of Life Questionnaire were used in the study. Results: A total of 100 neurological disorder patients were included in the study. The study found that the Major depressive disorder correlated with almost all domains of the quality of life, except the nausea and vomiting scores. Logistic regression showed that emotional functioning and pain were related to major depressive disorder. Different anxiety disorders also correlated with quality of life in specific domains. The leading anxiety disorders that associated mostly with quality of life scales were post traumatic stress disorder, panic disorder lifetime and current, panic disorder with agoraphobia, obsessive compulsive disorder, anxiety disorder and with agoraphobia current and social phobia current (p < 0.05). Conclusions: There is a significant relationship between psychiatric disorders and quality of life neurological disorder patients. Therefore treatment along with psychiatric intervention should be implemented to improve the overall curability of the neurological disorder patients.
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Amanat, Man, Mona Salehi i Nima Rezaei. "Neurological and psychiatric disorders in psoriasis". Reviews in the Neurosciences 29, nr 7 (25.09.2018): 805–13. http://dx.doi.org/10.1515/revneuro-2017-0108.

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Abstract Psoriasis used to be known as a skin disorder; however, it can now be considered as a systemic disease with the involvement of multiple organs. Neurological and psychiatric disorders are some of the associated problems that can be observed in patients with psoriasis. Stroke, multiple sclerosis, seizure, migraine, restless leg syndrome, Parkinson’s disease, Guillain-Barré syndrome, and myasthenia gravis are the reported neurological diseases, while depression, bipolar mood disorder, anxiety, psychosis, cognitive impairment, personality disorders, sexual disorders, sleep disturbance, and eating disorders are the recognized psychiatric presentations in patients with psoriasis. Herein, the neurological and psychiatric disorders of psoriasis are described.
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Eratne, Dhamidhu, Samantha M. Loi, Nirbaanjot Walia, Sarah Farrand, Qiao-Xin Li, Shiji Varghese, Mark Walterfang i in. "A pilot study of the utility of cerebrospinal fluid neurofilament light chain in differentiating neurodegenerative from psychiatric disorders: A ‘C-reactive protein’ for psychiatrists and neurologists?" Australian & New Zealand Journal of Psychiatry 54, nr 1 (21.06.2019): 57–67. http://dx.doi.org/10.1177/0004867419857811.

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Objective: Neurofilament light has shown promise as a biomarker for diagnosis, staging and prognosis in a wide range of neurological and neurodegenerative disorders. This study explored the utility of cerebrospinal fluid neurofilament light in distinguishing primary psychiatric disorders from neurodegenerative and neurological disorders, a common diagnostic dilemma for psychiatrists and neurologists. Methods: This cross-sectional retrospective pilot study assessed cerebrospinal fluid neurofilament light on patients referred to a tertiary neuropsychiatry service from 2009 to 2017 for diagnostic assessment of neuropsychiatric and neurocognitive symptoms, where a neurodegenerative disorder was a differential diagnosis, who received lumbar punctures as part of a comprehensive workup. The most recent gold-standard clinical consensus diagnosis was categorised into psychiatric disorder or neurodegenerative or neurological disorder. Data from healthy controls were available for comparison. Data extraction and diagnostic categorisation was blinded to neurofilament light results. Results: A total of 129 participants were included: 77 neurodegenerative or neurological disorder (mean age 57 years, including Alzheimer’s dementia, frontotemporal dementia), 31 psychiatric disorder (mean age 51 years, including schizophrenia, major depressive disorder) and 21 healthy controls (mean age 66 years). Neurofilament light was significantly higher in neurodegenerative or neurological disorder (M = 3560 pg/mL, 95% confidence intervals = [2918, 4601]) compared to psychiatric disorder (M = 949 pg/mL, 95% confidence intervals = [830, 1108]) and controls (M = 1036 pg/mL, 95% confidence intervals = [908, 1165]). Neurofilament light distinguished neurodegenerative or neurological disorder from psychiatric disorder with an area under the curve of 0.94 (95% confidence intervals = [0.89, 0.98]); a cut-off of 1332 pg/mL was associated with 87% sensitivity and 90% specificity. Conclusion: Cerebrospinal fluid neurofilament light shows promise as a diagnostic test to assist with the often challenging diagnostic dilemma of distinguishing psychiatric disorders from neurodegenerative and neurological disorders. Further studies are warranted to replicate and expand on these findings, including on plasma neurofilament light.
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Mihai Zamfir, Emilia, Anca Daniela Braila, Mihai Braila, Radu Nicolae Mateescu i Antoine Edu. "Neurological disorder in pregnancy". Romanian Journal of Medical Practice 13, nr 3 (30.09.2018): 206–8. http://dx.doi.org/10.37897/rjmp.2018.3.6.

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Saini, Arushi Gahlot, Naveen Sankhyan i Sameer Vyas. "PLEKHG2-associated neurological disorder". BMJ Case Reports 14, nr 7 (lipiec 2021): e244206. http://dx.doi.org/10.1136/bcr-2021-244206.

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Brang, David, Paul D. McGeoch i Vilayanur S. Ramachandran. "Apotemnophilia: a neurological disorder". NeuroReport 19, nr 13 (sierpień 2008): 1305–6. http://dx.doi.org/10.1097/wnr.0b013e32830abc4d.

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Charlier, Philippe, Donatella Lippi, Antonio Perciaccante, Otto Appenzeller i Raffaella Bianucci. "Neurological disorder? No, Mannerism". Lancet Neurology 18, nr 2 (luty 2019): 135. http://dx.doi.org/10.1016/s1474-4422(18)30447-2.

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Rozprawy doktorskie na temat "Neurological Disorder"

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Coy, G. "Emotion processing in functional neurological disorder". Thesis, Canterbury Christ Church University, 2018. http://create.canterbury.ac.uk/17707/.

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Objective: Alexithymia and hypomentalization, two traits associated with childhood emotional abuse/neglect, have not previously been studied in people with mixed-symptom functional neurological disorder (FND). This case-control study these traits in people with FND compared to healthy control participants, and explored the relationships between alexithymia, mentalization, and somatic and neurological symptoms of a generalized nature. Method: Twenty-nine participants with FND and 41 healthy control participants completed a battery of self-report measures. Between-group differences in alexithymia and hypomentalization were investigated using parametric tests, and binary logistic regression analyses examined whether alexithymia and hypomentalization were predictive of FND (vs control) group status, after controlling for depressive symptoms, anxiety symptoms and education attainment. Linear regression analyses examined whether alexithymia and hypomentalization were also associated with physical and neurological symptoms across the entire sample. Results: Participants with FND had significantly higher score on measures of alexithymia, hypomentalization, somatic symptoms and neurological symptoms compared to healthy control participants. Between-group differences in alexithymia and neurological symptoms remained significant after controlling for covariates. High scores on the alexithymia and mentalization measures were also predictive of high scores on the measures of somatic and neurological symptoms across the entire sample. Conclusion: Alexithymia and hypomentalization do appear to be significant issues for people with FND, and may contribute to the tendency to express distress via physical symptoms. Exploring these traits with individual service users may contribute to a more comprehensive conceptualisation of their difficulties, and inform treatment approaches that are engaging and supportive.
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Williams, Isobel Anne. "Emotion regulation in patients with Functional Neurological Disorder". Thesis, University of Sheffield, 2018. http://etheses.whiterose.ac.uk/21430/.

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Shaw, P. J. "Neurological and neurophysiological complications of coronary artery bypass graft surgery". Thesis, University of Newcastle Upon Tyne, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.380746.

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Ichikawa, Shoji. "The molecular genetic analysis of three human neurological disorders". free online free to MU campus, others may purchase, 2002. http://wwwlib.umi.com/cr/mo/preview?3074409.

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Phoenix, Danielle. "Exploring how males who encounter phenomena they identify as 'Conversion Disorder'/'Functional Neurological Disorder' experience agency in their lives". Thesis, Middlesex University, 2017. http://eprints.mdx.ac.uk/21822/.

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This research investigates the way that males who identify with the diagnostic label ‘conversion disorder/functional neurological disorder (CD/FND)’ experience agency in their lives. The historical developments, controversies and complexities around ‘CD/FND’ form the backdrop of this exploration into the lived experience of agency. A sample of eight participants were recruited via social networking sites and charities, and the data was collected through Skype-based interviews and analysed using the qualitative Interpretative Phenomenological Analysis (IPA) approach. The analysis showed the following five main themes: ‘paradox of control’, ‘living within a dualistic framework’, ‘disconnection from self and others’, ‘engaged in a battle or fight’ and ‘meaning and reality as dependent on other people’. These master themes and their related subordinate themes are presented in light of existing research. The findings highlight the difficulty experienced by participants who identify with a diagnostic label that is at odds with a medicalised approach to understanding and treating illness. The limitations of this study and the potential avenues for future research are also discussed.
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Beckh, Johanna [Verfasser]. "Is Functional Neurological Symptom Disorder (FNSD) a (somatic) stress disorder with altered emotion processing? : An approach to an answer via different methods, disorders an over time / Johanna Beckh". Konstanz : KOPS Universität Konstanz, 2019. http://d-nb.info/1206096772/34.

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Beasley, Brooke, Aubrey Sciara, Tiffani Carrasco, Gregory Dr Ordway i Michelle Dr Chandley. "Laser Capture Microdissection Analysis of Inflammatory-Related Alterations in Postmortem Brain Tissue of Autism Spectrum Disorder". Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/34.

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Autism spectrum disorder (ASD) is a social, sensory and developmental condition that affects one in 59 children and specifically one in 42 boys. Despite the 15% increase in prevalence in the last two years, there is no specific etiology, objective diagnostic criteria, or drug treatment. However, up-regulation of inflammation in ASD patients has been demonstrated in blood samples. Increased peripheral inflammation could have devastating effects on the developing brain. Peripheral inflammation in the blood could cross the blood-brain-barrier to stimulate microglia in the brain to produce aberrant levels of cytokines that regulate neuroinflammation such as insulin-like growth factor one (IGF1) that could alter neuronal cell-surface expression and neurotransmission. Additionally, arginase serves as a marker of inflammation, produced and expressed during cellular remodeling during brain injury. A balance of neurotransmitters, glutamate and gamma-aminobutyric acid (GABA), is critical to facilitate inter-regional signaling in the brain. Alterations of inflammatory molecules and the effects on glutamatergic neurons ability to uptake GABA in certain brain areas is currently unknown in ASD. Pathological changes in brain areas associated with social behaviors have been identified in postmortem tissue from ASD donors when compared to typically developing (TD) age and gender matched control tissue, as well as, in imaging scans of living individuals with ASD. We hypothesize that expression of inflammatory related molecules are increased in the identified brain areas related to symptoms of ASD and can be associated with altered gene expression changes in neurons as shown by gamma-aminobutyric acid type A receptor alpha 1 subunit (GABRA1). Dysfunction of GABRA1 on glutamatergic neurons could disrupt the typical neuronal balance of glutamate and GABA signaling. Inflammatory markers, IGF1 and insulin-like growth factor one receptor (IGF1R), were evaluated using quantitative polymerase chain reaction (QPCR). Additionally, IGF1 and arginase were evaluated using immunohistochemistry in both white and gray matter from the anterior cingulate cortex (ACC). Laser capture microdissection (LCM) was used to obtain single cell captures of glutamatergic neurons. IGF1R and GABRA1 gene expression was measured using end point PCR. A significant increase in IGF1 expression was obtained in the white matter punch in comparison to typically developed age-matched subjects using QPCR during initial statistical significance, however, was ultimately not significant. Additionally, IGF1R expression was significantly increased in ASD neurons in comparison to TD subjects utilizing the LCM method. However, a decrease expression in GABRA1 trended significance indicating a possible alteration in the neuron’s ability to facilitate proper signaling. These findings are the foundation of future investigations of signaling pathways in ASD that may uncover cell-specific etiologies and drug therapies for a condition that is only projected to increase in prevalence.
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Chetram, Sursatie. "Neurodevelopmental Basis of Autism Spectrum Disorder based on Age and Gender". ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/4720.

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Autism Spectrum Disorder (ASD) is a neurodevelopmental disorder that affects communication, socialization, and restricted/repetitive behaviors. In 2012, one out of every 55 children (1 in 42 boys and 1 in 189 girls) have been diagnosed with ASD in the United States. Only 30-40% of ASD has a known etiology (e.g., genetic predisposition) and the other 60-70% is unknown. Prior to this study, there was no known literature on age and gender differences related to neuro-developmental functioning of ASD. The purpose of this study was to examine how the differences in age and gender of people with ASD were related to total and domain scores, as measured by the Autism Diagnostic Observation Schedule, Second Edition (ADOS-2). This quantitative research study included a sample size of 80 and 2 independent variables: age groupings (ages 1-4, 5-8, 9-17, and 18-older), and gender (male and female). The 4 dependent variables were the total and domain scores measured by the ADOS-2. The statistical analyses included a multiple analysis of variance (MANOVA) and a 2-way analysis of variance (ANOVA) to examine age and gender differences in the ADOS-2 domain and total scores. There was a statistically signi�cant difference for age on the domain dependent variables, F(9, 171) = 2.64, p = .007; Wilks' Lambda = .73; partial η2 = .10. However, there were no statistically significant differences for gender on domain scores and there were no statistically significant differences for age and gender on the overall scores. Those with ASD between ages 5-8 were more severely impaired for socialization when compared to other age groups and other domains. This research can be used for the improvement of intervention strategies for the diverse ASD population, and to improve the understanding of the neurodevelopmental functioning of individuals with ASD based on age and gender.
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Law, Cecilia. "Everyday Memory Difficulties in Children and Adults with Neurological Disorders: Mixed Method Studies". Thesis, The University of Sydney, 2022. https://hdl.handle.net/2123/29743.

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Memory difficulties are prevalent in patients with neurological disorders across lifespan. However, little is known about how memory difficulties are experienced in everyday life, and their functional impacts. This thesis aimed to improve the assessment of children’s everyday memory and explore patients’ lived experience and coping with an under-researched memory disorder—accelerated long-term forgetting (ALF). Chapter 1 presented a general introduction to everyday memory difficulties in patients with neurological disorders. In Chapter 2, a systematic review evaluated currently available paediatric memory questionnaires and found that all, except one memory questionnaire, lacked evidence base to be used as assessment tools. Hence, exploratory factor analyses (EFA) were conducted to establish factorial structures of two commonly administered questionnaires, the Parent Memory Questionnaire (PMQ) and Child Memory Questionnaire (Child MQ), in a neurotypical paediatric sample (Chapter 3) and a clinical sample (Chapter 4). The two EFAs successfully established a comparable three-factor structure for the PMQ, but not the Child MQ. Next, Chapter 5 and 6 provided the first evidence of the lived experience of ALF of adults with epilepsy using a mixed method approach. Quantitative and qualitative findings from Chapter 5 revealed that ALF was associated with profound losses across personal, relationship and functional domains, and multiple psychosocial challenges. Chapter 6 documented findings from semi-structured interviews with patients, their family members, and health professionals on coping strategies for ALF and ALF-inflicted psychosocial difficulties. These coping strategies were categorised into avoidance, emotion-focused, memory-focused, seizure control. Overall, findings informed on the measurement of everyday memory problems and urged for the development of comprehensive intervention for patients with neurological disorders who also experience memory difficulties.
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Colson, Natalie, i n/a. "The Role of Hormonal and Vascular Genes in Migraine". Griffith University. School of Medical Science, 2007. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20071121.104112.

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Migraine is a frequent debilitating neurological disorder that is considered to be genetically complex with a multifactorial mode of inheritance. It has a high prevalence with approximately 18% of women and 6% of men suffering from the disorder. Migraine is characterized by severe head pain with associated nausea, emesis, photophobia, phonophobia, and neurological disturbances. The International Headache Society (IHS) has classified various types of migraine according to their clinical features. The two main subtypes of migraine are migraine without aura (MO), occurring in ~70-75% of migraineurs, and migraine with aura (MA) which occurs in ~25% of migraineurs. Some people experience both types of attack in their lives. While the precise pathogenesis of migraine is unknown, it is widely accepted that short-term alterations in neuronal activity occur in relation to the attack, along with temporary changes in the cerebral vasculature. Trigeminal nerve activation is also considered pivotal to progression of a migraine attack. Neurotransmitters, especially serotonin (5-hydroxytryptamine, 5-HT), platelet activation and sympathetic hyperactivity all appear to play a part, whether as part of the primary triggering event, or as a response mechanism. Migraine imparts a significant burden on society, both socially and financially. The World Health Organization has identified migraine among the world's top 20 leading causes of disability, with an impact that extends far beyond individual suffering. There is significant evidence from family and twin studies to indicate a strong genetic component to migraine. The current understanding of migraine is that it is a polygenic multifactorial disorder. It has been postulated that genetic factors set the individual migraine threshold, with environmental influences playing a modulating role. It is likely that many genes may provide an important although moderate contribution to an individual’s migraine susceptibility. The identification of migraine susceptibility genes has been the focus of substantial research to date and could eventually lead to improved treatments and greater understanding of the disorder. Several loci have shown promise, although these need to be followed up by both replication and functional studies to determine a definitive causative role. This research investigated the role of both hormonal and vascular related genes as candidate genes that may play a role in migraine susceptibility due to the well-known role of hormones and vascular changes in some migraineurs. The estrogen receptor (ESR) and progesterone receptor (PGR) genes are potential migraine candidates due to the recognized hormonal influence on migraine susceptibility. Migraines in women frequently occur during the childbearing years and are often influenced by significant hormonal milestones. The fluctuating hormone levels of the menstrual cycle have been implicated in migraine but a definitive role is yet to be established. It has been suggested that factors additional to circulating hormone levels may be at play. This research considered that variation in the ESR 1 and PGR genes may confer an increased migraine risk. To investigate the potential role of these genes in migraine, association studies investigating variants in ESR 1 and PGR were undertaken in two independent casecontrol cohorts. This was followed up by mutation screening and gene expression analysis in an effort to elucidate a functional role for these genes in the pathogenesis of migraine. Vascular genes also represent likely migraine candidates as alterations in both vascular function and cerebral blood flow are well known in migraine. Furthermore, cortical spreading depression (CSD), a depolarization wave that propagates across the brain cortex and has been speculated to cause the neurological symptoms that present in MA, has also been linked to vascular dysfunction. The methylenetetrahydrofolate reductase (MTHFR) and methionine synthase (MTRR) genes both play a role in vascular functioning and were thus considered potential migraine candidates for this study. Both are involved in the pathway of homocysteine metabolism. Impaired activity of these enzymes can lead to mild hyperhomocysteinemia which is believed to lead to oxidative arterial damage. This may in turn impact on migraine susceptibility, possibly through the activation of trigeminal fibres. The MTHFR 677T allele results in an amino acid change in the catalytic domain of the enzyme leading to mild hyperhomocysteinemia. This particular variant has been implicated in migraine in four separate studies. One of these studies also suggested a role for the MTHFR 1298C allele in migraine. This allele also results in an amino acid change and reduced enzyme activity. Similarly, the MTRR 66G allele results in an amino acid change and has been associated with reduced activity of MTRR and increased plasma homocysteine concentration. To investigate the role of the ESR 1, PGR, MTHFR and MTRR genes in migraine, samples from two large independent case control cohorts were investigated. Cohort 1 was comprised of 275 migraineur samples and 275 age, sex and ethnicity matched controls while cohort 2 comprised 300 cases and 300 matched controls. All individuals were collected at the Genomics Research Centre with migraine diagnosis undertaken by HIS criteria and migraine affected individuals designated as MO or MA. Results of analysis of ESR 1 indicated a positive association with migraine in the two large independent cohorts for the exon 8 G594A polymorphism (P = 0.003; P = 8x10-6). Similarly, the PGR analysis showed a positive association with migraine for the PROGINS allele (P = 0.02; P = 0.003). Results also showed that individuals with both ESR 1 and PGR susceptibility alleles were 3.2 times more likely to suffer migraine those those with no susceptibility alleles. As the ESR 1 variant is synonymous, a mutation analysis was undertaken in a small sub-sample of individuals carrying the susceptibility allele, but no mutations were detected in these particular samples. Detailed mutation analysis of ESR 1 in a larger study group may be warranted. An ESR 1 and PGR expression analysis by RT-PCR was undertaken to examine if there were any notable expression level changes in migraineurs versus controls and additionally whether the susceptibility genotypes influenced gene expression. Altered expression levels may point to a functional change in the gene. Although results did not show any significant difference in expression levels in the case/control group, nor any influence in gene expression conferred by the specific susceptibility genotypes, ESR 1 expression did appear to be down-regulated in the migraine group and more specifically in the migraine susceptibility genotype subgroup. A larger study group may therefore be warranted to detect any potential genuine changes in gene expression. Overall, these results suggested that these hormonal genes appear to play a role in migraine susceptibility, although further studies are needed to define this. Results of the MTHFR 677 analysis showed that the TT genotype was significantly associated with the MA subgroup in a joint analysis of the two independent cohorts (P = 0.004). Results of analysis of MTHFR 1298, which is tightly linked to the 677 locus, showed a significant association in female migraineurs (P = 0.009). Similarly, results of the MTRR analysis also showed a significant difference between the female case and control groups with the G allele over-represented in female migraineurs (P = 0.022) These results may indicate that a significant gender effect appears in this locus as well as the MTHFR 1298 locus although results may also be due to a larger number of female migraineurs conferring increased statistical power to the gender subgroup. Interaction analysis of the MTHFR 1298 locus and the MTRR locus showed that females who carried both variants under a recessive model were 5 times more likely to suffer migraine those those with no susceptibility genotypes. Overall these results indicated that these vascular genes appear to play a role in migraine susceptibility. The final study focused on 6 genetic variants that had shown a positive association with migraine and/or MA in the same large association population analysed in this research. The aim of this study was to provide preliminary data on the potential role of genetic profiling in migraine. Using the genotypic data to create vascular and hormonal risk profiles based on positive association and interaction of MTHFR 677 T and ACE D alleles, and MTHFR 1298 AA and MTRR GG genotypes as vascular variants; and positive association and interaction of ESR 1 594 A and PGR PROGINS as hormonal variants, this study was able to demonstrate the relevance of genetic risk profiling to migraine. Results showed a significantly higher proportion of individuals with at least one genetic risk profile in the migraine group compared to those in the control group (P = 6 x 10-6). Individuals who possessed either the vascular and/or hormonal genetic risk profile were 8.6 times more likely to suffer from migraine than those who possessed a ‘no risk’ profile. This indicated a greater effect than the individual effect of each of these variants. Furthermore individuals who possessed a vascular or both risk profiles were more likely to suffer nausea, emesis, phonophobia and photophobia, and have a mother who also suffered migraine. Overall, the genetic profiling approach provided interesting preliminary data on migraine susceptibility and indicated that such an approach may prove very useful for migraine diagnosis, particularly when all migraine genes have been identified. In conclusion this study provided the first indication that hormone receptor genes play a role in migraine susceptibility. Hormones have long been considered to play a role in the disorder but this study has provided the first molecular evidence to support this premise. In addition, this study showed that vascular related genes also play a role in migraine susceptibility. Finally, this study has clearly shown that migraine is a complex disorder involving multiple genes. Although a number of studies have implicated neurotransmitter related genes in the disorder, the present study is the first to show that both vascular and hormonal genes also play a role in migraine susceptibility. Thus there now appear to be three classes of genes that affect migraine susceptibility and although this study has implicated new variants, the preliminary genetic profiling study has shown that not all predisposing variants involved in the disorder have been defined.
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Książki na temat "Neurological Disorder"

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Translating mechanisms of orofacial neurological disorder. London: Academic Press, 2011.

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Roberto, Tuchman, i Rapin Isabelle, red. Autism: A neurological disorder of early brain development. London: MacKeith Press for the International Child Neurology Association, 2006.

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Motor disorder in psychiatry: Towards a neurological psychiatry. Chichester: J. Wiley & Sons, 1992.

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Boyle, Lia. A Precision Medicine Approach to Understanding KIF1A Associated Neurological Disorder. [New York, N.Y.?]: [publisher not identified], 2021.

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J, Frucht Steven, i Fahn Stanley 1933-, red. Movement disorder emergencies: Diagnosis and treatment. Totowa, N.J: Humana Press, 2005.

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author, Harrison Alexandra 1945, i Singletary William contributor, red. Autism spectrum disorder: Perspectives from psychoanalysis and neuroscience. Lanham: Jason Aronson, 2014.

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DeFelice, Karen L. Enzymes for autism and other neurological conditions: Practical guide to biomedical therapy. Philadelphia: Jessica Kingsley Publishers, 2002.

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Gardner, Anne. Bipolar strong: An empowering collection by and for teens & adults with bipolar disorder, depression, and other neurological brain disorders, or "hidden disabilites". [Philadelphia, PA]: Xlibris Corporation, 2008.

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Anne, Gardner, red. Bipolar strong: An empowering collection by and for teens & adults with bipolar disorder, depression, and other neurological brain disorders, or "hidden disabilites". [Bloomington, IN?]: Xlibris, 2008.

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Parkes, J. D., P. Jenner, D. N. Rushton i C. D. Marsden. Neurological Disorders. London: Springer London, 1987. http://dx.doi.org/10.1007/978-1-4471-3140-3.

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Części książek na temat "Neurological Disorder"

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Tondo, Leonardo, Matthew J. Albert, Alexia E. Koukopoulos, Christopher Baethge i Ross J. Baldessarini. "Bipolar Disorder". W Neurological and Psychiatric Disorders, 205–27. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-856-0:205.

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Khatri, B. O. "Plasmapheresis in Neurological Disorder". W Advances in haemapheresis, 69–77. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3904-9_9.

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Kalarani, Iyshwarya Bhaskar, i Ramakrishnan Veerabathiran. "Diabetes and Neurological Disorder". W Advances in Diabetes Research and Management, 63–79. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-0027-3_4.

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Husted, David S., Nathan A. Shapira i Wayne K. Goodman. "Obsessive-Compulsive Disorder". W Neurological and Psychiatric Disorders, 171–88. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-856-0:171.

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Zhang, Kehong, Eugen Davids i Ross J. Baldessarini. "Attention Deficit Hyperactivity Disorder". W Neurological and Psychiatric Disorders, 229–50. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-856-0:229.

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Gildenberg, P. L. "History of Movement Disorder Surgery". W Progress in Neurological Surgery, 1–20. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000062034.

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Raynor, Geoffrey, i Gaston Baslet. "Functional Neurological Disorder and Dissociative Disorders in Women". W Neurology and Psychiatry of Women, 15–26. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-04245-5_3.

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Hutchison, W. D., i A. M. Lozano. "Microelectrode Recordings in Movement Disorder Surgery". W Progress in Neurological Surgery, 103–17. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000062039.

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Saint-Cyr, J. A., i L. L. Tr�panier. "Neuropsychological Considerations in Movement Disorder Surgery". W Progress in Neurological Surgery, 266–71. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000062050.

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Clatterbuck, R. E., J. I. Lee i F. A. Lenz. "Movement Disorder Surgery: Lesions or Stimulation?" W Progress in Neurological Surgery, 227–35. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000062062.

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Streszczenia konferencji na temat "Neurological Disorder"

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Liu, Chuhua. "Depression: neurological disorder versus psychiatric disorder". W International Conference on Biological Engineering and Medical Science (ICBIOMed2022), redaktorzy Gary Royle i Steven M. Lipkin. SPIE, 2023. http://dx.doi.org/10.1117/12.2669636.

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Ferreira, Marcos Venâncio Araújo, Rafael Henrique Neves Gomes, Fabiana Carla dos Santos Correia, Mariana Beber Chamon, Sérgio Roberto Pereira da Silva Júnior, Isadora Chain Lima, Marcus Vinicius de Sousa, Murilo Justino de Almeida, Daniel Sabino de Oliveira i Thiago Cardoso Vale. "Idiopathic basal ganglia calcification and Hoarding disorder". W XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.499.

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Introduction: Basal ganglia calcifications are associated with many neurological and metabolic disorders, being present also on asymptomatic patients. It may present in its primary form, including familial and sporadic cases. Its secondary form is associated especially to hypoparathyroidism but also associated to infections, toxic exposure, rheumatologic diseases, mitochondrial disorders. It has an heterogenous clinical presentation with movement disorders and neuropsychiatric symptoms. Case presentation: A 66-year-old patient presented with a progressive hoarding disorder for the last six years. In the last 2 years started an aggressive behavior, loss of acquired skills, urinary incontinence, sleep-wake cicle disorder and one episode of focal seizure. Physical examination revealed bilateral asymmetrical tremor, bradykinesia and cogwheel rigidity. MoCA test was 23/30 for 12 years of schooling. Brain Computed Tomography showed calcifications in basal ganglia affecting predominantly pallidum e thalamus and cerebellar hemispheres. Brain Magnetic Resonance Imaging revealed hypointensites in the same regions and in nucleus caudate suggestive of calcification. Laboratory testing for endocrine and calcium metabolism was normal. No clinical signs of other disorders. Discussion: We presented a case of probable Idiopathic Basal Ganglia Calcification initially treated as a hoarding disorder. The normal laboratory results, lack of other clinical signs and familial history suggests a primary sporadic form that might be due to de novo mutations or transmitted by asymptomatic parent. The most commonly mutations in SLC20A2, PDGFB and PDGFRB but genetic testing is commonly unavailable. Parkinsonism is the most common movement disorder and the neuropsychiatric features include cognitive impairment, psychotic and obsessive compulsive disorders. Conclusion: This case demonstrates that attention is needed to the progression of psychiatric disorders suggesting some rare neurological disorders.
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Pereira, Luiza de Lima, Ana Flávia Silva e. Sousa, Anderson Pedrosa Mota Júnior, Giovanna Martins Romão Rezende, Marcella Ferreira Ribeiro i Carolina Ferreira Colaço. "Neurological conditions caused by microgravity". W XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.402.

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Background: Since Space Tourism is closer to reality, a review of the most prevalent neurological pathologies in microgravity is needed. Objective: Review major neurological afflictions in astronauts. Methods: Research into bibliographic reviews at PubMed, using the descriptors “astronauts” and “neurological disorders” Results: Several neurological alterations, such as ataxy, intracranial hypertension (ICH), neuromuscular disorders, ocular disturbances and changes in cognitive functions were assigned to a microgravity environment. Astronauts returning from space presented ICH; being the main pathophysiology hypothesis referred to a change in the liquor dynamics as a result of venous drainage obstruction and hematoencephalic barrier. Also, gravity doesn’t act on the neurovestibular system during space flights. This phenomenon can lead to Space Motion Sickness, situation in which astronauts report balance, coordination and sight disturbances, as well as movement illusions. A subset of this syndrome, called Space Movement Disorder, may occur; which includes symptoms, such as flushing, anorexia, nausea, vomiting, dizziness and malaise. Based on electromagnetic resonance, the occurrence of problems with movement time, balance, spatial working memory and motor coordination after the return of the space crew suggests alterations in cerebellum’s function and structure, which is responsible for the coordination and the fine motor control. Conclusion: Most studies presented disruptions of the neurofunctional homeostasis, for instance, changes in functional connectivity while in rest and alterations of the white and grey matter in sensor motor, somatosensory and cognitive regions of the brain.
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Greulich, Nicola, i Joerg Klepper. "Sudden Uncontrolled Laughter - Behavioral Disorder or Serious Neurological Symptom?" W Abstracts of the 45th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1698160.

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Dong, Yue, Kunjie Liu i Yushi Wu. "The Function of BDNF and Treatment in Neurological Disorder". W International Conference on Health Big Data and Intelligent Healthcare. SCITEPRESS - Science and Technology Publications, 2022. http://dx.doi.org/10.5220/0011269100003438.

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Aslam, Muhammad Arslan, Muhammad Zonain, Salman Muneer, Omar Sattar, Mohammad Salahat i Muhammad Saleem. "Neurological Disorder Detection Using OCT Scan Image of Eye". W 2022 International Conference on Cyber Resilience (ICCR). IEEE, 2022. http://dx.doi.org/10.1109/iccr56254.2022.9996016.

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Lellis, Caio de Almeida, Marco Alejandro Menacho Herbas, João Cesar Pereira da Cunha, Samyla Coutinho Paniago i Paulo Sérgio Machado Diniz. "Cannabidiol and the Management of Pediatric Neurological Disorders: A Systematic Review". W XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.266.

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Introduction: Due to an increase in cases of neurological disorders refractory to conventional treatments in pediatrics patients, other therapies have been sought. Objectives: To analyze the safety and efficacy of CBD in the management of neurological disorders in children. Design and setting: A systematic review conducted at the Pontifical Catholic University of Goiás. Methods: This is a systematic review carried out in PubMed, Lilacs and MedLine databases, with the descriptors: “(Pediatric OR Neurology) AND (Cannabidiol OR Cannabis)”, being selected only the randomized studies, meta-analysis and clinical trials published in the last 10 years. Results: One randomized trial concluded that daily oral CBD solution reduced the frequency of seizures in pediatric patients with Lennox-Gastaut syndrome (LGS). In addition, another study concluded that CBD (20 mg/kg/day) is associated with up to a 50% decrease in seizures in children with SLG and Dravet syndrome. Furthermore, it was pointed out that the association of oral CBD (2 to 5 mg/kg/day) with antiepileptic drugs caused a mean reduction of 36.5% of severe seizures. Finally, CBD enriched with 0.25% THC reduced spasticity, dystonia and pain intensity in children with complex motor disorder. Conclusion: CBD was shown to be safe and effective as a treatment for refractory epilepsies and complex motor disorder in pediatric patients, with no serious side effects reported.
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Zarkali, Angeliki, Mark Edwards i Mahinda Yogarajah. "42 Multi-agent allergies as predictor of functional neurological disorder". W The British Neuropsychiatry Association – Annual Meeting. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/jnnp-2019-bnpa.42.

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Mehmood, Aneesa, Liat Levita, Markus Reuber i Emily Mayberry. "44 Lifespan of negative experiences in functional neurological disorder patients". W The British Neuropsychiatry Association – Annual Meeting. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/jnnp-2019-bnpa.44.

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Verma, Anurag, i Devendra Kumar Chaturvedi. "Neurological disorder detection using EEG signal processing and Machine Learning". W 2023 International Conference on Recent Advances in Electrical, Electronics & Digital Healthcare Technologies (REEDCON). IEEE, 2023. http://dx.doi.org/10.1109/reedcon57544.2023.10151428.

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Raporty organizacyjne na temat "Neurological Disorder"

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Paul, Satashree. Autism Spectrum Disorder. Science Repository, luty 2021. http://dx.doi.org/10.31487/sr.blog.26.

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Jelsma, Dorothee, Reza Abdollahipour, Farhad Ghadiri, Fatemeh Alaei, Miriam Paloma Nieto, Zdenek Svoboda, Miguel Villa de Gregorio, Paola Violasdotter Nilsson, Dido Green i Kamila Banatova. Evidence-based practice interventions for children and young people with Developmental Coordination Disorder - A scoping review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, luty 2023. http://dx.doi.org/10.37766/inplasy2023.2.0028.

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Review question / Objective: The aim of this review was to identify, qualify, evaluate and synthesise interventions used for children and young people with Developmental Coordination Disorder. The PCC was used to develop the review question: Population – children and young people with disorders of motor coordination aged to 25 years, not due to neurological disease or disorder eg. Cerebral Palsy. Concept - any method aimed to improve/treat/intervene in areas of motor learning, motor control, motor coordination or motor skill. Context - information on methods of delivery of interventions to consider context and cultural factors influencing delivery as well as details of intervention timing and outcomes. Primary Question: What interventions are being used for children and young people with DCD? Secondary Questions: How are these interventions being implemented? What outcomes are evident?
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Bell, Ronald C. Diagnostic Exercise: Neurologic Disorder in a Cat. Fort Belvoir, VA: Defense Technical Information Center, grudzień 1989. http://dx.doi.org/10.21236/ada218031.

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Chae, May. A Needs Assessment for Adaptive Clothing: Women with Chronic Neurological Disorders. Ames (Iowa): Iowa State University. Library, styczeń 2019. http://dx.doi.org/10.31274/itaa.8250.

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Kuhl, D. E. New techniques for positron emission tomography in the study of human neurological disorders. Office of Scientific and Technical Information (OSTI), lipiec 1992. http://dx.doi.org/10.2172/10154585.

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Kuhl, D. E. New techniques for positron emission tomography in the study of human neurological disorders. Office of Scientific and Technical Information (OSTI), styczeń 1993. http://dx.doi.org/10.2172/6467222.

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Kuhl, D. E. New techniques for positron emission tomography in the study of human neurological disorders. Office of Scientific and Technical Information (OSTI), styczeń 1992. http://dx.doi.org/10.2172/5176116.

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Dr. Jogeshwar Mukherjee. Development of dopamine receptor radiopharmaceuticals for the study of neurological and psychiatric disorders. Office of Scientific and Technical Information (OSTI), styczeń 2009. http://dx.doi.org/10.2172/944919.

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Kuhl, D. New techniques for positron emission tomography in the study of human neurological disorders. Office of Scientific and Technical Information (OSTI), listopad 1989. http://dx.doi.org/10.2172/5361143.

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van Ginneken, Nadja, Simon Lewin i Vikram Patel. Do non-specialist health workers improve the care of people with mental, neurological and substance-use disorders? SUPPORT, 2017. http://dx.doi.org/10.30846/170213.

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Non specialist health workers (including doctors, nurses, lay health workers) who are not specialists in mental health or neurology, but who have some training in these fields, and other professionals, such as teachers, may have an important role to play in delivering mental, neurological or substance abuse care.
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