Gotowa bibliografia na temat „Neurodegenerative disease”
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Artykuły w czasopismach na temat "Neurodegenerative disease"
Homma, Hidenori, Hikari Tanaka, Kyota Fujita i Hitoshi Okazawa. "Necrosis Links Neurodegeneration and Neuroinflammation in Neurodegenerative Disease". International Journal of Molecular Sciences 25, nr 7 (24.03.2024): 3636. http://dx.doi.org/10.3390/ijms25073636.
Pełny tekst źródłaBakir, Sena, Gizem Catalkaya, Fatma D. Ceylan, Haroon Khan, Burcu Guldiken, Esra Capanoglu i Mohammad A. Kamal. "Role of Dietary Antioxidants in Neurodegenerative Diseases: Where are We Standing?" Current Pharmaceutical Design 26, nr 7 (25.03.2020): 714–29. http://dx.doi.org/10.2174/1381612826666200107143619.
Pełny tekst źródłaMarshall Moscon, Savannah L., i James R. Connor. "HFE Mutations in Neurodegenerative Disease as a Model of Hormesis". International Journal of Molecular Sciences 25, nr 6 (15.03.2024): 3334. http://dx.doi.org/10.3390/ijms25063334.
Pełny tekst źródłaAbitbol, Arjuna, Brody Mallard, Evelin Tiralongo i Joe Tiralongo. "Mushroom Natural Products in Neurodegenerative Disease Drug Discovery". Cells 11, nr 23 (6.12.2022): 3938. http://dx.doi.org/10.3390/cells11233938.
Pełny tekst źródłaPetrozzi, Lucia, Giulia Ricci, Noemi J. Giglioli, Gabriele Siciliano i Michelangelo Mancuso. "Mitochondria and Neurodegeneration". Bioscience Reports 27, nr 1-3 (13.06.2007): 87–104. http://dx.doi.org/10.1007/s10540-007-9038-z.
Pełny tekst źródłaBrkic, Marjana, Sriram Balusu, Claude Libert i Roosmarijn E. Vandenbroucke. "Friends or Foes: Matrix Metalloproteinases and Their Multifaceted Roles in Neurodegenerative Diseases". Mediators of Inflammation 2015 (2015): 1–27. http://dx.doi.org/10.1155/2015/620581.
Pełny tekst źródłaZhang, Minhua, Guangrui Luo, Yanjiao Zhou, Shaohui Wang i Zhong Zhong. "Phenotypic Screens Targeting Neurodegenerative Diseases". Journal of Biomolecular Screening 19, nr 1 (19.08.2013): 1–16. http://dx.doi.org/10.1177/1087057113499777.
Pełny tekst źródłaLemieszewska, Marta, Agnieszka Zabłocka i Joanna Rymaszewska. "Parkinson’s disease: Etiopathogenesis, molecular basis and potential treatment opportunities". Postępy Higieny i Medycyny Doświadczalnej 73 (15.05.2019): 256–68. http://dx.doi.org/10.5604/01.3001.0013.2021.
Pełny tekst źródłaBelyakin, Sergey, i Sergey Shuteev. "Application of a Time-Delay Model of the Plykin - Newhouse Attractor to Study the Dynamics of Neuro - Degeneration by Electroencephalography of the Brain". Psychology and Mental Health Care 6, nr 2 (22.01.2022): 01–06. http://dx.doi.org/10.31579/2637-8892/153.
Pełny tekst źródłaDoudet, D. J. "Neurodegenerative Disease". Molecular Imaging and Biology 9, nr 4 (20.04.2007): 159–60. http://dx.doi.org/10.1007/s11307-007-0099-y.
Pełny tekst źródłaRozprawy doktorskie na temat "Neurodegenerative disease"
Biro, Andrew J. "Specific aspects of neurodegenerative disease". Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/28919.
Pełny tekst źródłaMedicine, Faculty of
Anesthesiology, Pharmacology and Therapeutics, Department of
Graduate
Sleven, Hannah. "Models of neurodegenerative mitochondrial disease". Thesis, University of Oxford, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598048.
Pełny tekst źródłaRadakovic, Ratko. "Multidimensional apathy in neurodegenerative disease". Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25959.
Pełny tekst źródłaDury, R. J. "Understanding haemodynamics in neurodegenerative disease". Thesis, University of Nottingham, 2018. http://eprints.nottingham.ac.uk/50380/.
Pełny tekst źródłaVadnal, Jonathan. "Epigenetic Mechanisms in Neurodegenerative Disease". Kent State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=kent1353955013.
Pełny tekst źródłaGu, Mei. "Mitochondrial function in Parkinson's disease and other neurodegenerative diseases". Thesis, University College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322371.
Pełny tekst źródłaFranco, Iborra Sandra. "Mitochondrial quality control in neurodegenerative diseases: focus on Parkinson’s disease and Huntington’s disease". Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/565668.
Pełny tekst źródłaIn the past years, several important advances have expanded our understanding of the pathways that lead to cell dysfunction and death in Parkinson’s disease (PD) and Huntington’s disease (HD). Both diseases are movement disorders characterized by the loss of a specific subset of neurons within the basal ganglia, dopaminergic neurons in the substantia nigra pars compacta (SNpc), in the case of PD, and medium spiny neurons in the striatum, in the case of HD,. Despite distinct clinical and pathological features, these two neurodegenerative disorders share critical underlying pathogenic mechanisms such as the presence of misfolded and/or aggregated proteins, oxidative stress and mitochondrial anomalies. Mitochondria are the prime energy source in most eukaryotic cells, but these highly dynamic organelles are also involved in a multitude of cellular events. Disruption of mitochondrial homeostasis and the subsequent mitochondrial dysfunction plays a key role in the pathophysiology of neurodegenerative diseases. Therefore, maintenance of mitochondrial integrity through different surveillance mechanisms is critical for neuronal survival. In this thesis I have studied in depth some mitochondrial quality control mechanisms in the context of PD and HD, in order to broaden the knowledge about the pathomechanisms leading to cell death. In the first chapter I have studied mitochondrial protein import in in vitro and in vivo models of PD. In vitro, complex I inhibition, a characteristic pathological hallmark in PD, impaired mitochondrial protein import. This was associated with OXPHOS protein downregulation, accumulation of aggregated proteins inside mitochondria and downregulation of mitochondrial chaperones. Therefore, we aimed to reestablish the mitochondrial protein import by overexpressing two key components of the system: translocase of the outer membrane 20 (TOM20) and translocase of the inner membrane 23 (TIM23). Overexpression of TOM20 and TIM23 in vitro restored protein import into mitochondria and ameliorated mitochondrial dysfunction and cell death. Complex I inhibition also impaired mitochondrial protein import and led to dopaminergic neurodegeneration in vivo. Overexpression of TIM23 partially rescued protein import into mitochondria and slightly protected dopaminergic neurons in the SNpc. On the contrary, TOM20 overexpression did not rescue protein import into mitochondria and exacerbated neurodegeneration in both SNpc and striatum. These results highlight mitochondrial protein import dysfunction and the distinct role of two of their components in the pathogenesis of PD and suggest the need for future studies to target other elements in the system. In the second chapter, I have studied the role of huntingtin in mitophagy and how the polyglutamine expansion present in mutant huntingtin can affect its function. For such, I worked with differentiated striatal ST-Q7 (as control) and ST-Q111 (as mutant) cells, expressing full length huntingtin. In these conditions, induced mitophagy was not mediated by Parkin recruitment into depolarized mitochondria. Mutant huntingtin impaired induced mitophagy by altering wildtype huntingtin scaffolding activity at different steps of mitophagy process: (i) ULK1 activation through its release from the mTORC1, (ii) Beclin1-Vps15 complex formation, (iii) interaction of the mitophagy adapters OPTN and NDP52 with huntingtin and (iv) with LC3. As a result, mitochondria from ST-Q111 cells exhibited increased damage and altered mitochondrial respiration. These results uncover impaired mitophagy as a potential pathological mechanism linked with HD. In conclusion, we have discovered new mitochondrial targets for PD and HD emphasizing the important role that mitochondrial quality control plays in neurodegeneration
Ryan, Philip. "An Investigation Into Novel Molecular Strategies Targeting Neurodegenerative Diseases". Thesis, Griffith University, 2020. http://hdl.handle.net/10072/395102.
Pełny tekst źródłaThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Pharmacy and Pharmac
Griffith Health
Full Text
Blundell, James Michael. "Cognitive assessment of paediatric neurodegenerative disease". Thesis, University of Birmingham, 2015. http://etheses.bham.ac.uk//id/eprint/6042/.
Pełny tekst źródłaSTOCCORO, ANDREA. "Mitoepigenetics investigations in neurodegenerative diseases". Doctoral thesis, Università di Siena, 2019. http://hdl.handle.net/11365/1072183.
Pełny tekst źródłaKsiążki na temat "Neurodegenerative disease"
W, Langston J., i Young Anne B. 1947-, red. Neurotoxins and neurodegenerative disease. New York, N.Y: New York Academy of Sciences, 1992.
Znajdź pełny tekst źródłaAvila, Jesus, Jose J. Lucas i Felix Hernandez, red. Animal Models for Neurodegenerative Disease. Cambridge: Royal Society of Chemistry, 2011. http://dx.doi.org/10.1039/9781849732758.
Pełny tekst źródłaSaghir, Atif N. Free radicals in neurodegenerative disease. Birmingham: University of Birmingham, 1996.
Znajdź pełny tekst źródła1952-, Marwah J., i Teitelbaum Herman, red. Advances in neurodegenerative disorders. Scottsdale, Ariz: Prominent Press, 1998.
Znajdź pełny tekst źródłaBoard, National Radiological Protection, red. ELF electromagnetic fields and neurodegenerative disease. Didcot: NRPB, 2001.
Znajdź pełny tekst źródłaSingh, Sarika, i Neeraj Joshi, red. Pathology, Prevention and Therapeutics of Neurodegenerative Disease. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-0944-1.
Pełny tekst źródłaMauro, Fasano, red. The proteomic approach in neurodegenerative disease research. Trivandrum, Kerala, India: Research Signpost, 2007.
Znajdź pełny tekst źródłaMauro, Fasano, red. The proteomic approach in neurodegenerative disease research. Trivandrum, Kerala, India: Research Signpost, 2007.
Znajdź pełny tekst źródłaSelkoe, Dennis J., i Yves Christen, red. Immunization Against Alzheimer’s Disease and Other Neurodegenerative Disorders. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-59332-1.
Pełny tekst źródłavon, Bernhardi Rommy, i Inestrosa Nibaldo C, red. Neurodegenerative diseases: From molecular concepts to therapeutic targets. New York: Nova Biomedical Books, 2008.
Znajdź pełny tekst źródłaCzęści książek na temat "Neurodegenerative disease"
Clausen, Torben, José Luis Trejo, Mark P. Mattson, Alexis M. Stranahan, Joanna Erion, Rosa Maria Bruno, Stefano Taddei i Melinda M. Manore. "Neurodegenerative Disease". W Encyclopedia of Exercise Medicine in Health and Disease, 637–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_145.
Pełny tekst źródłaMeyer, Michael Andrew. "Neurodegenerative Diseases". W Neurologic Disease, 161–75. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39581-4_8.
Pełny tekst źródłaGallagher, Damien, Robert F. Coen i Brian A. Lawlor. "Alzheimer’s Disease". W Neurodegenerative Disorders, 57–74. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23309-3_4.
Pełny tekst źródłaOlszewska, Diana A., Stanley Fahn, Richard A. Walsh i Tim Lynch. "Parkinson’s Disease". W Neurodegenerative Disorders, 85–115. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23309-3_6.
Pełny tekst źródłaBurke, Tom, Colin P. Doherty, Walter Koroshetz i Niall Pender. "Huntington’s Disease". W Neurodegenerative Disorders, 167–79. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23309-3_9.
Pełny tekst źródłaGallagher, Damien J., Aine Ní Mhaolaín, Reisa A. Sperling i Brian A. Lawlor. "Alzheimer’s Disease". W Neurodegenerative Disorders, 43–64. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84996-011-3_3.
Pełny tekst źródłaWalsh, Richard A., Timothy Lynch i Stanley Fahn. "Parkinson’s Disease". W Neurodegenerative Disorders, 77–114. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84996-011-3_5.
Pełny tekst źródłaPender, Niall P., i Walter J. Koroshetz. "Huntington’s Disease". W Neurodegenerative Disorders, 167–79. London: Springer London, 2011. http://dx.doi.org/10.1007/978-1-84996-011-3_8.
Pełny tekst źródłaWeis, Serge, Michael Sonnberger, Andreas Dunzinger, Eva Voglmayr, Martin Aichholzer, Raimund Kleiser i Peter Strasser. "Neurodegenerative Diseases: Parkinson Disease". W Imaging Brain Diseases, 1001–20. Vienna: Springer Vienna, 2019. http://dx.doi.org/10.1007/978-3-7091-1544-2_37.
Pełny tekst źródłaWeis, Serge, Michael Sonnberger, Andreas Dunzinger, Eva Voglmayr, Martin Aichholzer, Raimund Kleiser i Peter Strasser. "Neurodegenerative Diseases: Huntington Disease". W Imaging Brain Diseases, 1059–68. Vienna: Springer Vienna, 2019. http://dx.doi.org/10.1007/978-3-7091-1544-2_40.
Pełny tekst źródłaStreszczenia konferencji na temat "Neurodegenerative disease"
Vidya, J., i Vamsidhar Yendapalli. "Comparative Analysis of Neurodegenerative disease detection using Artificial Intelligence". W 2024 International Conference on Knowledge Engineering and Communication Systems (ICKECS), 1–6. IEEE, 2024. http://dx.doi.org/10.1109/ickecs61492.2024.10616608.
Pełny tekst źródłaFaria, Gustavo Hugo de Souza. "The impact of epigenetics on the development of neurodegenerative diseases". W XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.654.
Pełny tekst źródłaSordillo, Laura A., Peter P. Sordillo, Lin Zhang i Robert R. Alfano. "Tryptophan and kynurenines in neurodegenerative disease". W Bio-Optics: Design and Application. Washington, D.C.: OSA, 2019. http://dx.doi.org/10.1364/boda.2019.jt4a.8.
Pełny tekst źródłaZhu, Xiangyun. "Role of MRI in neurodegenerative disease". W Radiopaedia 2023. Radiopaedia.org, 2023. http://dx.doi.org/10.53347/rposter-1636.
Pełny tekst źródłaRezende, Maria Clara Lopes, Maria Luiza Franco de Oliveira, Júlia Campos Fabri, Maria Júlia Filgueiras Granato, Mariana Vanon Moreira i Leandro Vespoli Campos. "Neuroprotective Effects of Creatine Supplementation in Neurodegenerative Diseases". W XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.234.
Pełny tekst źródłaBogdanović, Nenad, i Vladimira Vuletić. "Alzheimer disease and Alzheimer-like dementias". W Rijeka Forum on Neurodegenerative Diseases (2 ; 2018 ; Rijeka). Hrvatska akademija znanosti i umjetnosti, 2019. http://dx.doi.org/10.21857/m16wjc6j79.
Pełny tekst źródłaVuletić, Vladimira, i Nenad Bogdanović. "Dementia with Lewy bodies and Parkinson’s disease dementia". W Rijeka Forum on Neurodegenerative Diseases (2 ; 2018 ; Rijeka). Hrvatska akademija znanosti i umjetnosti, 2019. http://dx.doi.org/10.21857/m8vqrtzwv9.
Pełny tekst źródłaXavier, Lais de Paiva, Gabriella de Andrade Moreira, Luana Maria Amaral Cherain, Lucas Ferreira Flávio Souza, Tânia Maria da Silva Novaretti i Valdeci de Oliveira Santos Rigolin. "Preliminary study on the number of deaths from neurodegenerative diseases in former Brazilian soccer players." W XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.212.
Pełny tekst źródłaLei, Haijun, Yujia Zhao, Yuting Wen i Baiying Lei. "Adaptive Sparse Learning for Neurodegenerative Disease Classification". W 2017 IEEE International Symposium on Multimedia (ISM). IEEE, 2017. http://dx.doi.org/10.1109/ism.2017.51.
Pełny tekst źródłaLima, Aline Fonseca, Ana Luiza Almeida Carneiro, Bruna Torres Homem Fonseca, Alessandra Augusta Barroso Penna e. Costa, Fernanda Veiga Góes, Marcela Rodriguez de Freitas, Talys Jason Pinheiro, Tania Regina Dias Saad Salles i Ludimila Marins de Almeida Moura. "Neurodegenerative disease caused by the TRAPPC4 gene". W SBN Conference 2022. Thieme Revinter Publicações Ltda., 2023. http://dx.doi.org/10.1055/s-0043-1774598.
Pełny tekst źródłaRaporty organizacyjne na temat "Neurodegenerative disease"
Li, Jiawang, Xiangyun Chen, Hongrui Zhang, Wei Ding, Danni Chen, Ning Liang, Haiying Tong, Zhenhong Liu i Zhen Yang. A Systematic Review of the Capsaicin and other TRPV1 agonists effects on three neurodegenerative diseases: Alzheimer's disease, Parkinson's disease and Ischemic Stroke. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, kwiecień 2024. http://dx.doi.org/10.37766/inplasy2024.4.0115.
Pełny tekst źródłaDao, Kimberly. Relationship Between The Gut-Brain Axis, Parkinson's Disease, and Use of Canines as Animal Models in Neurodegenerative Disease Research. Ames (Iowa): Iowa State University, styczeń 2019. http://dx.doi.org/10.31274/cc-20240624-1212.
Pełny tekst źródłaMillhorn, David E. Signal Transduction and Gene Regulation During Hypoxic Stress: A Potential Role in Neurodegenerative Disease. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2001. http://dx.doi.org/10.21236/ada397765.
Pełny tekst źródłaMillhorn, David E. Signal Transduction and Gene Regulation During Hypoxia Stress: A Potential Role in Neurodegenerative Disease. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2002. http://dx.doi.org/10.21236/ada416979.
Pełny tekst źródłaMillhorn, David E. Signal Transduction and Gene Regulation During Hypoxic Stress: A Potential Role in Neurodegenerative Disease. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2000. http://dx.doi.org/10.21236/ada383039.
Pełny tekst źródłaRamos, Lauren. Therapeutic potential of cannabinoids in the management of Alzheimer's Disease and other neurodegenerative conditions. Ames (Iowa): Iowa State University, styczeń 2019. http://dx.doi.org/10.31274/cc-20240624-1505.
Pełny tekst źródłaSantos, Ana Lúcia Yaeko da Silva, Deyse Mayara Rodrigues Caron, Livia Shirahige i Abrahão Fontes Baptista. Alterations in Corticomotor Excitability in Amyotrophic Lateral Sclerosis: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, maj 2023. http://dx.doi.org/10.37766/inplasy2023.5.0078.
Pełny tekst źródłaLiu, Yangjun, Wei Xie, Zbigniew Ossowski, Juan Li, Juan Yang, Yiming Luo, Xia Wu i Liying Liu. Physical activity, abdominal obesity and inflammatory response in the elderly: a systematic review and meta-analysis of randomized-controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzec 2023. http://dx.doi.org/10.37766/inplasy2023.3.0051.
Pełny tekst źródłaFu, Chengwei, Ziqiang Xia, Menghan Feng, Zhicheng Zhou i Bo Liu. Comparative efficacy of noninvasive brain stimulation for the treatment of Parkinson disease: a systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzec 2022. http://dx.doi.org/10.37766/inplasy2022.3.0151.
Pełny tekst źródłaAndrades, Oscar, David Ulloa, Dario Martinez, Francisco Guede, Gustava Muñoz, Luis Javier Chirosa i Amador García. Effect of the manipulation of the variables that configure the stimulus of strength training on motor symptoms in people with Parkinson's disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, listopad 2022. http://dx.doi.org/10.37766/inplasy2022.11.0079.
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