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Artykuły w czasopismach na temat "(Na+,K+)-dependent ATPase"
Aizman, R. I., G. Celsi, L. Grahnquist, Z. M. Wang, Y. Finkel i A. Aperia. "Ontogeny of K+ transport in rat distal colon". American Journal of Physiology-Gastrointestinal and Liver Physiology 271, nr 2 (1.08.1996): G268—G274. http://dx.doi.org/10.1152/ajpgi.1996.271.2.g268.
Pełny tekst źródłaBenders, A. A., J. A. Timmermans, A. Oosterhof, H. J. Ter Laak, T. H. M. S. M. van Kuppevelt, R. A. Wevers i J. H. Veerkamp. "Deficiency of Na+/K+-ATPase and sarcoplasmic reticulum Ca2+-ATPase in skeletal muscle and cultured muscle cells of myotonic dystrophy patients". Biochemical Journal 293, nr 1 (1.07.1993): 269–74. http://dx.doi.org/10.1042/bj2930269.
Pełny tekst źródłaMathews, P. M., D. Claeys, F. Jaisser, K. Geering, J. D. Horisberger, J. P. Kraehenbuhl i B. C. Rossier. "Primary structure and functional expression of the mouse and frog alpha-subunit of the gastric H(+)-K(+)-ATPase". American Journal of Physiology-Cell Physiology 268, nr 5 (1.05.1995): C1207—C1214. http://dx.doi.org/10.1152/ajpcell.1995.268.5.c1207.
Pełny tekst źródłaMissiaen, L., F. Wuytack, H. De Smedt, M. Vrolix i R. Casteels. "AlF4- reversibly inhibits ‘P’-type cation-transport ATPases, possibly by interacting with the phosphate-binding site of the ATPase". Biochemical Journal 253, nr 3 (1.08.1988): 827–33. http://dx.doi.org/10.1042/bj2530827.
Pełny tekst źródłaAsano, Shinji, Satomi Hoshina, Yumi Nakaie, Toshiyuki Watanabe, Michihiko Sato, Yuichi Suzuki i Noriaki Takeguchi. "Functional expression of putative H+-K+-ATPase from guinea pig distal colon". American Journal of Physiology-Cell Physiology 275, nr 3 (1.09.1998): C669—C674. http://dx.doi.org/10.1152/ajpcell.1998.275.3.c669.
Pełny tekst źródłaKraut, J. A., F. Starr, G. Sachs i M. Reuben. "Expression of gastric and colonic H(+)-K(+)-ATPase in the rat kidney". American Journal of Physiology-Renal Physiology 268, nr 4 (1.04.1995): F581—F587. http://dx.doi.org/10.1152/ajprenal.1995.268.4.f581.
Pełny tekst źródłaCrambert, Gilles, Ciming Li, Dirk Claeys i Käthi Geering. "FXYD3 (Mat-8), a New Regulator of Na,K-ATPase". Molecular Biology of the Cell 16, nr 5 (maj 2005): 2363–71. http://dx.doi.org/10.1091/mbc.e04-10-0878.
Pełny tekst źródłaOno, S., J. Guntupalli i T. D. DuBose. "Role of H(+)-K(+)-ATPase in pHi regulation in inner medullary collecting duct cells in culture". American Journal of Physiology-Renal Physiology 270, nr 5 (1.05.1996): F852—F861. http://dx.doi.org/10.1152/ajprenal.1996.270.5.f852.
Pełny tekst źródłaVujisic, Ljubica, Danijela Krstic i Jovan Vucetic. "Chemical aspect of the influence of cobalt ions on atpase activity". Journal of the Serbian Chemical Society 65, nr 7 (2000): 507–15. http://dx.doi.org/10.2298/jsc0007507v.
Pełny tekst źródłaReinhardt, J., M. Kosch, M. Lerner, H. Bertram, D. Lemke i H. Oberleithner. "Stimulation of protein kinase C pathway mediates endocytosis of human nongastric H+-K+-ATPase, ATP1AL1". American Journal of Physiology-Renal Physiology 283, nr 2 (1.08.2002): F335—F343. http://dx.doi.org/10.1152/ajprenal.00226.2001.
Pełny tekst źródłaRozprawy doktorskie na temat "(Na+,K+)-dependent ATPase"
Hao, Jingping. "The electrical properties of Bufo marinus Na⁺, K⁺-ATPase". Ohio : Ohio University, 2009. http://www.ohiolink.edu/etd/view.cgi?ohiou1258151062.
Pełny tekst źródłaCheng, Sam Xian Jun. "Functional significance of phosphorylation of rat renal Na+,K+-ATPase by PKA and PKC /". Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-2971-8.
Pełny tekst źródłaNguyen, Khoa Thuy Diem. "Energy metabolism in the brain and rapid distribution of glutamate transporter GLAST in astrocytes". Thesis, The University of Sydney, 2008. http://hdl.handle.net/2123/3996.
Pełny tekst źródłaNguyen, Khoa Thuy Diem. "Energy metabolism in the brain and rapid distribution of glutamate transporter GLAST in astrocytes". University of Sydney, 2008. http://hdl.handle.net/2123/3996.
Pełny tekst źródłaGlutamate transporters play a role in removing extracellular excitatory neurotransmitter, L-glutamate into the cells. The rate of the uptake depends on the density of the transporters at the membrane. Some studies claimed that glutamate transporters could transit between the cytoplasm and the membrane on a time-scale of minutes. The present study examined the distribution of glutamate transporter GLAST predominantly expressed in rat cortical cultured astrocytes between the membrane and the cytoplasm by using deconvolution microscopy and then analyzing the images. The regulation of the distribution of GLAST was studied in the presence of glutamate transporter substrate (D-aspartate), purinergic receptor activators (α,β-methylene ATP, adenosine), neuroleptic drugs (clozapine, haloperidol), ammonia (hyperammonia) and Na+/K+-ATPase inhibitors (ouabain, digoxin and FCCP). It was demonstrated that the translocation of GLAST towards the plasma membrane was induced by D-aspartate, α,β-methylene ATP, adenosine, clozapine and ammonia (at 100 μM and very high concentrations of 10 mM). However, the inhibition of Na+/K+-ATPase activity had an opposite effect, resulting in redistribution of GLAST away from the membrane. It has previously been claimed that the membrane-cytoplasm trafficking of GLAST was regulated by phosphorylation catalysed by protein kinase C delta (PKC-delta). Involvement of this mechanism has, however, been put to doubt when rottlerin, a PKC-delta inhibitor, used to test the hypothesis showed to inhibit Na+/K+-ATPase-mediated uptake of Rb+, suggesting that rottlerin influenced the activity of Na+/K+-ATPase. As Na+/K+-ATPase converts ATP to energy and pumps Na+, K+ ions, thus helping to maintain normal electrochemical and ionic gradients across the cell membrane. Its inhibition also reduced D-aspartate transport and could impact on the cytoplasm-to-membrane traffic of GLAST molecules. Furthermore, rottlerin decreased the activity of Na+/K+-ATPase by acting as a mitochondrial inhibitor. The present study has focused on the inhibition of Na+/K+-ATPase activity by rottlerin, ouabain and digoxin in homogenates prepared from rat kidney and cultured astrocytes. The activity of Na+/K+-ATPase was measured by the absorption of inorganic phosphate product generated from the hydrolysis of ATP and the fluorescent transition of the dye RH421 induced by the movement of Na+/K+-ATPase. This approach has a potential to test whether the rottlerin effect on Na+/K+-ATPase is a direct inhibition of the enzyme activity. Rottlerin has been found to block the activity of Na+/K+-ATPase in a dose-dependent manner in both rat kidney and astrocyte homogenates. Therefore, rottlerin inhibited the activity of Na+/K+-ATPase directly in a cell-free preparation, thus strongly indicating that the effect was direct on the enzyme. In parallel experiments, ouabain and digoxin produced similar inhibitions of Na+/K+-ATPase activity in rat kidney while digoxin blocked the activity of Na+/K+-ATPase to a greater extent than ouabain in rat cortical cultured astrocytes. In a separate set of experiments, Na+/K+-ATPase in the astrocytic membrane was found to be unsaturated in E1(Na+)3 conformation in the presence of Na+ ions and this could explain the differences between the effects of digoxin and ouabain on the activity of Na+/K+-ATPase in rat astrocytes. In addition, it was found that at low concentrations of rottlerin, the activity of Na+/K+-ATPase was increased rather than inhibited. This effect was further investigated by studying rottlerin interactions with membrane lipids. The activity of Na+/K+-ATPase has been reported to be regulated by membrane lipids. The enzyme activity can be enhanced by increasing fluidity of the lipid membrane. I have, therefore, proposed that rottlerin binds to the membrane lipids and the effects of rottlerin on Na+/K+-ATPase are mediated by changes in the properties (fluidity) of the membrane. The hypothesis was tested by comparing rottlerin and a detergent, DOC (sodium deoxycholate), for their binding to the lipids by using a DMPC (1,2-Dimyristoyl-sn-Glycero-3-Phosphocholine) monolayer technique. DOC has been shown to both increase and inhibit activity of Na+/K+-ATPase in a manner similar to that displayed by rottlerin. The effects of rottlerin and DOC on the DMPC monolayers were studied by measuring the surface pressure of DMPC monolayers and surface area per DMPC molecule. I established that both rottlerin and DOC decreased the surface pressure of DMPC monolayers and increased the surface area per DMPC molecule. This indicates that both rottlerin and DOC penetrated into the DMPC monolayers. If rottlerin can interact with the lipids, changes in fluidity of the lipid membrane cannot be ruled out and should be considered as a possible factor contributing to the effects of rottlerin on the activity of Na+/K+-ATPase. Overall, the study demonstrates that rottlerin is not only a PKC-delta inhibitor but can have additional effects, both on the enzyme activities (Na+/K+-ATPase) and/or on lipid-containing biological structures such as membranes. The findings have implication not only for studies where rottlerin was used as a supposedly specific PKC-delta inhibitor but also for mechanisms of its toxicity.
Kuznetsova, Irina [Verfasser]. "Inhibition of the influenza A virus-induced, tubulin-dependent apical mislocalization of the Na+,K+-ATPase in infected cells: Improving vectorial water transport and pulmonary edema clearance / Irina Kuznetsova". Gießen : Universitätsbibliothek, 2020. http://d-nb.info/1216143633/34.
Pełny tekst źródłaSchneeberger, Anne. "Ionenbindung an die Na, K-ATPase". [S.l. : s.n.], 2000. http://deposit.ddb.de/cgi-bin/dokserv?idn=959955968.
Pełny tekst źródłaLiang, Man. "Na/K ATPase : signaling versus pumping". Toledo, Ohio : University of Toledo, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1173803261.
Pełny tekst źródła"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biomedical Sciences." Major advisor: Zi-Jian Xie. Includes abstract. Document formatted into pages: iii, 156 p. Title from title page of PDF document. Bibliography: pages 64-67, 97-100, 116-117, 125-155.
Tian, Jiang. "Na/K-ATPase : a signaling receptor". Connect to Online Resource-OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1175177603.
Pełny tekst źródła"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biomedical Sciences." Major advisor: Zi-Jian Xie. Includes abstract. Title from title page of PDF document. Bibliography: pages 64-70, 104-108, 121-158.
Aizman, Oleg. "Novel aspects of Na⁺,K⁺-ATPase /". Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-311-2.
Pełny tekst źródłaCarradus, Maria. "Structural studies of Na'+K'+-ATPase". Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342323.
Pełny tekst źródłaKsiążki na temat "(Na+,K+)-dependent ATPase"
Chakraborti, Sajal, i Naranjan S. Dhalla, red. Regulation of Membrane Na+-K+ ATPase. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-24750-2.
Pełny tekst źródłaHorisberger, Jean-Daniel. The Na,K-ATPase: Structure-function relationship. Austin: R.G. Landes, 1994.
Znajdź pełny tekst źródłaInternational Conference on the Na/K-ATPase and Related ATPases (9th 1999 Sapporo, Japan). Na/K-ATPase and related ATPases: Proceedings of the 9th International Conference on the Na/K-ATPase and Related ATPases, Sapporo, Japan, 18-23 August 1999. Redaktorzy Kaya Shunji i Taniguchi Kazuya 1966-. Amsterdam: Elsevier, 2000.
Znajdź pełny tekst źródłaJ, Shearer M., i Seghatchian M. J, red. Vitamin K and vitamin K-dependent proteins: Analytical, physiological, and clinical aspects. Boca Raton, Fla: CRC Press, 1993.
Znajdź pełny tekst źródłaGaretz, Susan Lynn. Variation in expression of Na+K+ATPase ł and ø subunit mRNAs in rat tissues and nervous system cell lines. [New Haven: s.n.], 1989.
Znajdź pełny tekst źródłaInternational, Conference on Na K.-ATPase. The sodium pump: Proceedings of the Fourth International Conference on Na, K-ATPase held at the Physiological Laboratory, Cambridge, in August 1984. Cambridge: Company of Biologists, 1985.
Znajdź pełny tekst źródłaIan, Glynn, Ellory J. C i Company of Biologists, red. The sodium pump: Proceedings of the Fourth International Conference on Na, K-ATPase, held at the Physiological Laboratory, Cambridge, in August 1984. Cambridge, U.K: Company of Biologists, 1985.
Znajdź pełny tekst źródłaYasunobu, Suketa, i Excerpta Medica (Firm), red. Control and diseases of sodium dependent transport proteins and ion channels: Proceedings of the First International Conference held in Shizuoka, Japan, 24-28th August 1999. Amsterdam: Elsevier, 2000.
Znajdź pełny tekst źródłaDhalla, Naranjan S., i Sajal Chakraborti. Regulation of Membrane Na+-K+ ATPase. Springer, 2015.
Znajdź pełny tekst źródłaChakraborti, Sajal, i Naranjan S. Dhalla. Regulation of Membrane Na+-K+ ATPase. Springer, 2015.
Znajdź pełny tekst źródłaCzęści książek na temat "(Na+,K+)-dependent ATPase"
Spieker, C., K. H. Rahn i W. Zidek. "Age-dependent secretion of a Na+ K+ ATPase inhibitor in spontaneously hypertensive rats". W Arteriosklerotische Gefäßerkrankungen, 410–16. Wiesbaden: Vieweg+Teubner Verlag, 1992. http://dx.doi.org/10.1007/978-3-663-19646-4_47.
Pełny tekst źródłaObradovic, Milan, Julijana Stanimirovic, Anastasija Panic, Bozidarka Zaric i Esma R. Isenovic. "Na+/K+-ATPase". W Encyclopedia of Signaling Molecules, 3338–43. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101543.
Pełny tekst źródłaObradovic, Milan, Julijana Stanimirovic, Anastasija Panic, Bozidarka Zaric i Esma R. Isenovic. "Na +/K+-ATPase". W Encyclopedia of Signaling Molecules, 1–6. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101543-1.
Pełny tekst źródłaScheiner-Bobis, G. "Na+/K+-ATPase". W Encyclopedia of Molecular Pharmacology, 1–10. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-21573-6_267-1.
Pełny tekst źródłaScheiner-Bobis, G. "Na+/K+-ATPase". W Encyclopedia of Molecular Pharmacology, 1062–71. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57401-7_267.
Pełny tekst źródłaLee, Chin Ok, i David C. Gadsby. "PKA Activation in Cardiac Myocytes Affects the Voltage Dependence of Na-K ATPase Pump and Na-Ca Exchange Currents Differently". W The Primo Vascular System, 271–83. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0601-3_37.
Pełny tekst źródłaDelamere, Nicholas A., i Shigeo Tamiya. "Lens Na+, K+-ATPase". W Ocular Transporters In Ophthalmic Diseases And Drug Delivery, 111–23. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-375-2_6.
Pełny tekst źródłade Pont, Jan Joep H. H. M., Jan B. Koenderink, Herman G. P. Swarts i Peter H. G. M. Willems. "Structure-Activity Relationships of Na,K-ATPase and H,K-ATPase". W Mechanisms and Consequences of Proton Transport, 71–82. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0971-4_7.
Pełny tekst źródłaSachs, George, i Björn Wallmark. "The gastric H, K ATPase". W Proton Pump Inhibitors, 23–45. Basel: Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-8795-3_2.
Pełny tekst źródłaChakraborti, Sajal, Sayed Modinur Rahaman, Md Nur Alam, Amritlal Mandal, Biswarup Ghosh, Kuntal Dey i Tapati Chakraborti. "Na+/K+-ATPase: A Perspective". W Regulation of Membrane Na+-K+ ATPase, 3–30. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-24750-2_1.
Pełny tekst źródłaStreszczenia konferencji na temat "(Na+,K+)-dependent ATPase"
Grzesik, Benno, Ramona Ruhl, Nieves Gabrielli, Werner Seeger, Wolfgang Kummer, Uwe Pfeil i Istvan Vadasz. "Intermedin Enhances Alveolar Edema Clearance By Promoting Na,K-ATPase Exocytosis In A Protein Kinase A-Dependent But CAMP-Independent Manner". W American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3521.
Pełny tekst źródłaGusarova, GA, LA Dada, A. Briva, H. Trejo i JI Sznajder. "Hypoxia Leads to Ca2+/Calmodulin-Dependent Kinase Kinase β (CaMKKβ) Activation/AMPK Phosphorylation, Na,K-ATPase Endocytosis and Impaired Alveolar Epithelial Reabsorbtion." W American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1927.
Pełny tekst źródłaHack, N., J. M. Wilkinson i N. Crawford. "A MONOCLONAL ANTIBODY (PL/IM 430) THAT BLOCKS THE ACTIVE TRANSL0CATI0N OF Ca2+ INTO HUMAN PLATELET INTRACELLULAR MEMBRANE (ER) VESICLES". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644678.
Pełny tekst źródłaFujimoto, T., B. Djuricic, K. Tanoue, Y. Fukushima i H. Yamazaki. "CHANGES IN ENZYMATIC ACTIVITIES IN BRAIN CAPILLARY ENDOTHELIAL CELLS INJURED BY PLATELET AGGREGATION IN VIVO". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643368.
Pełny tekst źródłaLestari, Silvia W., Aucky Hinting, Hamdani Lunardi, Debby Aditya, Dessy Noor Miati i Meidika Dara Rizki. "Sperm Na+,K+-ATPase and dynein ATPase activity: A study of embryo development in in vitro fertilization (IVF)". W SECOND INTERNATIONAL CONFERENCE OF MATHEMATICS (SICME2019). Author(s), 2019. http://dx.doi.org/10.1063/1.5096752.
Pełny tekst źródłaOnken, Horst. "V-ATPase and Na+/K+-ATPase energize postprandial fluid absorption from the isolated midgut of female yellow fever mosquitoes (Aedes aegypti)". W 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93236.
Pełny tekst źródłaKrastev, Dragomir, Shudong Li, Yilun Sun, Yves Pommier, Kristijan Ramadan i Christopher J. Lord. "Abstract P057: The ubiquitin-dependent ATPase p97 removes cytotoxic trapped PARP1 from chromatin". W Abstracts: AACR-NCI-EORTC Virtual International Conference on Molecular Targets and Cancer Therapeutics; October 7-10, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1535-7163.targ-21-p057.
Pełny tekst źródłaYang, Liqun, Olga Lopina, Daniel McStay, A. A. Boldyrev, Alan J. Rogers i Peter J. Quinn. "Time-resolved phosphorescence measurement of conformational transitions in cation transport Na+, K+-ATPase". W OE/LASE'93: Optics, Electro-Optics, & Laser Applications in Science& Engineering, redaktorzy Laurence A. Nafie i Henry H. Mantsch. SPIE, 1993. http://dx.doi.org/10.1117/12.145246.
Pełny tekst źródłaLi, Zhiqin, Alisa Litan, Seung Joon Lee, Bruce Graves, Sonali P. Barwe i Sigrid A. Langhans. "Abstract 707: Distinct roles of Na,K-ATPase function and expression in medulloblastoma". W Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-707.
Pełny tekst źródłaLestari, Silvia W., Manggiasih D. Larasati, Asmarinah i Indra G. Mansur. "Sperm Na+, K+-ATPase and Ca2+-ATPase activity: A preliminary study of comparison of swim up and density gradient centrifugation methods for sperm preparation". W 2ND BIOMEDICAL ENGINEERING’S RECENT PROGRESS IN BIOMATERIALS, DRUGS DEVELOPMENT, AND MEDICAL DEVICES: Proceedings of the International Symposium of Biomedical Engineering (ISBE) 2017. Author(s), 2018. http://dx.doi.org/10.1063/1.5023963.
Pełny tekst źródłaRaporty organizacyjne na temat "(Na+,K+)-dependent ATPase"
Colton, Joel S., Sara C. Gilman i Carol A. Colton. Effect of the Thiol-Oxidizing Agent, Diamide, on Cerebral Cortical Na(+) -K(+) ATPase. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1988. http://dx.doi.org/10.21236/ada205311.
Pełny tekst źródłaAubert, B. Measurement of Time-Dependent CP-Violating Asymmetries in B0 ->K*0 gamma (K*0->K0{sub S} pi0) Decays. Office of Scientific and Technical Information (OSTI), czerwiec 2004. http://dx.doi.org/10.2172/826956.
Pełny tekst źródłaAubert, B. Measurements of Branching Fractions and Time-Dependent CP-Violating Asymmetries in B to eta' K Decays. Office of Scientific and Technical Information (OSTI), luty 2005. http://dx.doi.org/10.2172/839753.
Pełny tekst źródłaGronau, M. Interpreting the Time-Dependent CP Asymmetry in B{sup 0} {yields} {pi}{sup 0} K{sub S}. Office of Scientific and Technical Information (OSTI), listopad 2003. http://dx.doi.org/10.2172/826518.
Pełny tekst źródłaIlic, J. Time-dependent Dalitz-Plot Analysis of the Charmless Decay B^0 -> K^0S Pi Pi- at BABAR. Office of Scientific and Technical Information (OSTI), październik 2009. http://dx.doi.org/10.2172/968531.
Pełny tekst źródłaPalombo, F. Measurement of Time-Dependent CP-Violating Asymmetries in B{sup 0} Meson Decays to eta' K{sup 0}. Office of Scientific and Technical Information (OSTI), sierpień 2004. http://dx.doi.org/10.2172/829746.
Pełny tekst źródłaAubert, B. Ambiguity-Free Measurement of cos2beta: Time-Intergrated and Time-Dependent Angular Analyses of B to J/psi K pi. Office of Scientific and Technical Information (OSTI), listopad 2004. http://dx.doi.org/10.2172/839607.
Pełny tekst źródłaT'Jampens, Stephane. Study of CP Symmetry Violation in the Charmonium-K*(892) Channel By a Complete Time Dependent Angular Analysis (BaBar Experiment). Office of Scientific and Technical Information (OSTI), wrzesień 2006. http://dx.doi.org/10.2172/891862.
Pełny tekst źródłaJoseph Marion Tuggle IV. Radiative B Meson Decay as a Probe of Physics Beyond the Standard Model: Time-Dependent CP Violation in B0 → K0S π0 γ and the B → Φ K γ Branching Fraction. Office of Scientific and Technical Information (OSTI), styczeń 2009. http://dx.doi.org/10.2172/946450.
Pełny tekst źródłaBiassoni, Pietro. Measurements of Time-Dependent CP-Asymmetry Parameters in B Meson Decays to η' K0 and of Branching Fractions of SU(3) Related Modes with BaBar Experiment at SLAC. Office of Scientific and Technical Information (OSTI), styczeń 2009. http://dx.doi.org/10.2172/946451.
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